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Books on the topic 'Media pathology'

Author: Grafiati

Published: 24 April 2022

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1

United States. Otitis Media Guideline Panel. Otitis media with effusion in young children. Rockville, Md: Agency for Health Care Policy and Research, Public Health Service, US Department of Health and Human Services, 1994.

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2

Parker, James N., and Philip M. Parker. Otitis media: A medical dictionary, bibliography and annotated research guide to Internet references. San Diego, CA: ICON Health Publications, 2004.

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3

Joyce, Emerson, ed. Does speech and language therapy work?: A review of the literature. London: Whurr, 1995.

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4

Maslov, A. Smertʹ ne postavila tochku: Rassledovanii︠a︡ sudebnogo medika. Moskva: Sampo, 1999.

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5

Maslov, Aleksandr Vasilʹevich. Smertʹ ne postavila tochku: Rassledovanii︠a︡ sudebnogo medika. Moskva: Sampo, 1999.

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6

L, Ochoa José, ed. Carpal tunnel syndrome and other disorders of the median nerve. Boston: Butterworth-Heinemann, 1993.

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7

Epizooties, Office International des. Animaux, pathologie et environnement =: Animals, pathogens and the environment = Animales, patología y medio ambiente. Paris: Office International des Epizooties, 1991.

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8

Gaag, Anna Van der. Communication and adults with learning disabilities: New map of an old country. London: Whurr, 1993.

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9

Entomology & palynology. Philadelphia, Pa: Mason Crest Publishers, 2014.

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10

Walker, Maryalice. Entomology and palynology: Evidence from the natural world. Philadelphia: Mason Crest Publishers, 2006.

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11

Schnorrenberger, Claus C. Pocket atlas of tongue diagnosis: With Chinese therapy guidelines for acupuncture, herbs, and nutrition. 2nd ed. Stuttgart: Thieme, 2011.

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12

Ric, Harnsberger H., ed. Imaging of the temporal bone. 3rd ed. New York: Thieme, 1998.

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13

Imaging of the temporal bone. New York: Thieme, 1986.

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14

Hugh, Selby, ed. The Aftermath of death: Coronials, law, pathology, counsellors, safety, media. Annandale, NSW: Federation Press, 1992.

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15

E, Stool Sylvan, Bluestone Charles D. 1932-, and Pittsburgh Otitis Media Research Center., eds. Studies in otitis media: Pittsburgh Otitis Media Research Center progress report, 1988. St. Louis, MO: Annals Pub. Co., 1988.

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16

United States. Otitis Media Guideline Panel, ed. Otitis media with effusion in children. Rockville, Md: U.S. Dept. of Health and Human Services, Public Health Service, Agency for Health Care Policy and Research, 1995.

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17

1925-, Stool Sylvan E., and Otitis Media Guideline Panel, eds. Otitis media with effusion in young children. Rockville, Md: U.S. Dept. of Health and Human Services, 1994.

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18

Stage, Carsten, and Tina Thode Hougaard. Language of Illness and Death on Social Media: An Affective Approach. Emerald Publishing Limited, 2018.

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19

Stage, Carsten, and Tina Thode Hougaard. Language of Illness and Death on Social Media: An Affective Approach. Emerald Publishing Limited, 2018.

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20

The Language of Illness and Death on Social Media: An Affective Approach. Emerald Publishing, 2018.

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21

Murder and the Making of English CSI. Johns Hopkins University Press, 2016.

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22

Honjo, Iwao, Goro Mogi, and Tetsuo Ishii. Recent Advances in Otitis Media. Demos Medical Publishing, 1994.

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23

Th, Balzer, Hamm Bernd Prof Dr, and Niendorf H. P, eds. Contrast agents in liver imaging. Dordrecht: Kluwer Academic, 1995.

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24

D, Martini Alessandro M., Read A. P, and Stephens Dafydd, eds. Genetics and hearing impairment. London: Whurr Publishers, 1996.

