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Books on the topic 'Metabolic activity'

Author: Grafiati

Published: 4 June 2021

Last updated: 26 July 2025

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1

Cudic, Predrag, ed. Peptide Modifications to Increase Metabolic Stability and Activity. Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-652-8.

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Predrag, Cudic. Peptide modifications to increase metabolic stability and activity. Humana Press, 2013.

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3

United States. National Aeronautics and Space Administration., ed. Studies of intercellular communication and intracellular metabolic responses by bone cells to simulated weightlessness: Final NASA report. National Aeronautics and Space Administration, 1997.

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4

Main, Fiona Gillian. The use of fluorescein diacetate and carboxy-fluorescein diacetate to analyse metabolic activity in oral bacteria using the flow cytometer. National Library of Canada, 1990.

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5

Daniel, Catherine W. J. Soil metabolic activity and properties of soil organic matter in the A1 horizon of reclaimed acid metalliferous mine tailings in the Sudbury area. Laurentian University, Department of Biology, 1997.

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6

Middaugh, Jeffrey A. A study of the metabolic adaptation provoked by decreased aconitase activity and increased ROS production as a consequence of aluminum stress in Pseudomonas fluorescens. Laurentian University, School of Graduate Studies, 2004.

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7

1921-, Montoye Henry J., ed. Measuring physical activity and energy expenditure. Human Kinetics, 1996.

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8

Gu, Lie. Caffeine metabolism as index of cytochrome P4501A2 activity. National Library of Canada = Bibliothèque nationale du Canada, 1993.

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9

Aldersons, A. A. Psikhofiziologicheskie reakt͡s︡ii ėnergoobmena. "Zinatne", 1989.

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10

Franco, Angeleri, ed. Analysis of the electrical activity of the brain. Wiley, 1997.

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11

G, Shulman R., and Rothman D. L, eds. Brain energetics and neuronal activity: Applications to fMRI and medicine. Wiley, 2004.

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12

Viru, A. A. Hormones in muscular activity. CRC Press, 1985.

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13

Marin, Eine. Adaptation of glutathione metabolism to physical activity in dog, rat and man. University of Kuopio, 1995.

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14

G, Burns Richard, and Dick Richard P. 1950-, eds. Enzymes in the environment: Activity, ecology, and applications. Marcel Dekker, 2002.

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15

Royal Society of Chemistry (Great Britain), ed. Polymeric materials with antimicrobial activity: From synthesis to applications. RSC Publishing, 2014.

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16

Cudic, Predrag. Peptide Modifications to Increase Metabolic Stability and Activity. Humana Press, 2016.

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17

Lilleker, James B., and Mark E. Roberts. Metabolic myopathies. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0005.

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Metabolic myopathies are caused by defects in the metabolic processes of energy storage and utilization, and can present with exercise intolerance, fatigue, muscle pain, and weakness. Metabolic myopathies are rare and can be difficult to diagnose. However, the clinical presentation can be similar to, and thus mimic, both the idiopathic inflammatory myopathies and other genetic muscle disorders including the muscular dystrophies. Careful enquiry about the nature and timing of muscle pain, as well as identification of other clinical ‘red-flags’, can highlight the possibility of a metabolic myopa

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18

Effective Presentation of Metabolic Rate Information for Lunar Extravehicular Activity (EVA). Independently Published, 2019.

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19

Martínez-François, Juan Ramón, Nika N. Danial, and Gary Yellen. Metabolic Seizure Resistance via BAD and KATP Channels. Edited by Detlev Boison. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0028.

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On a ketogenic diet, ketone bodies provide an alternative fuel, replacing much of the glucose used ordinarily by the brain. This switch is thought to underlie its anticonvulsant effects. Brain fuel utilization can also be modified by a nondietary approach: genetic alteration of the protein BAD, which has known roles in regulating both apoptosis and glucose metabolism. When the metabolic function of BAD is genetically altered in mice, it produces reduced glucose and increased ketone body metabolism in neurons and astrocytes. This effect is related to regulation of BAD by phosphorylation and is

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20

Franks, Paul W., and Helen C. Looker. Gene–physical activity interactions and their role in determining cardiovascular and metabolic health. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199232482.003.0026.

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Chapter 26 investigates the mechanisms that underlie children’s health-related behaviours and the way in which physical activity interacts with genetic factors, which may help improve our understanding of how and why children become obese and develop cardiovascular risk factors, how these children should be treated, and ultimately how the development of cardiovascular risk in childhood can be prevented.

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21

Emmerling, Marcel. Metabolic responses to manipulations of pyruvate kinase regulation and activity in Escherichia coli. 2000.

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22

Physical activity in disease prevention and treatment. Piccin, 1985.

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23

Sutter, Raoul, Trudy Pang, and Peter W. Kaplan. EEG in Metabolic Disorders, Intoxications, and Epileptic Encephalopathies. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0017.

