Relevant bibliographies by topics / Mechanism of infection

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Mechanism of infection'

Author: Grafiati
Published: 4 June 2025
Last updated: 1 August 2025

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Journal articles on the topic "Mechanism of infection"

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Palca, Joseph. "Infection mechanism?" Nature 319, no. 6050 (1986): 170. http://dx.doi.org/10.1038/319170b0.

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Jones, L., B. D. Braithwaite, B. Davies, B. P. Heather, and J. J. Earnshaw. "Mechanism of Late Prosthetic Vascular Graft Infection." Cardiovascular Surgery 5, no. 5 (1997): 486–89. http://dx.doi.org/10.1177/096721099700500511.

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This study was a retrospective analysis of 41 patients with late prosthetic graft infections (> 30 days after operation) from six hospitals in the south-west of England. The 41 patients had a median age of 66 years and generally accepted risk factors for infection were documented in 19 patients preoperatively. Thirteen patients had postoperative wound complications and three had early reoperation at the site of subsequent infection. The median time between index operation and symptoms of infection was 10 (range 1–224) months. Abscess (46%) was the most common presentation followed by false
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Dahiya, Sunita, Suman Kumari, Payal Rani, Suneel Kumar Onteru, and Dheer Singh. "Postpartum uterine infection & ovarian dysfunction." Indian Journal of Medical Research 148, Suppl 1 (2018): S64—S70. https://doi.org/10.4103/ijmr.ijmr_961_18.

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Postpartum uterine infections such as metritis, endometritis and mastitis have been considered as underlying causes for ovarian dysfunction in mammals. Almost all mammals, particularly dairy animals are susceptible to postpartum uterine infections, resulting in impaired fertility and economic loss. One of the factors for low fertility in females is ovarian dysfunction, which is exhibited as impaired growth and function of ovarian follicles by the postpartum infection. Immune system of mammals provides a host defence mechanism against pathogenic microbes through the recognition of pathogen-asso
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Yasukawa, Masaki. "3. Etiological Mechanism (Infection)." Nihon Naika Gakkai Zasshi 97, no. 7 (2008): 1531–36. http://dx.doi.org/10.2169/naika.97.1531.

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Tudor, Cristina Anca, Cristian Boros, Raluca Petre, Adriana Elena Nica, and Christina Chatzifilippidou. "The immune response in Acinetobacter baumannii pneumonia." Romanian Journal of Infectious Diseases 19, no. 1 (2016): 16–21. http://dx.doi.org/10.37897/rjid.2016.1.3.

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Acinetobacter baumannii is a bacterium that is commonly causes of nosocomial infections, the most common site of infection and colonization is the lower respiratory tract. Although it is present more often in immunocompromised patients, the defense mechanism against infection with Acinetobacter baumanii remains incomplete elucidated. Among the virulence factors involved in infection with Acinetobacter baumanii are production and release of exopolysaccharide, and ability to biofilm formation in tissues. Understanding of virulence mechanisms is important for early initiation of treatment.
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NOMOTO, KIKUO. "Host defense mechanism against infection." Nishi Nihon Hifuka 47, no. 3 (1985): 415–28. http://dx.doi.org/10.2336/nishinihonhifu.47.415.

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SUZUKI, Yasuo, and Takashi SUZUKI. "Mechanism of Influenza Virus Infection." Kagaku To Seibutsu 35, no. 5 (1997): 339–45. http://dx.doi.org/10.1271/kagakutoseibutsu1962.35.339.

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Yamada, Douglas, Heidi Elsaesser, and David Brooks. "Impaired antibody effector function during persistent viral infection (VIR7P.1049)." Journal of Immunology 192, no. 1_Supplement (2014): 208.1. http://dx.doi.org/10.4049/jimmunol.192.supp.208.1.

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Abstract Persistent viral infections are associated with dysfunctional B cell responses including polyclonal B cell activation, non-virus-specific hypergammaglobulinemia, and ultimately the inability to control infection. Using the lymphocytic choriomeningitis virus (LCMV) system of murine infection, we addressed whether similar to other antiviral mechanisms, the suppressive immune environment generated during persistent infection impacted antibody effector function. We demonstrate that high levels of circulating antibodies generated during persistent infection (termed hypergammaglobulinemia)
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Luo, Geyang, Bo Yan, and Yinzhong Shen. "The impact and mechanism of HIV infection on tuberculous granuloma formation." AIDS 39, no. 9 (2025): 1095–105. https://doi.org/10.1097/qad.0000000000004216.

