Relevant bibliographies by topics / Mitotic activity

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Mitotic activity'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Mitotic activity"

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Skoufias, Dimitrios A., Rose-Laure Indorato, Françoise Lacroix, Andreas Panopoulos, and Robert L. Margolis. "Mitosis persists in the absence of Cdk1 activity when proteolysis or protein phosphatase activity is suppressed." Journal of Cell Biology 179, no. 4 (2007): 671–85. http://dx.doi.org/10.1083/jcb.200704117.

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Cellular transition to anaphase and mitotic exit has been linked to the loss of cyclin-dependent kinase 1 (Cdk1) kinase activity as a result of anaphase-promoting complex/cyclosome (APC/C)–dependent specific degradation of its cyclin B1 subunit. Cdk1 inhibition by roscovitine is known to induce premature mitotic exit, whereas inhibition of the APC/C-dependent degradation of cyclin B1 by MG132 induces mitotic arrest. In this study, we find that combining both drugs causes prolonged mitotic arrest in the absence of Cdk1 activity. Different Cdk1 and proteasome inhibitors produce similar results,
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Long, John J., Anne Leresche, Richard W. Kriwacki, and Joel M. Gottesfeld. "Repression of TFIIH Transcriptional Activity and TFIIH-Associated cdk7 Kinase Activity at Mitosis." Molecular and Cellular Biology 18, no. 3 (1998): 1467–76. http://dx.doi.org/10.1128/mcb.18.3.1467.

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ABSTRACT Nuclear transcription is repressed when eukaryotic cells enter mitosis. Mitotic repression of transcription of various cellular and viral gene promoters by RNA polymerase II can be reproduced in vitro either with extracts prepared from cells arrested at mitosis with the microtubule polymerization inhibitor nocodazole or with nuclear extracts prepared from asynchronous cells and the mitotic protein kinase cdc2/cyclin B. Purified cdc2/cyclin B kinase is also sufficient to inhibit transcription in reconstituted transcription reactions with biochemically purified and recombinant basal tra
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Al Jord, Adel, Asm Shihavuddin, Raphaël Servignat d’Aout, et al. "Calibrated mitotic oscillator drives motile ciliogenesis." Science 358, no. 6364 (2017): 803–6. http://dx.doi.org/10.1126/science.aan8311.

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Cell division and differentiation depend on massive and rapid organelle remodeling. The mitotic oscillator, centered on the cyclin-dependent kinase 1–anaphase-promoting complex/cyclosome (CDK1-APC/C) axis, spatiotemporally coordinates this reorganization in dividing cells. Here we discovered that nondividing cells could also implement this mitotic clocklike regulatory circuit to orchestrate subcellular reorganization associated with differentiation. We probed centriole amplification in differentiating mouse-brain multiciliated cells. These postmitotic progenitors fine-tuned mitotic oscillator
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Fabisz-Kijowska, Anna A., Katherine Lumley-Sapanski, Margaret S. Halleck, and Robert A. Schlegel. "Cellular compartmentalization of protein kinase activity during the cell cycle." Biochemistry and Cell Biology 65, no. 12 (1987): 1070–79. http://dx.doi.org/10.1139/o87-140.

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The quantities and types of protein kinases found in the cytoplasmic and nuclear or chromosomal compartments of interphase and mitotic human culture cells were compared. Using histone as substrate, the total quantity of kinases recovered from cytoplasmic and chromosomal fractions of mitotic cells was several times greater than from cytoplasmic and nuclear fractions of interphase cells. In both mitotic and interphase cells, more activity was recovered from cytoplasmic fractions than from chromosomal or nuclear fractions, respectively. When activity against various substrates was examined, mitot
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Popov, Victor I., Igor V. Kraev, Dmitri A. Ignat'ev, and Michael G. Stewart. "Suspension of Mitotic Activity in Dentate Gyrus of the Hibernating Ground Squirrel." Neural Plasticity 2011 (2011): 1–7. http://dx.doi.org/10.1155/2011/867525.

