Relevant bibliographies by topics / Metastatic cancer

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Metastatic cancer'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "Metastatic cancer"

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Yuk, Hyeong-Dong, Kyoung-Hwa Lee, Hye-Sun Lee, et al. "PDLIM2 Suppression Inhibit Proliferation and Metastasis in Kidney Cancer." Cancers 13, no. 12 (2021): 2991. http://dx.doi.org/10.3390/cancers13122991.

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We evaluated the expression of PDLIM2 in human kidney cancer cell lines from primary or metastatic origins and found that PDLIM2 expression was highly elevated in metastatic kidney cancers. We evaluated the effect of PDLIM2 inhibition by RNA interference method. PDLIM2 knockdown showed the decreased proliferation and metastatic character in human metastatic kidney cancer cells. By repeated round of orthotopic injection of RenCa mouse kidney cancer cell line, we obtained metastatic prone mouse kidney cancer cell lines. PDLIM2 expression was highly expressed in these metastatic prone cells compa
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Bonni, Shirin, David N. Brindley, M. Dean Chamberlain, et al. "Breast Tumor Metastasis and Its Microenvironment: It Takes Both Seed and Soil to Grow a Tumor and Target It for Treatment." Cancers 16, no. 5 (2024): 911. http://dx.doi.org/10.3390/cancers16050911.

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Metastasis remains a major challenge in treating breast cancer. Breast tumors metastasize to organ-specific locations such as the brain, lungs, and bone, but why some organs are favored over others remains unclear. Breast tumors also show heterogeneity, plasticity, and distinct microenvironments. This contributes to treatment failure and relapse. The interaction of breast cancer cells with their metastatic microenvironment has led to the concept that primary breast cancer cells act as seeds, whereas the metastatic tissue microenvironment (TME) is the soil. Improving our understanding of this i
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Lim, Geok Hoon, Jing Xue Hoo, You Chan Shin, Rachel Zhi Ting Choo, Fuh Yong Wong, and John Carson Allen. "Is Metastatic Staging Needed for All Patients with Synchronous Bilateral Breast Cancers?" Cancers 16, no. 1 (2023): 17. http://dx.doi.org/10.3390/cancers16010017.

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Background: Patients with bilateral breast cancers are uncommon and are associated with a poorer prognosis. While metastatic staging guidelines in patients with unilateral cancer were established, the indication of metastatic staging in patients with bilateral breast cancers is unclear. We aimed to determine which patients with synchronous bilateral breast cancers require metastatic staging at diagnosis. This is the first such reported study, to the best of our knowledge. Methods: A retrospective review of newly diagnosed synchronous bilateral invasive breast cancer patients at our institution
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Ganguly, Debolina, Marcel Schmidt, Morgan Coleman, et al. "Abstract B016: Pleiotrophin drives a pro-metastatic immune niche within the breast tumor microenvironment." Cancer Research 83, no. 2_Supplement_2 (2023): B016. http://dx.doi.org/10.1158/1538-7445.metastasis22-b016.

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Abstract Metastatic cancer cells adapt to thrive in secondary organs. To investigate metastatic adaptation, we performed transcriptomic analysis of metastatic and non-metastatic murine breast cancer cells. We found that pleiotrophin (PTN), a neurotrophic cytokine, is a metastasis-associated factor that is expressed highly by aggressive breast cancers. Moreover, elevated PTN in plasma correlated significantly with metastasis and reduced survival of breast cancer patients. Mechanistically, we find that PTN activates NF-kB in cancer cells leading to altered cytokine production, subsequent neutrop
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Chahbounia, Imane, Saida Lamine, Khaoula Alaoui Slimani, et al. "Double Breast Neoplasia: RH+, HER2- Metastatic Left Breast Cancer and Triple Negative Metastatic Right Breast Cancer: Case Report." Scholars Journal of Medical Case Reports 12, no. 12 (2024): 2188–91. https://doi.org/10.36347/sjmcr.2024.v12i12.044.

