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Journal articles on the topic 'Mercury intoxication'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Korchuganova, E. N., E. V. Katamanova, N. V. Slivnitsyna, I. V. Kudaeva, and P. V. Kazakova. "Differential diagnosis of sleep disorders in the presence of chronic mercury intoxication." Limnology and Freshwater Biology, no. 3 (2022): 1326–29. http://dx.doi.org/10.31951/2658-3518-2022-a-3-1326.

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Abstract. Insomnia is a widespread pathological condition in the general population, which has numerous social and medical consequences. Sleep disorders in patients with chronic mercury intoxications are much more common than in the general population. This study aimed to determine the most significant differential diagnostic criteria for sleep disorders in patients with chronic mercury intoxications for their further effective treatment. In this regard, we examined 30 patients with chronic mercury intoxications, whose age averaged 56 0.71 years, and 30 patients of the control group, whose age

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2

P., Schutzmeier, Focil Baquerizo A., Castillo-Tandazo W., Focil N., and Bose-O'Reilly S. "Efficacy of N,N'bis-(2-mercaptoethyl) isophthalamide on mercury intoxication: a randomized controlled trial." Environmental Health 17, no. 1 (2018): 15. https://doi.org/10.1186/s12940-018-0358-1.

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BACKGROUND: Chronic mercury intoxication is a severe health issue and occurs especially in gold mining communities. Common chelators used for improving mercury elimination are not everywhere available and challenged by poor cell wall penetration. This study is part of a feasibility trial and the aim was to gather first information about the efficacy of the newly developed chelator N,N'bis-(2-mercaptoethyl) isophthalamide (NBMI) on chronic mercury intoxication. METHODS: In this three-armed, placebo-controlled randomized trial, 36 miners with mercury urine levels exceeding 15 μg/l we

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3

De Bont, B., R. Lauwerys, H. Govaerts, and D. Moulin. "Yellow mercuric oxide ointment and mercury intoxication." European Journal of Pediatrics 145, no. 3 (1986): 217–18. http://dx.doi.org/10.1007/bf00446069.

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4

Naumova, Olga V., Irina V. Kudaeva, Lyudmila B. Masnavieva, and Olga A. Dyakovich. "Role of intercellular adhesion molecules and antibodies to oxidized LDL in pathogenesis of cardiovascular diseases under mercury exposure." Hygiene and sanitation 99, no. 10 (2020): 1120–26. http://dx.doi.org/10.47470/0016-9900-2020-99-10-1120-1126.

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Introduction. Exposure to mercury and its compounds can be a risk factor for the development of cardiovascular diseases. The aim of the study is to investigate the levels of antibodies to oxidized LDL, intercellular adhesion molecules sICAM-1, sVCAM-1, and VEGF in individuals exposed to mercury. Material and Methods. A cross-sectional examination was carried out using biochemical methods in persons who have come into contact with metallic mercury with a work experience of more than five years, persons with a first established diagnosis of chronic mercury intoxication, and patients with chronic

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5

Guan, Rong, and Han Dai. "Article Commentary: Chelation Therapy for Mercury Poisoning." Biochemistry Insights 2 (January 2009): BCI.S3036. http://dx.doi.org/10.4137/bci.s3036.

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Chelation therapy has been the major treatment for heavy metal poisoning. Various chelating agents have been developed and tested for treatment of heavy metal intoxications, including mercury poisoning. It has been clearly shown that chelating agents could rescue the toxicity caused by heavy metal intoxication, but the potential preventive role of chelating agents against heavy metal poisoning has not been explored much. Recent paper by Siddiqi and colleagues has suggested a protective role of chelating agents against mercury poisoning, which provides a promising research direction for broader

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6

S., Doering, Bose-O'Reilly S., and Berger U. "Essential Indicators Identifying Chronic Inorganic Mercury Intoxication: Pooled Analysis across Multiple Cross-Sectional Studies." PLoS One 11, no. 8 (2016): 1–17. https://doi.org/10.1371/journal.pone.0160323.

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BACKGROUND: The continuous exposure to inorganic mercury vapour in artisanal small-scale gold mining (ASGM) areas leads to chronic health problems. It is therefore essential to have a quick, but reliable risk assessing tool to diagnose chronic inorganic mercury intoxication. This study re-evaluates the state-of-the-art toolkit to diagnose chronic inorganic mercury intoxication by analysing data from multiple pooled cross-sectional studies. The primary research question aims to reduce the currently used set of indicators without affecting essentially the capability to diagnose chronic inorganic

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7

Baum, Carl R. "Treatment of mercury intoxication." Current Opinion in Pediatrics 11, no. 3 (1999): 265–68. http://dx.doi.org/10.1097/00008480-199906000-00018.