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This chapter provides a systematic overview of the diagnostic and prognostic value of electroencephalography (EEG) in adult patients with different types of encephalopathies in association with metabolic, toxic, and epileptic disorders. Most encephalopathies present with a fluctuating course characterized by typical but not pathognomonic symptoms such as cognitive impairment, altered mental status or confusion, lethargy, decreased or rarely increased motor activity, and disturbed sleep/wake cycles. EEG enables rapid, bedside electrophysiological monitoring, providing dynamic real-time informat

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24

van Mil, Edgar GAH. Exercise, physical activity, and diabetes mellitus. Edited by Neil Armstrong and Willem van Mechelen. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198757672.003.0023.

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The main aim for the diabetic child is to live as normal a life as possible via four main targets of management: obtaining good metabolic control, preventing long-term complications, promoting social competence, and self-worth. The main challenges are that the child must receive appropriate insulin subcutaneously and there must be attention given to lifestyle. Participation in sports not only helps to reach those targets, but also forms a great challenge in maintaining good metabolic control as physical activity has an impact on glucose utilization. This chapter discusses the definition of dia

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25

Afrand, Mohammadhosain, Mohammad Afkhami-Ardekani, Ahmad Shojaoddiny-Ardekani, and Azita Ariaeinejad. Metabolic Syndrome and Ethnic Groups Second edition. Knowledge Kingdom Publishing, 2021. http://dx.doi.org/10.26415/978-9931-9446-4-5.

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Metabolic syndrome is defined as the co-occurrence of metabolic risk factors for both type 2 diabetes and cardiovascular disease (i.e. abdominal obesity, hyperglycemia, dyslipidemia, and hypertension). Indeed, metabolic syndrome is an important risk factor for subsequent development of type 2 diabetes and/or CVD. Thus, the key clinical implication of a diagnosis of metabolic syndrome is the identification of a patient who needs aggressive lifestyle modification focused on weight reduction and increased physical activity. Multiple different phenotypes and ethnic-specific values for waist circum

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26

Young, Allen Jack. In vitro hormonal and metabolic control of bovine mammary glucose 6-phosphate dehydrogenase activity. 1987.

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27

Katsagoni, Christina, Peter Kokkinos, and Labros S. Sidossis. Prevention and Management of Cardiovascular and Metabolic Disease: Physical Activity, Fitness and Healthy Aging. Wiley & Sons, Incorporated, John, 2023.

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28

Katsagoni, Christina, Peter Kokkinos, and Labros S. Sidossis. Prevention and Management of Cardiovascular and Metabolic Disease: Physical Activity, Fitness and Healthy Aging. Wiley & Sons, Incorporated, John, 2023.

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29

Katsagoni, Christina, Peter Kokkinos, and Labros S. Sidossis. Prevention and Management of Cardiovascular and Metabolic Disease: Physical Activity, Fitness and Healthy Aging. Wiley & Sons, Incorporated, John, 2023.

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30

Katsagoni, Christina, Peter Kokkinos, and Labros S. Sidossis. Prevention and Management of Cardiovascular and Metabolic Disease: Physical Activity, Fitness and Healthy Aging. Wiley & Sons, Limited, John, 2023.

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31

Mottram, Linda-Jayne, and Gavin G. Lavery. The metabolic and nutritional response to critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0202.

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The metabolic response to critical illness is complex and affects every body system. The first phase of this response is characterized by increased hypothalamic pituitary activity and resistance (decreased response) to effector hormones in many target tissues. Cytokines released in the early stages of such illness may be important as they appear to stimulate the hypothalamic pituitary axis directly as part of this ‘stress response’. This phase is considered ‘adaptive’ (helpful), increasing the availability of glucose, free fatty acids, and amino acids as substrates for vital organs. However, i

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32

Hilbert, Scott Allen. Use of ribosomal RNA content as an indicator of the metabolic activity of suspended and attached bacteria. 1993.

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33

Sturgill, James Fitzhugh. Activity-dependent regulation of synapse structure and function: Roles of PSD-95 and the metabolic sensor, AMPK. 2010.

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34

Studies of intercellular communication and intracellular metabolic responses by bone cells to simulated weightlessness: Final NASA report. National Aeronautics and Space Administration, 1997.

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35

O'Connor, Kieran G. Inter-relationships of spontaneous growth hormone axis activity, body fat and metabolic variables in health elderly women and men. 1997.

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36

Sloboda, Deborah M. The impact of prenatal glucocorticoid administration on the development and long term activity of the hypothalamic-pituitary-adrenal and metabolic axes. 2001.

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37

Insulin Resistance Diet Cookbook: Prevent Diabetes by Reversing Pre-Diabetes, Repairing Your Metabolic Activity, Losing Excess Weight and Boost Your Fertility. Independently Published, 2021.

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38

Testa, Bernard, and John Caldwell. Biochemistry of Redox Reactions. Elsevier Science & Technology Books, 1994.

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39

Armstrong, Neil, Alan R. Barker, and Alison M. McManus. Muscle metabolism during exercise. Edited by Neil Armstrong and Willem van Mechelen. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198757672.003.0006.

