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Books on the topic 'Monoamino oxidase'

Author: Grafiati

Published: 4 June 2021

Last updated: 18 February 2022

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1

Oreland, Lars, and Brian A. Callingham, eds. Monoamine Oxidase Enzymes. Springer Vienna, 1987. http://dx.doi.org/10.1007/978-3-7091-8901-6.

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2

Szelenyi, I., ed. Inhibitors of Monoamine Oxidase B. Birkhäuser Basel, 1993. http://dx.doi.org/10.1007/978-3-0348-6348-3.

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3

Ebrahimi, Soltan Ahmed. Characterisation of guinea pig liver monoamine oxidase. University of Portsmouth, School of Pharmacy and Biomedical Science, 1995.

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4

F, Tipton Keith, ed. Amine oxidases: Function and dysfunction : proceedings of the 5th International Amine Oxidase Workshop, Galway, Ireland, August 22-25, 1992. Springer-Verlag, 1994.

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5

Yu, Wai Haung. Desferrioxamine analysis by high performance liquid chromatography, stability and metabolic inhibition by monoamine oxidase inhibitors. National Library of Canada, 1994.

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6

Riederer, Peter, and Moussa Youdim. Monoamine Oxidases and Their Inhibitors. Elsevier Science & Technology Books, 2011.

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7

Monoamine Oxidase and Its Inhibition. Wiley & Sons, Incorporated, John, 2009.

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8

Monoamine Oxidase and their Inhibitors. Elsevier, 2011. http://dx.doi.org/10.1016/c2009-0-01862-5.

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9

H, Yasuhara, ed. Monoamine oxidase: Basic and clinical aspects. VSP, 1993.

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10

1939-, Oreland L., Callingham B. A, and Workshop on Amine Oxidases (2nd : 1986 : Biomedical Center, Uppsala University), eds. Monoamine oxidase enzymes: Review and overview. Springer-Verlag, 1987.

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11

Oreland, Lars. Monoamine Oxidase Enzymes: Review And Overview. Springer, 1987.

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12

1938-, Lieberman A. N., ed. Monoamine oxidase inhibitors in neurological diseases. M. Dekker, 1994.

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13

Parvez, Hasan. Monoamine Oxidase: Basic And Clinical Aspects. Brill Academic Publishers, 1993.

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14

H, Kennedy Sidney, ed. Clinical advances in monoamine oxidase inhibitor therapies. American Psychiatric Press, 1994.

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15

Depression and reversible monoamine oxidase inhibitors: New perspectives. Royal College of Psychiatrists, 1989.

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16

G, Priest R., ed. Depression and reversible monoamine oxidase inhibitors: New perspectives. Royal College of Psychiatrists, 1989.

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17

Tipton, K. F., M. B. H. Youdim, and C. J. Barwell. Amine Oxidases: Function and Dysfunction : Proceedings of the 5th International Amine Oxidase Workshop, Galway, Ireland, August 22-25, 1992 (Journal of Neural Transmission Supplementum). Springer-Verlag Telos, 1994.

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18

Michel, Bourin, ed. Les IMAO. Ellipses-Marketing, 1993.

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19

Sharma, Sushil K. Monoamine Oxidase Inhibitors: Clinical Pharmacology, Benefits, and Potential Health Risks. Nova Science Publishers, Incorporated, 2016.

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20

Oreland, L. Monoamine Oxidase Enzymes: Review and Overview (Journal of Neural Transmission Supplementum). Springer, 1987.

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21

Szelenyi and Istvan. Inhibitors of Monoamine Oxidase B: Pharmacology And Clinical Use In Neurodegenerative Disorders. Birkhäuser, 2013.

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22

1941-, Szelenyi I., ed. Inhibitors of monoamine oxidase B: Pharmacology and clinical use in neurodegenerative disorders. Birkhäuser, 1993.

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23

D, Burrows Graham, and Da Prada M, eds. Reversible MAO-A inhibitors as antidepressants: Basic advances and clinical perspectives. Springer-Verlag, 1989.

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24

O’Donnell, Aidan. Psychiatric disorders and drugs. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198719410.003.0012.

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This chapter describes the anaesthetic management of the patient with those psychiatric disorders which are relevant to anaesthetic practice, including dementia, alcoholism, and anorexia nervosa. Psychiatric medications which can affect anaesthetic practice are described, including monoamine oxidase inhibitors, antipsychotic drugs, and lithium. Anaesthesia for drug-misusing patients and electroconvulsive therapy is described. Sedation of agitated patients on the ward is discussed.

