Relevant bibliographies by topics / Met deficiency

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Met deficiency'

Author: Grafiati
Published: 4 June 2021
Last updated: 15 February 2022

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Journal articles on the topic "Met deficiency"

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Zhang, Zhilei, Yanyun Wang, Dingyuan Ma, Wei Cheng, Yun Sun, and Tao Jiang. "Analysis of five cases of hypermethioninemia diagnosed by neonatal screening." Journal of Pediatric Endocrinology and Metabolism 33, no. 1 (January 28, 2020): 47–52. http://dx.doi.org/10.1515/jpem-2019-0285.

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AbstractBackgroundHypermethioninemia is a group of diseases with elevated plasma methionine (Met) caused by hereditary and non-hereditary factors, although it could also be caused by administration of the amino acid Met. Among these, the disease caused by methionine adenosyltransferase (MAT) I/III deficiency is the most common, and is characterized by persistent, isolated hypermethioninemia as well as slightly elevated homocysteine. S-adenosylmethionine is the product of Met, which can be used as a direct methyl donor of many substances, such as choline and nucleotide, and essential in the development of the body. Among the patients, most have no symptoms, and a small number have central nervous system complications with high levels of plasma Met, including mental retardation, cognitive impairment and special breathing odor.MethodsIn this study, five cases of MAT I/III deficiency were diagnosed and retrospectively analyzed among 220,000 newborns. Patients with high Met levels received a Met-restricted diet treatment. Results and conclusionsMAT I/III deficiency is a common reason for Met elevation in neonatal screening by tandem mass spectrometry (MS/MS), which needs long-term follow-up except for these patients with explicitly benign mutations.
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Aroda, Vanita R., Sharon L. Edelstein, Ronald B. Goldberg, William C. Knowler, Santica M. Marcovina, Trevor J. Orchard, George A. Bray, et al. "Long-term Metformin Use and Vitamin B12 Deficiency in the Diabetes Prevention Program Outcomes Study." Journal of Clinical Endocrinology & Metabolism 101, no. 4 (April 1, 2016): 1754–61. http://dx.doi.org/10.1210/jc.2015-3754.

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Abstract Context: Vitamin B12 deficiency may occur with metformin treatment, but few studies have assessed risk with long-term use. Objective: To assess the risk of B12 deficiency with metformin use in the Diabetes Prevention Program (DPP)/DPP Outcomes Study (DPPOS). Design: Secondary analysis from the DPP/DPPOS. Participants were assigned to the placebo group (PLA) (n = 1082) or the metformin group (MET) (n = 1073) for 3.2 years; subjects in the metformin group received open-label metformin for an additional 9 years. Setting: Twenty-seven study centers in the United States. Patients: DPP eligibility criteria were: elevated fasting glucose, impaired glucose tolerance, and overweight/obesity. The analytic population comprised participants with available stored samples. B12 levels were assessed at 5 years (n = 857, n = 858) and 13 years (n = 756, n = 764) in PLA and MET, respectively. Interventions: Metformin 850 mg twice daily vs placebo (DPP), and open-label metformin in the metformin group (DPPOS). Main Outcome Measures: B12 deficiency, anemia, and peripheral neuropathy. Results: Low B12 (≤ 203 pg/mL) occurred more often in MET than PLA at 5 years (4.3 vs 2.3%; P = .02) but not at 13 years (7.4 vs 5.4%; P = .12). Combined low and borderline-low B12 (≤ 298 pg/mL) was more common in MET at 5 years (19.1 vs 9.5%; P < .01) and 13 years (20.3 vs 15.6%; P = .02). Years of metformin use were associated with increased risk of B12 deficiency (odds ratio, B12 deficiency/year metformin use, 1.13; 95% confidence interval, 1.06–1.20). Anemia prevalence was higher in MET, but did not differ by B12 status. Neuropathy prevalence was higher in MET with low B12 levels. Conclusions: Long-term use of metformin in DPPOS was associated with biochemical B12 deficiency and anemia. Routine testing of vitamin B12 levels in metformin-treated patients should be considered.
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Benight, Nancy M., Barbara Stoll, Shaji Chacko, Vanessa R. da Silva, Juan C. Marini, Jesse F. Gregory, Sally P. Stabler, and Douglas G. Burrin. "B-vitamin deficiency is protective against DSS-induced colitis in mice." American Journal of Physiology-Gastrointestinal and Liver Physiology 301, no. 2 (August 2011): G249—G259. http://dx.doi.org/10.1152/ajpgi.00076.2011.

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Vitamin deficiencies are common in patients with inflammatory bowel disease (IBD). Homocysteine (Hcys) is a thrombogenic amino acid produced from methionine (Met), and its increase in patients with IBD indicates a disruption of Met metabolism; however, the role of Hcys and Met metabolism in IBD is not well understood. We hypothesized that disrupted Met metabolism from a B-vitamin-deficient diet would exacerbate experimental colitis. Mice were fed a B6-B12-deficient or control diet for 2 wk and then treated with dextran sodium sulfate (DSS) to induce colitis. We monitored disease activity during DSS treatment and collected plasma and tissue for analysis of inflammatory tissue injury and Met metabolites. We also quantified Met cycle activity by measurements of in vivo Met kinetics using [1-13C-methyl-2H3]methionine infusion in similarly treated mice. Unexpectedly, we found that mice given the B-vitamin-deficient diet had improved clinical outcomes, including increased survival, weight maintenance, and reduced disease scores. We also found lower histological disease activity and proinflammatory gene expression (TNF-α and inducible nitric oxide synthase) in the colon in deficient-diet mice. Metabolomic analysis showed evidence that these effects were associated with deficient B6, as markers of B12 function were only mildly altered. In vivo methionine kinetics corroborated these results, showing that the deficient diet suppressed transsulfuration but increased remethylation. Our findings suggest that disrupted Met metabolism attributable to B6 deficiency reduces the inflammatory response and disease activity in DSS-challenged mice. These results warrant further human clinical studies to determine whether B6 deficiency and elevated Hcys in patients with IBD contribute to disease pathobiology.
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Garcia-Carretero, Rafael, Luis Vigil-Medina, Oscar Barquero-Perez, Inmaculada Mora-Jimenez, Cristina Soguero-Ruiz, Rebeca Goya-Esteban, and Javier Ramos-Lopez. "Logistic LASSO and Elastic Net to Characterize Vitamin D Deficiency in a Hypertensive Obese Population." Metabolic Syndrome and Related Disorders 18, no. 2 (March 1, 2020): 79–85. http://dx.doi.org/10.1089/met.2019.0104.

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Weight, Lindsay M., Peter Jacobs, and Timothy D. Noakes. "Dietary iron deficiency and sports anaemia." British Journal of Nutrition 68, no. 1 (July 1992): 253–60. http://dx.doi.org/10.1079/bjn19920082.