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25

Genetics and hearing impairment. London: Whurr, 1996.

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26

Martini, Alessandro, Dai Stephens, and Andrew Reed. Genetics and Hearing Impairment. Whurr Publishers, 1996.

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27

Recent advances in otitis media: Proceedings of the second extraordinary international symposium, March 31-April 3, 1993. Amsterdam: Kugler Publications, 1993.

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28

Samuel, Meerbaum, and Meltzer Richard S. 1921-, eds. Myocardial contrast two-dimensional echocardiography. Dordrecht: Kluwer Academic Publishers, 1989.

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29

Emerson, Joyce, Emerson, and Pamela Enderby. Does Speech and Language Therapy Work? Wiley, 1995.

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30

Carpal Tunnel Syndrome and Other Disorders of the Median Nerve. 2nd ed. Butterworth-Heinemann, 2002.

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31

Bertram, Edward H. Temporal Lobe Epilepsy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0038.

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Temporal lobe epilepsy, as discussed in this chapter, is a focal epilepsy that involves primarily the limbic structures of the medial temporal lobe (amygdala, hippocampus, and entorhinal cortex). In recent years animal models have been developed that mirror the pathology and pathophysiology of this disease. This chapter reviews the human condition, the structural and physiological changes that support the development of seizures. The neural circuitry of seizure initiation will be reviewed with a goal of creating a framework for developing more effective treatments for this disease.

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32

Coyne, Sarah M., and Jamie M. Ostrov, eds. The Development of Relational Aggression. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190491826.001.0001.

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The Development of Relational Aggression provides a rich and detailed literature review on developmental processes associated with the perpetration of relational aggression (and related terms of indirect aggression and social aggression) across childhood, adolescence, and emerging adulthood (with a brief mention of relational aggression in adulthood). Relational aggression is defined as behavior that is intended to harm another’s relationships or feelings of inclusion in a group. Unlike physical aggression, the scars of relational aggression are more difficult to see. However, victims (and aggressors) may experience strong and long-lasting consequences, including reduced self-esteem, loneliness, substance use, eating pathology, depression, and anxiety. This volume begins by providing an overview of the field, including a discussion of definitions, developmental trajectories, methodology, and theoretical approaches. Additionally, the volume examines the biobehavioral and evolutionary processes associated with this type of behavior. The book also examines a number of risk factors and socializing agents and contexts (e.g., family, peers, media, school, culture) that lead to the development of relational aggression over time. An understanding of how these behaviors develop will help inform intervention strategies to curb the use of relational aggression in schools, peer groups, and family relationships, which are addressed in an extended chapter.

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33

Crick, Alexandra, David Warwick, and Roderick Dunn. Nerves. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757689.003.0011.

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Examination of the nerves of the upper limb and localization of nerve lesions is mysterious to the unfamiliar. This chapter provides a scheme for the neuroanatomy of the upper limb, and for examination and investigation of nerve pathology including a section on neurophysiology. We discuss nerve injury, including pathophysiology and recovery. We describe common compression neuropathies affecting the median, ulnar, and radial nerves, and the brachial plexus lesions including thoracic outlet syndrome. Common tendon transfers are discussed for reconstruction following peripheral nerve injury or other loss of peripheral nerve function, and also for spinal injury at different levels.

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34

Raggi, Paolo, and Luis D’Marco. Imaging for detection of vascular disease in chronic kidney disease patients. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0116.

