Academic literature on the topic 'Minks – Reproduction – Endocrine aspects'

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Journal articles on the topic "Minks – Reproduction – Endocrine aspects"

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Klowden, Marc J. "Endocrine aspects of mosquito reproduction." Archives of Insect Biochemistry and Physiology 35, no. 4 (1997): 491–512. http://dx.doi.org/10.1002/(sici)1520-6327(1997)35:4<491::aid-arch10>3.0.co;2-5.

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Gilsanz, Paola, Catherine Lee, Maria M. Corrada, Claudia H. Kawas, Charles P. Quesenberry, and Rachel A. Whitmer. "Reproductive period and risk of dementia in a diverse cohort of health care members." Neurology 92, no. 17 (March 28, 2019): e2005-e2014. http://dx.doi.org/10.1212/wnl.0000000000007326.

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ObjectiveWomen have >50% greater lifetime risk of dementia than men but the role of female-specific endocrine milieu is not well-understood. This study evaluates associations between indicators of estrogen exposure from women's reproductive period and dementia risk in a large diverse population.MethodsWe evaluated 15,754 female members (29.9% nonwhite) of Kaiser Permanente with clinical examinations and health survey data from 1964 to 1973 and were members as of January 1, 1996. In midlife (mean age 51.1 years), women reported age at menarche and menopause and hysterectomy status. Reproductive span was calculated as menopause age minus menarche age. Dementia diagnoses were abstracted from January 1, 1996 to September 30, 2017 medical records (mean age at start of dementia follow-up 76.5 years). Cox proportional hazard models evaluated associations between aspects of reproductive span and dementia risk adjusting for demographics and life course health indicators.ResultsForty-two percent of women developed dementia. Compared to menarche at age 13.0 (mean menarche age), menarche at ≥16 was associated with 23% greater dementia risk (adjusted hazard ratio [HR] 1.23; 95% confidence interval [CI] 1.01–1.50) adjusting for demographics and life course health indicators. Natural menopause at age <47.4 (mean menopause age) was associated with 19% elevated dementia risk (HR 1.19; 95% CI 1.07–1.31). Reproductive spans <34.4 years (mean duration) were associated with 20% elevated dementia risk (HR 1.20; 95% CI 1.08–1.32). Hysterectomies were associated with 8% elevated dementia risk (HR 1.08; 95% CI 1.01–1.16).ConclusionIn this large prospective cohort study, endocrine events signaling less estradiol exposure (i.e., later age at menarche, younger age at menopause, shorter reproductive span, and hysterectomies) were associated with elevated risk of dementia.
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Tomatis, Veronica, Christian Battipaglia, and Alessandro D. Genazzani. "Thyroid, Adrenal, PRL Impairments and Ovarian Function." Endocrines 2, no. 3 (July 27, 2021): 212–25. http://dx.doi.org/10.3390/endocrines2030021.

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Endocrine axes (prolactin, thyroid and adrenal axes) directly and indirectly modulate and drive human female central functions, mainly behavior and reproduction. Though having distinct abilities, they greatly act both at peripheral as well as at neuroendocrine levels, so as to participate in the control of reproduction. Any event that changes these balanced activities produces specific peripheral signals that induce abnormal functions centrally, thus triggering menstrual disorders such as oligomenorrhea or amenorrhea. It is clear that the knowledge of the relationships that exist between the different endocrine axes becomes essential for the choice of therapeutical approach. This review aims to focus on the main aspects of the physiopathology of the endocrine diseases that might be at the basis of that interference with female reproductive capacity.
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Chetrite, Gérard S., and Bruno Fève. "Preface to special issue on Peripheral and Central Control of Human Reproduction: Endocrine Aspects – Part 1." Hormone Molecular Biology and Clinical Investigation 24, no. 1 (January 1, 2015): 1–3. http://dx.doi.org/10.1515/hmbci-2015-0059.

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Chetrite, Gérard S., and Bruno Fève. "Preface to special issue on Peripheral and Central Control of Human Reproduction: Endocrine Aspects – Part 2." Hormone Molecular Biology and Clinical Investigation 25, no. 1 (January 1, 2016): 1–3. http://dx.doi.org/10.1515/hmbci-2016-0011.

