Relevant bibliographies by topics / Membrane neuronale

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Membrane neuronale'

Author: Grafiati
Published: 4 June 2021
Last updated: 8 June 2024

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Journal articles on the topic "Membrane neuronale"

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Simone, I. L., C. Tortorella, F. Federico, et al. "Contributo della risonanza magnetica spettroscopica del protone (1H-RMS) nella infezione da HIV." Rivista di Neuroradiologia 13, no. 1 (2000): 51–56. http://dx.doi.org/10.1177/197140090001300109.

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È stato condotto uno studio combinato di RMI e 1H-RMS (Magnetom Siemens 1,5 Tesla) in 60 pazienti sieropositivi per HIV (n. 25 encefalopatie-HIV correlate, n. 20 toxoplasmosi, n. 8 PML, n. 7 linfomi) e 22 controlli neurologici sieronegativi per HIV. Gli spettri sono stati acquisiti su volumi singoli localizzati su lesioni focali in toxoplasmosi, PML, linfomi o su lesioni diffuse nelle encefalopatie da HIV (sequenza Spin Echo, TE 135 ms). In tutti i sottogruppi HIV si è evidenziato un significativo decremento del rapporto NAA/Cr rispetto ai controlli neurologici, suggerendo un danno neuronale e
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Martínez-Reyes, Harold, and Antonio Eblen-Zajjur. "Evaluación in silico del efecto de benzodiacepinas, ketamina y termo-dependencia sobre los patrones de descarga neuronal pre-Bötzinger de control respiratorio." Archivos de Neurociencias 23, no. 2 (2020): 25–35. http://dx.doi.org/10.31157/an.v23i2.8.

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Introducción: la fiebre y el uso de drogas como las benzodiacepinas (agonistas GABAA) y la ketamina (antagonista NMDA) se presentan con relativa frecuencia en anestesiología. La respiración es un patrón generado en el tallo cerebral, producto de una red compleja de centros ponto-medulares, que controlan las motoneuronas respiratorias gracias a la interacción de las neuronas inspiratorias del complejo pre-Bötzinger y las espiratorias del complejo Bötzinger. Estas neuronas poseen receptores de membrana del tipo GABAA y NMDA, que muestran alta termodependencia, sin embargo; se desconoce los cambi
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Martínez-Reyes, Harold, and Antonio Eblen-Zajjur. "Evaluación in silico del efecto de benzodiacepinas, ketamina y termo-dependencia sobre los patrones de descarga neuronal pre-Bötzinger de control respiratorio." Archivos de Neurociencias 23, no. 2 (2020): 25–35. http://dx.doi.org/10.31157/archneurosciencesmex.v23i2.8.

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Introducción: la fiebre y el uso de drogas como las benzodiacepinas (agonistas GABAA) y la ketamina (antagonista NMDA) se presentan con relativa frecuencia en anestesiología. La respiración es un patrón generado en el tallo cerebral, producto de una red compleja de centros ponto-medulares, que controlan las motoneuronas respiratorias gracias a la interacción de las neuronas inspiratorias del complejo pre-Bötzinger y las espiratorias del complejo Bötzinger. Estas neuronas poseen receptores de membrana del tipo GABAA y NMDA, que muestran alta termodependencia, sin embargo; se desconoce los cambi
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Morelli, Sabrina, Antonella Piscioneri, Enrico Drioli, and Loredana De Bartolo. "Neuronal Differentiation Modulated by Polymeric Membrane Properties." Cells Tissues Organs 204, no. 3-4 (2017): 164–78. http://dx.doi.org/10.1159/000477135.

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In this study, different collagen-blend membranes were successfully constructed by blending collagen with chitosan (CHT) or poly(lactic-co-glycolic acid) (PLGA) to enhance their properties and thus create new biofunctional materials with great potential use for neuronal tissue engineering and regeneration. Collagen blending strongly affected membrane properties in the following ways: (i) it improved the surface hydrophilicity of both pure CHT and PLGA membranes, (ii) it reduced the stiffness of CHT membranes, but (iii) it did not modify the good mechanical properties of PLGA membranes. Then, w
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Dasgupta, Raktim, Markus S. Miettinen, Nico Fricke, Reinhard Lipowsky, and Rumiana Dimova. "The glycolipid GM1 reshapes asymmetric biomembranes and giant vesicles by curvature generation." Proceedings of the National Academy of Sciences 115, no. 22 (2018): 5756–61. http://dx.doi.org/10.1073/pnas.1722320115.

