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Journal articles on the topic 'Mice Mice Mice Mice'

Author: Grafiati

Published: 4 June 2021

Last updated: 16 February 2022

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1

Park, Woo Teak, and Byeong Cheol Lee. "The conceptual approach of MICE in daily lives: A case of MICE village." Journal & Article Management System 17, no. 1 (2021): 225–45. http://dx.doi.org/10.31927/asec.17.1.12.

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2

Seo, Min-Ryeong, Mi-Seong Kim, Yoo-Shik Yoon, and Na-Young Baek. "Market Segmentation of MICE Organizer by MICE City Marketing Awareness." Korea Trade Exhibition Review 15, no. 3 (2020): 95–113. http://dx.doi.org/10.16938/ijtfs.2020.15.3.095.

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3

Šlosárková, S., V. Híbalová, I. Literák, et al. "Experimental toxoplasmosis in hypoiodemic mice." Veterinární Medicína 47, No. 2 - 3 (2012): 67–74. http://dx.doi.org/10.17221/5806-vetmed.

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The hypothesis, that hypoiodemia of goats causes such a compromise of the immune system, which during subsequent Toxoplasma gondii infections results in clinically more pronounced signs of toxoplasmosis, was verifying in laboratory mouse. Hypoiodemic mice (fed by wheat and supplied by water), normoiodemic mice (fed by wheat and supplied by water containing 1.25 mg KI/l) and the majority of standard mice (fed by commercial grain mixture containing 0.83 mg I/kg) were experimentally infected with T. gondii oocysts or tachyzoites. The susceptibility to acute T. gondii infection was evaluated according to mortality rate. As a criterion of cell-mediated immune function has been chosen the spleen-lymphocyte transformation test (LTT). We observed no difference in LTT between hypoiodemic and normoiodemic mice infected with T. gondii oocysts or tachyzoites and no difference in mortality of both infected groups. Four days after the exposure to 100 tachyzoites of T. gondii (K24 strain), all experimentally infected groups of mice showed statistically significant decrease (P = 0.004) in spleen cells responsiveness to stimulation by all mitogens used &ndash; as compared to non-infected standard mice group. Reduced responsiveness of cells was probably caused by T. gondii infection itself &ndash; the relation to iodine deficiency has not been found.

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4

Jang, Mi-hwa. "How is Daegu perceived as a MICE destination? A Study on the Placeness of MICE Destination on Social Media." Korea Trade Exhibition Review 16, no. 1 (2021): 21–39. http://dx.doi.org/10.16938/ijtfs.2021.16.1.021.

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5

Mghamis, Munqith Mazin. "Effect of Prenatal Ketamine Exposure on GFAP Marker Expression in Mice Prefrontal Cortex Mice Prefrontal Cortex." International Journal of Psychosocial Rehabilitation 24, no. 4 (2020): 3936–44. http://dx.doi.org/10.37200/ijpr/v24i4/pr201507.

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6

Ryu, Jae Hee, and Byeong Cheol Lee. "Conceptualizing MICE Supporters’ Sharing Economy Activity." Journal & Article Management System 14, no. 1 (2018): 173–95. http://dx.doi.org/10.31927/asec.14.1.11.

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7

Choi, In-Ho. "Japanese MICE Promotion Utilizing Unique Venues." Korea Trade Exhibition Review 11, no. 3 (2016): 217–35. http://dx.doi.org/10.16938/ijtfs.2016.11.3.217.

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8

Safaeva, Sayyora, and Diyora Adilova. "Mice Tourism: Opportunities, Priorities, Problems, Prospects." American Journal of Applied Sciences 02, no. 11 (2020): 116–21. http://dx.doi.org/10.37547/tajas/volume02issue11-21.

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The MICE sector is one of the most significantly developing tourism industries in recent years. The growth of business tourism strongly affects not only the industry as a whole, but also the economy of the host country. However, with the development and greater popularization of MICE tourism, various difficulties arise. The research work will reveal the main opportunities, prospects and priorities for the development of the MICE tourism industry, as well as analyze the problematic aspects that MICE agencies face when organizing their activities.