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8

Kanhiya Mahour. "Mercury retention after Panax ginseng treatment against mercuric chloride intoxication in hepato-haemato indices in albino rats." World Journal of Biology Pharmacy and Health Sciences 13, no. 2 (2023): 345–49. http://dx.doi.org/10.30574/wjbphs.2023.13.2.0113.

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Mercuric chloride was introduced per Os in albino rats (Rattus norvegicus) as per the LD50 (9.26 mg/kg b.w.). The assessment of mercuric chloride toxicity was done both after acute (0.926 mg/kg b.w.) and sub-acute (0.033 mg/kg b.w.) per Os treatment, while Panax ginseng was also introduced (10 mg/kg b.w.) per Os in the albino rats separately. Mercuric chloride treatment significantly increases mercury retention in liver and blood serum along with increase in liver weight, while Panax ginseng alone caused significant decrease in liver weight and mercury retention in liver and blood. Increase in

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9

Kostyniak, P. J., H. B. Greizerstein, J. Goldstein, et al. "Extracorporeal Regional Complexing Haemodialysis Treatment of Acute Inorganic Mercury Intoxication." Human & Experimental Toxicology 9, no. 3 (1990): 137–41. http://dx.doi.org/10.1177/096032719000900303.

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A 70-year-old white female presented approximately 24 h after ingesting three 475 mg tablets (1.425 g) of mercuric chloride in a suicide attempt. Acute renal failure necessitated the initiation of haemodialysis approximately 4 d after the ingestion. Treatment with BAL (2,3-dimercaptopropanol) resulted in only small increases in mercury output into dialysate. A new procedure involving the extracorporeal infusion of the chelating agent dimercaptosuccinic acid (DMSA) into the arterial blood line during haemodialysis was initiated. This procedure of Extracorporeal Regional Complexing Haemodialysis

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10

Wong, F., L. K. Hung, C. H. Wong, and P. C. Ho. "SUICIDAL MERCURY INJECTION INTO THE UPPER LIMB: A CASE STUDY." Hand Surgery 09, no. 02 (2004): 225–28. http://dx.doi.org/10.1142/s0218810404002352.

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We report a rare case of self-injection of mercury into the subcutaneous tissue of the upper limb. A multi-disciplinary management approach was adopted including cooperation between toxicologists, orthopaedic surgeons, radiologists and environment safety personnel. Surgical removal of mercury under radiological screening and systemic intoxication treated by chelating agents, namely dimercaprol and succimer. Serial serum and urine mercury levels showed an initial rise despite surgical removal and returned to normal after a prolonged period of time. Safety precautions were taken during surgery t

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11

Kudaeva, Irina V., L. B. Masnavieva, O. V. Naumova, and O. A. Dyakovich. "A SYSTEMATIC ANALYSIS OF RELATIONSHIPS BETWEEN BIOCHEMICAL INDICES IN PERSONS EXPOSED TO MERCURY." Hygiene and sanitation 97, no. 10 (2018): 990–94. http://dx.doi.org/10.18821/0016-9900-2018-97-10-990-994.

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Introduction. Changes in biochemical indices characterizing the state of lipid peroxidation processes - antioxidant protection and lipid metabolism are observed during the occupational contact with mercury. These changes play a certain pathogenic role in the formation of disorders in the nervous system during mercury intoxication. In this regard, the aim was to study the relationship between biochemical indices at different stages of the development of mercury intoxication to justify approaches to therapeutic and preventive measures. Material and methods. A prospective cohort examination of pe

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Napp, L. C., C. Moelgen, F. Wegner, et al. "Multimodal Elimination for Intoxication with a Lethal Dose of Organic Mercury." Case Reports in Critical Care 2019 (January 16, 2019): 1–4. http://dx.doi.org/10.1155/2019/4275918.