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In order to determine whether dietary inadequacies can explain the sub-optimal iron status widely documented in endurance-trained athletes, the food intake records of Fe-deficient and Fe-replete distance runners and non-exercising controls of both sexes were analysed. In all the male study groups the mean dietary Fe intake met the recommended dietary allowances (RDA; > 10 mg/d (US) Food and Nutrition Board, 1989). However, both female athletes and controls failed to meet the RDA with regard to Fe (< 15 mg/d) and folate (< 200 μg/d). There was no difference in the total Fe intakes of Fe-deficient and Fe-replete athletes and the controls of each sex. However, Fe-deficient male runners, but not female runners, consumed significantly less haem-Fe (P= 0.048) than their comparative groups. This suggests that the habitual consumption of Fe-poor diets is a factor in the aetiology of athletes' Fe deficiency.
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Wang, J., K. Cui, T. Ma, F. Zhang, S. Q. Wang, Y. Tu, Q. Y. Diao, and N. F. Zhang. "Effects of dietary methionine deficiency followed by replenishment on the growth performance and carcass characteristics of lambs." Animal Production Science 59, no. 2 (2019): 243. http://dx.doi.org/10.1071/an16643.

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Twelve pairs of male twin lambs were used to assess the effects of dietary methionine (Met) deficiency followed by replenishment on lamb growth performance and carcass characteristics. All lambs were weaned at 7 days of age and divided into the Control (CON) group and Met deficiency (MD) group. From 8 to 56 days of age, the lambs in the CON group were fed a milk replacer and starter feed containing 0.91% and 0.60% Met, respectively, whereas the lambs in the MD group were fed with a milk replacer and starter feed containing 0.21% and 0.20% Met, respectively. All lambs were fed a starter feed containing 0.60% Met from 57 to 84 days of age. Six twin pairs were slaughtered at 56 and 84 days of age, and their organ weights and carcass traits were measured. During 8 to 56 days of age, the Met-deficient diet decreased (P &lt; 0.05) Met intake, average daily gain, feed conversion ratio, shrunk bodyweight, empty bodyweight, hot carcass weight, and the apparent digestibility of crude protein, ether extract and neutral detergent fibre; however, no differences were detected in dressing percentage or in the percentage of visceral organ weight to shrunk bodyweight between the groups (P &gt; 0.05). During the follow-up Met replenishment stage, no differences in growth performance, nutrient digestibility, carcass characteristics, and percentages of visceral organs to shrunk bodyweight were found between the groups (P &gt; 0.05). In conclusion, dietary Met deficiency in early life retarded the growth and development of lambs. Growth rate was not retarded during the 28 days of subsequent Met replenishment, but the difference in bodyweight between the groups remained.
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Amirbaigloo, Alireza, Farhad Hosseinpanah, Farzaneh Sarvghadi, Maryam Tohidi, Parvin Sarbakhsh Eskandary, and Fereidoun Azizi. "Absence of Association Between Vitamin D Deficiency and Incident Metabolic Syndrome: Tehran Lipid and Glucose Study." Metabolic Syndrome and Related Disorders 11, no. 4 (August 2013): 236–42. http://dx.doi.org/10.1089/met.2012.0121.

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Henry, Rohan K. "Growth Hormone Deficiency and Nonalcoholic Fatty Liver Disease with Insights from Humans and Animals: Pediatric Implications." Metabolic Syndrome and Related Disorders 16, no. 10 (December 2018): 507–13. http://dx.doi.org/10.1089/met.2018.0056.

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Vreugdenhil-Hovenier, Priscilla. "De Organisatie Van de Woordenschat Bij Kinderen Met Een Cognitieve Deficientie." Toegepaste Taalwetenschap in Artikelen 56 (January 1, 1997): 23–34. http://dx.doi.org/10.1075/ttwia.56.03vre.

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With adults, many words in the memory are ordered on the basis of semantic relations. Especially co-ordinative relations between words (dog-cat) are strongly represented. In a child's lexicon, this arrangement is not clearly present yet. Studies into the development of lexical organization of normally developed children by means of word association tasks, have shown that co-ordinative relations are already present in a child's lexicon at an early stage (Eibers et al., 1993). However, these can only be addressed directly in a certain context (Contrast Association). The co-ordinations are not available independently yet (Free Association). This suggests that co-ordinative relations of adults have a contrastive origin. In these studies the development of lexical organization of children with a cognitive deficiency has been looked at. The group to be examined consists of 42 pupils. It has been examined whether their lexical organization deviates from normally developed children. A comparison with adults has been made as well. These studies consist of two kinds of word association: Free Association and Contrast Association. The most important findings point out that children with a cognitive deficiency do not deviate from normally developed children as far as their reactions in Free Association are concerned. Both groups of children mention the same small number of co-ordinations. With respect to the Contrast Association, however, the children with a cognitive deficiency mention considerably less co-ordinations than normally developed children. With the children with a cognitive deficiency, this results in a smaller number of co-ordinations in the Contrast Association, but apparently not in a smaller number of co-ordinations in the Free Association.
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Ruan, Tao, Lingjun Li, Yingnan Lyu, Qin Luo, and Bangyuan Wu. "Effect of methionine deficiency on oxidative stress and apoptosis in the small intestine of broilers." Acta Veterinaria Hungarica 66, no. 1 (March 2018): 52–65. http://dx.doi.org/10.1556/004.2018.006.

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The aim of this study was to investigate the effects of methionine (Met) deficiency on antioxidant functions (in the duodenal, jejunal and ileal mucosa) and apoptosis in the duodenum, jejunum and ileum of broiler chickens. A total of 120 one-day-old Cobb broilers were divided into two groups and fed a Metdeficient diet and a control diet, respectively, for six weeks. The activities of superoxide dismutase (SOD), catalase (CAT) and glutathione peroxidase (GSH-Px), the ability to inhibit hydroxyl radicals, and glutathione (GSH) content were significantly decreased in the Met-deficient group compared to the control. In contrast, malondialdehyde (MDA) content was significantly higher in the Met-deficient group. As measured by terminal deoxynucleotidyl transferase 2’-deoxyuridine 5’- triphosphate dUTP nick end-labelling (TUNEL) and flow cytometry (FCM), the percentages of apoptotic cells were significantly increased. In conclusion, dietary Met deficiency can cause oxidative stress and then induce increased apoptosis in the intestine. Oxidative stress contributes to intestinal apoptosis. This results in the impairment of local intestinal mucosal immunity due to oxidative stress and apoptosis in the small intestine. The results of this study provide new experimental evidence for understanding the negative effects of Met deficiency on mucosal immunity or the functions of other immune tissues.
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Dissertations / Theses on the topic "Met deficiency"

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Zhang, Shuai. "Physiological and Biochemical Aspects of Methionine Isomers and Precursors in Broilers." Diss., Virginia Tech, 2016. http://hdl.handle.net/10919/71816.

