Relevant bibliographies by topics / Mitochondria

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Mitochondria'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "Mitochondria"

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Amchenkova, A. A., L. E. Bakeeva, Y. S. Chentsov, V. P. Skulachev, and D. B. Zorov. "Coupling membranes as energy-transmitting cables. I. Filamentous mitochondria in fibroblasts and mitochondrial clusters in cardiomyocytes." Journal of Cell Biology 107, no. 2 (1988): 481–95. http://dx.doi.org/10.1083/jcb.107.2.481.

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An hypothesis considering mitochondria as intracellular power-transmitting protonic cables was tested in human fibroblasts where mitochondria are thin and long and in rat cardiomyocytes where they show cluster organization. Mitochondria in the cell were specifically stained with fluorescent-penetrating cation ethylrhodamine, which electrophoretically accumulates in the mitochondrial matrix. A 40-micron-long mitochondrial filament of fibroblast was illuminated by a very narrow (less than or equal to 0.5 micron) laser beam to induce local damage of the mitochondrial membranes. Such a treatment w
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Banerjee, Partha S., Junfeng Ma, and Gerald W. Hart. "Diabetes-associated dysregulation ofO-GlcNAcylation in rat cardiac mitochondria." Proceedings of the National Academy of Sciences 112, no. 19 (2015): 6050–55. http://dx.doi.org/10.1073/pnas.1424017112.

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Elevated mitochondrialO-GlcNAcylation caused by hyperglycemia, as occurs in diabetes, significantly contributes to mitochondrial dysfunction and to diabetic cardiomyopathy. However, little is known about the enzymology of mitochondrialO-GlcNAcylation. Herein, we investigated the enzymes responsible for cyclingO-GlcNAc on mitochondrial proteins and studied the mitochondrial transport of UDP-GlcNAc. Analyses of purified rat heart mitochondria from normal and streptozocin-treated diabetic rats show increased mitochondrialO-GlcNAc transferase (OGT) and a concomitant decrease in the mito-specific O
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Seo, Young Ah, Veronica Lopez, and Shannon L. Kelleher. "A histidine-rich motif mediates mitochondrial localization of ZnT2 to modulate mitochondrial function." American Journal of Physiology-Cell Physiology 300, no. 6 (2011): C1479—C1489. http://dx.doi.org/10.1152/ajpcell.00420.2010.

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Female reproductive tissues such as mammary glands, ovaries, uterus, and placenta are phenotypically dynamic, requiring tight integration of bioenergetic and apoptotic mechanisms. Mitochondrial zinc (Zn) pools have emerged as a central player in regulating bioenergetics and apoptosis. Zn must first be imported into mitochondria to modulate mitochondrion-specific functions; however, mitochondrial Zn import mechanisms have not been identified. Here we documented that the Zn transporter ZnT2 is associated with the inner mitochondrial membrane and acts as an auxiliary Zn importer into mitochondria
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Scanlon, David P., and Michael W. Salter. "Strangers in strange lands: mitochondrial proteins found at extra-mitochondrial locations." Biochemical Journal 476, no. 1 (2019): 25–37. http://dx.doi.org/10.1042/bcj20180473.

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Abstract The mitochondrial proteome is estimated to contain ∼1100 proteins, the vast majority of which are nuclear-encoded, with only 13 proteins encoded by the mitochondrial genome. The import of these nuclear-encoded proteins into mitochondria was widely believed to be unidirectional, but recent discoveries have revealed that many these ‘mitochondrial’ proteins are exported, and have extra-mitochondrial activities divergent from their mitochondrial function. Surprisingly, three of the exported proteins discovered thus far are mitochondrially encoded and have significantly different extra-mit
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Deng, Huichao, Xinhua Qiao, Ting Xie, et al. "SLC-30A9 is required for Zn2+ homeostasis, Zn2+ mobilization, and mitochondrial health." Proceedings of the National Academy of Sciences 118, no. 35 (2021): e2023909118. http://dx.doi.org/10.1073/pnas.2023909118.

