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Journal articles on the topic 'Mild Cognitive Impairment Alzheimer's disease'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Kelley, Brendan J., and Ronald C. Petersen. "Alzheimer's Disease and Mild Cognitive Impairment." Neurologic Clinics 25, no. 3 (2007): 577–609. http://dx.doi.org/10.1016/j.ncl.2007.03.008.

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2

Masumi, Jafar, Fatemeh Fekar Gharamaleki, Neda Gilani, Maryam Noroozian, and Ahmad Reza Khatoonabadi. "Exploring language performance in people with mild cognitive impairment and Alzheimer's disease: a prospective longitudinal study." International Journal of Therapy and Rehabilitation 30, no. 12 (2023): 1–13. http://dx.doi.org/10.12968/ijtr.2022.0058.

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Background/Aims Language changes are the earliest signs and symptoms of mild cognitive impairment and Alzheimer's disease. The aim of this study was to explore the language difference performance between people with mild cognitive impairment, people with Alzheimer's disease and healthy individuals as a control group. To accomplish this aim, the language profile of both mild cognitive impairment and Alzheimer's disease needs to be characterised to determine which feature is more affected by both mild cognitive impairment and Alzheimer's disease among expressive language, receptive language, exe

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3

Zancada-Menéndez, Clara, Patricia Sampedro-Piquero, Azucena Begega, Laudino López, and Jorge Luis Arias. "Attention and inhibition in Mild Cognitive Impairment and Alzheimer's Disease." Escritos de Psicología - Psychological Writings 6, no. 3 (2013): 43–50. http://dx.doi.org/10.24310/espsiescpsi.v6i3.13288.

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Mild cognitive impairment is understood as a cognitive deficit of insufficient severity to fulfil the criteria for Alzheimer’s disease. Many studies have attempted to identify which cognitive functions are most affected by this type of impairment and which is the most sensitive neuropsychological test for early detection. This study investigated sustained and selective attention, processing speed, and the inhibition process using a sample of people divided into three groups mild cognitive impairment, Alzheimer disease and cognitively healthy controls selected and grouped based on their scores

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4

Forlenza, Orestes V., Breno S. Diniz, Leda L. Talib, et al. "Clinical and biological predictors of Alzheimer's disease in patients with amnestic mild cognitive impairment." Revista Brasileira de Psiquiatria 32, no. 3 (2010): 216–22. http://dx.doi.org/10.1590/s1516-44462010005000002.

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OBJECTIVE: To identify predictors of the progression from pre-dementia stages of cognitive impairment in Alzheimer's disease is relevant to clinical management and to substantiate the decision of prescribing antidementia drugs. METHOD: Longitudinal study of a cohort of elderly adults with amnestic mild cognitive impairment and healthy controls, carried out to estimate the risk and characterize predictors of the progression to Alzheimer's disease. RESULTS: Patients with amnestic mild cognitive impairment had a higher risk to develop Alzheimer's disease during follow-up (odds ratio = 4.5, CI95%

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Snowden, J. S. "MILD COGNITIVE IMPAIRMENT: AGING TO ALZHEIMER'S DISEASE." Brain 127, no. 1 (2004): 231—a—233. http://dx.doi.org/10.1093/brain/awh010.

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Petersen, Ronald C. "AGING, MILD COGNITIVE IMPAIRMENT, AND ALZHEIMER'S DISEASE." Neurologic Clinics 18, no. 4 (2000): 789–805. http://dx.doi.org/10.1016/s0733-8619(05)70226-7.

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7

Jellinger, K. A. "Mild Cognitive Impairment. Aging to Alzheimer's disease." European Journal of Neurology 10, no. 4 (2003): 466. http://dx.doi.org/10.1046/j.1468-1331.2003.00603_3.x.

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8

Chertkow, Howard. "Mild cognitive impairment: Aging to Alzheimer's disease." Annals of Neurology 55, no. 4 (2004): 600. http://dx.doi.org/10.1002/ana.20057.

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Appel, Jason, Elizabeth Potter, Qian Shen, et al. "A Comparative Analysis of Structural Brain MRI in the Diagnosis of Alzheimer’s Disease." Behavioural Neurology 21, no. 1-2 (2009): 13–19. http://dx.doi.org/10.1155/2009/103123.