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Methionine (Met) is an essential amino acid for animals and also the first limiting amino acid in a broiler diet. The dietary supplemental Met sources include the natural isoform L-methionine (L-Met), the synthetic form DL-methionine (DL-Met) and the synthetic Met precursor DL-2-hydroxy-4 (methylthio)-butanoic acid (DL-HMTBA). The objective of this dissertation was to determine the effect of different dietary Met source supplementation and Met deficiency on a series of physiological and biochemical aspects, including growth performance, global DNA methylation and methyltransferase activity, blood antioxidant profile (e.g., acute phase protein, leukocyte count), intestinal nutrient transporter gene expression, Met converting enzyme gene expression and activity, oxidative stress markers, and a potential pathway related to amino acid signaling. To achieve this goal, male Cobb-500 broilers were raised from day of hatch (d0) to d35 post-hatch and fed a diet deficient in methionine and cysteine (Met + Cys) (control) or the same diet supplemented with 0.22% DL-Met, 0.22% L-Met or 0.31% DL-HMTBA (to provide 0.22% DL-Met equivalent) to meet Met + Cys requirements. Tissues (liver, breast muscle, duodenum, jejunum and ileum) and blood samples were collected at various ages from d0 to d35 for analysis. Met supplementation significantly improved body weight gain and feed efficiency compared to the Met deficient group, but no differences were observed among DL-Met, L-Met and DL-HMTBA for growth performance parameters (P > 0.05). Met supplementation had no effect on red blood cell packed cell volume, white blood cell differential count, hepatic total DNA methylation, DNA methyltransferase and Met oxidase activity, and had limited effects on activation of p70S6K, a key amino acid signaling protein (P > 0.05). Although dietary Met sources did not change oxidative status of the treated chickens, both L-Met and DL-Met but not DL-HMTBA supplementation decreased the level of acute phase protein serum amyloid A compared to the control group (P > 0.05). The effect of Met supplementation on gene expression of nutrient transporters and Met converting enzymes were complex and dynamic. Most of the target genes demonstrated tissue- and development-dependent expression patterns, with few significant treatment effects observed. L-Met and DL-Met but not DL-HMTBA supplementation enhanced the neutral amino acid transporters ATB0,+ and B0AT gene expression in various small intestinal segments. All three Met sources increased monocarboxylic acid transporter (MCT1) gene expression in the jejunum. DL-HMTBA and L-Met fed chickens showed greater hepatic L-HMTBA oxidase (HAO1) gene expression. DL-Met increased glutamic-oxaloacetic transaminase 2 (GOT2) gene expression in the duodenum. An in vitro study with tissue explants, however, did not demonstrate a similar gene expression pattern as that in the in vivo study. Lastly, RNA sequencing results illustrated that Met deficiency could lead to many differentially expressed genes but different supplemental Met sources had no influence on hepatic gene expression profiles. In conclusion, as common dietary supplemental Met sources, L-Met, DL-Met and DL-HMTBA exhibited similarity in impacting intestinal amino acid/peptide/monocarboxylic acid transporter gene expression and Met converting enzyme activity. The regulatory roles of Met as an antioxidant and nutrient signaling in cell metabolism were not affected by different dietary supplemental Met sources.
Ph. D.
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Shelby, R. Dennis. "The experiences of gay men whose partners contract acquired immune deficiency syndrome." Click here for text online. The Institute of Clinical Social Work Dissertations website, 1989. http://www.icsw.edu/_dissertations/shelby_1989.pdf.

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Dissertation (Ph.D.) -- The Institute for Clinical Social Work, 1989.
A dissertation submitted to the faculty of the Institute of Clinical Social Work in partial fulfillment for the degree of Doctor of Philosophy.
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Rashed, Ismail Ismail Ibrahim. "The association between vitamin D deficiency and tuberculosis : Effects of different levels of vitamin D deficiency - A meta-analysis." Thesis, Södertörns högskola, Institutionen för naturvetenskap, miljö och teknik, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:sh:diva-38922.

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Aim: To conduct a systematic review and meta-analysis of all published studies studying the risk of vitamin D deficiency in tuberculosis patients and in healthy controls. Additionally, subgroup meta-analysis was performed based on the level of vitamin D to test the risk in TB groups and in healthy groups. Methods: Pubmed was searched for observational studies in human and English that discussed the association between risk of low serum vitamin D and TB. Meta-analysis was performed on all relevant studies combined and for subgroups of each vitamin D level. Results: 22 studies were selected and pooled in the analysis. The results were consistent with previous studies examining the same risk. The overall log risk ratio (log RR) of low vitamin D was significantly higher in TB patients 1.68 times than healthy controls. In 4 subgroup meta- analyses based on vitamin D level below (20 nmol/L, 30 nmol/L, 50 nmol/L, and 75 nmol/L), the risk of having low vitamin D in TB patients was (1.82, 2.89, 1.38, 1.32) respectively. That subgroup analysis showed more clearly the higher RR were below 20 and 30 nmol/L. The smallest RR was at 75 nmol/L level. Conclusion: This study verified the association between risk of low vitamin D level and TB development. It also clarified that, the risk increased by decreasing vitamin D level.
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DUFFY, PAM REID. "SURVIVING SURVIVAL: A THEORY OF LIVING WITH THE THREAT OF AIDS (IMMUNE DEFICIENCY)." Diss., The University of Arizona, 1987. http://hdl.handle.net/10150/184231.

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The purpose of this study was to generate a grounded theory explaining the social and psychological processes employed by gay men in living with the threat of Acquired Immunodeficiency Syndrome (AIDS). As members of the high risk group for AIDS, gay men are living with a prolonged threat of unprecedented complexity which has unknown demands on health. An exhaustive preliminary literature review revealed a dearth of theoretical or empirical data addressing this problem. Theory discovery was accomplished using the grounded theory methodology. Two major data collection procedures were utilized: The conduct of increasingly structured interviews of healthy gay men, and an ongoing, progressive literature and media search. Data were sampled theoretically, as guided by the emergent theory. The constant comparative method of analytic induction was used for the analysis of data, in order to identify the elements and structure of the theory. Multiple procedures were incorporated into the analysis to ensure its trackability and credibility. A basic social process, Surviving Survival, was identified as the core category of the theory. Surviving Survival is the continuous process used by gay men to ensure mortal survival as well as outlive the extremity of the AIDS threat. The process is comprised of three subcategories: Vigilance, Safeguarding, and Balancing. The subcategory of Vigilance explains the work of monitoring the threat of AIDS and has both personal and social components. Safeguarding explains the behavior of protecting self and others from the AIDS threat, including AIDS' eventuality. The subcategory of Safeguarding contains Safer Sex, Reassuring Others, and Forecasting. The subcategory of Balancing explains efforts to conserve energy required to sustain affirmation of life and living in spite of the AIDS threat. In continuous interaction, the subcategories of Surviving Survival are interwoven into multiple aspects of gay living. This theory explains the profound impact AIDS has on the mental health of gay men, who both survive and perceive the extremity of the AIDS threat on a daily basis. These findings provide a knowledge base for the nursing discipline in becoming an informed and informing resource for the men who are outliving the threat of AIDS.
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Ismail, Rana H. "Vitamin D Deficiency and Immune Function in African American, HIV-Infected Men." ScholarWorks, 2015. https://scholarworks.waldenu.edu/dissertations/1222.