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The ganglioside GM1 is present in neuronal membranes at elevated concentrations with an asymmetric spatial distribution. It is known to generate curvature and can be expected to strongly influence the neuron morphology. To elucidate these effects, we prepared giant vesicles with GM1 predominantly present in one leaflet of the membrane, mimicking the asymmetric GM1 distribution in neuronal membranes. Based on pulling inward and outward tubes, we developed a technique that allowed the direct measurement of the membrane spontaneous curvature. Using vesicle electroporation and fluorescence intensi
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Nakada, C., Kenneth Ritchie, T. Fujiwara, et al. "LS3A1 Diffusion barrier in the neuronal cell membrane : a single molecule study." Seibutsu Butsuri 42, supplement2 (2002): S223. http://dx.doi.org/10.2142/biophys.42.s223_4.

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SARRADIN, P., P. BERTHON, and F. LANTIER. "Le point sur l’épidémiologie et la physiopathologie des encéphalopathies spongiformes des ruminants." INRAE Productions Animales 10, no. 2 (1997): 123–32. http://dx.doi.org/10.20870/productions-animales.1997.10.2.3988.

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L’épidémie d’encéphalopathie spongiforme bovine (ESB) résulte de la consommation par les bovins de farines de viandes et d’os contaminées. En recyclant l’agent infectieux, ces farines ont permis d’amplifier la dissémination d’une maladie dont l’origine et l’agent responsable demeurent inconnus. Les hypothèses sur la nature protéique ou/et virale de l’agent sont évoquées, ainsi que l’éventualité d’une transmission à l’homme. Une grande partie de nos connaissances des encéphalopathies spongiformes résulte des études réalisées de longue date sur la tremblante des ovins. En particulier, l’idée que
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Chen, Yuejun, Feifei Wang, Hui Long, Ying Chen, Ziyan Wu, and Lan Ma. "GRK5 promotes F-actin bundling and targets bundles to membrane structures to control neuronal morphogenesis." Journal of Cell Biology 194, no. 6 (2011): 905–20. http://dx.doi.org/10.1083/jcb.201104114.

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Neuronal morphogenesis requires extensive membrane remodeling and cytoskeleton dynamics. In this paper, we show that GRK5, a G protein–coupled receptor kinase, is critically involved in neurite outgrowth, dendrite branching, and spine morphogenesis through promotion of filopodial protrusion. Interestingly, GRK5 is not acting as a kinase but rather provides a key link between the plasma membrane and the actin cytoskeleton. GRK5 promoted filamentous actin (F-actin) bundling at the membranes of dynamic neuronal structures by interacting with both F-actin and phosphatidylinositol-4,5-bisphosphate.
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Harrison, R., A. Jehanli, G. G. Lunt, and J. Rutter. "Autoantibodies to neuronal membranes in motor neurone disease." Journal of Neuroimmunology 16, no. 1 (1987): 71. http://dx.doi.org/10.1016/0165-5728(87)90238-4.

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Sotnikov, Oleg S., Svetlana S. Sergeeva, and Tat'yana I. Vasyagina. "NEURONAL-GLIAL MEMBRANE CONTACTS DURING PESSIMAL ELECTRICAL STIMULATION." Morphological newsletter 28, no. 3 (2020): 35–50. http://dx.doi.org/10.20340/mv-mn.2020.28(3):35-50.

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After the creation of a method for obtaining inter-neuronal gap junctions in a nervous system devoid of glia, it is expedient to reproduce gap neuronal-glial contacts on a model that also contains hybrid neuronal-glial gap junctions, which, as you know, are functionally fundamentally different from inter-neuronal contacts. The experiments were carried out on the truncus sympathicus ganglia of laboratory rats using pessimal electrical stimulation and transmission electron microscopy. Electrical activation of ganglia with a frequency of up to 100 Hz revealed local and widespread variants of vari
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Dissertations / Theses on the topic "Membrane neuronale"

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Woiterski, Lydia. "Meeting at the Membrane – Confined Water at Cationic Lipids & Neuronal Growth on Fluid Lipid Bilayers." Doctoral thesis, Universitätsbibliothek Leipzig, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-132933.