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Dementia is a debilitating and life-altering disease which leads to both memory impairment and decline of normal executive functioning. While causes of dementia are numerous and varied, the leading cause among patients 60 years and older is Alzheimer’s disease. The gold standard for Alzheimer’s diagnosis remains histological identification of amyloid plaques and neurofibrillary tangles within the medial temporal lobe, more specifically the entorhinal cortex and hippocampus. Although no definitive cure for Alzheimer's disease currently exists, there are treatments targeted at preserving cogniti

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10

Zhou, Mengzhen. "The Relationship between Sleep Disturbance and Early Alzheimer's Disease – Mild Cognitive Impairment." Neuroscience and Neurological Surgery 14, no. 1 (2024): 01–06. http://dx.doi.org/10.31579/2578-8868/280.

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During sleep, the brain can clear toxins related to Alzheimer's disease. Compared with the baseline without sleep disorder, subjects with sleep disorder have a 1.49-fold increased risk of Alzheimer's disease. There is a two-way relationship between sleep/wakefulness disorder and Alzheimer's disease pathology. Sleep plays an important role in memory consolidation. Research shows that sleep disorder may have an important impact on the symptoms and course of Alzheimer's disease. Therefore, we need to better understand the mechanism of cognitive and behavioral impairment related to Alzheimer's dis

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11

T. S. Sindhu, N. Kumaratharan, P. Anandan, and P. Durga. "Performance Evaluation of CNN Models for Alzheimer’s Disease Detection with MRI Scans." Journal of Innovative Image Processing 5, no. 4 (2023): 390–402. http://dx.doi.org/10.36548/jiip.2023.4.004.

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The primary symptom of Alzheimer's disease is memory impairment, which is a neurodegenerative condition. The manifestation of these symptoms can be attributed to the impairment of the cerebral nerve responsible for cognitive functions such as learning, thinking and memory. Alzheimer’s disease is a prominent cause of mortality and lacks a definitive curve. However, appropriate medicinal interventions have demonstrated the potential to mitigate the progression and severity of the condition. This study presents the comparison of Convolutional Neural Network (CNN) models, namely ResNet50, VGG19 an

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12

Besser, Lilah M., Irene Litvan, Sarah E. Monsell, et al. "Mild cognitive impairment in Parkinson's disease versus Alzheimer's disease." Parkinsonism & Related Disorders 27 (June 2016): 54–60. http://dx.doi.org/10.1016/j.parkreldis.2016.04.007.

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13

Carvalho, Janessa O., Jing Ee Tan, Beth A. Springate, and Jennifer D. Davis. "Self-reported depressive syndromes in mild cognitive impairment and mild Alzheimer's disease." International Psychogeriatrics 25, no. 3 (2012): 439–44. http://dx.doi.org/10.1017/s1041610212001676.

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ABSTRACTBackground: There is suggestion that self-reported depressive syndromes can independently manifest in the general population as cognitive/affective or somatic/vegetative. The Beck Depression Inventory, 2nd edition (BDI-II), a self-report measure of depressive symptoms, has been shown to support this two-factor structure. However, this finding has not been examined in an older adult sample with cognitive impairment. In order to determine whether older adults with cognitive impairments exhibit similarly independent cognitive/affective and somatic/vegetative depressive syndromes, we explo

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14

Visser, Pieter Jelle, Frans R. J. Verhey, Rudolf W. H. M. Ponds, and Jellemer Jolles. "Diagnosis of Preclinical Alzheimer's Disease in a Clinical Setting." International Psychogeriatrics 13, no. 4 (2001): 411–23. http://dx.doi.org/10.1017/s1041610201007839.

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Introduction. The aim of the study was to investigate whether the preclinical stage of Alzheimer's disease (AD) can be diagnosed in a clinical setting. To this end we investigated whether subjects with preclinical AD could be differentiated from subjects with nonprogressive mild cognitive impairment and from subjects with very mild AD-type dementia. Methods. Twenty-three subjects with preclinical AD, 44 subjects with nonprogressive mild cognitive impairment, and 25 subjects with very mild AD-type dementia were selected from a memory clinic population. Variables that were used to differentiate

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15

Nelson, Aaron P., and Margaret G. O'Connor. "Mild Cognitive Impairment: A Neuropsychological Perspective." CNS Spectrums 13, no. 1 (2008): 56–64. http://dx.doi.org/10.1017/s1092852900016163.