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De Rensis, Fabio, Giorgio Morini, Irina Garcia-Ispierto, and Fernando López-Gatius. "To What Extent Does Photoperiod Affect Cattle Reproduction? Clinical Perspectives of Melatonin Administration – A Review." Annals of Animal Science 20, no. 3 (July 1, 2020): 797–809. http://dx.doi.org/10.2478/aoas-2020-0012.

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AbstractThe seasonality of reproduction in most mammals is dictated by photoperiod, temperature and nutrition. Melatonin, mainly synthesized in the pineal gland, is generally accepted as the active mediator of photoperiod responses including reproduction. While non-pregnant heifers and cows show continuous sexual activity and are therefore not seasonal breeders, it has been suggested that photo-periodicity may influence the appearance of puberty in heifers and the onset of parturition. Further, the light/dark ratio may influence endocrine patterns of gestation and a shorter light period correlates with the incidence of twin pregnancies. This review considers specific aspects of the effects of photoperiod and melatonin on reproduction in dairy cattle and discusses the clinical applications of melatonin.
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Santos, Jadson Pinheiro, Simone de Jesus Melo Almeida, Claryce Cunha Costa, Erick Cristofore Guimarães, Erivânia Gomes Teixeira, and Raimunda Nonata Fortes Carvalho-Neta. "Reproductive aspects of freshwater fishes exposed to pesticide-contamined environments: A systematic review." Revista Brasileira de Gestão Ambiental e Sustentabilidade 8, no. 19 (2021): 1155–68. http://dx.doi.org/10.21438/rbgas(2021)081935.

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Fish are the predominant group of vertebrates in aquatic environments, and they are directly affected by the presence of contaminants in water bodies, which undergo changes in the structure and function of communities and dynamics of the populations. The aim of the present study was to conduct a systematic review of studies that analyze the reproductive aspects of freshwater teleosts in environments contaminated by pesticides. A systematic search for scientific articles was carried out from 2000 to 2019 using the "Web of Science" database, using the descriptors "fish reproduction", "contamination", "toxicology", "ecotoxicity" and "biomonitoring", combined with the use of boolean operators "AND" and "OR". The content analysis was performed based on the titles and the summary, with a first screening. Then, 98 articles were read in full, with 40 being selected. 24 species of fish were recorded, with a predominance of Danio rerio (17.5%). Insecticides were present in 78% of the studies, mainly Endosulfan (35%) and Cypermethrin (13%). Reproductive endocrine disorders were the most reported pathways of action (57.5%). The effects of pesticides on the reproduction of freshwater teleosts are evident, requiring studies on structural changes in gonads and a decrease in the quality of gametes.
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Whitlock, BK, JA Daniel, RR Wilborn, TH Elsasser, JA Carroll, and JL Sartin. "Comparative Aspects of the Endotoxin- and Cytokine-Induced Endocrine Cascade Influencing Neuroendocrine Control of Growth and Reproduction in Farm Animals." Reproduction in Domestic Animals 43 (July 2008): 317–23. http://dx.doi.org/10.1111/j.1439-0531.2008.01180.x.

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Meier, Ursula, and Axel M. Gressner. "Endocrine Regulation of Energy Metabolism: Review of Pathobiochemical and Clinical Chemical Aspects of Leptin, Ghrelin, Adiponectin, and Resistin." Clinical Chemistry 50, no. 9 (September 1, 2004): 1511–25. http://dx.doi.org/10.1373/clinchem.2004.032482.