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The well-known severity of cardiovascular disease in patients suffering from chronic kidney disease (CKD) requires an accurate risk stratification of these patients in several clinical situations. Imaging has been used successfully for such purpose in the general population and it has demonstrated excellent potential among CKD patients as well. Two main forms of arterial pathology develop in patients with CKD: atherosclerosis, with accumulation of inflammatory cells, lipids, fibrous tissue and calcium in the subintimal space, and arteriosclerosis. The latter is characterized by accumulation of deposits of hydroxyapatite and amorphous calcium crystals in the muscular media of the vessel wall, and is believed to be more closely associated with alterations of mineral metabolism than with traditional atherosclerosis risk factors. The result is the development of what appears to be premature arterial ageing, with loss of elastic properties, increased stiffness, and increased overall fragility of the arterial system. Despite intensifying research and increasing awareness of these issues, the underlying pathophysiology of the aggressive vasculopathy of CKD remains largely unknown. As a consequence, there are currently very limited pathways to prevent progression of vascular damage in CKD. The indications, strengths and weaknesses of several imaging modalities employed to evaluate vascular disease in CKD are described, focusing on coronary arterial circulation and the peripheral arteries, with the exclusion of the intracranial arteries.

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35

Cheng, Ron Ron, and Abhay K. Varma. Ulnar Neuropathy—Cubital Tunnel Syndrome. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0004.

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The chapter presents the typical scenario of ulnar nerve entrapment at the elbow. The clinical picture can mimic pathology of nerve roots, of the brachial plexus, or of the ulnar nerve at different sites. Electrodiagnostic study helps to differentiate ulnar nerve entrapment from radiculopathy and to localize the site of compression, while imaging (ultrasound and MR imaging) are useful adjuncts to clinical examination. Conservative management is recommended for intermittent symptoms and absence of motor involvement. Surgical procedures include in situ, open, or endoscopic decompression and nerve transposition. Subluxation of the nerve over the medial epicondyle and recurrent or persistent neuropathy after in situ decompression are indications for transposition.

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36

Gala, Raj J., Lauren Szolomayer, and James Yue. Open Endoscopic Rhizotomy. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190626761.003.0015.

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The etiology of axial low back pain is multifactorial and includes pain arising from lumbar facet joints. The facet joints, capsules, and surrounding tissues are innervated by the medial branches of the dorsal rami. Rhizotomy of these nerves can provide pain relief in patients with lumbar facetogenic pain. The reported benefits of endoscopic approaches to the spine include minimal disruption of nonpathologic anatomy while simultaneously allowing for improved visualization of pathologic anatomy. Endoscopic techniques have been described for spinal stenosis, disc herniation, interbody fusion, infection, as well as dorsal medial branch rhizotomy. The goal of medial branch rhizotomy is to denervate lumbar facet joints that are contributing to axial back pain. The previous chapter focused on percutaneous techniques, while this chapter will describe endoscopic rhizotomy.

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37

Gaag, Anna van der, and Klara Dormandy. Communication and Adults with Learning Disabilities (Exc Business And Economy (Whurr)). Wiley, 1993.

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38

Alexander-Floyd, Nikol G. Re-Imagining Black Women. NYU Press, 2021. http://dx.doi.org/10.18574/nyu/9781479855858.001.0001.

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Re-Imagining Black Women dissects “post-politics”—the repertoire of dominant fantasies, frames, and narratives that hope for an afterlife beyond the social activism of the mid-twentieth century. This push for post-politics, such as post-feminism or post-racial thinking, serves as a form of race, gender, and class management that is uniquely suited for this neoliberal era. Alexander-Floyd centers black women as subjects, locating Moynihan’s black cultural pathology melodrama as the earliest basis for neoliberalism’s focus on self-regulation, solidifying patriarchal family formations, and the splitting of groups into virtuous victors who are worthy citizen subjects versus villainous, abject others in need of rehabilitation. Forging a unique methodology that fuses insights and approaches from political science, women’s studies, black studies, media studies, and most notably psychoanalysis, Re-Imagining Black Women provides a tour-de-force of black politics, exposing and addressing gender and other elements repressed or disavowed in the study of US race and politics. Each chapter traces the interplay of melodrama and liminality, examining political figures, such as Condoleezza Rice and Barack Obama and his My Brother’s Keeper initiative; cultural sites, such as The Help and Tyler Perry’s Madea; white male rape of black women and the social contract; and black women and the MeToo movement. This study helps to explain how some people were seduced by post-racial, post-feminist fantasies, exposing the primary ways in which they still operate. Re-Imagining Black Women also discusses post-politics in the COVID-19 era. It is a pioneering work that helps readers understand contemporary culture and politics and equips them to navigate turbulent political futures.

