Books: 'MET Matrix'

1

The homosexual matrix. 2nd ed. New York: New American Library, 1987.

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2

When mystic masters meet: Towards a new matrix for Christian-Muslim dialogue. New York: Blue Dome, 2011.

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3

Xu 99 wei chang yong zhong yao ru men. Guangzhou Shi: Guangdong ke ji chu ban she, 2007.

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4

Flückiger, Friedrich A. Pharmacographia: A history of the principal drugs of vegetable origin met with in Great Britain and British India. Dehra Dun, India: International Book Distributors, 1986.

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5

1970-, Huang Shixun, ed. Shi yong yao cao ru men tu jian. Taizhong Shi: Zhan du wen hua shi ye you xian gong si, 2007.

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6

Ljungqvist, Kerstin. Nyttans växter: Uppslagsbok med över tusen växter : historik om svensk medicinalväxtodling. [Dals Rostock]: Calluna, 2006.

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7

Hot plants: Nature's proven sex boosters for men and women. New York: St. Martin's Griffin, 2004.

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8

99 wei chang yong Zhong yao ru men: Yao xing xiang jie. Taibei Xian Banqiao Shi: Wen shen chu ban she, 2005.

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9

Yü chi yü cong ming: Jian yi mei wei yao shan. Taibei Xian San chong shi: Yüan qi zhai chu ban she, 1991.

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10

ʼBum-kho, ed. Gces bsdus ʼchi med nor buʼi phreṅ ba bźusgs so. Lan-chou: Kan-suʼu mi rigs dpe skrun khaṅ, 2001.

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11

Forsyth, Frederick. Ge ju mei ying zhi Manhadun mei ying. Taibei Xian Banqiao Shi: Ya shu tang wen hua shi ye, 2001.

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12

Bai zhong Zhong yao yang yan mei rong shi pu. Guangzhou Shi: Guangdong ke ji chu ban she, 2004.

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13

Deuber-Mankowsky, Astrid, and Anna Tuschling, eds. Conatus und Lebensnot. Wien: Turia + Kant, 2017. http://dx.doi.org/10.37050/ci-12.

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Als bewegende Bedingungen des Lebens stehen Conatus und Lebensnot im Zentrum aktueller medienanthropologischer Diskussionen über das Verhältnis von Leben und Medien. Spinoza definierte Conatus als Prinzip, nach dem das Wesen jedes Dings darin besteht, »in seinem Sein zu verharren«. Er begründete mit diesem Prinzip eine Denktradition, in der Leben und Materie, Rationalität und Vermögen koinzidieren. Der Begriff der Lebensnot dagegen entstammt Diskursen der Physiologie, der Medizin, der Urteilsphilosophie und schließlich der Psychoanalyse, die auf verschiedene Weise körperliche Bedingungen und Notwendigkeiten mit der Angewiesenheit auf das Andere und die Anderen verschränken. Conatus und Lebensnot erlauben in besonderer Weise zu situieren, warum und wie sich das Fragen nach dem Menschlichsein in Diskursen über Medientechnik immer wieder aktualisiert und dabei zugleich verändert.

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14

Laṅʻʺ, Kyoʻ. Lū tuiṅʻʺ kyanʻʺ mā reʺ ʼa tvakʻ Dhatʻ cā cāʺ naññʻ nhaṅʻʹ Vamʻʺ mīʺ Paññā Dhatʻ kyamʻʺ. Mrokʻ Ukkalāpa, Ranʻ kunʻ: Saraphū Cā pe, 2006.

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15

Laṅʻʺ, Kyoʻ. Lū tuiṅʻʺ kyanʻʺ mā reʺ ʼa tvakʻ Dhatʻ cā cāʺ naññʻ nhaṅʻʹ Vamʻʺ mīʺ Paññā Dhatʻ kyamʻʺ. Mrokʻ Ukkalāpa, Ranʻ kunʻ: Saraphū Cā pe, 2006.

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16

Chʻeng-chün, Liu, ed. Fruit as medicine: By Dai Yin-fang & Liu Cheng-jun ; translated from the Chinese by Ron Edwards & Gong Zhi-mei. 2nd ed. Kuranda, Australia: Rams Skull Press, 1987.

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17

Namja ŭi saengni. Sŏul Tʻŭkpyŏlsi: Tonghwa Munhwasa, 1989.

