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Dissertations / Theses on the topic 'Monoaminy'

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Published: 24 April 2022
Last updated: 30 July 2025

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1

CitÃ, Maria do Carmo de Oliveira. "AlteraÃÃes comportamentais e neuroquÃmicas provocadas por diferentes perÃodos de retirada apÃs tratamento subcrÃnico com cocaÃna em ratos: envolvimento dos sistemas dopaminÃrgico, serotonÃrgico e noradrenÃrgico." Universidade Federal do CearÃ, 2009. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=3986.

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CoordenaÃÃo de AperfeiÃoamento de Pessoal de NÃvel Superior<br>A cocaÃna à uma droga consumida mundialmente e considerada hoje como um problema de saÃde pÃblica. Uma das principais dificuldades enfrentadas no combate ao vÃcio da cocaÃna, na maioria dos casos, està relacionada aos sintomas de abstinÃncia da droga, como ansiedade, depressÃo, irritabilidade, fadiga e insÃnia, fazendo com que o indivÃduo volte a procurÃ-la. Para avaliar as alteraÃÃes comportamentais (ansiedade e depressÃo) e neuroquÃmicas, os ratos foram submetidos Ãs retiradas de 24 h, 7 d e 21 d apÃs o tratamento subcrÃnico por
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2

Kaushal, Setu. "Characterization of Three Putative Monoamine Oxidase Genes in Caenorhabditis elegans." Ohio University / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1218747926.

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3

Aguiar, Lissiana Magna Vasconcelos. "Efeitos comportamentais e neuroquÃmicos da melatonina em ratos submetidos à lesÃo estriatal com 6-ohda." Universidade Federal do CearÃ, 2002. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=19.

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Conselho Nacional de Desenvolvimento CientÃfico e TecnolÃgico<br>Os efeitos da melatonina (Mel) in vivo foram estudados no sistema dopaminÃrgico nigroestriatal de ratos, utilizando um modelo experimental da doenÃa de Parkinson que consiste na injeÃÃo intraestriatal da neurotoxina 6-hidroxidopamina (6-OHDA). Ratos Wistar, machos (200-250g) foram submetidos a lesÃo unilateral com 6-OHDA, tratados com melatonina nas doses de 2, 5, 10 e 25 mg/kg, i.p. 1 hora apÃs a lesÃo e depois, diariamente durante 7 dias, quatro semanas apÃs a lesÃo, foi realizado o teste rotacional e 24 horas depois os animais
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4

Figueira, Fernanda Hernandes. "EFEITO DO DISSELENETO DE DIFENILA SOBRE ALTERAÇÕES COMPORTAMENTAIS E BIOQUÍMICAS INDUZIDAS POR ANFETAMINA EM CAMUNDONGOS." Universidade Federal de Santa Maria, 2012. http://repositorio.ufsm.br/handle/1/11225.

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Selenium is an element that can modulate the dopaminergic neurotransmission. Studies show that diphenyl diselenide, an organic compound of selenium, has antioxidant activity improves depressive-like behavior and reduce the activity of the enzyme monoamine oxidase (MAO). However, there are few studies concerning about possible alterations of diphenyl diselenide in dopaminergic system. Thus, the purpose of the present study was to evaluate the effects of acute and sub-chronic treatment of diphenyl diselenide on amphetamine-induced behavioral and biochemical alterations in mice. In the acute trea
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5

Mullan, Elaine L. "Inhibitors of monoamine oxidase." Thesis, Queen's University Belfast, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337116.

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6

VenÃncio, Edith Teles. "Estudo dos efeitos comportamentais e neuroquÃmicos do extrato padronizado de Justicia pectoralis (chambÃ) em camundongos." Universidade Federal do CearÃ, 2009. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=3935.

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CoordenaÃÃo de AperfeiÃoamento de Pessoal de NÃvel Superior<br>O extrato padronizado de chambÃ, preparado a partir das partes aÃreas da Justicia pectoralis Jacq. var stenophylla Leonard, foi avaliado em modelos animais clÃssicos para screening de drogas com atividade em ansiedade, depressÃo, sedaÃÃo e convulsÃo, tais como, labirinto em cruz elevado (LCE), claro/escuro, campo aberto, rota rod, nado forÃado, suspensÃo da cauda, tempo de sono induzido por pentobarbital e convulsÃo induzida por pentilenotetrazol, e em estudo neuroquÃmico, atravÃs da concentraÃÃo de monoaminas e seus metabÃlitos, t
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7

Moura, Brinell Arcanjo. "AlteraÃÃes comportamentais, neuroquÃmicas e glicolipÃdicas em ratos tratados com Hoodia gordonii, um supressor natural do apetite." Universidade Federal do CearÃ, 2012. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=8886.

