Log in

Relevant bibliographies by topics / Misrouting / Journal articles

To see the other types of publications on this topic, follow the link: Misrouting.

Journal articles on the topic 'Misrouting'

Author: Grafiati

Published: 4 June 2025

Last updated: 23 June 2025

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'Misrouting.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

Stojanovic, Igor, and Goran Djordjevic. "In-channel misrouting suppression technique for deflection-routed networks on chip." Facta universitatis - series: Electronics and Energetics 29, no. 2 (2016): 309–23. http://dx.doi.org/10.2298/fuee1602309s.

Full text

Abstract:

Deflection routing, where port-contentions in routers are resolved by intentionally misrouting some of packets along unwanted directions instead of storing them, has been proposed as a promising approach for improving power and area efficiency of large-scale networks on chip (NoCs). However, at high network load, when packets are misrouted more frequently, the cost and energy benefits of this simple routing scheme are offset by the performance degradation. To address this problem, we propose a technique that uses small in-channel buffers to capture some of deflected packets before they take a misrouting hop. The captured packets are then looped-back to the routers where they suffered deflection and routed again. To improve the efficiency of this in-channel misrouting suppression scheme we also slightly modify the routing function of the deflection router by restricting the choice of productive directions for misrouted packets. Evaluations on synthetic traffic patterns show that the proposed misrouting suppression mechanism yields an improvement of 36.2% in network saturation throughput when implemented into the conventional deflection-routed network.

APA, Harvard, Vancouver, ISO, and other styles

2

Dorp, D. B. van, A. W. Eriksson, J. W. Delleman, et al. "Åland eye disease: no albino misrouting." Clinical Genetics 28, no. 6 (2008): 526–31. http://dx.doi.org/10.1111/j.1399-0004.1985.tb00421.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

van Genderen, M. M. "Chiasmal misrouting and foveal hypoplasia without albinism." British Journal of Ophthalmology 90, no. 9 (2006): 1098–102. http://dx.doi.org/10.1136/bjo.2006.091702.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Whitman, Mary C., Noriko Miyake, Elaine H. Nguyen, et al. "Decreased ACKR3 (CXCR7) function causes oculomotor synkinesis in mice and humans." Human Molecular Genetics 28, no. 18 (2019): 3113–25. http://dx.doi.org/10.1093/hmg/ddz137.

Full text

Abstract:

Abstract Oculomotor synkinesis is the involuntary movement of the eyes or eyelids with a voluntary attempt at a different movement. The chemokine receptor CXCR4 and its ligand CXCL12 regulate oculomotor nerve development; mice with loss of either molecule have oculomotor synkinesis. In a consanguineous family with congenital ptosis and elevation of the ptotic eyelid with ipsilateral abduction, we identified a co-segregating homozygous missense variant (c.772G>A) in ACKR3, which encodes an atypical chemokine receptor that binds CXCL12 and functions as a scavenger receptor, regulating levels of CXCL12 available for CXCR4 signaling. The mutant protein (p.V258M) is expressed and traffics to the cell surface but has a lower binding affinity for CXCL12. Mice with loss of Ackr3 have variable phenotypes that include misrouting of the oculomotor and abducens nerves. All embryos show oculomotor nerve misrouting, ranging from complete misprojection in the midbrain, to aberrant peripheral branching, to a thin nerve, which aberrantly innervates the lateral rectus (as seen in Duane syndrome). The abducens nerve phenotype ranges from complete absence, to aberrant projections within the orbit, to a normal trajectory. Loss of ACKR3 in the midbrain leads to downregulation of CXCR4 protein, consistent with reports that excess CXCL12 causes ligand-induced degradation of CXCR4. Correspondingly, excess CXCL12 applied to ex vivo oculomotor slices causes axon misrouting, similar to inhibition of CXCR4. Thus, ACKR3, through its regulation of CXCL12 levels, is an important regulator of axon guidance in the oculomotor system; complete loss causes oculomotor synkinesis in mice, while reduced function causes oculomotor synkinesis in humans.

APA, Harvard, Vancouver, ISO, and other styles

5

Spekreijse, H. "Misrouting in albinos; an integrated SPET/VEP study." Vision Research 35, no. 1 (1995): S118. http://dx.doi.org/10.1016/0042-6989(95)98449-j.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Derhab, Abdelouahid, Mohamed Guerroumi, Abdu Gumaei, et al. "Blockchain and Random Subspace Learning-Based IDS for SDN-Enabled Industrial IoT Security." Sensors 19, no. 14 (2019): 3119. http://dx.doi.org/10.3390/s19143119.

Full text

Abstract:

The industrial control systems are facing an increasing number of sophisticated cyber attacks that can have very dangerous consequences on humans and their environments. In order to deal with these issues, novel technologies and approaches should be adopted. In this paper, we focus on the security of commands in industrial IoT against forged commands and misrouting of commands. To this end, we propose a security architecture that integrates the Blockchain and the Software-defined network (SDN) technologies. The proposed security architecture is composed of: (a) an intrusion detection system, namely RSL-KNN, which combines the Random Subspace Learning (RSL) and K-Nearest Neighbor (KNN) to defend against the forged commands, which target the industrial control process, and (b) a Blockchain-based Integrity Checking System (BICS), which can prevent the misrouting attack, which tampers with the OpenFlow rules of the SDN-enabled industrial IoT systems. We test the proposed security solution on an Industrial Control System Cyber attack Dataset and on an experimental platform combining software-defined networking and blockchain technologies. The evaluation results demonstrate the effectiveness and efficiency of the proposed security solution.

