Relevant bibliographies by topics / Mental illness Nervous system Psychopharmacology

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  2. Dissertations / Theses
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Academic literature on the topic 'Mental illness Nervous system Psychopharmacology'

Author: Grafiati
Published: 4 June 2021
Last updated: 13 February 2022

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Journal articles on the topic "Mental illness Nervous system Psychopharmacology"

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Morrison, Diana P., Andre F. Joubert, Dave Swingler, et al. "Psychopharmacology 2003 Conference, 10-13 September 2003." South African Journal of Psychiatry 9, no. 2 (2003): 17. http://dx.doi.org/10.4102/sajpsychiatry.v9i2.136.

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List of abstracts and authors:1. Comparative benefits of Atypical antipsychoticsDiana P Morrison2. Evidence-based management of depression in SchizophreniaAndre F Joubert3. Second generation Antipsychotics: An African updateDave Swingler4. The drug management of patients with HIV/Aids in the Mental Health Care setting: A therapeutic challengeDenise White5. New developments in the treatment of Bipolar depressionJospeh R Calabrese6. Dual action antidepressants: Faster onset, more remission, better value?Roger M Pinder7. Antisocial personality disorder: A reviewDonald W Black8. The South African
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Braslow, Joel T., and Stephen R. Marder. "History of Psychopharmacology." Annual Review of Clinical Psychology 15, no. 1 (2019): 25–50. http://dx.doi.org/10.1146/annurev-clinpsy-050718-095514.

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We live in an age of psychopharmacology. One in six persons currently takes a psychotropic drug. These drugs have profoundly shaped our scientific and cultural understanding of psychiatric disease. By way of a historical review, we try to make sense of psychiatry's dependency on psychiatric drugs in the care of patients. Modern psychopharmacology began in 1950 with the synthesis of chlorpromazine. Over the course of the next 50 years, the psychiatric understanding and treatment of mental illness radically changed. Psychotropic drugs played a major part in these changes as state hospitals close
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Baruah, Jugajyoti, and Anju Vasudevan. "The Vessels Shaping Mental Health or Illness." Open Neurology Journal 13, no. 1 (2019): 1–9. http://dx.doi.org/10.2174/1874205x01913010001.

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The mammalian brain receives the lion’s share of the body’s blood supply and is a highly vascularized organ. The vascular and nervous systems arise at two distinct time points of embryogenesis; however, their functions tend to overlap or complement each other in the growth promoting milieu of the embryonic Central Nervous System (CNS). The pre-existing idea that mental disorders are a direct result from defects solely in neuronal populations and networks is gradually changing. Several studies have implicated blood vessel pathologies and blood flow changes in mental health disorders. Our own st
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Saugstad, Letten F. "Age at Puberty and Mental Illness." British Journal of Psychiatry 155, no. 4 (1989): 536–44. http://dx.doi.org/10.1192/bjp.155.4.536.

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The hypothesis of a neurodevelopmental aetiology of manic-depressive psychosis and schizophrenia is based on the relation between onset of puberty and the final regressive events in the central nervous system (elimination of 40% of neuronal synapses), and the discrepancy in body build in the two disorders which is similar to that between early- and late-maturing individuals. The marked rise in manic–depressive psychoses and decline in schizophrenia, particularly the non-paranoid categories, accompanying the decline in mean pubertal age by some four years during the past hundred years are taken
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Goodwin, F. K., and S. N. Ghaemi. "Prospects for a scientific psychiatry." Acta Neuropsychiatrica 9, no. 2 (1997): 49–51. http://dx.doi.org/10.1017/s0924270800036772.

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Herman van Praag has made major contributions to the scientific and conceptual development of psychiatry. Despite progress in delineating reliable diagnostic criteria, our classification system ignores clinical insights available from patients' subjective experiences of illness. Hence a ‘coarsening of diagnosis’ has occurred, weakening the nosologic infrastructure of scientific psychiatry. This paper will discuss these ideas and emphasize the central role of psychopharmacology in advancing psychiatry, with lithium's discovery as a paradigm.
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Hare, Edward. "The History of ‘Nervous Disorders' from 1600 to 1840, and a Comparison with Modern Views." British Journal of Psychiatry 159, no. 1 (1991): 37–45. http://dx.doi.org/10.1192/bjp.159.1.37.