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18

Deuber-Mankowsky, Astrid, and Reinhold Görling, eds. Denkweisen des Spiels. Wien: Turia + Kant, 2017. http://dx.doi.org/10.37050/ci-10.

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Gibt es eine spezifische Medialität des Spiels, die es sinnvoll macht, das Verhältnis jeden Ereignisses zu seiner Umgebung als Spiel zu beschreiben? In welcher Weise können Technik, Spiel und Ästhetik neu gedacht werden? Immer dann, wenn Technik mit mechanischen Bewegungen verbunden wird, sehen wir sie im Gegensatz zum Spiel. Wenn Technik jedoch mit Ästhetik assoziiert wird, taucht das Spiel als Teil der Technik auf. Spiel bringt Dinge in Relation und verändert sie dadurch. Wenn Materie als rhythmische Bewegung gedacht wird und Spiel auf Wiederholung basiert, rückt der Spielbegriff ins Zentrum einer medienwissenschaftlichen Auslegung der relationalen Beschaffenheit von Zeit und Raum. Vor dem Hintergrund der Aktualität, welche die Spieletheorien von Huizinga und Caillois in den Game Studies erlangten, unternimmt der vorliegende Band eine medienphilosophische Befragung des Spielbegriffs. Er behandelt dabei das Spiel unter Bezugnahme auf Fragen der Neuen Materialismen und wirft zugleich einen neuen Blick auf das Spiel in der Psychoanalyse.

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19

Indonesia. Kantor Menteri Negara Perumahan dan Permukiman. Pokok-pokok materi untuk dilaporkan pada sidang kabinet terbatas bidang ekonomi, keuangan, dan industri, Rabu, 5 Mei 1999. Jakarta: Kantor Menteri Negara Perumahan dan Permukiman, 1999.

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20

zhu, Han, ed. "Ben cao gang mu" qi xiao mei yan jing. Nanjing Shi: Jiangsu ke xue ji shu chu ban she, 2011.

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21

Yinfang, Dai. Fruit as medicine: By Dai Yin-fang & Liu Cheng-jun ; translated from the Chinese by Ron Edwards & Gong Zhi-mei. Kuranda, Australia: Rams Skull Press, 1986.

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22

Dictionnaire des plantes me dicinales. Outremont, Que bec: Quebecor, 1997.

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23

Kurizki, Gershon, and Goren Gordon. The Quantum Matrix. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780198787464.001.0001.

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“Meet Henry Bar, a physicist and … quantum superhero.” The title The Quantum Matrix refers to a central concept in quantum physics, but also (allegorically) to our enigmatic world. In this book, Henry Bar, physicist and the first quantum superhero, guides the reader through the amazing quantum world. Henry’s hair-raising adventures in his perilous struggle for quantum coherence are graphically depicted by comics and thoroughly explained to the lay reader. Behind each adventure lies a key concept in quantum physics. These concepts range from the basic quantum coherence and entanglement through tunneling and the recently discovered quantum decoherence control, to the principles of the emerging technologies of quantum communication and computing. The explanations of the concepts are popular, but nonetheless rigorous and detailed, and are followed by their broader context, their historic perspective, up-to-date status and forthcoming developments. Finally, thought-provoking philosophical and cultural implications of these concepts are discussed. The mathematical appendices of all chapters cover, in a straightforward manner, the core aspects of quantum physics at the level of a university introductory course. The Quantum Matrix presents an entertaining, popular yet comprehensive picture of quantum physics. It can be read as a light-hearted illustrated tale, a philosophical treatise or a textbook. Either way, the book lets the reader delve deeply into the wondrous quantum world from diverse perspectives and obtain glimpses into the quantum technologies that are about to reshape our lives. May the reader’s voyage through the quantum world charted by this book be both pleasant and rewarding.

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24

McQuistion, Hunter L. Recovery Orientation as the Clinical Matrix. Edited by Hunter L. McQuistion. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190610999.003.0001.

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At the core of working with people who experience mental illnesses is a collaboration in pursuit of helping them actualize their goals in work and love. Their recovery is therefore a unique process of personal development, with psychiatrists and other human services professionals practicing with recovery orientation. Specific skills, techniques, and services are identified as recovery oriented. In this chapter, through relating one person’s story, the reader can understand how recovery orientation becomes operational in a clearly systematic manner, using such techniques as motivational interviewing, shared decision-making, rehabilitative focus, person-centered treatment planning, and the use of peer supports. In this way, it is illustrated how recovery orientation is not only richly humanitarian but also evidence-based.