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Neurogenesis occurs in the adult mammalian hippocampus, a region of the brain important for learning and memory. Hibernation in Siberian ground squirrels provides a natural model to study mitosis as the rapid fall in body temperature in 24 h (from 35-36°C to +4–6°C) permits accumulation of mitotic cells at different stages of the cell cycle. Histological methods used to study adult neurogenesis are limited largely to fixed tissue, and the mitotic state elucidated depends on the specific phase of mitosis at the time of day. However, using an immunohistochemical study of doublecortin (DCX) and B
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Maddox, Amy Shaub, and Keith Burridge. "RhoA is required for cortical retraction and rigidity during mitotic cell rounding." Journal of Cell Biology 160, no. 2 (2003): 255–65. http://dx.doi.org/10.1083/jcb.200207130.

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Mitotic cell rounding is the process of cell shape change in which a flat interphase cell becomes spherical at the onset of mitosis. Rearrangement of the actin cytoskeleton, de-adhesion, and an increase in cortical rigidity accompany mitotic cell rounding. The molecular mechanisms that contribute to this process have not been defined. We show that RhoA is required for cortical retraction but not de-adhesion during mitotic cell rounding. The mitotic increase in cortical rigidity also requires RhoA, suggesting that increases in cortical rigidity and cortical retraction are linked processes. Rho-
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Shah, Rajvee, Sanne Jensen, Lisa M. Frenz, Anthony L. Johnson, and Leland H. Johnston. "The Spo12 Protein of Saccharomyces cerevisiae: A Regulator of Mitotic Exit Whose Cell Cycle-Dependent Degradation Is Mediated by the Anaphase-Promoting Complex." Genetics 159, no. 3 (2001): 965–80. http://dx.doi.org/10.1093/genetics/159.3.965.

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Abstract The Spo12 protein plays a regulatory role in two of the most fundamental processes of biology, mitosis and meiosis, and yet its biochemical function remains elusive. In this study we concentrate on the genetic and biochemical analysis of its mitotic function. Since high-copy SPO12 is able to suppress a wide variety of mitotic exit mutants, all of which arrest with high Clb-Cdc28 activity, we speculated whether SPO12 is able to facilitate exit from mitosis when overexpressed by antagonizing mitotic kinase activity. We show, however, that Spo12 is not a potent regulator of Clb-Cdc28 act
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Mohl, Dane A., Michael J. Huddleston, Therese S. Collingwood, Roland S. Annan, and Raymond J. Deshaies. "Dbf2–Mob1 drives relocalization of protein phosphatase Cdc14 to the cytoplasm during exit from mitosis." Journal of Cell Biology 184, no. 4 (2009): 527–39. http://dx.doi.org/10.1083/jcb.200812022.

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Exit from mitosis is characterized by a precipitous decline in cyclin-dependent kinase (Cdk) activity, dissolution of mitotic structures, and cytokinesis. In Saccharomyces cerevisiae, mitotic exit is driven by a protein phosphatase, Cdc14, which is in part responsible for counteracting Cdk activity. Throughout interphase, Cdc14 is sequestered in the nucleolus, but successful anaphase activates the mitotic exit network (MEN), which triggers dispersal of Cdc14 throughout the cell by a mechanism that has remained unknown. In this study, we show that a MEN component, protein kinase Dbf2–Mob1, prom
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Li, Jie, Hiroki Shima, Hironari Nishizawa, et al. "Phosphorylation of BACH1 switches its function from transcription factor to mitotic chromosome regulator and promotes its interaction with HMMR." Biochemical Journal 475, no. 5 (2018): 981–1002. http://dx.doi.org/10.1042/bcj20170520.

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The transcription repressor BACH1 performs mutually independent dual roles in transcription regulation and chromosome alignment during mitosis by supporting polar ejection force of mitotic spindle. We now found that the mitotic spindles became oblique relative to the adhesion surface following endogenous BACH1 depletion in HeLa cells. This spindle orientation rearrangement was rescued by re-expression of BACH1 depending on its interactions with HMMR and CRM1, both of which are required for the positioning of mitotic spindle, but independently of its DNA-binding activity. A mass spectrometry an
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Noree, Chalongrat, and Naraporn Sirinonthanawech. "Nuclear targeted Saccharomyces cerevisiae asparagine synthetases associate with the mitotic spindle regardless of their enzymatic activity." PLOS ONE 15, no. 12 (2020): e0243742. http://dx.doi.org/10.1371/journal.pone.0243742.