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In the 18th century the main varieties of nervous illness – hypochondria, hysteria, the spleen, the vapours and dyspepsia – became included under the general term ‘nervous disorders'. When no physical disorder of the nerves could be detected in such conditions, the hypothesis of nervous disorder was replaced by the more vague concept of ‘nervous temperament’. The fact that there is still no evidence of pathological change in such cases continues to expose physicians to the alternative hypothesis of a purely psychological cause. The modern era in our understanding of the nervous system may be s
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Quinn, Declan M. P. "Prevalence of Psychoactive Medication in Children and Adolescents." Canadian Journal of Psychiatry 31, no. 6 (1986): 575–80. http://dx.doi.org/10.1177/070674378603100616.

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Prevalence studies in Psychopharmacology are few and have many methodological flaws. This study examines the prevalence of psychoactive drugs in children and adolescents. Contrary to other studies, central nervous system stimulants are not the most widely prescribed psychoactive drugs in childhood and adolescence, but rather, minor tranquilizers, sedatives and hypnotics are the most widely prescribed psychoactive drugs. While this study overcomes some of the flaws of previous studies further studies regarding the prevalence of use of psychoactive drugs in childhood and adolescence are recommen
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Ayoub, Aimina, William D. Fraser, Nancy Low, Laura Arbour, Jessica Healy-Profitós, and Nathalie Auger. "Risk of central nervous system defects in offspring of women with and without mental illness." Archives of Women's Mental Health 21, no. 4 (2018): 437–44. http://dx.doi.org/10.1007/s00737-018-0819-0.

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Birmingham, Marion K., Madhabananda Sar, and Walter E. Stumpf. "Dexamethasone target sites in the central nervous system and their potential relevance to mental illness." Cellular and Molecular Neurobiology 13, no. 4 (1993): 373–86. http://dx.doi.org/10.1007/bf00711578.

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Cowen, Philip J. "Serotonin – 100 words." British Journal of Psychiatry 203, no. 1 (2013): 23. http://dx.doi.org/10.1192/bjp.bp.112.108506.

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Serotonin is a neurotransmitter conserved through at least 500 million years of nervous system evolution. Serotonin orchestrates adaptive responses to aversive stimuli in invertebrates and an analogous role can be discerned in the more complex behavioural repertoire displayed by mammals to adversity. However, this formulation fails to capture the range of human social behaviours influenced by serotonin, for example, affiliation, empathy and cooperation. In a psychopharmacology experiment I received paroxetine for three weeks. This boost in brain serotonin levels failed to alter my subjective r
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Dissertations / Theses on the topic "Mental illness Nervous system Psychopharmacology"

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Häidkind, Riina. "Monoaminergic mechanisms in mood-associated behaviours and neurochemistry in rats /." Tartu : Tartu University Press, 2004. http://dspace.utlib.ee/dspace/bitstream/10062/704/5/Haidkind.pdf.

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Brinkman, John J. "Psychiatric disorders as an outcome of neurological insult : a computation of relative risk." Virtual Press, 2004. http://liblink.bsu.edu/uhtbin/catkey/1301628.

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The purpose of this study was to examine the relative risk of psychiatric disorders occurring in patients diagnosed with neurological disorders. This study separately computed the relative risk ratios for identified psychiatric disorder (i.e., anxiety, mood disorders, somatization, schizophrenia, alcohol abuse, and antisocial personality disorder) on seven of the more common neurological disorders (i.e., brain tumor, closed head injury, stroke, dementia, multiple sclerosis, cerebral palsy, and Parkinson's disease). The six psychiatric disorders were chosen based on the epidemiological catchmen
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Matthews, Paul Richard Leonard. "Morphometric and molecular studies of schizophrenia and mood disorders." Thesis, University of Oxford, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.670182.

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Baldwin, P. Clive. "Narrative, ethics and severe mental illness." 2005. http://hdl.handle.net/10454/3268.

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No<br>Starting from the premise that people are essentially narrative beings, I argue that the onset of severe mental illness compromises the narrative enterprise of being able to construct one's Self and one's relationships inmeaningful and coherent ways. This is due to both the curtailment of opportunities for narrative engagement and the dispossession of those whose narratives do not conform to the current conceptualization of narrative and narrativity. In these circumstances, supporting the narrative enterprise is an ethical endeavour that requires that we examine not only which narratives
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Books on the topic "Mental illness Nervous system Psychopharmacology"

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Stahl's essential psychopharmacology. 3rd ed. Cambridge University Press, 2008.

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Essential psychopharmacology: The prescriber's guide. Cambridge University Press, 2006.

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Essential psychopharmacology: Neuroscientific basis and clinical applications. Cambridge University Press, 1998.

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Stahl, S. M. Essential psychopharmacology: Neuroscientific basis and clinical applications. Cambridge University Press, 1996.