APA, Harvard, Vancouver, ISO, and other styles

7

Spekreuse, H., and M. A. M. Hollants-Gilhuijs. "2445 Misrouting in albonos; An integrated spet/vep study." Vision Research 35 (October 1995): S118. http://dx.doi.org/10.1016/0042-6989(95)90241-4.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Schiff, Elena R., Vijay K. Tailor, Hwei Wuen Chan, Maria Theodorou, Andrew R. Webster, and Mariya Moosajee. "Novel Biallelic Variants and Phenotypic Features in Patients with SLC38A8-Related Foveal Hypoplasia." International Journal of Molecular Sciences 22, no. 3 (2021): 1130. http://dx.doi.org/10.3390/ijms22031130.

Full text

Abstract:

Biallelic pathogenic variants in solute carrier family 38 member 8, SLC38A8, cause a pan-ocular autosomal recessive condition known as foveal hypoplasia 2, FVH2, characterised by foveal hypoplasia, nystagmus and optic nerve chiasmal misrouting. Patients are often clinically diagnosed with ocular albinism, but foveal hypoplasia can occur in several other ocular disorders. Here we describe nine patients from seven families who had molecularly confirmed biallelic recessive variants in SLC38A8 identified through whole genome sequencing or targeted gene panel testing. We identified four novel sequence variants (p.(Tyr88*), p.(Trp145*), p.(Glu233Gly) and c.632+1G>A). All patients presented with foveal hypoplasia, nystagmus and reduced visual acuity; however, one patient did not exhibit any signs of chiasmal misrouting, and three patients had features of anterior segment dysgenesis. We highlight these findings in the context of 30 other families reported to date. This study reinforces the importance of obtaining a molecular diagnosis in patients whose phenotype overlap with other inherited ocular conditions, in order to support genetic counselling, clinical prognosis and family planning. We expand the spectrum of SLC38A8 mutations which will be relevant for treatment through future genetic-based therapies.

APA, Harvard, Vancouver, ISO, and other styles

9

Hou, Shurong, Franck Madoux, Louis Scampavia, Jo Ann Janovick, P. Michael Conn, and Timothy P. Spicer. "Drug Library Screening for the Identification of Ionophores That Correct the Mistrafficking Disorder Associated with Oxalosis Kidney Disease." SLAS DISCOVERY: Advancing the Science of Drug Discovery 22, no. 7 (2017): 887–96. http://dx.doi.org/10.1177/2472555217689992.

Full text

Abstract:

Primary hyperoxaluria is the underlying cause of oxalosis and is a life-threatening autosomal recessive disease, for which treatment may require dialysis or dual liver-kidney transplantation. The most common primary hyperoxaluria type 1 (PH1) is caused by genetic mutations of a liver-specific enzyme alanine:glyoxylate aminotransferase (AGT), which results in the misrouting of AGT from the peroxisomes to the mitochondria. Pharmacoperones are small molecules with the ability to modify misfolded proteins and route them correctly within the cells, which may present an effective strategy to treat AGT misrouting in PH1 disorders. We miniaturized a cell-based high-content assay into 1536-well plate format and screened ~4200 pharmacologically relevant compounds including Food and Drug Administration, European Union, and Japanese-approved drugs. This assay employs CHO cells stably expressing AGT-170, a mutant that predominantly resides in the mitochondria, where we monitor for its relocation to the peroxisomes through automated image acquisition and analysis. The miniaturized 1536-well assay yielded a Z′ averaging 0.70 ± 0.07. Three drugs were identified as potential pharmacoperones from this pilot screen, demonstrating the applicability of this assay for large-scale high-throughput screening.

APA, Harvard, Vancouver, ISO, and other styles

10

Zuccarini, Giulia, Ilaria D’Atri, Erika Cottone, et al. "Interference with the Cannabinoid Receptor CB1R Results in Miswiring of GnRH3 and AgRP1 Axons in Zebrafish Embryos." International Journal of Molecular Sciences 21, no. 1 (2019): 168. http://dx.doi.org/10.3390/ijms21010168.

Full text

Abstract:

The G protein-coupled cannabinoid receptors type 1 (CB1R) and type 2 (CB2R), and their endocannabinoid (eCBs) ligands, have been implicated in several aspects of brain wiring during development. Here we aim to assess whether interfering with CB1R affects development, neuritogenesis and pathfinding of GnRH and AgRP neurons, forebrain neurons that control respectively reproduction and appetite. We pharmacologically and genetically interfered with CB1R in zebrafish strains with fluorescently labeled GnRH3 and the AgRP1 neurons. By applying CB1R antagonists we observed a reduced number of GnRH3 neurons, fiber misrouting and altered fasciculation. Similar phenotypes were observed by CB1R knockdown. Interfering with CB1R also resulted in a reduced number, misrouting and poor fasciculation of the AgRP1 neuron’s axonal projections. Using a bioinformatic approach followed by qPCR validation, we have attempted to link CB1R functions with known guidance and fasciculation proteins. The search identified stathmin-2, a protein controlling microtubule dynamics, previously demonstrated to be coexpressed with CB1R and now shown to be downregulated upon interference with CB1R in zebrafish. Together, these results raise the likely possibility that embryonic exposure to low doses of CB1R-interfering compounds could impact on the development of the neuroendocrine systems controlling sexual maturation, reproduction and food intake.

