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Books on the topic 'Mechanism of infection'

Author: Grafiati

Published: 4 June 2025

Last updated: 1 August 2025

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1

Sinclair, Stephen Blair. Mechanism of the protective effect of prostagladin E2 in murine hepatitis virus strain 3 infection. National Library of Canada, 1991.

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2

Gerald, Keusch, and Farthing M. J. G, eds. Enteric infection: Mechanisms, manifestations and management. Chapman and Hall Medical, 1989.

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3

G, Farthing M. J., and Keusch Gerald, eds. Enteric infection: Mechanisms, manifestations, and management. Raven Press, 1989.

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4

Sasakawa, Chihiro, ed. Molecular Mechanisms of Bacterial Infection via the Gut. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-01846-6.

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5

J, Rolff, and Reynolds Stuart Edward, eds. Insect infection and immunity: Evolution, ecology, and mechanisms. Oxford University Press, 2009.

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6

Genco, C. A. Neisseria: Molecular mechanisms of pathogenesis. Caister Academic Press, 2010.

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7

H, Hunt Richard, Tytgat G. N. J, and Axcan Pharma, eds. Helicobacter pylori: Basic mechanisms to clinical cure 2002. Kluwer Academic, 2003.

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8

Giuseppe, Barbanti-Brodano, Bendinelli Mauro, and Friedman Herman 1931-, eds. DNA tumor viruses: Oncogenic mechanisms. Plenum Press, 1995.

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9

University, Johns Hopkins, and United States. Army Medical Research and Development Command, eds. Immunologic mechanisms of HTLV-III infection: Role of autoimmunity in AIDS. U.S. Army Medical Research and Development Command, 1992.

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10

Zemskov, Vladimir, Veronika Zemskova, Andrey Mironov, and Sergey Suchkov. Clinical immunology and allergology. INFRA-M Academic Publishing LLC., 2023. http://dx.doi.org/10.12737/1048793.

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The textbook "Clinical Immunology and Allergology" covers modern data on the phenomena, mechanisms of development, physiological changes in immunological reactivity, metabolic immunity, as well as on the diagnosis, prevention and treatment of immunodeficiency, allergies, immunosuppressive and immunoassociated non-infectious and infectious diseases. It consists of eight sections, 33 chapters, intermediate and final control tests, an electronic handbook of methods for assessing immune status, infections, immunotropic, antiallergic and other medications. Meets the requirements of the federa

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11

1946-, Matsumori Akira, ed. Cardiomyopathies and heart failure: Biomolecular, infectious, and immune mechanisms. Kluwer Academic Publishers, 2003.

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12

S, Paul Prem, Francis David H, Benfield David A, and International Rushmore Conference on Mechanisms in the Pathogenesis of Enteric Diseases (1st : 1995 : Rapid City, S.D.), eds. Mechanisms in the pathogenesis of enteric diseases. Plenum Press, 1997.

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13

H, Hunt Richard, Tytgat G. N. J, and Axcan Pharma, eds. Helicobacter pylori: Basic mechanisms to clinical cure, 1998 : the proceedings of a symposium. Kluwer Academic Publishers, 1998.

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14

Peters, C. J., and Charles H. Calisher, eds. Infectious Diseases from Nature: Mechanisms of Viral Emergence and Persistence. Springer Vienna, 2005. http://dx.doi.org/10.1007/3-211-29981-5.

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15

Gosztonyi, Georg, ed. The Mechanisms of Neuronal Damage in Virus Infections of the Nervous System. Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-662-10356-2.

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16

Pai, Manjunath P., Jennifer J. Kiser, Paul O. Gubbins, and Keith A. Rodvold, eds. Drug Interactions in Infectious Diseases: Mechanisms and Models of Drug Interactions. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-72422-5.

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17

S, Paul Prem, Francis David H, and International Rushmore Conference on Mechanisms in the Pathogenesis of Enteric Diseases (2nd : 1998 : Rapid City, S.D.), eds. Mechanisms in the pathogenesis of enteric diseases 2. Kluwer Academic/Plenum Publishers, 1999.

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18

Porter, Marty Harold. Mechanism and mediators of the anorexia during bacterial infection. 1998.

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19

Kosova, A. A., U. U. Kutlaeva, E. V. Fedorova, et al. Epidemiology of HIV infection and prevention directions. SIB-Expertise, 2021. http://dx.doi.org/10.12731/er0474.12072021.

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This electronic educational resource is intended for sixth-year students of the Faculty of Medicine and Prevention, but it can also be used to train medical students of other specialties, in order to form the knowledge, skills and abilities necessary for a specialist to work in the field of prevention of infections with a contact mechanism of transmission (HIV infection) to ensure the sanitary and epidemiological well-being of the population. The EOR consists of 2 disciplinary modules containing theoretical and practical material, as well as test control. The theoretical part is represented by

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20

Steiner, Lisa A. Infections of the Hand. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0047.

