Relevant bibliographies by topics / Microbiology and Parasitology

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Microbiology and Parasitology'

Author: Grafiati
Published: 4 June 2021
Last updated: 18 May 2024

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Journal articles on the topic "Microbiology and Parasitology"

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Garcia, Lynne S. "Parasitology." Reviews in Medical Microbiology 10, no. 1 (January 1999): 51–64. http://dx.doi.org/10.1097/00013542-199901000-00006.

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Kanungo, Reba. "MEDICAL PARASITOLOGY." Indian Journal of Medical Microbiology 26, no. 1 (January 2008): 99. http://dx.doi.org/10.1016/s0255-0857(21)02015-6.

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Castle, D. L. "Insecticide microbiology." Parasitology Today 2, no. 3 (March 1986): 90. http://dx.doi.org/10.1016/0169-4758(86)90173-0.

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Geissler, P. Wenzel. "Remembering Africanization: excerpt of reunion transcripts." Africa 90, no. 1 (January 2020): 35–50. http://dx.doi.org/10.1017/s0001972019000937.

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Professor Bukheti Swalehe Kilonzo, born 1949: joined Amani in 1968 as a Scientific Assistant. Studied microbiology and parasitology in London; PhD in plague epidemiology from Dar es Salaam in 1984. Left Amani in 1982 as Research Scientist and became professor at Sokoine University.
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Smits, Henk L., and Rudy A. Hartskeerl. "PCR amplification reactions in parasitology." Journal of Microbiological Methods 23, no. 1 (July 1995): 41–54. http://dx.doi.org/10.1016/0167-7012(95)00032-g.

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Richardson, Harold, Christine Fleming, Josephine Palmer, Erluo Chen, Robert Lannigan, and The Microbiology Committee. "An Assessment of the Utilization of Diagnostic Parasitology Laboratory Services in Ontario." Canadian Journal of Infectious Diseases 7, no. 4 (1996): 237–42. http://dx.doi.org/10.1155/1996/768923.

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OBJECTIVE: Diagnostic parasitology services in Ontario were assessed to determine whether the reduction in the number of provider laboratories from 209 to 70 over the period 1977 to 1994 has affected the availability and quality of service.DESIGN: A questionnaire on parasitology practice was distributed to all laboratories. Responses were validated against data from the requisitions, work cards and reports submitted by licensed parasitology laboratories in a pattern-of-practice survey.SETTING AND PARTICIPANTS: Laboratories in Ontario are licensed by the provincial Ministry of Health and subjected to mandatory proficiency testing. Laboratories not licensed in parasitology may receive specimens for referral to another facility for processing.MAIN OUTCOME MEASURES: Assessment included receipt of fecal specimens for parasitology, on-site testing, the limitation of testing, work referred elsewhere for primary investigation or confirmation, laboratory to which work was referred, and the turnaround time for both on-site and referred work.RESULTS: Ninety-eight per cent of 338 laboratories returned the questionnaire. Three hundred and six received specimens for parasitology. Sixty-four performed the testing. Of the remainder, 235 referred to 40 laboratories. Approximately 31,000 specimens were processed monthly, with 78% examined within private laboratories and 62% having a turnaround time of at least three days. The yield of positive findings was 3% or more of patients.CONCLUSIONS: The presence of fecal parasites has been reliably determined and the genus, species and stage of the organism correctly identified. Utilization must be improved through understanding of the factors increasing the pretest likelihood of a positive finding.
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Smith, Andrew G. "Tropical Microbiology." American Journal of Tropical Medicine and Hygiene 34, no. 2 (March 1, 1985): 413. http://dx.doi.org/10.4269/ajtmh.1985.34.2.tm0340020413a.

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Smith, Andrew G. "Medical Microbiology." American Journal of Tropical Medicine and Hygiene 36, no. 1 (January 1, 1987): 201. http://dx.doi.org/10.4269/ajtmh.1987.36.201.

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Fairley, Jessica K. "Human Parasitology, 5th Edition." Clinical Infectious Diseases 69, no. 5 (May 3, 2019): 906–7. http://dx.doi.org/10.1093/cid/ciz191.

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Chiodini, Peter L. "New diagnostics in parasitology." Infectious Disease Clinics of North America 19, no. 1 (March 2005): 267–70. http://dx.doi.org/10.1016/j.idc.2004.11.002.

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Dissertations / Theses on the topic "Microbiology and Parasitology"

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Getty, Troy A. "Life Cycle and Morphological Characterization of Colpodella sp. (ATCC 50594) in Hay Medium." Cleveland State University / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=csu1611795646162548.

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Rougeron, Amandine. "Distribution dans la mucoviscidose et écologie des différentes espèces du complexe Scedosporium apiospermum." Phd thesis, Université d'Angers, 2013. http://tel.archives-ouvertes.fr/tel-00966210.

