Journal articles: 'Morue – Physiologie'

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KENIGSBERG, DAVID N., and KENNETH A. ELLENBOGEN. "Physiologic Pacing: More Answers, More Questions." Journal of Cardiovascular Electrophysiology 18, no. 10 (October 2007): 1037–38. http://dx.doi.org/10.1111/j.1540-8167.2007.00948.x.

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Banki, Farzaneh, Rodney J. Mason, Steven R. DeMeester, Jeffrey A. Hagen, Nagammapudur S. Balaji, Peter F. Crookes, Cedric G. Bremner, Jeffrey H. Peters, and Tom R. DeMeester. "Vagal-Sparing Esophagectomy: A More Physiologic Alternative." Annals of Surgery 236, no. 3 (September 2002): 324–36. http://dx.doi.org/10.1097/00000658-200209000-00009.

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Torday, John S., and William B. Miller. "Life is determined by its environment." International Journal of Astrobiology 15, no. 4 (January 26, 2016): 345–50. http://dx.doi.org/10.1017/s1473550415000567.

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AbstractA well-developed theory of evolutionary biology requires understanding of the origins of life on Earth. However, the initial conditions (ontology) and causal (epistemology) bases on which physiology proceeded have more recently been called into question, given the teleologic nature of Darwinian evolutionary thinking. When evolutionary development is focused on cellular communication, a distinctly different perspective unfolds. The cellular communicative-molecular approach affords a logical progression for the evolutionary narrative based on the basic physiologic properties of the cell.Critical to this appraisal is recognition of the cell as a fundamental reiterative unit of reciprocating communication that receives information from and reacts to epiphenomena to solve problems. Following the course of vertebrate physiology from its unicellular origins instead of its overt phenotypic appearances and functional associations provides a robust, predictive picture for the means by which complex physiology evolved from unicellular organisms. With this foreknowledge of physiologic principles, we can determine the fundamentals of Physiology based on cellular first principles using a logical, predictable method. Thus, evolutionary creativity on our planet can be viewed as a paradoxical product of boundary conditions that permit homeostatic moments of varying length and amplitude that can productively absorb a variety of epigenetic impacts to meet environmental challenges.

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Coles, Gerald A. "Towards a More Physiologic Solution for Peritoneal Dialysis." Seminars in Dialysis 8, no. 6 (October 1, 2007): 333–35. http://dx.doi.org/10.1111/j.1525-139x.1995.tb00426.x.

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Bigam, D., A. Hemming, J. Sanabria, L. Wright, J. Karpinsky, R. Croxford, A. Carpentier, P. Greig, and M. Cattral. "Portal-enteric Pancreas Transplantation: A More Physiologic Approach." Transplantation 67, no. 7 (April 1999): S175. http://dx.doi.org/10.1097/00007890-199904150-00699.

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Nelson, David P., Steven M. Schwartz, and Anthony C. Chang. "Neonatal physiology of the functionally univentricular heart." Cardiology in the Young 14, S1 (February 2004): 52–60. http://dx.doi.org/10.1017/s1047951104006304.

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The term “functionally single ventricle” includes a variety of congenital cardiac anomalies where there is only one ventricle pumping blood to the systemic and pulmonary circulations. The physiology in this arrangement is a considerable challenge for the cardiac specialist, because the complexity encountered in patients with these lesions necessitates particularly specialized medical and surgical management. Patients with such functionally univentricular physiology often respond to common interventions, such as supplemental oxygen, mechanical ventilation, and vasoactive drugs, differently than patients with conventional circulations.1 Furthermore, these patients tend to be encountered more frequently by pediatricians and cardiologists because they undergo multiple operations, may be more adversely affected by intercurrent illnesses, or have chronic cardiac problems requiring frequent attention. A thorough understanding of the complexities of the physiology encountered is thus imperative for the pediatric cardiologist. In this review, we will address important physiologic and anatomic principles that influence care of neonates with functionally univentricular hearts. Although the anatomy and physiology of each reconstructive stage of palliation are unique, we will focus upon the pre- and post-operative physiology as encountered in the neonate.

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Robba, Chiara, Lorenzo Ball, and Paolo Pelosi. "Between hypoxia or hyperoxia: not perfect but more physiologic." Journal of Thoracic Disease 10, S16 (June 2018): S2052—S2054. http://dx.doi.org/10.21037/jtd.2018.05.129.

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Colwell, Alice. "To Bathe or Not to Bathe: The Neonatal Question." Neonatal Network 34, no. 4 (2015): 216–19. http://dx.doi.org/10.1891/0730-0832.34.4.216.

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AbstractAfter delivery, newborns go through a series of physiologic changes in an effort to adapt to extrauterine life, with preterm newborns more likely to experience medical problems following this transition. Neonatal hypothermia, defined as a temperature <36.5°C, is a major contributor to neonatal mortality and morbidity.1 Early bathing may be a contributing factor to hypothermia and interfere with the premature neonate’s ability to safely adapt to an extrauterine environment.2 Skin physiology, the physiologic changes that result from bathing, the importance of maintaining vernix for temperature stability, and how policy change and education-based programs for developmentally supportive care will be discussed in an attempt to improve patient care outcomes for neonates in the NICU.

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Anastasiadis, Kyriakos, Polychronis Antonitsis, Apostolos Deliopoulos, and Helena Argiriadou. "A multidisciplinary perioperative strategy for attaining “more physiologic” cardiac surgery." Perfusion 32, no. 6 (March 10, 2017): 446–53. http://dx.doi.org/10.1177/0267659117700488.