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Breast cancer is the most common cancer in women, and the proportion of metastatic cancer is not negligible; either metastasis of a cancer that was initially localized, or metastatic disease from the outset. The case of double breast neoplasia is not exceptional; but when faced with two breast cancers, both metastatic, and with different molecular profiles, several questions arise. Case report: We report the case of a diabetic patient with localized RH+, HER2- left breast cancer treated with surgery and adjuvant chemotherapy in 2003, which became metastatic to the lung in 2022, and for which t
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Park, Jinkyung, Dahee Jeong, Meeryoung Song, and Bonglee Kim. "Recent Advances in Anti-Metastatic Approaches of Herbal Medicines in 5 Major Cancers: From Traditional Medicine to Modern Drug Discovery." Antioxidants 10, no. 4 (2021): 527. http://dx.doi.org/10.3390/antiox10040527.

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Metastasis is the main cause of cancer-related death. Despite its high fatality, a comprehensive study that covers anti-metastasis of herbal medicines has not yet been conducted. The aim of this study is to investigate and assess the anti-metastatic efficacies of herbal medicines in the five major cancers, including lung, colorectal, gastric, liver, and breast cancers. We collected articles published within five years using PubMed, Google Scholar, and Web of Science with “cancer metastasis” and “herbal medicine” as keywords. Correspondingly, 16 lung cancer, 23 colorectal cancer, 10 gastric can
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Pein, Maren, and Thordur Oskarsson. "Microenvironment in metastasis: roadblocks and supportive niches." American Journal of Physiology-Cell Physiology 309, no. 10 (2015): C627—C638. http://dx.doi.org/10.1152/ajpcell.00145.2015.

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In many cancers, malignant cells can spread from the primary tumor through blood circulation and initiate metastasis in secondary organs. Metastatic colonization may depend not only on inherent properties of cancer cells, but also on suitable microenvironments in distant sites. Increasing evidence suggests that the nature of the microenvironment may determine the fate of disseminated cancer cells, providing either hindrance or support for cancer cell propagation. This can result in strong selective pressure where the vast majority of cancer cells, invading a secondary organ, are either elimina
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Hao, Shuang, Liqun Chen, Wenhui Du, and Huiyan Sun. "A Comprehensive Comparison between Primary Liver Cancer and Liver Metastases through scRNA-Seq Data Analysis." Metabolites 14, no. 2 (2024): 90. http://dx.doi.org/10.3390/metabo14020090.

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Metastasis is one of the leading causes of cancer-related deaths. A comprehensive comparison of the differences between primary and metastatic cancers within the same organ can aid in understanding the growth mechanisms of cancer cells at metastatic sites, thereby helping to develop more effective targeted treatment strategies. Primary liver cancer is one of the most common types of cancer, and the liver is also one of the main metastatic sites. In this paper, we utilize single-cell RNA-Seq data to compare primary liver cancer and colorectal liver metastases from multiple perspectives, includi
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Wan, Xiaochun, and Junxin Li. "DcR3 acts as a tumor marker of metastatic cancers." Journal of Immunology 200, no. 1_Supplement (2018): 178.45. http://dx.doi.org/10.4049/jimmunol.200.supp.178.45.

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Abstract Decoy receptor 3 (DcR3) is a soluble protein that competitively binds Fas ligand and has an anti-apoptotic role. Abnormally elevated DcR3 in malignant tumors may help cancer cells metastasize by suppressing immune responses and establishing metastatic lesions. However, whether DcR3 is valuable in differentiating cancer metastasis from non-metastasis remains largely unclear. In this study, we produced anti-DcR3 monoclonal antibodies and established a sensitive ELISA (11–12000 pg/ml, R2= 0.9941) to measure serum levels of DcR3 in cancer patients. DcR3 was significantly elevated in gastr
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Chen, Chao, Haozhen Liu, Qumiao Xu, Xiuqing Zhang, Feng Mu, and Jixian Liu. "Association of PTPRT Mutations with Cancer Metastasis in Multiple Cancer Types." BioMed Research International 2022 (June 25, 2022): 1–9. http://dx.doi.org/10.1155/2022/9386477.

