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Books on the topic 'Molecular radiotherapy'

Author: Grafiati
Published: 4 June 2021
Last updated: 7 July 2024

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1

J, Piccart Martine, ed. Breast cancer and molecular medicine. Berlin: Springer, 2006.

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2

Molecular imaging for integrated medical therapy and drug development. Tokyo: Springer, 2010.

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3

Marikki, Laiho, and SpringerLink (Online service), eds. Molecular Determinants of Radiation Response. New York, NY: Springer Science+Business Media, LLC, 2011.

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4

D, Ford Thomas, ed. New cancer research developments. Hauppauge, NY: Nova Science Publishers, 2009.

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5

1965-, Shumate Mark J., Kooby David A. 1967-, and Society of Nuclear Medicine (1953- ), eds. A clinician's guide to nuclear oncology: Practical molecular imaging and radionuclide therapies. Reston, VA: SNM, 2007.

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6

Molls, Michael. The Impact of Tumor Biology on Cancer Treatment and Multidisciplinary Strategies. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009.

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7

Vohra, Akhtar Abdullah. Observations on intercellular adhesion molecules in patients undergoing radiotherapy for cancer. Manchester: University of Manchester, 1996.

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8

Cancer Radiotherapy (Methods in Molecular Medicine). Humana Press, 2008.

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9

Schwaiger, Markus. From Morphological Imaging to Molecular Targeting. Springer, 2013.

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10

Cordes, Nils, Michael Baumann, and Mechthild Krause. Molecular Radio-Oncology. Springer London, Limited, 2016.

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11

Cordes, Nils, Michael Baumann, and Mechthild Krause. Molecular Radio-Oncology. Springer, 2018.

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12

Molecular Radio-Oncology. Springer, 2016.

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13

Schwaiger, Markus, Ludger Dinkelborg, and Hermann Schweinfurth. From Morphological Imaging to Molecular Targeting: Implications to Preclinical Development. Springer, 2013.

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14

Breast Cancer Management and Molecular Medicine. Springer, 2007.

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15

Hodgkiss, Andrew. Psychiatric consequences of cancer treatments: surgery and radiotherapy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198759911.003.0005.

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Surgery and radiotherapy for cancers can disrupt mental health through direct biological mechanisms in addition to the well-described psychological distress associated with the physical consequences of treatment. Upper bowel surgery and bilateral oophorectomy both frequently provoke psychopathology. Total gastrectomy, or ileal resection, causes an inevitable vitamin B12 deficiency. The molecular mechanisms by which these surgical treatments provoke depressive symptoms, or even a dementia, are considered. Raised homocysteine levels and reduced SAM availability are involved. Chronic gonadal oestrogen deprivation increases the risk of anxiety, depression, and dementia in later life. The likely molecular basis for this is discussed. Hypothyroidism, with its associated psychopathology, complicates radiotherapy for head & neck cancers in 40 per cent of patients. The chapter closes with a review of the effects of whole-brain radiotherapy on cognitive function, and the psychopathology arising from radiation-induced hypopituitarism.
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16

Tamaki, Nagara, and Yuji Kuge. Molecular Imaging for Integrated Medical Therapy and Drug Development. Springer, 2014.

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17

DeWeese, Theodore L., and Marikki Laiho. Molecular Determinants of Radiation Response. Springer, 2013.

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18

DeWeese, Theodore L., and Marikki Laiho. Molecular Determinants of Radiation Response. Springer, 2011.

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19

Connor, Thomas, and Patrick H. Maxwell. Molecular basis of renal tumour syndromes. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0328.

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Kidney cancer is among the most common adult malignancy. It accounts for over 3% of all new cases of cancer diagnosed in men and around 2% of all cancers in women in the United Kingdom. In the United States, 1 in 75 people will develop renal cancer in their lifetime and approximately one-third will have metastatic disease at presentation. Kidney cancer has a notoriously poor response to chemotherapy and radiotherapy, but treatment has evolved significantly in the past 10 years. Key to these recent developments in therapy has been a revolution in our understanding of the molecular basis of the renal tumour syndromes which are described in this chapter. Two to three per cent of cases of renal cancer are recognized as due to these syndromes, but they are likely to be recurrent, and to occur in other family members. Seven genes are currently implicated in these syndromes.
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20

Boterberg, Tom, Karin Dieckmann, and Mark Gaze, eds. Radiotherapy and the Cancers of Children, Teenagers, and Young Adults. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198793076.001.0001.