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The trace element zinc is essential for many aspects of physiology. The mitochondrion is a major Zn2+ store, and excessive mitochondrial Zn2+ is linked to neurodegeneration. How mitochondria maintain their Zn2+ homeostasis is unknown. Here, we find that the SLC-30A9 transporter localizes on mitochondria and is required for export of Zn2+ from mitochondria in both Caenorhabditis elegans and human cells. Loss of slc-30a9 leads to elevated Zn2+ levels in mitochondria, a severely swollen mitochondrial matrix in many tissues, compromised mitochondrial metabolic function, reductive stress, and induc
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Zorov, Dmitry B., Magdalena Juhaszova, and Steven J. Sollott. "Mitochondrial Reactive Oxygen Species (ROS) and ROS-Induced ROS Release." Physiological Reviews 94, no. 3 (2014): 909–50. http://dx.doi.org/10.1152/physrev.00026.2013.

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Byproducts of normal mitochondrial metabolism and homeostasis include the buildup of potentially damaging levels of reactive oxygen species (ROS), Ca2+, etc., which must be normalized. Evidence suggests that brief mitochondrial permeability transition pore (mPTP) openings play an important physiological role maintaining healthy mitochondria homeostasis. Adaptive and maladaptive responses to redox stress may involve mitochondrial channels such as mPTP and inner membrane anion channel (IMAC). Their activation causes intra- and intermitochondrial redox-environment changes leading to ROS release.
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Henderson, V., and M. J. Song. "Morphology of mitochondria in a teleost, salmo gairdneri." Proceedings, annual meeting, Electron Microscopy Society of America 44 (August 1986): 194–95. http://dx.doi.org/10.1017/s0424820100142591.

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Mitochondria have been observed at the ultrastructural level as spherical, oval, or sausagelike. Mitochondria average 0.3 to 1.0 um in diameter and 1.0 to 10.0 μm in length. Mitochondria may exceed these dimensions under certian physiological or pathological conditions. The number of mitochondria may reflect the metabolic condition of cells. Cells with high ATP demands display a large number of mitochondria. High energy requirements characterize muscles in both vertebrates and invertebrates. It has been established that yeast cells have but a single mitochondrion. This investigation was design
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Haseeb, Abdul, Hong Chen, Yufei Huang, et al. "Remodelling of mitochondria during spermiogenesis of Chinese soft-shelled turtle (Pelodiscus sinensis)." Reproduction, Fertility and Development 30, no. 11 (2018): 1514. http://dx.doi.org/10.1071/rd18010.

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Mitochondria are vital cellular organelles that have the ability to change their shape under different conditions, such as in response to stress, disease, changes in metabolic rate, energy requirements and apoptosis. In the present study, we observed remodelling of mitochondria during spermiogenesis and its relationship with mitochondria-associated granules (MAG). At the beginning of spermiogenesis, mitochondria are characterised by their round shape. As spermiogenesis progresses, the round-shaped mitochondria change into elongated and then swollen mitochondria, subsequently forming a crescent
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Fu, Ailing. "Mitotherapy as a Novel Therapeutic Strategy for Mitochondrial Diseases." Current Molecular Pharmacology 13, no. 1 (2020): 41–49. http://dx.doi.org/10.2174/1874467212666190920144115.

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Background: The mitochondrion is a multi-functional organelle that is mainly responsible for energy supply in the mammalian cells. Over 100 human diseases are attributed to mitochondrial dysfunction. Mitochondrial therapy (mitotherapy) aims to transfer functional exogenous mitochondria into mitochondria-defective cells for recovery of the cell viability and consequently, prevention of the disease progress. Conclusion: Mitotherapy makes the of modulation of cell survival possible, and it would be a potential therapeutic strategy for mitochondrial diseases. Objective: The review summarizes the e
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Qin, Lingyu, and Shuhua Xi. "The role of Mitochondrial Fission Proteins in Mitochondrial Dynamics in Kidney Disease." International Journal of Molecular Sciences 23, no. 23 (2022): 14725. http://dx.doi.org/10.3390/ijms232314725.

