Relevant bibliographies by topics / Minor blood antigens

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Minor blood antigens'

Author: Grafiati
Published: 4 June 2021
Last updated: 7 February 2022

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Journal articles on the topic "Minor blood antigens"

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Bleakley, Marie, Brith E. Otterud, Julia L. Richardt, et al. "Leukemia-associated minor histocompatibility antigen discovery using T-cell clones isolated by in vitro stimulation of naive CD8+ T cells." Blood 115, no. 23 (2010): 4923–33. http://dx.doi.org/10.1182/blood-2009-12-260539.

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Abstract T-cell immunotherapy that targets minor histocompatibility (H) antigens presented selectively by recipient hematopoietic cells, including leukemia, could prevent and treat leukemic relapse after hematopoietic cell transplantation without causing graft-versus-host disease. To provide immunotherapy that can be applied to a majority of transplantation recipients, it is necessary to identify leukemia-associated minor H antigens that result from gene polymorphisms that are balanced in the population and presented by common human leukocyte antigen alleles. Current approaches for deriving mi
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Verdijk, Rob M., Antoinette Kloosterman, Jos Pool, et al. "Pregnancy induces minor histocompatibility antigen–specific cytotoxic T cells: implications for stem cell transplantation and immunotherapy." Blood 103, no. 5 (2004): 1961–64. http://dx.doi.org/10.1182/blood-2003-05-1625.

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AbstractRecipients of HLA-identical stem cell transplants have a poorer transplant outcome if the donor is female rather than male. We analyzed whether pregnancy primes for minor histocompatibility (H) antigens. Peripheral blood mononuclear cells (PBMCs) from healthy multiparous female blood donors were depleted for CD4+, CD14+, CD16+, and CD19+ cells, stained with minor H antigen–specific HLA-A2 tetramers, sorted by fluorescence-activated cell sorting, and tested for cytotoxic activity. Minor H antigens HY-, HA-1–, and HA-2–specific cytotoxic T cells (CD8+, CD45RA–) were present in PBMCs from
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Roy, Denis Claude, and Claude Perreault. "Major vs minor histocompatibility antigens." Blood 129, no. 6 (2017): 664–66. http://dx.doi.org/10.1182/blood-2016-12-754515.

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Silvey, Michael, Rachel Beddard, Yidong Chen, and Melissa Frei-Jones. "Frequency of Minor Blood Group Antigens Among Hispanic-American Blood Donors in Southwest Texas." Blood 114, no. 22 (2009): 4188. http://dx.doi.org/10.1182/blood.v114.22.4188.4188.

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Abstract Abstract 4188 The frequency of minor blood group antigens has not been described among Hispanic blood donors. A retrospective cohort study was performed using the South Texas Blood and Tissue Center (STBTC) donor database, LifeTrak. Blood donors self-identified race and ethnicity. Caucasian and Hispanic donors were eligible. Donors who did not have minor antigen testing performed were not included. The STBTC has a high proportion of Hispanic blood donors (299047/779409, 38%), who are primarily of Mexican ancestry. Minor antigen testing was performed in 10% (74140/779409) of all donors
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Lamba, Divjot Singh, Ravneet Kaur, and Sabita Basu. "Clinically Significant Minor Blood Group Antigens amongst North Indian Donor Population." Advances in Hematology 2013 (2013): 1–5. http://dx.doi.org/10.1155/2013/215454.

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Background. Racial differences in blood group antigen distribution are common and may result in striking and interesting findings. These differences in blood group antigen distribution are important due to their influence on the clinical practice of transfusion medicine.Study Design and Methods. This is a prospective study, involving 1000 healthy regular repeat voluntary blood donors associated with the department. The clinically significant minor blood group antigens of these donors were studied.Results. Out of 1000 healthy regular repeat voluntary blood donors, 93% were D positive and 2.8% w
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Kumar, Mukesh, Palmer Shelia, Annie Marie Winkler, and James R. Eckman. "Minor Blood Group Antigen Disparities in Sickle Cell Disease Population." Blood 120, no. 21 (2012): 4381. http://dx.doi.org/10.1182/blood.v120.21.4381.4381.