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Die Zellmembran dient der Zelle nicht nur als äußere Hülle, sondern ist auch an einer Vielzahl von lebenswichtigen Prozessen wie Signaltransduktion oder Zelladhäsion beteiligt. Wasser als integraler Bestandteil von Zellen und der extrazellulären Matrix hat sowohl einen großen Einfluss auf die Struktur von Biomolekülen, als auch selbst besondere Merkmale in eingschränkter Geometrie. Im Rahmen dieser Arbeit wurden zwei Effekte an Modellmembranen untersucht: Erstens der Einfluss des Gegenions an kationischen Lipiden (DODAX, X = F, Cl, Br, I) auf die Eigenschaften des Grenzflächenwassers und zweit
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Ferreira, Brigham Marco Paulo. "Nonstationary Stochastic Dynamics of Neuronal Membranes." Thesis, Paris 6, 2015. http://www.theses.fr/2015PA066111/document.

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Les neurones interagissent à travers leur potentiel de membrane qui a en général une évolution temporelle complexe due aux nombreuses entrées synaptiques irrégulières reçues. Cette évolution est mieux décrite en termes probabilistes, en raison de ces entrées irrégulières ou «bruit synaptique». L'évolution temporelle du potentiel de membrane est stochastique mais aussi déterministe: stochastique, car conduite par des entrées synaptiques qui arrivent de façon aléatoire dans le temps, et déterministe, car un neurone biologique a une évolution temporelle très similaire quand soumis à une même séqu
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Morisset, Valérie. "Plasticité fonctionnelle dans la moelle épinière : caractéristiques et neuromodulation des propriétés membranaires intrinsèques des neurones de la corne dorsale." Bordeaux 2, 1999. http://www.theses.fr/1999BOR28644.

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Colin, Isabelle. "Formation de la membrane postsynaptique neuronale chez le rat : analyse morphologique de la mise en place des recepteurs glycinergiques centraux." Paris 6, 1997. http://www.theses.fr/1997PA066636.

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La formation des agregats de recepteurs de la glycine (rgly) postsynaptiques stabilises par une molecule associee (la gephyrine) a ete analysee par une etude immunocytochimique effectuee au cours du developpement in vitro et in vivo de la moelle epiniere de rat. Nous avons analyse ; i) les distributions cellulaires et subcellulaires des agregats de rgly et de gephyrine ; ii) les densites de leurs marquages postsynaptiques ; et ii) la nature des neurotransmetteurs inhibiteurs (glycine, gaba) presents dans les terminaisons presynaptiques a la gephyrine. In vitro nous avons caracterise l'etabliss
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Vigier, Maxime. "Influence des lipides membranaires sur les interactions protéiques liées aux anomalies endolysosomales dans un modèle neuronal de la maladie d'Alzheimer." Electronic Thesis or Diss., Université de Lorraine, 2022. http://www.theses.fr/2022LORR0331.

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La maladie d'Alzheimer (MA) est une pathologie complexe et multifactorielle pour laquelle il n'existe actuellement aucun traitement. Plusieurs hypothèses ont été proposées pour expliquer l'apparition et la progression de cette maladie, dont la cascade amyloïde, qui prédomine dans le domaine de la recherche depuis 30 ans. La voie amyloïdogène nécessite l'endocytose de la protéine APP dans les endosomes précoces où elle subit deux clivages protéolytiques, d'abord par la β-sécrétase pour produire le fragment C99, puis par la γ-sécrétase pour produire le peptide Aβ. L'une des hypothèses actuelles
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Colin, Julie. "Le vieillissement membranaire cérébral : conséquences fonctionnelles et protection par les acides gras polyinsaturés oméga-3 alimentaires." Thesis, Université de Lorraine, 2015. http://www.theses.fr/2015LORR0078/document.

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Un des phénomènes sociétaux marquants de ces dernières années est le vieillissement de la population et en conséquence, une hausse considérable du nombre de personnes âgées. Dans ce contexte, la recrudescence des pathologies chroniques liées au vieillissement, dont la maladie d’Alzheimer, est devenue un enjeu majeur de santé publique. L’impact de nombreux facteurs environnementaux modulables, l’aspect chronique et évolutif des mécanismes pathogènes mis en jeu, doivent inciter à développer des interventions préventives permettant de minimiser les risques de développer ces maladies liées au viei
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Casellas, Díaz Sergi. "Regulación de la bioenergética mitocondrial por Mfn2 mediante los contactos RE-mitocondria y su implicación en la fisiología neuronal." Doctoral thesis, Universitat de Barcelona, 2021. http://hdl.handle.net/10803/673820.