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Abstract Background: Recent studies point to the adipose tissue as a highly active endocrine organ secreting a range of hormones. Leptin, ghrelin, adiponectin, and resistin are considered to take part in the regulation of energy metabolism. Approach: This review summarizes recent knowledge on leptin and its receptor and on ghrelin, adiponectin, and resistin, and emphasizes their roles in pathobiochemistry and clinical chemistry. Content: Leptin, adiponectin, and resistin are produced by the adipose tissue. The protein leptin, a satiety hormone, regulates appetite and energy balance of the body. Adiponectin could suppress the development of atherosclerosis and liver fibrosis and might play a role as an antiinflammatory hormone. Increased resistin concentrations might cause insulin resistance and thus could link obesity with type II diabetes. Ghrelin is produced in the stomach. In addition to its role in long-term regulation of energy metabolism, it is involved in the short-term regulation of feeding. These hormones have important roles in energy homeostasis, glucose and lipid metabolism, reproduction, cardiovascular function, and immunity. They directly influence other organ systems, including the brain, liver, and skeletal muscle, and are significantly regulated by nutritional status. This newly discovered secretory function has extended the biological relevance of adipose tissue, which is no longer considered as only an energy storage site. Summary: The functional roles, structures, synthesis, analytical aspects, and clinical significance of leptin, ghrelin, adiponectin, and resistin are summarized.
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Rojas, Joselyn, Mervin Chávez-Castillo, Luis Carlos Olivar, María Calvo, José Mejías, Milagros Rojas, Jessenia Morillo, and Valmore Bermúdez. "Physiologic Course of Female Reproductive Function: A Molecular Look into the Prologue of Life." Journal of Pregnancy 2015 (2015): 1–21. http://dx.doi.org/10.1155/2015/715735.

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The genetic, endocrine, and metabolic mechanisms underlying female reproduction are numerous and sophisticated, displaying complex functional evolution throughout a woman’s lifetime. This vital course may be systematized in three subsequent stages: prenatal development of ovaries and germ cells up untilin uteroarrest of follicular growth and the ensuing interim suspension of gonadal function; onset of reproductive maturity through puberty, with reinitiation of both gonadal and adrenal activity; and adult functionality of the ovarian cycle which permits ovulation, a key event in female fertility, and dictates concurrent modifications in the endometrium and other ovarian hormone-sensitive tissues. Indeed, the ultimate goal of this physiologic progression is to achieve ovulation and offer an adequate environment for the installation of gestation, the consummation of female fertility. Strict regulation of these processes is important, as disruptions at any point in this evolution may equate a myriad of endocrine-metabolic disturbances for women and adverse consequences on offspring both during pregnancy and postpartum. This review offers a summary of pivotal aspects concerning the physiologic course of female reproductive function.
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Dissertations / Theses on the topic "Minks – Reproduction – Endocrine aspects"

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Pryor, Andrew William. "Reproduction and Endocrine Aspects of Early and Mid Lactation Holstein Cows." Thesis, Virginia Tech, 2002. http://hdl.handle.net/10919/32486.

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This study was designed to determine the effects of stage of lactation and subsequent energy status on metabolic and endocrine measures, follicular development, and the quality of oocytes obtained from Holstein cows. Holstein cows were selected prior to calving and assigned to the early lactation (EL) group (n=8) while, cows at d 90 postpartum were selected for the mid-lactation (ML) group (n=7). Blood samples were taken twice weekly from 4 wk prior to the start of follicular aspirations and then on through the aspiration periods for metabolite and hormone determination. Ultrasound-guided transvaginal follicular aspiration (TVFA) was conducted twice weekly for a 10-wk period on all cows. Follicular fluid samples were obtained from the largest follicle, > 10 mm in diameter, for hormone determination. All data were analyzed by ANOVA, using the general linear model procedures. Mean energy balance was positive for (2.43 ± 0.32 Mcal/kg) for ML cows and negative (-1.55 ± 0.33 Mcal/kg) for EL cows. In ML cows serum progesterone (P4) decreased rapidly from 2.7 ± 0.1 ng/ml at the first aspiration session to a nadir of 0.33 ± 0.1 ng/ml at wk 8, while follicular fluid P4 increased from 0.9 ± 0.5 to 5.6 ± 0.5 ng/ml. In the EL cows serum and follicular fluid P4 remained relatively constant over the course of aspirations. There was a linear increase in follicular fluid insulin-like growth factor I (IGF-I) for EL and ML cows, however the increase was more rapid for ML cows (159 ± 36 to 200 ± 36 ng/ml) than for EL cows (145 ± 36 to 164 ± 36 ng/ml). Over the aspiration period nonesterified fatty acids (NEFA) declined rapidly for the EL cows (0.32 ± 0.2 to 0.22 ± 0.2 mEq/L), while serum NEFA for the ML cows were relatively stable (0.19 ± 0.2 to 0.22 ± 0.2 mEq/L). The number of follicles observed during the aspiration sessions increased linearly for both EL and ML cows (P < 0.05) over the 10-wk period. However, the increase was larger for the ML cows than for the EL cows, going from 14.2 ± 0.5 to 18.1 ± 0.5 and 14.9 ± 0.3 to 15.7 ± 0.5, respectively. These results show that cows in early lactation are physiologically under more production stress than cows in mid lactation. Furthermore, increasing levels of serum and follicular fluid IGF-I in mid lactation may reflect differences in follicle and oocyte measures.
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Melnyk, Peter M. (Peter Michael). "Estrogen regulation of testicular function in the adult ram." Thesis, McGill University, 1989. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=59414.