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We here report on a case of massive organic mercury intoxication in a 40-year-old man that resulted in progressive multiorgan failure. We treated the patient intravenously and enterally with the chelating agent (RS)-2,3-bis(sulfanyl) propane-1-sulfonic acid (DMPS) in addition to hemodialysis. The patient was treated for 6 weeks and could successfully be weaned from mechanical ventilation and hemodialysis. He awoke and was sent to rehabilitation, but unfortunately died 7 months later from refractory status epilepticus. Autopsy revealed severe brain atrophy consistent with organ damage from mass

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13

Jindal, Mitali, Gobind Rai Garg, Pramod Kumari Mediratta, and Mohammad Fahim. "Protective role of melatonin in myocardial oxidative damage induced by mercury in murine model." Human & Experimental Toxicology 30, no. 10 (2010): 1489–500. http://dx.doi.org/10.1177/0960327110391685.

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This study was designed to investigate the electrophysiological, hemodynamic and biochemical parameters of mercuric chloride and methylmercury exposure on cardiovascular functions and its modulation by melatonin in vivo. Wistar albino rats were divided into six group containing 10 animals each. Mercuric chloride (3.75 µM/L) in drinking water and methylmercury (0.5 mg/kg/day) through gavage, given for 1 month, induced a statistically significant increase ( p < 0.001) in left ventricular end diastolic pressure, blood and cardiac tissue mercury content and myocardial lipid peroxides compared t

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14

Kanhiya, Mahour. "Mercury retention after Panax ginseng treatment against mercuric chloride intoxication in hepato-haemato indices in albino rats." World Journal of Biology Pharmacy and Health Sciences 13, no. 2 (2023): 345–49. https://doi.org/10.5281/zenodo.8027073.

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Mercuric chloride was introduced per Os in albino rats (<em>Rattus norvegicus</em>) as per the LD<sub>50</sub> (9.26 mg/kg b.w.). The assessment of mercuric chloride toxicity was done both after acute (0.926 mg/kg b.w.) and sub-acute (0.033 mg/kg b.w.) per Os treatment, while <em>Panax ginseng</em> was also introduced (10 mg/kg b.w.) per Os in the albino rats separately. Mercuric chloride treatment significantly increases mercury retention in liver and blood serum along with increase in liver weight, while <em>Panax ginseng</em> alone caused significant decrease in liv

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15

Schrallhammer-Benkler, K., J. Ring, B. Przybilla, M. Meurer, and M. Landthaler. "Acute mercury intoxication with lichenoid drug eruption followed by mercury contact allergy and development of antinuclear antibodies." Acta Dermato-Venereologica 72, no. 4 (1992): 294–96. http://dx.doi.org/10.2340/0001555572294296.

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A 31-year-old black man was examined for evaluation of a suspected occupational disease. Three years earlier he had been suffering from acute mercury intoxication during work in a mercury recycling factory. Skin symptoms then had been a lichenoid drug eruption, patchy alopecia and stomatitis, which had all disappeared rapidly after systemic glucocorticosteroid treatment. The examination revealed positive patch test reactions to metallic mercury and inorganic mercury compounds, an elevated titre of serum antinuclear antibodies and normal IgE levels. The induction of antinuclear antibodies by me

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16

Tolkach, Pavel G., Vadim A. Basharin, Sergey V. Chepur, Darya T. Sizova, and Nicolay G. Vengerovich. "Evaluate the role of mercury chloride in the development of toxic pulmonary edema in laboratory animals during intoxication with substances of acylating action." Medical academic journal 20, no. 4 (2021): 55–61. http://dx.doi.org/10.17816/maj50133.

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Relevance. Intoxication of acylate pulmonotoxicants causes disturbance of structure and function of air-blood barrier, the output of liquid in the interstitial and alveolar space and manifestation of lung edema. Aquaporins play an important role in the transportation of fluid through the alveolar-capillary membrane, including pathological conditions. Water permeability through aquaporins is blocked by mercury ions. Using mercury chloride may reduce severity of the acute lung edema after intoxication of the pulmonotoxicants. Intention. The goal is to evaluate the role of mercury dichlorid

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17

N., Steckling, Tobollik M., Plass D., et al. "Global Burden of Disease of Mercury Used in Artisanal Small-Scale Gold Mining." Annals of Global Health 83, no. 2 (2017): 234–47. https://doi.org/10.1016/j.aogh.2016.12.005.