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As most cancers occur in middle-aged or older adults, only a very small proportion of the overall radiotherapy workload relates to children and young people. As there is a wide spectrum of different cancer types in this age group, not only is paediatric cancer uncommon overall but each individual type is very rare. There are many ways in which to deliver radiotherapy, including advanced photon techniques, proton beam therapy, brachytherapy, and molecular radiotherapy. For these reasons, the care of children and young people requiring radiotherapy is limited to a small number of highly specialist centres. Delivery of high-quality paediatric radiotherapy requires a multiprofessional team including radiation or clinical oncologists, therapy radiographers, physicists, dosimetrists, anaesthetists, and play specialists. This team has to interact very closely with the wider paediatric and adolescent oncology multidisciplinary team, which includes oncologists, surgeons with different anatomical expertise, radiologists, and pathologists. Children, with their developing tissues and organs, are more susceptible to long-term radiation-induced complications, including second cancers, than adults. The art of paediatric radiotherapy, therefore, is to select treatment approaches which offer the maximum chance of cure while minimizing the risk of adverse effects. Careful teamwork, peer review of radiotherapy planning, and quality assurance within a clinical trial framework offer the best chances of achieving this balance. This book covers all these aspects, highlighting the need for highly specialist teams with the extensive knowledge and the broad skillset required to offer children and young people the best possible treatments.
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21

Theeler, Brett J., and Mark R. Gilbert. Primary Central Nervous System Tumors. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0129.

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Ependymomas are rare primary central nervous system (CNS) tumors that are thought to arise from ependymal cells lining the ventricular system located throughout the CNS. Ependymomas occur in all age groups but are more common in the pediatric population. Ependymomas typically present as mass lesions within the ventricular system, brain or spinal cord parenchyma. As with most central nervous system tumors, pathologic evaluation is required for definitive diagnosis. Ependymomas are typically treated with a combination of surgery and radiotherapy although this varies depending on tumor location, tumor grade, patient age, extent of tumor resection, and other pretreatment factors. Recent molecular studies demonstrate molecularly defined tumor heterogeneity that appears to have a region-specific pattern. Translating the emerging molecular profiles of ependymomas into improved treatment strategies is the primary goal of ongoing research efforts.
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22

Erba, Paola A., Steven M. Larson, Giuliano Mariani, Duccio Volterrani, and H. William Strauss. Nuclear Oncology: From Pathophysiology to Clinical Applications. Springer International Publishing AG, 2022.

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23

(Editor), Steven M. Larson, and H. William Strauss (Editor), eds. Nuclear Oncology: Basic Principles and Clinical Applications. Springer, 2006.

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24

Erba, Paola A., Steven M. Larson, Giuliano Mariani, Duccio Volterrani, and H. William Strauss. Nuclear Oncology: From Pathophysiology to Clinical Applications. Springer International Publishing AG, 2022.

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25

MacCallum, Niall S. Management of oncological complications in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0376.

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Important treatment complications relevant to critical care are discussed. Cancer-related pain is complex and requires multidisciplinary care, particularly in the peri-operative setting. Chemotherapeutic complications include pancytopenia, cardiac, pulmonary, renal, gastrointestinal, hepatic, and neurotoxicity. Radiotherapy complications include cardiac, pulmonary, and gastrointestinal toxicity. In general, management includes assessing the risk-benefit to cytotoxic therapy withdrawal and supportive care. There is a paucity of proven treatment options for most complications, althoughcertain therapies are used to prevent and/or treat complications (e.g. tumour lysis syndrome). Thromboembolic disease is a common cause of mortality and morbidity; low molecular weight heparin therapy may be superior to oral anticoagulation.
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26

Cassidy, Jim, Donald Bissett, Roy A. J. Spence OBE, Miranda Payne, and Gareth Morris-Stiff, eds. Oxford Handbook of Oncology. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199689842.001.0001.

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Oxford Handbook of Oncology has been the essential go-to guide for students and practitioners in oncology for over a decade. The scientific basis and diagnosis of cancers is covered, as well as drugs, biomarkers, and the presentation and psychosocial aspects of oncology. Concise, practical, and comprehensive, there is no better companion for both common conditions and challenging emergencies. This handbook incorporates changes such as the understanding of the science of cancer, novel therapies in breast, lung, renal, and melanoma, molecular sub-classification of common solid cancers, personalized therapy approaches, new agents in hard to treat cancers, the benefits of new technologies in radiotherapy, and the emerging data on the importance of the immune response.
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27

Brady, Luther W., Michael Molls, Carsten Nieder, Peter Vaupel, Mitchell Steven Anscher, and Hans-Peter Heilmann. The Impact of Tumor Biology on Cancer Treatment and Multidisciplinary Strategies. Springer, 2011.