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S, Musser William, and Gershon Samuel, eds. Maintenance pharmacotherapies for neuropsychiatric disorders. Brunner/Mazel, 1999.

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6

Steve, Parker. The brain and nervous system. Raintree Steck-Vaughn, 1997.

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Steve, Parker. The brain and nervous system. F. Watts, 1990.

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McArthur, Robert A., and Franco Borsini. Animal and translational models for CNS drug discovery. Elsevier/Academic Press, 2008.

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Michael-Titus, Adina. The nervous system. Churchill Livingstone, 2006.

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Patricia, Revest, and Shortland Peter, eds. The nervous system. 2nd ed. Churchill Livingstone, 2010.

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Book chapters on the topic "Mental illness Nervous system Psychopharmacology"

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Deutch, Ariel Y., and Robert H. Roth. "Neurochemical Systems in the Central Nervous System." In Neurobiology of Mental Illness. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199798261.003.0002.

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Deutch, Ariel Y., and Robert H. Roth. "Neurochemical Systems in the Central Nervous System." In Neurobiology of Mental Illness, edited by Eric J. Nestler. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199934959.003.0002.

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Chapter 2 describes the neurochemical organization of the brain. It summarizes the diverse types of molecules that neurons in the brain use as neurotransmitters and neurotrophic factors, and how these molecules are synthesized and metabolized. The chapter also presents the array of receptor proteins through which these molecules regulate target neuron functioning and the reuptake proteins that generally terminate the neurotransmitter signal. Today a large majority of all drugs used to treat psychiatric disorders, as well as most drugs of abuse, still have as their initial targets proteins involved directly in neurotransmitter function.
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Hickey, Miriam, Nicholas Franich, Vera Medvedeva, and Marie-Françoise Chesselet. "Mouse Models of Mental Illness and Neurological Disease." In The Mouse Nervous System. Elsevier, 2012. http://dx.doi.org/10.1016/b978-0-12-369497-3.10032-9.

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Rubenstein, John L. R. "Overview of Brain Development." In Neurobiology of Mental Illness, edited by Eric J. Nestler. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199934959.003.0001.

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This chapter highlights many of the major processes involved in brain development, including: induction of the central nervous system (CNS), patterning of the primordia of major brain regions, proliferation of neuroepithelial cells, differentiation and migration of immature neurons and glia, formation of axon tracts and synapses, and the establishment and plasticity of neuronal networks.
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Kwon, Ester J., Takahiro Soda, and Li-Huei Tsai. "Neurodevelopment and Schizophrenia." In Neurobiology of Mental Illness, edited by Pamela Sklar. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199934959.003.0025.

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A neurodevelopmental model of schizophrenia postulates that some of the key aspects of brain development that normally occur both pre- and post-natally are not occurring correctly, either in time or space. Complex neural circuitry needs to form and be modulated by experience. Classically, proliferation, migration, arborization and myelination occur prenatally. Elaboration and refining of dendritic trees and synapses as well as myelination of the nervous system continues through the first two-decade of life. There are opportunities for genetic and environmental abnormalities and variation to profoundly influence the trajectories of all of these critical functional processes.
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Mitchell, Amanda C., Yan Jiang, Cyril J. Peter, Ki A. Goosens, and Schahram Akbarian. "The Brain and Its Epigenome." In Neurobiology of Mental Illness, edited by Karl Deisseroth. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199934959.003.0013.

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Exploration of the epigenome—collectively defined by DNA methylation, post-translational histone modifications, histone variants and other regulators of genome organization and function—has emerged as one of the most prolific areas of the basic and clinical neurosciences alike. This is due to a number of recent developments, including a wealth of genetic information on psychiatric disorders indicating that many risk-associated DNA variants and mutations do not affect protein coding sequences. Furthermore, the hopeful prospect of chromatin modifying drugs to lead to novel therapeutic options—while largely based on preclinical studies in small laboratory animals such as rats and mice— has infiltrated many areas of medicine, including neurology and psychiatry. Here, we summarize current concepts and emerging insights on epigenetic regulation in the nervous system, with focus on the human brain and the neurobiology and pharmacology of cognitive and emotional disease.
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Hildebrandt, Thomas B., and Amanda Downey. "The Neurobiology of Eating Disorders." In Neurobiology of Mental Illness, edited by Dennis S. Charney. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199934959.003.0089.

