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Dissertations / Theses on the topic 'Monozyt'

Author: Grafiati

Published: 4 June 2021

Last updated: 11 February 2022

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1

Stehling, Martin. "Untersuchungen zur Regulation des CD163-Gens." [S.l. : s.n.], 2001. http://deposit.ddb.de/cgi-bin/dokserv?idn=967385334.

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2

Fröbe, Martin Leonhard. "Prokoagulante Aktivität des Monozyten : Fcgamma-Rezeptoren als Trigger und Variable." kostenfrei, 2008. http://www.opus-bayern.de/uni-regensburg/volltexte/2009/1222/.

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3

Göttling, Tanja Elke. "Einflüsse des humanen Zytomegalievirus auf Adhäsion und Chemotaxis von Monozyten und auf die reaktive Proliferation von humanen glatten Muskelzellen: Untersuchungen am koronaren Transfilter Co-Kultur-Modell." [S.l. : s.n.], 2006. http://nbn-resolving.de/urn:nbn:de:bsz:289-vts-56787.

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4

Ivanova, Nina Mihaylova. "Activation of receptors for advanced glycation endproducts (RAGEs) in human monocytes." [S.l. : s.n.], 2005. http://nbn-resolving.de/urn:nbn:de:bsz:289-vts-55812.

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5

Aberle, Dagmar Barbara. "Phagozytose und Rezeptorexpression von Monozyten unter dem Enfluss künstlicher Kolloide und LPS in vitro." [S.l. : s.n.], 2006.

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6

Tietzel, Illya. "Induktion und Signaltransduktion bei der Expression des Chemokins MCP-1 in Monozyten." [S.l.] : [s.n.], 2001. http://ArchiMeD.uni-mainz.de/pub/2001/0081/diss.pdf.

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7

Weber, Christian Michael. "Der Effekt von Mycophenolatmofetil im humanen koronaren 3D-Leukozytenangriffsmodell." [S.l. : s.n.], 2007. http://nbn-resolving.de/urn:nbn:de:bsz:289-vts-60399.

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8

Rösch, Daniela. "Regulation der Expression der Rezeptoren für advanced glycation end products (RAGE) auf humanen Monozyten." [S.l. : s.n.], 2006.

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9

Incekara, Nurcan. "PPAR-alpha-Aktivierung hemmt die Expression von Tissue Factor in humanen Monozyten, Makrophagen und THP-1 Zellen." [S.l. : s.n.], 2007. http://nbn-resolving.de/urn:nbn:de:bsz:289-vts-59513.

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10

Wurm, Sylvia Jutta Elisabeth. "Einfluss von oraler Glukoseaufnahme auf die monozytäre Genexpression." kostenfrei, 2008. http://www.opus-bayern.de/uni-regensburg/volltexte/2009/1319/.

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11

Zieglgänsberger, Dominik. "Monozyten-Lymphozyten-Koaggregate im akuten Myokardinfarkt." [S.l.] : [s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=973922842.

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12

Haddad, Ziad. "Monocytic cell responses to Aspergillus fumigatus investigation of phagocytosis, gene expression and peptide presentation /." [S.l. : s.n.], 2006.

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13

Hofer, Thomas. "Wirkung von partikulären Umweltnoxen auf die Immunabwehr monozytärer Zellen." [S.l.] : [s.n.], 2000. http://deposit.ddb.de/cgi-bin/dokserv?idn=961225173.

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14

Schönicke, Gerrit. "Einfluss alternativer Peritonealdialyse-Lösungen auf die monozytäre Zellvitalität und Zytokinfreisetzung /." Aachen : Shaker, 2000. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=009074420&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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15

Matthes, Constanze. "Immunmodulation Dendritischer Zellen durch Sekretionsprodukte mesenchymaler Stammzellen mit besonderem Focus auf hCyr61/CCN1." Doctoral thesis, kostenfrei, 2008. http://nbn-resolving.de/urn/resolver.pl?urn=nbn:de:bvb:20-opus-28129.

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16

Cappello, Christian. "Ozoniertes Low Density-Lipoprotien (OzLDL)." kostenfrei, 2009. http://mediatum2.ub.tum.de/node?id=679935.

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17

Krauß, Christine Anette. "Einfluss von Acetyl-alpha-Boswelliasäure auf die proinflammatorische Aktivierung von humanen, peripheren Monozyten: Ein Vergleich mit Dexamethason." [S.l.] : Universität Ulm , Medizinische Fakultät, 2003. http://www.bsz-bw.de/cgi-bin/xvms.cgi?SWB10913300.