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25

van der Kraan, Peter M., and Esmeralda N. Blaney Davidson. Cartilage (including meniscus). Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0004.

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This chapter provides an overview of tissues unique to synovial joints, articular cartilage, and meniscus. The development and cellular and (bio)chemical composition are described, as well as the role of mechanical stimuli. In addition, the role of growth factors in cartilage and meniscus homeostasis, cellular differentiation, and chondrocyte hypertrophy are discussed. Furthermore, the involvement of aggrecanases and matrix metalloproteinases in cartilage and meniscus matrix degradation and osteoarthritis are described. Finally, the current status of repair of articular cartilage and meniscus is provided. This chapter reflects the changes in cellular differentiation, growth factor signalling, and altered matrix composition that contribute to osteoarthritis.

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26

Kovanen, Petri T., and Magnus Bäck. Valvular heart disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0015.

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The heart valves, which maintain a unidirectional cardiac blood flow, are covered by endothelial cells and structurally composed by valvular interstitial cells and extracellular matrix. Valvular heart disease can be either stenotic, causing obstruction of the valvular flow, or regurgitant, referring to a back-flow through the valve. The pathophysiological changes in valvular heart disease include, for example, lipid and inflammatory cell infiltration, calcification, neoangiogenesis, and extracellular matrix remodelling. The present chapter addresses the biology of the aortic and mitral valves, and the pathophysiology of aortic stenosis and mitral valve prolapse.

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27

J. Singh, Parminder, and Rohit Kotnis. The musculoskeletal system: structure and function. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.0003.

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♦ Structure of bone is comprised of cells, matrix, and water♦ Bone consists broadly of three surfaces (periosteal, endosteal, and Haversian) and two membranes (periosteum and endosteum)♦ The blood supply of bone is derived from four main routes (nutrient, metaphyseal, epiphyseal, and periosteal arteries)♦ There are three main types of cells in bone (osteoblast, osteocyte, and osteoclast)♦ The matrix is a composite material consisting of an organic and an inorganic component♦ Two types of bone formation are intramembranous and endochondral ossification♦ The skeleton is also involved in the vital homeostasis of calcium and phosphate.

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28

Badano, Luigi P., Roberto M. Lang, and Alexandra Goncalves. Three-dimensional echocardiography. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0007.

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The advent of fully-sampled matrix array transthoracic transducers has enabled advanced digital processing and improved image formation algorithms and brought three-dimensional echocardiography (3DE) technology into clinical practice. Currently, 3DE is recognized as an important echocardiographic technique, demonstrated to be superior to two-dimensional echocardiography in various clinical scenarios. This chapter focuses on the technology of 3DE matrix transducers, physics of 3D imaging, data set acquisition (multiplane, real-time, full-volume, zoom, and colour), and display (volume rendering, surface rendering and multislice) modalities. The chapter also addresses the issues of training in 3DE, and main clinical indications and reporting of transthoracic and transoesophageal 3DE.

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29

Sánchez-Quintana, Damián, and José Angel Cabrera. Normal atrial and ventricular myocardial structures. Edited by Yen Ho. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0014.

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The heart functions by means of a three-dimensional arrangement of myofibres supported by an extracellular matrix which plays an important role in maintaining the size and shape of the heart. In both atria, the structure of the walls and the atrial septum confers a three-dimensional arrangement of muscle bundles and myoarchitecture that allows preferential electrical intra- and interatrial conduction which is important for a better understanding of atrial activation and arrhythmias. The myoarchitecture within the ventricular walls has a three-dimensional arrangement of myofibres, within a supporting matrix of fibrous tissue, which changes orientation from being oblique in the subepicardium to circumferential in the middle and to longitudinal in the subendocardium, allowing the chambers to change in shape and size through the cardiac cycle. Within each ventricle, the circumferential portion is the thickest transmurally, with the longitudinal portion the thinnest. The three-dimensional arrangement of the ventricular mesh serves to realign the myocytes during ventricular contraction, accounting for the extent of systolic mural thickening. Abnormal myoarchitecture in combination with alterations in the connective tissue matrix provide the structural basis for abnormalities in myocardial function.

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30

Lennon, Rachel, and Neil Turner. The molecular basis of glomerular basement membrane disorders. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0320_update_001.

