Relevant bibliographies by topics / MiR17-92

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  2. Dissertations / Theses
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  4. Conference papers

Academic literature on the topic 'MiR17-92'

Author: Grafiati
Published: 4 June 2021
Last updated: 15 February 2022

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Journal articles on the topic "MiR17-92"

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Huang, Yuanyuan, Hanlin Zhang, Meng Dong, et al. "miR17-92 cluster drives white adipose tissue browning." Journal of Molecular Endocrinology 65, no. 3 (2020): 97–107. http://dx.doi.org/10.1530/jme-20-0032.

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White adipose tissue (WAT) browning may have beneficial effects for treating metabolic syndrome. miRNA are important regulators of the differentiation, development, and function of brown and beige adipocytes. Here, we found that the cold-inducible miRNA17-92 cluster is enriched in brown adipose tissue (BAT) compared with WAT. Overexpression of the miR17-92 cluster in C3H10T1/2 cells, a mouse mesenchymal stem cell line, enhanced the thermogenic capacity of adipocytes. Furthermore, we observed a significant reduction in adiposity in adipose tissue-specific miR17-92 cluster transgenic (TG) mice.
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Ippolito, Thomas, Rodney R. Miles, Vladimir Rodic, Cory Mavis, Francisco J. Hernandez-Ilizaliturri, and Matthew J. Barth. "High Mir-17-92 Expression Is Associated with in Vitro chemoresistance in Burkitt Lymphoma." Blood 132, Supplement 1 (2018): 1586. http://dx.doi.org/10.1182/blood-2018-99-118932.

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Abstract Background: Copy number gain and/or MYC induction leads to increased expression of miRs in the miR-17~92 cluster. Development of Burkitt lymphoma (BL) is associated with translocation of MYC resulting in MYC expression and frequent recurrent copy number gains of chromosome 13q containing the miR-17~92 cluster of miRs. MiRs in the miR-17-92 cluster, including miR19 and miR17, have been associated with B-cell lymphomagenesis. Copy number gain of 13q and high expression of miR-17 have also been associated with possible inferior outcome in children with BL. MiR-17~92 miRs can decrease exp
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Ntoufa, Stavroula, Nikos Papakonstantinou, Christos Nikolaou, et al. "The Mir17∼92 Cluster Is an Immunomodulator in CLL Regulating Distinct Functional Responses to Toll-Like Receptors in Subsets with Stereotyped Antigen Receptors." Blood 120, no. 21 (2012): 3862. http://dx.doi.org/10.1182/blood.v120.21.3862.3862.

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Abstract Abstract 3862 Subgroups of CLL cases defined on the basis of the immunogenetic features of their B cell receptors (BcRs), especially those cases belonging to subsets with stereotyped BcRs, exhibit differential responses to immune stimulation through either the BcR or the Toll-Like Receptors (TLRs). This indicates distinct, subset-biased modalities of BcR collaboration with specific TLRs that may extend to the control of cell proliferation or apoptosis, B cell anergy and/or TLR tolerance. In both normal and CLL B cells, the final signaling outcome can be controlled by microRNAs (miRNAs
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Brandon-Warner, E., C. O. Field, C. R. Culberson, I. H. McKillop, and L. W. Schrum. "P626 PROCESSING OF miR17–92 CLUSTER IN HEPATIC STELLATE CELLS PROMOTES HEPATIC FIBROGENESIS." Journal of Hepatology 60, no. 1 (2014): S277. http://dx.doi.org/10.1016/s0168-8278(14)60788-1.

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Sawera, Milena, Jan Gorodkin, Susanna Cirera, and Merete Fredholm. "Mapping and expression studies of the mir17-92 cluster on pig Chromosome 11." Mammalian Genome 16, no. 8 (2005): 594–98. http://dx.doi.org/10.1007/s00335-005-0013-3.

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Mohan, Subburaman, Jon E. Wergedal, Subhashri Das, and Chandrasekhar Kesavan. "Conditional disruption of miR17-92 cluster in collagen type I-producing osteoblasts results in reduced periosteal bone formation and bone anabolic response to exercise." Physiological Genomics 47, no. 2 (2015): 33–43. http://dx.doi.org/10.1152/physiolgenomics.00107.2014.