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Metastasis is one of the characteristics of advanced cancer and the primary cause of cancer-related deaths from cancer, but the mechanism underlying metastasis is unclear, and there is a lack of metastasis markers. PTPRT is a protein-coding gene involved in both signal transduction and cellular adhesion. It is also known as a tumor suppressor gene that inhibits cell malignant proliferation by inhibiting the STAT3 pathway. Recent studies have reported that PTPRT is involved in the early metastatic seeding of colorectal cancer; however, the correlation between PTPRT and metastasis in other types
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Dissertations / Theses on the topic "Metastatic cancer"

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Wander, Seth A. "p27 and Metastatic Progression: Molecular Mechanisms Underlying Bone Metastasis." Scholarly Repository, 2011. http://scholarlyrepository.miami.edu/oa_dissertations/690.

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The complex PI3K/mTOR pathway regulates tumor progression via effects on cellular proliferation, apoptosis, autophagy, and motility. New drugs that inhibit the catalytic site of both PI3K and mTOR have shown promise in clinical trials. Here, we report the first use of a novel, dual PI3K/mTOR catalytic site inhibitor (PF-04691502, PF1502) in a xenograft model of breast cancer metastasis to bone. Metastatic MDA-MB-1833 cells showed PI3K/mTOR activation relative to parental MDA-MB-231. Low-dose PF1502 significantly impaired tumor cell motility and invasion in vitro without causing cell cycle arre
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Nordman, Ina IC Clinical School St Vincent's Hospital Faculty of Medicine UNSW. "Surrogate endpoints of survival in metastatic carcinoma." Publisher:University of New South Wales. Clinical School - St Vincent's Hospital, 2008. http://handle.unsw.edu.au/1959.4/42791.

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In most randomised controlled trials (RCTs), a large number of patients need to be followed over many years, for the clinical benefit of the drug to be accurately quantified (1). Using an early proxy, or a surrogate endpoint, in place of the direct endpoint of overall survival (OS) could theoretically shorten the duration of RCTs and minimise the exposure of patients to ineffective or toxic treatments (2, 3). This thesis examined the relationship between surrogate endpoints and OS in metastatic colorectal cancer (CRC), advanced non-small cell lung cancer (NSCLC) and metastatic breast cancer (M
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Solberg, Arne. "Outcome Assessments in Non- Metastatic Prostate Cancer." Doctoral thesis, Norges teknisk-naturvitenskapelige universitet, Institutt for kreftforskning og molekylær medisin, 2011. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-13881.

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Metoder for å måle utfall av behandling hos pasienter med prostatakreft uten spredning. Prostatakreft uten påvist spredning kan behandles med operasjon, strålebehandling med eller uten hormonbehandling, eller hormonbehandling alene. Svært mange menn med nylig påvist prostatakreft vil imidlertid kunne leve i mange år uten plager av sykdommen også uten behandling, og en stor andel dør til slutt av helt andre årsaker enn prostatkreft. Man mangler imidlertid helt sikre metoder for å velge ut pasienter som ikke trenger behandling, men i noen tilfeller hvor sykdommen er i tidlig fase er et regelmess
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Paon, Lenaic Marie Pierre. "Gene expression profiling of metastatic gastric cancer." Thesis, University of Liverpool, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.490659.

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Gastric cancer is one of the main cancers in the world, and is responsible for a large number of deaths each year. In Europe, the main issue is that it is only detected at latter stages, when metastases have developed. Although metastasis is the main cause of death from such tumours, the process is a very complex one and still not fully understood. In order to unravel this mechanism, microarrays have been used to study the expression pattern of a large number of genes in primary tumours and metastatic samples. These studies aim at indentifying genes that may playa role in metastasis. A number
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Pelon, Floriane. "Fibroblastic heterogeneity and metastatic spread in breast cancers Fibroblast heterogeneity drives metastatic spread in breast cancer through distinct mechanisms." Thesis, Sorbonne Paris Cité, 2018. https://wo.app.u-paris.fr/cgi-bin/WebObjects/TheseWeb.woa/wa/show?t=2392&f=17330.