APA, Harvard, Vancouver, ISO, and other styles

11

Keeling, Eloise, Perry Carter, Abdi Malik Musa, Fatima Shawkat, Helena Lee, and Jay E. Self. "Can a Portable Flash Visual Evoked Potential (VEP) Device Identify Chiasmal Decussation Anomalies in Albinism?" Diagnostics 15, no. 11 (2025): 1395. https://doi.org/10.3390/diagnostics15111395.

Full text

Abstract:

Background: Visual evoked potentials (VEPs) are used to detect chiasmal misrouting associated with albinism. However, VEPs are only performed in specialist centres and typically have long waiting lists. The portable electrophysiology device RETeval® shows promise as a clinical screening tool across a range of ophthalmic conditions. Here, we explore its utility in detecting chiasmal abnormalities associated with albinism. Methods: Flash VEPs were recorded on the RETeval® and by standard ISCEV techniques for 27 patients with suspected albinism and 40 control patients as part of routine appointments. We retrospectively investigated the agreeability between the two methods. The amplitude/latency of the main component was measured for standard VEPs whilst a correlation value of interhemispheric difference was calculated for the RETeval® data. Results: We demonstrate a significant difference between albinism patients and controls (p < 0.001) with respect to the interhemispheric difference identified by the RETeval®. By applying a threshold of 0.001865 to the correlation value, the RETeval® detected chiasmal misrouting in all 27 patients with albinism and had 97% agreeability to standard testing. Conclusions: This study shows the potential of using the RETeval® as a clinical tool for the diagnosis of chiasmal anomalies in albinism. The RETeval® has significant time/cost savings which could hasten diagnoses.

APA, Harvard, Vancouver, ISO, and other styles

12

Apkarian, P., P. Eckhardt, and M. van Schooneveld. "Detection of Optic Pathway Misrouting in the Human Albino Neonate." Neuropediatrics 22, no. 04 (1991): 211–15. http://dx.doi.org/10.1055/s-2008-1071443.

Full text

APA, Harvard, Vancouver, ISO, and other styles

13

Muruganandam, D., and J. Martin Leo Manickam. "Detection and Countermeasure of Packet Misrouting in Wireless Adhoc Networks." Sensor Letters 17, no. 9 (2019): 696–700. http://dx.doi.org/10.1166/sl.2019.4127.

Full text

Abstract:

A MANET is an infrastructure-less type network, which consists of number of mobile nodes connected through wireless network interfaces. The Communication among nodes is made successfully when the nodes dynamically set up route among one another. The open nature and infrastructureless type of such networks causes the attacker's interest to penetrate through the network and decrease the network performance. Thus Security becomes a major concern for protected communication between mobile nodes. Packet misrouting stops the packet from reaching the destination by a malicious intermediate node. But the malicious node makes the intuition to its neighbors that it has done the genuine packet forwarding action. Moreover the malicious node makes the neighbours to suspect the normal node as malicious one. The proposed work ensures the detection of malicious nodes and avoids suspecting the trustworthy.

APA, Harvard, Vancouver, ISO, and other styles

14

Prieur, Delphine S., and Alexandra Rebsam. "Retinal axon guidance at the midline: Chiasmatic misrouting and consequences." Developmental Neurobiology 77, no. 7 (2017): 844–60. http://dx.doi.org/10.1002/dneu.22473.

Full text

APA, Harvard, Vancouver, ISO, and other styles

15

Nomura, T. "Misrouting of mitral cell progenitors in the Pax6/small eyerat telencephalon." Development 131, no. 4 (2004): 787–96. http://dx.doi.org/10.1242/dev.00984.

Full text

APA, Harvard, Vancouver, ISO, and other styles

16

Takizawa, Satohiro, Yurika Nakanishi, Yumeno Koga, et al. "CRMP2 and its phosphorylation prevent axonal misrouting of the corticospinal tract." Neuroscience Letters 855 (April 2025): 138231. https://doi.org/10.1016/j.neulet.2025.138231.

Full text

APA, Harvard, Vancouver, ISO, and other styles

17

Ezhilvendhan, Dr K., Dr Harshitha S, Dr Kamalabala Krishnan, and Dr Aprajita Gautam. "An Interesting Case of Hermansky Pudlak Syndrome- A Rare Case Report." Scholars Journal of Medical Case Reports 9, no. 11 (2021): 1105–8. http://dx.doi.org/10.36347/sjmcr.2021.v09i11.017.

Full text

Abstract:

Oculo cutaneous albinism [OCA] is a autosomal recessive disorder associated with reduction in the pigment of skin, hair and eyes with strabismus and nystagmus leading to misrouting of optic nerve fibers from eye to brain during development. Reduction in skin pigmentation leads to the increased risk of skin cancer due to photosensitivity. They can occur in isolated forms or associated with syndromes. Syndromic OCA includes oculocutaneous albinism with hermansky pudlak syndrome. Seven types of OCA [1-6] with mutation in different genes have been identified.