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The co-infection of Mycobacterium tuberculosis (MTB) and HIV continues to pose a major challenge to healthcare systems. Currently, the effects of HIV infection on tuberculous granulomas are not fully understood. This review discusses the impact of HIV infection on the formation and function of tuberculous granulomas, highlighting key immunological mechanisms and the interactions between HIV and MTB infections. The co-infection results in atypical granulomas with weakened immune defenses, which facilitate the dissemination of MTB and accelerate the progression of tuberculosis. Additionally, thi
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Wu, Jian, Huiqing Wang, Ze Xiang, et al. "Role of viral hepatitis in pregnancy and its triggering mechanism." Journal of Translational Internal Medicine 12, no. 4 (2024): 344–54. http://dx.doi.org/10.2478/jtim-2024-0015.

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Abstract Hepatitis viral infection can cause severe complications, even mortality in pregnant women and their offspring. Multiple studies have shown that vertical transmission can cause viral hepatitis infections in newborns, especially in hepatitis B, C, and E. Screening for hepatitis viral infection in pregnant women is essential. Once infected, pregnant women should be given timely antiviral treatments, which could effectively alleviate the disease progression and reduce adverse outcomes. Besides, the mechanism of viral hepatitis mediating adverse pregnancy outcomes has been a hot topic. He
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Dissertations / Theses on the topic "Mechanism of infection"

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Fernandes, Isabel Oliveira. "Infection mechanism of Diplodia corticola." Doctoral thesis, Universidade de Aveiro, 2015. http://hdl.handle.net/10773/15487.

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Doutoramento em Biologia<br>Diplodia corticola is regarded as the most virulent fungus involved in cork oak decline, being able to infect not only Quercus species (mainly Q. suber and Q. ilex), but also grapevines (Vitis vinifera) and eucalypts (Eucalyptus sp.). This endophytic fungus is also a pathogen whose virulence usually manifests with the onset of plant stress. Considering that the infection normally culminates in host death, there is a growing ecologic and socio-economic concern about D. corticola propagation. The molecular mechanisms of infection are hitherto largely unknown. Accordi
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Shammakhi, Sahar. "The Infection and Uncoating Mechanism of the Giant Melbournevirus." Thesis, Uppsala universitet, Institutionen för biologisk grundutbildning, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-428745.

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Since their 'discovery' at the turn of the 21st century, giant viruses of the amoeba have captured the fascination of virologists. They have raised a plethora of questions regarding their evolution and ecological significance and have greatly defied a century's old definition of viruses. By now, it is understood that a handful of giant viruses enter the amoeba via the phagosomal pathway. This thesis chooses to focus on the giant Melbournevirus (MelV) regarding its entry and uncoating pathway. We now conclude that the initial attachment between MelV and amoeba cells is built upon glycan interac
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Charsou, Chara. "Autophagy as a control mechanism in human papilloma virus infection." Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/25878.

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Human Papilloma Virus (HPV) is a conserved DNA virus, which infects mucosal and cutaneous epithelia. Although over 200 types of HPV have been identified which can infect humans, only around 15 high-risk (HR) types have been shown to be responsible for the development of cancer. HPV-16 is the most abundant HR-HPV type being responsible for almost 70% of cervical cancers. HPV-16 consists of 8 genes, the early genes (E1, E2, E4, E5 and the potential oncogenes E6 and E7) responsible for the infection, amplification and proliferation and the late genes (L1 and L2) responsible for the packaging and
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Scala, Carlo. "Mechanism of DC-SIGN mediated enhancement of HIV-1 infection." Thesis, King's College London (University of London), 2015. http://kclpure.kcl.ac.uk/portal/en/theses/mechanism-of-dcsign-mediated-enhancement-of-hiv1-infection(1cf4fa70-8144-4828-810c-e28900c962bb).html.