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Conselho Nacional de Desenvolvimento CientÃfico e TecnolÃgico<br>Hoodia gordonii à uma planta da famÃlia das apocinÃceas. OriginÃria do sudeste da Ãfrica, onde tem sido historicamente usada para suprimir o apetite durante longas jornadas de caÃa, sendo utilizada em diversos paÃses com o objetivo de emagrecer. No Brasil foi retirada do mercado devido à falta de estudos que comprovem sua eficÃcia e seguranÃa para o uso. O objetivo deste trabalho foi avaliar as alteraÃÃes comportamentais, neuroquÃmicas e glicolipÃdicas em ratos tratados com Hoodia gordonii. Para a realizaÃÃo deste estudo H. gordo
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8

Linhares, Maria Isabel. "Estudo da Ritalina (Cloridrato de Metilfenidato) sobre o sistema nervoso central de animais jovens e adultos: aspectos comportamentais e neuroquÃmicos." Universidade Federal do CearÃ, 2012. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=9347.

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CoordenaÃÃo de AperfeiÃoamento de Pessoal de NÃvel Superior<br>O Transtorno de DÃficit de AtenÃÃo/ Hiperatividade (TDAH) Ã um transtorno prevalente e debilitante, diagnosticado com base em persistentes nÃveis de hiperatividade, desatenÃÃo e impulsividade. FÃrmacos estimulantes tÃm sido eficazes no tratamento desse transtorno, sendo que o metilfenidato (MFD) Ã o agente terapÃutico mais prescrito e seu uso aumentou significativamente nos Ãltimos anos, entretanto, as conseqÃÃncias da sua utilizaÃÃo ainda sÃo pouco conhecidas. O MFD foi avaliado em modelos animais clÃssicos para screening de droga
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9

Meschin, Pierre. "Régulations monoaminergiques AMPc-dépendantes du coeur sain et pathologique." Thesis, Montpellier 1, 2014. http://www.theses.fr/2014MON1T010.

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La fonction cardiaque est finement régulée par des hormones de type monoamines qui constituent des régulateurs cruciaux de l’activité cardiaque (chronotropie et inotropie). Ces hormones dérivées d’acides aminés aromatiques comprenant les catécholamines et la sérotonine maintiennent l’activité du myocarde dans un cadre physiologique tout en lui permettant de s’adapter aux contraintes environnementales. Les récepteurs cellulaires des monoamines sont couplés à des voies de signalisation qui impliquent un nucléotide cyclique, l’AMPc, et modulent la contractilité des cardiomyocytes par l’intermédia
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10

Vacher, Claire-Marie. "Régulation par les monoamines de l'expression de la vasopressine dans l'hypothalamus neuroendocrine : étude cytophysiologique chez la souris Tg8, knock-out pour la monoamine oxydase A (MAO-A)." Paris 6, 2002. http://www.theses.fr/2002PA066359.

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11

Fitzgerald, Julia. "Monoamine oxidase in neuronal cell death." Thesis, Nottingham Trent University, 2008. http://irep.ntu.ac.uk/id/eprint/51/.

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Monoamine oxidase (MAO) is an oxidative enzyme that deaminates a variety of amine substrates, including the neurotransmitter dopamine. The enzymatic reaction requires molecular oxygen and produces hydrogen peroxide as a by-product. MAO is localised in the outer mitochondrial membrane and exists as two isoforms, MAO-A and MAO-B, which are differentially expressed in the body and differ in their substrate and inhibitor specificities. Previous studies have suggested that MAO-generated reactive oxygen species (ROS) contribute to oxidative stress in the cell and can directly inhibit electron transp
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12

Stenström, Anders. "Intra- and extraneuronal monoamine oxidase (MAO)." Umeå : [University of Umeå], 1986. http://catalog.hathitrust.org/api/volumes/oclc/15239401.html.

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13

Yeomanson, K. B. "An immunochemical analysis of monoamine oxidase profiles." Thesis, Nottingham Trent University, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.254741.

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14

Maurel, Agnès. "Monoamine oxydases rénales et cardiaques : implications physiopathologiques." Paris 7, 2004. http://www.theses.fr/2004PA077127.