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BACKGROUND: Artisanal small-scale gold mining (ASGM) is the world's largest anthropogenic source of mercury emission. Gold miners are highly exposed to metallic mercury and suffer occupational mercury intoxication. The global disease burden as a result of this exposure is largely unknown because the informal character of ASGM restricts the availability of reliable data. OBJECTIVE: To estimate the prevalence of occupational mercury intoxication and the disability-adjusted life years (DALYs) attributable to chronic metallic mercury vapor intoxication (CMMVI) among ASGM gold miners globally and i

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18

Kudaeva, Irina, Olga Naumova, Olga Dyakovich, and Liudmila Masnavieva. "The role of neurochemical factors in the chronic mercury intoxication development." E3S Web of Conferences 210 (2020): 17004. http://dx.doi.org/10.1051/e3sconf/202021017004.

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The search of pathogenetic targets for the diagnosis and treatment of mercury intoxication is an actual problem. The aim of the investigation was to study the dynamics of changes in neurochemical parameters in workers in contact with mercury and in persons in the early and distant period of intoxication. A prospective cohort examination included 69 men, exposed to mercury vapors in the workplace (1) and 36 patients in the distant (2) and 18 – in initial period of chronic mercury intoxication (3). Serotonin, histamine, catecholamines and their metabolites were determined in blood plasma. The ma

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19

P., Schutzmeier, Berger U., and Bose-O'Reilly S. "Gold Mining in Ecuador: A Cross-Sectional Assessment of Mercury in Urine and Medical Symptoms in Miners from Portovelo/Zaruma." International Journal of Environmental Research and Public Health 14, no. 1 (2016): 34. https://doi.org/10.3390/ijerph14010034.

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Mercury is a toxic metal and is used in small scale gold mining. In Portovelo, Ecuador, mercury has been an environmental and health problem for decades. The target of this study was to assess the mercury concentration in the urine of miners from Portovelo/Zaruma to establish a prevalence of high values. Eight hundred and sixty-five (865) urine samples were collected and analysed for their mercury content, using cold vapor atom absorption spectroscopy. The prevalence of high mercury values (>25 μg/L) was estimated. Forty-four (44) miners with mercury levels >15 μg/L filled in a questionn

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20

Magnavita, Nicola, Mario Sabatelli, Egeria Scoditti, and Francesco Chirico. "Personalized Prevention in Mercury-Induced Amyotrophic Lateral Sclerosis: A Case Report." Applied Sciences 10, no. 21 (2020): 7839. http://dx.doi.org/10.3390/app10217839.

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Chronic exposure to low levels of mercury is involved in the development of motor neuron diseases (MND). Genetic alterations may have a crucial role in the onset and progression. We presented a case of a TANK-binding kinase 1 (TBK1)-mutated 54-year-old male worker who developed a MND due to chronic mercury exposure at work. He was employed in a chlor-alkali plant in Central Italy. After two years of employment he had acute mercury intoxication with suggestive neurological symptoms and a high urinary level of the metal. Through years, many episodes of intoxication occurred, but he continued to

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21

Celebi, Nalan, Ozgur Canbay, Ilker O. Aycan, Altan Sahin, and Ulku Aypar. "Mercury intoxication and neuropathic pain." Pediatric Anesthesia 18, no. 5 (2008): 440–42. http://dx.doi.org/10.1111/j.1460-9592.2008.02452.x.

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22

Beck, Carolyn, Bernice Krafchik, Jeffrey Traubici, and Sheila Jacobson. "Mercury Intoxication: It Still Exists." Pediatric Dermatology 21, no. 3 (2004): 254–59. http://dx.doi.org/10.1111/j.0736-8046.2004.21314.x.

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23

Ekawanti, Ardiana, and Seto Priyambodo. "Intoxication Of Mercury: Risk Factors, Pathophysiology And Its Impact For Pregnant Women In The Artisanal And Small Scale Gold Mining." Unram Medical Journal 9, no. 2 (2020): 158–65. http://dx.doi.org/10.29303/jku.v9i2.418.

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Mercury exposure of pregnant women comes from various sources including pollution from gold mining, from the consumption of fish and equipment in the neighborhood where they live. Intoxication that occurs as a result of exposure is determined by the type of mercury, route, dose, duration of exposure and age of the host. Mercury exposure in a pregnant woman not only causes intoxication for her but also affects the fetus . The fetus is a developmental phase that is very susceptible to mercury exposure of all kinds from its mother. Various effects that can arise from this exposure on the fetus in

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24

D., Pelclová, Lukáš E., Urban P., et al. "Mercury intoxication from skin ointment containing mercuric ammonium chloride." International Archives of Occupational and Environmental Health 75 (July 1, 2002): 54–59. http://dx.doi.org/10.1007/s00420-002-0349-x.