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During the nonbreeding season (July), three groups of five Dorset x Leicester x Suffolk rams were assessed over a period of 5 days. One group of rams (control) was implanted (sc) with five 5cm empty Silastic capsules (i.d. 3.4mm, o.d. 4.6mm); two other groups, designated as Low-E$ sb2$ and High-E$ sb2$, received five estradiol filled capsules of either 5cm or 10cm, respectively for 4 days. Estradiol treatment elevated serum estradiol concentration about 150% in the Low-E$ sb2$ groups (15.7 $ pm$ 1.3 pg/ml) and 300% in the High-E$ sb2$ groups (26.6 $ pm$ 2.4 pg/ml) compared with controls (6.3 $ pm$ 0.8 pg/ml). In the absence of LH pulsing, mean LH, FSH and testosterone concentrations were all decreased significantly (P $<$.05) with increasing estradiol concentration, while PRL concentration was increased (P $<$.05) by as much as 105%. In the LH-pulsed groups, LH-peak height on day 4 was comparable for all three groups of rams and peak frequency was, as expected, consistently increased to 4 peaks per 6 hours. The increase in mean testosterone concentration (P $<$.05) in all three groups was due to an increase in testosterone baseline concentration and testosterone peak frequency.
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Hamudikuwanda, Humphrey. "Endocrine and metabolic mediators of dietary energy status and reproduction in dairy cows." Thesis, McGill University, 1995. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=28770.

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Five experiments were undertaken to identify metabolites and hormones that could mediate the effect of dietary energy status on reproduction, particularly pulsatile secretion of luteinizing hormone (LH) postpartum dairy cows.
In the first two experiments, the concentration of progesterone (P4) in tailhead adipose tissue and plasma in 12 cows at different stages of pregnancy and lactation was determined as was P4 produced in vitro by explants of tailhead adipose tissue. Concentration of P4 in adipose tissue was correlated with that of plasma P4 near estrus and during the luteal phase of the estrous cycle, and P4 was released in vitro by fat mobilization.
In the third and fourth experiments, blood was collected continuously for 16 h from four ovariectomized cows offered maintenance or restricted energy diets after priming with P4 or estradiol (E2) using a crossover experimental design. The results indicated that P4 released during body fat mobilization is minor and is not related to LH secretion. Dietary energy restriction influenced plasma LH concentration and pulse amplitude but the effect was modulated by P4 and E2 priming. Dietary energy restriction decreased glucose concentration but did not influence plasma non-esterified fatty acids (NEFA), cortisol, P4 and insulin levels. Cortisol was negatively related to LH pulse frequency. Glucose and insulin were positively and negatively correlated with LH pulse amplitude, respectively. Cortisol, NEFA and glucose jointly had a negative correlation with LH concentration.
In the fifth experiment, blood samples were collected daily for 60 d and every 10 min for 8 h on 18, 36 and 54 d postpartum from 24 cows (12 ovariectomized) fed low (1.4 Mcal/kg DM) (L) or high (1.7 Mcal/kg DM) (H) energy in a 2 x 2 factorial treatment design. LH pulse frequency was reduced at 18 d postpartum in ovariectomized cows, but not in intact cows, fed L. First postpartum ovulation occurred later in intact cows fed L compared to those fed H. Energy balance and plasma glucose concentration were lower, but plasma NEFA, $ beta$-hydroxybutyrate (BHB) and E2 concentrations higher, in cows fed L compared to those fed H. E2 concentration in intact cows fed L was elevated for a prolonged period prior to first ovulation. Diet had no influence on plasma P4 and insulin concentrations. Plasma E2 and BHB concentrations were positively correlated with LH pulse frequency in intact cows across diets and ovariectomized cows fed L, respectively. NEFA were negatively correlated with LH pulse amplitude in ovariectomized cows fed L. Glucose, NEFA and P4 were negatively, but BHB, E2 and insulin positively correlated, individually or in association, with LH concentration.
Overall, the results suggest that the effect of dietary energy status on LH patterns and timing of onset of postpartum ovulation is modulated by priming with or presence of ovarian steroids. The relationships of metabolites and hormones with LH patterns appear to change with dietary energy level, ovarian status and mutual associations among the metabolites and hormones. These parameters, especially glucose and BHB, may be potential mediators of the effect of dietary energy status on LH patterns. (Abstract shortened by UMI.)
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Cassidy, Carrie. "Further evidence that prostaglandin F2-alpha is the obligatory eicosanoid in porcine ovulation." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape11/PQDD_0004/MQ44139.pdf.

