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Journal articles on the topic 'Minimal brain dysfunctions'

Author: Grafiati

Published: 28 May 2022

Last updated: 30 July 2025

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1

RAISS, JOHN C., and DENNIS P. CANTWELL. "The Minimal Brain Dysfunctions: Diagnosis and Treatment." American Journal of Psychiatry 142, no. 5 (1985): 651–52. http://dx.doi.org/10.1176/ajp.142.5.651.

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2

Linden, M. "Minimal emotional dysfunctions (MED) in personality disorders." European Psychiatry 21, no. 5 (2006): 325–32. http://dx.doi.org/10.1016/j.eurpsy.2006.01.010.

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AbstractThe concept of minimal emotional dysfunctions (MED) refers to traditional psychopathology in order to describe, classify, and understand personality disorders. Emotional dysfunctions encompass disorders of affect predominance, production, expression, experience, modulation, and regulation. MED can explain the dimensional nature of personality disorders, their multidimensionality and problems with categorical classifications. It can stimulate research on the etiology of personality disorders in reference to modern developmental brain research and trauma psychology. It can guide new deve

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Shimchenko, E. V., E. I. Kleshchenko, E. P. Apalkova, et al. "THE ALLERGIC REACTIONS IN CHILDREN WITH MINIMAL BRAIN DYSFUNCTIONS, FEATURES OF TREATMENT." Kubanskij nauchnyj medicinskij vestnik, no. 4 (January 1, 2017): 173–75. http://dx.doi.org/10.25207/1608-6228-2017-24-4-173-175.

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Tokareva, Yulia A., and Svetlana V. Volik. "Typology of Primary School Children with Identified Developmental Disability and Learning Difficulties." Lurian Journal 4, no. 1 (2023): 21–32. http://dx.doi.org/10.15826/lurian.2023.4.1.2.

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At the present stage of the development of the system of psychological and pedagogical support for the subjects of the educational process, the goal of timely diagnosis and identification children with developmental problems that are the reason for the lack of the necessary psychological basis to master the educational program becomes the most urgent. According to research over the past 10 years, the number of primary school pupils who fail to cope with the program with preserved intelligence is steadily growing. The study aimed at identifying the psychological characteristics of children with

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Marczak, Elżbieta, Maria Szarras-Czapnik, Małgorzata Wójcik, et al. "Hypopituitarism—A rare manifestation in Joubert syndrome: about 4 cases." AIMS Medical Science 11, no. 3 (2024): 318–29. http://dx.doi.org/10.3934/medsci.2024022.

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<p>Joubert syndrome (JS) is a complex medical condition characterized by a pathognomonic midbrain-hindbrain malformation visible on brain imaging, which is known as the “molar tooth sign” (MTS). The presence of the MTS in the brain is the defining diagnostic criterion for JS. Individuals with JS commonly exhibit a developmental delay, hypotonia, and abnormal eye movements. In addition, neonatal breathing dysregulation is observed in about half of the cases. Midline brain defects associated with JS can lead to pituitary hormone abnormalities, thereby manifesting as multiple pituitary insu

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Dovzhikova, I. V., I. A. Andrievskaya, N. A. Ishutina, I. N. Gorikov, S. V. Medvedeva, and M. T. Lutsenko. "DISTURBANCE OF THE PROGESTERONE AND ITS METABOLITES SYNTHESIS IN THE DEVELOPMENT OF NEUROLOGICAL DISORDERS IN CHILDREN AFTER CYTOMEGALOVIRUS INFECTION DURING PREGNANCY." Acta Biomedica Scientifica 3, no. 4 (2018): 78–84. http://dx.doi.org/10.29413/abs.2018-3.4.12.

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Cytomegalovirus (CMV) infection is one of the causes of congenital neurological disorders and the virus itself is the most common viral agent causing an imbalance in the production of placenta progesterone and its neuroactive metabolites – allopregnenolone and 5α-dihydroprogesterone. The aim was to evaluate the concentration of progesterone and its metabolites – 5α-dihydroprogesterone and allopregnenolone – in placenta during exacerbation of CMV infection in the first trimester of pregnancy, and the impact of these disturbances on the development of neurological disorders in children. We exami

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Dummler, Bettina, Oliver Tschopp, Debby Hynx, Zhong-Zhou Yang, Stephan Dirnhofer, and Brian A. Hemmings. "Life with a Single Isoform of Akt: Mice Lacking Akt2 and Akt3 Are Viable but Display Impaired Glucose Homeostasis and Growth Deficiencies." Molecular and Cellular Biology 26, no. 21 (2006): 8042–51. http://dx.doi.org/10.1128/mcb.00722-06.