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18

Engelhardt, Lars. "Einfluss von Methylprednisolon und Tirilazad Mesylat auf immunologische Parameter nach koronarer Bypassoperation." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 2002. http://dx.doi.org/10.18452/14721.

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Seit vielen Jahren werden Glukokortikoide routinemäßig eingesetzt, um Zeichen der inflammatorischen Reaktion nach kardiochirurgischen Eingriffen unter extrakorporaler Zirkulation (EKZ) zu mildern. Glukokortikoide sind jedoch für ihre immunsuppressiven Wirkungen bekannt, und bisher blieben die möglichen Auswirkungen auf immunologische Funktionen weitgehend hypothetisch. Ziel der vorliegenden Studie war es daher, den Einfluss von Methylprednisolon (MP) und Tirilazad Mesylat (TM), einer antiinflammatorischen Substanz aus der Klasse der Aminosteroide auf immunologische Funktionen nach koronarchiru

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19

Kraus, Stephan Georg. "Funktionelle Charakterisierung von CD16+ Monozytensubpopulationen." Doctoral thesis, Universitätsbibliothek Leipzig, 2011. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-77454.

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Seit 20 Jahren unterteilt man Monozyten in eine klassische CD14++/CD16-Population und eine proinflammatorische CD16+ Population. Letztere macht 10-20 % der peripheren Blutmonozyten aus und ist unter verschiedenen pathologischen Zuständen drastisch erhöht. Seit Kurzem wird die CD16+ Fraktion in zwei weitere Untergruppen aufgeteilt, die CD14++/CD16+ und die CD14+/CD16+ Monozyten. In der hier vorliegenden Arbeit wurde versucht, die relativ neue und wenig charakterisierte Subpopulation der CD14++/CD16+ Monozyten näher zu beschreiben. Es sollte die Frage beantwortet werden, ob diese sich von den üb

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20

Herbst, Andreas Sebastian. "Untersuchungen zu in vitro modifizierten humanen Blutmonozyten : Immunhistochemisch-morphologische Charakterisierung und funktioneller Nachweis von Insulin." Doctoral thesis, kostenfrei, 2008. http://nbn-resolving.de/urn/resolver.pl?urn=nbn:de:bvb:20-opus-28404.

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21

Lesky, Thomas. "In vitro Differenzierung von Monozyten der Zelllinine RAW 264.7 zu Osteoklasten, deren Charakterisierung und Wechselwirkung mit Osteoblasten." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2006. http://nbn-resolving.de/urn:nbn:de:swb:14-1158674933492-53949.

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Das RANKL/RANK/OPG-System spielt eine entscheidende Rolle in der Steuerung der Osteoklastendifferenzierung und -aktivierung durch Osteoblasten/ Knochenmarkbindegewebszellen im Rahmen des Knochenremodelings. Osteoblasten/Knochenmarkbindegewebszellen exprimieren RANKL. Dieses hat im Körper zwei Rezeptoren: RANK und OPG. RANKL kann durch Bindung an RANK auf Osteoklasten/Osteoklastenvorläuferzellen in Gegenwart von M-CSF seine osteoklastenstimulierende Wirkung entfalten. Der ebenfalls von Osteoblasten gebildete „decoy“-Rezeptor OPG blockiert als freies Protein durch Bindung an RANKL dessen Interak

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22

Lesky, Thomas. "In vitro Differenzierung von Monozyten der Zelllinine RAW 264.7 zu Osteoklasten, deren Charakterisierung und Wechselwirkung mit Osteoblasten." Doctoral thesis, Technische Universität Dresden, 2005. https://tud.qucosa.de/id/qucosa%3A24881.

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Das RANKL/RANK/OPG-System spielt eine entscheidende Rolle in der Steuerung der Osteoklastendifferenzierung und -aktivierung durch Osteoblasten/ Knochenmarkbindegewebszellen im Rahmen des Knochenremodelings. Osteoblasten/Knochenmarkbindegewebszellen exprimieren RANKL. Dieses hat im Körper zwei Rezeptoren: RANK und OPG. RANKL kann durch Bindung an RANK auf Osteoklasten/Osteoklastenvorläuferzellen in Gegenwart von M-CSF seine osteoklastenstimulierende Wirkung entfalten. Der ebenfalls von Osteoblasten gebildete „decoy“-Rezeptor OPG blockiert als freies Protein durch Bindung an RANKL dessen Interak

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23

Bickenbach, Annette. "Experimentelle Untersuchungen zum Einfluss von Autoantikörpern gegen Proteinase 3 auf monozytäre Zellfunktionen bei der Wegenerschen Granulomatose." Doctoral thesis, Universitätsbibliothek Leipzig, 2011. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-68222.