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Zhang, Song. "Peripheral and central pathways linking metabolic status and reproduction in male sheep." University of Western Australia. School of Animal Biology, 2005. http://theses.library.uwa.edu.au/adt-WU2005.0037.

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[Truncated abstract] Reproductive activity is affected by external factors such as photoperiod, social cues, stress and nutrition, all of which can alter the pulsatile activity of the GnRH neurons, which is the major neuroendocrine system used by the brain to control gonadal function. In the male Merino sheep, nutrition is one of the most powerful factors that affect pulsatile LH secretion, used commonly to bioassay GnRH neuronal activity. More accurately, the reproductive system responds to “metabolic status”, rather than “nutrition”, and the three factors that contribute to metabolic status are food intake, the amount of body reserves and the rate of energy expenditure ... In this thesis, I tested the general hypothesis that the metabolic hormones and hypothalamic neuropeptides that are known to control food intake also mediate the effect of metabolic status on the activity of the GnRH neurons ... In conclusion, the results from my experiments provide some insight into the mechanisms by which metabolic status affects reproductive activity in male sheep. Plasma insulin, which changes with alterations in metabolic status, appears to play a critical role in the regulation of GnRH neuronal activity. The level of leptin seems to have a permissive role only in lean animals. Orexins acting via OX2 receptors could be involved in the activation of reproductive function following an acute increase in nutrition. However, the neuropeptidergic systems can not be ruled out because they might be involved in very early steps of responses to nutrition.
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Ferasyi, Teuku Reza. "Mathematical model of the reproductive endocrine system in male sheep." University of Western Australia. School of Animal Biology, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0080.

