Relevant bibliographies by topics / Methylphenidate interaction

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Methylphenidate interaction'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Methylphenidate interaction"

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&NA;. "Methylphenidate interaction." Reactions Weekly &NA;, no. 746 (1999): 9. http://dx.doi.org/10.2165/00128415-199907460-00031.

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&NA;. "Methylphenidate interaction." Reactions Weekly &NA;, no. 1173 (2007): 18. http://dx.doi.org/10.2165/00128415-200711730-00054.

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&NA;. "Methylphenidate interaction." Reactions Weekly &NA;, no. 1181 (2007): 25. http://dx.doi.org/10.2165/00128415-200711810-00073.

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&NA;. "Methylphenidate interaction." Reactions Weekly &NA;, no. 1136 (2007): 20. http://dx.doi.org/10.2165/00128415-200711360-00062.

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&NA;. "Methylphenidate interaction." Reactions Weekly &NA;, no. 1152 (2007): 17. http://dx.doi.org/10.2165/00128415-200711520-00055.

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&NA;. "Methylphenidate interaction." Reactions Weekly &NA;, no. 655 (1997): 10. http://dx.doi.org/10.2165/00128415-199706550-00032.

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&NA;. "Methylphenidate interaction." Reactions Weekly &NA;, no. 803 (2000): 10. http://dx.doi.org/10.2165/00128415-200008030-00025.

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&NA;. "Methylphenidate/sertraline interaction." Reactions Weekly &NA;, no. 1200 (2008): 24. http://dx.doi.org/10.2165/00128415-200812000-00067.

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&NA;. "Methylphenidate/pseudoephedrine interaction." Reactions Weekly &NA;, no. 1303 (2010): 25. http://dx.doi.org/10.2165/00128415-201013030-00077.

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&NA;. "Disulfiram/methylphenidate interaction." Reactions Weekly &NA;, no. 1424 (2012): 24. http://dx.doi.org/10.2165/00128415-201214240-00079.

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Dissertations / Theses on the topic "Methylphenidate interaction"

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Freeman, Elizabeth D. "An Analysis of the Interaction of Methylphenidate and Nicotine in Adolescent Rats: Effects on BDNF." Digital Commons @ East Tennessee State University, 2015. https://dc.etsu.edu/etd/2572.

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This investigation was an analysis of the interaction of adolescent exposure to methylphenidate (MPH; trade name: Ritalin) on nicotine sensitization and conditioned place preference (CPP) in a rodent model and underlying mechanisms of this effect. Animals were treated IP with 1 mg/kg MPH or saline using a ―school day‖ regimen of five days on, two days off, from postnatal day (P) 28-50. During the final two weeks of MPH treatment, animals were either behaviorally sensitized to nicotine (0.5 mg/kg free base) or saline for 10 days, or conditioned to nicotine or saline using the CPP behavioral par
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Books on the topic "Methylphenidate interaction"

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Chrzanowski, Daniel T., Elisabeth B. Guthrie, Matthew B. Perkins, and Moira A. Rynn. Child and Adolescent Psychiatry. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199326075.003.0015.

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Common disorders of children and adolescents include neurodevelopmental disorders (e.g., intellectual disability, autistic spectrum disorder, and learning disorders), internalizing disorders (e.g., mood and anxiety disorders), and externalizing disorders (e.g., oppositional defiant disorder and conduct disorder). The assessment of a child or adolescent patient always includes multiple informants, the context in which the child’s difficulties occur, and a functional behavioral assessment. Patients with autism spectrum disorder tend to have persistent deficits in social communication and social
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Book chapters on the topic "Methylphenidate interaction"

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"Methylphenidate." In Meyler's Side Effects of Drugs: The International Encyclopedia of Adverse Drug Reactions and Interactions. Elsevier, 2006. http://dx.doi.org/10.1016/b0-44-451005-2/00021-8.

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Grace, Anthony A. "Psychostimulant Actions on Dopamine and Limbic System Function: Relevance to the Pathophysiology and Treatment of ADH D." In Stimulant Drugs and ADHD. Oxford University PressNew York, NY, 2000. http://dx.doi.org/10.1093/oso/9780195133714.003.0006.

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Abstract Understanding the mechanism of action of psychostimulants must begin with a comprehension of their neurochemical action at the site of the nerve terminal in the central nervous system. It is the catecholamine terminal at which drugs such as methylphenidate have their primary site of action. Moreover, the action of psychostimulants on the dopamine (DA) neurotransmitter system is believed to mediate the rewarding properties of these drugs (Colle & Wise, 1988; Gratton et al., 1988; Olds, 1990). However, to understand the effects of such compounds on nor mal behavior and their therape
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Ansari, Arash, and David N. Osser. "Stimulants and Other ADHD Medicines." In Psychopharmacology. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780197537046.003.0006.

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The chapter on adult attention-deficit/hyperactivity disorder (ADHD) medicines discusses and reviews the use of psychostimulants (such as methylphenidate and amphetamines), and nonstimulants (such as atomoxetine, guanfacine, and clonidine). It reviews their mechanisms of action, clinical characteristics, potential medication interactions, and adverse effects. It further reviews stimulants’ risk of misuse and dependence. The chapter also briefly discusses complementary and alternative pharmacotherapies. It includes an in-depth review of the clinical use of these medications for ADHD (particular
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Conference papers on the topic "Methylphenidate interaction"

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Abrimian, Anna, Eleni Papadopoulos, Christopher P. Knapp, J. Loweth, Barry Waterhouse, and Rachel Navarra. "Interactions Between Repetitive Mild Traumatic Brain Injury and Methylphenidate Administration on Catecholamine Transporter Protein Levels Within the Rodent Prefrontal Cortex." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.10_2024.

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It is theorized that low concentrations of dopamine (DA) and norepinephrine (NE) within in the prefrontal cortex (PFC) following traumatic brain injury (TBI) leads to increased risky behavior. Our lab has shown that repeated mild TBI (rmTBI) sex-differentially increases risky behavior in a rodent model. Methylphenidate (MPH) is a psychostimulant drug used to treat symptoms of Attention-Deficit Hyperactivity Disorder (ADHD), also driven by a hypo-catecholaminergic PFC. MPH elevates catecholamine levels by blocking DA and NE transporters, DAT and NET. While the potential of psychostimulants to t
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Reports on the topic "Methylphenidate interaction"

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Babkoff, H., T. L. Kelly, L. T. Mattseon, S. Gomez, and A. Lopez. Pemoline and Methylphenidate: Interaction With Mood, Sleepiness, and Cognitive Performance During 64 Hours of Sleep Deprivation. Defense Technical Information Center, 1992. http://dx.doi.org/10.21236/ada256601.

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You might also be interested in the extended bibliographies on the topic 'Methylphenidate interaction' for particular source types:
Journal articles
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