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ABSTRACT To address the issues of isoform redundancy and isoform specificity of the Akt family of protein kinases in vivo, we generated mice deficient in both Akt2 and Akt3. In these mice, only the Akt1 isoform remains to perform essential Akt functions, such as glucose homeostasis, proliferation, differentiation, and early development. Surprisingly, we found that Akt2 −/− Akt3 −/− and even Akt1 +/− Akt2 −/− Akt3 −/− mice developed normally and survived with minimal dysfunctions, despite a dramatic reduction of total Akt levels in all tissues. A single functional allele of Akt1 appears to be s

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8

Jr., Leon Oettinger. "Minimal Brain Dysfunction." Developmental Medicine & Child Neurology 20, no. 4 (2008): 531–32. http://dx.doi.org/10.1111/j.1469-8749.1978.tb15264.x.

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Sillevis, Rob, Joshua Unum, Valerie Weiss, Eric Shamus, and Francisco Selva-Sarzo. "The effect of a spinal thrust manipulation’s audible pop on brain wave activity: a quasi-experimental repeated measure design." PeerJ 12 (June 28, 2024): e17622. http://dx.doi.org/10.7717/peerj.17622.

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Introduction High velocity thrust manipulation is commonly used when managing joint dysfunctions. Often, these thrust maneuvers will elicit an audible pop. It has been unclear what conclusively causes this audible sound and its clinical meaningfulness. This study sought to identify the effect of the audible pop on brainwave activity directly following a prone T7 thrust manipulation in asymptomatic/healthy subjects. Methods This was a quasi-experimental repeated measure study design in which 57 subjects completed the study protocol. Brain wave activity was measured with the Emotiv EPOC+, which

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10

Дасуев, И. Л., and М. Л. Хайдарова. "Features of identifying negative experiences in primary school students." Management of Education, no. 8(66) (June 30, 2023): 116–22. http://dx.doi.org/10.25726/q2003-2539-4644-e.

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Статья посвящена теоретическому обоснованию и изучению особенностей негативных переживаний у младших школьников. Авторы пришли к выводу, что негативные переживания младших школьников следует рассматривать как эмоции, основанные на неприятных субъективных и объективных переживаниях, которые активируют механизм адаптивного поведения, направленного на устранение причины опасности (физической или психической). Особенностью негативных переживаний в младшем школьном возрасте является неудовлетворение сверхситуативных потребностей психологического характера в общении, одобрении, уважении, а также пот

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11

Dragos, F. Voicu, Anghel Lucretia, Baroiu Liliana, and Stan Dorina. "About Minimal Hepatic Encephalopathy." BRAIN. Broad Research in Artificial Intelligence and Neuroscience 11, No 1 Sup 1 (2020): 70–77. https://doi.org/10.18662/brain/11.1Sup1/30.

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Introduction. Hepatic encephalopathy comprises a broad spectrum of neuropsychiatric manifestations, commonly encountered in patients with hepatic dysfunction, in the absence of other concomitant causes of cerebral infarction. Under these conditions, varying degrees of consciousness, neuromuscular dysfunction, cognitive, behavioral and emotional deficits coexist. Minimal hepatic encephalopathy (MHE) is defined by a normal neurological clinical examination but with cognitive deficits that can be highlighted by neuropsychological testing. Material and Methods. A retrospective study was conducted

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12

Saravia-Campos, J. "‘MINIMAL BRAIN DYSFUNCTION’: AN OVER-SIMPLIFICATION?" Developmental Medicine & Child Neurology 18, no. 2 (2008): 246–48. http://dx.doi.org/10.1111/j.1469-8749.1976.tb03638.x.

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13

Carey, William B., Sean C. McDevitt, and David Baker. "Differentiating Minimal Brain Dysfunction and Temperament." Developmental Medicine & Child Neurology 21, no. 6 (2008): 765–72. http://dx.doi.org/10.1111/j.1469-8749.1979.tb01699.x.