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Les cancers du sein, premiers cancers féminins et causes aujourd’hui encore de nombreux décès, sont classés en 3 sous-types moléculaires : luminaux –les plus répandus, HER2 et triple-négatifs (TN) –les plus agressifs. Lors du diagnostic, l’envahissement des ganglions lymphatiques axillaires par les cellules tumorales est établi. Il s’agit, en plus de la classification moléculaire, d’un marqueur pronostique utilisé en clinique pour stratifier les patientes, car il informe sur le risque de développement ultérieur de métastases, cause majeure des décès à l’heure actuelle. Les tumeurs solides, et
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Chiang, Yan Ting. "Identification of metastasis-driving genes as potential therapeutic targets/ biomarkers for metastatic prostate cancer." Thesis, University of British Columbia, 2015. http://hdl.handle.net/2429/52901.

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Metastatic prostate cancer is currently incurable. Metastasis is thought to result from changes in the expression of specific metastasis-driving genes, leading to a cascade of activated downstream genes setting the metastatic process in motion. As such, metastasis-driving genes could provide effective therapeutic targets and prognostic biomarkers for improved disease management. In search of potential metastasis-driving genes, genes with elevated expression in patient-derived metastatic LTL-313H prostate cancer tissues, as distinct from non-metastatic LTL-313B tissues, were identified. Among t
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Nielsen, S. R. "Metastasis-associated macrophages orchestrate the formation of a hospitable metastatic niche in pancreatic cancer." Thesis, University of Liverpool, 2017. http://livrepository.liverpool.ac.uk/3007527/.

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Pancreatic ductal adenocarcinoma (PDAC) is one of the most lethal cancers with an overall 5-year survival rate < 5%, a rate that has not improved for a long time. The dismal prognosis for PDAC is part due to late detection, often at advanced stages of the disease where patients have developed distant metastases, but also due to chemotherapeutical resistance. Patients eligible for surgical resection of the pancreatic tumour has the best prognosis, but even when resection is successful, patients often relapse with distant metastases within 2 years after surgery. Macrophages promote tumourigenesi
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Reed, Elizabeth. "The experience of living with metastatic breast cancer." Thesis, University of Southampton, 2012. https://eprints.soton.ac.uk/344746/.

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Over the last 10-15 years the medical management of metastatic breast cancer has improved survival, so women are living longer with progressive disease. Little is understood about women’s problems and needs and how they live their everyday lives. This study aimed to explore the experience of women with metastatic breast cancer by applying three phases: a cross-sectional survey exploring quality of life, experience of care and where they turned for support; exploration of the narratives of 30 women considering the social consequences of living with progressive breast cancer on identity; and fin
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He, Felicia Jane. "Targeting Metastatic Breast Cancer Using Dual-Ligand Nanoparticles." Case Western Reserve University School of Graduate Studies / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=case1499699087340348.

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Donald, Carlton Dewitt. "Metastatic characteristics of tumor progression in Prostate Cancer." DigitalCommons@Robert W. Woodruff Library, Atlanta University Center, 1995. http://digitalcommons.auctr.edu/dissertations/3299.

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Tumor biologist have long appreciated that both cell to cell and cell to extracellular matrix (ECM) interactions are involved in the invasive and metastatic events that are characteristic of malignancy. Cancer cell attachment to and invasion of an ECM has been associated with metastatic potential of cell lines of the Dunning rat prostate model. It was postulated that differences observed in the metastatic potential of four Dunning cell lines may correlate with cell-matrix interactions. Four cell lines, highly metastatic ML, MLL, AT-3 and non-metastatic AT-1 were studied. The adhesive, invasive
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Books on the topic "Metastatic cancer"

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Sherbet, G. V. The Metastatic Spread of Cancer. Palgrave Macmillan UK, 1987. http://dx.doi.org/10.1007/978-1-349-09577-3.