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This thesis describes an investigation of the mechanisms by which DC-SIGN mediates enhancement of HIV-1 infection. Mannose-binding C-type lectin receptors, expressed on Langerhans cells and subepithelial dendritic cells (DCs), play an important role in HIV-1capture and subsequent dissemination to lymph nodes. DC-SIGN mediates both productive infection of DCs and trans-infection of CD4+ T cells that occurs in the absence of replication. The molecular events involved in transmission have not been fully defined. In this study, surface plasmon resonance analyses demonstrated that DC-SIGN, but not
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Mitchell, Peter James. "Inappropriate Regulation as a Mechanism for Persistence of Helicobacter Pylori Infection." Thesis, Imperial College London, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.486573.

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Ogbechi, J. "Investigating the mechanism behind the tissue necrosis in Mycobacterium ulcerans infection." Thesis, University of Surrey, 2017. http://epubs.surrey.ac.uk/813238/.

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Buruli ulcer (BU) is a progressive necrotising skin disease caused by Mycobacterium ulcerans. The bacteria produce a lipid virulence factor, mycolactone which is responsible for the tissue necrosis seen in the disease but the trigger for this is not known. Endothelial cells play a critical role in maintaining tissue homeostasis but prior to this thesis, the effect of mycolactone on endothelial cells had not been investigated. Mycolactone caused a reduction in the expression of proteins involved in the regulation of clotting as well as adhesion molecules in endothelial cells. The mycolactone-me
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Asakura, Makoto. "Studies on metabolic function required for infection mechanism of Colletotrichum lagenarium." Kyoto University, 2007. http://hdl.handle.net/2433/136577.

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Kyoto University (京都大学)<br>0048<br>新制・課程博士<br>博士(農学)<br>甲第13454号<br>農博第1665号<br>新制||農||949(附属図書館)<br>学位論文||H19||N4310(農学部図書室)<br>UT51-2007-S485<br>京都大学大学院農学研究科応用生物科学専攻<br>(主査)教授 奥野 哲郎, 教授 遠藤 隆, 准教授 田中 千尋<br>学位規則第4条第1項該当
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Sneyd, Hannah, and Hannah Sneyd. "Influenza A: Mechanism of Infection and Development of M2 Ion Channel Inhibitors." Thesis, The University of Arizona, 2017. http://hdl.handle.net/10150/626385.

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Influenza viral infection causes several hospitalizations and claims the lives of many people each year. The threat of epidemic and pandemic are more pressing than ever with newly mutated strains developing every year. Understanding the mechanism of infection of influenza can help identify new potential drug targets and help progress the development of antivirals. Currently there are two classes of FDA approved drugs, neuraminidase inhibitors and M2 ion channel inhibitors, to combat influenza infection. Unfortunately, viral resistance to M2 ion channel blockers has caused them to stop being us
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Sundaravaradan, Vasudha. "Molecular Mechanism of HIV-1 Infection: Role of Viral and Host Determinants." Diss., Tucson, Arizona : University of Arizona, 2006. http://etd.library.arizona.edu/etd/GetFileServlet?file=file:///data1/pdf/etd/azu%5Fetd%5F1685%5F1%5Fm.pdf&type=application/pdf.

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Leung, Hiu-lan Nancy, and 梁曉灡. "Mechanism of antibody-dependent enhancement in severe acute respiratory syndrome coronavirus infection." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B47327066.

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Severe lymphopenia is a clinical feature of Severe Acute Respiratory Syndrome (SARS) patients. However, lymphocytes do not express receptor for SARS-CoV, neither the widely accepted viral receptor angiotensin converting enzyme 2 (ACE2) nor the putative receptors Dendritic Cell- and Liver/lymph-Specific Intercellular adhesion molecule-3-Grabbing Non-integrin (DC-SIGN and L-SIGN). Our group previously showed in vitro that, SARS-CoV Spike pseudotyped particles (SARSCoVpp) could infect human B cells only when inoculated in presence of anti-SARSCoV Spike immune serum. Such observations rais
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Books on the topic "Mechanism of infection"

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Sinclair, Stephen Blair. Mechanism of the protective effect of prostagladin E2 in murine hepatitis virus strain 3 infection. National Library of Canada, 1991.

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Gerald, Keusch, and Farthing M. J. G, eds. Enteric infection: Mechanisms, manifestations and management. Chapman and Hall Medical, 1989.