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Antonis, Theofilidis. "The Hypothesis of Unexplained Brain Damage and Learning Difficulties." Neuroscience and Neurological Surgery 8, no. 5 (2021): 01–08. http://dx.doi.org/10.31579/2578-8868/165.

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Introduction: The term learning disabilities refers to a complex and multidimensional phenomenon that affects many thousands of students. Due to the rich symptomatology of learning difficulties and the increased differences between individuals, it has not been possible to analyze all cases exclusively from the perspective of the neuropsychological approach. Aim. The aim of this study was to present research conducted in the context of theories on the brain function of people with learning disabilities. Supporting the hypothesis of brain dysfunction. Methodology: Literature review was carried o

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15

Rye, Henning, and Liv Hundevadt. "Emotional problems and treatment in minimal brain dysfunction." Nordic Journal of Psychiatry 47, no. 2 (1993): 123–32. http://dx.doi.org/10.3109/08039489309095022.

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16

Kelly, M. Kathleen. "Neuroanatomical and Neurophysiological Correlates of Minimal brain Dysfunction." Physical & Occupational Therapy In Pediatrics 7, no. 4 (1988): 101–8. http://dx.doi.org/10.1080/j006v07n04_08.

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17

Voinov, V. B. "Psychophysiology of maladaptation to school: Minimal brain dysfunction." Doklady Biological Sciences 419, no. 1 (2008): 77–79. http://dx.doi.org/10.1134/s0012496608020026.

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18

Schlager, G., D. E. Newman, H. G. Dunn, J. U. Crichton, and M. Schulzer. "Bone Age in Children with Minimal Brain Dysfunction." Developmental Medicine & Child Neurology 21, no. 1 (2008): 41–51. http://dx.doi.org/10.1111/j.1469-8749.1979.tb01579.x.

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19

Cakirpaloglu, P., and T. Radill. "Visual-motor organization in Minimal Brain Dysfunction (MBD) children." International Journal of Psychophysiology 11, no. 1 (1991): 16. http://dx.doi.org/10.1016/0167-8760(91)90083-a.

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20

Shamansurov, S., N. Sokhieva, N. Ziyamuhamedova, and N. Mirdjuraeva. "P26.14 Bioelectric brain activity in children of preschool age with minimal brain dysfunction." European Journal of Paediatric Neurology 15 (May 2011): S137. http://dx.doi.org/10.1016/s1090-3798(11)70481-0.

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21

Schmidt, M. H., G. Esser, W. Allehoff, B. Geisel, M. Laught, and W. Woerner. "EVALUATING THE SIGNIFICANCE OF MINIMAL BRAIN DYSFUNCTION?RESULTS OF AN EPIDEMIOLOGICAL STUDY." Journal of Child Psychology and Psychiatry 28, no. 6 (1987): 803–21. http://dx.doi.org/10.1111/j.1469-7610.1987.tb00670.x.

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22

Cakirpaloglu, P., and T. Radil. "Perceptual-Motor Coordination and Style in Healthy Boys and Those with Minimal Brain Dysfunction." Perceptual and Motor Skills 75, no. 2 (1992): 448–50. http://dx.doi.org/10.2466/pms.1992.75.2.448.

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Differences in visual-motor tracking by 8- to 9-yr.-old boys diagnosed with Minimal Brain Dysfunction or healthy have been analyzed. The subject's task was to use two computer keys to control a cursor line within a target which moved horizontally in two directions over the display. Target size and speed were the experimental variables. Off-target incidences and durations were measured and evaluated. The latter were longer and the former were more frequent on small targets for boys with brain dysfunction than for healthy boys. The two measures correlated negatively in most situations. The perce

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23

Rakhimbaeva, G. S., and N. M. Tujchibaeva. "CBP022 EEG – video monitoring in diagnostics of the minimal brain dysfunction at children." European Journal of Paediatric Neurology 11 (September 2007): 92. http://dx.doi.org/10.1016/s1090-3798(08)70603-2.

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Cakirpaloglu, P., T. Radil, J. Langmeier, and Z. Matějček. "Visual-motor disturbances in minimal Brain Dysfunction (MBD) children compared with normal ones." International Journal of Psychophysiology 7, no. 2-4 (1989): 156. http://dx.doi.org/10.1016/0167-8760(89)90103-7.

