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Journal articles on the topic 'Metabolic therapy'

Author: Grafiati

Published: 28 May 2022

Last updated: 31 July 2025

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1

Tanashyan, Tanashyan M. M., Antonova K. V. Antonova, Spryshkov N. E. Spryshkov, and Panina A. A. Panina. "Metabolic markers of cerebrovascular pathology." Therapy 5_2024 (July 18, 2024): 84–92. http://dx.doi.org/10.18565/therapy.2024.5.84-92.

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2

Malimon, Malimon V. V., and Kokorin V. A. Kokorin. "Antihypertensive therapy in a patient with metabolic syndrome." Therapy 7_2024 (October 14, 2024): 105–12. http://dx.doi.org/10.18565/therapy.2024.7.105-112.

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3

Tiselius, Hans-Göran. "Metabolic evaluation and therapy." Current Opinion in Urology 10, no. 6 (2000): 545–49. http://dx.doi.org/10.1097/00042307-200011000-00002.

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4

Antonova, Antonova K. V., Lagoda O. V. Lagoda, Shishkina T. S. Shishkina, and Tanashyan M. M. Tanashyan. "Metabolic comorbidity in case of cerebrovascular pathology." Therapy 5_2024 (July 18, 2024): 101–10. http://dx.doi.org/10.18565/therapy.2024.5.101-110.

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5

Lapshina, Lapshina S. A., Feyskhanova L. I. Feyskhanova, Nurmieva A. R. Nurmieva, and Sadriev K. A. Sadriev. "Osteoarthritis in patients with components of metabolic syndrome." Therapy 8_2019 (December 31, 2019): 52–58. http://dx.doi.org/10.18565/therapy.2019.8.52-58.

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6

Rühle, Annika, Adrian T. Billeter, and Beat P. Müller-Stich. "Metabolic Surgery: Paradigm Shift in Metabolic Syndrome/Diabetes Therapy." Visceral Medicine 38, no. 1 (2022): 56–62. http://dx.doi.org/10.1159/000521707.

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Background: Obesity and metabolic disorders as type 2 diabetes (T2D), nonalcoholic fatty liver disease (NAFLD) or better called metabolic dysfunction fatty liver disease (MAFLD), arterial hypertension (AHT), and obstructive sleep apnea syndrome (OSAS) show a rising prevalence. The increased cardiovascular risk is one of the main causes for death of obese, metabolic ill patients. Sustainable and efficient therapeutic options are needed. Summary: Metabolic surgery not only permits a substantial and lasting weight loss but also ameliorates metabolic co-morbidities and reduces cardiovascular risk

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7

Shishkova, Shishkova V. N., and Martynov A. I. Martynov. "Modern opportunities of therapy for metabolic cardiomyopathy and heart failure." Therapy 6_2020 (September 22, 2020): 139–49. http://dx.doi.org/10.18565/therapy.2020.6.139-149.

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8

Lopatin, Yury. "Metabolic Therapy in Heart Failure." Cardiac Failure Review 1, no. 2 (2015): 112. http://dx.doi.org/10.15420/cfr.2015.1.2.112.

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Metabolic impairments play an important role in the development and progression of heart failure. The use of metabolic modulators, the number of which is steadily increasing, may be particularly effective in the treatment of heart failure. Recent evidence suggests that modulating cardiac energy metabolism by reducing fatty acid oxidation and/or increasing glucose oxidation represents a promising approach to the treatment of patients with heart failure. This review focuses on the role of metabolic modulators, in particular trimetazidine, as a potential additional medication to conventional medi

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9

Chandler, Randy J., and Charles P. Venditti. "Gene therapy for metabolic diseases." Translational Science of Rare Diseases 1, no. 1 (2016): 73–89. http://dx.doi.org/10.3233/trd-160007.

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10

&NA;. "Metabolic consequences of antihypertensive therapy." Inpharma Weekly &NA;, no. 1045 (1996): 24. http://dx.doi.org/10.2165/00128413-199610450-00055.

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11

Vertkin, A. L., A. S. Sycheva, A. V. Nosova, G. U. Knorring, and G. V. Zavyalov. "Metabolic therapy for comorbid pathology." Medical alphabet 1, no. 19 (2020): 19–23. http://dx.doi.org/10.33667/2078-5631-2020-19-19-23.