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9

Кузнецова, Ольга, Olga Kuznetsova, Людмила Сильчева, et al. "Aspects on MICE-tourism and its management." Services in Russia and abroad 8, no. 2 (2014): 40–52. http://dx.doi.org/10.12737/3585.

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The article highlights the issues of MICE-related business travel, such as business tourism, conference-, exhibition- and travel-study tourism. The authors consider the historical preconditions for business travel birth and development, as well as the peculiarities of incentive tourism and the types and purposes of incentive programmes. The article provides statistics concerning the current state of the MICE-industry, and a comprehensive aspect-by-aspect study of a sample offsite seminar management in the framework of business tourism.

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10

Kang, Hae-Sang, and Kang-Young Song. "Analysis on the Satisfaction by MICE Participants in Busan Metropolitan." Journal of the Korea Contents Association 10, no. 11 (2010): 414–23. http://dx.doi.org/10.5392/jkca.2010.10.11.414.

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11

Wang, Xianmei, Di Tang, Fei Wang, et al. "Microneme Protein 6 Is Involved in Invasion and Egress by Neospora caninum." Pathogens 10, no. 2 (2021): 201. http://dx.doi.org/10.3390/pathogens10020201.

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Background: Neospora caninum, is the etiological agent of neosporosis, an infection that causes abortions in cattle and nervous system dysfunction in dogs. Invasion and egress are the key steps of the pathogenesis of N. caninum infection. Microneme proteins (MICs) play important roles in the recognition, adhesion, and invasion of host cells in other apicomplexan parasites. However, some MICs and their functions in N. caninum infection have rarely been reported. Methods: The homologous recombination strategy was used to investigate the function of MIC6 in N. caninum infection. Results: ΔNcMIC6 showed a smaller plaque size and weakened capacities of invasion and egress than Nc1. Transcription levels of the egress-related genes CDPK1, PLP1, and AMA1 of ΔNcMIC6 were downregulated. Due to the lack of NcMIC6, virulence of the pathogen in the infected mouse was weakened. The subcellular localization of NcMIC1 and NcMIC4 in ΔNcMIC6, however, did not change. Nevertheless, the transcription levels of MIC1 and MIC4 in ΔNcMIC6 were downregulated, and the expression and secretion of MIC1 and MIC4 in ΔNcMIC6 were reduced compared with that in Nc1. Furthermore, the absence of NcMIC6 weakened the virulence in mice and lower parasite load detected in mice brains. Conclusions: NcMIC6 is involved in host cell invasion and egress in N. caninum and may work synergistically with other MICs to regulate the virulence of the pathogen. These data lay a foundation for further research into the function and application of NcMIC6.

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12

Shakya, Pratibha, Neelesh Kumar Shakya, and C. Mohanty. "VALPROATE INDUCED NEPHROTOXICITY ON FETAL MICE KIDNEY." International Journal of Anatomy and Research 8, no. 1.3 (2020): 7383–85. http://dx.doi.org/10.16965/ijar.2020.108.

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13

Kim, Chul-Won, and Ji-Yeon Park. "A Study on Exploratory Paradigm for the MICE Serviceology." Korea Trade Exhibition Review 10, no. 1 (2015): 1–20. http://dx.doi.org/10.16938/ijtfs.2015.10.1.001.

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14

Ha, Myung hee, and Chul Won Kim. "A Study on the Development of the Network Governance Model - Focusing on Seoul MICE Industry -." Journal & Article Management System 14, no. 1 (2018): 23–45. http://dx.doi.org/10.31927/asec.14.4.2.

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15

Kim, Seokyung, Yooshik Yoon, Miseong Kim, and Yoonjung Kim. "The Study of the MICE Brand Equity and Brand Loyalty by Creating Shared Value in MICE Industry." Journal of Tourism Management Research 22, no. 5 (2018): 69–88. http://dx.doi.org/10.18604/tmro.2018.22.5.4.