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A, Rosenberg Steven, ed. Surgical treatment of metastatic cancer. Lippincott, 1987.

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Swanton, Charles, and Stephen R. D. Johnston. Handbook of metastatic breast cancer. 2nd ed. Informa Healthcare, 2011.

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Leong, Stanley P. L., ed. From Local Invasion to Metastatic Cancer. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-087-8.

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National Cancer Institute (U.S.), ed. Questions and answers about metastatic cancer. National Cancer Institute, 1990.

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Kaufmann, M. Therapeutic management of metastatic breast cancer. Walter de Gruyter & Co, 1989.

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H, Schroeder Fritz, and Richards B. A, eds. Therapeutic principles in metastatic prostatic cancer. A.R. Liss, 1985.

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1949-, Enghofer E., Henderson I. Craig, and Kaufmann M. 1946-, eds. Therapeutic management of metastatic breast cancer. Walter de Gruyter, 1989.

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G, Ward William, and Peabody Terrance D, eds. Orthopedic management of metastatic disease. W.B. Saunders, 2000.

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Chu, Edward. New treatment strategies for metastatic colorectal cancer. CMP Medica, 2008.

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Book chapters on the topic "Metastatic cancer"

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Szmulewitz, Russell, Jennifer Taylor, and Carrie Rinker-Schaffer. "Metastatic Colonization." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_3676-2.

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Szmulewitz, Russell, Jennifer Taylor, and Carrie Rinker-Schaffer. "Metastatic Colonization." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-46875-3_3676.

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Szmulewitz, Russell, Jennifer Taylor, and Carrie Rinker-Schaffer. "Metastatic Colonization." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_3676.

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Alcin, Adem, and Halil Lutfi Canat. "Bone Health and Bone Targeted Therapies for Metastatic Prostate Cancer." In Current Management of Metastatic Prostate Cancer. Nobel Tip Kitabevleri, 2024. http://dx.doi.org/10.69860/nobel.9786053359142.15.

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This section provides information about bone health and bone-targeted therapies in metastatic prostate cancer. The most common site of metastasis for prostate cancer is the bone. Treatments used in cancer management can also have adverse effects on bones. Bone-related conditions in metastatic prostate cancer are one of the most important causes of morbidity and mortality. In bone-targeting treatments, resorption modulators and radiotherapy are the main components. Radionuclide agents are innovative treatments for targeting bones.
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Field, Kathryn M., and John R. Zalcberg. "Chemotherapy: Metastatic Disease." In Rectal Cancer. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-567-5_12.

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Sumpter, K., and D. Cunningham. "Metastatic Rectal Cancer." In Modern Management of Cancer of the Rectum. Springer London, 2001. http://dx.doi.org/10.1007/978-1-4471-0331-8_14.

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Halfdanarson, Thorvardur R., and Joleen M. Hubbard. "Metastatic Rectal Cancer." In Modern Management of Cancer of the Rectum. Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-6609-2_20.

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Patel, Tejal A., and Edith A. Perez. "Metastatic Breast Cancer." In Management of Breast Diseases. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-69743-5_23.

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Stephens, Frederick O., and Karl Reinhard Aigner. "Metastatic (Secondary) Cancer." In Basics of Oncology. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-92925-3_22.

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Stephens, Frederick O., and Karl Reinhard Aigner. "Metastatic (Secondary) Cancer." In Basics of Oncology. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-23368-0_22.

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Conference papers on the topic "Metastatic cancer"

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Yoshimoto, T., T. Ishikawa, N. Matsuki, et al. "Rheology of Cancer Cells With Different Metastatic Properties." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206593.