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Autoantikörper gegen Proteinase 3 (cANCA) stellen einen hochsensitiven und spezifischen Seromarker für das Krankheitsbild der Wegenersche Granulomatose dar. Während die Ätiologie dieser systemischen Vaskulitis unbekannt ist, weisen zahlreiche klinische und experimentelle Daten darauf hin, daß cANCA an der Entstehung und Chronifizierung dieser Erkrankung beteiligt sind. Insbesondere die Konsequenzen einer cANCA-Ligation an Proteinase 3 (PR3)-exprimierende Neutrophile wurden intensiv untersucht und man geht davon aus, daß Anti-PR3-Antikörper über die Aktivierung inflammatorischer, neutrophiler Z

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24

Ehrlich, Stefan. "Die Bedeutung löslicher TNF-Familienmitglieder für die multiple Sklerose." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 2006. http://dx.doi.org/10.18452/15484.

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Bei Autoimmunkrankheiten wie der Multiplen Sklerose (MS) kommt es zu einer fehlgesteuerten Immunantwort mit Aktivierung und Persistenz autoreaktiver T-Zellen. Apoptose-regulierende Mechanismen wie das CD95-Rezeptor/CD95-Ligand- und TRAIL-Rezeptor/TRAIL-System könnten dabei eine wichtige Rolle spielen. Die lösliche Form des CD95-Rezeptor (sCD95) kann an CD95L binden und so die Apoptose aktivierter T-Zellen verhindern. Die systemische Blockade von TRAIL führt zur Exazerbation von Autoimmunerkrankungen in Tierversuchen. In der vorliegenden Arbeit wurden deshalb die Expression, Regulation und Bed

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25

Bonfield, Tracey Leigh. "Human monocyte interaction with biomedical polymers: Induction of monocyte-derived growth factors." Case Western Reserve University School of Graduate Studies / OhioLINK, 1991. http://rave.ohiolink.edu/etdc/view?acc_num=case1059154908.

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26

Johnson-Tidey, Ruth R. "Monocyte adhesion in atherosclerosis." Thesis, King's College London (University of London), 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.387903.

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27

Wrigley, Benjamin J. "Monocyte subsets in heart failure." Thesis, University of Leicester, 2013. http://hdl.handle.net/2381/28383.

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Introduction: Monocytes play important roles in inflammation, thrombosis, angiogenesis and tissue repair and may contribute to the pathophysiology of heart failure (HF). Functional diversity is likely to stem from the presence of three distinct monocyte subsets, defined by flow cytometry (FC) as CD14++CD16-CCR2+ (Mon1), CD14++CD16+CCR2+ (Mon2) and CD14+CD16++CCR2- (Mon3). The aims of this thesis were to study the following parameters in patients with ischaemic HF: 1) monocyte subset numbers, 2) monocyte subset expression of surface receptors for inflammation, angiogenesis, cell adhesion molecu

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28

Orr, N. J. "Characterisation of monocyte esterase deficiency." Thesis, Queen's University Belfast, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.403433.

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29

Dempster, Anne Margaret. "Endothelial influences on monocyte maturation." Thesis, University of Aberdeen, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.425102.

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The aims of this project were to determine the phenotype of migrating monocytes and examine the influence transmigration has on their differentiation to macrophages or DC. To determine stages of differentiation and activation of monocytes cultured in the presence of autologous serum without endothelium nine cell surface receptors were used. These cells matured over time with an increase in CD16<sup>+</sup> cells and increased major histocompatibility complex II and macrophage mannose receptor (MMR) expression suggesting alternative activation over time compared to cells cultured in foetal cal

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30

Carvalho, Maria das Gracas. "Monocyte tissue factor in malignancy." Thesis, University of Southampton, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.296364.

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31

McCann, Katelyn J. "IFNγ Mediated Monocyte Metabolic Reprogramming". eScholarship@UMMS, 2021. https://escholarship.umassmed.edu/gsbs_diss/1146.

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IFNγ is an essential and pleiotropic activator of monocytes, but little is known about the effects IFNγ on cellular metabolism. Therefore, we sought to characterize and elucidate the mechanisms by which IFNγ reprograms monocyte metabolism to support its immunologic activities. First, we identified a critical role for IFNγ in the induction of immunoresponsive gene 1 (IRG1) and its product, itaconate. The immunometabolite, itaconate, has been reported to have antibacterial, anti-inflammatory and antioxidant activity. Irg1-/- mice, lacking itaconate, are highly susceptible and phenotypically simi

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32

Schuldhaus, Christopher [Verfasser]. "Mögliche Rolle der Serinprotease Granzym B in Monozyten und Monozyten-abgeleiteten dendritischen Zellen / Christopher Schuldhaus." Ulm : Universität Ulm, 2019. http://d-nb.info/1190727285/34.