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25

Yu-Cun, Shen, Wang Yu-Feng, and Yang Xiao-Ling. "AN EPIDEMIOLOGICAL INVESTIGATION OF MINIMAL BRAIN DYSFUNCTION IN SIX ELEMENTARY SCHOOLS IN BEIJING." Journal of Child Psychology and Psychiatry 26, no. 5 (1985): 777–87. http://dx.doi.org/10.1111/j.1469-7610.1985.tb00591.x.

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26

Erickson, Marilyn T. "Reading Disability in Relation to Performance on Neurological Tests for Minimal Brain Dysfunction." Developmental Medicine & Child Neurology 19, no. 6 (2008): 768–75. http://dx.doi.org/10.1111/j.1469-8749.1977.tb08391.x.

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27

Elsner, Jürg. "Tactile-kinesthetic system of rats as an animal model for minimal brain dysfunction." Archives of Toxicology 65, no. 6 (1991): 465–73. http://dx.doi.org/10.1007/bf01977358.

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28

Rommelse, N. N. J. "Doet minimal brain dysfunction zijn herintrede in de DSM-5 als neurobiologische ontwikkelingsstoornis?" Kind en adolescent 41, no. 4 (2020): 402–4. http://dx.doi.org/10.1007/s12453-020-00253-5.

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Millichap, Lauren Elizabeth, Elisabetta Damiani, Luca Tiano, and Iain P. Hargreaves. "Targetable Pathways for Alleviating Mitochondrial Dysfunction in Neurodegeneration of Metabolic and Non-Metabolic Diseases." International Journal of Molecular Sciences 22, no. 21 (2021): 11444. http://dx.doi.org/10.3390/ijms222111444.

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Many neurodegenerative and inherited metabolic diseases frequently compromise nervous system function, and mitochondrial dysfunction and oxidative stress have been implicated as key events leading to neurodegeneration. Mitochondria are essential for neuronal function; however, these organelles are major sources of endogenous reactive oxygen species and are vulnerable targets for oxidative stress-induced damage. The brain is very susceptible to oxidative damage due to its high metabolic demand and low antioxidant defence systems, therefore minimal imbalances in the redox state can result in an

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30

Weissenborn, Karin. "The Clinical Relevance of Minimal Hepatic Encephalopathy - A Critical Look." Digestive Diseases 33, no. 4 (2015): 555–61. http://dx.doi.org/10.1159/000375348.

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Minimal hepatic encephalopathy (mHE) is diagnosed in patients with severe liver disease but no clinical symptoms of encephalopathy if either neuropsychological or neurophysiological tests indicate cerebral dysfunction and other possible causes of brain dysfunction have been excluded. mHE is characterized by deficits in attention, visuospatial orientation, visuoconstructive abilities and motor function. Accordingly, mHE can be expected to interfere with a subject's working ability, especially in those occupations that require handiwork, and driving ability. Indeed, about 60% of blue-collar work

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Cakirpaloglu, Panajotis, and Tomáš Radil. "Some Neuropsychological and Psychological Parameters in Children with Minimal Brain Dysfunction: A Transcultural Comparison." Perceptual and Motor Skills 72, no. 1 (1991): 80–82. http://dx.doi.org/10.2466/pms.1991.72.1.80.

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Cakirpaloglu, Panajotis, and Tomáš Radil. "Correlations between Symbolic Motor Performance and Achievement in School for Children with Minimal Brain Dysfunction." Perceptual and Motor Skills 73, no. 3 (1991): 952–54. http://dx.doi.org/10.2466/pms.1991.73.3.952.

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Significant correlations were found between grades for reading, writing, and calculating and each child's individual mean reaction time for symbolic motor performance tasks with letters and numbers as stimuli for 240 elementary school boys with minimal brain dysfunction and in first to fourth classes in Prague (Czechoslovakia) and Skopje (Yugoslavia) For age(class)-matched groups of 382 healthy boys the correlations disappeared by the age of ten years. Similar results were noted for groups of boys in both cities.

