Relevant bibliographies by topics / Motor Neuron differentiation

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  2. Dissertations / Theses
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  5. Conference papers

Academic literature on the topic 'Motor Neuron differentiation'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Motor Neuron differentiation"

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Nango, Hiroshi, Yasuhiro Kosuge, Masaki Sato, et al. "Highly Efficient Conversion of Motor Neuron-Like NSC-34 Cells into Functional Motor Neurons by Prostaglandin E2." Cells 9, no. 7 (2020): 1741. http://dx.doi.org/10.3390/cells9071741.

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Motor neuron diseases are a group of progressive neurological disorders that degenerate motor neurons. The neuroblastoma × spinal cord hybrid cell line NSC-34 is widely used as an experimental model in studies of motor neuron diseases. However, the differentiation efficiency of NSC-34 cells to neurons is not always sufficient. We have found that prostaglandin E2 (PGE2) induces morphological differentiation in NSC-34 cells. The present study investigated the functional properties of PGE2-differentiated NSC-34 cells. Retinoic acid (RA), a widely-used agent inducing cell differentiation, facilita
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Lin, Yu-Lung, Yi-Wei Lin, Jennifer Nhieu, Xiaoyin Zhang, and Li-Na Wei. "Sonic Hedgehog-Gli1 Signaling and Cellular Retinoic Acid Binding Protein 1 Gene Regulation in Motor Neuron Differentiation and Diseases." International Journal of Molecular Sciences 21, no. 11 (2020): 4125. http://dx.doi.org/10.3390/ijms21114125.

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Cellular retinoic acid-binding protein 1 (CRABP1) is highly expressed in motor neurons. Degenerated motor neuron-like MN1 cells are engineered by introducing SODG93A or AR-65Q to model degenerated amyotrophic lateral sclerosis (ALS) or spinal bulbar muscular atrophy neurons. Retinoic acid (RA)/sonic hedgehog (Shh)-induced embryonic stem cells differentiation into motor neurons are employed to study up-regulation of Crabp1 by Shh. In SODG93A or AR-65Q MN1 neurons, CRABP1 level is reduced, revealing a correlation of motor neuron degeneration with Crabp1 down-regulation. Up-regulation of Crabp1 b
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Bax, Monique, Jessie McKenna, Dzung Do-Ha, et al. "The Ubiquitin Proteasome System Is a Key Regulator of Pluripotent Stem Cell Survival and Motor Neuron Differentiation." Cells 8, no. 6 (2019): 581. http://dx.doi.org/10.3390/cells8060581.

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The ubiquitin proteasome system (UPS) plays an important role in regulating numerous cellular processes, and a dysfunctional UPS is thought to contribute to motor neuron disease. Consequently, we sought to map the changing ubiquitome in human iPSCs during their pluripotent stage and following differentiation to motor neurons. Ubiquitinomics analysis identified that spliceosomal and ribosomal proteins were more ubiquitylated in pluripotent stem cells, whilst proteins involved in fatty acid metabolism and the cytoskeleton were specifically ubiquitylated in the motor neurons. The UPS regulator, u
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Hallam, S., E. Singer, D. Waring, and Y. Jin. "The C. elegans NeuroD homolog cnd-1 functions in multiple aspects of motor neuron fate specification." Development 127, no. 19 (2000): 4239–52. http://dx.doi.org/10.1242/dev.127.19.4239.

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The basic helix-loop-helix transcription factor NeuroD (Neurod1) has been implicated in neuronal fate determination, differentiation and survival. Here we report the expression and functional analysis of cnd-1, a C. elegans NeuroD homolog. cnd-1 expression was first detected in neuroblasts of the AB lineage in 14 cell embryos and maintained in many neuronal descendants of the AB lineage during embryogenesis, diminishing in most terminally differentiated neurons prior to hatching. Specifically, cnd-1 reporter genes were expressed in the precursors of the embryonic ventral cord motor neurons and
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Martinez-Morales, J. R., J. A. Barbas, E. Marti, P. Bovolenta, D. Edgar, and A. Rodriguez-Tebar. "Vitronectin is expressed in the ventral region of the neural tube and promotes the differentiation of motor neurons." Development 124, no. 24 (1997): 5139–47. http://dx.doi.org/10.1242/dev.124.24.5139.