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Recently, human asparagine synthetase has been found to be associated with the mitotic spindle. However, this event cannot be seen in yeast because yeast takes a different cell division process via closed mitosis (there is no nuclear envelope breakdown to allow the association between any cytosolic enzyme and mitotic spindle). To find out if yeast asparagine synthetase can also (but hiddenly) have this feature, the coding sequences of green fluorescent protein (GFP) and nuclear localization signal (NLS) were introduced downstream of ASN1 and ASN2, encoding asparagine synthetases Asn1p and Asn2
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Dissertations / Theses on the topic "Mitotic activity"

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Hewitt, Laura. "Using a novel small molecule inhibitor to investigate the role of Mps1 kinase activity." Thesis, University of Manchester, 2011. https://www.research.manchester.ac.uk/portal/en/theses/using-a-novel-small-molecule-inhibitor-to-investigate-the-role-of-mps1-kinase-activity(fcacfefc-90d9-4e92-9af4-a57897737329).html.

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During mitosis, accurate chromosome segregation is essential: gain or loss of genetic information can be detrimental to cell viability, or promote tumourigenesis. The mitotic checkpoint (also known as the spindle assembly checkpoint or SAC) ensures accurate chromosome segregation by delaying cell cycle progression until accuracy can be guaranteed. Mps1 is a protein kinase that is crucial for mitotic checkpoint signalling and also for proper chromosome alignment at metaphase. However, the precise role of Mps1’s catalytic activity is still unclear. Here, I present AZ3146, a novel small molecule
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David, Alain. "Exploring the Regulation of Mitotic PP2A-Rts1 Activity in Saccharomyces cerevisiae." Thesis, Université d'Ottawa / University of Ottawa, 2021. http://hdl.handle.net/10393/42437.

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Protein phosphorylation is an essential post-translational modification used in cells for regulating multiple biological processes in all organisms. Particularly, mitotic onset is regulated in all eukaryotes by an increase in cyclin-dependent kinase 1 (Cdk1) activity caused by the dephosphorylation of Cdk1 on a conserved tyrosine residue. PP2ARts1 is a phosphatase that participates in dephosphorylating the conserved tyrosine residue, tyrosine-19 (Y19). PP2ARts1 dephosphorylates phosphorylated serine and threonine residues. However, in vitro experiments suggest that in conjunction with the mamm
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Nöske, Katharina [Verfasser], and Petra [Akademischer Betreuer] Boukamp. "Mitotic activity of keratinocytes in regeneration and tissue homestasis / Katharina Nöske ; Betreuer: Petra Boukamp." Heidelberg : Universitätsbibliothek Heidelberg, 2015. http://d-nb.info/1180500814/34.

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Nurmenniemi, P. (Petri). "Inflammatory cells and mitotic activity of keratinocytes in gingival overgrowth induced by immunosuppressive- and nifedipine medication." Doctoral thesis, University of Oulu, 2006. http://urn.fi/urn:isbn:9514279964.

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Abstract Both immunosuppressive and nifedipine medication have been associated with drug-induced gingival overgrowth. There are several hypothetical mechanisms for drug-induced gingival overgrowth, such as the influence of genetic predisposition, alterations in gingival tissue homeostasis, especially in the function of fibroblasts, and drug-induced action on growth factors. Clinical studies have also shown that, those with poor oral hygiene status drug-induced gingival overgrowth is more prevalent and severe than those with good oral hygiene status. The working hypothesis was that immunosupp
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Potapova, Tamara. "Exploring mechanisms that control the activity of cyclin-dependent kinase 1 during mitotic transitions in somatic cells." Oklahoma City : [s.n.], 2009.