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Vitamin D deficiency is common in individuals diagnosed with HIV and is known for its detrimental health effects. Its recognition as a potent immune-modulator with possible immune health implications in HIV disease progression was the main impetus for this study. The association between Vitamin D and CD4 count falls short of being consistent and is too weak to allow conclusions. Similarly, the literature is inconsistent with regard to the impact of Vitamin D supplementation on CD4. This observational, retrospective chart review study aimed to explore the relationship between Vitamin D deficiency and CD4 count/percent, and to evaluate whether changes in Vitamin D levels after supplementation corresponds with significant changes in CD4 count/percent in a cohort of African American, HIV-infected men who attended an HIV clinic in southeast Michigan (N = 70). The conceptual framework was based on the role of Vitamin D in regulating the immune responses through Vitamin D nuclear receptors on the CD4 cells. It postulated that an increase in Vitamin D level might enhance immune function, promote cellular anti-inflammatory state, and decelerate CD4 destruction. Data analysis included descriptive statistics, bivariate correlation, logistic and linear regression, t test, repeated measures ANOVA, and ANCOVA. Findings of the study did not support the hypotheses of significant correlation between Vitamin D and CD4 count (p = 0.458) and percent (p = 0.776), or of any impact of supplementation on CD4 count (p = 0.216) and percent (p = 0.918). Social change implications include providing health professionals, researchers, and policymakers with knowledge to tailor health promotion interventions aiming to reduce Vitamin D deficiency in favor of improving the overall health of HIV patients, especially high-risk groups such as African American HIV-infected patients.
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Törnroth, John, and Mohammed Alshaer. "Upplevelser av egenvård hos personer med hjärtsvikt." Thesis, Hälsohögskolan, Högskolan i Jönköping, HHJ, Avd. för omvårdnad, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:hj:diva-35779.

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Bakgrund: I dagens samhälle är det många individer som lider av hjärtsvikt och det är många patienter med hjärtsvikt som behöver vård. Patienter med hjärtsvikt upplever både fysiska och psykiska påfrestningar som påverkar deras välbefinnande.  För patienter med hjärtsvikt är egenvård viktig del för att möjliggöra att vardagen fungerar. Syfte: Att beskriva upplevelser av egenvård hos patienter med hjärtsvikt. Metod: litteraturöversikt med induktiv ansats. Databaserna CINHALL och MEDLINE användes och tretton kvalitativa artiklar inkluderades i litteraturöversikt. Resultat: Litteraturöversiktens resultat beskriver patienters upplevelse av egenvård. Bland annat framkommer att motivation är viktigt för att utöva egenvård. Patienter uppskattade stödet de fick från familj, vänner och vårdpersonal i samband med egenvård. Utifrån resultatet framkommer egenvårds- och insiktsbrist hos patienter med hjärtsvikt, vilket leder till dålig följsamhet till egenvård. Resultatet presenteras i följande tre huvudrubriker: motivation, stöd, kunskap. Slutsats: Patienter med hjärtsvikt har hinder och begränsningar i samband med egenvård. Patienterna har också bristande insikt kring hjärtsvikt och egenvård vilket medför nedsatt välbefinnande. Patienter med hjärtsvikt och bristande egenvård är i stort behov av stöd, vägledning och kunskap i syftet att öka patientens makt och självbestämmande över situationen. Sjuksköterskor bör utifrån litteraturöversiktens resultat ha kunskap om värdet att förstå patientens upplevelse av egenvård i samband med hjärtsvikt för att kunna anpassa vården tillsammans med patienter och utefter individens behov.
Background: In today's society, there are many people who suffer from heart failure and many people with heart failure who need care. Patients with heart failure experience both physical and mental stresses that also affect the quality of life. For patients with heart failure, self-care is an important part for enabling a functioning life. Aim: To describe experiences of self-care in people with heart failure. Method: Literature overview with inductive approach. Thirteen qualitative articles were included and downloaded through the CINHALL and MEDLINE databases. Result: The essay result describes patients' self-care experience and shows that motivation is a driving force in self-care practice. Patients appreciated the support they received from family, friends and care personnel in connection with self-care. Based on the results, self-awareness and lack of knowledge results in poor compliance with self-care. The results are presented in the following three main headings: motivation, knowledge and support. Conclusion: The essay showed that patients with heart failure have barriers and constraints associated with self-care. Another problem in patients was lack of knowledge about heart failure and self-care which results in reduced quality of life. Patients with heart failure and lack of self-care are in great need of support, guidance and knowledge in order to increase patient's power and self-determination over the situation. Nurses should understand the patient's perception of self-care in heart failure in order to be able to adapt care together with the patient and to the needs of the individual person.
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Wallin, Marianne. "Musiken skapar en bro mellan människor : en intervjustudie med personal inom Daglig Verksamhet." Thesis, Kungl. Musikhögskolan, Institutionen för musik, pedagogik och samhälle, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:kmh:diva-1304.

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Studien är en undersökning om hur personal beskriver musikverksamheten i den omsorgsgren som erbjuder sysselsättning för personer med grav till lindrig intellektuell funktionsnedsättning. Studien söker också svar på vilka reflektioner personalen har kring musikverksamhet. Uppsatsens problemområde uppstår i skärningspunkten mellan det praktiska, vardagliga arbetet med musikanvändning i mötet med personer med intellektuell funktionsnedsättning och uppsatsens problemområde utifrån det musikterapeutiska ämnesområdets teori och tillämpning. Studien har den kvalitativa forskningsintervjun som metod. Det teoretiska vetenskapsperspektivet har hämtat inspiration ur hermeneutiken. Forskningsresultaten visar på att det finns en kunskap som helt bygger på beprövad erfarenhet bland den grupp av individer som undersökningen omfattar och att det finns ett uttalat behov av en kompletterande teoretisk baserad kunskap. Forskningsresultaten belyser olika perspektiv på reflektioner kring musikanvändning. Dessa resultat har sedan bearbetats under diskussionsdelen i ljuset av de teoribildningar som presenteras under ämnesområdet.
The study is a research project on how personnel appreciate music in the branch of care that provides activities for persons suffering from severe to mild intellectual deficiency. The study also attempts to find answers to what kind of observations the personnel have concerning the use of music as activity. The problem area of the study arises from the meeting point between on the one hand the practical, everyday application of music on persons with reduced intellectual capacity. On the other hand, the study will illustrate the adaptional possibilities of the theories of the topic area. The research results show that there is information which is completely based on tested experience among the group of individuals included in the research project, and further that there is an explicit need for supplementary theoretically based knowledge. The research results point to different perspectives concerning observations of the application of music. These results have later in the study been treated in the discussion part and shed light on by theories which are presented in the topic area. The method being used in the study is the qualitative research interview. The theoretical scientific perspective is taken from the hermeneutics.
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Ortman, Kerstin. "Organic vs. inorganic selenium in farm animal nutrition with special reference to supplementation of cattle /." Uppsala : Swedish Univ. of Agricultural Sciences (Sveriges lantbruksuniv.), 1999. http://epsilon.slu.se/avh/1999/91-576-5411-5.pdf.