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Le complexe Pseudallescheria boydii / Scedosporium apiospermum se situe au deuxième rang des champignons filamenteux isolés des expectorations de patients atteints de mucoviscidose, maladie génétique la plus fréquente dans la population caucasienne et dont le pronostic est étroitement lié à l'atteinte respiratoire. La distribution des cinq espèces constituant ce complexe a été étudiée au sein d'une cohorte de patients français atteints de mucoviscidose. Nos résultats suggèrent que P. boydii est l'espèce prédominante (62%), suivie de S. apiospermum (24%), Scedosporium aurantiacum (10%) et Pseudallescheria minutispora (4%). Nous avons également démontré la capacité de ces deux dernières espèces à coloniser de manière chronique les voies respiratoires de ces patients. Les niches naturelles et les réservoirs de ces espèces constituant des sources potentielles de contamination humaine ont ensuite été étudiées dans la région Pays-dela- Loire. Les densités les plus importantes de ces espèces ont été retrouvées dans les zones impactées par les activités humaines. Scedosporium dehoogii est l'espèce la plus abondante ; S. aurantiacum, P. boydii et S. apiospermum ont été principalement retrouvées dans les zones agricoles, les plages et les aires de jeux, respectivement. Les techniques de PCR étant plus rapides et plus sensibles que les méthodes culturales et permettant une semi-quantification, nous avons enfin développé une méthode moléculaire pour la détection et la quantification du complexe P. boydii / S. apiospermum. Cependant, une optimisation de l'étape d'extraction d'ADN génomique est nécessaire pour permettre l'application de cette méthode à des échantillons de sol.
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Mendonça, Daiane Barros Dias. "Análise do microbioma de bactérias de luz intestinal de hamsteres e sua correlação com LPS circulante, decorrente da translocação microbiana na leishmaniose visceral experimental." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/99/99131/tde-22012018-114555/.

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A leishmaniose visceral, na sua forma clinica ativa, caracteriza-se por febre de longa duração, hepatoesplenomegalia e caquexia. No Brasil, a letalidade é, em média, de 7% e as principais causas de morte são: hemorragia, comorbidade com doenças imunossupressoras e infecção bacteriana. O mecanismo de aumento de infecção bacteriana na LV não está claro e uma das hipóteses, é que pode haver translocação bacteriana da mucosa intestinal para o lúmen dos vasos sanguíneos e ocasionar uma maior severidade da resposta imuno-inflamatória e com consequente piora clínica. O objetivo deste trabalho foi avaliar a ocorrência de translocação microbiana em hamsteres infectados experimentalmente com Leishmania (L.) infantum e correlacionar com as alterações histopatológicas encontradas no intestino dos animais infectados. Hamsteres (Mesocricetus auratus) foram infectados intraperitonealmente com 2x107 amastigotas de L. (L.) infantum e eutanasiados após 48, 72 horas e 15, 45 e 90 dias de infecção. Como grupo controle foram utilizados hamsteres inoculados intraperitonealmente com meio de cultura RPMI. Foram coletados: sangue, fezes, baço, intestinos grosso e delgado. Para detecção de amastigotas na mucosa intestinal, foi utilizada a técnica de PCR em tempo real (qPCR), imunohistoquímica e análise histopatológica, sendo que nesta técnica também foram avaliadas alterações histológicas no tecido intestinal. O baço foi utilizado para determinar a carga parasitária através da técnica de Stauber. Para detecção da translocação microbiana ou produtos desde, foi realizada a quantificação de lipopolissacarídeo (LPS) em plasma. Para avaliar a possível mudança da flora bacteriana intestinal, foi realizado sequenciamento bacteriano de amostra de fezes de hamsteres controles e infectados nos vários tempos de infecção. Observamos aumento da carga parasitária em baço e em intestino com o decorrer da infecção, sendo a diferença significativa aos 90 dias de infecção. Paralelamente, observamos aumento de LPS circulante nos animais infectados em diferentes tempos, 48 horas, 72 horas, 45 dias e 90 dias, com diminuição no período intermediário de 15 dias, porém com diferença significante somente aos 90 dias após a infecção em relação ao grupo controle. Alterações histopatológicas foram observadas no intestino grosso e delgado, variando de infiltrado inflamatório leve a grave, enterite, histiocitose e ainda presença de amastigotas. As alterações observadas ocorreram a partir de 48 horas de infecção, diferenciando a população do infiltrado inflamatório entre neutrófilos, linfócitos, e ainda eosinófilos em intestino grosso de animais com 90 dias de infecção. O sequenciamento de DNA bacteriano das fezes mostra que houve alteração no microbioma dos animais, porém não há identidade significante, ou seja, acima de 95% na maioria das bactérias. Concluímos que as alterações de histologia da mucosa, a invasão de amastigotas neste tecido e o aumento do LPS, sugerem que a translocação microbiana é um evento ocorrente durante a infecção por L. (L.) infantum neste modelo experimental.
Visceral leishmaniasis, in its active clinical form, is characterized by long-lasting fever, hepatosplenomegaly and cachexia. In Brazil, the lethality is, on average, 7% and the main causes of death are hemorrhage, comorbidity with immunosuppressive diseases and bacterial infection. The mechanism of increased bacterial infection in LV is unclear and one of the hypotheses is that there may be bacterial translocation of the intestinal mucosa to the lumen of the blood vessels and cause a greater severity of the immune-inflammatory response and consequent clinical worsening. The objective of this work was evaluate the occurrence of microbial translocation in Leishmania (L.) infantum infected-hamsters and correlate with the histopathological changes found in the gut of infected animals. Hamsters (Mesocricetus auratus) were infected intraperitoneally with 2x107 amastigotes of L. (L.) infantum and euthanized after 48, 72 hours and 15, 45 and 90 days of infection. As a control group, hamsters were inoculated intraperitoneally with RPMI culture medium. Were collected: blood, feces, spleen, large and small intestines. To detection amastigotes in intestinal mucosa, real-time PCR (qPCR), immunohistochemistry and histopathological analysis were used, and histological alterations in intestinal tissue were also evaluated. Spleen was used to determine the parasitic load through the Stauber technique. To detection of microbial translocation or products related, was performed quantification of lipopolysaccharide (LPS) in plasma. In order to evaluate the possible change in intestinal bacterial flora, bacterial sequencing of sample faeces from control and infected hamsters was carried. We observed increased parasite load on spleen and intestine as the infection progressed, the difference being significant at 90 days of infection. At the same time, we observed increased circulating LPS in infected animals at different times, 48 hours, 72 hours, 45 days and 90 days, with decrease in the intermediate period of 15 days, howeversignificant difference was observed only at 90 days post-infection in relation to control group. Histopathological changes were observed in the large and small intestine, ranging from mild to severe inflammatory infiltrate, enteritis, histiocytosis, and amastigotes. The changes occurred from 48 hours of infection, differentiating the population of the inflammatory infiltrate between neutrophils, lymphocytes, and even eosinophils in the large intestine of animals with 90 days of infection. |Bacterial sequencing shows that there was a change in the microbiome of the animals, but there is no significant identity, ie, above 95% in most bacteria. We conclude that changes in mucosal histology, invasion of amastigotes in this tissue and increase in LPS, suggest that microbial translocation is an event occurring during L. (L.) infantum infection in this experimental model.
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Swenerton, Ryan Kells. "Biochemical and functional characterization of serine proteases in Leishmania." Diss., Search in ProQuest Dissertations & Theses. UC Only, 2009. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:3390080.