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25

Aoi, T., T. Higuchi, R. Kidokoro, et al. "An Association of Mercury with Selenium in Inorganic Mercury Intoxication." Human Toxicology 4, no. 6 (1985): 637–42. http://dx.doi.org/10.1177/096032718500400612.

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1 Energy dispersive X-ray analysis was performed on the renal tubular cells of two patients with inorganic mercury intoxication. 2 Some lysosomes of these cells consisted of unusual matrices of aggregated electron-dense grains which were positive for mercury, selenium and sulphur. 3 Though maps of the specific X-rays of both mercury and selenium coincided exactly with these lysosomes, the molecular ratio of selenium to mercury ranged between zero and 2.9. 4 It is unlikely that the trace element of selenium and exogenous inorganic mercury are deposited in the lysosomes independent of each other

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Murray, Kim M., and Jennifer C. Hedgepeth. "Intravenous Self-Administration of Elemental Mercury: Efficacy of Dimercaprol Therapy." Drug Intelligence & Clinical Pharmacy 22, no. 12 (1988): 972–75. http://dx.doi.org/10.1177/106002808802201208.

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Deliberate parenteral self-injection of mercury is extremely rare, and is associated with a high degree of mortality and morbidity. Because mercury depresses cellular enzymatic mechanisms by combining with sulfhydryl groups, soluble mercuric salts are toxic to all cells. Embolization of mercury in the lungs has been reported with varying degrees of changes in pulmonary function. Mercury causes urticaria progressing to weeping dermatitis, leukopenia, anemia, diarrhea, salivation, liver damage, and renal damage progressing to acute renal failure with anuria. Dimercaprol is an effective antidote

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27

Kyaw, Win Thiri, Xiaoxu Kuang, and Masayuki Sakakibara. "Health Impact Assessment of Artisanal and Small-Scale Gold Mining Area in Myanmar, Mandalay Region: Preliminary Research." International Journal of Environmental Research and Public Health 17, no. 18 (2020): 6757. http://dx.doi.org/10.3390/ijerph17186757.

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Increasing artisanal and small-scale gold mining (ASGM) in developing countries has raised health concerns in mining communities. A preliminary health survey was conducted in Thabeikkyin Township, Mandalay Region, Myanmar, in February 2020 to assess the health conditions of an ASGM community. Respiratory function and other clinical assessments were evaluated in miners and non-miners, and participants’ hair was analyzed for heavy metals. Respiratory function of miners was similar to that of non-miners. However, miners’ respiratory function declined with longer mining activity duration. In total

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28

YALLOUZ, ALLEGRA V., TATIANA CALIXTO, and SANDRA HACON. "Semiquantitative mercury determination in fish: a tool for poisoning prevention." Anais da Academia Brasileira de Ciências 74, no. 1 (2002): 187–91. http://dx.doi.org/10.1590/s0001-37652002000100015.

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Human exposure to mercury intoxication through contaminated fish ingestion has been well studied, mainly among Japanese population. The Brazilian population, particulaly in the Amazon region, is now in focus due to findings of fish contamination. Major health impacts caused by mercury affect mostly people who have a regular fish diet. A continuous checking for mercury content in the most consumed fish could prevent human intoxication. A simple, non-instrumental method to allow a continuous checking of the mercury content in fish was developed. Based on this method, we are proposing a preventio

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29

Boklazhenko, Elena V., and G. M. Bodienkova. "PHENOTYPIC COMPOSITION OF PERIPHERAL BLOOD LYMPHOCYTES AND THEIR COOPERATION IN PATIENTS WITH CHRONIC MERCURY INTOXICATION IN A POST-CONTACT PERIOD." Hygiene and sanitation 98, no. 10 (2019): 1091–95. http://dx.doi.org/10.18821/0016-9900-2019-98-10-1091-1095.

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Introduction. Based on the current understanding of the progression of professional chronic mercury intoxication, it is extremely important to study the regulatory activity of immunocompetent cells after the cessation contact with mercury to develop effective therapeutic measures. The purpose of the study was to study the population and subpopulation spectrum of peripheral blood lymphocytes and their cooperation in patients with chronic mercury intoxication in a distant postexposure period. Material and methods. Phenotyping of lymphocytes in the blood of the subjects was carried out by the met

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30

Hoet, Perrine, and Dominique Lison. "A Nonoccupational Source of Mercury Intoxication." Clinical Chemistry 43, no. 7 (1997): 1248. http://dx.doi.org/10.1093/clinchem/43.7.1248.