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The extracellular matrix protein vitronectin and its mRNA are present in the embryonic chick notochord, floor plate and in the ventral neural tube at the time position of motor neuron generation. When added to cultures of neural tube explants of developmental stage 9, vitronectin promotes the generation of motor neurons in the absence of either notochord or exogenously added Sonic hedgehog. Conversely, the neutralisation of endogenous vitronectin with antibodies inhibits over 90% motor neuron differentiation in co-cultured neural tube/notochord explants, neural tube explants cultured in the pr
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Pattyn, A., M. Hirsch, C. Goridis, and J. F. Brunet. "Control of hindbrain motor neuron differentiation by the homeobox gene Phox2b." Development 127, no. 7 (2000): 1349–58. http://dx.doi.org/10.1242/dev.127.7.1349.

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Motor neurons are a widely studied model of vertebrate neurogenesis. They can be subdivided in somatic, branchial and visceral motor neurons. Recent studies on the dorsoventral patterning of the rhombencephalon have implicated the homeobox genes Pax6 and Nkx2.2 in the early divergence of the transcriptional programme of hindbrain somatic and visceral motor neuronal differentiation. We provide genetic evidence that the paired-like homeodomain protein Phox2b is required for the formation of all branchial and visceral, but not somatic, motor neurons in the hindbrain. In mice lacking Phox2b, both
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Lee, S., R. Shen, H. H. Cho, et al. "STAT3 promotes motor neuron differentiation by collaborating with motor neuron-specific LIM complex." Proceedings of the National Academy of Sciences 110, no. 28 (2013): 11445–50. http://dx.doi.org/10.1073/pnas.1302676110.

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Jungbluth, S., G. Koentges, and A. Lumsden. "Coordination of early neural tube development by BDNF/trkB." Development 124, no. 10 (1997): 1877–85. http://dx.doi.org/10.1242/dev.124.10.1877.

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Neurotrophins signal through members of the trk family of tyrosine kinase receptors and are known to regulate several neuronal properties. Although initially characterized by their ability to prevent naturally occurring cell death of subsets of neurons during development, neurotrophins can also regulate the proliferation and differentiation of precursor cells. Here we report a novel involvement of neurotrophins in early development of the neural tube. We demonstrate that a functional trkB receptor is expressed by motor neuron progenitors in the ventral neural tube and that treatment of ventral
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Cave, Clinton, and Shanthini Sockanathan. "Transcription Factor Hand-offs “Enhance” Motor Neuron Differentiation." Neuron 92, no. 6 (2016): 1149–51. http://dx.doi.org/10.1016/j.neuron.2016.12.009.

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Rao, M. "Transmembrane Protein GDE2 Induces Motor Neuron Differentiation in Vivo." Science 309, no. 5744 (2005): 2212–15. http://dx.doi.org/10.1126/science.1117156.

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Dissertations / Theses on the topic "Motor Neuron differentiation"

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Toli, Diana Eleni. "Directed differentiation and purification of motor neurons from human induced pluripotent stem cells to model Amyotrophic Lateral Sclerosis." Thesis, Paris 5, 2013. http://www.theses.fr/2013PA05T044/document.

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La sclérose latérale amyotrophique (SLA) est une maladie neurodégénérative incurable de l’adulte qui affecte principalement les motoneurones. Les mécanismes conduisant à la mort des motoneurones restent mal connus, notamment du fait de l'hétérogénéité de la maladie et du manque d'accès aux neurones humains affectés. La technologie des cellules souches pluripotentes induites humaines (iPSc) est un outil prometteur pour la modélisation de la SLA, car elle offre la possibilité unique d'obtenir et d’étudier des motoneurones humains.Des clones d’iPSc de deux sujets témoins ont été générés et nous a
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Hallgren, Henrik. "Characterization of NeuN expression in the mouse neuronal NSC-34 cell line using RT-qPCR, immunological staining and siRNA-mediated gene suppression." Thesis, Uppsala universitet, Institutionen för kvinnors och barns hälsa, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-389757.