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Goldowitz, Ilana Sarah. "Plasmodium's Crossroads: Deciphering the Molecular Pathway That Leads to Malaria Transmission." Thesis, Harvard University, 2015. http://nrs.harvard.edu/urn-3:HUL.InstRepos:17467311.

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Plasmodium falciparum is the causative agent of the most severe form of malaria. Transmission from humans to mosquito vectors is an essential step in this eukaryotic parasite‘s life cycle and in the spread of malaria disease, which killed nearly 600,000 people in 2013. I investigated the developmental switch parasites make to the transmission stage or gametocyte, with the goal of identifying molecular mechanisms and environmental triggers of gametocyte formation. In Chapter 2 of this dissertation, I discuss the completion of a genetic mutagenesis screen leading to the discovery of a putative E3 ubiquitin ligase enzyme which is likely a negative regulator of gametocyte formation. Chapter 3 presents findings on population-based regulation of gametocyte production and on parasite-derived microvesicles that transfer between cells and stimulate gametocyte production. In Chapter 4, I and coauthors present a protocol for measuring parasite growth and gametocyte production in response to drugs or other treatments.
Chemical Biology
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Hamidou, Soumana Illiassou. "La Trypanosomose Humaine Africaine (maladie du sommeil) : caractérisation de gènes impliqués dans les interactions symbiontes - glossines - trypanosomes." Thesis, Montpellier 2, 2014. http://www.theses.fr/2014MON20182.