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Las mitofusinas 1 y 2 son unas GTPasas que se encuentran en la membrana mitocondrial externa, donde llevan a cabo el proceso de fusión mitocondrial. La existencia de mutaciones en Mfn2 es la principal causa de la enfermedad de Charcot- Marie-Tooth tipo 2A, un grupo de distintas neuropatías caracterizadas por degeneración axonal. Por otro lado, otras alteraciones en Mfn2 también han sido descritas en múltiples enfermedades neurodegenerativas crónicas, en episodios traumáticos agudos como accidentes cerebrovasculares e incluso otras enfermedades no neurológicas como las car
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Prado, Gustavo R. "Neuronal Plasma Membrane Disruption in Traumatic Brain Injury." Diss., Georgia Institute of Technology, 2004. http://hdl.handle.net/1853/7260.

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During a traumatic insult to the brain, tissue is subjected to large stresses at high rates which often surpass cellular thresholds leading to cell dysfunction or death. Cellular events that occur at the time of and immediately after an insult are poorly understood. Immediately following traumatic brain injury (TBI), the neuronal plasma membrane may become disrupted and potentiate detrimental pathways by allowing extracellular contents to gain access to the cytosol. In the current study, neuronal plasma membrane disruption was assessed in vivo following moderate unilateral controlled cortical
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Farber, Steven Arthur. "Neuronal activity and membrane turnover in rat brain." Thesis, Massachusetts Institute of Technology, 1993. http://hdl.handle.net/1721.1/12673.

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Raju, Rajeswari. "Optimisation of image processing networks for neuronal membrane detection." Thesis, University of Nottingham, 2016. http://eprints.nottingham.ac.uk/33948/.

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This research dealt with the problem of neuronal membrane detection, in which the core challenge is distinguishing membranes from organelles. A simple and efficient optimisation framework is proposed based on several basic processing steps, including local contrast enhancement, denoising, thresholding, hole-filling, watershed segmentation, and morphological operations. The two main algorithms proposed Image Processing Chain Optimisation (IPCO) and Multiple IPCO (MIPCO)combine elements of Genetic Algorithms, Differential Evolution, and Rank-based uniform crossover. 91.67% is the highest recorde
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Books on the topic "Membrane neuronale"

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Wright, William Neil. Nonlinear models of the electrical properties of neuronal membranes. National Library of Canada = Bibliothèque nationale du Canada, 1993.

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Neurobiology. 3rd ed. Oxford University Press, 1994.

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Neurobiology. 2nd ed. Oxford University Press, 1988.

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Montgomery, Erwin B. Controlling the Flow of Electrical Charges. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190259600.003.0004.

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In depolarization to effect neuronal activations,, electrical charges are delivered to the neuron to affect the electrical potential across the neuronal membrane to subsequently affect voltage-gated ionic conductance channels. The orientation of the field of electrical charges to the neuronal membrane is critical. Electrical charges flow from the negative contact to the positive contact. The negative electrostatic charge “pushes” negative charges onto the outer surface of the neuron, which results in depolarization of the neuronal membrane. Neurons near the positive contact will not have negat
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Laktionova, A. A., and O. S. Sotnikov. Membrane Fusion and Syncytial Neuronal Cytoplasmic Connection. Trans Tech Publications, Limited, 2016.

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Laktionova, A. A., and O. S. Sotnikov. Membrane Fusion and Syncytial Neuronal Cytoplasmic Connection. Trans Tech Publications, Limited, 2016.

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D'Aguanno, Aldo. The passive electrical properties of neuronal membranes. 1987.

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Slimp, Jefferson C. Neurophysiology of Multiple Sclerosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199341016.003.0003.

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Any discussion of the pathomechanisms and treatments of MS benefits from an understanding of the physiology of the neuronal membrane and the action potential. Neurons and glia, are important for signal propagation, synaptic function, and neural development. The neuronal cell membrane, maintains different ionic environments inside and outside the cell, separating charge across the membrane and facilitating electrical excitability. Ion channels allow flow of sodium, potassium, and calcium ions across the membrane at selected times. At rest, potassium ion efflux across the membrane establishes th
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Mason, Peggy. The Neuron at Rest. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190237493.003.0009.

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Neuronal membrane potential depends on the distribution of ions across the plasma membrane and the permeability of the membrane to those ions afforded by transmembrane proteins. Ions cannot pass through a lipid bilayer but enter or exit neurons through ion channels. When activated by voltage or a ligand, ion channels open to form a pore through which selective ions can pass. The ion channels that support a resting membrane potential are critical to setting a cell’s excitability. From the distribution of an ionic species, the Nernst potential can be used to predict the steady-state potential fo
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Orchinik, Miles. Biochemical and behavioral characterization of steroid receptors in neuronal membranes. 1992.