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Background: Cardiac surgery is, by definition, a “non-physiologic” intervention associated with systemic adverse effects. Despite advances in surgical technique, cardiopulmonary bypass (CPB) technology as well as anaesthesia management and patient care, there is still significant morbidity and subsequent mortality. Aim: We consider that the contemporary demand for further improving patient outcome mandates the upgrade from optimal perfusion during the procedure as the gold standard to the concept of a “more physiologic” cardiac surgery. Our policy is a multidisciplinary perioperative strategy based on goal-directed perfusion throughout surgery incorporating in-line monitoring. This translates to “prevent rather than correct” malperfusion through real-time adjustment rather than correction of derangement detected late by incremental evaluation. Method: The strategy is based on continuous monitoring of cardiac index, SvO2, DO2i, DO2i/VCO2i and rSO2. Data acquisition is followed by action when needed; this includes stepwise: transfusion, increase of cardiac output and initiation of inotropic/vasoactive support. Moreover, implementation of minimally invasive extracorporeal circulation (MiECC) is considered as a fundamental component of physiologic perfusion when on-CPB, providing improved circulatory support and end-organ protection. Conclusion: We consider that, with this strategy which establishes optimal perfusion perioperatively, we attain the goal of a “more physiologic” cardiac surgery.

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Anastasiadis, Kyriakos, Polychronis Antonitsis, Marco Ranucci, and John Murkin. "Minimally Invasive Extracorporeal Circulation (MiECC): Towards a More Physiologic Perfusion." Journal of Cardiothoracic and Vascular Anesthesia 30, no. 2 (April 2016): 280–81. http://dx.doi.org/10.1053/j.jvca.2016.01.018.

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Kovács, Sándor J. "Diastolic Function in Heart Failure." Clinical Medicine Insights: Cardiology 9s1 (January 2015): CMC.S18743. http://dx.doi.org/10.4137/cmc.s18743.

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Heart failure has reached epidemic proportions, and diastolic heart failure or heart failure with preserved ejection fraction (HFpEF) constitutes about 50% of all heart failure admissions. Long-term prognosis of both reduced ejection fraction heart failure and HFpEF are similarly dismal. No pharmacologic agent has been developed that actually treats or repairs the physiologic deficit(s) responsible for HFpEF. Because the physiology of diastole is both subtle and counterintuitive, its role in heart failure has received insufficient attention. In this review, the focus is on the physiology of diastole in heart failure, the dominant physiologic laws that govern the process in all hearts, how all hearts work as a suction pump, and, therefore, the elucidation and characterization of what actually is meant by “diastolic function”. The intent is for the reader to understand what diastolic function actually is, what it is not, and how to measure it. Proper measurement of diastolic function requires one to go beyond the usual E/A, E/E′, etc. phenomenological metrics and employ more rigorous causality (mathematical modeling) based parameters of diastolic function. The method simultaneously provides new physiologic insight into the meaning of in vivo “equilibrium volume” of the left ventricle (LV), longitudinal versus transverse volume accommodation of the chamber, diastatic “ringing” of the mitral annulus, and the mechanism of L-wave generation, as well as availability of a load-independent index of diastolic function (LIIDF). One important consequence of understanding what diastolic function is, is the recognition that all that current therapies can do is basically alter the load, rather than actually “repair” the functional components (chamber stiffness, chamber relaxation). If beneficial (biological/structural/metabolic) remodeling due to therapy does manifest ultimately as improved diastolic function, it is due to resumption of normal physiology (as in alleviation of ischemia) or activation of compensatory pathways already devised by evolution. In summary, meaningful quantitative characterization of diastolic function in any clinical setting, including heart failure, requires metrics based on physiologic mechanisms that quantify the suction pump attribute of the heart. This requires advancing beyond phenomenological global indexes such as E/A, E/E′, Vp, etc. and employing causality (mathematical modeling) based parameters of diastolic function easily obtained via the parametrized diastolic function (PDF) formalism.

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Busch, Richard F. "Frontal Sinus Osteoma: Complete Removal via Endoscopic Sinus Surgery and Frontal Sinus Trephination." American Journal of Rhinology 6, no. 4 (July 1992): 139–43. http://dx.doi.org/10.2500/105065892781874612.

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Endoscopic sinus surgery has improved our understanding of normal sinus physiology and enabled us to provide better surgical treatment of sinus disease. A method for removal of frontal sinus osteomas was sought that would be less invasive and more physiologic than the conventional osteoplastic flap procedure. Endoscopic sinus surgery has been combined with conventional frontal sinus trephination to achieve total osteoma removal while maintaining normal sinus mucociliary flow. Two successive patients have been treated in this manner with gratifying results.

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Diniz Behn, Cecilia, Eunsook S. Jin, Kate Bubar, Craig Malloy, Elizabeth J. Parks, and Melanie Cree-Green. "Advances in stable isotope tracer methodology part 1: hepatic metabolism via isotopomer analysis and postprandial lipolysis modeling." Journal of Investigative Medicine 68, no. 1 (September 24, 2019): 3–10. http://dx.doi.org/10.1136/jim-2019-001109.

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Stable isotope tracers have been used to gain an understanding of integrative animal and human physiology. More commonly studied organ systems include hepatic glucose metabolism, lipolysis from adipose tissue, and whole body protein metabolism. Recent improvements in isotope methodology have included the use of novel physiologic methods/models and mathematical modeling of data during different physiologic states. Here we review some of the latest advancements in this field and highlight future research needs. First we discuss the use of an oral [U-13C3]-glycerol tracer to determine the relative contribution of glycerol carbons to hepatic glucose production after first cycling through the tricarboxylic acid cycle, entry of glycerol into the pentose phosphate pathway or direct conversion of glycerol into the glucose. Second, we describe an adaptation of the established oral minimal model used to define postprandial glucose dynamics to include glycerol dynamics in an oral glucose tolerance test with a [2H5]-glycerol tracer to determine dynamic changes in lipolysis. Simulation results were optimized when parameters describing glycerol flux were determined with a hybrid approach using both tracer-based calculations and constrained parameter optimization. Both of these methodologies can be used to expand our knowledge of not only human physiology, but also the effects of various nutritional strategies and medications on metabolism.

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Ordoñez, Maria Victoria, Giovanni Biglino, Massimo Caputo, Brenda Kelly, Aarthi Mohan, Johanna Trinder, and Stephanie L. Curtis. "Case of placental insufficiency and premature delivery in a Fontan pregnancy: physiological insights and considerations on risk stratification." Open Heart 8, no. 1 (February 2021): e001211. http://dx.doi.org/10.1136/openhrt-2019-001211.