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Les glossines (mouches tsétsé) sont les vecteurs des trypanosomes africains, responsables de la Trypanosomose Humaine Africaine (THA) ou maladie du sommeil en Afrique sub-saharienne. De nouvelles stratégies de lutte contre la THA visent à utiliser les symbiontes de la glossine pour augmenter sa réfraction à l'infection par les trypanosomes. La mise en place de telles approches nécessite une bonne connaissance des bases moléculaires et cellulaires des interactions entre les symbiontes, la glossine et le trypanosome. Les objectifs de cette thèse étaient, i) d'évaluer l'évolution des densités des symbiontes (Wigglesworthia glossinidia et Sodalis glossinidius) au cours du cycle de développement du vecteur et ii) de caractériser les gènes de Sodalis, Glossina palpalis gambiensis et Trypanosome brucei gambiense en interaction et qui s'expriment différentiellement au cours de l'infection. Nous avons pu montrer la présence permanente des deux symbiontes quel que soit le stade de développement de la glossine, ce qui permet leur utilisation dans le cadre du contrôle des vecteurs. Par la suite, des infections expérimentales ont été réalisées sur des glossines d'insectarium. Des glossines de l'espèce G. p. gambiensis ont été gorgées sur des souris infectées par T. b. gambiense. L'analyse des métatranscriptomes des glossines infectées versus réfractaires à l'infection nous ont permis de mettre en évidence les gènes de Sodalis, G. p. gambiensis et T. b. gambiense différentiellement exprimés aux étapes clé de l'infection. Les résultats qui découlent de cette thèse mettent la lumière sur la complexité des interactions Sodalis - G. p. gambiensis - T. b. gambiense et soulignent l'implication des bactériophages du symbionte S. glossinidius dans la réfraction des glossines à l'infection. Mots clés : maladie du sommeil, mouche tsétsé, trypanosome, symbiontes, compétence vectorielle, expression de gènes
Tsetse flies are the vectors of African trypanosomes, the causative agents of human African trypanosomiasis (sleeping sickness)in sub-saharan Africa. New sleeping sickness control strategies plan to use tsetse gut symbionts to increase tsetse flies refractoriness to trypanosomes infection. Such approaches require good knowledge on the molecular and cellular basis of interactions between symbionts, tsetse fly and trypanosome. This thesis aimed to i) assess the evolution of Glossina palpalis gambiensis symbionts (Wigglesworthia glossinidia and Sodalis glossinidius) densities throughout the host fly development cycle and ii) to characterize genes of Sodalis, G. p. gambiensis and Trypanosoma brucei gambiense in interaction, which are differentially expressed during the infection. We showed that both symbionts are present in all tsetse fly development stages, allowing their use in the context of vector control. Subsequently, experimental infections were performed on colonies flies. G. p. gambiensis female flies were fed on T. b. gambiense hosting mice. Transcriptome of infected flies and flies that have cleared trypanosome they ingested were analysed. This allow us identifying genes of Sodalis, G. p. gambiensis and T. b. gambiense differentially expressed at the infection key stages. Our results highlight the complexity of interactions between Sodalis, G. p. gambiensis, T. b. gambiense and underline the involvement of bacteriophages hosted by S. glossinidius in tsetse fly refractoriness to trypanosome infection. Key words: sleeping sickness; tsetse fly; trypanosome; symbionts; vector competence; gene expression
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Krishnamurthy, Shruthi. "FUNCTIONAL CONSEQUENCES OF AMA1-RON2 INTERACTION DURING HOST CELL INVASION BY TOXOPLASMA." ScholarWorks @ UVM, 2016. http://scholarworks.uvm.edu/graddis/462.

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T.gondii is a model organism of the phylum Apicomplexa that infects one third of the human population. While the majority of infections are asymptomatic or manifest with mild flu-like symptoms, toxoplasmosis can be fatal in immunocompromised individuals and in the developing fetus. The lytic cycle of tachyzoite-stage parasites causes damage to the host by repeated rounds of host cell invasion, intracellular replication and lysis of the host cell upon egress. Invasion is a key step for the parasite to maintain its intracellular lifestyle. Apical Membrane Antigen 1 (AMA1) is an adhesin released from a unique set of secretory organelles called micronemes. AMA1 plays a central role in the initial stages of host cell invasion. Although parasites without AMA1 are viable in culture, virulence in an animal model of infection is completely attenuated, highlighting AMA1's functional importance. AMA1 is a type I transmembrane protein with a large ectodomain and a short cytoplasmic tail. The ectodomain of AMA1 interacts with domain 3 (D3) of rhoptry neck protein 2 (RON2), which in turn complexes with RONs 4, 5, and 8 in the host cell. Together, this complex of proteins forms the moving junction, through which the parasite pushes itself during invasion. Rhomboid proteases on the parasite surface cleave AMA1 within its transmembrane domain and parasites expressing a non-cleavable form of AMA1 show reduced invasion of host cells and a growth defect. While much is known about the ectodomain of T. gondii AMA1 (TgAMA1), the fate of the TgAMA1 cytoplasmic tail after cleavage remains unclear, its interacting partners remain unidentified, and its role in invasion or thereafter remains a mystery. To address these questions, we: (a) explored the consequences of TgAMA1-TgRON2 interaction during invasion and (b) generated allelic replacement (AR) parasites with point mutations across the tail of TgAMA1 to determine the effect of these mutations on the parasite's ability to invade host cells. Quantitative proteomic techniques were used to analyze the proteins that bind to the tail of TgAMA1 under these different experimental conditions. The results from this work highlight the importance of TgAMA1 post-translational modifications, and potentially TgAMA1-binding proteins, in regulating invasion-related processes in T. gondii.
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Thomas, Vincent. "Amibes libres de l'environnement : écologie et interactions avec des micro-organismes pathogènes émergents." Habilitation à diriger des recherches, Université Paris Sud - Paris XI, 2012. http://tel.archives-ouvertes.fr/tel-00694418.