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Cancer is the leading cause of death in Japan as well as many other countries. One of the most serious problem of cancer is that cancer cells often migrate to a distant part of the body, referred to as metastasis. The rheological properties of cancer cells have been investigated by some reserchers [1,2]. However, the correlations between the metastasis and the rheological properties are still unclear, because of limited number of experimental cases reported so far. In this study, we used two kinds of human breast cancer cell lines, MCF-7 and KPL-4. It is known that KPL-4 has much higher metast
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Costa, Rafael Everton Assunção Ribeiro da, Fergus Tomás Rocha de Oliveira, Eduarda Norberto Siqueira, Ana Lúcia Nascimento Araújo, and Sabas Carlos Vieira. "CUTANEOUS AND BONE METASTASIS OF OCCULT BREAST CANCER: CASE REPORT." In Abstracts from the Brazilian Breast Cancer Symposium - BBCS 2021. Mastology, 2021. http://dx.doi.org/10.29289/259453942021v31s2078.

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Introduction: Occult breast cancer (OBC) is defined as a metastatic carcinoma that occurs mainly in the axillary lymph nodes, derived from a primary malignant breast tumor undetectable by clinical and radiological analyses. OBC is a rare disease accounting for 0.3%–1.0% of all breast cancers, which occurs more commonly at the age of around 55 years. The OBC represents a rare event (especially with the manifestation of systemic metastases) and a major diagnostic challenge. Thus, the aim of this study was to report a case of OBC with the primary manifestation of cutaneous metastases and the subs
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Inufusa, H., N. Sagara, K. Nakano, and M. Yasutomi. "CHARACTERISATION OF PLATELET AGGREGATING MATERIAL EXTRACTED FROM HUMAN LUNG ADENOCARCINOMA CELL LINE WHICH METASTASIZED IN NUDE MICE S,C, IMPLANTATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643199.

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It has been reported in animal experimental system that Platelet Aggregating Material (PAM) of cancer cell play important role in cancer metastasis. Many human cancer cell lines has been also studied the platelet aggregation activity of PAM. But correlation between the platelet aggregation activity and metastatic potential of human cancer cells were usually unkncwn. We established a human lung adenocarcinoma cell line KUM-LK-2 which produce spontaneous lung metastasis when cells implanted into subcutaneous of nude mice. PAM was extracted from KUM-LK-2 cells following the method of D.Mbhanty an
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Sayyad, Megan, Madhavi Puchalapalli, Chris Canal, Theresa Swift-Scanlan, Andrew Ottens, and Jennifer E. Koblinski. "Abstract 1988: Metastatic v non-metastatic cancer exosomes in brain metastasis." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1988.

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Sayyad, Megan, Madhavi Puchalapalli, Chris Canal, Theresa Swift-Scanlan, Andrew Ottens, and Jennifer E. Koblinski. "Abstract 1988: Metastatic v non-metastatic cancer exosomes in brain metastasis." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-1988.

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Hoskins, Meghan H., Shile Liang, Robert F. Kunz, and Cheng Dong. "Cellular Mechanics and Biology of Tumor Cell-Leukocyte Interactions in the Near Wall Region Under Shear Flow Conditions." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176376.

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Malignant melanoma has a high propensity for metastatic spread, making it the most deadly form of skin cancer. Currently, no effective treatment exists to prevent melanoma metastasis or to inhibit metastatic tumors growing in distant organs [1,2].
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Shareef, Sarah J., Mihai Nita-Lazar, and Maria A. Kukuruzinska. "E-cadherin N-glycosylation Modulates the Strength of Adherens Junctions." In ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13013.

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Metastasis, the spread of cancer cells throughout the body, claims 90% of solid tumor deaths. During metastasis, cancer cells undergo discohesion. An understanding of how to control and strengthen cell adhesion through junction formation could lead to methods for decreasing metastatic tendencies.
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McClain, Jacob, Sara L. Tuell, and Susan N. Thomas. "Tumors Change the Elastic and Viscoelastic Properties of Draining Lymph Node Tissues." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14419.