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31

Kamensky, Olivia L., Destiny Horton, Donald P. Kingsley, and Christy C. Bridges. "A Case of Accidental Mercury Intoxication." Journal of Emergency Medicine 56, no. 3 (2019): 275–78. http://dx.doi.org/10.1016/j.jemermed.2018.12.039.

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Dias, Daniel, José Bessa, Susana Guimarães, Maria Elisa Soares, Maria de Lourdes Bastos, and Helena M. Teixeira. "Inorganic mercury intoxication: A case report." Forensic Science International 259 (February 2016): e20-e24. http://dx.doi.org/10.1016/j.forsciint.2015.12.021.

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Triunfante, Paula, Maria Elisa Soares, Agostinho Santos, Susana Tavares, Helena Carmo, and Maria de Lourdes Bastos. "Mercury fatal intoxication: Two case reports." Forensic Science International 184, no. 1-3 (2009): e1-e6. http://dx.doi.org/10.1016/j.forsciint.2008.10.023.

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Muhlendahl, KarlErnstV. "Intoxication from mercury spilled on carpets." Lancet 336, no. 8730-8731 (1990): 1578. http://dx.doi.org/10.1016/0140-6736(90)93352-p.

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Chan, M. H. M., R. C. K. Cheung, I. H. S. Chan, and C. W. K. Lam. "An unusual case of mercury intoxication." British Journal of Dermatology 144, no. 1 (2001): 192–93. http://dx.doi.org/10.1046/j.1365-2133.2001.03978.x.

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Praline, Julien, Anne-Marie Guennoc, Nadège Limousin, Habib Hallak, Bertrand de Toffol, and Philippe Corcia. "ALS and mercury intoxication: A relationship?" Clinical Neurology and Neurosurgery 109, no. 10 (2007): 880–83. http://dx.doi.org/10.1016/j.clineuro.2007.07.008.

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Chernyak, Yu I., and A. P. Merinova. "CYP3A Polymorphism and Chronic Mercury Intoxication." Bulletin of Experimental Biology and Medicine 168, no. 4 (2020): 492–95. http://dx.doi.org/10.1007/s10517-020-04738-4.

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38

Arik, Elif, Olcay Gungor, Fatih Temiz, Ergul Kurutas, and Cengiz Dilber. "Evaluation of Blood Nitrotyrosine and Nitric Oxide Levels in Acute Mercury Intoxication in Children." Annals of Medical Research 31, no. 7 (2024): 486. http://dx.doi.org/10.5455/annalsmedres.2024.01.07.

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Aim: Multiple processes have been demonstrated to elucidate the biological toxicity of mercuric chloride, among which oxidative stress has been identified as a contributing factor. The superoxide radical has the potential to induce peroxidation of lipid membranes, alter the activities of proteins and antioxidant enzymes, and modulate gene transcription. Additionally, it has the ability to swiftly deactivate Nitric Oxide (NO), resulting in impairment of endothelial function and causing harm to macromolecules, membranes, and DNA by generating more harmful radicals including peroxynitrite and hyd

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39

Coelho, Cideli de Paula, and Rejane Cristina Lucco. "Zebrafish Research Model: Homeopathy in Heavy Metal Intoxication." International Journal of High Dilution Research - ISSN 1982-6206 24, no. 1 (2024): 30–31. https://doi.org/10.51910/ijhdr.v24i1.1501.

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Environmental contamination by heavy metals is a significant global concern due to its adverse impacts on human, animal, and ecosystem health. Heavy metals such as lead (Pb), mercury (Hg), cadmium (Cd), and arsenic (As) are particularly worrying because of their persistence in the environment and toxic potential. These metals can be found in water bodies, affecting the quality of drinking water and aquatic ecosystems. Exposure to these metals through water can cause acute and chronic poisoning, damage to kidneys, liver, and nervous system, and increase the risk of cancer. Homeopathic medicines

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40

Protasova, Elena N. "The functional activity of the brain in patients with occupational chronic mercury intoxication against background of the treatment of insomnia disorders." Hygiene and sanitation 103, no. 9 (2024): 973–79. http://dx.doi.org/10.47470/0016-9900-2024-103-9-973-979.