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ObjectivesThe coexistence of two complex physiologies such as Fontan and pregnancy is still not fully understood. We aim to add a unique and essential knowledge to help our colleagues in the management of Fontan patients that undergo pregnancy as well as the fetus and the placenta perfusion.Methods and resultsWe analyse the coexistence of Fontan and pregnancy physiology on a complex case of a woman with hypoplastic left heart syndrome palliated with a univentricular repair who became pregnant, delivered very prematurely and had atypical placental findings.ConclusionHistopathological analysis of the placenta could help us to refine the understanding of Fontan physiology adaptation during pregnancy, predict women and fetal outcomes as well as to plan a better pre-pregnancy status. However, further evidence is needed in order to reach a more solid and unified conclusion.

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Goldstein, David S. "How does homeostasis happen? Integrative physiological, systems biological, and evolutionary perspectives." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 316, no. 4 (April 1, 2019): R301—R317. http://dx.doi.org/10.1152/ajpregu.00396.2018.

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Homeostasis is a founding principle of integrative physiology. In current systems biology, however, homeostasis seems almost invisible. Is homeostasis a key goal driving body processes, or is it an emergent mechanistic fact? In this perspective piece, I propose that the integrative physiological and systems biological viewpoints about homeostasis reflect different epistemologies, different philosophies of knowledge. Integrative physiology is concept driven. It attempts to explain biological phenomena by continuous formation of theories that experimentation or observation can test. In integrative physiology, “function” refers to goals or purposes. Systems biology is data driven. It explains biological phenomena in terms of “omics”–i.e., genomics, gene expression, epigenomics, proteomics, and metabolomics–it depicts the data in computer models of complex cascades or networks, and it makes predictions from the models. In systems biology, “function” refers more to mechanisms than to goals. The integrative physiologist emphasizes homeostasis of internal variables such as Pco2 and blood pressure. The systems biologist views these emphases as teleological and unparsimonious in that the “regulated variable” (e.g., arterial Pco2 and blood pressure) and the “regulator” (e.g., the “carbistat” and “barostat”) are unobservable constructs. The integrative physiologist views systems biological explanations as not really explanations but descriptions that cannot account for phenomena we humans believe exist, although they cannot be observed directly, such as feelings and, ultimately, the conscious mind. This essay reviews the history of the two epistemologies, emphasizing autonomic neuroscience. I predict rapprochement of integrative physiology with systems biology. The resolution will avoid teleological purposiveness, transcend pure mechanism, and incorporate adaptiveness in evolution, i.e., “Darwinian medicine.”

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Kanat, Mustafa. "Is daytime insulin more physiologic and less atherogenic than bedtime insulin?" Medical Hypotheses 68, no. 6 (January 2007): 1228–32. http://dx.doi.org/10.1016/j.mehy.2006.10.037.

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Rapin, Isabelle, and Judith Gravel. "“Auditory neuropathy”: physiologic and pathologic evidence calls for more diagnostic specificity." International Journal of Pediatric Otorhinolaryngology 67, no. 7 (July 2003): 707–28. http://dx.doi.org/10.1016/s0165-5876(03)00103-4.

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Marozkina, Nadzeya, and Benjamin Gaston. "An Update on Thiol Signaling: S-Nitrosothiols, Hydrogen Sulfide and a Putative Role for Thionitrous Acid." Antioxidants 9, no. 3 (March 10, 2020): 225. http://dx.doi.org/10.3390/antiox9030225.

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Long considered vital to antioxidant defenses, thiol chemistry has more recently been recognized to be of fundamental importance to cell signaling. S-nitrosothiols—such as S-nitrosoglutathione (GSNO)—and hydrogen sulfide (H2S) are physiologic signaling thiols that are regulated enzymatically. Current evidence suggests that they modify target protein function primarily through post-translational modifications. GSNO is made by NOS and other metalloproteins; H2S by metabolism of cysteine, homocysteine and cystathionine precursors. GSNO generally acts independently of NO generation and has a variety of gene regulatory, immune modulator, vascular, respiratory and neuronal effects. Some of this physiology is shared with H2S, though the mechanisms differ. Recent evidence also suggests that molecules resulting from reactions between GSNO and H2S, such as thionitrous acid (HSNO), could also have a role in physiology. Taken together, these data suggest important new potential targets for thiol-based drug development.

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Zhang, Jianhua, John C. Chatham, and Martin E. Young. "Circadian Regulation of Cardiac Physiology: Rhythms That Keep the Heart Beating." Annual Review of Physiology 82, no. 1 (February 10, 2020): 79–101. http://dx.doi.org/10.1146/annurev-physiol-020518-114349.

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On Earth, all life is exposed to dramatic changes in the environment over the course of the day; consequently, organisms have evolved strategies to both adapt to and anticipate these 24-h oscillations. As a result, time of day is a major regulator of mammalian physiology and processes, including transcription, signaling, metabolism, and muscle contraction, all of which oscillate over the course of the day. In particular, the heart is subject to wide fluctuations in energetic demand throughout the day as a result of waking, physical activity, and food intake patterns. Daily rhythms in cardiovascular function ensure that increased delivery of oxygen, nutrients, and endocrine factors to organs during the active period and the removal of metabolic by-products are in balance. Failure to maintain these physiologic rhythms invariably has pathologic consequences. This review highlights rhythms that underpin cardiac physiology. More specifically, we summarize the key aspects of cardiac physiology that oscillate over the course of the day and discuss potential mechanisms that regulate these 24-h rhythms.

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Coles, Jonathan P., Alison S. Cunningham, Raymond Salvador, Doris A. Chatfield, Adrian Carpenter, John D. Pickard, and David K. Menon. "Early Metabolic Characteristics of Lesion and Nonlesion Tissue after Head Injury." Journal of Cerebral Blood Flow & Metabolism 29, no. 5 (March 18, 2009): 965–75. http://dx.doi.org/10.1038/jcbfm.2009.22.