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ABSTRACTMild cognitive impairment (MCI) is a clinical diagnosis in which deficits in cognitive function are evident but not of sufficient severity to warrant a diagnosis of dementia. For the majority of patients, MCI represents a transitional state between normal aging and mild dementia, usually Alzheimer's disease. Multiple subtypes of MCI are now recognized. In addition to presentations featuring memory impairment, symptoms in other cognitive domains (eg, executive function, language, visuospatial) have been identified. Neuropsychological testing can be extremely useful in making the MCI dia

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16

Capucho, Patrícia Helena Figueirêdo Vale, and Sonia Maria Dozzi Brucki. "Judgment in Mild Cognitive Impairment and Alzheimer's disease." Dementia & Neuropsychologia 5, no. 4 (2011): 297–302. http://dx.doi.org/10.1590/s1980-57642011dn05040007.

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Abstract Judgment is the capacity to make decisions after considering available information, contextual factors, possible solutions and probable outcomes. Our aim was to investigate previous research studies regarding assessment of judgment in older adults with different degrees of cognitive impairment. To this end, a search of Pubmed and Lilacs electronic databases for studies published from January 1990 until August 2011 in English, Spanish and Portuguese was carried out. The terms used were "judgment" combined with the terms "dementia" or "Mild Cognitive Impairment" (MCI) or "Alzheimer's di

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17

Ryan, Joanne, Jaqueline Scali, Isabelle Carriere, Karen Ritchie, and Marie-Laure Ancelin. "Hormonal treatment, mild cognitive impairment and Alzheimer's disease." International Psychogeriatrics 20, no. 1 (2008): 47–56. http://dx.doi.org/10.1017/s1041610207006485.

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ABSTRACTA plethora of in vitro and in vivo studies have supported the neuroprotective role of estrogens and their impact on the neurotransmitter systems implicated in cognition. Recent hormonal replacement therapy (HRT) trials in non-demented postmenopausal women suggest a temporary positive effect (notably on verbal memory), and four meta-analyses converge to suggest a possible protective effect in relation to Alzheimer's disease (reducing risk by 29 to 44%). However, data from the only large randomized controlled trial published to date, the Women's Health Initiative Memory Study, did not co

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18

Reisberg, Barry, Leslie Prichep, Lisa Mosconi, et al. "The pre-mild cognitive impairment, subjective cognitive impairment stage of Alzheimer's disease." Alzheimer's & Dementia 4, no. 1S1 (2008): S98—S108. http://dx.doi.org/10.1016/j.jalz.2007.11.017.

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19

Adlam, Anna-Lynne R., Sasha Bozeat, Robert Arnold, Peter Watson, and John R. Hodges. "Semantic Knowledge in Mild Cognitive Impairment and Mild Alzheimer's Disease." Cortex 42, no. 5 (2006): 675–84. http://dx.doi.org/10.1016/s0010-9452(08)70404-0.

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20

Choi, M. S. "Neuropsychiatric symptoms in mild Alzheimer's disease and mild cognitive impairment." Journal of the Neurological Sciences 333 (October 2013): e357. http://dx.doi.org/10.1016/j.jns.2013.07.1309.

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21

Priya, P. Lakshmi, G. Veerapandu, and Sanjeev Kumar. "Predictive Biomarker Grade Transfer Alzheimer's disease and Mild Cognitive Impairment." International Journal of Electrical and Electronics Research 10, no. 4 (2022): 853–57. http://dx.doi.org/10.37391/ijeer.100416.

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The disease of Alzheimer’s is a neurodegenerative disease that affects the brain. This participated in the progress to Mild Cognitive Impairment (MCI) in Alzheimer's disease (AD) with effect is not solitary critical in medical observation but also has a considerable perspective to improve medical trials. This learning intends to establish an efficient biomarker for predicting accurately the conversion of AD in MCI to Magnetic Resonance Image (MRI). This learning executed an Event-Related Potential (ERP) study on patient and control collection commencing 32 channel EEG obtained throughout N-bac

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22

Spíndola, Lívia, and Sonia Maria Dozzi Brucki. "Prospective memory in Alzheimer's disease and Mild Cognitive Impairment." Dementia & Neuropsychologia 5, no. 2 (2011): 64–68. http://dx.doi.org/10.1590/s1980-57642011dn05020002.