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Background: Acute spinal trauma is followed by a secondary injury that causes additional damage to the tissue. The mouse neuronal hybrid cell line NSC-34 is planned for studies regarding this process, wherefore the cell line needed to be established in the laboratory and a proof-of-concept study needed to be performed. A suitable target gene for this study was Neuronal Nucleus (NeuN), a neuronal marker expressed in nearly all neuronal cells although not yet studied in NSC-34. Aim: The aim of this project was to characterize the expression of NeuN in differentiated and undifferentiated NSC-34 c
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Dai, Xizi. "Fiber Scaffolds of Poly (glycerol-dodecanedioate) and its Derivative via Electrospinning for Neural Tissue Engineering." FIU Digital Commons, 2015. http://digitalcommons.fiu.edu/etd/1852.

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Peripheral nerves have demonstrated the ability to bridge gaps of up to 6 mm. Peripheral Nerve System injury sites beyond this range need autograft or allograft surgery. Central Nerve System cells do not allow spontaneous regeneration due to the intrinsic environmental inhibition. Although stem cell therapy seems to be a promising approach towards nerve repair, it is essential to use the distinct three-dimensional architecture of a cell scaffold with proper biomolecule embedding in order to ensure that the local environment can be controlled well enough for growth and survival. Many approaches
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Martinou, Jean-Claude. "Purification de motoneurones embryonnaires : caracterisation de facteurs de survie et de differenciation in vitro." Toulouse 3, 1988. http://www.theses.fr/1988TOU30140.

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Des motoneurons d'embryons de rat (14 jours) ont ete marques de maniere retrograde, apres injection dans les pattes d'un compose liposoluble fluorescents, puis enrichies sur gradient de densite ou purifies a homogeneite avec un tri de cellules. Les motoneurones purifies sont maintenus en survie a long terme sur des monocouches d'astrocytes. Le substratum joue un role important dans leur differenciation, ainsi cultives, les motoneurones lyses presentent une activite choline acetylcholinesterase. Deux facteurs de differenciation qui stimulent l'activite choline-acetylstransferase, sous effet sur
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O'Brien, Laura. "Mitochondrial biogenesis and electrical properties of hPSC-derived motor neurons." VCU Scholars Compass, 2015. http://scholarscompass.vcu.edu/etd/3804.

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Human pluripotent stem cells (hPSCs), including human embryonic stem cells (hESCs) and induced pluripotent stem cells (iPSCs) hold great promise in the fields of drug development and regenerative medicine. If iPSCs reprogrammed from patient cells replicate what is seen in vivo they may be used as a model of disease. A process that is disrupted in many neurodegenerative diseases is mitochondrial biogenesis. One of these diseases is amyotrophic lateral sclerosis (ALS), which is characterized by loss of motor neurons in the brain and spinal cord. Differentiation of hPSCs into motor neurons offers
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Lee, Hyojin. "Directed differentiation and functional characterization of embryonic stem cell-derived motoneurons /." Access full-text from WCMC:, 2007. http://proquest.umi.com/pqdweb?did=1296098331&sid=4&Fmt=2&clientId=8424&RQT=309&VName=PQD.

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Sadegh, Cameron. "Directed differentiation of mouse embryonic stem cells into neocortical output neurons." Thesis, Harvard University, 2013. http://dissertations.umi.com/gsas.harvard:11064.

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During development of the neocortex, many diverse projection neuron subtypes are generated under regulation of cell-extrinsic and cell-intrinsic controls. One broad projection neuron class, corticofugal projection neurons (CFuPN), is the primary output neuron population of the neocortex. CFuPN axons innervate sub-cortical targets including thalamus, striatum, brainstem, and spinal cord.
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Podrygajlo, Grzegorz. "Differentiation of human teratocarcinoma cell line into motor neurons: investigation of cellular phenotype in vitro and in transplantation studies." Hannover Bibliothek der Tierärztlichen Hochschule Hannover, 2009. http://d-nb.info/1000125572/34.

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Podrygajlo, Grzegorz [Verfasser]. "Differentiation of human teratocarcinoma cell line into motor neurons : investigation of cellular phenotype in vitro and in transplantation studies / Grzegorz Podrygajlo." Hannover : Bibliothek der Tierärztlichen Hochschule Hannover, 2009. http://d-nb.info/1000125572/34.

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Chen, J. A. "Cyclin Dx, a novel cell-type specific cyclin identified from an enhanced functional screen, regulates the differentiation of motor neurons in Xenopus." Thesis, University of Cambridge, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.597520.