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Mitochondria have many forms and can change their shape through fusion and fission of the outer and inner membranes, called “mitochondrial dynamics”. Mitochondrial outer membrane proteins, such as mitochondrial fission protein 1 (FIS1), mitochondrial fission factor (MFF), mitochondrial 98 dynamics proteins of 49 kDa (MiD49), and mitochondrial dynamics proteins of 51 kDa (MiD51), can aggregate at the outer mitochondrial membrane and thus attract Dynamin-related protein 1 (DRP1) from the cytoplasm to the outer mitochondrial membrane, where DRP1 can perform a scissor-like function to cut a comple
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Dissertations / Theses on the topic "Mitochondria"

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Jugé, Romain. "Étude de la dynamique mitochondriale dans des cellules cutanées humaines : Mise en place de modèles pour des applications en cosmétologie." Thesis, Lyon, 2016. http://www.theses.fr/2016LYSEN007.

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La peau est un épithélium spécialisé vital et fragile, qui évolue avec l’âge et est influencé par l’environnement, notamment les radiations solaires. Des données sont disponibles sur la réponse du réseau mitochondrial et le devenir des mitochondries endommagées en réponse à des stress chimiques et environnementaux dans plusieurs systèmes expérimentaux, mais ces processus restent peu étudiés dans les cellules cutanées. Dans ce contexte, le projet de thèse visait à analyser l’effet (i) de l’irradiation UVB sur la dynamique mitochondriale (en particulier la fragmentation des mitochondries) dans d
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Mortz, Mathieu. "Flexibilités bioénergétique et génomique mitochondriales chez l’oiseau." Thesis, Lyon, 2019. https://n2t.net/ark:/47881/m6v40tjz.

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Les oiseaux sont des organismes endothermes faisant preuve d’une flexibilité métabolique remarquable en réponse aux contraintes énergétiques inhérentes à leur mode de vie. Cette flexibilité est notamment stimulée lors de transitions nutritionnelles pour adapter leur intensité métabolique selon les ressources énergétiques disponibles, est déterminante pour la survie chez ces animaux. Les mitochondries constituant le principal carrefour métabolique, en permettant l’essentiel de la production d’ATP cellulaire, sont largement impliquées dans les modulations observées au cours d’un jeûne et lors de
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Al, Amir Dache Zahra. "Étude de la structure de l'ADN circulant d'origine mitochondriale." Thesis, Montpellier, 2019. http://www.theses.fr/2019MONTT059.

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Le plasma transporte des cellules sanguines avec un mélange de composés, y compris les nutriments, déchets, anticorps, et messagers chimiques... dans tout l'organisme. Des facteurs non solubles tels que l’ADN circulant et les vésicules extracellulaires ont récemment été ajoutés à la liste de ces composants et ont fait l'objet d'études approfondies en raison de leur rôle dans la communication intercellulaire. Or, l’ADN circulant (ADNcir) est composé de fragments d’ADN libres ou associés à d’autres particules, libérés par tous les types cellulaires. Cet ADN est non seulement de l'ADN génomique m
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Beinat, Marine. "Caractérisation génétique des atteintes hépatiques mitochondriales." Thesis, Paris 5, 2013. http://www.theses.fr/2013PA05T007/document.

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Les maladies mitochondriales sont les anomalies congénitales du métabolisme les plus fréquentes. Elles sont caractérisées par une très grande hétérogénéité clinique et génétique, et le gène responsable de la maladie n’a pu être identifié que pour seulement 30% d’entre elles. Malgré l’hétérogénéité de ces maladies, il est possible d’identifier des groupes de patients cliniquement homogènes. C’est notamment le cas des atteintes hépatiques mitochondriales, qui peuvent se présenter sous une forme syndromique ou isolée. Les patients ayant une forme isolée ont soit une déplétion de l’ADNmt, soit une
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Reinhardt, Camille. "Impact de la voie d’import mitochondrial contrôlée par le complexe AIF/CHCHD4 sur la survie des cellules cancéreuses et la réponse aux traitements anticancéreux." Thesis, Université Paris-Saclay (ComUE), 2019. http://www.theses.fr/2019SACLS542.