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Abstract Prospective memory (PM) is defined as remembering to carry out intended actions at an appropriate point in the future, and can be categorized into three types of situation: time-, event-, and activity-based tasks. PM involves brain structures such as frontal and medial temporal cortices. The aim of this study was to review the currently available literature on PM in Alzheimer's disease and Mild Cognitive Impairment patients. We performed a search on Pubmed, Medline, ScieLO, LILACS and the Cochrane Library electronic databases from January 1990 to December 2010. The key terms used were

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23

Verma, Nishant, S. Natasha Beretvas, Belen Pascual, Joseph C. Masdeu, Mia K. Markey, and The Alzheimer's Disease Neuroimaging Initiative. "A Biomarker Combining Imaging and Neuropsychological Assessment for Tracking Early Alzheimer's Disease in Clinical Trials." Current Alzheimer Research 15, no. 5 (2018): 429–42. http://dx.doi.org/10.2174/1567205014666171106150309.

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Background: Combining optimized cognitive (Alzheimer's Disease Assessment Scale- Cognitive subscale, ADAS-Cog) and atrophy markers of Alzheimer's disease for tracking progression in clinical trials may provide greater sensitivity than currently used methods, which have yielded negative results in multiple recent trials. Furthermore, it is critical to clarify the relationship among the subcomponents yielded by cognitive and imaging testing, to address the symptomatic and anatomical variability of Alzheimer's disease. Method: Using latent variable analysis, we thoroughly investigated the relatio

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24

Petersen, Ronald C., Ronald G. Thomas, Paul S. Aisen, Richard C. Mohs, Maria C. Carrillo, and Marilyn S. Albert. "Randomized controlled trials in mild cognitive impairment." Neurology 88, no. 18 (2017): 1751–58. http://dx.doi.org/10.1212/wnl.0000000000003907.

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Objective:To examine the variability in performance among placebo groups in randomized controlled trials for mild cognitive impairment (MCI).Methods:Placebo group data were obtained from 2 National Institute on Aging (NIA) MCI randomized controlled trials, the Alzheimer's Disease Cooperative Study (ADCS) MCI trial and the Alzheimer's Disease Neuroimaging Initiative (ADNI), which is a simulated clinical trial, in addition to industry-sponsored clinical trials involving rivastigmine, galantamine, rofecoxib, and donepezil. The data were collated for common measurement instruments. The performance

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25

Pavlovic, Dragan, and Aleksandra Pavlovic. "Mild cognitive impairment." Srpski arhiv za celokupno lekarstvo 137, no. 7-8 (2009): 434–39. http://dx.doi.org/10.2298/sarh0908434p.

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Mild cognitive impairment (MCI) is a syndrome that spans the area between normal ageing and dementia. It is classified into amnestic and non-amnestic types, both with two subtypes: single domain and multiple domains. Prevalence of MCI depends on criteria and population and can vary from 0.1 to 42% persons of older age. In contrast to dementia, cognitive deterioration is less severe and activities of daily living are preserved. Most impaired higher cognitive functions in MCI are memory, executive functions, language, visuospatial functions, attention etc. Also there are depression, apathy or ps

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26

Lonie, J. A., K. M. Tierney, L. L. Herrmann, et al. "Dual task performance in early Alzheimer's disease, amnestic mild cognitive impairment and depression." Psychological Medicine 39, no. 1 (2008): 23–31. http://dx.doi.org/10.1017/s0033291708003346.

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BackgroundThe dual task paradigm (Baddeleyet al.1986; Della Salaet al.1995) has been proposed as a sensitive measure of Alzheimer's dementia, early in the disease process.MethodWe investigated this claim by administering the modified dual task paradigm (utilising a pencil-and-paper version of a tracking task) to 33 patients with amnestic mild cognitive impairment (aMCI) and 10 with very early Alzheimer's disease, as well as 21 healthy elderly subjects and 17 controls with depressive symptoms. All groups were closely matched for age and pre-morbid intellectual ability.ResultsThere were no group

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27

Tangen, Gro Gujord, Knut Engedal, Astrid Bergland, Tron Anders Moger, and Anne Marit Mengshoel. "Relationships Between Balance and Cognition in Patients With Subjective Cognitive Impairment, Mild Cognitive Impairment, and Alzheimer Disease." Physical Therapy 94, no. 8 (2014): 1123–34. http://dx.doi.org/10.2522/ptj.20130298.