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Abstract Abstract 4381 Introduction: Red blood cell (RBCs) transfusion is the mainstay of treatment for sickle cell disease (SCD) patients and indications continue to expand. Differences in geographic origin between blood donors and sickle recipients has been posited as a cause of the high rate of alloantibodies in patients with sickle cell disease (Vichinsky et al. NEJM 1990; 322: 1617–21). Alloimmunization is one of the major limiting factors of obtaining compatible blood for transfusion and exchange transfusion compromising the management of patients with SCD. Mismatch in minor antigen dist
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Veerapathran, Anandharaman, Joseph Pidala, Francisca Beato, William E. Janssen, Xue-Zhong Yu, and Claudio Anasetti. "Tregs Specific for Minor Histocompatibility Antigens." Blood 120, no. 21 (2012): 1891. http://dx.doi.org/10.1182/blood.v120.21.1891.1891.

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Abstract Abstract 1891 Background: The risk of acute GVHD after HSCT is increased in male recipients of female grafts. Disparities for the male-associated H-Y and other minor histocompatibility antigens (mHAs) have the capacity to sensitize alloreactive donor T cells and cause GVHD in HLA-matched recipients. These mHAs are polymorphic proteins that differ between donor and recipient and are presented as peptides by HLA molecules on recipient or donor antigen-presenting cells to donor immune cells. Currently, there is no evidence that minor histocompatibility antigen specific Tregs exist. Earli
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van Halteren, Astrid G. S., Ewa Jankowska-Gan, Antoinette Joosten, et al. "Naturally acquired tolerance and sensitization to minor histocompatibility antigens in healthy family members." Blood 114, no. 11 (2009): 2263–72. http://dx.doi.org/10.1182/blood-2009-01-200410.

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Abstract Bidirectional cell transfer during pregnancy frequently leads to postpartum persistence of allogeneic cells and alloimmune responses in both the mother and in her offspring. The life-long consequences of naturally acquired alloimmune reactivity are probably of importance for the outcome of allogeneic stem cell transplantation. We investigated the presence of CD8pos minor histocompatibility (H) antigen–specific cytotoxic T lymphocytes (TCTL) and CD8pos minor H antigen–specific T regulator cells (TREG) in peripheral blood cells obtained from 17 minor H antigen–disparate mother-offspring
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Kawase, Takakazu, Yasuhito Nannya, Hiroki Torikai, et al. "Identification of human minor histocompatibility antigens based on genetic association with highly parallel genotyping of pooled DNA." Blood 111, no. 6 (2008): 3286–94. http://dx.doi.org/10.1182/blood-2007-10-118950.

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Abstract Minor histocompatibility (H) antigens are the molecular targets of allo-immunity responsible both for the development of antitumor effects and for graft-versus-host disease (GVHD) in allogeneic hematopoietic stem cell transplantation (allo-HSCT). However, despite their potential clinical use, our knowledge of human minor H antigens is largely limited by the lack of efficient methods of their characterization. Here we report a robust and efficient method of minor H gene discovery that combines whole genome association scans (WGASs) with cytotoxic T-lymphocyte (CTL) assays, in which the
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Propper, D. J., M. C. Jones, K. N. Stewart, G. R. D. Catto, and D. A. Power. "Cyclosporin A and the humoral response to blood transfusion in the rat: definition of antigens which suppress alloantibody formation." Clinical Science 80, no. 1 (1991): 9–15. http://dx.doi.org/10.1042/cs0800009.