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We defined lesion and structurally normal regions using magnetic resonance imaging at follow-up in patients recovering from head injury. Early metabolic characteristics in these regions of interest (ROIs) were compared with physiology in healthy volunteers. Fourteen patients with severe head injury had positron emission tomography within 72 h, and magnetic resonance imaging at 3 to 18 months after injury. Cerebral blood flow (CBF), oxygen utilization (CMRO2), and oxygen extraction fraction (OEF) were all lower in lesion ROIs, compared with nonlesion and control ROIs ( P < 0.001); however, there was substantial overlap in physiology. Control ROIs showed close coupling among CBF, blood volume (CBV), and CMRO2, whereas relationships within lesion and nonlesion ROIs were abnormal. The relationship between CBF and CMRO2 generally remained coupled but the slope was reduced; that for CBF and OEF was variable; whereas that between CBF and CBV was highly variable. There was considerable heterogeneity between and within patients. Although irreversibly damaged tissue is characterized by marked derangements in physiology, a more detailed analysis shows acute changes in physiology and physiologic relationships within regions of the brain that appear structurally normal at follow-up. Such pathophysiological derangements may result in selective neuronal loss and impact on functional outcome.

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Goldberger, Jeffrey J., Haris Subacius, and Jason Ng. "HEART RATE VERSUS RR INTERVAL RECOVERY AFTER EXERCISE: WHICH IS MORE PHYSIOLOGIC?" Journal of the American College of Cardiology 59, no. 13 (March 2012): E1944. http://dx.doi.org/10.1016/s0735-1097(12)61945-7.

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Johnson, Tiffany L., Gilda A. Barabino, and Robert M. Nerem. "Engineering more physiologic in vitro models for the study of vascular biology." Progress in Pediatric Cardiology 21, no. 2 (March 2006): 201–10. http://dx.doi.org/10.1016/j.ppedcard.2005.11.011.

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Sigmon, Hilary D., and Patricia A. Grady. "Physiologic Variables in Nursing Research: Progress, Yes…But More Work is Needed." Image: the Journal of Nursing Scholarship 28, no. 2 (June 1996): 94. http://dx.doi.org/10.1111/j.1547-5069.1996.tb01198.x.

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Weiss, Sara O., Carmen C. Sucharov, and Carlin S. Long. "HDAC inhibitors promote a more differentiated and physiologic genotype in cardiac hypertrophy." Journal of Cardiac Failure 10, no. 4 (August 2004): S61. http://dx.doi.org/10.1016/j.cardfail.2004.06.157.

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Gardner, Reed M., and Marianne Hujcs. "Fundamentals of Physiologic Monitoring." AACN Advanced Critical Care 4, no. 1 (February 1, 1993): 11–24. http://dx.doi.org/10.4037/15597768-1993-1002.

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For centuries, medical practitioners had no electronic medical instruments and had to rely on their senses of sight, hearing, smell, taste, and touch to obtain physiologic measurements. Although it is possible to estimate blood pressure by palpating the pulse at the radial or brachial artery, such estimates are not accurate. Determining arterial oxygen saturation of hemoglobin is more complex: how “blue” a patient appears depends on skin coloration, lighting, and the examiner’s sense of color. Finally, using radiographic images to validate pulmonary edema when clinicians suspect that there is an elevated left atrial or pulmonary artery wedge pressure also challenges human senses. However, today’s medical instruments use transducers and signal processors to convert patient information into a form that clinicians can easily perceive and understand. This article defines terms used with biomedical instrumentation and discusses the components of ideal physiologic patient monitoring systems

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Gilbert, Donald L., David A. Huddleston, Steve W. Wu, Ernest V. Pedapati, Paul S. Horn, Kathryn Hirabayashi, Deanna Crocetti, Eric M. Wassermann, and Stewart H. Mostofsky. "Motor cortex inhibition and modulation in children with ADHD." Neurology 93, no. 6 (July 17, 2019): e599-e610. http://dx.doi.org/10.1212/wnl.0000000000007899.

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ObjectiveCompared to typically developing (TD) peers, children with attention-deficit/hyperactivity disorder (ADHD) consistently demonstrate impaired transcranial magnetic stimulation (TMS)-evoked short interval cortical inhibition (SICI) of motor evoked potentials (MEPs) in resting motor cortex (M1). To determine whether perturbed M1 physiology also reflects clinically relevant behavioral dysfunction, we evaluated M1 physiology during a cognitive control task taxing motor response selection/inhibition.MethodsIn this case-control study, behavioral ratings, motor skill (assessed using standardized examination), and left M1 physiology were evaluated in 131 right-handed, 8- to 12-year-old children (66 ADHD: mean 10.5 years, 43 male; 65 TD: mean 10.6 years, 42 male). The primary outcomes were MEP amplitudes and SICI, evaluated during rest and during a modified “racecar” Slater-Hammel stop signal reaction task, with TMS pulses administered 150 ms prior to the target go action and after the dynamic stop cue.ResultsGo responses were significantly slower (p = 0.01) and more variable (p = 0.002) in ADHD. Children with ADHD showed less M1 SICI at rest (p = 0.02) and during go (p = 0.03) and stop trials (p = 0.02). Rest M1 excitability increased during response inhibition task engagement (p < 0.0001). This Task-Related Up-Modulation (TRUM) was less robust across and within groups, with diminished task upmodulation associated with significantly more severe ADHD behavioral ratings and slower stop signal reaction times.ConclusionChildren with ADHD show anomalous motor cortex physiology, with deficient SICI across behavioral states and less TRUM from rest to action selection. Associations of these physiologic measures with ADHD symptoms and cognitive control measures support further investigation into biological mechanisms.

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Turner, J. N., D. H. Szarowski, W. Shain, M. Davis-Cox, D. O. Carpenter, and M. Fejtl. "Three-dimensional imaging and physiology of live neurons and glia: Confocal light and correlative high-voltage Electron Microscopy." Proceedings, annual meeting, Electron Microscopy Society of America 51 (August 1, 1993): 162–63. http://dx.doi.org/10.1017/s0424820100146655.