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Ethical considerations and the lack, until recently, of accessible non-invasive techniques of interrogating exercising muscles have limited research into developmental muscle metabolism during exercise. Current evidence supports an anaerobic/aerobic energy metabolism interplay in exercise in which children present a relatively higher oxidative capacity than adolescents or adults. There is a progressive increase in anaerobic glycolytic flux with age at least into adolescence and, possibly into young adulthood. Independent effects of biological maturation on muscle metabolism during exercise rem

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40

Veech, Richard L., and M. Todd King. Alzheimer’s Disease. Edited by Detlev Boison. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0026.

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Deficits in cerebral glucose utilization in Alzheimer’s disease (AD) arise decades before cognitive impairment and accumulation of amyloid plaques and neurofibrillary tangles in brain. Addressing this metabolic deficit has greater potential in treating AD than targeting later disease processes – an approach that has failed consistently in the clinic. Cerebral glucose utilization requires numerous enzymes, many of which have been shown to decline in AD. Perhaps the most important is pyruvate dehydrogenase (PDH), which links glycolysis with the Krebs cycle and aerobic metabolism, and whose activ

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41

(Editor), Bernard Testa, and John Caldwell (Editor), eds. Biochemistry of Redox Reactions (Metabolism of Drugs and Other Xenobiotics). Academic Press, 1994.

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42

Clarke, Andrew. Endothermy. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780199551668.003.0010.

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Endothermy is the maintenance of a high and relatively constant internal body temperature, where the principal source of heat is a high metabolic rate at rest. The main sources of this heat are the visceral organs (especially the liver, spleen and gut), which tend to be larger and with greater metabolic capacity than in ectotherms. An important contribution also comes from heat produced by muscular activity during routine daily activity. Among living animals, only mammals and birds are true endotherms. Body temperatures are generally higher in bird than in mammals, and in both groups mean body

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43

Bender, David A. 2. Energy nutrition. Oxford University Press, 2014. http://dx.doi.org/10.1093/actrade/9780199681921.003.0002.

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Apart from water, the body’s first requirement under all conditions is for an energy source to perform physical and chemical work. ‘Energy nutrition’ explains that the metabolic fuels to provide this energy are derived from fats, carbohydrates, protein, and alcohol in the diet. The constituents of a meal provide these fuels directly for a few hours. Simultaneously, reserves of fat and carbohydrate are laid down for use during fasting between meals. Only about one-third of the average person’s energy expenditure is for voluntary activity; two-thirds is required for maintenance of the body’s fun

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44

³¹P metabolic responses to activity of nonspecifically trained muscle tissue in elite endurance athletes and in healthy, sedentary subjects as observed by ³¹P magnetic resonance spectroscopy. 1992.

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45

³¹P metabolic responses to activity of nonspecifically trained muscle tissue in elite endurance athletes and in healthy, sedentary subjects as observed by ³¹P magnetic resonance spectroscopy. 1992.

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46

Keshav, Satish, and Alexandra Kent. Unintentional weight loss. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0080.

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Body weight is determined by the combination of metabolic rate, calorie intake, and activity levels. Natural weight loss is usually due to declining muscle mass, with the redistribution of muscle mass in the extremities, leading to greater truncal fat stores. Unintentional weight loss refers to weight loss that is not voluntary, and can reflect serious underlying pathology. It can be caused by inadequate nutritional intake, increased metabolism, malabsorption, or a combination of these factors. Weight loss of 5% of body weight over 6–12 months should be investigated. Cachexia is a complex synd

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47

L, Brown Richard. p31sP metabolic responses to activity of nonspecifically trained muscle tissue in elite endurance athletes and in healthy, sedentary subjects as observed by p31sP magnetic resonance spectroscopy. 1992.

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48

(Editor), Artemis P. Simopoulos, and Konstantinos N. Pavlou (Editor), eds. Nutrition and Fitness, Diet, Genes, Physical Activity and Health Metabolic Studies in Health and Disease: 4th International Conference on Nutrition and ... 2000 (World Review of Nutrition & Dietetics). 2nd ed. Not Avail, 2001.

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49

Albright, Robert C. Acid-Base and Electrolyte Disorders. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0474.

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The most important principle in understanding disorders of water balance is that sodium balance is determined by the adequacy of the effective circulating volume, while water balance is determined by osmoregulation and the interplay between vasopressin activity, renal concentrating and diluting ability, and thirst. Disorders of sodium balance can be determined only by clinical examination. Orthostatic hypotension implies volume depletion and sodium deficiency. Edema implies volume excess and sodium excess. Potassium is predominantly an intracellular cation. The intracellular balance of potassi

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50

Maquet, Pierre, and Julien Fanielle. Neuroimaging in normal sleep and sleep disorders. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0011.

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Functional neuroimaging techniques include methods that probe various aspects of brain function and help derive models of brain organization in health and disease. These techniques can be grouped in two categories. Some are mainly based on electromagnetic signals (electroencephalography, magnetoencephalography), recording brain activity using a large number of sensors with exquisite temporal resolution (usually of the order of a kilohertz) but allowing only indirect characterization of three-dimensional brain activity by resorting to mathematical models. The second type includes different tech

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