APA, Harvard, Vancouver, ISO, and other styles

18

Apaja, Pirjo M., Jussi T. Tuusa, E. Maritta Pietilä, Hannu J. Rajaniemi, and Ulla E. Petäjä-Repo. "Luteinizing Hormone Receptor Ectodomain Splice Variant Misroutes the Full-Length Receptor into a Subcompartment of the Endoplasmic Reticulum." Molecular Biology of the Cell 17, no. 5 (2006): 2243–55. http://dx.doi.org/10.1091/mbc.e05-09-0875.

Full text

Abstract:

The luteinizing hormone receptor (LHR) is a G protein–coupled receptor that is expressed in multiple RNA messenger forms. The common rat ectodomain splice variant is expressed concomitantly with the full-length LHR in tissues and is a truncated transcript corresponding to the partial ectodomain with a unique C-terminal end. Here we demonstrate that the variant alters the behavior of the full-length receptor by misrouting it away from the normal secretory pathway in human embryonic kidney 293 cells. The variant was expressed as two soluble forms of Mr 52,000 and Mr 54,000, but although the protein contains a cleavable signal sequence, no secretion to the medium was observed. Only a very small fraction of the protein was able to gain hormone-binding ability, suggesting that it is retained in the endoplasmic reticulum (ER) by its quality control due to misfolding. This was supported by the finding that the variant was found to interact with calnexin and calreticulin and accumulated together with these ER chaperones in a specialized juxtanuclear subcompartment of the ER. Only proteasomal blockade with lactacystin led to accumulation of the variant in the cytosol. Importantly, coexpression of the variant with the full-length LHR resulted in reduction in the number of receptors that were capable of hormone binding and were expressed at the cell surface and in targeting of immature receptors to the juxtanuclear ER subcompartment. Thus, the variant mediated misrouting of the newly synthesized full-length LHRs may provide a way to regulate the number of cell surface receptors.

APA, Harvard, Vancouver, ISO, and other styles

19

Apkarian, P. "A Practical Approach to Albino Diagnosis VEP Misrouting Across the Age Span." Ophthalmic Paediatrics and Genetics 13, no. 2 (1992): 77–88. http://dx.doi.org/10.3109/13816819209087608.

Full text

APA, Harvard, Vancouver, ISO, and other styles

20

Anguelova, Galia V., Martijn J. A. Malessy, Erik W. van Zwet, and J. Gert van Dijk. "Extensive motor axonal misrouting after conservative treatment of obstetric brachial plexus lesions." Developmental Medicine & Child Neurology 56, no. 10 (2014): 984–89. http://dx.doi.org/10.1111/dmcn.12490.

Full text

APA, Harvard, Vancouver, ISO, and other styles

21

Whitlock, K. E. "Development of Drosophila wing sensory neurons in mutants with missing or modified cell surface molecules." Development 117, no. 4 (1993): 1251–60. http://dx.doi.org/10.1242/dev.117.4.1251.

Full text

Abstract:

The neurons of the sensory receptors on the wing of Drosophila melanogaster have highly characteristic axon projections in the central nervous system (CNS). The morphology of these projections was studied in flies bearing mutations that affect cell surface molecules thought to be important in axon guidance. The animals used were mutant for the fasciclinI (fasI), fasciclinII (fasII), fasciclinIII (fasIII) and neurally altered carbohydrate (nac) genes. Axon populations were visualized by staining with DiI and light-reacting the dye with diaminobenzidine to yield permanent preparations. The fasI, fasII and fasIII mutants as well as the nac mutant display altered axonal trajectories in the CNS. One phenotype seen in fasII mutants and in animals mutant for both fasI and fasIII was extra branching within the axon projection pattern. A second phenotype observed was a reduction or complete loss of one of the tracts, apparently due to the axons shifting to a neighboring tract. This was seen in the most extreme form in nac mutants and to a lesser degree in fasIII mutants. To determine if the mutations discussed here affected axon guidance, wing discs were analyzed using the antibody 22C10 to label sensory neurons in the wing during metamorphosis. Both misrouting of axons and the appearance of ectopic neurons in the wing were observed. In the fasI:fasIII, the fasII and the nac mutants, there was misrouting of sensory axons in the developing wing. In addition, the fasII and nac mutants displayed ectopic sensory neurons in the wing. This implies that the cell surface molecules missing (fasciclins) or modified (by the nac gene product), in these mutants may play a role in both neurogenesis and axon guidance.

APA, Harvard, Vancouver, ISO, and other styles

22

Poulter, James A., Musallam Al-Araimi, Ivan Conte, et al. "Recessive Mutations in SLC38A8 Cause Foveal Hypoplasia and Optic Nerve Misrouting without Albinism." American Journal of Human Genetics 93, no. 6 (2013): 1143–50. http://dx.doi.org/10.1016/j.ajhg.2013.11.002.

Full text

APA, Harvard, Vancouver, ISO, and other styles

23

Sinha, Sumit, G. Lakshmi Prasad, and Sanjeev Lalwani. "A cadaveric microanatomical study of the fascicular topography of the brachial plexus." Journal of Neurosurgery 125, no. 2 (2016): 355–62. http://dx.doi.org/10.3171/2015.6.jns142181.