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G, Farthing M. J., and Keusch Gerald, eds. Enteric infection: Mechanisms, manifestations, and management. Raven Press, 1989.

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Sasakawa, Chihiro, ed. Molecular Mechanisms of Bacterial Infection via the Gut. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-01846-6.

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J, Rolff, and Reynolds Stuart Edward, eds. Insect infection and immunity: Evolution, ecology, and mechanisms. Oxford University Press, 2009.

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Genco, C. A. Neisseria: Molecular mechanisms of pathogenesis. Caister Academic Press, 2010.

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H, Hunt Richard, Tytgat G. N. J, and Axcan Pharma, eds. Helicobacter pylori: Basic mechanisms to clinical cure 2002. Kluwer Academic, 2003.

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Giuseppe, Barbanti-Brodano, Bendinelli Mauro, and Friedman Herman 1931-, eds. DNA tumor viruses: Oncogenic mechanisms. Plenum Press, 1995.

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University, Johns Hopkins, and United States. Army Medical Research and Development Command, eds. Immunologic mechanisms of HTLV-III infection: Role of autoimmunity in AIDS. U.S. Army Medical Research and Development Command, 1992.

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Zemskov, Vladimir, Veronika Zemskova, Andrey Mironov, and Sergey Suchkov. Clinical immunology and allergology. INFRA-M Academic Publishing LLC., 2023. http://dx.doi.org/10.12737/1048793.

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The textbook "Clinical Immunology and Allergology" covers modern data on the phenomena, mechanisms of development, physiological changes in immunological reactivity, metabolic immunity, as well as on the diagnosis, prevention and treatment of immunodeficiency, allergies, immunosuppressive and immunoassociated non-infectious and infectious diseases. It consists of eight sections, 33 chapters, intermediate and final control tests, an electronic handbook of methods for assessing immune status, infections, immunotropic, antiallergic and other medications.&#x0D; Meets the requirements of the federa
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Book chapters on the topic "Mechanism of infection"

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Lemonnier, Jérôme, and Nicolas Lemonnier. "The Mechanism of Viral Infection." In The Marathon of the Messenger. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-39300-6_4.

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Lang, C. H. "Mechanism of Insulin Resistance in Infection." In Pathophysiology of Shock, Sepsis, and Organ Failure. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-76736-4_44.

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Wang, Ge, and Diane E. Taylor. "Mechanism of Antibiotic Resistance in Helicobacter pylori." In Helicobacter pylori Infection and Immunity. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0681-2_6.

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Lehmann-Grube, Fritz. "Mechanism of Recovery from Acute Virus Infection." In Modern Trends in Virology. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73745-9_7.

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Upadhyay, Vinod, and Elangbam Premabati Devi. "Mechanism of Microbial Infection in Vegetables Diseases." In The Vegetable Pathosystem. Apple Academic Press, 2019. http://dx.doi.org/10.1201/9780429022999-11.

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Kiatyingangsulee, Thawanrut, Shabbir Simjee, Rungtip Chuanchuen, Faye Swinbourne, and Fergus Allerton. "Antibiotic Mechanism of Action and Resistance." In Infection Control in Small Animal Clinical Practice. CABI, 2023. http://dx.doi.org/10.1079/9781789244977.0016.

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Chambers, R. M. "The mechanism of infection in the urethra, prostate and epididymis." In Common Infections. Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-4878-5_19.

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Yang, Jinkui, Lianming Liang, Chenggang Zou, and Ke-Qin Zhang. "Molecular Mechanism of Nematophagous Fungi Infection of Nematodes." In Nematode-Trapping Fungi. Springer Netherlands, 2014. http://dx.doi.org/10.1007/978-94-017-8730-7_6.

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Salazar, Norman, Juan A. Rodriguez-Aguilar, and Josep Ll Arcos. "An Infection-Based Mechanism in Large Convention Spaces." In Coordination, Organizations, Institutions and Norms in Agent Systems V. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-14962-7_18.

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Manju and Pranav Kumar Prabhakar. "Targeting Molecular and Cellular Mechanism in Rhinovirus Infection." In Targeting Cellular Signalling Pathways in Lung Diseases. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-33-6827-9_22.