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15

Wu, Bo. "Structure-function relationships in monoamine oxidase B /." Digital version accessible at:, 1998. http://wwwlib.umi.com/cr/utexas/main.

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16

Ruchala, Iwona. "EXPANDING MONOAMINE TRANSPORTERS PHARMACOLOGY USING CALCIUM CHANNELS." VCU Scholars Compass, 2017. http://scholarscompass.vcu.edu/etd/5032.

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Research in drug development meets many challenges including lengthy, complex and costly procedures to identify novel pharmacotherapies. In our lab, we developed a method for fast screening of small molecules that interact with monoamine transports – dopamine and serotonin (DAT, SERT). These membrane proteins play important roles in brain neurotransmission responsible for cognition, motion and pleasure. Dysfunction in dopaminergic and serotonergic systems result in neurological disorders such as depression, Attention Deficit Hyperactivity Disorder (ADHD), schizophrenia and addiction. DAT and S
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17

Saiyudthong, Somrudee. "Mechanisms underlying the antidepressant properties of St. John's wort (Hypericum perforatum)." Thesis, University of Nottingham, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.275319.

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18

Foka, Germaine Boulenoue. "Synthesis and evaluation of novel coumarin-donepezil derivatives as dual acting monoamine oxidase B and cholinesterase in Alzheimer's disease." University of the Western Cape, 2016. http://hdl.handle.net/11394/5549.

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Magister Pharmaceuticae - MPharm<br>Alzheimer's disease is a progressive neurodegenerative disease characterised by low acetylcholine (ACh) levels in the hippocampus and cortex of the brain, causing symptoms like progressive memory loss, decline in language skills and other cognitive impairments to occur. The hallmarks of AD include the presence of extracellular insoluble amyloid beta plaques, intracellular neurofibrillary tangles, and the decrease in ACh concentration. The pathophysiology of AD is not well understood, however, acetylcholinesterase (AChE), butyrylcholinesterase (BuChE) and mon
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19

Backwell, Colette. "Labels for the active site of monoamine oxidase." Thesis, Queen's University Belfast, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.336043.

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20

Abdel-Razaq, Wesam. "Molecular mechanisms of monoamine neurotransmitter modulatory antidepressant actions." Thesis, University of Nottingham, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.435767.

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21

Jackson, Gillian. "Monoamine release and turnover in broiler chicken brain." Thesis, University of Bristol, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.411086.

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22

Vince, Matthew Joseph Kline. "Examining Monoamine Oxidase Inhibitor Targets Using Caenorhabditis elegans." Ohio University Art and Sciences Honors Theses / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ouashonors1598384776888279.

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23

Villarinho, Jardel Gomes. "Avaliação do envolvimento da enzima monoamina oxidase b em modelos de dor pós-operatória e neuropática em camundongos." Universidade Federal de Santa Maria, 2010. http://repositorio.ufsm.br/handle/1/11124.

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Coordenação de Aperfeiçoamento de Pessoal de Nível Superior<br>Monoamines appear to play an important modulatory role on pain descending pathways and are involved in the antinociceptive mechanism of several drugs commonly used for the management of pain. In this study, we assessed the involvement of monoamine oxidase B (MAO-B), a key enzyme implicated in monoamine metabolism, on models of postsurgical and neuropathic pain in mice. For this purpose, we evaluated the effects of the selective and irreversible MAO-B inhibitor selegiline on mechanical sensitivity and ex vivo MAO-B activity in diffe
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24

Bacalhau, Patrícia Alexandra Anico Gazimba. "Biochemical mechanisms and target drugs in neurodegenerative diseases." Doctoral thesis, Universidade de Évora, 2018. http://hdl.handle.net/10174/31045.

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Neurodegenerative diseases, namely Alzheimer’s and Parkinson’s diseases are a major challenge for medicine and public health due to their prevalence in developed countries. Thus, the research for therapies for neurodegenerative diseases, such as Parkinson’s and Alzheimer’s diseases, should be based on understanding their molecular and biochemical pathogenesis. The research conducted in this thesis involves screening of different families of compounds (isoquinolinones, azepanones, indolinones, diether-esters, chromanones, chromanols and rivastigmine derivatives) based on their ability to inhibi
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25

Calefi, Atilio Sersun. "Avaliação dos efeitos do estresse por calor sobre a imunidade de frangos de corte em modelos experimentais de enterite necrótica aviária." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/10/10133/tde-07102016-124200/.