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16

Trisetiyono, Yuli, Widjiati Widjiati, Syarief Thaufik Hidayat, and Noor Pramono. "Antioxidant Herbs Supplementation Inhibits Endometriosis Extension in Mice." Journal of Biomedicine and Translational Research 5, no. 2 (2019): 53–61. http://dx.doi.org/10.14710/jbtr.v5i2.4716.

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Background: Increased oxidative stress causes inflammation and increases angiogenesis. It presumed to promote the proliferation of endometriosis tissue. Kebar grass (Biophytum petersianum) and other herbs such as green tea and Cucumis melo, which contain high antioxidants, are expected to decrease oxidative stress, inflammation, angiogenesis, and reduced endometriosis implants.Objective: To investigate the effects of Kebar grass, green tea, and Cucumis melo to malondialdehyde serum, tumor necrosis factor alpha, and vascular endothelial growth factor expression, and the area of the endometriotic implants.Methods: Twenty-eight mice were divided into four groups, i.e., the first group of endometriosis mice was given Kebar grass extract; the second group was assigned green tea extract, the third group was given the combination of Cucumis melo extract–gliadin, and the last containing the untreated endometriosis mice as the control. Each treatment was given for 14 days. The data of MDA serum level, the area of the endometriotic implants, TNF-α, and VEGF expression were collected and analyzed.Results: The MDA serum levels of the groups treated with Kebar grass extract, green tea extract, and Cucumis melo extract – gliadin were significantly lower (p=0.001) than the control group. TNF-α expression of the groups provided with each treatment also lower than the control groups (p=0.002). However, only the administration of the Cucumis melo extract–gliadin resulted in lower VEGF expression compare with the control (p=0.017). Finally, the area of the endometriotic implants of the mice models administered with each treatment was smaller than the control group (p=0.003).Conclusion: Kebar grass as well as green tea and Cucumis melo–gliadin inhibits endometriotic implants extension by decreasing MDA serum and TNF-α expression.

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17

Mustafa, Snoor Jalal, and Kameel Mate Naoum. "EFFECT OF ACYCLOVIR ON EMBRYO IMPLANTATION IN MICE." Journal of Sulaimani Medical College 3, no. 2 (2013): 103–7. http://dx.doi.org/10.17656/jsmc.10038.

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18

Vohra, Prerna, and Navneet Kumar Gupta. "Carbontetrachloride induced toxicity in liver of albino mice." Indian Journal of Applied Research 3, no. 9 (2011): 273–75. http://dx.doi.org/10.15373/2249555x/sept2013/80.

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19

Arévalo Rodríguez, Montserrat, Montserrat Civit Torruella, and Maria Antònia Martí. "MICE." International Journal of Corpus Linguistics 9, no. 1 (2004): 53–68. http://dx.doi.org/10.1075/ijcl.9.1.03are.

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In the field of corpus linguistics, Named Entity treatment includes the recognition and classification of different types of discursive elements like proper names, date, time, etc. These discursive elements play an important role in different Natural Language Processing applications and techniques such as Information Retrieval, Information Extraction, translations memories, document routers, etc.

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20

Kim, Jun-Hyun, Bong-Seok Kim, and Ji-Yeon Lee. "A Study on Determinants of Burnout of MICE Industry Workers." Korea Trade Exhibition Review 16, no. 1 (2021): 131–56. http://dx.doi.org/10.16938/ijtfs.2021.16.1.131.

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21

Park, Hyo-Yeun. "The Study on Improvements of Statistics in the MICE Industry: Focused on『MICE Industry Statistics Survey』." Journal of MICE & Tourism Research 20, no. 3 (2020): 253–68. http://dx.doi.org/10.35176/jmtr.20.3.13.

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22

Heller, Stefan, and A. J. Hudspeth. "Two deaf mice, two deaf mice…" Nature Medicine 4, no. 5 (1998): 560–61. http://dx.doi.org/10.1038/nm0598-560.