Full text

Abstract:

OBJECT Mapping of the fascicular anatomy of the brachial plexus could provide the nerve surgeon with knowledge of fascicular orientation in spinal nerves of the brachial plexus. This knowledge might improve the surgical outcome of nerve grafting in brachial plexus injuries by anastomosing related fascicles and avoiding possible axonal misrouting. The objective of this study was to map the fascicular topography in the spinal nerves of the brachial plexus. METHODS The entire right-sided brachial plexus of 25 adult male cadavers was dissected, including all 5 spinal nerves (C5–T1), from approximately 5 mm distal to their exit from the intervertebral foramina, to proximal 1 cm of distal branches. All spinal nerves were tagged on the cranial aspect of their circumference using 10-0 nylon suture for orientation. The fascicular dissection of the C5–T1 spinal nerves was performed under microscopic magnification. The area occupied by different nerve fascicles was then expressed as a percentage of the total cross-sectional area of a spinal nerve. RESULTS The localization of fascicular groups was fairly consistent in all spinal nerves. Overall, 4% of the plexus supplies the suprascapular nerve, 31% supplies the medial cord (comprising the ulnar nerve and medial root of the median nerve [MN]), 27.2% supplies the lateral cord (comprising the musculocutaneous nerve and lateral root of the MN), and 37.8% supplies the posterior cord (comprising the axillary and radial nerves). CONCLUSIONS The fascicular dissection and definitive anatomical localization of fascicular groups is feasible in plexal spinal nerves. The knowledge of exact fascicular location might be translatable to the operating room and can be used to anastomose related fascicles in brachial plexus surgery, thereby avoiding the possibility of axonal misrouting and improving the results of plexal reconstruction.

APA, Harvard, Vancouver, ISO, and other styles

24

Ambasudhan, Rajesh, XiaoFei Wang, Monica M. Jablonski, et al. "Atrophic macular degeneration mutations in ELOVL4 result in the intracellular misrouting of the protein." Genomics 83, no. 4 (2004): 615–25. http://dx.doi.org/10.1016/j.ygeno.2003.10.004.

Full text

APA, Harvard, Vancouver, ISO, and other styles

25

Bach, Michael, and Guntram Kommerell. "Albino-type misrouting of the optic nerve fibers not found in dissociated vertical deviation." Graefe's Archive for Clinical and Experimental Ophthalmology 230, no. 2 (1992): 158–61. http://dx.doi.org/10.1007/bf00164655.

Full text

APA, Harvard, Vancouver, ISO, and other styles

26

Rachel, Rivka A., Jennifer N. Murdoch, Friedrich Beermann, Andrew J. Copp, and Carol A. Mason. "Retinal axon misrouting at the optic chiasm in mice with neural tube closure defects." genesis 27, no. 1 (2000): 32–47. http://dx.doi.org/10.1002/1526-968x(200005)27:1<32::aid-gene50>3.0.co;2-t.

Full text

APA, Harvard, Vancouver, ISO, and other styles

27

Benahmed, Khelifa, Madjid Merabti, and Hafid Haffaf. "Inspired Social Spider Behavior for Secure Wireless Sensor Networks." International Journal of Mobile Computing and Multimedia Communications 4, no. 4 (2012): 1–10. http://dx.doi.org/10.4018/jmcmc.2012100101.

Full text

Abstract:

Last year’s biologically inspired systems had received a great interest. Behavior of social insects, DNA computation, artificial neural networks, evolutionary computation, and artificial immune systems are some of the interests which can be highlighted. Wireless sensor networks have no clear line of defense and no fixed infrastructure; therefore, the known security techniques used for cabled networks might not work perfectly. While wireless sensor networks, node misbehavior can cause the packet dropping, packet modification, packet misrouting, selfish node behavior, and so on. A biologically-inspired algorithm for detecting misbehaving nodes in a wireless sensor network is presented. Such an algorithm, inspired by the behavior of some social spiders from Congo, a specially their strategy to collaborate for detecting an intrusion in their web.

APA, Harvard, Vancouver, ISO, and other styles

28

Sharma, Ms Shreya, and Ms Rupali Rohankar. "Vehicular Traffic Monitoring Using VANET." INTERNATIONAL JOURNAL OF COMPUTERS & TECHNOLOGY 13, no. 1 (2014): 4068–73. http://dx.doi.org/10.24297/ijct.v13i1.2928.

Full text

Abstract:

During last few years, continuous progresses in wireless communication have open new research fields in computer network. VANET proposes communication between vehicles by wireless means has a large potential to improve safety and travel comfort of passenger. Moreover automobile traffic safety is a major issue in developed area in which there may be misbehaving of traffic safety due to lack of controls or misguiding from the attacker end. Due to this, there is high amount of wastage of time and resources travelling through traffic congestion and misrouting of the vehicles by external source. For the sake of this, we propose an algorithm for VANET in which we show the effect of and further remove it by actual path routing and communication using distance algorithm to further improve the traffic pattern.Â

APA, Harvard, Vancouver, ISO, and other styles

29

Aslam Shaikh, Nikhat Naaz, and Vaishali Bagade. "Performance Evaluation and Detection of Grey, Warm, Flooding, Misrouting & Modification of Attacks in Vanet." International Journal of Electronics and Communication Engineering 8, no. 4 (2021): 10–17. http://dx.doi.org/10.14445/23488549/ijece-v8i4p103.