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M. Fikar, Rosniati Hakim, Dasrizal Dahlan, and Ahmad Lahmi. "GANGGUAN OTAK YANG BERKAITAN DENGAN PROSES BELAJAR." AL-MUADDIB: Jurnal Kajian Ilmu Kependidikan 7, no. 2 (2025): 279–87. https://doi.org/10.46773/muaddib.v7i2.1629.

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This study explores the relationship between brain disorders and the learning process from the perspective of neuroscience in Islamic education. Highlighting the concepts of intellect and brain based on the Qur'an, it explains how brain function influences human learning ability. The neuroscience approach, particularly in the anatomy of the brain, such as the cerebrum, cerebellum, brainstem, and limbic system, is elaborated to understand the brain's role in learning. Several brain disorders, including Minimal Brain Dysfunction (MBD), dyslexia, dyscalculia, and ADHD, are also described as facto

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Cakirpaloglu, Panajotis, and Tomáš Radil. "On Development of Visual-Motor Performance in Normal Boys and Boys with Minimal Brain Dysfunction." Perceptual and Motor Skills 70, no. 2 (1990): 426. http://dx.doi.org/10.2466/pms.1990.70.2.426.

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35

Gazda, Jakub, Peter Drotar, Sylvia Drazilova, et al. "Artificial Intelligence and Its Application to Minimal Hepatic Encephalopathy Diagnosis." Journal of Personalized Medicine 11, no. 11 (2021): 1090. http://dx.doi.org/10.3390/jpm11111090.

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Hepatic encephalopathy (HE) is a brain dysfunction caused by liver insufficiency and/or portosystemic shunting. HE manifests as a spectrum of neurological or psychiatric abnormalities. Diagnosis of overt HE (OHE) is based on the typical clinical manifestation, but covert HE (CHE) has only very subtle clinical signs and minimal HE (MHE) is detected only by specialized time-consuming psychometric tests, for which there is still no universally accepted gold standard. Significant progress has been made in artificial intelligence and its application to medicine. In this review, we introduce how art

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Xu, Su, Wenjun Zhu, Yamin Wan, et al. "Decreased Taurine and Creatine in the Thalamus May Relate to Behavioral Impairments in Ethanol-Fed Mice: A Pilot Study of Proton Magnetic Resonance Spectroscopy." Molecular Imaging 17 (January 1, 2018): 153601211774905. http://dx.doi.org/10.1177/1536012117749051.

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Minimal hepatic encephalopathy (MHE) is highly prevalent, observed in up to 80% of patients with liver dysfunction. Minimal hepatic encephalopathy is defined as hepatic encephalopathy with cognitive deficits and no grossly evident neurologic abnormalities. Clinical management may be delayed due to the lack of in vivo quantitative methods needed to reveal changes in brain neurobiochemical biomarkers. To gain insight into the development of alcoholic liver disease–induced neurological dysfunction (NDF), a mouse model of late-stage alcoholic liver fibrosis (LALF) was used to investigate changes i

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37

Jarmołowicz, Jakub, Agata Boczar, Patryk Dryja, Sven Solisch, Izabela Stawicka, and Izabela Orzołek. "Minimal hepatic encephalopathy - from cirrhosis to neurological disorders." Journal of Education, Health and Sport 76 (December 8, 2024): 56382. https://doi.org/10.12775/jehs.2024.76.56382.

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Introduction and aim Minimal hepatic encephalopathy (MHE) is a subtle but significant brain dysfunction resulting from chronic liver damage, with symptoms that can be difficult to detect. It affects patients' quality of life by causing problems with concentration, memory, and motor coordination. Untreated MHE may progress to more severe neurological disorders, such as overt hepatic encephalopathy. The importance of this condition lies in its impact on patients' daily functioning and the increased risk of accidents, particularly in situations requiring quick responses, such as driving.Neurologi

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Hong, Jeong-Min. "Perioperative brain health: strategies to prevent perioperative neurocognitive disorders." Journal of the Korean Medical Association 63, no. 9 (2020): 540–47. http://dx.doi.org/10.5124/jkma.2020.63.9.540.