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Correlating physiologic measures with three-dimensional (3D) imaging at the light and electron microscopic levels is a powerful combination of methods for studying the structure and function of biological systems. Neurobiology is an ideal field for the application of these methods because neurons and glia have complex and extensive 3D structure, and their physiology is under intense study. Neurons, such as those studied here from Aplysia, can be more than 100 μm in diameter, and glia undergo large scale 3D shape change as a function of a number of physiologic parameters. The ability to accurately quantitate the 3D structure, volume and surface area of live neurons and glia is important to our understanding of the complex function of these cells.Neurons were isolated from the major ganglia of juvenile Aplysia Californica and glia were obtained from long term cultures of LRM 55 cells or as primary isolates from rats. Cultures were exposed to Dil dissolved in DMSO with or without 20% Pluronic F-127 and added to the culture media. The imaging instrument was an Olympus IMT-2 and a Bio-Rad MRC-600.

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Wieser, Fritz, Leslie Waite, Christophe Depoix, and Robert N. Taylor. "PPAR Action in Human Placental Development and Pregnancy and Its Complications." PPAR Research 2008 (2008): 1–14. http://dx.doi.org/10.1155/2008/527048.

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During pregnancy crucial anatomic, physiologic, and metabolic changes challenge the mother and the fetus. The placenta is a remarkable organ that allows the mother and the fetus to adapt to the new metabolic, immunologic, and angiogenic environment imposed by gestation. One of the physiologic systems that appears to have evolved to sustain this metabolic regulation is mediated by peroxisome proliferator-activated receptors (PPARs). In clinical pregnancy-specific disorders, including preeclampsia, gestational diabetes, and intrauterine growth restriction, aberrant regulation of components of the PPAR system parallels dysregulation of metabolism, inflammation and angiogenesis. This review summarizes current knowledge on the role of PPARs in regulating human trophoblast invasion, early placental development, and also in the physiology of clinical pregnancy and its complications. As increasingly indicated in the literature, pregnancy disorders, such as preeclampsia and gestational diabetes, represent potential targets for treatment with PPAR ligands. With the advent of more specific PPAR agonists that exhibit efficacy in ameliorating metabolic, inflammatory, and angiogenic disturbances, further studies of their application in pregnancy-related diseases are warranted.

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Birukova, Anastasiya, Jaime Cyphert-Daly, Robert Ian Cumming, Yen-Rei Yu, Kymberly M. Gowdy, Loretta G. Que, and Robert M. Tighe. "Sex Modifies Acute Ozone-Mediated Airway Physiologic Responses." Toxicological Sciences 169, no. 2 (March 2, 2019): 499–510. http://dx.doi.org/10.1093/toxsci/kfz056.

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Abstract Sex differences clearly exist in incidence, susceptibility, and severity of airway disease and in pulmonary responses to air pollutants such as ozone (O3). Prior rodent O3 exposure studies demonstrate sex-related differences in the expression of lung inflammatory mediators and signaling. However, whether or not sex modifies O3-induced airway physiologic responses remains less explored. To address this, we exposed 8- to 10-week-old male and female C57BL/6 mice to either 1 or 2 ppm O3 or filtered air (FA) for 3 h. At 12, 24, 48, and 72 h following exposure, we assessed airway hyperresponsiveness to methacholine (MCh), bronchoalveolar lavage fluid cellularity, cytokines and total protein/albumin, serum progesterone, and whole lung immune cells by flow cytometry. Male mice generated consistent airway hyperresponsiveness to MCh at all time points following exposure. Alternatively, females had less consistent airway physiologic responses to MCh, which were more variable between individual experiments and did not correlate with serum progesterone levels. Bronchoalveolar lavage fluid total cells peaked at 12 h and were persistently elevated through 72 h. At 48 h, bronchoalveolar lavage cells were greater in females versus males. Bronchoalveolar lavage fluid cytokines and total protein/albumin increased following O3 exposure without sex differences. Flow cytometry of whole lung tissue identified dynamic O3-induced immune cell changes also independent of sex. Our results indicate sex differences in acute O3-induced airway physiology responses and airspace influx without significant difference in other injury and inflammation measures. This study highlights the importance of considering sex as a biological variable in acute O3-induced airway physiology responses.

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Kuschner, Cyrus E., and Lance B. Becker. "Recent advances in personalizing cardiac arrest resuscitation." F1000Research 8 (June 21, 2019): 915. http://dx.doi.org/10.12688/f1000research.17554.1.

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Cardiac arrest remains a significant cause of death and disability throughout the world. However, as our understanding of cardiac arrest and resuscitation physiology has developed, new technologies are fundamentally altering our potential to improve survival and neurologic sequela. Some advances are relatively simple, requiring only alterations in current basic life support measures or integration with pre-hospital organization, whereas others, such as extra-corporeal membrane oxygenation, require significant time and resource investments. When combined with consistent rescuer and patient-physiologic monitoring, these innovations allow an unprecedented capacity to personalize cardiac arrest resuscitation to patient-specific pathophysiology. However, as more extensive options are established, it can be difficult for providers to incorporate novel resuscitation techniques into a cardiac arrest protocol which can fit a wide variety of cases with varying complexity. This article will explore recent advances in our understanding of cardiac arrest physiology and resuscitation sciences, with particular focus on the metabolic phase after significant ischemia has been induced. To this end, we establish a practical consideration for providers seeking to integrate novel advances in cardiac arrest resuscitation into daily practice.

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Weinger, M., and K. Gurushanthaiah. "GRAPHICAL DISPLAYS MAY ALLOW MORE RAPID AND ACCURATE DETECTION OF ACUTE PHYSIOLOGIC CHANGES." Anesthesiology 81, SUPPLEMENT (September 1994): A580. http://dx.doi.org/10.1097/00000542-199409001-00579.