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Pokrovskii, Daniil [Verfasser], and Ralph [Akademischer Betreuer] Rupp. "Stage-specific histone modification profiles are shaped by mitotic activity during Xenopus development / Daniil Pokrovskii ; Betreuer: Ralph Rupp." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2019. http://d-nb.info/1238016863/34.

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Ridgway, Ellen. "Investigation into the role of Aurora A kinase activity during mitosis." Thesis, University of Manchester, 2010. https://www.research.manchester.ac.uk/portal/en/theses/investigation-into-the-role-of-aurora-a-kinase-activity-during-mitosis(58d806c8-8393-4bab-b91d-dcf6f58c0012).html.

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Aurora A is an important mitotic regulator that has been found to be up-regulated in a variety oftumours provoking a great deal of attention and the development of a number of small moleculeAurora kinase inhibitors. Most of these inhibitors though have predominantly targeted Aurora B,meaning that our understanding of the role of the kinase activity of Aurora A is comparatively lesswell developed.MLN8054 however, is a small molecule inhibitor that has been reported in vitro to have a highdegree of specificity towards Aurora A activity. In this thesis, I show in vivo that MLN8054 can beused to s
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Hafizi, Fatima. "Characterization of the Interactions of the Bacterial Cell Division Regulator MinE." Thèse, Université d'Ottawa / University of Ottawa, 2012. http://hdl.handle.net/10393/23189.

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Symmetric cell division in gram-negative bacteria is essential for generating two equal-sized daughter cells, each containing cellular material crucial for growth and future replication. The Min system, comprised of proteins MinC, MinD and MinE, is particularly important for this process since its deletion leads to minicells incapable of further replication. This thesis focuses on the interactions involving MinE that are important for allowing cell division at the mid-cell and for directing the dynamic localization of MinD that is observed in vivo. Previous experiments have shown that the MinE
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Wang, Yun-Chi, and 王韻琪. "Aurora kinase A increased CDK5 activity in mitotic phase." Thesis, 2016. http://ndltd.ncl.edu.tw/handle/46895235741016649470.

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碩士<br>國立中興大學<br>生命科學系所<br>104<br>Mitosis is a very important part in cell cycle and regulated by not only extracellular factors, but also intracellular signaling. The Aurora kinase A (Aurora A) is a mitosis associated protein kinase, which localizes to centrosomes and nearby spindle microtubules, plays an important role in the centrosome maturation and spindle separation during mitosis (M phase). In that phase, the amount of Aurora A shows the highest expression compared to other phases. Cyclin-dependent kinases (CDKs) are Ser/Thr protein kinases that associate with specific cyclin subunits in
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Saft, Mallory Sue. "Two studies of mitotic apparatus isolated from sea urchin embryos CPK activity and localized Ca⁺²-sequestration." 1986. http://catalog.hathitrust.org/api/volumes/oclc/13477739.html.

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Thesis (M.S.)--University of Wisconsin--Madison, 1986.<br>Typescript. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 81-88).
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Books on the topic "Mitotic activity"

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Burton, Derek, and Margaret Burton. The integument. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198785552.003.0002.

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The skin is the boundary between fish and environment and possesses important boundary functions such as protection and camouflage. Fish skin is mucigenic, contrasting with keratinized skin in terrestrial vertebrates. Structurally, there is an outer epidermis, a dermis and an inner hypodermis, the entire mucigenic epidermis remaining alive, with mitotic cells, unlike a keratinized epidermis. A variety of specialized epidermal cells are described, and the role of the ‘bias-sleeve’ orientation of dermal collagen is discussed. Scales, scutes and bony plates have protective roles. The variety of m
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Freifeld, Alison G. An Introduction. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199938568.003.0100.