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Cognitive changes in patients after anesthesia and surgery have been recognized for over 100 years. Research on postoperative cognitive changes accelerated in the 1980s and the term postoperative cognitive dysfunction emerged, which was used until recently. Postoperative cognitive dysfunction has been used in research to describe an objectively measurable decline in cognitive function using neuropsychological tests. This dysfunction had significant heterogeneity in the type, number of tests, timing of tests, and the criteria for change. Therefore, a recent article recommended a new nomenclatur

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Li, Jing-Li, Heng Jiang, Xiao-Dong Zhang, et al. "Microstructural brain abnormalities correlate with neurocognitive dysfunction in minimal hepatic encephalopathy: a diffusion kurtosis imaging study." Neuroradiology 61, no. 6 (2019): 685–94. http://dx.doi.org/10.1007/s00234-019-02201-4.

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Shariff, Erum, and Nehad Mahmoud Soltan. "Posterior Reversible Encephalopathy Syndrome (PRES) following blood transfusion in a polytrauma victim, an atypical occurrence." Turkish Journal of Emergency Medicine 24, no. 3 (2024): 180–84. http://dx.doi.org/10.4103/tjem.tjem_30_24.

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Abstract Posterior reversible encephalopathy syndrome (PRES) is a neuroradiological syndrome, clinically present by impaired consciousness, headache, visual disturbances, and seizures, and radiologically brain edema. Cases of PRES induced by blood transfusion are rarely documented. We report this case to increase the awareness of treating physicians for the possible complications of rapid blood transfusion. A 29-year-old man presented with polytrauma and was in hemorrhagic shock. He was transfused with multiple transfusions. Later, he was found to have quadriplegia with minimal movement of fin

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Bakker, Nelleke. "Minimal Brain Damage/Dysfunction (MBD) en de ontwikkeling van de wetenschappelijke kinderstudie in Nederland, ca. 1950–1990." Studium 7, no. 2 (2014): 82. http://dx.doi.org/10.18352/studium.9569.

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Lobanov, I. Y. "MINIMAL BRAIN DYSFUNCTION AS THE PREDICTOR FOR FORMATION OF ALCOHOL AND SUBSTANCE USE DISORDERS IN YOUNG ADULTS." Bulletin of Problems Biology and Medicine 1.1, no. 142 (2018): 45. http://dx.doi.org/10.29254/2077-4214-2018-1-1-142-45-50.

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Gustavson, K. H., F. Rasmussen, Y. Flodérus, S. B. Ross, B. Bille, and L. Wetterberg. "Catechol-O-methyltransferase activity in erythrocytes and plasma dopamine-B-hydroxylase activity in familial Minimal Brain Dysfunction." Clinical Genetics 23, no. 1 (2008): 75–77. http://dx.doi.org/10.1111/j.1399-0004.1983.tb00440.x.

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44

Kondziolka, Douglas, Steven N. Kalkanis, Minesh P. Mehta, Manmeet Ahluwalia, and Jay S. Loeffler. "It Is Time to Reevaluate the Management of Patients With Brain Metastases." Neurosurgery 75, no. 1 (2014): 1–9. http://dx.doi.org/10.1227/neu.0000000000000354.

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Abstract There are many elements to the science that drives the clinical care of patients with brain metastases. Although part of an understanding that continues to evolve, a number of key historical misconceptions remain that commonly drive physicians' and researchers' attitudes and approaches. By understanding how these relate to current practice, we can better comprehend our available science to provide both better research and care. These past misconceptions include: Misconception 1: Once a primary cancer spreads to the brain, the histology of that primary tumor does not have much impact o

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Yuan, Xiaodong, Lu Wang, Neha Tandon, et al. "Triheptanoin Mitigates Brain ATP Depletion and Mitochondrial Dysfunction in a Mouse Model of Alzheimer’s Disease." Journal of Alzheimer's Disease 78, no. 1 (2020): 425–37. http://dx.doi.org/10.3233/jad-200594.

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Background: Brain energy failure is an early pathological event associated with synaptic dysfunction in Alzheimer’s disease (AD). Thus, mitigation or enhancement of brain energy metabolism may offer a therapeutic avenue. However, there is uncertainty as to what metabolic process(es) may be more appropriate to support or augment since metabolism is a multiform process such that each of the various metabolic precursors available is utilized via a specific metabolic pathway. In the brain, these pathways sustain not only a robust rate of energy production but also of carbon replenishment. Objectiv

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Haisraely, Ory, Yaccov Lawerence, and Alicia Taliansky. "RADT-10. CEREBELLAR RE-IRRADIATION AFTER WHOLE BRAIN RADIOTHERAPY SIGNIFICANT SYMPTOM RELIEF WITH MINIMAL TOXICITY IN METASTATIC BRAIN PATIENTS." Neuro-Oncology 26, Supplement_8 (2024): viii74. http://dx.doi.org/10.1093/neuonc/noae165.0294.