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Doenças, como a enterite necrótica aviária (NE), têm se tornado reemergentes em função não apenas do sistema de criação intensivo de frangos de corte atualmente em uso, como também da restrição imposta ao uso dos aditivos antimicrobianos por diversos países, dentre os quais, aqueles da União Européia. A NE é uma doença que acomete aves de produção e seu agente etiológico primário é o Clostridium perfringens tipo A. Pouco se conhece a respeito dos mecanismos pelos quais o estresse modula o desenvolvimento da NE. O presente trabalho foi realizado para avaliar os efeitos do estresse por calor sob
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26

Booysen, Hermanus Perold. "Thiocaffeine derivatives as inhibitors of monoamine oxidase / Booysen H.P." Thesis, North-West University, 2011. http://hdl.handle.net/10394/7348.

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Parkinson’s disease (PD) is a neurodegenerative disorder which is characterized by selective loss of dopaminergic neurons in the substantia nigra pars compacta (SNpc) of the brain and reduced striatal dopamine (DA). Neuropathologically, PD is characterized by the presence of intraneuronal inclusions called Lewy Bodies (LBs). While the pathogenesis of PD is unknown, it is thought that monoamine oxidase (MAO) may play an important role in the neurodegenerative process. In the basal ganglia DA is oxidized by MAO, a process which is associated with the formation of toxic metabolic by–products. For
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27

Quinn, T. G. "Characterisation of the vesicular monoamine transporters 1 and 2." Thesis, Queen's University Belfast, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368550.

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28

Rein, Glen. "Monoamine metabolism in normal tissues and neural crest tumours." Thesis, Imperial College London, 1987. http://hdl.handle.net/10044/1/47814.

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29

Odhar, Hasanain. "Identification of novel scaffolds for Monoamine oxidase B inhibitors." Kent State University / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=kent1394416913.

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30

Courtney, Nicholas A. "Mechanisms of Autoreceptor-Mediated Inhibition in Central Monoamine Neurons." Case Western Reserve University School of Graduate Studies / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=case1447955036.

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31

Pizzinat, Nathalie. "Les monoamine oxydases renales et adipocytaire : caracterisation et regulation." Toulouse 3, 1999. http://www.theses.fr/1999TOU30005.

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Les monoamine oxydases (mao) sont des enzymes cles du catabolisme de neuromediateurs tels que la noradrenaline, la dopamine et la serotonine, et d'amines exogenes. Deux isoformes de l'enzyme, la mao-a et la mao-b, codees par deux genes distincts ont ete identifiees. Les maos sont fortement exprimees au niveau peripherique. Toutefois, l'expression relative et la fonction de chaque isoforme dans les organes presentant une importante heterogeneite morphologique tel que le rein ont ete tres peu etudiees. A ce niveau, nous avons montre que les cellules mesangiales glomerulaires de rat expriment tre
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32

Hansson, Stefan R. "The serotonin transporter and vesicular monoamine transporters during development." Lund : Lund University, 1998. http://catalog.hathitrust.org/api/volumes/oclc/68945023.html.

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33

Holloway, Alexa. "Pharmacodynamics of Monoamine Transporter Releasing Agents and Reuptake Inhibitors." VCU Scholars Compass, 2019. https://scholarscompass.vcu.edu/etd/5880.

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Ligands of the human monoamine transporters encompass a wide range of both illicit and therapeutic drugs that act upon neural circuitry related to reward, motivation, and the processing of salient stimuli. The present study utilizes two methods for analyzing transporter substrates and inhibitors in order to characterize activity and assess potency. The first measures transient changes in intracellular calcium as a surrogate for transporter activity by harnessing the electrical coupling of monoamine transporters and L-type calcium channels. This is used to analyze novel chimera of the strong hD
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34

Pesarico, Ana Paula. "ENVOLVIMENTO DOS SISTEMAS SEROTONINÉRGICO E DOPAMINÉRGICO NA AÇÃO DO TIPO ANTIDEPRESSIVA DO 7-FLÚOR-1,3 DIFENILISOQUINOLINA-1-AMINO EM CAMUNDONGOS." Universidade Federal de Santa Maria, 2014. http://repositorio.ufsm.br/handle/1/11237.