Full text

APA, Harvard, Vancouver, ISO, and other styles

30

Kruijt, Charlotte C., Gerard C. de Wit, Herman E. Talsma, Nicoline E. Schalij-Delfos, and Maria M. van Genderen. "The Detection Of Misrouting In Albinism: Evaluation of Different VEP Procedures in a Heterogeneous Cohort." Investigative Opthalmology & Visual Science 60, no. 12 (2019): 3963. http://dx.doi.org/10.1167/iovs.19-27364.

Full text

APA, Harvard, Vancouver, ISO, and other styles

31

Banihani, Saleh M. "Loss of binocular vision as direct cause for misrouting of temporal retinal fibers in albinism." Medical Hypotheses 85, no. 4 (2015): 458–62. http://dx.doi.org/10.1016/j.mehy.2015.06.028.

Full text

APA, Harvard, Vancouver, ISO, and other styles

32

Schwahn, U. "Mutations in the X-linked RP2 gene cause intracellular misrouting and loss of the protein." Human Molecular Genetics 10, no. 11 (2001): 1177–83. http://dx.doi.org/10.1093/hmg/10.11.1177.

Full text

APA, Harvard, Vancouver, ISO, and other styles

33

Hoffmann, Michael B., Birgit Lorenz, Antony B. Morland, and Linda C. Schmidtborn. "Misrouting of the Optic Nerves in Albinism: Estimation of the Extent with Visual Evoked Potentials." Investigative Opthalmology & Visual Science 46, no. 10 (2005): 3892. http://dx.doi.org/10.1167/iovs.05-0491.

Full text

APA, Harvard, Vancouver, ISO, and other styles

34

Kim, Sooyeon, and Tim Moses. "An Investigation of the Impact of Misrouting Under Two-Stage Multistage Testing: A Simulation Study." ETS Research Report Series 2014, no. 1 (2014): 1–13. http://dx.doi.org/10.1002/ets2.12000.

Full text

APA, Harvard, Vancouver, ISO, and other styles

35

Sathianesan, Godfrey Winster, S. Gnanavel, R. Salini, V. Raji, R. Vijay Anand, and Md Salah Uddin. "Detection and Counter Measure of Packet Misrouting by GPS-Free Positioning of Ad-Hoc Nodes." Mobile Information Systems 2023 (April 19, 2023): 1–12. http://dx.doi.org/10.1155/2023/1029875.

Full text

Abstract:

The ad-hoc networks are a developing area of research with a large and wide variety of applications and related requirements. The nodes participating in an ad-hoc network use different routing protocols to send their packets from one node to another. But most of the time, the ad-hoc network is not suitable for urgent needs. For this, an ad-hoc network GPS-free positioning system can be used in emergency situations to save people in danger. Nodes participating in an ad-hoc network choose the best route from various nodes near them to send information through the complex system. For this, we have used trust dynamic source routing (TDSR) in our research work to determine and design the best route to transmit information, and we designed this system with the aim of being able to be used in emergencies. We have compared our proposed routing protocols with dynamic source routing (DSR) and found out that TDSR is working excellent.

APA, Harvard, Vancouver, ISO, and other styles

36

Moeller, Hanne B., Emma T. B. Olesen, and Robert A. Fenton. "Regulation of the water channel aquaporin-2 by posttranslational modification." American Journal of Physiology-Renal Physiology 300, no. 5 (2011): F1062—F1073. http://dx.doi.org/10.1152/ajprenal.00721.2010.

Full text

Abstract:

The cellular functions of many eukaryotic membrane proteins, including the vasopressin-regulated water channel aquaporin-2 (AQP2), are regulated by posttranslational modifications. In this article, we discuss the experimental discoveries that have advanced our understanding of how posttranslational modifications affect AQP2 function, especially as they relate to the role of AQP2 in the kidney. We review the most recent data demonstrating that glycosylation and, in particular, phosphorylation and ubiquitination are mechanisms that regulate AQP2 activity, subcellular sorting and distribution, degradation, and protein interactions. From a clinical perspective, posttranslational modification resulting in protein misrouting or degradation may explain certain forms of nephrogenic diabetes insipidus. In addition to providing major insight into the function and dynamics of renal AQP2 regulation, the analysis of AQP2 posttranslational modification may provide general clues as to the role of posttranslational modification for regulation of other membrane proteins.

APA, Harvard, Vancouver, ISO, and other styles

37

Quoc, Nguyen Hong, and Le Van Hoa. "A Hybrid Model of Limited Burst Retransmission and Deflection Routing in Optical Switching Network." Cybernetics and Information Technologies 25, no. 1 (2025): 97–109. https://doi.org/10.2478/cait-2025-0006.

Full text

Abstract:

Abstract Optical Burst Switching (OBS) is considered a promising optical switching technology for the future. However, a key issue of the OBS network is reducing dropped bursts due to contentions because there is no optical buffer at intermediate nodes. Several methods have been proposed to address burst contention, such as wavelength conversion, Fiber Delay Line (FDL) usage, deflection routing, or burst retransmission. Among these methods, deflection routing and burst retransmission are two approaches that do not modify the network infrastructure and can take advantage of idle resources on alternative connections. However, uncontrolled burst retransmissions and misrouting can lead to increased collisions, and potentially endless collision handling loops. This paper proposes a hybrid model of limited burst retransmission and deflection routing. Simulation results show that the proposed model has significantly improved resource utilization efficiency, burst-dropping probability, and end-to-end transmission delay.