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33

Owen, Caroline Ann. "Monocyte adherence to fibronectin : role of CD11/CD18 integrins and relationship to other monocyte functions." Thesis, University of Birmingham, 1993. http://etheses.bham.ac.uk//id/eprint/36/.

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Regulated adherence of monocytes to extracellular matrix macromolecules is a prerequisite for their accumulation at sites of pulmonary infection and inflammation. To begin to assess the pathobiological importance of alterations in monocyte adherence to extracellular matrix in inflammatory lung diseases, the adherence properties of monocytes from patients with an inflammatory lung disease (bronchiectasis) and healthy subjects to a representative matrix component (fibronectin) were compared. Spontaneous adherence of monocytes from the control subjects was 20 to 25%, whereas that of the patients'

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34

Peveri, Ottavio. "Monocyte-neutrophil interactions through cytokines, with special reference to a novel monocyte-derived neutrophil-activating peptide /." [S.l : s.n.], 1988. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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35

Follot, Sébastien. "Analyse des propriétés physico-chimiques et pharmacologiques d'Imidazole [1,2-¯]pyridine : évaluation de l'apport de la RMN HR-MAS pour la compréhension de ces mécanismes." Thesis, Grenoble, 2011. http://www.theses.fr/2011GRENS003/document.

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La MAP kinase p38 régule la transduction du signal en réponse à un stress environnemental. Les inhibiteurs spécifiques de p38, qui sont connus pour bloquer la production de cytokines pro-inflammatoires, mais peuvent également intervenir sur le phénomène d'apoptose. C'est dans cette dernière optique que de nouvelles structures d'inhibiteurs, ont été synthétisées. Ces structures, après caractérisation par RMN, ont été ensuite évaluées en termes de mécanisme d'action et de disponibilité biologique. Outre les méthodes classiques cette évaluation a finalement fait appel aux techniques HR-MAS. Les s

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36

Bogner, Viktoria. "Differentielle Genexpression in Monozyten polytraumatisierter Patienten." Diss., lmu, 2008. http://nbn-resolving.de/urn:nbn:de:bvb:19-79678.

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37

McQuibban, Angus. "Matrix metalloproteinase processing of monocyte chemokines." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/NQ61145.pdf.

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38

Hussen, Jemal [Verfasser]. "Charakterisierung boviner Monozyten-Subpopulationen / Jemal Hussen." Hannover : Bibliothek der Tierärztlichen Hochschule Hannover, 2017. http://d-nb.info/113749638X/34.

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39

Smith, Graham. "Early monocyte responses to bacterial lipopolysaccharides." Thesis, Open University, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.259890.

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40

Fairclough, Marianne Elizabeth. "Diversity among monocyte derived stromal cells." Thesis, University of Birmingham, 2010. http://etheses.bham.ac.uk//id/eprint/679/.

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Fibrocytes are monocyte-derived cells that morphologically look like fibroblasts, express both stromal and haematopoietic markers, and have been reported as being involved in wound healing and fibrosis. In-vitro derived fibrocytes can be differentiated in both serum-containing and serum-free environments and we wanted to study the relationship between these two fibrocytes; which potentially could be involved at different time points at a wound healing site. To investigate the relationship between serum-free and serum-containing derived fibrocytes monocytes were differentiated without serum. Wh

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41

Al-Reshoudi, Reem Hamoud. "Monocyte and fibrocyte characterisation in asthma." Thesis, University of Aberdeen, 2017. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=233753.

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Monocytes and their subsets play an important role in immune and inflammatory diseases. In this study the focus was on their role of driving pathogenesis in asthmatic patients. The hypothesis was that although monocytes have not been identified as key players in atopic disease they are likely to be pro-inflammatory, be readily recruited to tissue and have a major role in the exacerbation of disease. This may relate to the prevailing pro-inflammatory microenvironment. Chemokines such as CCL2 and CX3CL1 are involved in monocyte recruitment and survival respectively in the inflamed tissues. Three

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42

Crawshaw, Anjali Priya. "Monocyte profile and function in sarcoidosis." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:3378bf46-a494-45a0-b68e-81b37c1dae49.