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Introduction. In patients with chronic mercury intoxication, insomnia disorders are much more common than in the general population. Persistent emotional disorders in combination with severe sleep disturbances may contribute to the progression of cognitive dysfunction in patients in the late post-exposure period of chronic mercury intoxication. The purpose of the study was to analyze the trend in electroencephalography indices and study cognitive evoked potentials in patients with occupational chronic mercury intoxication to optimize the treatment of insomnia. Materials and methods. Two groups

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Dalkiran, Tahir, Kursat Bora Carman, Velid Unsal, Ergul Belge Kurutas, Yasar Kandur, and Cengiz Dilber. "Evaluation of oxidative stress biomarkers in acute mercury intoxication." Folia Medica 63, no. 5 (2021): 704–9. http://dx.doi.org/10.3897/folmed.63.e56110.

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Objective:&nbsp;Very few studies have evaluated the association between mercury exposure and oxidative stress in humans, particularly in children. This is the first report where we aimed to determine the oxidative stress status of children who were accidently exposed to elemental mercury. Methods: In the present study, the study group was composed of 86 randomly selected children poisoned by mercury; the control group was composed of 78 children who had no history of mercury exposure. At admission, blood samples were collected. Blood superoxide dismutase activity, catalase enzyme activity,

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SINGH, NEETU. "Alterations in Acid Phosphatase Activity Following Mercuric Chloride Toxicity in Albino Rats." WORLD JOURNAL OF APPLIED SCIENCE AND RESEARCH 10-11, no. 01-02 (2021): 59. https://doi.org/10.59467/wjasr.2020-21.10-11.59.

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Metals are prevalent in our environment and pose a significant risk to human health, particularly due to their ability to accumulate in the body. We are continuously exposed to toxic heavy metals such as lead (Pb), cadmium (Cd), and mercury (Hg) through food, water, and air, respectively. Additional sources of contamination include leaching from metallic cookware and utensils, as well as personal habits such as smoking and alcohol consumption, which amplify the risk of exposure. Mercury, a silver-white metal, exists in various physical and chemical forms, making it a crucial industrial materia

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SINGH, NEETU. "Alterations in Acid Phosphatase Activity Following Mercuric Chloride Toxicity in Albino Rats." WORLD JOURNAL OF APPLIED SCIENCE AND RESEARCH 10-11, no. 01-02 (2021): 59. https://doi.org/10.59467/wjasr.2021.10-11.59.

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Metals are prevalent in our environment and pose a significant risk to human health, particularly due to their ability to accumulate in the body. We are continuously exposed to toxic heavy metals such as lead (Pb), cadmium (Cd), and mercury (Hg) through food, water, and air, respectively. Additional sources of contamination include leaching from metallic cookware and utensils, as well as personal habits such as smoking and alcohol consumption, which amplify the risk of exposure. Mercury, a silver-white metal, exists in various physical and chemical forms, making it a crucial industrial materia

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Bourgeois, M., A. Dooms-Goossens, D. Knockaert, D. Sprengers, M. Van Boven, and T. Van Tittelboom. "Mercury Intoxication after Topical Application of a Metallic Mercury Ointment." Dermatology 172, no. 1 (1986): 48–51. http://dx.doi.org/10.1159/000249292.

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Takahashi, Tetsuya, and Takayoshi Shimohata. "Vascular Dysfunction Induced by Mercury Exposure." International Journal of Molecular Sciences 20, no. 10 (2019): 2435. http://dx.doi.org/10.3390/ijms20102435.

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Methylmercury (MeHg) causes severe damage to the central nervous system, and there is increasing evidence of the association between MeHg exposure and vascular dysfunction, hemorrhage, and edema in the brain, but not in other organs of patients with acute MeHg intoxication. These observations suggest that MeHg possibly causes blood–brain barrier (BBB) damage. MeHg penetrates the BBB into the brain parenchyma via active transport systems, mainly the l-type amino acid transporter 1, on endothelial cell membranes. Recently, exposure to mercury has significantly increased. Numerous reports suggest

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Katamanova, Elena V., Elena N. Korchuganova, Natalia V. Slivnitsyna, Irina V. Kudaeva, and Oleg L. Lakhman. "The interrelation of neurophysiological, psychological, and biochemical indicators in patients with occupational chronic mercury intoxication suffering from insomnia." Russian Journal of Occupational Health and Industrial Ecology 60, no. 12 (2020): 904–10. http://dx.doi.org/10.31089/1026-9428-2020-60-12-904-910.