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23

Bléry, Mathieu, Manel Mrabet-Kraiem, Ariane Morel, et al. "Targeting MICA/B with cytotoxic therapeutic antibodies leads to tumor control." Open Research Europe 1 (September 13, 2021): 107. http://dx.doi.org/10.12688/openreseurope.13314.1.

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Background: MICA and MICB are tightly regulated stress-induced proteins that trigger the immune system by binding to the activating receptor NKG2D on cytotoxic lymphocytes. MICA and MICB are highly polymorphic molecules with prevalent expression on several types of solid tumors and limited expression in normal/healthy tissues, making them attractive targets for therapeutic intervention. Methods: We have generated a series of anti-MICA and MICB cross-reactive antibodies with the unique feature of binding to the most prevalent isoforms of both these molecules. Results: The anti-MICA and MICB antibody MICAB1, a human IgG1 Fc-engineered monoclonal antibody (mAb), displayed potent antibody-dependent cellular cytotoxicity (ADCC) and antibody-dependent cellular phagocytosis (ADCP) of MICA/B-expressing tumor cells in vitro. However, it showed insufficient efficiency against solid tumors in vivo, which prompted the development of antibody-drug conjugates (ADC). Indeed, optimal tumor control was achieved with MICAB1-ADC format in several solid tumor models, including patient-derived xenografts (PDX) and carcinogen-induced tumors in immunocompetent MICAgen transgenic mice. Conclusions: These data indicate that MICA and MICB are promising targets for cytotoxic immunotherapy.

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24

Masopust, David, Christine P. Sivula, and Stephen C. Jameson. "Of Mice, Dirty Mice, and Men: Using Mice To Understand Human Immunology." Journal of Immunology 199, no. 2 (2017): 383–88. http://dx.doi.org/10.4049/jimmunol.1700453.

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25

Parashar, Vivek, Buddhadeb Ghosh, Natwar lal Gaur, Satya Narayan Shamal, S. K. Pandey, and Girdhari Lal Shah. "TERATOLOGICAL EFFECTS OF CARBOPLATIN: A MORPHOLOGICAL STUDY IN MICE." International Journal of Anatomy and Research 4, no. 2.2 (2016): 2358–64. http://dx.doi.org/10.16965/ijar.2016.218.

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26

Cherif H.S, Cherif H. S., Saidi F. Saidi F, and Guedioura A. Guedioura A. "Toxicological evaluation of Aristolochia longa L. extract in mice." Indian Journal of Applied Research 4, no. 5 (2011): 26–30. http://dx.doi.org/10.15373/2249555x/may2014/8.

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27

KUMAR, DEEPAK, and SURESH KUMAR. "Neuropharmacological Activities of Abies pindrow Aerial Parts in Mice." JOURNAL OF PHARMACEUTICAL TECHNOLOGY, RESEARCH AND MANAGEMENT 3, no. 2 (2015): 141–51. http://dx.doi.org/10.15415/jptrm.2015.32011.

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28

DHINGRA, D., and S. PARSHAD. "Improvement of Learning and Memory of Mice by Plumbagin." Journal of Pharmaceutical Technology, Research and Management 4, no. 2 (2016): 147–59. http://dx.doi.org/10.15415/jptrm.2016.42010.

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29

Ha, Myungehee, and Chulwon Kim. "Directing the Revision of Local Governments’ MICE-related Ordinances." Korea Trade Exhibition Review 15, no. 4 (2020): 81–103. http://dx.doi.org/10.16938/ijtfs.2020.15.4.081.

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30

Seo, Kwang-Seoug, and Chul-Mo Koo. "MICE Venue Satisfaction and Its Influence on Expenditures in the Destination." Journal of Tourism Sciences 41, no. 2 (2017): 99–119. http://dx.doi.org/10.17086/jts.2017.41.2.99.119.

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31

Oh, Chang-Ho, Se-Jun Lee, Kyung-Hwa Nam, Eui-Joong Kim, and Yu-Jin Shin. "The Analysis of Economic impact effect of Busan MICE - Emphasis on incentive tours -." Journal of Tourism and Leisure Research 30, no. 6 (2018): 149–64. http://dx.doi.org/10.31336/jtlr.2018.06.30.6.149.