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Dans la majorité des cas, les mitochondries sont nécessaires à la tumorigenèse et à la réponse des cellules cancéreuses aux signaux générés par les facteurs micro-environnementaux (exemples : privation de nutriments, hypoxie) ou par les traitements anticancéreux (exemples : chimiothérapie, radiothérapie). Presque toutes les protéines mitochondriales sont codées par le génome nucléaire et importées dans l'organelle. Des machineries d'import ont donc évolué afin de répondre aux besoins d'import protéique. Dans ce contexte, la machinerie régulée par CHCHD4/Mia40 fonctionne dans l’espace intermemb
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Renken, Christian Wolfgang. "The structure of mitochondria /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2004. http://wwwlib.umi.com/cr/ucsd/fullcit?p3141929.

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Craig, Elaine. "Protein import into cardiac mitochondria." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ39261.pdf.

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Esteves, Pauline. "Etude de l’action anti-tumorale de la protéine mitochondriale UCP2." Thesis, Paris 5, 2014. http://www.theses.fr/2014PA05T024.

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Les cellules tumorales sont caractérisées par un métabolisme hautement glycolytique à l’inverse des cellules normales qui utilisent préférentiellement un métabolisme oxydatif. Cette adaptation métabolique permet aux cellules d’être moins dépendantes de l’oxygène et favorise les processus d’invasion. D’autres mécanismes associés aux espèces réactives de l’oxygène qui induisent des dommages de l’ADN contribuent à la transformation cellulaire et à l’initiation tumorale. Le transporteur mitochondrial UCP2 est le deuxième membre identifié dans la famille des UCP dont la fonction mitochondriale rest
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Ruby, Vincent. "Étude des évènements mitochondriaux impliqués dans le contrôle de l'apoptose par rbf1, l'homologue de drosophile du gène suppresseur de tumeur rb." Thesis, Université Paris-Saclay (ComUE), 2018. http://www.theses.fr/2018SACLV039/document.

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Le gène rb est le premier suppresseur de tumeur découvert chez l’homme. Il prévient l’apparition de tumeurs notamment en régulant négativement le cycle cellulaire. Le rôle de pRb dans le contrôle de l’apoptose est plus complexe et les mécanismes moléculaires contrôlés par ce facteur de transcription ne sont pas complétement élucidés. Il existe un homologue de rb chez la drosophile : rbf1. J’ai contribué à caractériser les évènements mitochondriaux induits au cours de l’activation de l’apoptose par Rbf1 dans le disque imaginal d'aile, un tissu en prolifération de la larve de drosophile. Dans ce
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Heller, Anne Sabine [Verfasser], and Achim [Akademischer Betreuer] Göpferich. "Targeting mitochondria by mitochondrial fusion, mitochondria-specific peptides and nanotechnology / Anne Sabine Heller. Betreuer: Achim Göpferich." Regensburg : Universitätsbibliothek Regensburg, 2013. http://d-nb.info/103321664X/34.

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Books on the topic "Mitochondria"

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St. John, Justin C., ed. Mitochondrial DNA, Mitochondria, Disease and Stem Cells. Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-101-1.

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John, Justin C. St. Mitochondrial DNA, mitochondria, disease, and stem cells. Humana Press, 2013.

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Tomar, Namrata, ed. Mitochondria. Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2309-1.

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Mokranjac, Dejana, and Fabiana Perocchi, eds. Mitochondria. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6824-4.

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Schaffer, Stephen W., and M.-Saadeh Suleiman, eds. Mitochondria. Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-69945-5.

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Scheffler, Immo E. Mitochondria. John Wiley & Sons, Inc., 1999. http://dx.doi.org/10.1002/0471223891.

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Leister, Dario, and Johannes M. Herrmann, eds. Mitochondria. Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-365-3.

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A, Pon Liza, and Schon Eric A, eds. Mitochondria. Academic, 2001.

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W, Schaffer S., and Suleiman M. -Saadeh, eds. Mitochondria: The dynamic organelle. Springer, 2007.

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Van Aken, Olivier, and Allan G. Rasmusson, eds. Plant Mitochondria. Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-1653-6.

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Book chapters on the topic "Mitochondria"

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Hangay, George, Susan V. Gruner, F. W. Howard, et al. "Mitochondrion, (pl., Mitochondria)." In Encyclopedia of Entomology. Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6359-6_4638.

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Valk, Jacob, and Marjo S. van der Knaap. "Mitochondria and Mitochondrial Dysfunction." In Magnetic Resonance of Myelin, Myelination, and Myelin Disorders. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-662-02568-0_23.