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Coordenação de Aperfeiçoamento de Pessoal de Nível Superior<br>Depression is a psychiatric disorder associated with a negative impact on quality of life. Monoaminergic system has been involved in this disease and in the action of antidepressants. This study aimed to investigate the potential antidepressant-like of 7-fluoro-1,3-diphenylisoquinoline-1-amine (FDPI) and the possible involvement of monoaminergic system. Results showed that FDPI (1, 10 and 20 mg/kg, intragastric (i.g.)) reduced the immobility time, increased swimming time, but did not alter climbing time of mice in the modified forc
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35

Baulieu, Jean-Louis. "Méta-iodobenzylguanidine et captage des monoamines." Tours, 1988. http://www.theses.fr/1988TOUR3302.

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36

Sørensen, Izel Fourie. "Influence of tobacco smoke constituents on monoamine oxidase activity in vitro and on human placental monoamine oxidase A activity in vivo / Izel Fourie Sørensen." Thesis, North-West University, 2004. http://hdl.handle.net/10394/294.

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The aim of this study was to investigate the influence of tobacco smoke on monoamine oxidase activity, and of smoking during pregnancy on placental monoamine oxidase A (MAO-A) activity. The enzyme MA0 exists in two isoforms, MAO-A and MAO-B, both of which are important in regulating monoamine status in the brain and periphery. Some substrate selectivity for particular monoamines is a characteristic of these isoforms, and one form of the enzyme may predominate in particular organs. Recreational tobacco smoking influences the status of monoamine oxidases in the body. In vivo studies using PET im
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37

Parboosingh, Jillian S. "Biochemical and molecular analysis of monoamine oxidase in alcoholics, high risk subjects and low risk controls." Thesis, McGill University, 1991. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=61088.

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Alcoholism is a prevalent multifactorial disease with both genetic and environmental components. Monoamine oxidase (MAO) has been proposed as a susceptibility marker for familial alcoholism but consistent evidence of either specific MAO variants in alcoholics or allelic segregation in at-risk families has not been presented. Two structural genes on the X chromosome encode two forms of the enzyme, MAO-A and MAO-B. Kinetic constants for platelet MAO-B and restriction fragment length polymorphisms for MAO-A were determined in alcoholics with multigenerational family histories of alcoholism, high
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38

Shearman, M. S. "Pharmacological and biochemical characterisation of serotonin-5-HT2̲ receptors from rat frontal cortex and human platelets." Thesis, University of Nottingham, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.377497.

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39

Escrig, Ayuso Miguel Ángel. "Implicación de la monoamino oxidasa en conductas inducidas por etanol." Doctoral thesis, Universitat Jaume I, 2016. http://hdl.handle.net/10803/669006.

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La catalasa utiliza como cosubstrato el peróxido de hidrógeno (H2O2) para formar el compuesto I (catalasa- H2O2) que es el principal sistema de metabolismo de etanol (EtOH) en el sistema nervioso central (SNC). De este modo, el etanol se metaboliza a acetaldehído por la actividad del compuesto I. Se ha demostrado que las manipulaciones farmacológicas que varían los niveles de catalasa central producen una modificación de los efectos que el EtOH produce en roedores. También se han observado resultados similares cuando se han comparado estirpes de ratones con diferentes niveles de catalasa cen
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Gretton, Heather Margaret. "Platelet monoamine oxidase type B (MAO-B) activity in psychopathy." Thesis, University of British Columbia, 1991. http://hdl.handle.net/2429/30619.

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Studies of platelet MAO-B activity have revealed a link between low platelet activity and psychiatric syndromes characterized by an inability to control impulses and to anticipate future consequences of behavior (Oreland, 1980; Gottfries, von Knorring, & Oreland, 1980). These characteristics are fundamental to the construct of psychopathy, and we might therefore expect that psychopathy is associated with low MAO activity. Indeed, some investigators have suggested that low platelet MAO-B activity is a potential marker for vulnerability to psychopathy (Schalling, Asberg, Edman, & Oreland, 1987).
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Mejia, Jose. "Monoamine oxidases and aggressive behaviour : clinical studies and animal models." Thesis, McGill University, 2002. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=38238.

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Monoamine oxidases (MAOs) are phylogenetically old enzymes which catalyze the deamination of monoamines. Interest in a relationship between MAO and aggressive behaviour derives from the report of a single family with a mutation which obliterates the activity of MAO A, as well as a long history of studies which substantiate a relationship between MAO activity and impulsive aggressive behaviour. The goals of this thesis were: (1) to examine the generalizability of the specific MAO mutation noted above; (2) to evaluate the relationship between platelet MAO activity and genetic polymorphisms in MA
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Finch, Cheryl Christine. "An immunochemical analysis of monoamine oxidase in health and disease." Thesis, Nottingham Trent University, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.298903.