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Le risque de contamination des réseaux par les légionelles est aujourd'hui bien connu. Ces bactéries sont naturellement résistantes à un certain nombre de stress, elles peuvent survivre de façon prolongée dans les biofilms et proliférer de façon très importante via une croissance intracellulaire dans les amibes. Les amibes peuvent elles-mêmes présenter une résistance élevée aux traitements de décontamination usuels et offrent donc une protection supplémentaire aux légionelles. Si l'association amibes-légionelles dans les réseaux a été largement étudiée, il n'en est pas de même pour un certain nombre d'autres associations existant entre les amibes et des micro-organismes pathogènes avérés ou pathogènes émergents. Parmi les agents pathogènes émergents associés aux amibes, de nouvelles espèces bactériennes apparentées aux Chlamydia pourraient être responsables de pneumonies et d'avortements à répétition chez l'Homme et l'animal. Ces nouvelles espèces de Chlamydia présentent de nombreuses particularités, dont une survie à l'état libre dans l'environnement beaucoup plus importante que celles des Chlamydia " classiques " et une résistance importante aux températures élevées. De nouveaux virus " géants " associés aux amibes ont aussi été récemment décrits. Ils présentent de nombreuses particularités biologiques par rapport aux virus plus classiques et, bien que les preuves formelles de leur pathogénicité restent à apporter, certains de ces virus pourraient être responsables de pneumonies. Enfin, l'association amibes libres - mycobactéries soulève des questions importantes. Des travaux récents démontrent que des mycobactéries atypiques résistantes à certains traitements de décontamination (glutaraldéhyde notamment) sont aussi plus résistantes à certains traitements antibiotiques et plus à même de survivre dans les amibes. Par ailleurs la plupart des espèces de mycobactéries présentent la capacité de survivre dans les kystes amibiens, eux-mêmes très résistants à la plupart des traitements de décontamination. Les travaux de recherche présentés dans ce mémoire portent sur l'étude de l'écologie des amibes libres de l'environnement ainsi que sur l'étude de leur résistance à différents types de traitements biocides. Le rôle des amibes en tant que lieu d'échange de matériel génétique entre micro-organismes est abordé, ainsi que l'interaction des amibes avec les légionelles, les espèces apparentées aux Chlamydia, les virus d'amibes et les mycobactéries.
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Vergnes, Mike. "Plasticité fonctionnelle et structurale chez Legionella pneumophila - Impact des protéines de type histone sur la virulence et génotypage par les séquences d'insertion." Phd thesis, Grenoble, 2010. http://tel.archives-ouvertes.fr/tel-00670047.

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Le genre Legionella regroupe des bactéries pouvant causer chez l'homme une pneumonie fatale dans 10% des cas, la légionellose. Elles sont capables de coloniser tous les réseaux d'eau. Le génome de ces bactéries révèle une forte plasticité génomique, aux niveaux fonctionnel et structural. La première partie de cette thèse analyse l'impact des protéines de type histone sur la régulation de la virulence chez L. pneumophila. Ces protéines structurent le chromosome bactérien et influencent l'expression génique. Des mutants des gènes codant les protéines Dps et IHF ont été obtenus chez L. pneumophila et analysés pour leur sensibilité aux stress et leurs propriétés de virulence. Ces deux protéines sont impliquées dans la régulation de la virulence chez Legionella. De plus, Dps permet de diminuer la sensibilité au stress oxydatif et IHF régulerait l'entrée dans l'état VBNC (viable but non-culturable), un état physiologique dans lequel les bactéries sont viables mais ne sont plus cultivables. La seconde partie vise à utiliser la plasticité structurale, notamment celle induite par les éléments génétiques mobiles IS, comme outil épidémiologique. A l'heure actuelle, les méthodes d'identification ne permettent pas de discriminer les isolats de même espèce. Afin d'éviter de nouvelles contaminations, il est impératif d'identifier rapidement et avec précision l'installation contaminée, à partir des prélèvements de patients comme élément comparatif. L'utilisation d'une méthode RFLP-IS a permis de mettre en évidence une IS particulière, ISLpn11, possédant un taux de discrimination de 80% au sein de la souche L. pneumophila Paris, qui est responsable de plus de 10 % des épidémies en Europe.
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Wang, Dong ying. "Diversité génétique et sensibilité aux antifongiques d'isolats d'Aspergillus spp. provenant d'élevages aviaires du Guangxi , Chine." Phd thesis, AgroParisTech, 2012. http://pastel.archives-ouvertes.fr/pastel-00779051.