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Demographic changes, the increasing frequency of comorbidity make us pay close attention to the prescribed therapy and control of drug to drug interactions. The use of polymodal drugs with pleiotropic effects that affect the key points of development of comorbid pathology may be promising.

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12

&NA;. "Metabolic consequences of antihypertensive therapy." Reactions Weekly &NA;, no. 609 (1996): 3. http://dx.doi.org/10.2165/00128415-199606090-00005.

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13

Diggins, Caroline E., Samuel C. Russo, and Janet Lo. "Metabolic Consequences of Antiretroviral Therapy." Current HIV/AIDS Reports 19, no. 2 (2022): 141–53. http://dx.doi.org/10.1007/s11904-022-00600-6.

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14

Olin, Jeffrey W. "Metabolic consequences of antihypertensive therapy." Journal of the American Osteopathic Association 87, no. 2 (1987): 150–52. http://dx.doi.org/10.1515/jom-1987-870233.

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15

Alpers, David H. "Vitamin therapy for metabolic diseases." Current Opinion in Gastroenterology 19, no. 2 (2003): 130–33. http://dx.doi.org/10.1097/00001574-200303000-00005.

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16

Macdougall, Iain. "Metabolic Adjuvants to Erythropoietin Therapy." Mineral and Electrolyte Metabolism 25, no. 4-6 (1999): 357–64. http://dx.doi.org/10.1159/000057475.

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17

Galluzzi, Lorenzo, Oliver Kepp, Matthew G. Vander Heiden, and Guido Kroemer. "Metabolic targets for cancer therapy." Nature Reviews Drug Discovery 12, no. 11 (2013): 829–46. http://dx.doi.org/10.1038/nrd4145.

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18

Mancini, Mario, Teodoro Marotta, and L. Aldo Ferrara. "Metabolic Neutrality in Nitrendipine Therapy." Journal of Cardiovascular Pharmacology 18 (1991): S30—S33. http://dx.doi.org/10.1097/00005344-199100181-00007.

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19

Kaehny, William D., and Robert J. Anderson. "Bicarbonate therapy of metabolic acidosis." Critical Care Medicine 22, no. 10 (1994): 1525–27. http://dx.doi.org/10.1097/00003246-199410000-00003.

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20

Kaehny, William D., and Robert J. Anderson. "Bicarbonate therapy of metabolic acidosis." Critical Care Medicine 22, no. 10 (1994): 1525–27. http://dx.doi.org/10.1097/00003246-199422100-00003.

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21

Agrillo, Corrado. "Metabolic Training and Herbal Therapy." Journal of Nutritional Ecology and Food Research 4, no. 1 (2017): 77–83. http://dx.doi.org/10.1166/jnef.2017.1136.

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22

BOSCHERT, SHERRY. "Apnea Therapy Improves Metabolic Measures." Internal Medicine News 43, no. 4 (2010): 42. http://dx.doi.org/10.1016/s1097-8690(10)70230-4.

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23

Bowles, Neil, and Savio L. C. Woo. "Gene therapy for metabolic disorders." Advanced Drug Delivery Reviews 17, no. 3 (1995): 293–302. http://dx.doi.org/10.1016/0169-409x(95)00063-d.

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24

Kay, Mark A., and Savio L. C. Woo. "Gene therapy for metabolic disorders." Trends in Genetics 10, no. 7 (1994): 253–57. http://dx.doi.org/10.1016/0168-9525(94)90173-2.

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25

Mancini, Mario, Teodoro Marotta, and L. Aldo Ferrara. "Metabolic Neutrality in Nitrendipine Therapy." Journal of Cardiovascular Pharmacology 18 (1991): S30—S33. http://dx.doi.org/10.1097/00005344-199106181-00007.

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26

Fragasso, Gabriele, Anna Salerno, Roberto Spoladore, Giorgio Bassanelli, Francesco Arioli, and Alberto Margonato. "Metabolic Therapy of Heart Failure." Current Pharmaceutical Design 14, no. 25 (2008): 2582–91. http://dx.doi.org/10.2174/138161208786071245.

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27

Kay, M. A., and S. L. C. Woo. "Gene Therapy for Metabolic Diseases." ILAR Journal 36, no. 3-4 (1994): 47–53. http://dx.doi.org/10.1093/ilar.36.3-4.47.