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The formation of mesoderm is an important development process of vertebrate embryos, which can be broken down into several steps; mesoderm induction, patterning, morphogenesis and differentiation. Although mesoderm formation in <i>Xenopus</i> has been intensively studied, much remains to be learned about the molecular events responsible for each of these steps. Furthermore, the interplay between mesoderm induction, patterning and morphogenesis remains obscure. Here, I describe an enhanced functional screen in <i>Xenopus</i> designed for large-scale identification of genes controlling mesoderm
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Books on the topic "Motor Neuron differentiation"

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Shaibani, Aziz. Proximal Arm Weakness. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199898152.003.0012.

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Proximal arm muscles include supra and infra spinatii, pectoralis major and minor, teres major and minor, rhomboids, serratus anterior, deltoids, biceps, and triceps. The main function of these muscles is to lift the arms. The first sign of proximal weakness is difficulty in raising the arms above a horizontal level. Shoulder conditions like supraspinatus tendonitis are often confused as proximal weakness. In myopathies, usually proximal arm weakness is associated with proximal leg weakness. Motor neuron diseases like ALS and SMA and neuropathies like CIDP may present with symmetrical proximal
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Shaibani, Aziz. Proximal Arm Weakness. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0012.

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Proximal arm muscles include supra and infra spinatii, pectoralis major and minor, teres major and minor, rhomboids, serratus anterior, deltoids, biceps, and triceps. The main function of these muscles is to abduct the arms. The first sign of proximal weakness is difficulty raising arms above the horizontal level. Shoulder conditions like supraspinatus tendonitis are often confused as proximal weakness. In myopathies, usually proximal arm weakness is associated with proximal leg weakness. Motor neuron diseases (MNDs) like amyotrophic lateral sclerosis (ALS) and spinal muscular atrophy (SMA) an
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Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 72-Year-Old Female with Facial Weakness and Droopy Eyelids. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0030.

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Fatigable weakness is the hallmark of myasthenia gravis (MG). It may present with false localizing signs leading to an itinal incorrect diagnosis of a brainstem stroke. MRI scanning of the brain with specific sequences can rule out the diagnosis of stroke. Differential diagnosis of MG may also include also motor neuron disease. Electromyography is very helpful in confirming the diagnosis of motor neuron disease. The two major diseases of the neuromuscular junction are MG and Lambert-Eaton syndrome (LEMS). A table presents the differing characteristics of each. LEMS can be associated with malig
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Banerjee, Ashis, and Clara Oliver. Neurological emergencies. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198786870.003.0011.

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A number of neurological conditions can present to the emergency department in a variety of presentations. This chapter summarizes the common neurological problems that may be examined in the Intermediate FRCEM short-answer question (SAQ) paper. This chapter includes the pathophysiology and management of an unconscious patient which may commonly appear in the SAQ paper. In addition, it also includes sections of epilepsy, headaches and strokes, and their subclassification and diagnosis. Many individuals find the differentiation of the cause of motor weakness complicated. This chapter summarizes
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G, Holstege, Bandler Richard, and Saper C. B, eds. The emotional motor system. Elsevier, 1996.

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(Editor), T. Kumazawa, L. Kruger (Editor), and K. Mizumura (Editor), eds. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Elsevier, 1996.

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Book chapters on the topic "Motor Neuron differentiation"

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Okamoto, Hitoshi, Hiroshi Segawa, and Shin-ichi Higashijima. "Toward genetic dissection of motor neuron differentiation." In Aquatic Genomics. Springer Japan, 2003. http://dx.doi.org/10.1007/978-4-431-65938-9_13.

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Rossoll, Wilfried, and Gary J. Bassell. "Spinal Muscular Atrophy and a Model for Survival of Motor Neuron Protein Function in Axonal Ribonucleoprotein Complexes." In Results and Problems in Cell Differentiation. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/400_2009_4.

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Miranda, Helen Cristina, and Albert R. La Spada. "Motor Neuron Differentiation from Pluripotent Stem Cells: Development of the Technique, Synopsis of Protocols, and Summary of Current Applications." In Working with Stem Cells. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-30582-0_11.

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Pfaff, S. L., T. Yamada, T. Edlund, and T. M. Jessell. "Induction and Differentiation of Motor Neurons." In Neural Cell Specification. Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1929-4_9.