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Conference papers on the topic "Mechanism of infection"

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Kane, R. D., and P. Surinach. "A Field Study of Microbiological Growth and Reservoir Souring." In CORROSION 1997. NACE International, 1997. https://doi.org/10.5006/c1997-97208.

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Abstract The souring of normally sweet production systems is a significant problem which can have implications to continued oilfield operations. Such problems are commonly approached by gathering of field sample and laboratory analysis or by simple test kits. This paper describes an alternative approach which includes the use of specialized field sampling and analysis procedures and portable equipment that can be move from site to site. A case study is presented that illustrates the use of these procedures and equipment. In the present case, it was learned that whereas souring was occurring in
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Syed, S. Syed Abdul, E. Syed Mohamed, A. Nazreen, C. Tamilarasi, P. Joy Kirupa, and I. Karthiga. "AI-Based Infectious-Disease Surveillance Monitoring Using Koipe Mechanism." In 2025 International Conference on Computing and Communication Technologies (ICCCT). IEEE, 2025. https://doi.org/10.1109/iccct63501.2025.11020310.

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Mitkovskaya, O. A. "Hepatitis and allergic process – immune interaction." In General question of world science. НИЦ "LJournal", 2023. http://dx.doi.org/10.18411/gqws-10-2023-04.

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Studies of the interaction effect in patients with infectious hepatitis, other infections with subsequent development of allergic inflammation (allergic nosological entities, atopic form of bronchial asthma) were going for several years. In clinical and epidemiological studies, two types of the effect of infection on immunity were studied: inhibition of the development of allergic diseases or enhancement of the mechanism of allergy progression. An inverse correlation was revealed between bacterial infections and atopic mechanisms of development of allergic diseases and bronchial asthma
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Long, Changjiang, Huan Qi, and Sheng-He Huang. "Modeling the Mechanism of HBV Infection." In 2010 International Conference on Biomedical Engineering and Computer Science (ICBECS). IEEE, 2010. http://dx.doi.org/10.1109/icbecs.2010.5462433.

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Novak, M. D., O. V. Evdokimova, and A. I. Novak. "THE CORRECTNESS FOR THE USE OF CONCEPTS AND TERMS IN EPIDEMIOLOGY AND PARASITOLOGY." In THEORY AND PRACTICE OF PARASITIC DISEASE CONTROL. VNIIP – FSC VIEV, 2025. https://doi.org/10.31016/978-5-6053355-1-1.2025.26.223-227.

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The Russian scientific literature on the epizootic and epidemic process in parasitic and infectious diseases has been studied. The general patterns of the epizootic and epidemic process have been confirmed using the methodology of comparative analysis of the transmission mechanism, pathways, and factors of zoonotic infectious and invasive pathogens. A comparison of the fundamentally sound terminology used in epizootology and epidemiology which developed before the 70s and 80s of the 20th century and which is currently in use allowed us to identify a number of erroneous interpretations and inac
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Elbashir, Israa, Heba Al Khatib, and Hadi Yassine. "Replication Dynamics, Pathogenicity, and Evolution of Influenza Viruses in Intestinal Caco-2 Cells." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0166.

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Background: Influenza virus is a major cause of respiratory infections worldwide. Besides the common respiratory symptoms, namouras cases with gastrointestinal symptoms have been reported. Moreover, influenza virus has been detected in feces of up to 20.6 % of influenza-infected patients. Therefore, direct infection of intestinal cells with influenza virus is suspected; however, the mechanism of this infection has not been explored. AIM: To investigate influenza virus replication, cellular responses to infection, and virus evolution following serial infection in human Caucasian colon adenocarc
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Salazar, Norman, Juan A. Rodriguez-Aguilar, and Josep Lluis Arcos. "An Infection-Based Mechanism for Self-Adaptation in Multi-agent Complex Networks." In 2008 Second IEEE International Conference on Self-Adaptive and Self-Organizing Systems (SASO). IEEE, 2008. http://dx.doi.org/10.1109/saso.2008.65.

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Lusher, J. M., L. M. Aledort, M. Hiltgartner, J. Mosley, and E. Operskalski. "TRANSMISSION OF HUMAN IMMUNODEFICIENCY VIRUS INFECTION TO HOUSEHOLD CONTACTS OF PERSONS WITH CONGENITAL HEMATOLOGIC DISORDERS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644679.