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Morava, E., and J. A. M. Smeitink. "Mitochondria and Mitochondrial Disorders." In Magnetic Resonance of Myelination and Myelin Disorders. Springer Berlin Heidelberg, 2005. http://dx.doi.org/10.1007/3-540-27660-2_23.

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van der Knaap, Marjo S., and Jacob Valk. "Mitochondria and Mitochondrial Disorders." In Magnetic Resonance of Myelin, Myelination, and Myelin Disorders. Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-03078-3_20.

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Mattedi, Francesca, George Chennell, and Alessio Vagnoni. "Detailed Imaging of Mitochondrial Transport and Precise Manipulation of Mitochondrial Function with Genetically Encoded Photosensitizers in Adult Drosophila Neurons." In Methods in Molecular Biology. Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-1990-2_20.

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AbstractPrecise distribution of mitochondria is essential for maintaining neuronal homeostasis. Although detailed mechanisms governing the transport of mitochondria have emerged, it is still poorly understood how the regulation of transport is coordinated in space and time within the physiological context of an organism. How alteration in mitochondrial functionality may trigger changes in organellar dynamics also remains unclear in this context. Therefore, the use of genetically encoded tools to perturb mitochondrial functionality in real time would be desirable. Here we describe methods to in
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Hassinen, Ilmo. "Regulation of Mitochondrial Respiration in Heart Muscle." In Mitochondria. Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-69945-5_1.

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O’Rourke, Brian. "Mitochondrial Ion Channels." In Mitochondria. Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-69945-5_10.

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Halestrap, Andrew P., Samatha J. Clarke, and Igor Khalilin. "The Mitochondrial Permeability Transition Pore – from Molecular Mechanism to Reperfusion Injury and Cardioprotection." In Mitochondria. Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-69945-5_11.

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Suleiman, M.-Saadeh, and Stephen W. Schaffer. "The Apoptotic Mitochondrial Pathway – Modulators, Interventions and Clinical Implications." In Mitochondria. Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-69945-5_12.

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Murphy, Elizabeth, and Charles Steenbergen. "The Role of Mitochondria in Necrosis Following Myocardial Ischemia-Reperfusion." In Mitochondria. Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-69945-5_13.

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Conference papers on the topic "Mitochondria"

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Bae, Kideog, Muzaffer Ozbey, Mark A. Anastasio, Stephen A. Boppart, and Edita Aksamitiene. "Label-free mitochondria segmentation for morphological and functional study of mitochondria (Conference Presentation)." In Dynamics and Fluctuations in Biomedical Photonics XXII, edited by Valery V. Tuchin, Martin J. Leahy, and Ruikang K. Wang. SPIE, 2025. https://doi.org/10.1117/12.3041285.

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Pope, Nathaniel J., Joshua Lalonde, Gary D. Noojin, and Michael L. Denton. "Photostimulation of mitochondria: a Raman microspectroscopy approach." In Mechanisms of Photobiomodulation Therapy XIX, edited by James D. Carroll, Ann Liebert, and Jeri-Anne Lyons. SPIE, 2025. https://doi.org/10.1117/12.3039302.

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Balan, Ion, Peter Surai, Monica Marin, et al. "Morfofuncționalitatea mitocondrială în evoluția spermatogenezei la mamifere." In "Fiziologia şi sănătatea", congresul fiziologilor. Bulletin of the Academy of Sciences of Moldova. Medical Sciences, 2025. https://doi.org/10.52692/cfzl2024.10.

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In spermatogenesis, mitochondria are particularly important for the development and maintenance of germ cells and the production of healthy gametes. Deficiencies in these processes lead to mitochondrial dysfunction and abnormal spermatogenesis. The classical roles of mitochondria in energy production, metabolism and apoptosis have been well defined, but they also have various other functions. Mitochondria are also involved in testosterone production, regulating proton secretion in the lumen to maintain an acidic state in the epididymis, and condensing sperm DNA. In the available literature on
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Li, Ching-Wen, Pao-Hsin Yen, and Gou-Jen Wang. "A Cascade Microfluidic Device for High Quality Mitochondria Extraction." In ASME 2015 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/detc2015-46117.