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Book chapters on the topic "Membrane neuronale"

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De Bartolo, Loredana, and Antonietta Messina. "Neuronal Regeneration." In Encyclopedia of Membranes. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-40872-4_410-1.

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Bear, Mark F., Barry W. Connors, and Michael A. Paradiso. "Die neuronale Membran im Ruhezustand." In Neurowissenschaften. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-662-49933-7_3.

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Bear, Mark F., Barry W. Connors, and Michael A. Paradiso. "Die neuronale Membran im Ruhezustand." In Neurowissenschaften. Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-57263-4_3.

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Bear, Mark F., Barry W. Connors, and Michael A. Paradiso. "Die neuronale Membran im Ruhezustand." In Neurowissenschaften. Spektrum Akademischer Verlag, 2009. http://dx.doi.org/10.1007/978-3-8274-2228-6_3.

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Cole, Gregory J., and Richard W. Bond. "Neuronal Antigens Involved in Cell Adhesion and Cell Recognition." In Cell Membranes. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1915-3_3.

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Moore, J. W., and N. E. Berthier. "Purkinje Cell Activity and the Conditioned Nictitating Membrane Response." In Cerebellum and Neuronal Plasticity. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-0965-9_22.

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Yu, Robert K., James R. Goldenring, John Y. H. Kim, and Robert J. DeLorenzo. "Gangliosides as Differential Modulators of Membrane-Bound Protein Kinase Systems." In Gangliosides and Neuronal Plasticity. Springer New York, 1986. http://dx.doi.org/10.1007/978-1-4757-5309-7_8.

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Levin, Ellis R. "Nuclear Receptor Versus Plasma Membrane Oestrogen Receptor." In Neuronal and Cognitive Effects of Oestrogens. John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/0470870818.ch5.

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Ando, Susumu, Yasukazu Tanaka, and Kazuo Kon. "Membrane Aging of the Brain Synaptosomes with Special Reference to Gangliosides." In Gangliosides and Neuronal Plasticity. Springer New York, 1986. http://dx.doi.org/10.1007/978-1-4757-5309-7_9.

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Breer, H., and M. Knipper. "Synaptosomes and Neuronal Membranes from Insects." In Springer Series in Experimental Entomology. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70045-3_6.

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Conference papers on the topic "Membrane neuronale"

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Cecílio, Samyra Giarola, Jéssica Naiara Lara, Camila Raianna Justiniana Rocha, and Juliana Pereira Cardoso. "EPILEPSIAS E A HOMEOSTASE DE CLORETO." In II Congresso Brasileiro de Ciências Biológicas On-line. Revista Multidisciplinar Educação e Meio Ambiente, 2021. http://dx.doi.org/10.51189/rema/1662.

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Introdução: Os íons sódio, potássio, cálcio e cloreto são os principais envolvidos na determinação do potencial de membrana das células neuronais. Uma vez que o potencial de equilíbrio do cloreto (ECl), de - 80 mV, é próximo ao potencial de repouso da membrana (- 75 mV), pequenas variações na concentração intracelular do íon interferem de modo significativo no gradiente transmembrânico e, consequentemente, na excitabilidade celular. Objetivo: A proposta do presente estudo consistiu em uma revisão da literatura sobre a influência do desequilíbrio iônico de cloreto. Método: Realizou-se levantame
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Allen, Kathleen B., and Bradley Layton. "A Mechanical Model for Cytoskeleton and Membrane Interactions in Neuronal Growth Cones." In ASME 2007 International Mechanical Engineering Congress and Exposition. ASMEDC, 2007. http://dx.doi.org/10.1115/imece2007-42008.

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Revealing the molecular events of neuronal growth is critical to obtaining a deeper understanding of nervous system development, neural injury response, and neural tissue engineering. Central to this is the need to understand the mechanical interactions among the cytoskeleton and the cell membrane, and how these interactions affect the overall growth mechanics of neurons. Using ANSYS, the force produced by a cytoskeletal protein acting against a deformable membrane was modeled, and the deformation, stress, and strain were computed for the membrane. Parameters to represent the flexural rigiditi
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Cao, Guoxin, You Zhou, Jeong Soon Lee, Jung Yul Lim, and Namas Chandra. "Mechanical Model of Neuronal Function Loss." In ASME 2010 International Mechanical Engineering Congress and Exposition. ASMEDC, 2010. http://dx.doi.org/10.1115/imece2010-39447.