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43

Taylor, Julian Scott. "Opioid and monoamine modulation of spinal reflexes in the rabbit." Thesis, University of Nottingham, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.385277.

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44

Gibb, Celia Margaret. "Environment and neurological illness : role of monoamine oxidase and phenolsulphotransferase." Thesis, Imperial College London, 1988. http://hdl.handle.net/10044/1/47072.

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45

Wang, Zhixia, Gregory A. Ordway, and William Woolverton. "Effects of Cocaine on Monoamine Uptake as Measured Ex Vivo." Digital Commons @ East Tennessee State University, 2007. https://dc.etsu.edu/etsu-works/8611.

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The increase in extracellular dopamine (DA) following cocaine administration plays a major role in cocaine abuse. In vitro, cocaine binds to DA transporters (DAT) and blocks DA uptake. Moreover, cocaine can increase extracellular DA concentration as measured by in vivo neurochemical methods. The present study examined the effects of cocaine and other drugs on DA, NE and 5-HT uptake using an ex vivo assay. Rats were injected i.v. with saline or drug and sacrificed at various time points after injections. Brains were dissected for regional monoamine uptake studies ex vivo. In most brain regions,
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46

Coatrieux, Christelle. "Monoamine oxydases et athérosclérose : signalisation mitogène et étude in vivo." Toulouse 3, 2007. http://thesesups.ups-tlse.fr/38/.

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Les espèces réactives de l'oxygène (EROs) activent de nombreuses voies de signalisation cellulaires, comme la prolifération et sont impliquées dans l'athérosclérose. Les monoamine oxydases (MAOs), dégradent les amines biogènes, libèrant des EROs. Elles peuvent être impliquées dans la prolifération cellulaire ou l'apoptose par le stress oxydant qu'elles génèrent. Ce travail a montré que la MAO-A, en dégradant son substrat, active une voie de signalisation mitogène particulière : la voie MMP2/sphingolipides, et contribue à la prolifération de cellules musculaire lisses vasculaires. Cet effet a é
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47

Wan, Li. "Monoamine Oxidase and Sensory Gating: Psychophysiological Vulnerabilities among Teenage Smokers." Diss., Virginia Tech, 2006. http://hdl.handle.net/10919/27380.

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Smoking is one of the leading causes of death in the world. About 80% of smokers start smoking before the age of 18. In the Appalachian area and the South in the United States, smoking percentages among adults and adolescents are higher than in other regions. Female smoking shows a variety of different trends from male smoking, and smoking brings particular health problems related to production to female smokers. These findings highlighted the importance of studying female teenage smokers in southwest Virginia. The initial project aimed to identify risk factors that might prevent smoking in an
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48

Beaver, Jasmin Nicole. "BEHAVIORAL RESPONSES TO PSYCHOSTIMULANTS IN PLASMA MEMBRANE MONOAMINE DEFICIENT MICE." Kent State University / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=kent1627571055518516.

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49

Bauer, Clayton T. "Determinants of Abuse-Related Effects of Monoamine Releasers in Rats." VCU Scholars Compass, 2013. http://scholarscompass.vcu.edu/etd/522.

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Monoamine releasers constitute a class of compounds that promote release of dopamine, serotonin, and/or norepinephrine. These compounds have a range of different uses in the medical setting, including treatment of attention deficit hyperactivity disorder, narcolepsy, and obesity. A major limitation of many of these compounds (i.e. amphetamine, methamphetamine, phenmetrazine) is their propensity for abuse; however, not all monoamine releasers are abused (i.e. fenfluramine). The goal of this dissertation was to examine pharmacological determinants of abuse-related effects produced by monoamine r
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50

Peters, Jennifer Margaret 1956. "Synthesis and monoamine uptake inhibiting properties of perisubstituted tricyclic compounds." Thesis, The University of Arizona, 1988. http://hdl.handle.net/10150/291828.

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The synthesis of 1-methyl-promazine, 4-hydroxymethyl-iminodibenzyl, and 4-bromo-5-trimethylsilyl-iminodibenzyl via dilithiation and ¹H-NMR's are described. Molecular modeling was done for the latter compound. The heat of dissociation was 30.6 kcal/mole for the lowest energy conformer. Rotational energies were examined for three bonds. The IC₅₀ values for inhibition of neurotransmitter uptake by rat brain synaptosomes were determined for a series of 1-substituted promazines, and 4-substituted imipramines. 1-Substituted promazines were fair inhibitors of serotonin uptake with an average IC₅₀ of
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