Relevant bibliographies by topics / MGluR-mediated priming of LTP

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Academic literature on the topic 'MGluR-mediated priming of LTP'

Author: Grafiati
Published: 4 June 2021
Last updated: 15 February 2022

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Journal articles on the topic "MGluR-mediated priming of LTP"

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Auerbach, Benjamin D., and Mark F. Bear. "Loss of the Fragile X Mental Retardation Protein Decouples Metabotropic Glutamate Receptor Dependent Priming of Long-Term Potentiation From Protein Synthesis." Journal of Neurophysiology 104, no. 2 (2010): 1047–51. http://dx.doi.org/10.1152/jn.00449.2010.

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Fragile X Syndrome (FXS), the most common inherited form of intellectual disability, is caused by loss of the fragile X mental retardation protein (FMRP). FMRP is a negative regulator of local mRNA translation downstream of group 1 metabotropic glutamate receptor (Gp1 mGluR) activation. In the absence of FMRP there is excessive mGluR-dependent protein synthesis, resulting in exaggerated mGluR-dependent long-term synaptic depression (LTD) in area CA1 of the hippocampus. Understanding disease pathophysiology is critical for development of therapies for FXS and the question arises of whether it i
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Cohen, A. S., and W. C. Abraham. "Facilitation of long-term potentiation by prior activation of metabotropic glutamate receptors." Journal of Neurophysiology 76, no. 2 (1996): 953–62. http://dx.doi.org/10.1152/jn.1996.76.2.953.

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1. The influence of prior metabotropic glutamate receptor (mGluR) activation on subsequent long-term potentiation (LTP) induction was investigated with the use of the mGluR agonist 1-amino-cyclopentane-1S,3R-dicarboxylic acid (ACPD, 20 microM). Field potential recordings were made in the stratum radiatum of CA1 slices taken from young adult male rats and from which CA3 was routinely dissected. Theta burst stimulation (TBS) just above threshold was used to induce LTP. 2. A 10-min bath application of ACPD begun 30 min before the TBS facilitated the induction of LTP in a dose-dependent manner and
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Cohen, Akiva S., Christine M. Coussens, Clarke R. Raymond, and Wickliffe C. Abraham. "Long-Lasting Increase in Cellular Excitability Associated With the Priming of LTP Induction in Rat Hippocampus." Journal of Neurophysiology 82, no. 6 (1999): 3139–48. http://dx.doi.org/10.1152/jn.1999.82.6.3139.

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The mechanisms underlying the facilitation (priming) of long-term potentiation (LTP) by prior activation of metabotropic glutamate receptors (mGluRs) were investigated in area CA1 of rat hippocampal slices. In particular, we focused on whether a long-lasting increase in postsynaptic excitability could account for the facilitated LTP. Administration of the mGluR agonist 1S,3R-aminocyclopentanedicarboxylic acid (ACPD) produced rapid decreases in the amplitude of both the slow spike afterhyperpolarization (AHPslow) and spike frequency adaptation recorded intracellularly from CA1 pyramidal cells.
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Hu, Bin, Sergei Karnup, Lei Zhou, and Armin Stelzer. "Reversal of Hippocampal LTP by Spontaneous Seizure-Like Activity: Role of Group I mGluR and Cell Depolarization." Journal of Neurophysiology 93, no. 1 (2005): 316–36. http://dx.doi.org/10.1152/jn.00172.2004.

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Memory impairment is a common consequence of epileptic seizures. The hippocampal formation is particularly prone to seizure-induced amnesia due to its prominent role in mnemonic processes. We used the isolated CA1 slice preparation to examine effects of seizure-like activity on hippocampal plasticity, long-term potentiation (LTP), and long-term depression (LTD). Repeated spontaneous ictal events, generated in the presence of antagonists of GABAA receptor function, led to a stepwise erasure of LTP (termed spontaneous depotentiation, SDP). SDP could be initiated at various stages of LTP consolid
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IV, Paul M. Lea, Barbara Wroblewska, John M. Sarvey та Joseph H. Neale. "β-NAAG Rescues LTP From Blockade by NAAG in Rat Dentate Gyrus via the Type 3 Metabotropic Glutamate Receptor". Journal of Neurophysiology 85, № 3 (2001): 1097–106. http://dx.doi.org/10.1152/jn.2001.85.3.1097.

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N-Acetylaspartylglutamate (NAAG) is an agonist at the type 3 metabotropic glutamate receptor (mGluR3), which is coupled to a Gi/o protein. When activated, the mGluR3 receptor inhibits adenylyl cyclase and reduces the cAMP-mediated second-messenger cascade. Long-term potentiation (LTP) in the medial perforant path (MPP) of the hippocampal dentate gyrus requires increases in cAMP. The presence of mGluR3 receptors and NAAG in neurons of the dentate gyrus suggests that this peptide transmitter may inhibit LTP in the dentate gyrus. High-frequency stimulation (100 Hz; 2 s) of the MPP resulted in LTP
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Neugebauer, Volker, N. Bradley Keele, and Patricia Shinnick-Gallagher. "Loss of Long-Lasting Potentiation Mediated by Group III mGluRs in Amygdala Neurons in Kindling-Induced Epileptogenesis." Journal of Neurophysiology 78, no. 6 (1997): 3475–78. http://dx.doi.org/10.1152/jn.1997.78.6.3475.