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Les champignons du genre Aspergillus sont des moisissures banales de l'environnement. Elles sont présentes dans le sol et sur des végétaux en décomposition. Les Aspergillus se propagent par l'intermédiaire de spores microscopiques en suspension dans l'air. L'Homme et les animaux sont exposés en permanence aux spores aspergillaires mais les défenses immunes empêchent leur développement dans l'organisme. Lorsque ces défenses sont amoindries, une aspergillose est possible. Dans ce cas, Aspergillus fumigatus et A. flavus sont le plus souvent incriminés. Les oiseaux sont beaucoup plus sensibles que les mammifères et l'environnement représenté par les élevages aviaires est propice à la prolifération des moisissures du genre Aspergillus. L'objectif de ce travail de thèse a été de caractériser la diversité génétique et la sensibilité aux antifongiques d'isolats d'Aspergillus provenant d'élevages aviaires dans la province du Guangxi en Chine. La première partie de la thèse est une analyse bibliographique sur les champignons du genre Aspergillus, les aspergilloses et les caractéristiques de l'élevage aviaire en Chine. Une première enquête a été réalisée dans 3 élevages près de la ville de Nanning et dans un élevage (incluant un éclosoir) à proximité de la ville de Guilin. Des écouvillonnages pharyngés et des prélèvements d'air ont été réalisés pendant plusieurs semaines. Des prélèvements ont également été faits sur des œufs dans l'éclosoir. Cette enquête a montré que le niveau de contamination fongique dépendait du type d'élevage. De nombreux isolats fongiques ont pu être collectés : 188 isolats d'A. fumigatus et 159 isolats d'A. flavus. La seconde partie du travail expérimental a porté sur la caractérisation de la diversité génétique d'A. fumigatus et d'A. flavus. Pour cela, la technique MLVA (multiple locus VNTR analysis) a été utilisée. Pour A. flavus, 8 marqueurs VNTR (variable-number tandem-repeat) ont été sélectionnés et une réaction PCR multiplex a été mise au point. Au total, 91 isolats d'A. flavus, incluant 6 souches de référence, ont été caractérisées avec le panel des 8 marqueurs VNTR. Cette analyse a permis de définir 78 génotypes distincts et un index de discrimination de 0,993. L'analyse de 188 isolats d'A. fumigatus avec 10 marqueurs VNTR a permis de définir 142 génotypes distincts. Certains génotypes d'A. flavus ou d'A. fumigatus sont clairement regroupés dans le nuage de point généré par l'analyse MST (minimum spanning tree). La troisième partie du travail expérimental a porté sur la sensibilité aux antifongiques de 177 isolats d'A. fumigatus. Ces isolats ont été récupérés dans des élevages aviaires en Chine et en France. Les isolats de Chine sont pour la plupart sensibles avec des valeurs minimales inhibitrices (vis-à-vis de l'itraconazole) comprises entre 0,38 et 0,75 µg/mL. Les isolats de France sont pour la plupart sensibles avec des valeurs minimales inhibitrices (vis-à-vis de l'itraconazole) comprises entre 0.19 and 1 µg/mL. Quatre souches ont été considérées comme résistantes : 2 souches provenant de deux élevages en Chine et 2 souches provenant de deux élevages en France. Des mutations sur le gène Cyp51A ont été détectées pour 11 isolats (3 résistants et 8 sensibles). Vingt et une mutations nucléotidiques ont été identifiées. Onze de ces mutations sont silencieuses et 9 sont à l'origine d'un changement de la composition de la protéine. Sept substitutions ont déjà été décrites dans la littérature ; les mutations A116R, E130D et Q131H sont originales.
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Books on the topic "Microbiology and Parasitology"

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R, Murray Patrick, ed. Medical microbiology. 3rd ed. St. Louis, Mo: Mosby, 1998.

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R, Murray Patrick, ed. Medical microbiology. 2nd ed. St. Louis: Mosby, 1994.

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R, Murray Patrick, ed. Medical microbiology. 2nd ed. St. Louis: Mosby, 1993.

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R, Murray Patrick, ed. Medical microbiology. St. Louis: Mosby, 1990.

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S, Rosenthal Ken, and Pfaller Michael A, eds. Medical microbiology. 5th ed. Philadelphia: Elsevier Mosby, 2005.

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S, Rosenthal Ken, and Pfaller Michael A, eds. Medical microbiology. 6th ed. Philadelphia: Mosby/Elsevier, 2009.

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S, Rosenthal Ken, and Pfaller Michael A, eds. Medical microbiology. Philadelphia: Elsevier/Saunders, 2013.

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Swanson, Todd A. Microbiology I: immunology, parasitology, urology, and mycology. 5th ed. Philadelphia: Lippincott Williams & Wilkins, 2008.

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Swanson, Todd A. Microbiology I: immunology, parasitology, urology, and mycology. 5th ed. Philadelphia: Lippincott Williams & Wilkins, 2008.

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Manual of practical medical microbiology and parasitology. New York: Wiley, 1985.

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Book chapters on the topic "Microbiology and Parasitology"

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Garcia, Lynne S., and Gary W. Procop. "Diagnostic Medical Parasitology." In Manual of Commercial Methods in Clinical Microbiology, 284–308. Hoboken, NJ: John Wiley & Sons, Inc, 2016. http://dx.doi.org/10.1002/9781119021872.ch15.

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Garcia, Lynne S. "Diagnostic Medical Parasitology." In Manual of Commercial Methods in Clinical Microbiology, 274–305. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555817961.ch11.

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Linscott, Andrea J., and Susan E. Sharp. "Reagents, Stains, and Media: Parasitology." In Manual of Clinical Microbiology, 2310–16. Washington, DC, USA: ASM Press, 2015. http://dx.doi.org/10.1128/9781555817381.ch134.

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Pritt, Bobbi S. "Advanced Techniques in Diagnostic Parasitology." In Advanced Techniques in Diagnostic Microbiology, 199–218. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-95111-9_8.

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Shimizu, Robyn Y., and Lynne S. Garcia. "Specimen Collection, Transport, and Processing: Parasitology." In Manual of Clinical Microbiology, 2293–309. Washington, DC, USA: ASM Press, 2015. http://dx.doi.org/10.1128/9781555817381.ch133.

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Gillespie, S. H. "Parasitology." In Medical Microbiology Illustrated, 125–45. Elsevier, 1994. http://dx.doi.org/10.1016/b978-0-7506-0187-0.50016-x.

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Bhatia, Rajesh, and Rattan Ichhpujani. "Parasitology." In Microbiology for Nurses, 151. Jaypee Brothers Medical Publishers (P) Ltd., 2003. http://dx.doi.org/10.5005/jp/books/10529_33.