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Sarcoidosis is a multisystem inflammatory disorder of unknown aetiology. The immune pathology is characterised by dysregulated T cell (T<sub>H</sub>1) activity, macrophage activation and granuloma formation, resulting in systemic inflammation, and organ dysfunction. I hypothesised that, as the systemic precursor to the macrophage, altered monocyte activity in sarcoidosis may contribute to the early immune pathology of the disease. In this thesis, I examined their phenotype, four key monocytic functions: cytokine production, suppression of T cell proliferation, phagocytosis and fusion (as a pre

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43

Baugh, John Andrew. "Differential regulation of monocyte cytokine release." Thesis, University of Bath, 1999. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.285313.

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44

Woollard, Kevin J. "Mediators of monocyte activity in inflammation." Thesis, Aston University, 2003. http://publications.aston.ac.uk/11001/.

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The effects of incubation of CRP with human primary and monocytic cell lines were examined using monocytic cytokine expression, adhesion molecule expression and adhesion to endothelial cells and intracellular peroxide formation, as end points. Monocytic intracellular signalling events were investigated after interaction of CRP with specific CRP receptors on monocytes. These initial signalling events were examined for their role in modulating monocyric adhesipn molecule and cytokine expression. Monocyte recruitment and retention in the vasculature is also influenced by oxidative stress. Therefo

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45

Passacquale, Gabriella. "Platelet-monocyte interaction : relevance to atherosclerosis." Thesis, King's College London (University of London), 2012. http://kclpure.kcl.ac.uk/portal/en/theses/plateletmonocyte-interaction-relevance-to-atherosclerosis(13e9b6f5-be4b-4dca-b9e7-baefd1a4d695).html.

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Despite recent advances in diagnosis and therapy, atherosclerosis-related cardiovascular disease remains a leading cause of morbidity and mortality worldwide. An important challenge is to detect silent atherosclerosis in asymptomatic people at risk, so that preventative strategies can be more effectively targeted. This thesis examines the pro-inflammatory/pro-atherogenic role of platelet hyperactivity and the consequent interaction of activated platelets with circulating monocytes to form monocyte-platelet aggregates (MPA), and investigates the potential usefulness of MPA measurement and/or mo

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46

Karagianni, Anna Eleonora. "Characterisation of the equine macrophage/monocyte." Thesis, University of Edinburgh, 2015. http://hdl.handle.net/1842/15961.

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Inflammatory airway disease (IAD) is a common performance limiting pulmonary disorder in young racehorses in training. Although the precise aetiopathogenesis is poorly understood, proposed mechanisms include opportunistic bacterial infections and/or suboptimal air-hygiene. Since alveolar macrophages (AMs) are the first line defence in the lungs of mammalian species, they may constitute an appropriate therapeutic target cell in the treatment and the prevention of opportunistic airway infections. This thesis aimed to investigate the basic biology of the equine AM. A series of experiments were co

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47

Patel, Dilipkumar. "Cholesterol metabolism in monocyte-derived macrophages." Thesis, Imperial College London, 1990. http://hdl.handle.net/10044/1/46492.

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48

Aldraihim, Asaad. "Development of monocyte and macrophage subpopulations." Thesis, University of Leicester, 2007. http://hdl.handle.net/2381/30494.

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The mechanism by which monocytes develop into different macrophages in vivo is not well understood. This study examines the in vitro development of different types of monocytes-derived macrophages (MDM) from the two main blood monocyte subsets (CD14++ Mo and CD14+16+Mo). Culture of CD14++Mo and CD14+16 +Mo with macrophage colony-stimulating factor (M-CSF) for 7 days generated two phenotypically and functionally different types of macrophage. The hallmark of CD14+16+MDM is the expression of CD209 (DC-SIGN), while CD14++MDM remains CD209 negative. In addition, CD14 +16+MDM show higher expression

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49

Galle, Lauriane. "Caractérisation du rôle de SR-BI dans les macrophages dans le développement de l'athérosclérose." Thesis, Paris 6, 2015. http://www.theses.fr/2015PA066283.

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L’athérosclérose est une pathologie chronique inflammatoire qui résulte du dérèglement d’une réaction inflammatoire non résolue ayant pour but initial d’éliminer l’accumulation excessive de lipides au niveau de l’intima. Cette élimination est exercée par les monocytes/macrophages, dont l’infiltration et l’accumulation au niveau des lésions contribue à l’inflammation chronique locale.SR-BI est un récepteur scavenger multi-fonction capable de reconnaître un large spectre de ligands allant des lipoprotéines natives et modifiées jusqu’aux endotoxines. Outre de jouer un rôle crucial dans l’homéosta

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50

Hundelshausen, Philipp von. "Interaktionen von Monozyten und Endothelzellen unter Flußbedingungen." Diss., lmu, 2003. http://nbn-resolving.de/urn:nbn:de:bvb:19-10376.

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