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Neugebauer, Volker, N. Bradley Keele, and Patricia Shinnick-Gallagher. Loss of long-lasting potentiation mediated by group III mGluRs in amygdala neurons in kindling-induced epileptogenesis. J. Neurophysiol. 78: 3475–3478, 1997. Long-lasting modifications of synaptic transmission can be induced in the amygdala by electrical stimulation as done in the long-term potentiation (LTP) model of learning and memory and the kindling model of epilepsy. The present study reports for the first time a long-lasting potentiation (LLP) of synaptic transmission that is induced pharmacologically by the activati
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Harrison, John M., Richard G. Allen, Michael J. Pellegrino, John T. Williams, and Olivier J. Manzoni. "Chronic Morphine Treatment Alters Endogenous Opioid Control of Hippocampal Mossy Fiber Synaptic Transmission." Journal of Neurophysiology 87, no. 5 (2002): 2464–70. http://dx.doi.org/10.1152/jn.2002.87.5.2464.

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Synaptic adaptations are thought to be an important component of the consequences of drug abuse. One such adaptation is an up-regulation of adenylyl cyclase that has been shown to increase transmitter release at several inhibitory synapses. In this study the effects of chronic morphine treatment were studied on mossy fiber synapses in the guinea pig hippocampus using extracellular field potential recordings. This opioid-sensitive synapse was chosen because of the known role of the adenylyl cyclase cascade in the regulation of glutamate release. Long-term potentiation (LTP) at the mossy fiber s
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Vickery, R. M., Shanida H. Morris, and Lynn J. Bindman. "Metabotropic Glutamate Receptors Are Involved in Long-Term Potentiation in Isolated Slices of Rat Medial Frontal Cortex." Journal of Neurophysiology 78, no. 6 (1997): 3039–46. http://dx.doi.org/10.1152/jn.1997.78.6.3039.

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Vickery, R. M., Shanida H. Morris, and Lynn J. Bindman. Metabotropic glutamate receptors are involved in long-term potentiation in isolated slices of rat medial frontal cortex. J. Neurophysiol. 78: 3039–3046, 1997. The prelimbic region of medial frontal cortex in the rat receives a direct input from the hippocampus and this functional connection is essential for aspects of spatial memory. Activity-dependent changes in the effectiveness of synaptic transmission in the medial frontal cortex, namely long-term potentiation (LTP) and long-term depression (LTD) can persist for tens of minutes or hou
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Chinestra, P., L. Aniksztejn, D. Diabira, and Y. Ben-Ari. "(RS)-alpha-methyl-4-carboxyphenylglycine neither prevents induction of LTP nor antagonizes metabotropic glutamate receptors in CA1 hippocampal neurons." Journal of Neurophysiology 70, no. 6 (1993): 2684–89. http://dx.doi.org/10.1152/jn.1993.70.6.2684.

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1. The effects of the putative antagonist of metabotropic glutamate receptors (mGluR), (RS)-alpha-methyl-4-carboxyphenylglycine (MCPG), were investigated in CA1 hippocampal neurons using intracellular and extracellular recordings. 2. MCPG (0.5 mM) did not antagonize the characteristic block of the slow afterhyperpolarization and spike accomodation produced by the selective mGluR agonist, 1S,3R-1-aminocyclopentane-1,3-dicarboxylic acid (1S,3R-ACPD) (30 microM). 3. MCPG (0.5 mM) did not prevent the inward current produced by 1S,3R-ACPD (30 microM) [240 +/- 14 and 255 +/- 21 pA (mean +/- SD) in t
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Cumiskey, Derval, Mark Pickering, and John J. O’Connor. "Interleukin-18 mediated inhibition of LTP in the rat dentate gyrus is attenuated in the presence of mGluR antagonists." Neuroscience Letters 412, no. 3 (2007): 206–10. http://dx.doi.org/10.1016/j.neulet.2006.11.007.

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Dissertations / Theses on the topic "MGluR-mediated priming of LTP"

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Privitera, Lucia, Ellen L. Hogg, M. Gaestel, M. J. Wall, and Sonia A. L. Corrêa. "The MK2 cascade regulates mGluR-dependent synaptic plasticity and reversal learning." 2019. http://hdl.handle.net/10454/17110.

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Yes<br>The ability to either erase or update the memories of a previously learned spatial task is an essential process that is required to modify behaviour in a changing environment. Current evidence suggests that the neural representation of such cognitive flexibility involves the balancing of synaptic potentiation (acquisition of memories) with synaptic depression (modulation and updating previously acquired memories). Here we demonstrate that the p38 MAPK/MAPK-activated protein kinase 2 (MK2) cascade is required to maintain the precise tuning of long-term potentiation and long-term depressi
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