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In this study, we evaluated the role of the microRNA (miR)17-92 cluster in osteoblast lineage cells using a Cre-loxP approach in which Cre expression is driven by the entire regulatory region of the type I collagen α2 gene. Conditional knockout (cKO) mice showed a 13–34% reduction in total body bone mineral content and area with little or no change in bone mineral density (BMD) by DXA at 2, 4, and 8 wk in both sexes. Micro-CT analyses of the femur revealed an 8% reduction in length and 25–27% reduction in total volume at the diaphyseal and metaphyseal sites. Neither cortical nor trabecular vol
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Li, Y., M. Lauriola, D. Kim та ін. "Adenomatous polyposis coli (APC) regulates miR17-92 cluster through β-catenin pathway in colorectal cancer". Oncogene 35, № 35 (2016): 4558–68. http://dx.doi.org/10.1038/onc.2015.522.

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Brinkmann, Kerstin, Ashley P. Ng, Carolyn A. de Graaf, et al. "miR17~92 restrains pro-apoptotic BIM to ensure survival of haematopoietic stem and progenitor cells." Cell Death & Differentiation 27, no. 5 (2019): 1475–88. http://dx.doi.org/10.1038/s41418-019-0430-6.

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Brandon-Warner, Elizabeth, Nicole A. Feilen, Catherine R. Culberson, et al. "Processing of miR17-92 Cluster in Hepatic Stellate Cells Promotes Hepatic Fibrogenesis During Alcohol-Induced Injury." Alcoholism: Clinical and Experimental Research 40, no. 7 (2016): 1430–42. http://dx.doi.org/10.1111/acer.13116.

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Xu, Z., P. P. Sharp, Y. Yao, et al. "BET inhibition represses miR17-92 to drive BIM-initiated apoptosis of normal and transformed hematopoietic cells." Leukemia 30, no. 7 (2016): 1531–41. http://dx.doi.org/10.1038/leu.2016.52.

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Dissertations / Theses on the topic "MiR17-92"

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Gapihan, Guillaume. "Etude du cluster oncogénique miR17-92 dans les lymphomes B agressifs humains." Thesis, Sorbonne Paris Cité, 2016. http://www.theses.fr/2016USPCC321.

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Les lymphomes à grandes cellules B primitifs du médiastin (PMBL) partagent des caractéristiques pathologiques avec les lymphomes diffus à grandes cellules B (DLBCL), et des caractéristiques moléculaires communes aux lymphomes de Hodgkin classiques (cHL). Le cluster oncogénique miR-17-92, localisé au niveau du chromosome 13q31, est un gène amplifié dans les DLBCL. Dans notre étude, nous avons comparé le niveau d’expression de chaque membre du clustermiR-17-92 dans une série de prélèvements de patients de 40 PMBL, 20 DLBCL et 20 cHL, et étudié les gènes cibles liés aux microARN dérégulés dans le
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Kabbout, Mohamed Nazih. "ETS1 AND ETS2 ROLE IN RAS ONCOGENIC TRANSFORMATION IN MOUSE EMBRYONIC FIBROBLASTS." The Ohio State University, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=osu1275408102.

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Books on the topic "MiR17-92"

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Reckman, Yolan J., and Yigal M. Pinto. The role of non-coding RNA/microRNAs in cardiac disease. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, et al. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0031.

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In the past two decades, our knowledge about non-coding DNA has increased tremendously. While non-coding DNA was initially discarded as ‘junk DNA’, we are now aware of the important and often crucial roles of RNA transcripts that do not translate into protein. Non-coding RNAs (ncRNAs) play important functions in normal cellular homeostasis and also in many diseases across all organ systems. Among the different ncRNAs, microRNAs (miRNAs) and long non-coding RNAs (lncRNAs) have been studied the most. In this chapter we discuss the role of miRNAs and lncRNAs in cardiac disease. We present example
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Conference papers on the topic "MiR17-92"

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Schwentner, Raphaela, Maximilian Kauer, David Herrero-Martin, and Heinrich Kovar. "Abstract 1918: The role of miR17-92 in the miRegulatory landscape of Ewing sarcoma." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-1918.

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Haws, Hillary, Hubert F. Arokium, James L. Bogenberger, et al. "Abstract 883: Alvocidib-mediated inhibition of CDK9 upregulates BIM via suppression of miR17-92." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-883.

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Yang, HY, CY Wu, and JL Huang. "56 Micromanaging lupus nephritis: mir17–92 modulates regulatory t cell activity by targeting foxp3 co-regulators." In LUPUS 2017 & ACA 2017, (12th International Congress on SLE &, 7th Asian Congress on Autoimmunity). Lupus Foundation of America, 2017. http://dx.doi.org/10.1136/lupus-2017-000215.56.

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Shapira, Iuliana, Annette Lee, Michaela Oswald, Janaki Parameswaran, Keith Sultan, and Daniel Budman. "Abstract 1843: Surgical and chemotherapy influence on circulating developmental microRNA miR17-92 and long term survival in breast and ovarian cancer patients." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-1843.

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Journal articles
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