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Background Balance impairments are common in patients with Alzheimer disease (AD), but which aspects of balance are affected, at which stage of cognitive impairment, and their associations with cognitive domains remain unexplored. Objectives The aims of this study were: (1) to explore differences in balance abilities among patients with subjective cognitive impairment (SCI) or mild cognitive impairment (MCI), mild AD, and moderate AD and (2) to examine the relationship between the various aspects of balance and cognitive domains. Design This was a cross-sectional study. Methods Home-dwelling p

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28

Arai, Hiroyuki. "Biomarkers, mild cognitive impairment and early diagnosis of Alzheimer's disease." Nippon Ronen Igakkai Zasshi. Japanese Journal of Geriatrics 40, no. 1 (2003): 22–26. http://dx.doi.org/10.3143/geriatrics.40.22.

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29

Lee, Jun Ho, Min Soo Byun, Dahyun Yi, et al. "Frequency of Depressive Syndromes in Elderly Individuals with No Cognitive Impairment, Mild Cognitive Impairment, and Alzheimer's Disease Dementia in a Memory Clinic Setting." Dementia and Geriatric Cognitive Disorders 42, no. 3-4 (2016): 135–45. http://dx.doi.org/10.1159/000449155.

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Aims: The aims of this study were to investigate the frequency of various depressive syndromes in elderly individuals with no cognitive impairment (NC), mild cognitive impairment (MCI), and Alzheimer's disease dementia (AD) in a memory clinic setting, and then to test whether severe and milder forms of depressive syndromes are differentially associated with the cognitive groups. Methods: For 216 NC, 478 MCI, and 316 AD subjects, we investigated the frequency of depressive syndromes, defined by three different categories: major and minor depressive disorder (MaDD and MiDD) according to the Diag

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30

Yarnall, Alison Jane, Lynn Rochester, and David John Burn. "Mild cognitive impairment in Parkinson's disease." Age and Ageing 42, no. 5 (2013): 567–76. http://dx.doi.org/10.1093/ageing/aft085.

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Abstract The concept of mild cognitive impairment (MCI) in the general population has received increased attention over recent years, and is associated with risk of progression to Alzheimer's disease. Within Parkinson's disease (PD), MCI (PD-MCI) is also now recognised to be relatively common, with certain subtypes predicting progression to Parkinson's disease dementia (PDD). Recently, criteria to better characterise PD-MCI and its subtypes have been produced by the Movement Disorder Society. In contrast to the population as a whole, where amnestic MCI is the most common subtype, non-amnestic

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31

Baldeiras, Inês, Isabel Santana, Maria Teresa Proença, et al. "Peripheral Oxidative Damage in Mild Cognitive Impairment and Mild Alzheimer's Disease." Journal of Alzheimer's Disease 15, no. 1 (2008): 117–28. http://dx.doi.org/10.3233/jad-2008-15110.

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&NA;. "Levacecarnine is effective in mild cognitive impairment and mild Alzheimer's disease,." Inpharma Weekly &NA;, no. 1380 (2003): 18. http://dx.doi.org/10.2165/00128413-200313800-00043.

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Peltsch, Alicia, Alisha Hemraj, Angeles Garcia, and Douglas P. Munoz. "Saccade deficits in amnestic mild cognitive impairment resemble mild Alzheimer's disease." European Journal of Neuroscience 39, no. 11 (2014): 2000–2013. http://dx.doi.org/10.1111/ejn.12617.

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34

Santana, Isabel, Rita Guerreiro, Artur Paiva, et al. "P4-208 Inflammation in mild cognitive impairment and mild Alzheimer's disease." Neurobiology of Aging 25 (July 2004): S534. http://dx.doi.org/10.1016/s0197-4580(04)81766-4.

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35

Werner, P., S. Rosenblum, G. Bar-On, J. Heinik, and A. Korczyn. "Handwriting Process Variables Discriminating Mild Alzheimer's Disease and Mild Cognitive Impairment." Journals of Gerontology Series B: Psychological Sciences and Social Sciences 61, no. 4 (2006): P228—P236. http://dx.doi.org/10.1093/geronb/61.4.p228.

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36

Britt III, William G., Anne M. Hansen, Sofia Bhaskerrao, et al. "Mild Cognitive Impairment: Prodromal Alzheimer's Disease or Something Else?" Journal of Alzheimer's Disease 27, no. 3 (2011): 543–51. http://dx.doi.org/10.3233/jad-2011-110740.

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37

Morris, John C., and Jeffrey Cummings. "Mild cognitive impairment (MCI) represents early-stage Alzheimer's disease." Journal of Alzheimer's Disease 7, no. 3 (2005): 235–39. http://dx.doi.org/10.3233/jad-2005-7306.