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39

Brusselaers, Nele, and Eric A. J. Hoste. Acute kidney injury in patients with severe burn injury. Edited by Norbert Lameire. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0253_update_001.

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Acute kidney injury (AKI) occurs in approximately one-quarter of all patients with severe burn injury (as defined by the RIFLE consensus classification), and approximately 3% of paediatric burn patients. Overall, a three- to six-fold higher mortality for burn patients with AKI is observed, depending on the applied definition. When AKI is defined by the sensitive RIFLE classification, median mortality of AKI is approximately 35%. This chapter describes the general pathophysiology of AKI in burns, particularly the severe form of burn shock, and discusses in addition the roles of intra-abdominal hypertension, rhabdomyolysis, and the potentially negative impact of povidone-iodine burn dressing. Finally the definitions used in burn pathology, the prevention of AKI with a discussion of the fluid therapy in burned patients, and the role of renal replacement therapy in these patients is discussed.

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40

Parr, Bartholomew. The London Medical Dictionary, Vol. 2: Including, Under Distinct Heads, Every Branch of Medicine, Viz. Anatomy, Physiology, and Pathology, the ... and Materia Medica (Classic Reprint). Forgotten Books, 2017.

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41

Okada, Kazuo, ed. Shock: FROM MOLECULAR AND CELLULAR LEVEL TO WHOLE BODY (International Congress Series (Excerpta Medica)). Elsevier Science Publishing Company, 1996.

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42

M, Cox Timothy, and Sinclair John Ph D, eds. Molecular biology in medicine. Oxford [England]: Blackwell Science, 1997.

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43

Covic, Adrian, Mugurel Apetrii, Luminita Voroneanu, and David J. Goldsmith. Vascular calcification. Edited by David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0120_update_001.

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Vascular calcification (VC) is a common feature of patients with advanced CKD and it could be, at least in part, the cause of increased cardiovascular mortality in these patients. From a morphologic point of view, there are at least two types of pathologic calcium phosphate deposition in the arterial wall—namely, intima calcification (mostly associated with atherosclerotic plaques) and media calcification (associated with stiffening of the vasculature, resulting in significantly adverse cardiovascular outcomes). Although VC was viewed initially as a passive phenomenon, it appears to be a cell-mediated, dynamic, and actively regulated process that closely resembles the formation of normal bone tissue, as discovered recently. VC seems to be the result of the dysregulation of the equilibrium between promoters and inhibitors. The determinants are mostly represented by altered calcium and phosphorus metabolism, secondary hyperparathyroidism, vitamin D excess, high fibroblast growth factor 23, and high levels of indoxyl sulphate or leptin; meanwhile, the inhibitors are vitamin K, fetuin A, matrix G1a protein, osteoprotegerin, and pyrophosphate. A number of non-invasive imaging techniques are available to investigate cardiac and vascular calcification: plain X-rays, to identify macroscopic calcifications of the aorta and peripheral arteries; two-dimensional ultrasound for investigating the calcification of carotid arteries, femoral arteries, and aorta; echocardiography, for assessment of valvular calcification; and, of course, computed tomography technologies, which constitute the gold standard for quantification of coronary artery and aorta calcification. All these methods have a series of advantages and limitations. The treatment/ prevention of VC is currently mostly around calcium-mineral bone disease interventions, and unproven. There are interesting hypotheses around vitamin K, Magnesium, sodium thiosulphate and other potential agents.

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44

R, Scriver Charles, ed. The metabolic and molecular bases of inherited disease. 7th ed. New York: McGraw-Hill, Health Professions Division, 1995.

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45

D, Willard Michael, and Tvedten Harold, eds. Small animal clinical diagnosis by laboratory methods. 4th ed. St. Louis, Mo: Saunders, 2004.

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