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32

Hester, James, Jeremi Mullins, Qila Sa, et al. "Toxoplasma gondii Antigens Recognized by IgG Antibodies Differ between Mice with and without Active Proliferation of Tachyzoites in the Brain during the Chronic Stage of Infection." Infection and Immunity 80, no. 10 (2012): 3611–20. http://dx.doi.org/10.1128/iai.00604-12.

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ABSTRACTWe examined whether tachyzoite proliferation in the brains of immunocompetent hosts during the chronic stage of infection withToxoplasma gondiiinduces production of IgG antibodies that recognize parasite antigens different from those recognized by the antibodies of infected hosts that do not have tachyzoite growth. For this purpose, two groups of CBA/J mice, which display continuous tachyzoite growth in their brains during the later stage of infection, were infected, and one group received treatment with sulfadiazine to prevent tachyzoite proliferation during the chronic stage of infection.T. gondiiantigens recognized by the IgG antibodies from these two groups of mice were compared using immunoblotting following separation of tachyzoite antigens by two-dimensional gel electrophoresis. Several antigens, including the microneme protein MIC2, the cyst matrix protein MAG1, and the dense granule proteins GRA4 and GRA7, were commonly recognized by IgG antibodies from both groups of mice. There were multiple antigens recognized mostly by IgG antibodies of only one group of mice, either with or without cerebral tachyzoite growth. The antigens recognized only by or mostly by the antibodies of mice with cerebral tachyzoite growth include MIC6, the rhoptry protein ROP1, GRA2, one heat shock protein HSP90, one (putative) HSP70, and the myosin heavy chain. These results indicate that levels of IgG antibody to only selectedT. gondiiantigens increase in association with cerebral tachyzoite proliferation (reactivation of infection) in immunocompetent hosts with chronic infection.

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33

Shen, Mengdie, Bibi Zhang, Mengyao Wang, Li’na Meng, and Bin Lv. "Mica Can Alleviate TNBS-Induced Colitis in Mice by Reducing Angiotensin II and IL-17A and Increasing Angiotensin-Converting Enzyme 2, Angiotensin 1-7, and IL-10." Mediators of Inflammation 2020 (October 10, 2020): 1–8. http://dx.doi.org/10.1155/2020/3070345.

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Aim. To explore the treatment effect of mica on 2,4,6-trinitrobenzenesulfonic acid solution- (TNBS-) induced colitis in mice. Materials and Methods. Thirty male BALB/C mice were randomly divided into the control group, the TNBS group, and the mica group. Control mice were treated with saline solution. Experimental colitis was induced by TNBS (250 mg/kg/d) in the TNBS group and the mica group. After modeling, the mica group was treated with mica (180 mg/kg/d) for 3 days, while the TNBS group continued the treatment with TNBS. All solutions were injected intrarectally. During treatment, body weight and mice activity were monitored daily. After treatment, the colon tissues of mice were collected; angiotensin II (Ang II), angiotensin-converting enzyme 2 (ACE2), angiotensin 1-7 (Ang (1-7)), IL-17A, and IL-10 expression was analyzed by ELISA and immunohistochemistry. Results. Food intake, activity, and body weight gradually decreased in the TNBS group compared to the control group and the mica group (all P<0.05). Also, black stool adhesion in the anus and thin and bloody stool were observed in the TNBS group, but not in the other two groups. Moreover, the expression of Ang II, ACE2, Ang (1-7), IL-17A, and IL-10 in the TNBS group increased compared to that in the control group. Compared to the TNBS group, ACE2, Ang (1-7), and IL-10 in the mica group increased, while Ang II and IL-17A decreased (all P<0.05). Conclusion. Mica can alleviate TNBS-induced colitis in mice by regulating the inflammation process; it reduces Ang II and IL-17A and increases ACE2, IL-10, and Ang (1-7).