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The glomerular basement membrane (GBM) is a condensed network of extracellular matrix molecules which provides a scaffold and niche to support the function of the overlying glomerular cells. Within the glomerulus, the GBM separates the fenestrated endothelial cells, which line capillary walls from the epithelial cells or podocytes, which cover the outer aspect of the capillaries. In common with basement membranes throughout the body, the GBM contains core components including collagen IV, laminins, nidogens, and heparan sulphate proteoglycans. However, specific isoforms of these proteins are required to maintain the integrity of the glomerular filtration barrier.Across the spectrum of glomerular disease there is alteration in glomerular extracellular matrix (ECM) and a number of histological patterns are recognized. The GBM can be thickened, expanded, split, and irregular; the mesangial matrix may be expanded and glomerulosclerosis represents a widespread accumulation of ECM proteins associated with loss of glomerular function. Whilst histological patterns may follow a sequence or provide diagnostic clues, there remains limited understanding about the mechanisms of ECM regulation and how this tight control is lost in glomerular disease. Monogenic disorders of the GBM including Alport and Pierson syndromes have highlighted the importance of both collagen IV and laminin isoforms and these observations provide important insights into mechanisms of glomerular disease.

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31

MacGrogan, Donal, José Maria Pérez-Pomares, Bill Chaudhry, José Luis de la Pompa, and Deborah J. Henderson. From cushions to leaflets: morphogenesis of cardiac atrioventricular valves. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso, and Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0017.

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At the looping stage of heart development, tissue patterning of myocardium and endocardium at the atrioventricular (AV) junction defines a morphogenic field competent to form valves that initially appear as protrusions of proteoglycan-rich extracellular matrix (ECM) called endocardial cushions (ECs) which are cellularized by an endocardial-mesenchymal transition (EMT). Cellular proliferation results in fusion of the major AV mesenchymal cushions and AV septation, whereas smaller cushions receive a supply from epicardially derived cells. These various sources of mesenchyme precursors give rise to most of the valve structures, leaflets, annuli, and supporting tension apparatus. During valve leaflet maturation, the ECM matrix accumulates collagen and elastin and assembles into a thin flexible fibrous structure, which is remarkably tough. Valve development is regulated by the cross-talk between developmental signalling pathways. Pathogenic mutations in a subset of developmentally important genes have been linked to valve disease, suggesting that developmental defects may underlie valve disease in adulthood.

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32

Wallace, Don, and Andrew Carr. Calcifying tendinitis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.004006.

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♦ Calcifying tendonitis remains an enigmatic condition♦ The pathology shows hydroxyapatite crystals lying in extra-cellular matrix vesicles♦ There are two presentations, acute and chronic♦ Acute episodes can be treated by analgesia and barbottage♦ Chronic lesions can be treated conservatively, or by excision of the calcific deposit, arthroscopically or by barbottage.

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33

Bebiano, Natália. Applied and Computational Matrix Analysis: MAT-TRIAD, Coimbra, Portugal, September 2015 Selected, Revised Contributions. Springer, 2018.

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34

Bebiano, Natália. Applied and Computational Matrix Analysis: MAT-TRIAD, Coimbra, Portugal, September 2015 Selected, Revised Contributions. Springer, 2017.

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35

Zhong yi yao ru men. Jiangxi ke xue ji shu chu ban she, 1989.

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36

Wordsworth, B. P. Skeletal dysplasias. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0150.

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Bone is metabolically active throughout life and metabolic disturbances may have wide-ranging consequences that are not restricted to altering its mechanics. The study of some genetic bone diseases has already provided remarkable insights into the normal regulation of bone metabolism. Skeletal dysplasias are developmental disorders of the chondro-osseous tissues commonly resulting in short stature, which is often disproportionate. The underlying mutations are often in the structural genes encoding components of the matrix but may also involve growth factors or cell signalling. In contrast, the dysostoses tend to affect single bones or groups of bones, reflecting the transient nature of the many different signalling factors to which they are responsive during development. Abnormalities of bone density (high or low) may be due to primary deficiency of bone matrix synthesis (e.g. osteogenesis imperfecta and hypophosphatasia) but may also reflect an imbalance between bone formation and resorption. This may be caused by abnormalities of bone formation (e.g. hyperostosis/sclerosteosis and osteoporosis pseudoglioma syndrome) or bone resorption (e.g. classic osteopetrosis and fibrous dysplasia).