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Recent researches have demonstrated that cells ingest mitochondrial by endocytosis to repair cell damage. This new mitochondrial therapy approach can be used for curing particular disease of neuropathy related diseases. Hence the obtainment of high quality and healthy mitochondria play an important role in mitochondrial based disease therapy. In this study, we propose a cascade microfluidic device for green extraction of healthy mitochondria. The geometry of the device was designed using the commercially available COMSOL package. Soft lithography process was than conducted to realize the devic
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Vitkovac, Aleksandra, Tanja Pajić, Marta Bukumira, Marina Stanić, Mihailo Rabasović, and Nataša Todorović. "Slight cooling during growth induced changes in filamentous fungi hypha mitochondrial morphology." In 2nd International Conference on Chemo and Bioinformatics. Institute for Information Technologies, University of Kragujevac, 2023. http://dx.doi.org/10.46793/iccbi23.334v.

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Adaptive changes in mitochondrial morphology are associated with changes in the mitochondrial function and metabolic fitness of eukaryotic cells. We previously described in young hyphae of the filamentous fungus Phycomyces blakesleeanus a dramatic effect of an increase in ambient temperature during growth: a 3°C warmer environment compared with a control temperature of 22°C resulted in the appearance of long elongated (“tubular”) mitochondria accompanied by an increase in lipid droplet density. Here, we examined how cooler ambient temperature (18°C) during growth affects mitochondrial morpholo
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Getmanskaya, Alexandra, Nikolai Sokolov, and Vadim Turlapov. "Multiclass U-Net Segmentation of Brain Electron Microscopy Data." In 31th International Conference on Computer Graphics and Vision. Keldysh Institute of Applied Mathematics, 2021. http://dx.doi.org/10.20948/graphicon-2021-3027-508-518.

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This work focuses on multi-class labeling and segmentation of electron microscopy data. The well-known and state-of-the-art EPFL open dataset has been labeled for 6 classes (instead of 1) and a multi-class version of the U-Net was trained. The new labeled classes are mitochondrion together with its border, mitochondrion’s border (separately), membrane, PSD, axon, vesicle. Our labeling results are available on GitHub. Our study showed that the quality of segmentation is affected by the presence of a sufficient number of specific features that distinguish the selected classes and the representat
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Ermoshina, A. A., A. D. Okhalnikov, A. O. Motorina, M. S. Gavrish, and A. A. Babaev. "ASSESSMENT OF THE ACTIVATION TYPE OF GLIAL CELLS AND THE STATE OF MITOCHONDRIA IN ALZHEIMER’S DISEASE MODELING IN VITRO." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-235.

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Alzheimer’s disease is the most common neurodegenerative disease. According to new data, mitochondria are actively involved in the pathogenesis of Alzheimer’s disease, while disorders accumulate in organoids. It is believed that there is a link between the accumulation of mitochondrial defects and inflammation. We conducted a study of the type of glial cell activation, evaluation of the expression patterns of structural mitochondrial genes, auto- and mitophagy genes.
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Lvova, I. D., D. A. Sidorenko, S. A. Tyganov, and K. A. Sharlo. "THE ACCUMULATION OF INTRAMITOCHONDRIAL FORMATION DURING FUNCTIONAL UNLOADING DOES NOT AFFECT THE DEVELOPMENT OF INCREASED FATIGUE OF THE SOLEUS MUSCLE." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-254.

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It was assumed that increased generation of ROS after the first day of functional unloading in m. soleus can lead to disruption of mitochondrial functions and increased muscle fatigue. It was found that mitochondrial ROS during a 7-day functional unloading contribute to the accumulation of Ca2+ inside mitochondria and tropomyosin oxidation, but do not have a significant effect on the functional properties of m. soleus against the background of 7-day functional unloading.
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Sityaeva, D. V., and N. A. Stefanova. "STRUCTURAL AND FUNCTIONAL PARAMETERS OF MITOCHONDRIA IN HIPPOCAMPAL NEURONS OF OXYS RATS DURING EARLY ONTOGENY AND THE INFLUENCE OF EXOGENOUS MELATONIN." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-270.