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Balachandran, M., K. L. Cross, and M. Podar. "Single-Cell Genomics and the Oral Microbiome." Journal of Dental Research 99, no. 6 (February 24, 2020): 613–20. http://dx.doi.org/10.1177/0022034520907380.

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The human oral cavity is one of the first environments where microbes have been discovered and studied since the dawn of microbiology. Nevertheless, approximately 200 types of bacteria from the oral microbiota have remained uncultured in the laboratory. Some are associated with a healthy oral microbial community, while others are linked to oral diseases, from dental caries to gum disease. Single-cell genomics has enabled inferences on the physiology, virulence, and evolution of such uncultured microorganisms and has further enabled isolation and cultivation of several novel oral bacteria, including the discovery of novel interspecies interactions. This review summarizes some of the more recent advances in this field, which is rapidly moving toward physiologic characterization of single cells and ultimately cultivation of the yet uncultured. A combination of traditional microbiological approaches with genomic-based physiologic predictions and isolation strategies may lead to the oral microbiome being the first complex microbial community to have all its members cultivable in the laboratory. Studying the biology of the individual microbes when in association with other members of the community, in controlled laboratory conditions and in vivo, should lead to a better understanding of oral dysbiosis and its prevention and reversion.

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Gupta, Madhulika A., and Harvey Moldofsky. "Dysthymic Disorder and Rheumatic Pain Modulation Disorder (Fibrositis Syndrome): A Comparison of Symptoms and Sleep Physiology." Canadian Journal of Psychiatry 31, no. 7 (October 1986): 608–16. http://dx.doi.org/10.1177/070674378603100702.

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It has been suggested that “fibrositis” or rheumatic pain modulation disorder (RPMD) is a varient of depressive illness. Both disorders are associated with abnormalities in sleep physiology. Since the clinical features of RPMD do not meet all the criteria for a major depressive disorder, the symptoms and sleep phsyiology in subjects with dysthmic disorder (DSM III criteria) (N = 6), and RMPD (N = 6) were compared, in order to determine the similarity between the two groups. The sleep physiology in dysthymic disorder was first examined over three consecutive nights since a systematic evaluation of the sleep physiology in this group of disorders has not yet been reported. All dysthymic patients showed episodic bursts of high-amplitude (75–150 microvolts) theta (3–5 Hz) bursts in stage 2 sleep, and REM onset latency was abbreviated only on night 2. The theta bursts have not been previously reported, and may be an early marker of disorganization of non-REM sleep in the dysthymic subjects. The comparison of the two groups revealed that RPMD subjects reported more pre- and post-sleep pain (p < 0.01), lighter sleep (p < 0.01), and more physical ailments during sleep (p < 0.01), and had more alpha (7–11.5 Hz) in non-REM sleep (p < 0.01). The dysthymic subjects who reported deeper sleep (p < 0.01), had a greater sleep continuity disturbance with longer stage 2 onset latency (p < 0.05), fewer hours of sleep (p < 0.05), more wakefulness after sleep onset (p < 0.05), more awakenings per hour of sleep (p < 0.01) and more stage changes per hour of sleep (p < 0.01), and showed theta bursts in stage 2 (p < 0.01). The distinctive symptoms and sleep physiologies in the two groups suggest that the two disorders are not related.

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Shah, Samit, and Steven Pfau. "Coronary Physiology in the Cardiac Catheterization Laboratory." Journal of Clinical Medicine 8, no. 2 (February 18, 2019): 255. http://dx.doi.org/10.3390/jcm8020255.

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Coronary angiography has been the principle modality for assessing the severity of atherosclerotic coronary artery disease for several decades. However, there is a complex relationship between angiographic coronary stenosis and the presence or absence of myocardial ischemia. Recent technological advances now allow for the assessment of coronary physiology in the catheterization laboratory at the time of diagnostic coronary angiography. Early studies focused on coronary flow reserve (CFR) but more recent work has demonstrated the physiologic accuracy and prognostic value of the fractional flow reserve (FFR) and instantaneous wave free ratio (iFR) for the assessment of coronary artery disease. These measurements have been validated in large multi-center clinical trials and have become indispensable tools for guiding revascularization in the cardiac catheterization laboratory. The physiological assessment of chest pain in the absence of epicardial coronary artery disease involves coronary thermodilution to obtain the index of microcirculatory resistance (IMR) or Doppler velocity measurement to determine the coronary flow velocity reserve (CFVR). Physiology-based coronary artery assessment brings “personalized medicine” to the catheterization laboratory and allows cardiologists and referring providers to make decisions based on objective findings and evidence-based treatment algorithms. The purpose of this review is to describe the theory, technical aspects, and relevant clinical trials related to coronary physiology assessment for an intended audience of general medical practitioners.

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Klein, Deborah Goldenberg. "Physiologic Response to Traumatic Shock." AACN Advanced Critical Care 1, no. 3 (November 1, 1990): 505–21. http://dx.doi.org/10.4037/15597768-1990-3006.

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Traumatic injury results in major physiologic alterations that begin at the time of injury and persist until recovery is complete. The response of the body is divided into two phases — the acute phase and the flow phase. The acute phase is characterized by shock with changes in hormone concentration. These hormones, either alone or in combination, result in lipolysis, amino acid release, gluconeogenesis, and glycolysis. The flow phase of injury is a catabolic process that is characterized by an increased protein metabolism. Hypermetabolism and increased nitrogen losses are seen. The magnitude of these alterations is directly related to the severity of injury. Tissues with the highest oxygen consumption are more susceptible to injury and death. Cellular function does not depend on oxygen alone but also on the ability of the cells to use available oxygen. If the body is unable to compensate through biochemical, hormonal, and metabolic activities, an irreversible state results unless appropriate interventions are instituted promptly

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Binette, Tanya M., Fletcher B. Taylor, Glenn Peer, and Laszlo Bajzar. "Thrombin-thrombomodulin connects coagulation and fibrinolysis: more than an in vitro phenomenon." Blood 110, no. 9 (November 1, 2007): 3168–75. http://dx.doi.org/10.1182/blood-2007-03-078824.