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38

Petersen, Ronald C., and David Bennett. "Mild Cognitive Impairment: Is it Alzheimer's disease or Not?" Journal of Alzheimer's Disease 7, no. 3 (2005): 241–45. http://dx.doi.org/10.3233/jad-2005-7307.

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39

Van der Mussele, Stefan, Peter Mariën, Jos Saerens, et al. "Behavioral Syndromes in Mild Cognitive Impairment and Alzheimer's Disease." Journal of Alzheimer's Disease 38, no. 2 (2013): 319–29. http://dx.doi.org/10.3233/jad-130596.

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40

Mitchell, Alexandra G., Stephanie Rossit, Suvankar Pal, et al. "Peripheral reaching in Alzheimer's disease and mild cognitive impairment." Cortex 149 (April 2022): 29–43. http://dx.doi.org/10.1016/j.cortex.2022.01.003.

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Popp, Julius, Karsten Schaper, Heike Kölsch, et al. "CSF cortisol in Alzheimer's disease and mild cognitive impairment." Neurobiology of Aging 30, no. 3 (2009): 498–500. http://dx.doi.org/10.1016/j.neurobiolaging.2007.07.007.

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42

Blacioti, M. A., R. Moraru, and L. Spiru. "Personality changes in mild cognitive impairment versus alzheimer's disease." European Psychiatry 26, S2 (2011): 1009. http://dx.doi.org/10.1016/s0924-9338(11)72714-6.

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IntroductionEstimates for Romanian patients with dementia range from 200,000 to 300,000, but reports warn that actual numbers may be higher.ObjectivesBased on our experience we inferred that specific personality changes may occur in patients with mild cognitive impairment (MCI) and Alzheimer (AD).AimsWe tried to quantify such personality changes in MCI and AD patients.MethodWe designed an observational study (n = 60 patients, aged 55+) with MCI (n = 30; 50% male; mean age ∼ 73) and moderate AD (n = 30; 43.3% male (n = 13); mean age ∼74). We “profiled” patient personality (Woodworth - Mathews q

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43

Zhang, Daoqiang, Yaping Wang, Luping Zhou, Hong Yuan, and Dinggang Shen. "Multimodal classification of Alzheimer's disease and mild cognitive impairment." NeuroImage 55, no. 3 (2011): 856–67. http://dx.doi.org/10.1016/j.neuroimage.2011.01.008.

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Heckemann, Rolf A., Shiva Keihaninejad, Paul Aljabar, et al. "Automatic morphometry in Alzheimer's disease and mild cognitive impairment." NeuroImage 56, no. 4 (2011): 2024–37. http://dx.doi.org/10.1016/j.neuroimage.2011.03.014.

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45

Bucholtz, Nina, and Ilja Demuth. "DNA-repair in mild cognitive impairment and Alzheimer's disease." DNA Repair 12, no. 10 (2013): 811–16. http://dx.doi.org/10.1016/j.dnarep.2013.07.005.

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46

Song, Fei, Anne Poljak, George A. Smythe, and Perminder Sachdev. "Plasma biomarkers for mild cognitive impairment and Alzheimer's disease." Brain Research Reviews 61, no. 2 (2009): 69–80. http://dx.doi.org/10.1016/j.brainresrev.2009.05.003.

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Stam, C. J., Y. Van Der Made, Y. A. L. Pijnenburg, and Ph Scheltens. "EEG synchronization in mild cognitive impairment and Alzheimer's disease." Acta Neurologica Scandinavica 108, no. 2 (2003): 90–96. http://dx.doi.org/10.1034/j.1600-0404.2003.02067.x.

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Chalermpalanupap, Termpanit, Becky Kinkead, William T. Hu, et al. "Targeting norepinephrine in mild cognitive impairment and Alzheimer's disease." Alzheimer's Research & Therapy 5, no. 2 (2013): 21. http://dx.doi.org/10.1186/alzrt175.

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Beinhoff, U., H. Tumani, J. Brettschneider, D. Bittner, and M. W. Riepe. "Gender-specificities in Alzheimer's disease and mild cognitive impairment." Journal of Neurology 255, no. 1 (2008): 117–22. http://dx.doi.org/10.1007/s00415-008-0726-9.

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50

Nestor, Peter J., Tim D. Fryer, Peter Smielewski, and John R. Hodges. "Limbic hypometabolism in Alzheimer's disease and mild cognitive impairment." Annals of Neurology 54, no. 3 (2003): 343–51. http://dx.doi.org/10.1002/ana.10669.

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