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[Truncated abstract] The activity of the reproductive endocrine axis is the result of interactions among many organs and tissues, particularly the hypothalamus, pituitary gland and gonad. However, it depends on more than the communication between anatomical structures because it is also affected by genotype, internal factors (e.g., metabolic inputs) and external factors (e.g., photoperiod, socio-sexual cues, stress, nutrition). This multifactorial complexity makes it difficult to use animal experimentation to investigate the pathways and mechanisms involved. Therefore, in this study, I have turned to mathematical modelling. The general hypothesis was that, by modelling the hormonal feedback loop that links the hypothalamus, pituitary gland and gonad, I would be able to discover the critical control points in this homeostatic system. This would allow me to inform and direct research into the processes that control reproduction, including inputs from environmental factors. My studies began with the development of a model of the negative feedback loop through which testosterone controls the secretion of pulses of gonadotrophin-releasing hormone (GnRH) by the hypothalamus. The model incorporated two critical factors: testosterone concentration and a time delay in the inhibition of the activity of the GnRH 'pulse generator' by testosterone. The general assumptions were: i) there are two positive feedforward processes (GnRH pulses stimulate LH pulses, and, in turn, LH pulses stimulate testosterone secretion); ii) testosterone exerts negative feedback that reduces the frequency of GnRH pulses. The model incorporated a group of equations that represent the GnRH pulse generator, through which the inhibitory effect of testosterone acted to reduce GnRH pulse frequency. Simulations were run with various values for the time delay in feedback and, as model development progressed, the simulations were extended to include combinations of time delays and levels of sensitivity of the GnRH pulse generator to inhibition by testosterone. The output of the simulations showed clearly that a time delay in negative feedback, as well as the concentration of testosterone, can greatly affect the frequency of GnRH pulses and the shape of the GnRH secretory profile. Importantly, the effect of the time delay depends on the sensitivity of the pulse generator to testosterone. In addition, the simulations suggested two additional components that might be involved in the control of the GnRH pulse generator: i) a delay in the rate of adaptation to a change in steroid feedback; and ii) a minimum pulse interval (maximum frequency). These studies iii therefore suggest that the regulation of the activity of the GnRH pulse generator, and thus the frequency and profile of GnRH and LH pulses, requires interactions among these four components. These interactions should be tested in animal experimentation. In the next stage, I extended the model so I could test whether the feedback delay might involve the process of aromatization in which testosterone is converted to oestradiol at brain level. ... This information can be used to direct future experimental studies that will help us to understand the factors that underlie the dynamic behaviour of the hypothalamic and pituitary systems that control reproduction.
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Cote, Fabienne. "Induction of prostaglandin endoperoxide synthase 2 in the follicles of equine chorionic gonadotropinhuman chorionic gonadotropin treated prepubertal gilts." Thesis, McGill University, 2001. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=33741.

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Prostaglandin G/H synthase-2 (PGHS-2) is a key rate limiting enzyme in the prostaglandin (PG) biosynthetic pathway, and PG synthesis is required for ovulation in pigs. The objective of this study was to characterize the expression and regulation of PGHS-2 in porcine follicles prior to ovulation. The combination of equine chorionic gonadotropin (eCG; 750 IU) followed by human chorionic gonadotropin (hCG; 500 IU) 72 h later was used to induce ovulation in prepubertal gilts. Previous studies have shown that ovulation is generally induced between 40 and 44 h post-hCG in this model. Ovariectomies were performed at 0, 24, 30, 34 and 38 h post-hCG (n = 4 or 5 animals per time-point), and all follicles larger than 4 mm in diameter were isolated. The regulation of PGHS-1 and PGHS-2 proteins was studied by immunohistochemistry and Western blot analyses, whereas the regulation of PGHS-2 mRNA was studied by Northern blot. PG production was assessed by radioimmunoassay (RIA). (Abstract shortened by UMI.)
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Algire, James Edgar. "Prostaglandins in follicular development and ovulation in cattle." Thesis, McGill University, 1989. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=61849.

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Mootoo, Judy E. (Judy Elizabeth). "Lipoxygenase metabolites of arachidonic acid in the porcine ovulatory process." Thesis, McGill University, 1994. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=22779.