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37

Ronen, Boaz, Joseph S. Pliskin, and Shimeon Pass. The Pareto Rule, the Pareto Based Methodology, the Focusing Table, and the Focusing Matrix (DRAFT). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190843458.003.0003.

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This chapter introduces one of the most powerful tools in management—the Pareto methodology and its derivatives. This upgrade of the Pareto (80/20) rule enables managers to use different policies for different entities. For the “vital few” cases, managers should use policies that are different from those used for the “trivial many” cases. The Pareto analysis classifies sources of the phenomenon into classes A, B, and C. Then, a differential policy is applied to members of each class. Finally, resources are allocated based on the first two steps. This chapter also presents the focusing table and the focusing matrix, which are among the simplest, nicest, and easiest to implement tools that allow focusing and enable good communication and empowerment within the organization.

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38

Lai, Kar Neng, and Sydney C. W. Tang. Immunoglobulin A nephropathy. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0065.

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Immunoglobulin A (IgA) nephropathy is the most common primary glomerulonephritis. It runs a slow and sometimes relentless clinical course with consequent end-stage renal failure in 35–40% of patients 25–30 years after first clinical presentation. The pathology is characterized by deposition of macromolecular (polymeric) IgA1 in the glomerular mesangium, proliferation of mesangial cells, increased synthesis of extracellular matrix, and infiltration of macrophage, monocytes, and T cells.

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39

Beza, Dingani. Diamonds in the Rough: Men of Extraordinary Value, Hidden Deep Beneath the Matrix of Sin. Essence Publishing (Canada), 2004.

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40

Zhong yao di ying yong (Zhong yi ru men cong shu). Ren min wei sheng chu ban she, 1989.

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41

Devlin, Hugh, and Rebecca Craven. Bone. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759782.003.0004.

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Bone in relation to dentistry is the topic of this chapter. This chapter describes the mineral, cells, vascular and matrix components of bone. Throughout the chapter, the clinical relevance of these features and how they interact in health and disease are emphasized. The later parts of the chapter describe bone healing, bone grafts, healing of the extraction socket, orthodontic tooth movement, periodontal bone loss in chronic periodontitis, and the effect of bisphosphonates. A final section summarizes age changes in bone and bone cells.

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42

Black, Helen K., John T. Groce, and Charles E. Harmon. The Strategies of Coping in Suffering and Caregiving. Oxford University Press, 2018. http://dx.doi.org/10.1093/acprof:oso/9780190602321.003.0005.

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If an experience is distressful enough to be called suffering, does it truly end, or does residue of the experience continue to assault the person’s wholeness? In this chapter, we offer three themes that that emerged as means to resolve suffering or to protect themselves from experiencing suffering despite distress. They are: (1) having goals, (2) sharing the power of the family story and (3) maintaining friendships with other African-American men. The three themes were often interrelated in men’s accounts. Men interviewed for this research revealed the importance of looking toward the future with hope and plans in hand, keeping previous generations alive through reminiscence and, seeking friendship with other men, particularly during caregiving (Black, 2015; Mattis et al., 2001) and showed older African-American male caregivers to be involved in reciprocal friendships with other men, which one man described as “what keeps me going.”

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43

Abhishek, Abhishek, and Michael Doherty. Pathophysiology of calcium pyrophosphate deposition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0049.

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Calcium pyrophosphate (CPP) dihydrate crystals form extracellularly. Their formation requires sufficient extracellular inorganic pyrophosphate (ePPi), calcium, and pro-nucleating factors. As inorganic pyrophosphate (PPi) cannot cross cell membranes passively due to its large size, ePPi results either from hydrolysis of extracellular ATP by the enzyme ectonucleotide pyrophosphatase/phosphodiesterase 1 (also known as plasma cell membrane glycoprotein 1) or from the transcellular transport of PPi by ANKH. ePPi is hydrolyzed to phosphate (Pi) by tissue non-specific alkaline phosphatase. The level of extracellular PPi and Pi is tightly regulated by several interlinked feedback mechanisms and growth factors. The relative concentration of Pi and PPi determines whether CPP or hydroxyapatite crystal is formed, with low Pi/PPi ratio resulting in CPP crystal formation, while a high Pi/PPi ratio promotes basic calcium phosphate crystal formation. CPP crystals are deposited in the cartilage matrix (preferentially in the middle layer) or in areas of chondroid metaplasia. Hypertrophic chondrocytes and specific cartilage matrix changes (e.g. high levels of dermatan sulfate and S-100 protein) are related to CPP crystal deposition and growth. CPP crystals cause inflammation by engaging with the NALP3 inflammasome, and with other components of the innate immune system, and is marked with a prolonged neutrophilic inflitrate. The pathogenesis of resolution of CPP crystal-induced inflammation is not well understood.