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The Transfusion Safety Study is collecting data concerning the transmission of transfusion-acquired infections from patients with congenital hematologic disorders to household members. Of 233 patients for whom information is presently available, 128 (55%) were anti-HIV-positive. The 128 positive patients lived in 123 households with 174 members; 16 contacts were positive by EIA and immunoblot.These data provide further evidence of relatively high risk of HIV infection of sexual contacts. The three anti-HIV-positive children are all infants born to anti-HIV-positive wivesof infected hemophiliac
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Maltri, Rebecca, Fernanda Peronti Marino, Jorge Ygor Gonçalves dos Santos, et al. "Brain stroke in patients with Covid-19 disease." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.625.

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Background: Currently, the world is facing a pandemic caused by the SARSCoV-2, which has already infected millions of people and has accumulated countless deaths. Association has been reported between severe conditions of this disease and the occurrence of neurological manifestations, including stroke. The mechanisms that trigger this cerebrovascular disease in infected people are not defined yet, but it’s believed that they involve inflammatory reaction, vascular endothelial dysfunction and hypercoagulopathy present in SARS-CoV-2 infection. Objectives: Verify the studies which relate the cere
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Xie, Jinyang, and Guifang Xie. "Research and Implementation of Virus Infection Detection Mechanism Based on Brute-Force Algorithm." In 2023 IEEE International Conference on Control, Electronics and Computer Technology (ICCECT). IEEE, 2023. http://dx.doi.org/10.1109/iccect57938.2023.10140924.

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Reports on the topic "Mechanism of infection"

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Sjogren, Maria H., and Kent Holtzmuller. Hepatitis C Virus Infection: Mechanism of Disease Progression. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada406083.

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Sjogren, Maria H. Hepatitis C. Virus Infection: Mechanism of Disease Progression. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada433067.

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Citovsky, Vitaly, and Yedidya Gafni. Suppression of RNA Silencing by TYLCV During Viral Infection. United States Department of Agriculture, 2009. http://dx.doi.org/10.32747/2009.7592126.bard.

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The Israeli isolate of Tomato yellow leaf curl geminivirus (TYLCV-Is) is a major tomato pathogen, causing extensive (up to 100%) crop losses in Israel and in the south-eastern U.S. (e.g., Georgia, Florida). Surprisingly, however, little is known about the molecular mechanisms of TYLCV-Is interactions with tomato cells. In the current BARD project, we have identified a TYLCV-Is protein, V2, which acts as a suppressor of RNA silencing, and showed that V2 interacts with the tomato (L. esculentum) member of the SGS3 (LeSGS3) protein family known to be involved in RNA silencing. This proposal will
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Lapidot, Moshe, and Vitaly Citovsky. molecular mechanism for the Tomato yellow leaf curl virus resistance at the ty-5 locus. United States Department of Agriculture, 2016. http://dx.doi.org/10.32747/2016.7604274.bard.

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Tomato yellow leaf curl virus (TYLCV) is a major pathogen of tomato that causes extensive crop loss worldwide, including the US and Israel. Genetic resistance in the host plant is considered highly effective in the defense against viral infection in the field. Thus, the best way to reduce yield losses due to TYLCV is by breeding tomatoes resistant or tolerant to the virus. To date, only six major TYLCV-resistance loci, termed Ty-1 to Ty-6, have been characterized and mapped to the tomato genome. Among tomato TYLCV-resistant lines containing these loci, we have identified a major recessive quan
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Splitter, Gary A., Menachem Banai, and Jerome S. Harms. Brucella second messenger coordinates stages of infection. United States Department of Agriculture, 2011. http://dx.doi.org/10.32747/2011.7699864.bard.