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The mechanism of mild traumatic brain injury (mTBI) is directly related to the relationship between the mechanical response of neurons and their biological/chemical functions since the neuron is the main functional component of brain.1 The hypotheses is that the external mechanical load will firstly cause the mechanical deformation of neurons, and then, when the mechanical deformation of neurons reaches to a critical point (the mechanical deformation threshold), it will initiate the chemical/biological response (e.g. neuronal function loss). Therefore, defining and measuring the mechanical def
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Roke, S. "Probing Neuronal Activity using Membrane Interfacial Water." In 2019 Conference on Lasers and Electro-Optics Europe & European Quantum Electronics Conference (CLEO/Europe-EQEC). IEEE, 2019. http://dx.doi.org/10.1109/cleoe-eqec.2019.8873282.

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Dolotova, S. M., Y. V. Bershatsky, Yu A. Zagryadskaya, et al. "OBTAINING MUTANT FORMS OF THE TRANSMEMBRANE DOMAIN OF THE RECEPTOR FOR ADVANCED GLYCATION END PRODUCTS (RAGE) FOR STRUCTURAL NMR STUDIES." In X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-173.

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The membrane receptor RAGE of immunoglobulin family is involved in regulation of neuronal homeostasis and inflammatory processes in human body. To understand the molecular mechanism of signal transduction across the membrane by RAGE, we performed structural-dynamic NMR studies of recombinant transmembrane fragments of RAGE with oncogenic mutations G365R and G368E in membrane-mimicking media.
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LaPlaca, M. C., G. R. Prado, D. Kacy Cullen, and C. M. Simon. "Plasma membrane damage as a marker of neuronal injury." In 2009 Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2009. http://dx.doi.org/10.1109/iembs.2009.5334457.

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Lee, Sung Jin, Jingjing Sun, Michael King, Huikai Xie, and Malisa Sarntinoranont. "Viscoelastic Property Changes of Acute Rat Brain Tissue Slices as a Function of Cell Viability." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53909.

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Changes in mechanical properties within brain tissues after losses in cell viability have not been well investigated. Lack of oxygen and nutrient transport can induce hypoxic neuronal injury and increase cell membrane permeability, and cell membranes and matrix components can lose their structural and mechanical integrity. These physical changes may have an effect on mechanical properties of brain tissue [1]. In this study, the viscoelastic behavior of two anatomical regions (cerebral cortex and hippocampus) in acute rat brain tissue slices were measured as a function of cell viability using i
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Wallace, Ron. "Microgravity and charge transfer in the neuronal membrane - Implications for computational neurobiology." In Life Sciences and Space Medicine Conference. American Institute of Aeronautics and Astronautics, 1995. http://dx.doi.org/10.2514/6.1995-1053.

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Tkachenko, A. D. "Photosensitivity of bilirubin incorporated in neuronal membrane particles or in biological gel." In Optical Information Science and Technology, edited by Andrei L. Mikaelian. SPIE, 1998. http://dx.doi.org/10.1117/12.304985.

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Me´ndez-Rojas, Miguel A., Claudia Cravioto Guzman, and Oscar Arias-Carrion. "Synthesis and Chemical Functionalization of Ferromagnetic Nanoparticles to Manipulate Stem Cells Using External Magnetic Fields." In ASME 2007 5th International Conference on Nanochannels, Microchannels, and Minichannels. ASMEDC, 2007. http://dx.doi.org/10.1115/icnmm2007-30188.

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The human brain is able to respond to several situations that promotes neural survivance, growth and neuronal cells regeneration. The potential of stem cells from the subventricular zone to be used for neuronal cell substitution has been widely studied. However, migration of endogen or transplanted cells is random, not efficient and with a very low survivance rate. The use of ferromagnetic nanoparticles may be of interest to guide the stem cells to the desired site using external fields. In this project, we have synthesized ferromagnetic nanoparticles (magnetite) with an average diameter of 10
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Reports on the topic "Membrane neuronale"

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Esteban, José Antonio. Del tráfico de membrana a la comunicación neuronal (Especial Premio Nobel de Medicina 2013). Sociedad Española de Bioquímica y Biología Molecular (SEBBM), 2014. http://dx.doi.org/10.18567/sebbmdiv_anc.2014.07.1.

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