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Mukherjee, PK, and Preeti Bandyopadya. "Ocular Parasitology." In Ocular Microbiology, 121. Jaypee Brothers Medical Publishers (P) Ltd., 2010. http://dx.doi.org/10.5005/jp/books/11027_7.

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Worthington, Tony. "Clinical parasitology." In Medical Microbiology. Oxford University Press, 2019. http://dx.doi.org/10.1093/hesc/9780198818144.003.0011.

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This chapter focuses on clinical parasitology, which is an important sub-discipline of pure parasitology and may be defined as the study of human infections caused by ecto- and endoparasites. During the twenty-first century, parasites have come to contribute to an ever-increasing number of human blood, tissue, and gastrointestinal infections that are often associated with important risk factors. The chapter begins by differentiating between protozoa, nematodes, cestodes, and trematodes. It looks at the diagnosis of gastrointestinal parasitic infections acquired through the gastrointestinal tract, before describing the examination of faeces for ova, cysts, and parasites (OCP). The chapter then examines immunological and molecular techniques, and discusses the treatment of parasitic infections acquired through the gastrointestinal tract.
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"Parasitology." In Practical Medical Microbiology for Clinicians, 303–11. Hoboken, NJ: John Wiley & Sons, Inc, 2015. http://dx.doi.org/10.1002/9781119066767.ch22.

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Conference papers on the topic "Microbiology and Parasitology"

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Liu, Limei, Liyuan Sun, Yun Yu, Wei Xia, and Chunhe Li. "The Application of E-Learning System Based on the Network Teaching Platform in Microbiology and Parasitology Teaching." In 2015 7th International Conference on Information Technology in Medicine and Education (ITME). IEEE, 2015. http://dx.doi.org/10.1109/itme.2015.66.

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Aguiar, Agila de Oliveira, Fabiana Sanches Furtado, Gabriele Caroline Nunes Miranda, Gelson Pinto Alves, and Luciana Mendes Fernandes. "PESQUISA DE FORMAS EVOLUTIVAS DE PARASITOS COM POTENCIAL ZOONÓTICO NO SOLO DE BAIRROS DO MUNICÍPIO DE TUCURUÍ, PARÁ, BRASIL." In II Congresso Brasileiro de Parasitologia Humana On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/conbrapah/19.

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Introdução: O aumento da população de cães e gatos em áreas urbanas tem papel epidemiológico importante na contaminação do solo de áreas públicas e na disseminação de infecções pelos mais variados gêneros de parasitos. A confirmação da presença de parasitos de importância médico veterinária serve de alerta a população humana a respeito dos riscos em que a mesma encontra-se exposta. Objetivo: Neste sentido, esta pesquisa teve como escopo principal avaliar a incidência de ovos, cistos e/ou larvas de parasitos no solo de bairros do município de Tucuruí, no Pará. Material eMétodos: Foram coletadas 15 amostras de solo de vias públicas e domicílios de cada bairro, no período de Novembro a Dezembro de 2021, totalizando 30 amostras. Após a coleta, as amostras foram acondicionadas em isopor, devidamente identificadas e encaminhadas ao Laboratório de Microbiologia e Parasitologia do IFPA/Campus Tucuruí. Em seguida, foram processadas pela técnica flutuação no açúcar (método de Sheather) e observadas ao microscópio óptico nas objetivas de 10X e 40X. Resultados: Do total de amostras coletadas, 50% (15 amostras) foram positivas para parasitas de solo com potencial zoonótico. O bairro com maior contaminação foi o Cohab (10 amostras positivas). Os principais protozoários encontrados nas amostras positivas foram: Iodamoeba butschlii (11,76%); Balantidium coli (8,82%); Hymenolepis diminuta (5,88%) e Entamoeba sp. (2,94%). Os helmintos encontrados foram: Taenia sp. (20,58%); Ancylostoma sp.(11,76%); Toxocara sp. (11,76%); Ascaris sp. (5,88%); Dipylidium caninum (5,88%); Trichuris vulpis (5,88%); Enterobius vermicularis (2,94%), Fasciola hepatica (2,94%) e Strongyloides stercoralis (2,94%). Conclusão: Os achados desse estudo comprovaram que em vias públicas e domicílios dos bairros estudados em Tucuruí, há parasitos com potencial zoonótico, ressaltando a necessidade de implantar ações integradas ligadas à saúde pública para a conscientização da população humana quanto às zoonoses, bem como suas formas de transmissão e prevenção.
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Alves, Matheus Tenório, Taleny dos Santos Moreira, Ivânia Emanuele Louzada Caldas, Janilis Lima Carvalho, and Luciana Mendes Fernandes. "AVALIAÇÃO PARASITOLÓGICA DA ÁGUA DE ABASTECIMENTO E DO SOLO PERIDOMICILIAR DE RIBEIRINHOS DO LAGO DA UHE TUCURUÍ – PARÁ." In II Congresso Brasileiro de Parasitologia Humana On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/conbrapah/27.