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25

M, Yu Peter, Boulton A. A, and Tipton Keith F, eds. Current neurochemical and pharmacological aspects of biogenic amines: Their function, oxidative deamination, and inhibition. Elsevier, 1995.

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26

M, Yu Peter, Boulton A. A, and Tipton Keith F, eds. Current neurochemical and pharmacological aspects of biogenic amines: Their function, oxidative deamination, and inhibition. Elsevier, 1995.

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27

Moller, H. J. Renaissance of Monoamine Oxidase Inhibitors: The New Selective And Reversible Generation (International Congress & Symposium). Royal Society of Medicine Press Ltd, 1992.

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28

Jes, Olesen, and Edvinsson Lars, eds. Headache pathogenesis: Monoamines, neuropeptides, purines, and nitric oxide. Lippincott-Raven, 1997.

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29

Hodgkiss, Andrew. Psychiatric consequences of cancer treatments: conventional chemotherapy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198759911.003.0006.

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The psychiatric consequences of a range of conventional chemotherapy agents are reviewed. Anti-folates can cause frank demyelination (methotrexate-induced leucoencephalopathy) or more subtle cognitive impairment. The latter is attributed to reduced hippocampal neurogenesis due to the excitotoxic effects of homocysteine. Low mood, due to reduced availability of SAM, is also found during anti-folate chemotherapy. Ifosfamide-induced encephalopathy is described. Depression and encephalopathies are found with mitotic spindle poisons. Procarbazine is considered as a monoamine oxidase inhibitor. Fina

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30

Szelenyi, I. Inhibitors of Monoamine Oxidase B: Pharmacology and Clinical Use in Neurodegenerative Disorders (Milestones in Drug Therapy). Birkhauser, 1993.

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31

Series, Hugh. The Treatment of Depression. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198801900.003.0010.

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This chapter considers some of the physical options for the treatment of the affective disorders, depression, and mania. It first provides an historical overview of physical treatments for depression, including drugs such as opium, morphine, and diamorphine, chloral, and barbiturates, as well as electroconvulsive therapy (ECT). It then examines whether antidepressants work and how they are used before describing the biological basis of depression. It also looks at different classes of antidepressants, including monoamine oxidase inhibitors (MAOIs), reuptake inhibitors, and mood stabilizers. Fi

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32

Dependence of developing motoneutrons on tropic support: Actions of ciliary neurotropic factor and some monoamine oxidase inhibitors. National Library of Canada, 1994.

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33

Koen, N., T. Amos, J. Ipser, and D. Stein. Antidepressants in Post-Traumatic Stress Disorder. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0034.

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This chapter discusses the use of antidepressants in treating symptoms of posttraumatic stress disorder (PTSD). Tricyclic antidepressants were the first psychotropic agents to be studied systematically and rigorously for the treatment of PTSD. While early studies focused both on the tricyclics and monoamine oxidase inhibitors (MAOIs), more recent work has centered on the selective serotonin reuptake inhibitors (SSRIs); and paroxetine and sertraline are currently approved by the U.S. Food and Drug Administration (FDA) for use in this disorder. However, given the relatively small effect sizes in

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34

(Editor), Jes Olesen, and Lars Edvinsson (Editor), eds. Headache Pathogenesis: Monoamines, Neuropeptides, Purines, and Nitric Oxide (Frontiers in Headache Research). J. A. Majors Company, 1997.

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35

Steinberg, Martin. Treatment of Depression. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199959549.003.0006.

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Most depression in the elderly can be effectively treated in the primary care setting. Psychiatric referral should be considered in the setting of severe depression, suicidal ideation, prior suicide attempts, multiple risk factors, psychotic symptoms, bipolar disorder, poor response to prior treatment, or high medical comorbidity. Combining pharmacological and psychosocial interventions is most likely to be effective. Available antidepressants include serotonin-specific reuptake inhibitors, serotonin norepinephrine reuptake inhibitors, novel mechanism agents, tricyclic antidepressants, and mon

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36

Thornton, Kevin, and Michael Gropper. Diagnosis, assessment, and management of hyperthermic crises. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0247.