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Wikström, Daniel, and Ida Sandeberg. "Accessibility, With or Without Colour : A qualitative look on existing accessibility guidelines for colour vision deficiency and its effect on Swedish e-health services." Thesis, Tekniska Högskolan, Jönköping University, JTH, Datateknik och informatik, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:hj:diva-48237.

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Purpose WCAG is a set of guidelines to assist in developing websites accessible for everyone, for example people with colour vision deficiency (CVD). However, there are few guidelines relevant for CVD and with increasing dependant on websites the importance of accessibility on these websites increases as well. The purpose of the research study is therefore to investigate the impact the relevant guidelines for CVD in WCAG 2.1 has on Swedish health care websites and what the consequences are for people who live with CVD. Method An initial literature study was conducted to gain knowledge of previous findings and also evaluate the best approach for the research study. The research study is based on a qualitative approach to gain a deep understanding of the area and to gain insight into the feelings and experiences of the people affected. The methods used to gain the necessary knowledge are an evaluation of 1177.se and e-tjanster.1177.se also observations and interviews around these two websites. Findings The results show that WCAG 2.1 has a positive impact on Swedish health care websites, as well as on users with CVD. From evaluations, interviews and observations it is concluded that 1177.se and e-tjanster.1177.se are easy to perceive for people with CVD. Implications This research study was limited to the mobile version of the websites. However, the results could be different if the research was conducted on the desktop version due to the different structure of the websites and the inclusion of a cursor on desktop. The evaluation could have been affected by the sampling of pages while the interviews and observations should not have been. Limitations The research study was limited to Swedish health care website 1177.se and its subdomain etjanster. 1177.se. Further it was decided to evaluate the mobile version of these websites, although the elements should look similar on desktop and mobile. While not the initial intention, due to over representation of red-green CVD the study was also limited to people with red-green CVD. Also, the experience of browsing the web on a mobile device was considered to reduce the variables for frustrations. Further research For further research, another selection of WCAG guidelines could be considered to evaluate etjanster. 1177.se since the results shows that 1177.se’s and e-tjanster.1177.se's weaknesses seems to be in the structure of the website. A study researching the “blue-yellow colour blindness” compared to websites could contribute with new, relevant knowledge as well.
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Selvin, David. "Regulation of Myoplasmic Ca2+ During Fatigue in KATP Channel Deficient FDB Muscle Fibres." Thèse, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/26174.

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It is known that muscles that lack KATP channel activity generate much greater unstimulated [Ca2+]i and force than normal muscles during fatigue. The increase in unstimulated force in KATP channel deficient muscles is abolished by a partial inhibition of L-type Ca2+ channels, suggesting that it is due to a Ca2+ influx through L-type Ca2+ channels and a subsequent increased myoplasmic Ca2+. However, there is also evidence that the increase in resting force is abolished by NAC, a ROS scavenger. The objective of this study was to reconcile these observations by studying the hypothesis that “the increase in resting [Ca2+]i during fatigue in KATP channel deficient muscles starts with an excess Ca2+ influx through L-type Ca2+ channels, followed by an excess ROS production that causes a further increase in resting [Ca2+]i”. To test the hypothesis, single FDB fibres were fatigued with one tetanic contraction/sec for 180 sec. KATP channel deficient fibres were obtained i) by exposing wild type muscle fibers to glibenclamide, a KATP channel blocker and ii) by using fibres from Kir6.2-/- mice, which are null mice for the Kir6.2 gene that encodes for the protein forming the channel pore. Verapamil, a L-type Ca2+ channel blocker, applied at 1 μM, significantly reduced resting [Ca2+]i during fatigue in glibenclamide-exposed wild type fibres. NAC (1 mM) also reduced resting [Ca2+]i in glibenclamide-exposed muscles. The results suggest that the increase in resting [Ca2+]i during fatigue in KATP channel deficient FDB fibres is due to an influx through L-type Ca2+ channels, and an excess ROS production.
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Books on the topic "Met deficiency"

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translator, You Yixuan, ed. Mei de qi ji: The magnesium miracle / Carolyn Dean. Taibei Shi: Shi zi wen hua shi ye you xian gong si, 2015.

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Brahm, Amanda J., and Robert A. Hegele. Monogenic Chylomicronemia: Deficiency of Lipoprotein Lipase and Related Factors. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0033.

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Monogenic chylomicronemia is an autosomal recessive condition characterized by severely elevated fasting triglyceride that carries lifelong elevated risk of developing pancreatitis. The majority of cases are caused by mutations in the LPL gene encoding lipoprotein lipase, the enzyme primarily responsible for chylomicron clearance. Mutations in genes encoding associated proteins (APOC2, APOA5, GPIHBP1 and LMF1) may also present with a very similar phenotype. Current management, which includes restriction of dietary fat intake and standard pharmacologic interventions, has met with limited success, but new therapies under development may prove to be more effective.
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Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. Vitamin B3 (niacin) in pregnancy and breastfeeding. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0009.

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Niacin (as nicotinamide) is a component of coenzyme systems that function in the reductive biosynthesis of fatty acids and steroids, including cholesterol, and are involved in cell signalling. Niacin deficiency is rare, as the daily requirement can usually be met by food sources, and also via synthesis from tryptophan, which is present in dietary proteins. The prevalence of niacin deficiency is higher in populations consuming mainly corn or sorghum as a dietary staple. Corn contains niacin, but only in a bound form that is nutritionally unavailable. The additional needs for niacin during pregnancy are mirrored by the increased energy intake needs, and dietary supplementation is only necessary in cases of overall poor nutritional intake.
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Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. Vitamin B2 (riboflavin) in pregnancy and breastfeeding. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0008.