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Bridgman, Paul C. "Myosin Motor Proteins in the Cell Biology of Axons and Other Neuronal Compartments." In Results and Problems in Cell Differentiation. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/400_2009_10.

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Cai, Jun, and Mengsheng Qiu. "Role of Nkx Homeodomain Factors in the Specification and Differentiation of Motor Neurons and Oligodendrocytes." In Transcription Factors in the Nervous System. Wiley-VCH Verlag GmbH & Co. KGaA, 2006. http://dx.doi.org/10.1002/3527608036.ch9.

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Li, Xue-Jun, Dali Yang, and Su-Chun Zhang. "Motor Neuron and Dopamine Neuron Differentiation." In Human Stem Cell Manual. Elsevier, 2007. http://dx.doi.org/10.1016/b978-012370465-8/50020-x.

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Levitan, Irwin B., and Leonard K. Kaczmarek. "Neuronal Growth and Trophic Factors." In The Neuron. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199773893.003.0015.

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Neural development requires the participation of growth factors that regulate neuronal determination, proliferation, migration, and differentiation. Molecular genetic approaches using Drosophila, as well as other creatures whose genetics is well understood, have provided insights into the mechanisms of action of some of these developmental factors. Other factors are soluble and are secreted by nearby cells or other neurons. These include neurotrophins such as NGF and BDNF, cytokines such as CNTF, as well as GDNF and steroid hormones. Current research aims to identify key growth factors required for producing different types of neurons, and different patterns of transcription factor activated by different combinations of these factors. This knowledge may eventually allow medical therapies to convert a stem cell into a sympathetic neuron, a motor neuron, or any one of the thousands of other types of neurons that make up a mature nervous system.
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Swetenburg, R. L., S. L. Stice, and L. Karumbaiah. "Molecular and Extracellular Cues in Motor Neuron Specification and Differentiation." In Molecular and Cellular Therapies for Motor Neuron Diseases. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-12-802257-3.00001-8.

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Atkinson, Martin E. "Development of the central nervous system." In Anatomy for Dental Students. Oxford University Press, 2013. http://dx.doi.org/10.1093/oso/9780199234462.003.0027.

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The early development of the nervous system, the process of neurulation, has already been outlined in Chapter 8 and illustrated in Figure 8.4. To briefly recap, an area of dorsal ectoderm is induced by the underlying notochord to form the neural plate during the third week of development. The lateral edges of the neural plate rise to form the neural folds which eventually fold over and unite in the midline by the end of the fourth week to produce the neural tube. A distinct cell population on the crest of the neural folds, the neural crest, migrates from the forming neural tube to form various structures, including components of the peripheral nervous system. The closed neural tube consists of a large diameter anterior portion that will become the brain and a longer cylindrical posterior section, the future spinal cord. Initially, the neural plate is a single cell layer, but concentric layers of cells can be recognized by the time the neural tube has closed. An inner layer of ependymal cells surrounds the central spinal canal. Neuroblasts, the precursors of neurons, make up the bulk of the neural tube called the mantle layer; this will become the grey matter of the spinal cord. Neuroblasts do not extend processes until they have completed their differentiation. When the cells in a particular location are fully differentiated, the neuronal processes emerging from the neuroblasts form an outer marginal layer which ultimately becomes the white matter of the spinal cord. Figure 19.1B shows that the neural tube changes shape due to proliferation of cells in the mantle layer. This figure also indicates two midline structures in the roof and floor of the tube, known as the roof plate and floor plate. They are important in the determination of the types of neurons that develop from the mantle layer. The floor plate is induced by the expression of a protein product of a gene called sonic hedgehog (SHH) produced by the underlying notochord; the floor plate then expresses the same gene itself. Neuroblasts nearest to the floor plate receive a high dose of SHH protein and respond by differentiating into motor neurons; as seen in Figure 19.1B, these cells group together to form bilateral ventrolateral basal plates.
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Conference papers on the topic "Motor Neuron differentiation"

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Demers, Christopher J., Greg Cox, Scott D. Collins, and Rosemary L. Smith. "Directing the spatial patterning of motor neuron differentiation in engineered microenvironments." In 2016 38th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2016. http://dx.doi.org/10.1109/embc.2016.7590743.

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