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Introduction. Despite the apparent connection of the existing neurological disorders and changes in the psycho-emotional sphere with sleep disorders in patients with chronic mercury intoxication (CRI), these relationships remain the least studied in the clinic neurointoxications. The study aimed to establish a connection between neurophysiological, biochemical, and psychopathological indicators in patients with occupational chronic mercury intoxication and insomnia. Materials and methods. Thirty-six patients took part in the examination in the remote period of CRI. The average age of patients

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S., Bose-O'Reilly, Schierl R., Nowak D., et al. "A preliminary study on health effects in villagers exposed to mercury in a small-scale artisanal gold mining area in Indonesia." Environmental Research 149 (May 9, 2016): 274–81. https://doi.org/10.1016/j.envres.2016.04.007.

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Cisitu is a small-scale gold mining village in Indonesia. Mercury (Hg) is used to extract gold from ore, heavily polluting air, soil, fish and rice paddy fields with Hg. Rice in Cisitu is burdened with mercury. The main staple food of the inhabitants of Cisitu is this polluted rice. Villagers were concerned that the severe diseases they observed in the community might be related to their mining activities, including high mercury exposure. Case report of the medical examinations and the mercury levels in urine and hair of 18 people with neurological symptoms. Typical signs and symptoms of chron

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Toet, A. E., A. van Dijk, T. J. F. Savelkoul, and J. Meulenbelt. "Mercury Kinetics in a Case of Severe Mercuric Chloride Poisoning Treated with Dimercapto-1-propane Sulphonate (DMPS)." Human & Experimental Toxicology 13, no. 1 (1994): 11–16. http://dx.doi.org/10.1177/096032719401300103.

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1 A case of severe mercuric chloride poisoning with clinical signs of mucosal damage of the gastrointestinal tract and anuric renal failure, is presented. The initial whole blood mercury concentration was 14,300 ?g 1-1. This concentration is supposed to be associated with fatal outcome due to multiple organ failure. 2 Because of anuric renal failure, haemodialysis was necessary. Kidney function returned to normal within 10 days. Haemodialysis proved to be ineffective with regard to total mercury elimination. 3 Treatment with DMPS was started because of very severe poisoning, anuric renal failu

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Habibia, Alif Maulida, Ferry Yundiarto, Hen Sania, Karimah Permata Arinda Putri, and Wia Bunga Ramadhan. "Penyakit Parkinson Akibat Merkuri pada Pekerja Penambangan Emas Skala Kecil." CoMPHI Journal: Community Medicine and Public Health of Indonesia Journal 2, no. 1 (2021): 170–76. http://dx.doi.org/10.37148/comphijournal.v2i1.31.

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Introduction: Parkinson's disease is one of the fastest-growing diseases in terms of prevalence, disability, and mortality. Exposure to environmental toxic substances, such as mercury (Hg) can increase the risk of neuronal damage and neurodegenerative diseases. Purpose: The purpose of writing this article is to determine the impact of mercury exposure on the incidence of Parkinson's disease in small-scale gold mining workers. Methods: The author searches for material through the Google Scholars, NCBI, and ScienceDirect search pages by limiting the journals published in 2015-2020. In this revie

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Habibia, Alif Maulida, Ferry Yundiarto, Hen Sania, Karimah Permata Arinda Putri, and Wia Bunga Ramadhan. "Penyakit Parkinson Akibat Merkuri pada Pekerja Penambangan Emas Skala Kecil." CoMPHI Journal: Community Medicine and Public Health of Indonesia Journal 1, no. 3 (2021): 170–76. http://dx.doi.org/10.37148/comphijournal.v1i3.31.

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Introduction: Parkinson's disease is one of the fastest-growing diseases in terms of prevalence, disability, and mortality. Exposure to environmental toxic substances, such as mercury (Hg) can increase the risk of neuronal damage and neurodegenerative diseases. Purpose: The purpose of writing this article is to determine the impact of mercury exposure on the incidence of Parkinson's disease in small-scale gold mining workers. Methods: The author searches for material through the Google Scholars, NCBI, and ScienceDirect search pages by limiting the journals published in 2015-2020. In this revie

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