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Mitochondrial dysfunction is considered one of the key factors initiating the development of Alzheimer’s disease (AD), the most common neurodegenerative disorder. This study aims to assess the structural and functional state of mitochondria in hippocampal neurons of OXYS rats during the early postnatal period, their potential contribution to the development of AD symptoms, and the neuroprotective potential of exogenous melatonin.
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Altaee, Muddather, Samaraa Youns, Abdullah Al-Nuaymia, Sara Dabdob, and Muhammad Alkataan. "The Protective Effects of a Phenolic Clove Extract on Mitochondria: An Animal Study." In 5th International Conference on Biomedical and Health Sciences. Cihan University-Erbil, 2024. http://dx.doi.org/10.24086/biohs2024/paper.1201.

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Background: Plants represent a treasure chest with many unrevealed medical applications that have made them traditionally used for thousands of years to treat different diseases. One of these applications is the hypoglycemic and antioxidant roles of some plants. A pathological condition characterized by hyperglycemia, insulin resistance, and dyslipidemia known as metabolic syndrome. Objectives: This work focuses on examining the hypoglycemic effect of Syzygium aromaticum on a metabolic syndrome-induced animal model and studying the role of the extracted phenolic compounds on different mitochon
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Reports on the topic "Mitochondria"

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Ostersetzer-Biran, Oren, and Alice Barkan. Nuclear Encoded RNA Splicing Factors in Plant Mitochondria. United States Department of Agriculture, 2009. http://dx.doi.org/10.32747/2009.7592111.bard.

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Mitochondria are the site of respiration and numerous other metabolic processes required for plant growth and development. Increased demands for metabolic energy are observed during different stages in the plants life cycle, but are particularly ample during germination and reproductive organ development. These activities are dependent upon the tight regulation of the expression and accumulation of various organellar proteins. Plant mitochondria contain their own genomes (mtDNA), which encode for a small number of genes required in organellar genome expression and respiration. Yet, the vast ma
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Chitayat, Liyam. Mitochondria Are Alive. Asimov Press, 2024. http://dx.doi.org/10.62211/38pe-75hu.

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The physical world is an intricate dance between matter, information, and energy. We have learned to control matter and information, but energy is still a missing pillar. Recognizing that mitochondria are alive will open new horizons into how we learn about, and build with, biology.
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Aachi, Venkat. Preliminary Characterization of Mitochondrial ATP-sensitive Potassium Channel (MitoKATP) Activity in Mouse Heart Mitochondria. Portland State University Library, 2000. http://dx.doi.org/10.15760/etd.1666.

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Kumar, Om. do you know about mitochondria ? ResearchHub Technologies, Inc., 2023. http://dx.doi.org/10.55277/researchhub.11eubx1d.

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Kurtz, Andreas. Mitochondria Polymorphism in Neurofibromatosis Type 1. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada400622.

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Kurtz, Andreas. Mitochondria Polymorphism in Neurofibromatosis Type 1. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada411354.

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Kurtz, Andreas C. Mitochondria Polymorphism in Neurofibromatosis Type 1. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada420949.

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Siedow, J. N. Molecular studies of functional aspects of plant mitochondria. Office of Scientific and Technical Information (OSTI), 1992. http://dx.doi.org/10.2172/7194216.

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Dr. Carol Lynn George, Dr Carol Lynn George. Can our cell's mitochondria power a cell phone? Experiment, 2013. http://dx.doi.org/10.18258/1755.

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Ostersetzer-Biran, Oren, and Jeffrey Mower. Novel strategies to induce male sterility and restore fertility in Brassicaceae crops. United States Department of Agriculture, 2016. http://dx.doi.org/10.32747/2016.7604267.bard.

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Abstract Mitochondria are the site of respiration and numerous other metabolic processes required for plant growth and development. Increased demands for metabolic energy are observed during different stages in the plants life cycle, but are particularly ample during germination and reproductive organ development. These activities are dependent upon the tight regulation of the expression and accumulation of various organellar proteins. Plant mitochondria contain their own genomes (mtDNA), which encode for rRNAs, tRNAs and some mitochondrial proteins. Although all mitochondria have probably evo
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