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Books on the topic 'Mobility of temporomandibular joints'

Author: Grafiati

Published: 24 April 2022

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1

Antoniotti, Terri. Exercise and total well being for vertebral and craniomandibular disorders. Tucson, AZ: International Fundamental Rocabado Center, 1990.

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2

Schneider, W. Mobility: Theory and practice. Stuttgart: G. Thieme Verlag, 1992.

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3

Schroeder, Hubert E. Oral structure biology: Embryology, structure, and function of normal hard and soft tissues of the oral cavity and temporomandibular joints. Stuttgart: G. Thieme Verlag, 1991.

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4

Halligan, Eugene P. A study of collagen abnormalities in subjects with limited joint mobility and their role in the development of diabetic complications. Dublin: University College Dublin, 1995.

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5

1931-, Horns John Willard, and Gold Richard H, eds. Clinical arthrography. 2nd ed. Baltimore: Williams & Wilkins, 1985.

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6

Arndt, Rolf-D. Clinicalarthrography. 2nd ed. Baltimore: Williams & Wilkins, 1985.

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7

Manual physical therapy of the spine. St. Louis: Saunders, 2009.

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8

Frawley, Geoff. Mucopolysaccharidoses. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0064.

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The mucopolysaccharidoses (MPS) are a group of seven chronic progressive diseases caused by deficiencies of 11 different lysosomal enzymes required for the catabolism of glycosaminoglycans (GAGs). Hurler syndrome (MPS IH) is an autosomal recessive storage disorder caused by a deficiency of α‎-L-iduronidase. Hunter syndrome (MPS II) is an X-linked recessive disorder of metabolism involving the enzyme iduronate-2-sulfatase. Many of the MPS clinical manifestations have potential anesthetic implications. Significant airway issues are particularly common due to thickening of the soft tissues, enlarged tongue, short immobile neck, and limited mobility of the cervical spine and temporomandibular joints. Spinal deformities, hepatosplenomegaly, airway granulomatous tissue, and recurrent lung infections may inhibit pulmonary function. Odontoid dysplasia and radiographic subluxation of C1 on C2 is common and may cause anterior dislocation of the atlas and spinal cord compression.

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9

Isberg, Annika. Temporomandibular Joint Dysfunction: A Practitioner's Guide. Martin Dunitz Publishers, 2002.

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10

deBurgh, Norman John Edgar, and Bramley Paul, eds. Textbook and color atlas of the temporomandibular joint: Diseases, disorders, surgery. Chicago: Year Book Medical Publishers, 1990.

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11

Radiologic reproduction: Of healthy and arthritic temporomandibular joints of children and adolescents. Stockholm: Gothia, 1997.

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12

Tsatsouline, Pavel. Super Joints: Russian Longevity Secrets for Pain-Free Movement, Maximum Mobility & Flexible Strength. Dragon Door Publications, 2001.

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13

Schneider, Werner, Thomas Tritschler, and Hans Spring. Mobility: Theory and Practice (Trauma Management). Thieme Medical Publishers, 1992.

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14

author, Smith William 1976, and Volkmar Michael author, eds. Mobility workout handbook: With over 100 workouts for the everyday athlete : improve performance, reduce injury, increase flexibility. 2016.

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15

Restoring flexibility: A gentle yoga-based practice to increase mobility at any age. Ulysses Press, 2015.

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16

Manual Physical Therapy of the Spine. Elsevier - Health Sciences Division, 2015.

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17

Jordan, Joanne M., Kelli D. Allen, and Leigh F. Callahan. Age, gender, race/ethnicity, and socioeconomic status in osteoarthritis and its outcomes. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0010.

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Osteoarthritis (OA) is the most common joint condition worldwide. It can impair mobility and result in significant disability, need for total joint replacement, and healthcare utilization. OA is unusual in those younger than 40 years, then commonly the result of an underlying metabolic disorder or a prior joint injury. Some geographic and racial/ethnic variation exists in the prevalence and incidence of OA for specific joints, likely due to variation in genetics, anatomy, and environmental exposures. Many OA outcomes vary by socioeconomic status and other social factors. This chapter describes demographic and social determinants of knee, hip, and hand OA, including how these factors impact radiographic and symptomatic OA, OA-related pain and function, and its treatment.

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18

Kloppenburg, Margreet. Clinical assessment: signs, symptoms, and patient perceptions in osteoarthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0015.

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Osteoarthritis (OA) is a disorder that can affect any joint. It results in a high clinical burden in many patients. Patients with OA experience a wide range of symptoms and clinical signs such as pain, disability, stiffness, tenderness, crepitus, and decreased mobility and strength in their osteoarthritic joints, where the impact depends on the involved joint. Also general symptoms such as fatigue and psychosocial consequences are experienced by OA patients. The impact of symptoms and signs does not only depend on osteoarthritic abnormalities, but also on patient factors, such as coping strategies and illness perceptions, and co-morbidities. In this chapter, general and joint-specific symptoms and signs as well as the impact of patient factors are discussed.

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19

Shaibani, Aziz. Distal Arm Weakness. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199898152.003.0015.

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Distal arm weakness may be caused by involvement of the intrinsic hand muscles (interossi, thenar and hypothenar muscles, lumbricals) or extrinsic hand muscles (long finger flexors and extensors). ALS is typical for the former type, and IBM is typical for the latter type. Incoordination of skilled finger movement due to cerebellar disease is associated with normal strength. Poor mobility due to joints pain and swelling should not be confused with muscle weakness. Mononeuropathies such as ulnar, radial, median, and AIN,lesions usually cause differential loss of function. Myasthenia sometimes causes weakness of the wrist and finger extensors. A small but distinct group of distal hereditary myopathies should always be borne in mind. Progressive sensorimotor neuropathies are usually associated with sensory symptoms. Multifocal motor neuropathy can be a diagnostic challenge.

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20

Hems, T. E. J. Reconstruction after nerve injury. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.006009.

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♦ Late reconstructive procedures may improve function if there is persisting paralysis after nerve injury♦ Transfer of a functioning musculotendinous unit to the tendon of the paralysed muscle is the most common type of procedure♦ Passive mobility must be maintained in affected joints before tendon transfer can be performed♦ The transferred muscle should be expendable, have normal power, and have properties appropriate to the function it is required to restore♦ Tendon transfers can provide reliable improvement in function after isolated radial nerve palsy♦ A number of procedures have been described for reconstruction of thumb opposition but impaired sensation after median nerve injury may limit gain in function♦ Tendon transfers are possible to improve clawing of fingers and lateral pinch of the thumb after ulnar nerve palsy or other cases of intrinsic paralysis.

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