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Aim 1: To determine levels of this second messenger in: a) B. melitensiscyclic-dimericguanosinemonophosphate-regulating mutants (BMEI1448, BMEI1453, and BMEI1520), and b) B. melitensis16M (wild type) and mutant infections of macrophages and immune competent mice. (US lab primary) Aim 2: To determine proteomic differences between Brucelladeletion mutants BMEI1453 (high cyclic-dimericguanosinemonophosphate, chronic persistent state) and BMEI1520 (low cyclicdimericguanosinemonophosphate, acute virulent state) compared to wild type B. melitensisto identify the role of this second messenger in esta
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Joel, Daniel M., John C. Steffens, and Alfred M. Mayer. Host-Elicited Germination and Mechanism of Penetration in Broomrape (Orobanche Spp.). United States Department of Agriculture, 1993. http://dx.doi.org/10.32747/1993.7568107.bard.

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Orobanche is an important parasitic weed. For developing novel methods for its control, a thorough understanding of crucial stages of its development is needed. Therefore, the objectives of this project were characterization of Orobanche germination stimulants, analysis of mechanisms of haustorial penetration, and characterization and isolation of penetration enzymes. The first highly potent natural germination stimulant for Orobanche was isolated from sunflower and identified by high-field 1D (1H and 13C), 2D (1H-1H COSY, HMQC, HMBC)-NMR, GC.FT-IR, and GC.MS as costuslactone, a guaiane type s
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Yogev, David, Ricardo Rosenbusch, Sharon Levisohn, and Eitan Rapoport. Molecular Pathogenesis of Mycoplasma bovis and Mycoplasma agalactiae and its Application in Diagnosis and Control. United States Department of Agriculture, 2000. http://dx.doi.org/10.32747/2000.7573073.bard.

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Mycoplasma bovis and M. agalactiae are two phylogenetically related mycoplasmas which cause economically significant diseases in their respective bovine or small ruminant hosts. These organisms cause persistent asymptomatic infections that can result in severe outbreaks upon introduction of carrier animals into susceptible herds. Little is known about the mechanisms underlying mycoplasma-host interaction, variation in virulence, or of the factors enabling avoidance of the host immune system. In recent years it has become apparent that the ability of pathogenic microorganisms to rapidly alter s
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Whitham, Steven A., Amit Gal-On, and Tzahi Arazi. Functional analysis of virus and host components that mediate potyvirus-induced diseases. United States Department of Agriculture, 2008. http://dx.doi.org/10.32747/2008.7591732.bard.

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The mechanisms underlying the development of symptoms in response to virus infection remain to be discovered in plants. Insight into symptoms induced by potyviruses comes from evidence implicating the potyviral HC-Pro protein in symptom development. In particular, recent studies link the development of symptoms in infected plants to HC-Pro's ability to interfere with small RNA metabolism and function in plant hosts. Moreover, mutation of the highly conserved FRNK amino acid motif to FINK in the HC-Pro of Zucchini yellow mosaic virus (ZYMV) converts a severe strain into an asymptomatic strain,
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Cheepsunthorn, Poonlarp, and Yong Poovorawan. Identification of receptors for H5N1 virus on human nerve cells using protromics-based approaches. Chulalongkorn University, 2013. https://doi.org/10.58837/chula.res.2013.12.

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In this study, we investigated neuroinfectious capacity of H5N1 (A/Thailand/NK165/2005) isolated from plasma of infected individual during the third wave of Thailand outbreaks using human neuroblastoma SH-SY5Y cells. This was due to lack of information regarding neuroinfectivity of this variant. Results demonstrated that H5N1/NK165 induced servere CPEs in these neuronal cells. H5N1-specific hemagglutinin was found in the cytoplasm of the infected cells as early as 12 h post-infection. By 24 h post-infection, all cells in the cultures were infected. These findings coincided with time course of
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Mawassi, Munir, and Valerian Dolja. Role of RNA Silencing Suppression in the Pathogenicity and Host Specificity of the Grapevine Virus A. United States Department of Agriculture, 2010. http://dx.doi.org/10.32747/2010.7592114.bard.

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RNA silencing is a defense mechanism that functions against virus infection and involves sequence-specific degradation of viral RNA. Diverse RNA and DNA viruses of plants encode RNA silencing suppressors (RSSs), which, in addition to their role in viral counterdefense, were implicated in the efficient accumulation of viral RNAs, virus transport, pathogenesis, and determination of the virus host range. Despite rapidly growing understanding of the mechanisms of RNA silencing suppression, systematic analysis of the roles played by diverse RSSs in virus biology and pathology is yet to be completed
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