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28

Fischer, Alain, Salima Hacein-Bey-Abina, and Marina Cavazzana-Calvo. "Gene therapy of metabolic diseases." Journal of Inherited Metabolic Disease 29, no. 2-3 (2006): 409–12. http://dx.doi.org/10.1007/s10545-006-0270-7.

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29

FAADIELESSOP, M., and L. OPIE. "Metabolic therapy for heart failure." European Heart Journal 25, no. 20 (2004): 1765–68. http://dx.doi.org/10.1016/j.ehj.2004.08.019.

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30

Wang, Fubao, R. Michael Raab, Michael W. Washabaugh, and Barry C. Buckland. "Gene Therapy and Metabolic Engineering." Metabolic Engineering 2, no. 2 (2000): 126–39. http://dx.doi.org/10.1006/mben.1999.0144.

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31

BOSCHERT, SHERRY. "Apnea Therapy Improves Metabolic Measures." Clinical Endocrinology News 5, no. 1 (2010): 15. https://doi.org/10.1016/s1558-0164(10)70016-x.

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32

Vertkin, Vertkin A. L., Shishkova V. N. Shishkova, Sycheva A. S. Sycheva, et al. "Options of metabolic support in case of coronaviral infection." Therapy 7_2020 (October 26, 2020): 146–55. http://dx.doi.org/10.18565/therapy.2020.7.146-155.

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33

Řiháčková, Eva, Lubomír Elbl, Michal Řiháček, Mária Holická, and Petr Kala. "Anti-cancer therapy-induced metabolic syndrome." Vnitřní lékařství 67, no. 6 (2021): 334–38. http://dx.doi.org/10.36290/vnl.2021.089.

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34

Řiháčková, Eva, Lubomír Elbl, Michal Řiháček, Mária Holická, and Petr Kala. "Anti-cancer therapy-induced metabolic syndrome." Vnitřní lékařství 67, no. 6 (2021): 334–38. http://dx.doi.org/10.36290/vnl.2021.089.

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35

NODE, KOICHI. "Clinical condition of metabolic syndrome. 5 Therapy of metabolic syndrome." Nihon Naika Gakkai Zasshi 95, no. 9 (2006): 1737–41. http://dx.doi.org/10.2169/naika.95.1737.

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36

Node, Koichi. "Clinical Diagnosis of Metabolic Syndrome 4. Therapy for Metabolic Syndrome." Internal Medicine 46, no. 16 (2007): 1289. http://dx.doi.org/10.2169/internalmedicine.46.1906.

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37

Petković, Z., and M. Mirković. "GOUT - DIAGNOSTIC DIAGNOSTIC AND THERAPY THERAPY - case raport." Praxis medica 34, no. 2 (2006): 107–8. http://dx.doi.org/10.70949/pramed200602164p.

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&lt;p&gt;Gout is a metabolic arthropathy caused by abnormal deposition of crystal histological they are urats (salts) of mononatrium in the joints and periatricular connective tissue. In 70% cases, decreased renal function in urats&amp;rsquo;excretion is responsible for hyperuricaemya. The other causes are mostly metabolic: increased (take in) of food wich contains mostly meat (purins) and increased synthesis of purin due to defective enzyme system (i.e. Lesch-Nyhan Sy)&lt;/p&gt;

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38

MANGYO, Rika. "Metabolic Syndrome Prevention:." Rigakuryoho Kagaku 25, no. 6 (2010): 957–64. http://dx.doi.org/10.1589/rika.25.957.

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39

Kurdanov, Kurdanov Kh A., Elbaeva A. D. Elbaeva, Elbaev A. D. Elbaev, and Kurdanova M. Kh Kurdanova. "Simulation modeling of the correlation between the components of metabolic syndrome." Therapy 5_2025 (July 21, 2025): 73–83. https://doi.org/10.18565/therapy.2025.5.73-83.

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40

Rathore, Richa, and Brian Van Tine. "Developing a Metabolic Therapy for Osteosarcoma." Oncology Times 43, no. 8 (2021): 1,4–4. http://dx.doi.org/10.1097/01.cot.0000749860.15197.c8.

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41

Perez-Martinez, Pablo, and Jose Lopez-Miranda. "Editorial Nutritional Therapy in Metabolic Syndrome." Current Vascular Pharmacology 11, no. 6 (2014): 838–41. http://dx.doi.org/10.2174/157016111106140128113839.