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Abstract Thrombin activatable fibrinolysis inhibitor (TAFI), when activated, forms a basic carboxypeptidase that can inhibit fibrinolysis. Potential physiologic activators include both thrombin and plasmin. In vitro, thrombomodulin and glycosaminoglycans increase the catalytic efficiency of TAFI activation by thrombin and plasmin, respectively. The most relevant (patho-) physiologic activator of TAFI has not been disclosed. Our purpose was to identify the physiologic activator of TAFI in vivo. Activation of protein C (a thrombin-thrombomodulin–dependent reaction), prothrombin, and plasminogen occurs during sepsis. Thus, a baboon model of Escherichia coli–induced sepsis, where multiple potential activators of TAFI are elaborated, was used to study TAFI activation. A monoclonal antibody (mAbTAFI/TM#16) specifically inhibiting thrombin-thrombomodulin–dependent activation of TAFI was used to assess the contribution of thrombin-thrombomodulin in TAFI activation in vivo. Coinfusion of mAbTAFI/TM#16 with a lethal dose of E coli prevented the complete consumption of TAFI observed without mAbTAFI/TM#16. The rate of fibrin degradation products formation is enhanced in septic baboons treated with the mAbTAFI/TM#16; therefore, TAFI activation appears to play a key role in the extent of fibrin(ogen) consumption during E coli challenge, and thrombin-thrombomodulin, in a baboon model of E coli–induced sepsis, appears to be the predominant activator of TAFI.

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Jain, Atul, Vinod Aurwade, Rachana Bahuguna, and Abhinay Agarwal. "Biomarkers of Healthy and Diseased Pulp and Periapical Tissue: A Review." UNIVERSITY JOURNAL OF DENTAL SCIENCES 6, no. 2 (August 27, 2020): 94–100. http://dx.doi.org/10.21276/ujds.2020.6.2.14.

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Establishing an accurate diagnosis is the first and prime requisite, in initiating the correct treatment, but unfortunately due to the limitations of the diagnostic procedure, this step can be highly confounding. Biological marker serves as a parameter, that is indicative of underlying physiology and health of the tissue. Healthy and diseased pulp and periapical tissue, tend to reflect physiologic and pathologic agents, which on isolation and estimation, can help diagnose, the condition more conclusively. Use of these agents, along with signs, symptoms, result of various tests, can aid in establishing the condition, more precisely. This review article focuses upon, identification of biomarkers, that play important role in pathogenesis of dental caries, pulpal and periapical pathosis and help to diagnose the pathological and healthy condition accurately, thus guide decisions, regarding clinical treatment. Key words: biological products, biomarkers, dental Caries, dental Pulp, diagnostic Tests

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Beck, Alan M., and Dalton Morgan. "Utility of Mobility in Post Open-Heart Surgery Patients: A Pilot Trial." Journal of Clinical Exercise Physiology 8, no. 2 (June 1, 2019): 82–85. http://dx.doi.org/10.31189/2165-6193-8.2.82.

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ABSTRACT Background: As the role of the clinical exercise physiologist expands, early mobility is an area of potential focus. A rural Midwestern intensive care unit began a mobility program alongside its open-heart surgery program. The mobility specialist, who was trained as a clinical exercise physiologist, was tasked to ambulate the open-heart surgery patients. The purpose of this pilot study was to determine the effectiveness of a mobility specialist on ambulation frequency and distance on post open-heart surgery patients. Methods: Data were collected retrospectively for 1 month on ambulation frequency and distance to determine the mobility specialist's impact on the variables. Results: Data was collected on 18 patients (15 male, 3 female) over the month. Overall, when the mobility specialist was present, patients ambulated further (M = 421 feet versus 189 feet, P = 0.039) and more often (M = 3.32 versus 1.43 ambulations per day, P < 0.001). Conclusion: In this study, having a mobility specialist with a background in exercise physiology led to more frequent and distant ambulation. Therefore, a mobility specialist should be considered an integral member of a multidisciplinary clinical team in rural intensive care units.

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Diodato, M. D., N. R. Shah, S. M. Prasad, S. Mizutani, E. L. Racen, J. L. Lawton, and R. J. Damiano. "Progress towards a more physiologic approach to heart preservation: the advantages of hyperpolarized arrest." Journal of Heart and Lung Transplantation 22, no. 1 (January 2003): S103. http://dx.doi.org/10.1016/s1053-2498(02)00766-0.

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Schwartz, Joyce G., William T. Phillips, and Oded Langer. "Use of a More Physiologic Oral Glucose Solution During Testing for Gestational Diabetes Mellitus." American Journal of Clinical Pathology 97, no. 6 (June 1, 1992): 831–35. http://dx.doi.org/10.1093/ajcp/97.6.831.

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41

Schwartz, Joyce G., William T. Phillips, Michael R. Blumhardt, and Oded Langer. "Use of a more physiologic oral glucose solution during screening for gestational diabetes mellitus." American Journal of Obstetrics and Gynecology 171, no. 3 (September 1994): 685–91. http://dx.doi.org/10.1016/0002-9378(94)90082-5.

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Drozdowicz, Zbigniew. "Od spekulatywnego humoryzmu do eksperymentalnego fizjologizmu." Humaniora. Czasopismo Internetowe 34, no. 2 (June 15, 2021): 61–75. http://dx.doi.org/10.14746/h.2021.2.4.

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Pointing out the boundary after crossing which physiology of man stops being pseudoscience and becomes true science had been a task undertaken for a long time. Some of those attempts caused a greater and some a lesser reservation. Relatively limited ways of recognizing life processes, including the preveiling for centuries ban of doing autopsy, on many occasions opened widely a door for various pseudo and quasi scientific speculations. Numerous of them had survived through the centuries. Although with time passing by it had been proved that some of them a scientifically groundless they had been replaced with new ones. In my remarks I recall the example of speculative humourism as well several examples of experimental physiologism, including behaviorism which is still popular in science. Certainly, more of such examples can be given at any time. We cannot say however that diverse pseudoscientific speculations in physiology have been eradicated. We might say that today we are given a much smaller space for speculativism in that or other areas of biological research.