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Riboflavin is a cofactor for enzymes involved in energy generation, biosynthesis, detoxification, and electron-scavenging pathways, as well as in the metabolism of other B vitamins. Deficiency is rare in developed countries; it is encountered almost invariably in combination with deficit of other B vitamins in areas of poor overall nutrition. Deficiency is endemic in populations whose staple diet consists of rice and wheat, with low or no consumption of meat and dairy products. Infants of riboflavin-deficient mothers tend to be deficient themselves at birth and remain deficient through breastfeeding and weaning. To ensure adequate riboflavin supply in both mother and infant, an increase in dairy products and/or meat consumption, particularly during lactation, may be necessary in some women.
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1954-, Jones T. Hugh, ed. Testosterone deficiency in men. Oxford: Oxford University Press, 2008.

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Jones, Hugh, ed. Testosterone Deficiency in Men. Oxford University Press, 2008. http://dx.doi.org/10.1093/med/9780199545131.001.0001.

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Testosterone Deficiency in Men. Oxford University Press, 2013.

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Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Iron deficiency. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0007.

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Diagnosis 54Management 55Iron deficiency is the most common nutritional deficiency in the world, affecting around 5 billion people, most of them from developing countries. The prevalence of iron deficiency anaemia in UK preschool children is ~8%, increasing considerably in inner city children; ~9% of under 5s in the USA are thought to be iron deficient. Depletion of iron stores is followed by the development of anaemia, initially with a normal mean cell volume (MCV). Continuing deficiency leads to impairment of erythropoiesis, with hypochromia and microcytosis apparent on blood film. Iron is essential in haemoglobin for oxygen transport, and is also found in myoglobin, and some enzymes (peroxidase, catalase, and cytochromes). Iron from red blood cell breakdown is recycled and excess iron stored as ferritin and haemosiderin....
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Puntis, John. Iron deficiency. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759928.003.0009.

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Iron deficiency is the most common nutritional deficiency in the world, affecting around 5 billion people mostly in developing countries. Risk factors in infants include low birthweight, high cow milk consumption, low intake of iron containing complementary foods, low socioeconomic status, and immigrant status. Developmental delay and poor educational achievement are among the long-term complications. Preventative strategies include promotion of breastfeeding, use of iron-fortified formula if breast milk not available, encouraging intake of iron-rich foods, vitamin C-rich drinks with meals to promote iron absorption, and avoiding whole cow’s milk in the first year of life. Poor response to oral iron treatment is most likely due to poor compliance (iron ingestion may cause abdominal pain diarrhoea or constipation) but should also raise the possibility of underlying disease causing inflammation, malabsorption, or blood loss.
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Bunch, Chris. Deficiency anaemias. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0279.

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This chapter addresses the diagnosis, investigation, and management of anaemia due to a deficiency in iron, vitamin B12, or folate. Erythropoiesis requires an adequate supply of iron for haem formation, as well as vitamin B12 and folic acid (folate) to support high levels of DNA synthesis, and a lack of any of these will result in anaemia. Iron-deficient anaemias are typically microcytic, while a deficiency in vitamin B12 or folate results in megaloblastic haemopoiesis and a macrocytic anaemia. Iron deficiency results from poor dietary iron intake, poor absorption, increased demands, blood loss, or combinations of these. The usual cause of severe vitamin B12 deficiency in Western countries is an autoimmune atrophic gastritis, in which there is a loss of gastric parietal cell numbers and an absence of intrinsic factor production, which effectively prevents vitamin B12 absorption. This is the classical pernicious anaemia, and it is often seen in association with other autoimmune disorders. Folate deficiency may result from poor diet, malabsorption, or when demand for folate is increased, for example, during pregnancy, or with increased haemopoiesis in haemolytic anaemias or myeloproliferative disorders.
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Book chapters on the topic "Met deficiency"

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Trost, Landon. "Hyperprolactinemia in Men with Testosterone Deficiency." In Controversies in Testosterone Deficiency, 35–43. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-77111-9_4.

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Cambareri, Gina, Jane Ashby, David Goldmeier, and Hossein Sadeghi-Nejad. "HIV and Testosterone in Men." In Androgen Deficiency and Testosterone Replacement, 103–28. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-179-0_9.

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Rochira, Vincenzo, and Cesare Carani. "Estrogen Deficiency in Men." In Endocrinology, 1–32. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-29456-8_27-1.

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Rochira, Vincenzo, and Cesare Carani. "Estrogen Deficiency in Men." In Endocrinology, 797–828. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-44441-3_27.

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Jarrett, Simon, and Jan Walmsley. "Intellectual disability policy and practice in twentieth-century United Kingdom." In Intellectual Disability in the Twentieth Century, 177–94. Policy Press, 2019. http://dx.doi.org/10.1332/policypress/9781447344575.003.0015.

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In the first half of the twentieth century UK policy, dominated by the passing of the Mental Deficiency Act in 1913, was not driven solely by eugenic discourse but by a commonly held assumption across the ideological spectrum that the ‘mentally deficient’ population needed ‘fixing’ in some way. Lurking beneath this desire for completeness and the tidying up of the social sphere were deep anxieties about urban modernity and human capacity to meet its challenges. In the second half of the century we see the voice of the person with intellectual disability finally emerge as self-advocacy, as the institutions met their end and care in the community became policy. A so-called ‘golden period’ in the 1990s, characterized by thoughtful policy and a commitment to inclusion, raised hopes of genuine citizenship and improved support. The chapter ends however with a cautionary warning from the 21st century, where gains seem to be receding and the institution rising from its grave.
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Krone, Nils. "Congenital adrenal hyperplasia." In Oxford Textbook of Endocrinology and Diabetes, 865–78. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199235292.003.0616.

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Congenital adrenal hyperplasia (CAH) represents a group of autosomal recessive disorders of steroidogenesis caused by defects in steroidogenic enzymes involved in glucocorticoid synthesis or in enzymes providing cofactors to steroidogenic enzymes (1, 2). Congenital lipoid adrenal hyperplasia (CLAH) caused by steroidogenic acute regulatory protein (StAR) deficiency is distinct in origin and presentation from the conventional variants of CAH, with the unique feature of lipid accumulation subsequently leading to destruction of adrenal function. This chapter will also mention aldosterone synthase deficiency, which is the only defect in adrenal steroidogenesis causing deficient mineralocorticoid biosynthesis without affecting glucocorticoid biosynthesis. The disorder cannot strictly be considered a CAH variant as it does not result in increased ACTH drive and thus not in adrenal hyperplasia. Novel forms of CAH have emerged during recent years. These include P450 oxidoreductase deficiency (ORD), P450 side-chain cleavage (CYP11A1) deficiency, the nonclassic form of CLAH (StAR deficiency), and apparent cortisone reductase deficiency. All forms of congenital adrenal hyperplasia resemble a disease continuum spanning from mild nonclassic presentations to classic onset with severe signs and symptoms.
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Jackson, Alan A. "Severe malnutrition." In Oxford Textbook of Medicine, edited by Katherine Younger, 1880–90. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0221.