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1. Blood transfusions improve renal allograft survival rates, but may induce antibodies which are directed to class I major histocompatibility complex antigens and mediate hyperacute transplant rejection. A model to study the development of such antibodies was developed in inbred strains of rats. 2. The influence of transplantation antigens shared between an initial course of blood transfusions, given with cyclosporin A, and a subsequent antigenic challenge (blood transfusion), given without cyclosporin A, on alloantibody responses to class I major histocompatibility complex antigens was then
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Dissertations / Theses on the topic "Minor blood antigens"

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Mongrain, Ian. "Development of a Blood Antigen Molecular Profiling Panel using Genotyping Technologies for Patients Requiring Frequent Transfusions." Thèse, 2009. http://hdl.handle.net/1866/3541.

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Contexte. Les phénotypes ABO et Rh(D) des donneurs de sang ainsi que des patients transfusés sont analysés de façon routinière pour assurer une complète compatibilité. Ces analyses sont accomplies par agglutination suite à une réaction anticorps-antigènes. Cependant, pour des questions de coûts et de temps d’analyses faramineux, les dons de sang ne sont pas testés sur une base routinière pour les antigènes mineurs du sang. Cette lacune peut résulter à une allo-immunisation des patients receveurs contre un ou plusieurs antigènes mineurs et ainsi amener des sévères complications pour de futures
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Book chapters on the topic "Minor blood antigens"

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Colin, Yves, and Caroline Le Van Kim. "Gerbich Blood Groups and Minor Glycophorins." In Molecular Basis of Human Blood Group Antigens. Springer US, 1995. http://dx.doi.org/10.1007/978-1-4757-9537-0_12.

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Conference papers on the topic "Minor blood antigens"

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Shainoff, John R., Youko Hishikawa-Itoh, Fred M. Lucas, Robert Graor, and Bernadine Healy. "IMMUNOELECTROPHORETIC CHARACTERIZATION OF SYSTEMIC FIBRINOGEN DURING THROMBOLYSIS OF PERIPHERAL ARTERIAL EMBOLI WITH tPA." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643889.

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Prompted by uncertainties over the possibility of ex-vivo alterations of fibrinogen in plasma samples from patients receiving thrombolytic agents, we sought to 1) use sodium dodecyl sulfate (SDS) and mild acidification to pH 5 as a general means for inhibiting post-sampling proteolysis, and 2) assess the chemical state of fibrinogen by immuno-electrophoretically profiling the molecular weight distribution of the fibrinogen-related antigens in the sample. Blood samples taken into EDTA and PPACK were immediately centrifuged, and plasma diluted 5X in 2% SDS and 0.04% monochloroacetic acid. Such s
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Gunda, Naga Siva Kumar, and Sushanta K. Mitra. "Microfluidic Based Biosensor for Detection of Cardiac Markers." In ASME 2013 Fluids Engineering Division Summer Meeting. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/fedsm2013-16270.

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Myocardial Infarction (MI) occurs when the blood flow to the heart is blocked. It is major threat to human kind. Current laboratory and ELISA tests are expensive, time consuming, and are not very sensitive. Biosensors can play an important role in the diagnosis of MI without relying on hospital visits. Therefore, researchers are focusing to develop rapid, hand-held, inexpensive biosensors for detecting cardiac markers. In the present study, one of the cardiac markers (Troponin T) is detected using microfluidic based biosensor. Troponin T (cTnT) releases in to the blood serum within 4–6 h after
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Giddings, J. C. "AN IMMUNORADIOMETRIC ASSAY (IRMA) FOR HUMANTHROMBOMODULIN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643963.

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Thrombomodulin was separated from detergent-soluble fractions of human placenta using a combination of DEAE-Sepharose and thrombin-Sepharose chromatography. The final product demonstrated one major band (Mr approximately 100000) and three minor bands (Mr 40000 - 80000) on SDS-polyacrylamide gel electrophoresis. The purified protein markedly enhanced the rate of activation of human protein C by thrombin in the presence of calcium ions. Polyclonal antibodies to the isolated thrombomodulin were raised in rabbits and were shown to inhibit thrombin co-factor activity. Immunofluorescence of fibrobla
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