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This chapter focuses on solid tumors and how they can be treated. Solid tumors, lymphomas, and leukemias represent a widely diverse array of cancers. Until recently, the general approach to treating all of them was to administer cytotoxic anticancer drugs that damage proliferating cells by interfering with mitosis and other essential steps in cellular replication. Localized solid tumors are largely treated by surgical resection and radiotherapy, with cytotoxic chemotherapy being commonly used adjunctively or in cases of metastatic disease. A major drawback of this approach has been the lack of
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Book chapters on the topic "Mitotic activity"

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Cater, D. B., and M. P. Stack-Dunne. "Mitotic Activity in the Adrenal Cortex Studied in the Rat." In Novartis Foundation Symposia. John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470715208.ch3.

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Aufderheide, M., M. Kohler, S. Hammann, and M. Riebe. "Effects of Carbendazim on the Mitotic Activity of the Small Intestine." In Environmental Hygiene II. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-46712-7_21.

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Kiyomitsu, Tomomi. "Using Optogenetics to Spatially Control Cortical Dynein Activity in Mitotic Human Cells." In Methods in Molecular Biology. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-2958-1_5.

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Sinzinger, H. "Inhibition of Mitotic and Proliferative Activity of Smooth Muscle Cells by Prostaglandin E1." In Prostaglandin E1 in Atherosclerosis. Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71679-9_6.

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Vallade, Jean. "Quantitative Data on Petunia Embryogenesis: Mitotic Activity and Characteristics of the Cell Cycles." In Sexual Reproduction in Higher Plants. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73271-3_62.

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Borchard, F., A. S. C. Barreto, and P. Pfitzer. "Nuclear Diameter, Mitotic Activity, and DNA Content in Various Lesions of the Gastric Mucosa." In Progress in Surgical Pathology. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-662-12820-6_7.

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Sperling, Karl, Sabine Henschel, Ilka Schulze, and Heidemarie Neitzel. "Constitutive Heterochromatin of Microtus agrestis: Molecular Organization and Genetic Activity in Mitotic and Meiotic Cells." In Chromosomes Today. Springer Netherlands, 2004. http://dx.doi.org/10.1007/978-94-017-1033-6_22.

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Schuyler, Scott C., and Andrew W. Murray. "An In Vitro Assay for Cdc20-Dependent Mitotic Anaphase-Promoting Complex Activity from Budding Yeast." In Methods in Molecular Biology. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-993-2_17.

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Fatma, Homa, and Hifzur R. Siddique. "Luciferase-Based Reporter Assay for the Assessment of Aurora A-Kinase Activity in Mitotic Cycle." In Methods in Molecular Biology. Springer US, 2024. http://dx.doi.org/10.1007/978-1-0716-4236-8_12.

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Nurse, C. A., and C. Vollmer. "Effects of Hypoxia on Cultured Chemoreceptors of the Rat Carotid Body: DNA Synthesis and Mitotic Activity in Glomus Cells." In Advances in Experimental Medicine and Biology. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2966-8_12.

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Conference papers on the topic "Mitotic activity"

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Tsomartova, Dibakhan, Ekaterina Timokhina, Nataliya Yaglova, Sergey Obernikhin, and Valentin Yaglov. "MITOTIC ACTIVITY OF LYMPHOCYTES IN PREPUBERTAL RATS EXPOSED TO LOW DOSES OF DDT." In NEW TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. Institute of information technology, 2021. http://dx.doi.org/10.47501/978-5-6044060-1-4.13.

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Low-dose developmental exposure to DDT results in inhibited mitotic activity of thymic lymphocytes in prepubertal rats. Decreased number of proliferating cells in deeper layers of the cortex and in the medulla, not subcapsular lymphoblasts, contributes to lowered proliferation rate.
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Sinzinger, H., T. Zidek, P. Fitscha, O. Wagner, and Waltraud Rogatti. "PGI2 AND PGE1 INHIBIT SMOOTH MUSCLE CELL PROLIFERATION MITOTIC ACTIVITY AND EXTRACELLULAR MATRIX FORMATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643798.