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Hand infections can be delineated by type and location of infection, by a polymicrobial vs single microbial colonization, and by the type of organism. They are most often caused by superficial injury or trauma. Early identification and timely treatment can significantly improve the morbidity associated with hand infections. In addition to determining the source and mechanism of infection, it is important to identify tetanus immunization status, prior injury to the affected area, immune status, occupation, and hand dominance. Some hand infections (eg, paronychia, felon, herpetic whitlow, and ce

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21

Wijdicks, Eelco F. M., and Sarah L. Clark. Antimicrobial Therapy for Central Nervous System Infections. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190684747.003.0011.

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Broad-spectrum antibiotics—those that are bactericidal and able to penetrate the blood–brain barrier—must be administered to any patient with a suspected infection of the central nervous system (CNS). Antimicrobials should be selected with consideration of spectrum of activity, pharmacokinetics, mechanism of action, and need to be administered in a timely fashion. This chapter discusses antibiotics that provide good penetration of the central nervous system, and appropriate doses for these infections are outlined. Antiviral therapy for CNS infections is limited but complex. Antimicrobial thera

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22

Guerrero Manriquez, Gloria G., ed. Anti-infective Research and Development: Updates on Infection Mechanisms and Treatments. BENTHAM SCIENCE PUBLISHERS, 2020. http://dx.doi.org/10.2174/97898114695891200201.

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23

Guerrero, Gloria G. Anti-Infective Research and Development: Updates on Infection Mechanisms and Treatments. Bentham Science Publishers, 2020.

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24

Guerrero, Gloria G. Anti-Infective Research and Development: Updates on Infection Mechanisms and Treatments. Bentham Science Publishers, 2020.

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25

Guerrero, Gloria G. Anti-Infective Research and Development: Updates on Infection Mechanisms and Treatments. Bentham Science Publishers, 2020.

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26

Longmore, Murray, Ian B. Wilkinson, Andrew Baldwin, and Elizabeth Wallin. Renal medicine. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199609628.003.0007.

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Introduction to nephrologyUrineUrinary tract infection (uti)Acute kidney injury (aki):DiagnosisManagementChronic kidney disease (ckd)ManagementRenal replacement therapy (rrt)Glomerulonephritis (gn)Nephrotic syndromeDiuretics and their mechanism of actionInterstitial nephritides and nephrotoxins...

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27

Nambiar, Puja, and William R. Short. Mechanisms of HIV Transmission. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0003.

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HIV is a sexually transmitted infection. Most new HIV infections in the United States are the result of sex, but it is rare for HIV to be transmitted through oral sex. The risk of HIV transmission to a receptive partner remains higher than that to an insertive one; however, both are at risk. Anything that compromises the integrity of mucous membranes, such as sexually transmitted infections, may increase the risk of transmission. Although not 100% effective, keeping an infected partner’s viral load low reduces the risk of transmission to an HIV-negative partner. Maternal transmission is a larg

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28

Vodopivec, Ivana, and Tracey A. Cho. Neurobiology of Transverse Myelitis and Infectious Myelopathies. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0153.

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Infectious agents cause spinal cord pathology by three different mechanisms: direct invasion/infection of neural tissues (i.e., infective myelitis), secondary inflammation and tissue bystander damage with or without autoimmune pathogenesis (parainfectious myelitis), or involvement of extra-axial structures (including the pia-arachnoid, the dura, the epidural space, or the adjacent spinal bones or intervertebral discs), resulting in compressive or ischemic myelopathy. This chapter describes the pathogenesis and treatment of these disorders.

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29

Host Response Mechanisms in Infectious Diseases (Contributions to Microbiology). S. Karger Publishers (USA), 2002.

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30

Nolan, T. J., T. B. Nutman, and G. A. Schad. Strongyloidosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0064.

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Strongyloidosis is an intestinal parasitism caused by the threadworm, Strongyloides stercoralis. The parasite, occurring in dogs, primates and man, is found throughout the moist tropics, as well as in temperate areas where poor sanitation or other factors facilitate the occurrence of faecally transmitted organisms. In some parts of the world, notably Africa and New Guinea, human infections caused by S. fülleborni have been reported. In Africa, the latter is primarily a parasite of primates, but in New Guinea, no animal host is known. S. stercoralis is unique among zoonotic nematodes, in that l

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31

McCulloh, Russell J., and Steven M. Opal. Drug-induced depression of immunity in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0290.

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Immunosuppressive drugs are among the fastest-growing category of drugs in medicine today, both in terms of new medication development and in terms of use. Glucocorticoids, calcineurin inhibitors, and biological agents, among others, are used in a variety of diseases. . Although often of great benefit to patients, immunosuppressive agents also pose significant long-term risks for opportunistic infection. These drugs can also blunt normal host responses to infection, and patients receiving immunosuppressive medications are at high risk for severe illness, sepsis, and death from opportunistic in

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32

Farthing, M. J. G., and Gerald T. Keusch. Enteric Infection Mechanisms Manifestations and Management. Raven Pr, 1989.