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It is widely accepted that prostaglandins (PGs), produced via the cyclooxygenase pathway from arachidonic acid, are essential to the ovulatory process in the pig. In support of this, ovulation is preceded by an increase in follicular fluid (FF) PG concentration, indomethacin (INDO) suppresses both the PG increase and ovulation, and ovulation can be restored by administration of exogenous PGs (Downey and Ainsworth, 1980; Prostaglandins 19: 17-22). Recent studies in the rat have shown that ovulation is also preceded by a rise in ovarian concentrations of 15-hydroxyeicosatetraenoic acid (15-HETE), a product of the lipoxygenase pathway (Tanaka et al., 1989; Endocrinology 15: 1373-1377) and inhibition of this pathway suppresses ovulation (Reich et al., 1983; Prostaglandins 26: 1011-1020). Furthermore, INDO, a cyclooxygenase inhibitor, inhibits 15-lipoxygenase as well as PG synthesis (Tanaka et al., 1989 Endocrinology 15: 1373-1377). The PMSG/hCG prepuberal gilt model was used to investigate the involvement of 15-HETE in the procine ovulatory process, and the effect of INDO on the 15-lipoxygenase pathway. Follicular fluid concentrations of 15-HETE were elevated 40 h post hCG (p $<$ 0.01). The effects of INDO and nordihydroguaiaretic acid (NDGA), an inhibitor of lipoxygenase activity, on ovulation rate, FF 15-HETE and FF PGF$ rm sb{2a}$ were investigated by intraovarian administration of INDO or NDGA. INDO inhibited ovulation rate (p $<$ 0.01) and PGF$ rm sb{2a}$ (p $<$ 0.01) as well as 15-HETE (p $<$ 0.01). NDGA also suppressed ovulation rate (p $<$ 0.01) but did not inhibit 15-HETE or PGF$ rm sb{2a}$ production. In in vitro experiments, 15-HETE production by both granulosa cell (GC) and theca interna cell (TIC) cultures 40 h post hCG was greater (p $<$ 0.01) than at 0 h post hCG. INDO inhibited 15-HETE production in 40 h post hCG TIC cultures (p $<$ 0.01) but not GC cultures, while NDGA inhibited 15-HETE production by both cell types (p $<$ 0.01). These results sugges
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Grant, Gerald F. "The association between prostaglandins and the plasminogen activator/plasmin system in the porcine ovulatory process /." Thesis, McGill University, 1993. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=69759.

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The objectives were: (1) to determine the pre-ovulatory changes in plasminogen activator (PA) and (PA) inhibitor (PAI) activities in the porcine follicle, and, (2) to determine if changes in the PA/plasmin system associated with ovulation were prostaglandin (PG)-dependent. PA activity (change in absorbance/h/mg wet tissue weight, three gilts per treatment group) was elevated in both granulosa cells (GC) and theca interna cells (TIC) prior to human chorionic gonadotropin (hCG) administration (0.582 $ pm$ 0.171 and 0.718 $ pm$ 0.221, respectively) but returned to basal levels in these two compartments (0.023 $ pm$ 0.013 and 0.052 $ pm$ 0.024, respectively) at 29 h post-hCG. PA activity remained basal thereafter in GC but increased approximately ten-fold in the TIC (0.549 $ pm$ 0.239) at the time of ovulation (three gilts at 41 h and one of three gilts at 38 h). PAI activity did not change in TIC over the pre-ovulatory period but increased in GC as ovulation approached. PAI activity in GC peaked at 38 h (being significantly different (p $<$ 0.05) to all other times except 41 h). Although indomethacin (INDO) effectively inhibited both PG synthesis (1.1 $ pm$ 0.2 vs. 9.2 $ pm$ 0.9 ng/ml in controls) and ovulation (0 vs. 27-61% in controls), elevated PA activity (0.801 and 0.349) was detected in the TIC of two out of nine INDO-treated gilts. Levels were basal (0.074 $ pm$ 0.028) in the other gilts. These inconclusive results are believed to reflect the occurrence of ovulation earlier than predicted, in as many as 40% of control gilts, and the short duration of increased PA activity at this time. In conclusion, elevated PA activity, in GC and TIC prior to ovulation induction, may play a role in follicular development. Elevated TIC PA activity may play an important role in the ovulatory process, but is probably PG-independent.
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Books on the topic "Minks – Reproduction – Endocrine aspects"

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Agadzhani︠a︡n, N. A. Stress, adaptat︠s︡ii︠a︡, reproduktivnai︠a︡ sistema: Monografii︠a︡. Nizhniĭ Novgorod: Nizhegorodskai︠a︡ gos. medit︠s︡inskai︠a︡ akademii︠a︡, 2009.

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Fellows Conference (1992 Tucson, Ariz.). A Fellows Conference, Tucson, Arizona, January 17 and 18, 1992: Proceedings of a Fellows Conference. [Birmingham, Ala.]: American Fertility Society, 1992.