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Earlier findings that activated smooth muscle cells (SMC) generate more PGI2 indicate a pathophysiological role of PG's in SMC-proliferation. In a rabbit experiment enhanced mitotic activity (MA) induced by ACTH or desoxycorticosterone acetate(DCS) is significantly depressed by both PGI2 and El.most effective.Similarly, a decrease in collagen and glycosamino-glycan-formation canbe noted. PGI2 is able to inhibit quite selectively liberation of platelet derived growthfactor (PDGF). PDGF itself stimulates vascular PGI2-generation,inhibiting further PGDF-relese long-1lasting intermittent alternati
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Pati, Pushpak, Raul Catena, Orcun Goksel, and Maria Gabrani. "A deep learning framework for context-aware mitotic activity estimation in whole slide images." In Digital Pathology, edited by John E. Tomaszewski and Aaron D. Ward. SPIE, 2019. http://dx.doi.org/10.1117/12.2512705.

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Ben-Salem, Salma, Salam Bachour, Varadha Balaji Venkadakrishnan, et al. "Abstract 390: Prostate cancer progression depends on the activity of the mitotic kinase citron kinase." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-390.

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Ben-Salem, Salma, Salam Bachour, Varadha Balaji Venkadakrishnan, et al. "Abstract 390: Prostate cancer progression depends on the activity of the mitotic kinase citron kinase." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-390.

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Venugopal, Anand, Satish Ramalingam, Dharmalingam Subramaniam, Zhiyun He, Roy Jensen, and Shrikant Anant. "Abstract 3080: Novel splicing activity for RNA binding protein CUGBP2 in radiation-induced mitotic catastrophe." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-3080.

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Klintman, M., C. Strand, E. Gudlaugsson, et al. "Abstract P2-10-19: Are the mitotic factors Mitotic Activity Index (MAI) and Phosphohistone 3 (PPH3) stronger prognostic proliferation factors than Ki67 in node-negative breast cancer?" In Abstracts: Thirty-Fifth Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 4‐8, 2012; San Antonio, TX. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/0008-5472.sabcs12-p2-10-19.

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Dergacheva, N. I., I. O. Suchkova, L. K. Sasina, and E. L. Patkin. "CADMIUM CHLORIDE EXPOSURE FOR 48 AND 72 HOURS AFFECTS THE MITOTIC ACTIVITY OF HEPG2 AND HEK293 CELLS." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-316.

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Venkatasubbu, Thirulogachandar. "Floral development and growth dynamics are influenced by the spatio-temporal mitotic activity of the inflorescence meristem in barley." In ASPB PLANT BIOLOGY 2020. ASPB, 2020. http://dx.doi.org/10.46678/pb.20.1332422.

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Chan, Tim Hon Man, Leilei Chen, and Xin-Yuan Guan. "Abstract 2985: CHD1L upregulates TCTP and induces mitotic defects and chromosome missegregation through decreasing Cdk1 activity by Cdc25C ubiquitination." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-2985.

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Reports on the topic "Mitotic activity"

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Grafi, Gideon, and Brian Larkins. Endoreduplication in Maize Endosperm: An Approach for Increasing Crop Productivity. United States Department of Agriculture, 2000. http://dx.doi.org/10.32747/2000.7575285.bard.

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The focus of this research project is to investigate the role of endoreduplication in maize endosperm development and the extent to which this process contributes to high levels of starch and storage protein synthesis. Although endoreduplication has been widely observed in many cells and tissues, especially those with high levels of metabolic activity, the molecular mechanisms through which the cell cycle is altered to produce consecutive cycles of S-phase without an intervening M-phase are unknown. Our previous research has shown that changes in the expression of several cell cycle regulatory
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Levy, Avraham A., and Virginia Walbot. Regulation of Transposable Element Activities during Plant Development. United States Department of Agriculture, 1992. http://dx.doi.org/10.32747/1992.7568091.bard.

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We have studied the regulation of the maize Ac and MuDR transposable elements activities during plant development. Ac was studied in an heterologous system (transgenic tobacco plants and cell suspensions) while MuDR was studied in the native maize background. The focus of this study was on the transcriptional regulation of Ac and MuDR. For Ac, the major achievements were to show that 1-It is autoregulated in a way that the Ac-encoded transposase can repress the activity of its own promoter; 2-It is expressed at low basal level in all the plant organs that were studied, and its activity is stro
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