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Malignant hyperthermia, the neuroleptic malignant syndrome (NMS), and the serotonin syndrome are the principal disorders associated with life-threatening hyperthermia in the intensive care unit. While each is a clinically unique entity, all can progress to multisystem organ dysfunction with acidosis, shock, and death. MH usually results from exposure to halogenated volatile anaesthetics and/or succinylcholine and symptoms of increased CO2 production and respiratory acidosis progress rapidly without prompt intervention, including the administration of dantrolene. NMS is a syndrome of rigidity a

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37

Keck, Paul E., and Susan L. McElroy. Pharmacological Treatments for Bipolar Disorder. Oxford University Press, 2015. http://dx.doi.org/10.1093/med:psych/9780199342211.003.0008.

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The majority of clinical trials in patients with bipolar disorders have been conducted in groups with bipolar I illness, although a few trials have included patients with bipolar II disorder. Pharmacological management of bipolar disorder involves the treatment of acute manic, hypomanic, mixed, and depressive episodes, as well as the prevention of further episodes and subsyndromal symptoms. Lithium, divalproex, carbamazepine, haloperidol, risperidone, olanzapine, quetiapine, ziprasidone, aripiprazole, and asenapine have demonstrated efficacy in the treatment of acute mania in randomized contro

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38

Praag, Herman M. van 1929-, Plutchik Robert, and Apter Alan, eds. Violence and suicidality: Perspectives in clinical and psychobiological research. Brunner/Mazel, 1990.

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39

Herman M. Van Praag (Editor), Robert Plutchik (Editor), and Alan Apter (Editor), eds. Violence And Suicidality : Perspectives In Clinical And Psychobiological Research: Clinical And Experimental Psychiatry (Clinical and Experimental Psychiatry). Brunner/Mazel, 1990.

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40

(Editor), H. Parvez, and P. Riederer (Editor), eds. Oxidative Stress and Neuroprotection (Journal of Neural Transmission. Supplementa). Springer, 2006.

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41

Nielsen, David A., Dmitri Proudnikov, and Mary Jeanne Kreek. The Genetics of Impulsivity. Edited by Jon E. Grant and Marc N. Potenza. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195389715.013.0080.

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Impulsivity is a complex trait that varies across healthy individuals, although when excessive, it is generally regarded as dysfunctional. Impulsive behavior may lead to initiation of drug addiction that interferes with inhibitory controls, which may in turn result in facilitation of the individual’s impulsive acts. Although environmental factors play a considerable role in impulsive behavior, a body of evidence collected in twin studies suggests that about 45% of the variance in impulsivity is accounted for by genetic factors. Genetic variants studied in association with impulsivity include t

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42

F, Tipton Keith, and Youdim Moussa B. H, eds. Biochemical and pharmacological aspects of depression. Taylor & Francis, 1989.

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43

P, Riederer, and Przuntek H. 1938-, eds. MAO-B-inhibitor selegiline (R-(-)-deprenyl): A new therapeutic concept in the treatment of Parkinson's disease : proceedings of the international symposium in Berlin, January 23-25, 1987. Springer-Verlag, 1987.

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44

Riederer, P., and H. Przuntek. Mao-B-Inhibitor Selegiline (Journal of Neural Transmission Supplementum). Springer, 1988.

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45

H, Youdim Moussa B., Da Prada M, Amrein R. 1937-, and International Conference on New Directions in Affective Disorders (1987 : Jerusalem), eds. The Cheese effects and selective MAO-A inhibitors: Proceedings of the round table of the International Conference on New Directions in Affective Disorders, Jerusalem, April 5-9, 1987. Springer-Verlag, 1988.

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46

Hans-Jürgen, Möller, Paykel Eugene S, Geigy Pharmaceuticals, and World Congress of Biological Psychiatry. (5th : 1991 : Florence, Italy), eds. Renaissance of monoamine oxidase inhibitors: The new selective and reversible generation : proceedings of an international symposium supported by an educational grant from Ciba-Geigy, held during the 5th World Congress of Biological Psychiatry, Florence, Italy, 9-14 June 1991. Royal Society of Medicine Services, 1992.

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47

1949-, Frost J. James, and Wagner Henry N. 1927-, eds. Quantitative imaging: Neuroreceptors, neurotransmitters, and enzymes. Raven Press, 1990.

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48

Quantitative Imaging: Neuroreceptors Neurotransmitters and Enzymes. Raven Pr, 1990.

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49

(Editor), T. Kumazawa, L. Kruger (Editor), and K. Mizumura (Editor), eds. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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50

Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Elsevier, 1996.

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