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Changes in tissue mechanical properties are often the first indication of malignant disease, with the detection of a stiff lump by a patient. These changes include growth-induced solid stresses, increased matrix stiffness, high fluid pressure, and increased interstitial flow, which in turn enhance fluid flux away from the tumor to downstream lymph nodes (LNs). But in addition to changing the way a tumor feels to a patient, altered tumor tissue mechanics promote cancer cell invasion into lymphatic vessels, allowing their metastatic dissemination to draining LNs. LN swelling and stiffening is an
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Barros, Jordana Joab Alencar, Alexandre Bravin Moreira, Paulo Roberto Moreira Sousa, Tatiane Oliveira Borges, Luciana Carla Belém dos Santos, and Catharina Maria Facciolli Blum. "METASTATIC BREAST CANCER TO THE UTERINE CERVIX: A CASE REPORT." In Brazilian Breast Cancer Symposium 2022. Mastology, 2022. http://dx.doi.org/10.29289/259453942022v32s2084.

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A 49-year-old woman was diagnosed with invasive ductal carcinoma in the left breast with positive estrogen and progesterone receptors (ER, PR) and negative HER2, and Ki-67 10%. Computed tomography of the abdomen, chest, and bone scintigraphy did not show metastasis. Treatment was established with neoadjuvant chemotherapy, followed by sectorectomy, sentinel lymph node biopsy, and oncoplasty. Freezing was negative, but in the anatomopathological result, metastasis in the axillary lymph node was observed. Then, axillary lymphadenectomy was performed, and she underwent radiotherapy and hormone the
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Cruz, Marcelo Ribeiro da Luz, Valeria Fernandes Roppa Cruz, Alfredo Almeida Cunha, and Renato Souza Bravo. "CAN AXILLARY ULTRASONOGRAPHY WITH CORE NEEDLE BIOPSY BE A USEFUL TOOL IN THE APPROACH OF BREAST CANCER PATIENTS?" In Scientifc papers of XXIII Brazilian Breast Congress - 2021. Mastology, 2021. http://dx.doi.org/10.29289/259453942021v31s1004.

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Introduction: Metastatic involvement of axillary lymph nodes in patients with breast cancer is an important prognostic factor and it also has therapeutic implications. Nevertheless, the use of ultrasound for evaluating axillary lymph node status has been questioned. Objective: Evaluate the performance of ultrasound with core needle biopsy in the diagnosis of axillary metastasis. Patients and Method: A diagnostic validation study was performed to compare axillary ultrasound with core biopsy versus the surgical procedure. The scenario was a quaternary hospital in Rio de Janeiro where breast canc
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Reports on the topic "Metastatic cancer"

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Tinker, Anna V., Neesha C. Dhani, Prafull Ghatage, et al. Immune Checkpoint Inhibitors in Pretreated Metastatic Endometrial Cancer. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.1.0038.

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Review question / Objective: Efficacy and safety of immune checkpoint inhibitors alone or in combination with tyrosine kinase inhibitors in patients with pretreated advanced, persistent, or recurrent metastatic endometrial cancer. Condition being studied: Advanced, persistent, or recurrent metastatic endometrial cancer. Study designs to be included: Non-randomized studies of monotherapy in populations selected for relevant biomarkers such as MMR, microsatellite stability, and PD-L1 expression status and randomized trials of ICI combinations in unselected patients.
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Khan, Shafiq. Biomarkers for Metastatic Prostate Cancer. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada427158.

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Williams, Myron N. Biomarkers for Metastatic Prostate Cancer. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada416624.

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Deisseroth, Albert B. Vectors for Treatment of Metastatic Breast Cancer. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada448903.

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Deisseroth, Albert B. Vectors for Treatment of Metastatic Breast Cancer. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada429189.

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Takeshita, Kenichi. Retinoic Acid Receptors in Metastatic Prostate Cancer. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada413284.

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Marchetti, Dario. Heparanase Mechanisms in Brain-metastatic Breast Cancer. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada585985.

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Pang, Shen. Gene Delivery for Metastatic Prostate Cancer Cells. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada390852.

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Takeshita, Kenichi. Retinoic Acid Receptors in Metastatic Prostate Cancer. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada392639.

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Marchetti, Dario. Heparanase Mechanisms in Brain-Metastatic Breast Cancer. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada596541.

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