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Abstract INTRODUCTION Whole-brain irradiation (WBRT) remains a standard treatment for patients with extensive brain metastases, providing symptom relief and prolonging survival. However, patients often experience recurrent metastases, necessitating re-irradiation. This study evaluates the efficacy and safety of cerebellar-only re-irradiation for improving symptoms and reducing toxicity in patients with recurrent cerebellar metastases following WBRT. METHODS A retrospective review was conducted on patients who underwent cerebellar re-irradiation between 2017 and 2023 after prior WBRT for sympto

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Liebman, Tracey N., and Sara C. Crystal. "What Is the Evidence that Riboflavin Can Be Used for Migraine Prophylaxis?" Einstein Journal of Biology and Medicine 27, no. 1 (2016): 7. http://dx.doi.org/10.23861/ejbm20112718.

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Individuals with migraine often have recurrent, painful symptoms, and symptomatic treatments have detrimentalside effects and do not prevent further attacks. Studies indicate that riboflavin can be used to decrease headachefrequency and lessen the need for symptomatic treatment. Mitochondrial dysfunction may play a role in migrainepathogenesis by interfering with oxygen metabolism. Daily doses of riboflavin, vitamin B2, may improve mitochondrialfunction by increasing the reserve of brain mitochondrial energy, and there are minimal side effects with dailytreatment. However, there is a need for

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Shenoy, Anitha. "Care of the brain dead organ donor." Indian Journal of Respiratory Care 05, no. 02 (2022): 705–12. http://dx.doi.org/10.5005/jp-journals-11010-05202.

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Organ donation is becoming more common but there is still a large gap between the number of people requiring transplants and the organs donated. There are set criteria for organ donation. When organ donation is considered after brain death, the physician must ensure that the prerequisites for testing are met and proceed to establish brain death using standard guidelines. The pathophysiological changes that occur after brain death must be borne in mind and utmost care should be given to counter those changes that would result in dysfunction of the donated organs. The brain dead patient must be

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Fuller, Joshua T., Tabina K. Choudhury, Deborah A. Lowe, and Steve Balsis. "Hallucinations and Delusions Signal Alzheimer’s Associated Cognitive Dysfunction More Strongly Compared to Other Neuropsychiatric Symptoms." Journals of Gerontology: Series B 75, no. 9 (2019): 1894–904. http://dx.doi.org/10.1093/geronb/gbz032.

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AbstractObjectivesNeuropsychiatric symptoms (NPS) are common among individuals with dementia of the Alzheimer’s type (DAT). We sought to characterize which NPS more purely relate to cognitive dysfunction in DAT, relative to other NPS.MethodDemographic, neurocognitive, neuroimaging, and NPS data were mined from the Alzheimer’s Disease Neuroimaging Initiative database (n = 906). Using factor analysis, we analyzed the degree to which individual NPS were associated with DAT-associated cognitive dysfunction. We also employed item response theory to graphically depict the ability of individual NPS t

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Upadhyaya, A. K., M. T. Abou-Saleh, K. Wilson, S. J. Grime, and M. Critchley. "A Study of Depression in Old Age Using Single-Photon Emission Computerised Tomography." British Journal of Psychiatry 157, S9 (1990): 76–81. http://dx.doi.org/10.1192/s0007125000291873.

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There is evidence to suggest that organic cerebral changes occur in elderly depressed patients. Quantitative studies of elderly depressed patients have demonstrated that they have reduced ‘radio-attenuation’ on computerised tomography (CT) scans and as a group resembled more closely a group of demented patients than normal controls (Jacoby et al, 1983). Many biochemical changes associated with ageing are similar to the pathophysiological abnormalities in depression and in dementia (Alexopoulos et al, 1988). These changes, coupled with the high frequency of depressive syndromes in demented pati

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