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28

Kerr, David J., Daniel G. Haller, Cornelis J. H. van de Velde, and Michael Baumann, eds. Oxford Textbook of Oncology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199656103.001.0001.

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This new 3rd edition of the Oxford Textbook of Oncology is a major piece of scholarly work that details the evolution, evidence base and current best practice in multidisciplinary practice in oncology. Oncology is the largest hospital multi-specialty and the contributors to this 3rd edition are all internationally recognised key opinion leaders in their field. It includes all aspects of oncology, including surgery, radiotherapy, and medical oncology. It focuses on the clinical aspects of oncology while also covering the basic sciences. It provides introductory chapters covering basic science (cell and molecular biology and genetics), translational science (pharmacology, pharmo-genetics, drug discovery, drug development, radiation oncology), general principles of surgical radiation and medical oncology, cancer imaging and pathology, followed by a series of disease-associated chapters.
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29

Weller, Michael, Michael Brada, Tai-Tong Wong, and Michael A. Vogelbaum. Astrocytic tumours: diffuse astrocytoma, anaplastic astrocytoma, glioblastoma, and gliomatosis cerebri. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0003.

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Astrocytic gliomas are primary brain tumours thought to originate from neural stem or progenitor cells. They are assigned grades II, III, or IV by the World Health Organization according to degree of malignancy as defined by histology. The following molecular markers are increasingly used for diagnostic subclassification or clinical decision-making: 1p/19q co-deletion status, O6-methylguanine-DNA methyltransferase (MGMT) promoter methylation status, and isocitrate dehydrogenase 1 and 2 mutation status. Extent of resection is a favourable prognostic factor, but surgery is never curative. Radiotherapy prolongs progression-free survival across all astrocytic glioma entities. Alkylating agent chemotherapy is an active treatment in particular for patients with MGMT promoter-methylated tumours. Anti-angiogenic therapies have failed to improve survival, and the current focus of major clinical trials is on novel targeted agents or on immunotherapy.
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30

Soffietti, Riccardo, Hugues Duffau, Glenn Bauman, and David Walker. Neuronal and mixed neuronal–glial tumours. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0008.

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Neuronal and mixed neuronal-glial tumours are rare tumours of the central nervous system that occur more commonly in children. Despite a generally benign course, most tumours cause medically intractable seizures, and have been denominated as ‘long-term epilepsy-associated tumours’. The World Health Organization classification distinguishes nine histological variants: dysplastic gangliocytoma of the cerebellum/Lhermitte–Duclos disease, desmoplastic infantile astrocytoma and ganglioglioma, dysembryoplastic neuroepithelial tumour, gangliocytoma and ganglioglioma, central neurocytoma and extraventricular neurocytoma, cerebellar liponeurocytoma, papillary glioneuronal tumour, rosette-forming glioneuronal tumour of the fourth ventricle, and spinal paraganglioma. Early surgery with complete resection may significantly improve the likelihood of postoperative epilepsy freedom. Conformal radiotherapy can be considered in case of patients with incompletely resected symptomatic tumours, atypical or high-grade tumours, or in the case of multiple recurrences despite resections. The role of chemotherapy in these lesions remains poorly defined, while targeted therapies are now available to impact some molecular alterations.
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31

Grant, Robert. Tumours of the brain and skull. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0624.

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Over the last 10 years, there have been several important advances in cell biology, molecular genetics, and targeted therapies in neuro-oncology. Improved neurosurgical techniques such as frameless stereotaxy, awake craniotomy, and intra-operative MRI, safer methods of directing radiotherapy, new chemotherapy approaches, and novel modalities of therapy provide optimism that there will eventually be some improvements in treatment-related morbidity and survival. There has also been an increasing change from individual clinician decision making to decision making by multidisciplinary teams of neurosurgeons, neurologists, clinical oncologists, neuropathologists, neuroradiologists, and specialist nurses with the aim of improving decision making, management planning across specialties, communication, and enrolment in suitable clinical trials. In addition, Good Clinical Practice guidelines, an international ethical and scientific quality standard for designing, conducting, recording, and reporting trials, increases the onus and responsibilities on clinical investigators to perform trials to the highest standard and to have the trials externally monitored, and the trial conduct and results audited. While these obligatory and statutory responsibilities are labour intensive and time consuming, they should improve the quality of trials by limiting the possibility of unintentional bias or fraud. Improving the recording of serious adverse event reporting through trial quality assurance and quality control procedures will help ensure that a balanced view of the effects of a drug or procedure is identified earlier than in the past. It will be interesting to see how research develops over the next decade.
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