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33

Kaushik, Sanket, and Nagendra Singh, eds. Current Developments in the Detection and Control of Multi Drug Resistance. BENTHAM SCIENCE PUBLISHERS, 2022. http://dx.doi.org/10.2174/97898150498791220101.

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The rise in the incidence of infections is caused by multi drug resistant (MDR) bacteria, it is essential to elucidate the basic mechanism of antibiotic resistance to discover effective methods for diagnosis and treatment of infections. The use of pathogen-specific probes offers a faster alternative for pathogen detection and could improve the diagnosis of infection. High resolution melting analysis techniques are useful for the detection of multi drug resistant pathogens. Rational Structural Based Drug Design is a common method to identify a lead compound and take it forward for further devel

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34

Hasbun, Rodrigo, Richard Dunham, Joseph S. Kass, et al. HIV-Associated Neurocognitive Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0038.

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HIV causes a chronic form of encephalitis (HIVE) that is clinically characterized by either dementia or mild neurocognitive impairment. Since the introduction of antiretroviral therapy in 1996, the incidence of HIV dementia has decreased by 50%, but the prevalence of mild neurocognitive disorder has increased up to 39%. HIVE is the result of direct microglial infection, interruption of trophic factors, or caused by inflammatory cytokines. HIV enters the brain primarily by the “Trojan horse mechanism”; it is carried by monocytes and lymphocytes that cross the blood–brain barrier. HIV has a pred

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35

Chandra, R. K., and P. M. Newberne. Nutrition, Immunity, and Infection: Mechanisms of Interactions. Springer, 2012.

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36

Chandra, R. K., and P. M. Newberne. Nutrition, Immunity, and Infection: Mechanisms of Interactions. Springer, 2012.

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37

Wang, Yufeng, Zhan Zhou, and Jianying Gu, eds. Evolutionary Mechanisms of Infectious Diseases. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88966-985-1.

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38

Cole, Michael F. Unifying Microbial Mechanisms: Shared Strategies of Pathogenesis. CRC Press LLC, 2019.

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39

Cole, Michael F. Unifying Microbial Mechanisms: Shared Strategies of Pathogenesis. CRC Press LLC, 2019.

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40

Cole, Michael. Unifying Microbial Mechanisms: Shared Strategies of Pathogenesis. Taylor & Francis Group, 2019.

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41

Cole, Michael. Unifying Microbial Mechanisms: Shared Strategies of Pathogenesis. Taylor & Francis Group, 2019.

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42

Rolff, Jens, and Stuart Reynolds. Insect Infection and Immunity: Evolution, Ecology, and Mechanisms. Oxford University Press, 2009.

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43

Sasakawa, Chihiro. Molecular Mechanisms of Bacterial Infection via the Gut. Springer, 2012.

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44

Molecular Mechanisms Of Bacterial Infection Via The Gut. Springer, 2009.

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45

Lalvani, Ajit, and Katrina Pollock. Defences against infection. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0303.

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The immune system is classified into a series of component parts, each specialized to defend the host against infection. Cells of the innate immune system are distributed throughout the body, in the tissues, and in the circulation, to defend against the first signs of danger, combining the acute inflammatory response with the ability to kill and remove invading pathogens. Monocytes, macrophages, and neutrophils phagocytose and kill exogenous and endogenous targets, using both oxygen-dependent and oxygen-independent mechanisms. The adaptive immune system creates a structurally specific and prol

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46

Herwald, H., ed. Host Response Mechanisms in Infectious Diseases. S. Karger AG, 2002. http://dx.doi.org/10.1159/isbn.978-3-318-00904-0.

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47

Hedges, Dawson W., Shawn D. Gale, and Lance D. Erickson, eds. Infectious Diseases in Neurocognitive and Neuropsychiatric Medicine. Oxford University PressOxford, 2024. http://dx.doi.org/10.1093/oso/9780192870414.001.0001.

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Abstract Neurocognitive and neuropsychiatric disorders contribute to a substantial amount of global morbidity. Unfortunately, available treatments for these diseases are often less than optimal. As such, identification of the causes of neurocognitive and neuropsychiatric disorders with goals of better understanding underlying pathophysiology and preventive strategies are paramount. Infectious Diseases in Neurocognitive and Neuropsychiatric Medicine describes the role of infectious diseases in neurocognitive and neuropsychiatric disorders, including possible mechanisms by which infectious disea

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48

Török, M. Estée, Fiona J. Cooke, and Ed Moran. Antibiotics. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199671328.003.0002.

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This chapter provides a systematic summary of antibiotics, primarily those used in bacterial infections, grouped by class and mechanism of action. Each summary provides information on the mode of action, mechanisms of resistance, pharmacology, and clinical use.

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49

Covid-19 Loci of Infection: Transport Pathways and Mechanisms. Indy Pub, 2020.

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50

Currier, Angela R. Interferon-induced effector mechanisms in murine Chlamydia psittaci infection. 1998.

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