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44

Qiyou, Liu, ed. Zhong yi yao ru men. Nanchang Shi: Jiangxi ke xue ji shu chu ban she, 1989.

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45

Goeral, Katharina, Ali Fatemi, and Michael V. Johnston. Neonatal Brain Injuries. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0072.

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Neonatal brain injuries (NBIs) are important causes of motor and neurocognitive disabilities in children worldwide. This chapter discusses the most common types of NBIs in developed countries, namely neonatal encephalopathy associated with birth asphyxia in term infants, perinatal arterial ischemic stroke, perinatal white matter injury, and germinal matrix hemorrhage and neonatal hydrocephalus. Epidemiologic studies suggest that prenatal stressors, including intrauterine inflammation, maternal drug/toxin exposure, and placental pathologies are key players in the etiology of NBIs, whereas genetic contributors are yet to be identified. Hypothermia has become a standard neuroprotective strategy in neonatal encephalopathy, and novel therapeutics are currently being evaluated.

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46

Hasan, David. The Natural History of Cerebral Aneurysms. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0109.

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Subarachnoid hemorrhage (SAH) secondary to rupture of cerebral aneurysms represents a relatively small fraction of strokes (5%) but morbidity and mortality associated with aneurysm rupture remain very high despite advances in the treatment of aneurysmal SAH. Cerebral vasospasm (CV) is the leading cause of delayed morbidity and mortality following aneurysmal subarachnoid hemorrhage, as well as delayed neurological dysfunction 1 to 2 weeks after rupture. Endothelial dysfunction is one of the primary contributing factors to CV following aneurysmal SAH, and this is associated with alterations in intracellular adhesion molecule-1 (ICAM-1), matrix metalloproteinases (MM), and the blood-brain barrier[p63].

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47

Feili, Huang, and Yan Shixiang, eds. Shi yong mei rong Zhong yao xue. Shenyang: Liaoning ke xue ji shu chu ban she, 2001.

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48

Organizer, Creative. Super Dot Grid Journal: 150 Pages Dotted Matrix Planner and Notebook Diary for Women, Men, Boys and Girls. Independently Published, 2020.

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49

Riley, Peter. The role of the microbiology laboratory in antimicrobial stewardship. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198758792.003.0010.

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Microbiology laboratories play an important role in antimicrobial stewardship at the level of individual patients and the population as a whole. When empiric therapy has been started, rapid results can lead to earlier targeted treatment. Accumulated results of susceptibility tests can be analysed and used to generate local or national guidelines on empiric treatment and prophylaxis. Several methods can be used to determine microbial identity and antimicrobial susceptibility, including traditional culture-based methods and newer molecular methods such as matrix-assisted laser desorption ionization time-of-flight (MALDI-TOF) mass spectrometry, polymerase chain reaction (PCR), and whole-genome sequencing. These methods and potential advantages are reviewed. Before results are reported, expert rules are applied and results edited. At this point the laboratory can influence prescribing practices.

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50

Poretti, Andrea, and Michael V. Johnston. Genetic Disorders and Stroke. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0110.

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A variety of monogenic and polygenic genetic disorders have been linked to stroke, making it important for the clinician to keep up with the new discoveries and the potential to provide new gene-based therapies. Hematologic disorders such as sickle cell disease and thrombophilia due to mutations in prothrombin, factor V Leiden, and homocysteine metabolism are fairly well known, but mutations in mitochondrial metabolism and matrix metalloproteinases are less recognized. In addition, results of genome-wide association studies (GWAS) in stroke populations are revealing mutations that could predispose to stroke in specific ethnic populations. These studies are also revealing some crossover in mutations between stroke and familial hemiplegic migraine as well as mutations in growth factors such as brain derived neurotrophic factor (BDNF) that appear to influence the recovery from stroke by altering cortical plasticity.

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Books on the topic 'MET Matrix'

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