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Chmielewska, Ewa, and Paweł Kafarski. "Physiologic Activity of Bisphosphonates – Recent Advances." Open Pharmaceutical Sciences Journal 3, no. 1 (May 30, 2016): 56–78. http://dx.doi.org/10.2174/1874844901603010056.

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Background: Bisphosphonates are drugs commonly used for the medication and prevention of diseases caused by decreased mineral density. Despite such important medicinal use, they display a variety of physiologic activities, which make them promising anti-cancer, anti-protozoal, antibacterial and antiviral agents. Objective: To review physiological activity of bisphosphonates with special emphasis on their ongoing and potential applications in medicine and agriculture. Method: Critical review of recent literature data. Results: Comprehensive review of activities revealed by bisphosphonates. Conclusion: although bisphosphonates are mostly recognized by their profound effects on bone physiology their medicinal potential has not been fully evaluated yet. Literature data considering enzyme inhibition suggest possibilities of far more wide application of these compounds. These applications are, however, limited by their low bioavailability and therefore intensive search for new chemical entities overcoming this shortage are carried out.

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Morin, Scott J. "Reduction in oxygen tension to 2% in extended culture: a more physiologic system may mean more blastocysts available for transfer." Fertility and Sterility 109, no. 6 (June 2018): 1002–3. http://dx.doi.org/10.1016/j.fertnstert.2018.03.011.

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Moore, Ryan A., Saul Flores, and David S. Cooper. "Critical care of patients with paediatric valvar cardiac disease." Cardiology in the Young 24, no. 6 (December 2014): 1071–76. http://dx.doi.org/10.1017/s1047951114002169.

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AbstractValvar cardiac disease accounts for a large percentage of heart lesions in the paediatric population. There are a variety of congenital and acquired valvar lesions produced by different mechanisms that produce unique physiologies. With the development of more advanced cardiac interventional procedures and congenital cardiac surgery techniques, most of these lesions can be repaired in a single or multi-stage approach. Given the potential for significant symptomatology before intervention, it is imperative to review the critical care management of paediatric valvar heart disease. The purpose of this article is to provide an overview of the current critical care management of valvar cardiac disease. We approach each cardiac valve individually in order to review the pathophysiology caused by valve dysfunction (obstruction and incompetence) and elaborate on the most frequent diagnoses, specific cardiac physiology, potential for other organ system associations, and perioperative management.

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Evans, R. Scott, Kathryn G. Kuttler, Kathy J. Simpson, Stephen Howe, Peter F. Crossno, Kyle V. Johnson, Misty N. Schreiner, et al. "Automated detection of physiologic deterioration in hospitalized patients." Journal of the American Medical Informatics Association 22, no. 2 (August 27, 2014): 350–60. http://dx.doi.org/10.1136/amiajnl-2014-002816.

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Abstract Objective Develop and evaluate an automated case detection and response triggering system to monitor patients every 5 min and identify early signs of physiologic deterioration. Materials and methods A 2-year prospective, observational study at a large level 1 trauma center. All patients admitted to a 33-bed medical and oncology floor (A) and a 33-bed non-intensive care unit (ICU) surgical trauma floor (B) were monitored. During the intervention year, pager alerts of early physiologic deterioration were automatically sent to charge nurses along with access to a graphical point-of-care web page to facilitate patient evaluation. Results Nurses reported the positive predictive value of alerts was 91–100% depending on erroneous data presence. Unit A patients were significantly older and had significantly more comorbidities than unit B patients. During the intervention year, unit A patients had a significant increase in length of stay, more transfers to ICU (p = 0.23), and significantly more medical emergency team (MET) calls (p = 0.0008), and significantly fewer died (p = 0.044) compared to the pre-intervention year. No significant differences were found on unit B. Conclusions We monitored patients every 5 min and provided automated pages of early physiologic deterioration. This before–after study found a significant increase in MET calls and a significant decrease in mortality only in the unit with older patients with multiple comorbidities, and thus further study is warranted to detect potential confounding. Moreover, nurses reported the graphical alerts provided information needed to quickly evaluate patients, and they felt more confident about their assessment and more comfortable requesting help.

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Turko, Andy J., Giulia S. Rossi, and Patricia A. Wright. "More than Breathing Air: Evolutionary Drivers and Physiological Implications of an Amphibious Lifestyle in Fishes." Physiology 36, no. 5 (September 1, 2021): 307–14. http://dx.doi.org/10.1152/physiol.00012.2021.

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Amphibious and aquatic air-breathing fishes both exchange respiratory gasses with the atmosphere, but these fishes differ in physiology, ecology, and possibly evolutionary origins. We introduce a scoring system to characterize interspecific variation in amphibiousness and use this system to highlight important unanswered questions about the evolutionary physiology of amphibious fishes.

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Maazouzi, W., A. Boutayeb, H. Benyoussef, A. Benyass, H. Yakin, and O. Amar. "OP-019 THE FAST-TEC RING: TOWARDS A MORE PHYSIOLOGIC TRICUSPID VALVE REPAIR INITIAL RESULTS." International Journal of Cardiology 155 (March 2012): S4. http://dx.doi.org/10.1016/s0167-5273(12)70013-x.

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Martin, Turf D. "IGIV: Contents, properties, and methods of industrial production—evolving closer to a more physiologic product." International Immunopharmacology 6, no. 4 (April 2006): 517–22. http://dx.doi.org/10.1016/j.intimp.2005.11.005.

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Diodato, Michael D., Nirav R. Shah, Sunil M. Prasad, Erica L. Racen, Shinichi Mizutani, Jennifer S. Lawton, and Ralph J. Damiano. "Progress Towards a More Physiologic Approach to Donor Heart Preservation: The Advantages of Hyperpolarized Arrest." Journal of Heart and Lung Transplantation 24, no. 9 (September 2005): 1362–68. http://dx.doi.org/10.1016/j.healun.2004.10.011.

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Journal articles on the topic 'Morue – Physiologie'

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