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A, Aruti͡u︡ni͡a︡n N., and Stepanov M. G, eds. Ėksperimentalʹnoe besplodie: Ėndokrinologicheskie aspekty. Sankt-Peterburg: "Nauka," S.-Peterburgskoe otd-nie, 1992.

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R, Olson Erik, and Naugle Jennifer E, eds. Endocrine and reproductive systems. Philadelphia: Elsevier Mosby, 2006.

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Carrell, Douglas T., and C. Matthew Peterson. Reproductive endocrinology and infertility: Integrating modern clinical and laboratory practice. New York: Springer, 2010.

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Shinkōkyoku, Japan Monbu Kagakushō Kenkyū. Naibunpitsu kakuran busshitsu ni yoru seishoku e no eikyō to sono sayō kikō ni kansuru kenkyū (Heisei 10--12-nendo): Seika hōkokusho. [Tokyo]: Monbu Kagakushō Kenkyū Shinkōkyoku, 2001.

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1924-, Iizuka Rihachi, Semm K. 1927-, and International Academy of Human Reproduction., eds. Human reproduction: Current status/future prospect : proceedings of the VIth World Congress on Human Reproduction, held in Tokyo, Japan, 25-30 October 1987. Amsterdam: Excerpta Medica, 1988.

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French-Israeli Symposium (1986 Tel Aviv, Israel). Reproduction in fish: Basic and applied aspects in endocrinology and genetics : proceedings of the French-Israeli Symposium, Tel-Aviv (Israel), 10-12 November 1986. Edited by Zohar Y and Breton B. Paris: Institut national de la recherche agronomique, 1988.

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Odent, Michel. The functions of the orgasms: The highways to transcendence. London: Pinter & Martin, 2009.

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Odent, Michel. The functions of the orgasms: The highways to transcendence. London: Pinter & Martin, 2009.

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Book chapters on the topic "Minks – Reproduction – Endocrine aspects"

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Pivonello, Claudia, Cristina de Angelis, Francesco Garifalos, Rosario Pivonello, and Annamaria Colao. "Environmental Factors’ Interference in Endocrine Aspects of Male Reproduction." In Beyond Our Genes, 37–51. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-35213-4_3.

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"Reproduction and development." In Oxford Assess and Progress: Medical Sciences, edited by Jade Chow, John Patterson, Kathy Boursicot, and David Sales. Oxford University Press, 2012. http://dx.doi.org/10.1093/oso/9780199605071.003.0025.

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Abstract:
Reproduction and development are large topics, knowledge of which underpins several medical specialities including sexual health, fertility, gynaecology, urology, reproductive endocrinology, obstetrics, and neonatology. Doctors need to know the structure, function, and endocrine control of both male and female systems in order to diagnose and manage conditions specific to either male or female organs, as well as conditions such as impotence and infertility. Not surprisingly, the reproductive system is the only body system that shows major differences in both structure and function between males and females. However, sexual differences go beyond the primary sexual characteristics present at birth and the secondary sexual characteristics that emerge under the influence of sex hormones at puberty. Sexual dimorphism in some brain structures commences at an early age, and differences in the endocrine profiles of males and females produce characteristic changes in morphology, physiology, and behaviour that go beyond simple sexual dimorphism to affect many aspects of life, including sexual differences in susceptibility to disease and the longer life expectancy of women as compared to men that is seen around the world. Whether these differences, mainly beneficial to women, are because females are ‘biologically superior’ or because of a complex mix of genetic, behavioural, and social factors is a matter for discussion and research. Some knowledge of embryology is important to every medical student. As a minimum it provides explanations for the congenital malformations and their consequences that are encountered in many areas of clinical practice. Deeper knowledge will assist those seeking real insights into the structure of the human body. It is the study of embryological development and the knowledge of how each tissue type arises, how one tissue meets another, and how tissues move and change shape during development that explains the relations between tissues and organs in the adult human form. Achieving a full understanding of the dynamics of the formation of the body’s organs and tissues is demanding, but it can replace some of the rote learning of anatomical structures, familiar to many students, with a deeper understanding of form and function.
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