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Severe malnutrition is the consequence of systemic deficiency of energy and nutrients over a prolonged period: in children development is stunted and the individual is at risk of fatal (often clinically ‘silent’) infection and other illnesses. It is a medical and societal emergency: mortality is high, despite attempts to provide appropriate care. When severe malnutrition affects several individuals in a society, it reflects a state in which basic needs and justice are not met. Severe malnutrition may also result from clinical disorders affecting a single person with gastrointestinal disease, poor appetite, or reduced food intake for other reasons. The World Health Organization has produced guidelines for facility-based care of patients suffering severe malnutrition, and this chapter explains the diagnosis, treatment, and management of the condition, along with its possible complications.
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Spranger, Jürgen W., Paula W. Brill, Christine Hall, Gen Nishimura, Andrea Superti-Furga, and Sheila Unger. "Spondylo-Epi-Metaphyseal and Spondylo-Metaphyseal Dysplasias." In Bone Dysplasias, 157–244. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190626655.003.0007.

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This chapter further discusses bone dysplasias and explores achondrogenesis type 1A, odontochondrodysplasia, Schneckenbecken dysplasia, opsismodysplasia, spondylometaphyseal dysplasia (Sedaghatian type), spondyloenchondrodysplasia, SEMD (PAPSS2 type) and brachyolmia (autosomal recessive type), Dyggve-Melchior-Clausen dysplasia, spondylometaepiphyseal dysplasia (short limb-abnormal calcification type), spondylometaphyseal dysplasia with cone-rod dystrophy, dyssegmental dysplasia, Schwartz-Jampel syndrome, spondyloepiphyseal dysplasia tarda (X-linked), aggrecan-associated skeletal dysplasias, Wolcott-Rallison syndrome, Schimke immunoosseous dysplasia, progressive pseudorheumatoid chondrodysplasia, spondylometaphyseal dysplasia (corner fracture type), sponastrime dysplasia, CODAS syndrome, N-acetyl-neuraminic acid synthase (NANS) deficiency, and spondylo-epi-metaphyseal dysplasia with immune deficiency and developmental disability (EXTL3-deficient type). Each discussion includes major radiographic features, major clinical findings, genetics, major differential diagnoses, and a bibliography.
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Pollak, Eleanor S., and Katherine A. High. "Genetic disorders of coagulation." In Oxford Textbook of Medicine, edited by Chris Hatton and Deborah Hay, 5532–46. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0546.

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Haemophilia is a familial X-linked disorder due to deficiency of either factor VIII (haemophilia A) or factor IX (haemophilia B), components of the intrinsic enzymatic complex that activates factor X. Clinical features and diagnosis—the main manifestations are bleeding into joints and soft tissues, with haemophilic arthropathy and joint deformity being inevitable complications in untreated patients. Other features include pseudotumours, bleeding into the urinary system, and bleeding following clinical procedures. Laboratory diagnosis is based on a modification of the classic activated partial thromboplastin time (APTT) assay, with inhibitor screening used to exclude other causes of prolonged APTT. Treatment—involves the administration of the deficient factor VIII or factor IX, most commonly ‘on demand’ in response to bleeding, with prophylactic treatment given before surgery. Von Willebrand’s disease is a common autosomal dominant disorder of platelet function caused by a functional deficiency of von Willebrand factor (VWF). VWF, normally synthesized by megakaryocytes, prevents degradation of factor VIII; VWF, also made by endothelial cells, enhances platelet activation and recruitment at sites of tissue damage. Treatment—mild von Willebrand’s disease is treated with desmopressin 1-deamino-8-d-arginine vasopressin (DDAVP), which releases factor VIII and VWF from endothelial cells. Other treatments include ε‎-aminocaproic acid, oestrogens, and factor VIII concentrates. Other hereditary disorders of coagulation, including (1) hereditary deficiency of the plasma metalloproteinase ADAMTS13; (2) combined deficiency of coagulation factors V and VIII; (3) factor XI deficiency; (4) inherited deficiencies of factors II, V, VII, and X; and (5) deficiency of the contact activating factors, factor XIII, and fibrinogen, and hypercoagulable diseases due to deficiencies of anticoagulants or propensity to thrombosis are discussed in this chapter.
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Jadon, Deepak R., Tehseen Ahmed, and Ashok K. Bhalla. "Disorders of bone mineralization—osteomalacia." In Oxford Textbook of Rheumatology, 1252–58. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0146_update_002.

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Disorders of bone mineralization cause rickets in children and osteomalacia in adults. Both remain common in developing countries. Incidence in Western countries had declined since the fortification of foodstuffs, but appears to be increasing again. Calcium and inorganic phosphate are the key precursors for bone mineralization and growth. The commonest aetiology of osteomalacia is vitamin D deficiency, primarily due to low dietary intake and inadequate sun exposure. In the last decade gene mutations have been identified that are responsible for inherited rickets and osteomalacia, particularly those that result in phosphate deficiency, hypophosphatasia, and vitamin D receptor or metabolizing enzyme mutations. Additionally, the pathogenesis of tumour-induced osteomalacia is becoming better understood. Osteomalacia may present as bone pain and tenderness, muscle pain and weakness, and skeletal deformity or fracture. Key investigations include biochemical assessment and plain radiographs. Radioisotope bone scans and bone biopsy may be considered in selected cases. Differential diagnoses include osteoporosis, seronegative arthritides, and localized soft tissue disorders. Treatment, guided by the underlying aetiology, aims to reduce symptoms, fracture risk, bone deformity and sequelae. Vitamin D deficient patients require vitamin D and calcium replacement.
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Conference papers on the topic "Met deficiency"

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Kaufman, Steven G., and Vern Martin. "Rated Fan Performance: Determining If It Is Accurate and Acceptable — A Case for Laboratory Verification Testing." In ASME 2005 Power Conference. ASMEDC, 2005. http://dx.doi.org/10.1115/pwr2005-50202.

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With few exceptions, fan vendors do not normally provide a written guarantee for the aerodynamic performance of a fan or even identify tolerances for the rated performance. When requested, the vendor often refers to acceptance criteria established in accordance with fan industry recognized standards and publications issued by the Air Movement and Control Association, Inc. (AMCA). AMCA acceptance criteria are very subjective, allow for very wide tolerances and can result in a very low probability that the purchaser’s performance requirements are met. There are three options available to the purchaser. • Assume the risk that the fan may not meet the performance requirement. • Purchase an oversized fan such that the performance can be met even if the fan performs at the low end of its performance tolerance. • Insure that the purchased equipment will perform as specified and mitigate the risks of deficiency by requiring the vendor comply with a performance guarantee. Test procedures suitable for conducting fan performance guarantee tests are available in many published AMCA, ANSI, ISO and ASME performance test standards. These performance test standards typically provide a procedure to estimate the accuracy of the measurement in the form of an uncertainty analysis. However, the accuracy of the measurement does not necessarily address whether the measurement meets the requirement within an acceptable tolerance. Except for AMCA, these test standards generally do not stipulate acceptable performance tolerances. A tolerance stipulates how much the measured value is allowed to fall short or exceed the required specified value and still be deemed to be acceptable. The system designer should specify suitable acceptance tolerances with consideration of the design margins that have been applied to the flow, pressure and power requirements of the system. This technical paper outlines a guideline of how to assess the accuracy and validity of a vendor’s quoted performance and the implications on system design margins. A case is made for laboratory verification testing in situations where a performance guarantee is required. • Allows proactive verification before final design, manufacture or shipment takes place. • Verification and corrective actions do not interfere with plant startup and production. • Allows for proactive modifications to be made at a fixed cost to the buyer.
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Zhong, Xian, Cheng Gu, Wenxin Huang, Lin Li, Shuqin Chen, and Chia-Wen Lin. "Complementing Representation Deficiency in Few-shot Image Classification: A Meta-Learning Approach." In 2020 25th International Conference on Pattern Recognition (ICPR). IEEE, 2021. http://dx.doi.org/10.1109/icpr48806.2021.9412416.