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Eating disorders are recognized by a primary dysregulation in the ability to eat and the inability to maintain a healthy weight without significant and impairing levels of distress. Commonly beginning in adolescence and affecting primarily females, these illnesses have debated diagnostic criteria but retain core features including disturbances in the maintenance of a healthy weight, episodic binge eating and/or compensatory behaviors, and body image disturbances. The pathogenesis of eating disorders is not well understood, having historically been attributed to primarily sociocultural pressures to be thin. The dearth of effective interventions for eating disorders may stem from the lack of understanding of the complex neurobiological processes that contribute to the psychopathology of these illnesses. This chapter will provide a summary of the current research of the genetic, hormonal, neurotransmitter, and functional neurocircuitry of eating disorders. Dysregulation in appetite hormones and peptides, adrenal hormones, ovarian hormones, the serotonergic and dopaminergic systems and several central nervous system neurocircuits all show strong correlation to the development and maintenance of these chronic and frequently relapsing disorders. Future directions for laboratory, clinical, and translational research are discussed.
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Konofagou, Elisa E. "Blood–Brain Barrier Opening and Drug Delivery Using Focused Ultrasound and Microbubbles." In Neurobiology of Mental Illness, edited by Karl Deisseroth. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199934959.003.0011.

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Current treatments of neurological and neurodegenerative diseases are limited due to the lack of a truly non-invasive, transient, and regionally selective brain drug delivery method. The brain is particularly difficult to deliver drugs to because of the blood-brain barrier (BBB). The impermeability of the BBB is due to the tight junctions connecting adjacent endothelial cells and highly regulatory transport systems of the endothelial cell membranes. The main function of the BBB is ion and volume regulation to ensure conditions necessary for proper synaptic and axonal signaling. However, the same permeability properties that keep the brain healthy also constitute the cause of the tremendous obstacles posed in its pharmacological treatment. The BBB prevents most neurologically active drugs from entering the brain and, as a result, has been isolated as the rate-limiting factor in brain drug delivery. Until a solution to the trans-BBB delivery problem is found, treatments of neurological diseases will remain impeded. Over the past decade, methods that combine Focused Ultrasound (FUS) and microbubbles have been shown to offer the unique capability of noninvasively, locally and transiently opening the BBB so as to treat central nervous system (CNS) diseases. Four of the main challenges that lie ahead are to: 1) assess its safety profile, 2) unveil the mechanism by which the BBB opens and closes, 3) control and predict the opened BBB properties and duration of the opening and 4) assess its premise in brain drug delivery. All these challenges will be discussed, findings in both small (mice) and large (non-human primates) animals will be shown and finally the case for this technique for clinical applications will be made.
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Davis, Mellar P., and John L. Shuster. "Pain and Comorbid Psychiatric Illnesses in Elderly People." In Overlapping Pain and Psychiatric Syndromes, edited by Martin D. Cheatle, Simmie L. Foster, and Nicole K. Y. Tang. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780190248253.003.0021.

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Chronic pain is often associated with anxiety, depression, and frailty. The relationship between pain and mental illness is complex and bidirectional. In elderly people, poor self-rated health is strongly associated with pain severity, and pain-related interference with daily activities leads to depression. There is a shared neural substrate within the central nervous system (CNS) between pain and depression, which have a common neuroanatomical organization within the CNS. The close association between pain and depression means that assessment of pain should be accompanied by assessment of depression even if by the single question, “Are you depressed?” The physiological changes in aging influence the pain experience and analgesic tolerance, which diminishes in the presence of comorbidities. Tolerance to antidepressants is also diminished, with a greater risk for drug–drug interactions due to polypharmacy, which accompanies older age.
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Patel, Rachel, and Neil A. Harrison. "Immunology relevant to neuropsychiatry." In Oxford Textbook of Neuropsychiatry, edited by Niruj Agrawal, Rafey Faruqui, and Mayur Bodani. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198757139.003.0014.

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Neuroimmunology is a rapidly developing field, challenging the age-old notion of the central nervous system (CNS) as an immune-privileged site with limited immune function. In the last two decades, new technologies have demonstrated that immune processes play a critical role in normal CNS development, are integral to fundamental neurological processes, such as long-term potentiation and synaptic plasticity, and are implicated in pathologies ranging from narcolepsy to depression, schizophrenia, and Alzheimer’s disease. They indicate that immunology and mental illness are fundamentally intertwined, opening exciting new avenues for future immunotherapies tackling psychiatric disease. Here the chapter reviews basic immune mechanisms, specifically highlighting processes that are of particular relevance to neuropsychiatry. It is hoped that this chapter will serve as an accessible primer to the related fields of psychoneuroimmunology and immunopsychiatry and stimulate readers’ interest in this increasingly important and fast-evolving field.
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