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Introdução: A água e o solo podem transportar formas evolutivas de parasitos, representando um perigo para a transmissão de bioagentes, especialmente entre populações socioeconomicamente desfavorecidas, situação comum no cotidiano dos ribeirinhos da região do Lago de Tucuruí. Portanto, a presença desses endoparasitas no meio ambiente torna-se um sério problema de saúde pública. Objetivo: Neste sentido, o objetivo desta pesquisa foi avaliar a qualidade parasitológica da água de abastecimento e solo peridomiciliar em uma comunidade ribeirinha do lago da UHE-Tucuruí/PA. Material e Métodos: Foram coletadas 25 amostras de água (volume final 2L) nas captações das residências e 25 amostras de solo, obtidas por meio da escavação de 15cm do solo em quatro pontos da casa, totalizando 600g/amostra, durante o mês de novembro/2021. Em seguida, as amostras foram devidamente identificadas, acondicionadas e encaminhadas ao Laboratório de Microbiologia e Parasitologia do IFPA/Campus Tucuruí. Para observação microscópica dos parasitas, utilizou-se as técnicas de sedimentação espontânea, técnica flutuação no açúcar (método de Sheather) e métodos flotação centrífuga em solução de sacarose. Resultados: Os resultados forampositivos para parasitos nas amostras de água, detectando-se Balantidium coli (5,26%) e Cryptosporidium sp (94,74%). Do total de amostras coletadas do solo, 72% (18 amostras) apresentaram positividade para helmintos e protozoários. Pelo método flotação centrífuga em solução de sacarose foi identificado Strongyloides stercoralis (75%) e Toxocara sp (25%). No método de Sheather, os mais encontrados foramEndolimax nana (48,52%), Iodamoeba butschlii (12,43%), Balantidium coli (10,65%) e Entamoeba histolytica (6,51%). Finalmente, no método sedimentação espontânea, detectaram-se Ancylostoma sp. (50%) e Trichuris trichiura (50%). Conclusão: Os resultados indicam elevada contaminação ambiental e deficiência no tratamento de água de consumo dos ribeirinhos da UHE Tucuruí – Pará. Com isso, medidas devem serem tomadas para minimizar fatores de riscos para a saúde de consumidores de água oriunda do lago da UHE Tucuruí – Pará.
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Freire, Isabela Franco, JÚLIA LEITÃO CABRAL, FRANCISCO WAGNER VASCONCELOS FREIRE FILHO, and MARIA DE FÁTIMA FIGUEIREDO. "CARDIOMIOPATIA CHAGÁSICA: ETIOPATOGENIA E MANIFESTAÇÕES CLÍNICAS." In II Congresso Nacional de Microbiologia Clínica On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/ii-conamic/14.

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Introdução: A Doença de Chagas é uma antropozoonose americana predominantemente rural causada pelo protozoário intracelular facultativo Trypanosoma cruzi, cujo vetor é o Triatoma infestans, hematófago conhecido como “barbeiro”. A infecção se manifesta em duas fases. Na aguda, ocorre multiplicação parasitária no miocárdio. Na crônica, acontece um processo de miocardite focal de baixa intensidade, cuja persistência promove o desenvolvimento de cardiomiopatia dilatada. Objetivos: Demonstrar a etiopatogenia e as manifestações cardíacas da forma crônica da Doença de Chagas. Material e métodos: Trata-se de pesquisa bibliográfica realizada na base de dados PubMed, utilizando “Cardiomiopatia Chagásica” como descritor, encontrando-se 6 artigos, e em 4 livros-texto de Parasitologia, Patologia, Semiologia e Clínica Médica, datados de 2017 a 2021. Resultados: A Cardiomiopatia Chagásica (CC) consiste em lesão fibrosante progressiva ao miocárdio decorrente da presença da forma amastigota do T. cruzi nos cardiomiócitos. A infecção provoca infiltrado inflamatório de neutrófilos, linfócitos e macrófagos. A reação imune - celular e humoral - à permanência tecidual do parasita é um relevante processo etiopatogênico. Isso resulta, a longo prazo, em cardiomiopatia dilatada, que consiste na perda de cardiomiócitos e em sua substituição por tecido fibrótico, desprovido de inotropismo. A miocardite consequente dessa necrose das miofibrilas pode evoluir, após 10 a 30 anos, para insuficiência cardíaca congestiva, mais comumente à esquerda, que pode chegar a nível IV NYHA. A CC associa-se ao desenvolvimento de desordens arrítmicas, especialmente bloqueios atrioventriculares. Os mais comuns são BRD, BDAS-E e BAV Total, sendo os dois últimos marcadores de pior prognóstico. Nessa perspectiva, pacientes apresentam frequentemente a Síndrome de Stokes Adams, uma síncope bradicárdica cuja diminuição do débito cardíaco pode resultar em morte súbita. Tal fatalidade, de alta incidência em pacientes chagásicos, também pode decorrer do comprometimento da microvasculatura em consequência da resposta imune contínua, que diminui a perfusão miocárdica e resulta em fenômenos tromboembólicos. Conclusão: A CC trata-se de uma doença crônica prevenível, grave, de comprometimento progressivo da função cardíaca, limitadora da qualidade de vida e associada a morte súbita. O conhecimento da história natural da doença se faz importante para a prevenção, diagnóstico precoce e tratamento da doença, podendo diminuir sua morbimortalidade.
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