APA, Harvard, Vancouver, ISO, and other styles

38

Huang, Y. Y., O. Rinner, P. Hedinger, S. C. Liu, and S. C. F. Neuhauss. "Oculomotor Instabilities in Zebrafish Mutant belladonna: A Behavioral Model for Congenital Nystagmus Caused by Axonal Misrouting." Journal of Neuroscience 26, no. 39 (2006): 9873–80. http://dx.doi.org/10.1523/jneurosci.2886-06.2006.

Full text

APA, Harvard, Vancouver, ISO, and other styles

39

MEIJ, IWAN C., JAN B. KOENDERINK, JOKE C. JONG та ін. "Dominant Isolated Renal Magnesium Loss Is Caused by Misrouting of the Na+,K+-ATPase γ-Subunit". Annals of the New York Academy of Sciences 986, № 1 (2003): 437–43. http://dx.doi.org/10.1111/j.1749-6632.2003.tb07226.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

40

Meij, Iwan C., Jan B. Koenderink, Hans van Bokhoven та ін. "Dominant isolated renal magnesium loss is caused by misrouting of the Na+,K+-ATPase γ-subunit". Nature Genetics 26, № 3 (2000): 265–66. http://dx.doi.org/10.1038/81543.

Full text

APA, Harvard, Vancouver, ISO, and other styles

41

Leaños-Miranda, Alfredo, Jo Ann Janovick, and P. Michael Conn. "Receptor-Misrouting: An Unexpectedly Prevalent and Rescuable Etiology in Gonadotropin-Releasing Hormone Receptor-Mediated Hypogonadotropic Hypogonadism." Journal of Clinical Endocrinology & Metabolism 87, no. 10 (2002): 4825–28. http://dx.doi.org/10.1210/jc.2002-020961.

Full text

APA, Harvard, Vancouver, ISO, and other styles

42

Selvavinayaki, Dr K. "An Effectual Data Transmission Approach to Prevent Black Hole Attack in Mobile Adhoc Networks." INFORMATION TECHNOLOGY IN INDUSTRY 9, no. 1 (2021): 526–40. http://dx.doi.org/10.17762/itii.v9i1.166.

Full text

Abstract:

The dynamic nature of infrastructure in MANET makes security as a critical issue. Due to mobility of nodes, network is easily affected by several types of attacks. In particular black hole attack cause packet dropping, misrouting the information from source to destination. To prevent a network from the effects of the black hole attack, we propose a most reliable data transfer scheme to detect the black hole nodes in the network and ensure the availability, reliability, confidentiality of the information. First, the proposed scheme detects the black hole node using trust active and node recommendation values. Second, the reliability and confidentiality of the information is achieved by using verifiable secret sharing scheme. The proposed algorithm is implemented on AOMDV protocol. The simulation results show the proposed algorithm achieves the better packet delivery ratio, misbehavior detection efficiency, fewer packets overhead and low end to end delay than the existing schemes.

APA, Harvard, Vancouver, ISO, and other styles

43

Cetin-Berber, Dee Duygu, Halil Ibrahim Sari, and Anne Corinne Huggins-Manley. "Imputation Methods to Deal With Missing Responses in Computerized Adaptive Multistage Testing." Educational and Psychological Measurement 79, no. 3 (2018): 495–511. http://dx.doi.org/10.1177/0013164418805532.

Full text

Abstract:

Routing examinees to modules based on their ability level is a very important aspect in computerized adaptive multistage testing. However, the presence of missing responses may complicate estimation of examinee ability, which may result in misrouting of individuals. Therefore, missing responses should be handled carefully. This study investigated multiple missing data methods in computerized adaptive multistage testing, including two imputation techniques, the use of full information maximum likelihood and the use of scoring missing data as incorrect. These methods were examined under the missing completely at random, missing at random, and missing not at random frameworks, as well as other testing conditions. Comparisons were made to baseline conditions where no missing data were present. The results showed that imputation and the full information maximum likelihood methods outperformed incorrect scoring methods in terms of average bias, average root mean square error, and correlation between estimated and true thetas.

APA, Harvard, Vancouver, ISO, and other styles

44

Su, Jianmin, Naomi E. Charalambakis, Ubadah Sabbagh, et al. "Retinal inputs signal astrocytes to recruit interneurons into visual thalamus." Proceedings of the National Academy of Sciences 117, no. 5 (2020): 2671–82. http://dx.doi.org/10.1073/pnas.1913053117.

Full text

Abstract:

Inhibitory interneurons comprise a fraction of the total neurons in the visual thalamus but are essential for sharpening receptive field properties and improving contrast-gain of retinogeniculate transmission. During early development, these interneurons undergo long-range migration from germinal zones, a process regulated by the innervation of the visual thalamus by retinal ganglion cells. Here, using transcriptomic approaches, we identified a motogenic cue, fibroblast growth factor 15 (FGF15), whose expression in the visual thalamus is regulated by retinal input. Targeted deletion of functional FGF15 in mice led to a reduction in thalamic GABAergic interneurons similar to that observed in the absence of retinal input. This loss may be attributed, at least in part, to misrouting of interneurons into nonvisual thalamic nuclei. Unexpectedly, expression analysis revealed that FGF15 is generated by thalamic astrocytes and not retino-recipient neurons. Thus, these data show that retinal inputs signal through astrocytes to direct the long-range recruitment of interneurons into the visual thalamus.