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42

Galeffi, Francesca, and Dennis A. Turner. "Exploiting Metabolic Differences in Glioma Therapy." Current Drug Discovery Technologies 9, no. 4 (2012): 280–93. http://dx.doi.org/10.2174/157016312803305906.

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43

FUJII, Satoshi. "Anti-thrombotic therapy for metabolic syndrome." Japanese Journal of Thrombosis and Hemostasis 18, no. 6 (2007): 607–13. http://dx.doi.org/10.2491/jjsth.18.607.

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44

Mananko, E. I., E. A. Bushkova, E. M. Idrisova, and A. I. Vengerovsky. "COMBINED ANTIHYPERTENSIVE THERAPY IN METABOLIC SYNDROME." Rational Pharmacotherapy in Cardiology 4, no. 3 (2008): 52–58. http://dx.doi.org/10.20996/1819-6446-2008-4-3-52-58.

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45

Chubenko, V. A., and V. M. Moiseyenko. "Metabolic cancer therapy: proof of concept." Practical oncology 23, no. 1 (2022): 51–61. http://dx.doi.org/10.31917/2301051.

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Today cancer is considered as a genetic disease without taking into account some metabolic features of the cells due to these molecular abnormalities. At the same time, accumulated scientific data indicate that energy homeostasis of the malignant cells and their microenvironment plays one of the leading roles in the pathogenesis of the neoplasms and the potential treatment sensitivity. Multiple heterogeneity and plasticity of the metabolic signals, as well as their similarity to the energy pathways in normal cells notes the need to find an optimal model for their further research and implement

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46

Pragier, Uriel. "METABOLIC IMPACT OF CARDIAC HORMON THERAPY." Revista de la Sociedad Argentina de Diabetes 53, no. 2Sup (2020): 13. http://dx.doi.org/10.47196/diab.v53i2sup.227.

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Gender dysphoria (DG) implies a discordance between the gender assigned at birth and the self-perceived one. Male transgender or male transsexual (FTM) are identified with the male gender and have been assigned as female at birth. Transgender women or transsexual women (MTF) are identified as women but have been assigned as men at birth. There are those who do not seek extreme masculinization or feminization, which allows the term "sexual diversity" to cover these cases.

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47

Tolstov, S. N., and V. B. Mychka. "Metabolic (cytoprotective) therapy of menopausal disturbances." Cardiovascular Therapy and Prevention 10, no. 3 (2011): 72–75. http://dx.doi.org/10.15829/1728-8800-2011-3-72-75.

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The paper presents modern views on cardiovascular aspects of menopause and metabolic therapy of menopausal disturbances with meldonium (Mildronate®). The role of estrogen deficiency in climacteric disturbance development, key pathogenetic mechanisms of menopausal metabolic syndrome (MS), and relevant features of arterial hypertension and endothelial dysfunction development are discussed. The data on Mildronate® clinical use for cardiovascular prevention are summarized. The wide prevalence and multiple clinical manifestations of menopausal disturbances point to the need for their complex therap

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48

Coons, Barbara, and William H. Peranteau. "Prenatal Gene Therapy for Metabolic Disorders." Clinical Obstetrics & Gynecology 64, no. 4 (2021): 904–16. http://dx.doi.org/10.1097/grf.0000000000000662.

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49

Евсегнеев, Р. А. "Metabolic side effects of Antipsychotic therapy." Психиатрия, психотерапия и клиническая психология, no. 1 (June 16, 2020): 146–54. http://dx.doi.org/10.34883/pi.2020.11.1.014.

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Статья посвящена метаболическим побочным эффектам, возникающим в ходе терапии антипсихотиками. Обсуждены причины, повышающие актуальность проблемы, механизмы возникновения и факторы риска метаболических нарушений, критерии их диагностики, а также особенности в этом плане отдельных антипсихотических препаратов. Приведен порядок действий психиатра для раннего выявления, профилактики и лечения метаболических побочных эффектов антипсихотической терапии. The article is devoted to metabolic side effects of antipsychotic medication. Some reasons of aggravation of the problem, mechanisms and risk fact

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50

Zhao, Yuhua. "Emerging Metabolic Targets in Cancer Therapy." Frontiers in Bioscience 16, no. 1 (2011): 1844. http://dx.doi.org/10.2741/3826.

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