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34

Yu, Sun-Nyoung, Jeong-Bin Ahn, Eun-Young Park, et al. "Effect of Trans-unsaturated Fatty Acid on Serum Lipid Levels in Mice." Journal of Life Science 22, no. 8 (2012): 1126–31. http://dx.doi.org/10.5352/jls.2012.22.8.1126.

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35

Kim, Yung-Moon, and Chul-Won Kim. "Developing Indicators to Forecast the Optimal Number of Employees for Hotel MICE." Journal of Tourism Sciences 43, no. 1 (2019): 13–35. http://dx.doi.org/10.17086/jts.2019.43.1.13.35.

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36

Hickman-Davis, Judy M., and Ian C. Davis. "Transgenic mice." Paediatric Respiratory Reviews 7, no. 1 (2006): 49–53. http://dx.doi.org/10.1016/j.prrv.2005.09.005.

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37

Cunniff, Peggy. "Multiplying mice." New Scientist 201, no. 2699 (2009): 24. http://dx.doi.org/10.1016/s0262-4079(09)60711-9.

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38

Travis, John. "Model Mice." Science News 164, no. 24 (2003): 371. http://dx.doi.org/10.2307/4019057.

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39

PALMITER, R., and R. BRINSTER. "Transgenic mice." Cell 41, no. 2 (1985): 343–45. http://dx.doi.org/10.1016/s0092-8674(85)80004-0.

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40

Hines, P. J. "Macho Mice." Science 326, no. 5951 (2009): 341. http://dx.doi.org/10.1126/science.326_341a.

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41

Babineau, Brooke A., Mu Yang, and Jacqueline N. Crawley. "Mainstreaming Mice." Neuropsychopharmacology 37, no. 1 (2011): 300–301. http://dx.doi.org/10.1038/npp.2011.168.

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42

López, Juan Carlos. "Dipsomaniac mice?" Nature Reviews Neuroscience 3, no. 6 (2002): 415. http://dx.doi.org/10.1038/nrn860.

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43

Corcoran, Elizabeth. "Making Mice." Scientific American 258, no. 6 (1988): 112–14. http://dx.doi.org/10.1038/scientificamerican0688-112.

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44

Stebbins, Michael. "Degenerate mice." Nature Medicine 8, no. 7 (2002): 670. http://dx.doi.org/10.1038/nm0702-670.

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45

LeBrasseur, Nicole. "Titinless mice." Journal of Cell Biology 173, no. 4 (2006): 455b. http://dx.doi.org/10.1083/jcb.1734iti3.

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46

Macchiarini, Francesca, Markus G. Manz, A. Karolina Palucka, and Leonard D. Shultz. "Humanized mice." Journal of Experimental Medicine 202, no. 10 (2005): 1307–11. http://dx.doi.org/10.1084/jem.20051547.

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Animal models have been instrumental in increasing the understanding of human physiology, particularly immunity. However, these animal models have been limited by practical considerations and genetic diversity. The creation of humanized mice that carry partial or complete human physiological systems may help overcome these obstacles. The National Institute of Allergy and Infectious Diseases convened a workshop on humanized mouse models for immunity in Bethesda, MD, on June 13–14, 2005, during which researchers discussed the benefits and limitations of existing animal models and offered insights into the development of future humanized mouse models.

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47

Sreenivasan, Aparna. "Mineralized mice." Journal of Cell Biology 165, no. 5 (2004): 602–3. http://dx.doi.org/10.1083/jcb1655iti2.

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Bashyam, Hema. "Jinxed mice." Journal of Experimental Medicine 204, no. 4 (2007): 697a. http://dx.doi.org/10.1084/jem.2044iti4.

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Marron, Karen. "Mega mice." Lab Animal 36, no. 9 (2007): 8. http://dx.doi.org/10.1038/laban1007-8b.

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Habeck, Martina. "Digital mice." Drug Discovery Today 7, no. 9 (2002): 491–92. http://dx.doi.org/10.1016/s1359-6446(02)02277-8.

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