APA, Harvard, Vancouver, ISO, and other styles

45

Asadi, Farzad, та Savita Dhanvantari. "Misrouting of glucagon and stathmin-2 towards lysosomal system of α-cells in glucagon hypersecretion of diabetes". Islets 14, № 1 (2021): 40–57. http://dx.doi.org/10.1080/19382014.2021.2011550.

Full text

APA, Harvard, Vancouver, ISO, and other styles

46

CONESA, Magali, Annik PRAT, John S. MORT, Jacques MARVALDI, Jean-Claude LISSITZKY, and Nabil G. SEIDAH. "Down-regulation of alphav/beta3 integrin via misrouting to lysosomes by overexpression of a beta3Lamp1 fusion protein." Biochemical Journal 370, no. 2 (2003): 703–11. http://dx.doi.org/10.1042/bj20021301.

Full text

Abstract:

We present a general strategy for the dominant negative reduction in the levels of type-1 membrane-bound heterodimeric proteins within the secretory pathway through fusion of the soluble ectodomain of one of the partners to the transmembrane-cytosolic tail of the lysosomal protein Lamp1. Thus, in human embryonic kidney (HEK)-293 cells, overexpression of an integrin β3Lamp1 chimera resulted in a drastic reduction of its endogenous partner, the integrin αv subunit. The mechanism involves the formation in the endoplasmic reticulum of a αv/β3Lamp1 complex that is subsequently sorted towards a lysosomal/endosomal degradation pathway. The specificity of this approach is afforded by the invariance in the levels of the endogenous integrins α5 and β1 as compared with control cells. Conversely overexpression of integrin β3 in HEK-293 cells led to an increased level of αvβ3 at the cell surface. Functionally β3Lamp1 and β3 overexpressors exhibit decreased and increased adhesion to vitronectin, respectively, as well as diminished cellular aggregation. The application of this technology should enable the analysis of the functional importance of homodimers or heterodimers in the cell types of choice and the identification of novel partner proteins by proteomic approaches.

APA, Harvard, Vancouver, ISO, and other styles

47

Whitman, Mary C., Elaine H. Nguyen, Jessica L. Bell, Alan P. Tenney, Alon Gelber, and Elizabeth C. Engle. "Loss of CXCR4/CXCL12 Signaling Causes Oculomotor Nerve Misrouting and Development of Motor Trigeminal to Oculomotor Synkinesis." Investigative Opthalmology & Visual Science 59, no. 12 (2018): 5201. http://dx.doi.org/10.1167/iovs.18-25190.

Full text

APA, Harvard, Vancouver, ISO, and other styles

48

Beermann, Friedrich, Seth J. Orlow, Raymond E. Boissy, Andrea Schmidt, Ying L. Boissy, and M. Lynn Lamoreux. "Misrouting of tyrosinase with a truncated cytoplasmic tail as a result of the murine platinum (cp) mutation." Experimental Eye Research 61, no. 5 (1995): 599–607. http://dx.doi.org/10.1016/s0014-4835(05)80053-3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

49

Simmen, T., A. Schmidt, W. Hunziker, and F. Beermann. "The tyrosinase tail mediates sorting to the lysosomal compartment in MDCK cells via a di-leucine and a tyrosine-based signal." Journal of Cell Science 112, no. 1 (1999): 45–53. http://dx.doi.org/10.1242/jcs.112.1.45.

Full text

Abstract:

Tyrosinase is a type I membrane protein found in melanosomes, which are lysosomal-like organelles and specific for pigment cells. A mutation of mouse tyrosinase, platinum (cp), leads to truncation of tyrosinase's cytosolic tail, and results in misrouting to the cell periphery. In this study, we expressed chimeras of wild-type and mutant cytosolic tails of mouse tyrosinase fused to rat lysosome-associated membrane protein-1 luminal and transmembrane domain to study sorting of tyrosinase in Madin-Darby canine kidney cells. The study shows that the mouse tyrosinase cytosolic tail is necessary and sufficient to mediate sorting of a heterologous type I membrane protein to compartments of the lysosomal lineage. Whereas deletions of 7 or 10 C-terminal amino acids of the tail still result in sorting to lysosomes, a deletion mutant corresponding to platinum (cp) tail fails to sort correctly and corroborates the in situ findings in cp homozygous mutant mice. Correct sorting of tyrosinase-lysosome-associated membrane protein-1 chimeras is mediated by the interplay of a di-leucine signal and a tyrosine motif of the Y-X-X-O type.

APA, Harvard, Vancouver, ISO, and other styles

50

Bulow, H. E., K. L. Berry, L. H. Topper, E. Peles, and O. Hobert. "Heparan sulfate proteoglycan-dependent induction of axon branching and axon misrouting by the Kallmann syndrome gene kal-1." Proceedings of the National Academy of Sciences 99, no. 9 (2002): 6346–51. http://dx.doi.org/10.1073/pnas.092128099.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!