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Murphy, Adam B., Yaw Nyame, Brian Kelley, Chiledum A. Ahaghotu, and Rick A. Kittles. "Abstract A93: Correlates of total testosterone and testosterone deficiency in African American men." In Abstracts: AACR International Conference on the Science of Cancer Health Disparities‐‐ Sep 18-Sep 21, 2011; Washington, DC. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1055-9965.disp-11-a93.

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Nonn, Larisa. "Abstract PL01-03: Vitamin D deficiency and prostate cancer in men with African ancestry." In Abstracts: Sixth AACR Conference: The Science of Cancer Health Disparities; December 6–9, 2013; Atlanta, GA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7755.disp13-pl01-03.

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De Stefano, V., G. Leone, R. Ferrelli, and B. Bizzi. "A NEW VARIANT OF FACTOR X DEFICIENCY (FACTOR X ROMA)." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643296.

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A 13 years-old-girl was admitted in our Hospital for a large muscle hematoma of left psoas. At age 3 she had a severe tonsillar bleeding following angina. Afterwards she suffered from easy bruisability and recurrent epistaxis. Prothrombin time (PT) was slightly prolonged (14.7 sec, control 12 sec, INR 1.5), while a more marked prolongation in aPTT was noticed (54.7 sec, control 30.3 sec). All clotting factors resulted within the normal range but Factor X. Factor X antigen was 0.95 U/ml (Laurell), whereas Factor X activity was 0.52 U/ml by extrinsic system assay (rabbit brain and lung thromboplastin), 0.06 U/ml by intrinsic system assay (rabbit brain cephalin) and 1.15 U/ml by activation by RW-cephalin. Chromogenic assays (S-2222 and CBS 3139) perfor med after activation with RW gave 1.02 U/ml and 1.00 U/ml, respectively. The patient plasma had no inhibitory activity against Factor X. The parents of the proposita (first cousins) had Factor X antigen levels and RW functional activity around 1.00 U/ ml, whereas Factor X activity tested by extrinsic and intrinsic system assay was about 0.60 U/ml and 0.50 U/ml, respectively. This Factor X variant seems different from the other ones previously described, showing normal antigen levels and RW activation, a severe defect in the intrinsic activation and only a partial defect in the extrinsic activation. In particular the two defects more close to it (Factor X Friuli and Factor X Melbourne), both found in patients with Italian ancestry, were dif ferent because of a very prolonged PT (Factor X Friuli) or a normal PT (Factor X Melbourne).
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Sun, Fengmei, Juan Zhang, and Yuepu Pu. "Glucose-6-phosphate dehydrogenase deficiency and the risk of malaria: A meta-analysis and trial sequential analysis." In 2ND INTERNATIONAL CONFERENCE ON MATERIALS SCIENCE, RESOURCE AND ENVIRONMENTAL ENGINEERING (MSREE 2017). Author(s), 2017. http://dx.doi.org/10.1063/1.5005230.

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Wannamethee, SG, Paul Welsh, O. Papacosta, LT Lennon, and PH Whincup. "P61 Vitamin D deficiency, impaired lung function and total mortality in older men: the british regional heart study." In Society for Social Medicine 62nd Annual Scientific Meeting, Hosted by the MRC/CSO Social and Public Health Sciences Unit, University of Glasgow, 5–7 September 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/jech-2018-ssmabstracts.184.

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Hilario, Angelica Mae A. "Maternal Vitamin D Supplementation Among Lactating Mothers in the Prevention of Vitamin D Deficiency Among Breastfed Term Infants: A Meta Analysis." In Selection of Abstracts From NCE 2016. American Academy of Pediatrics, 2018. http://dx.doi.org/10.1542/peds.141.1_meetingabstract.293.

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Ningrum, Fiki Cahya, Bhisma Murti, and Vitri Widyaningsih. "Relative Efficacy of Intravenous Iron Versus Oral Iron on Increasing Hemoglobin Level among Pregnant Women with Iron Deficiency Anemia: A Meta-Analysis." In The 6th International Conference on Public Health 2019. Masters Program in Public Health, Graduate School, Universitas Sebelas Maret, 2019. http://dx.doi.org/10.26911/the6thicph.03.18.

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Charoenngam, Nipith, Ben Ponvilawan, and Patompong Ungprasert. "SAT0452 VITAMIN D INSUFFICIENCY AND DEFICIENCY ARE ASSOCIATED WITH A HIGHER LEVEL OF SERUM URIC ACID: A SYSTEMATIC REVIEW AND META-ANALYSIS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.696.

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Reports on the topic "Met deficiency"

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Shi, Shanzhen, Jiaxing Feng, Yu Li, and Huaxiu Shi. Risk factors for vitamin D deficiency in inflammatory bowel disease: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2020. http://dx.doi.org/10.37766/inplasy2020.6.0028.

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Ferdinand, Nyankoue, Arroye Fabrice, Mazou Temgoua, and Ndip Valerie. Association between melatonin deficiency and progressive adolescent idiopathic scoliosis: protocol for a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, July 2020. http://dx.doi.org/10.37766/inplasy2020.7.0083.

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TAO, Zhuang, Xiaojie HUANG, Ying LIU, Ru WANG, Jipeng DONG, Biyan LIANG, Wen ZOU, et al. The Clinical Effect of Integrated Traditional Chinese Medicine and Western Medicine in Immuno-reconstitution Deficiency of HIV/AIDS: A Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2020. http://dx.doi.org/10.37766/inplasy2020.6.0057.

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Bai, Yiling, L. L. Han, H. Z. Lu, H. Z. Wang, and C. Jiang. Efficacy and safety of traditional Chinese medicine in the treatment of immune infertility based on the theory of "kidney deficiency and blood stasis": systematic review and meta. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, January 2021. http://dx.doi.org/10.37766/inplasy2021.1.0098.

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