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Relevant bibliographies by topics / Motor neurons ; Muscle receptors ; Neural transmission

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Journal articles

Academic literature on the topic 'Motor neurons ; Muscle receptors ; Neural transmission'

Author: Grafiati

Published: 4 June 2021

Last updated: 11 February 2022

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Journal articles on the topic "Motor neurons ; Muscle receptors ; Neural transmission"

1

Majeed, Zana R., Esraa Abdeljaber, Robin Soveland, et al. "Modulatory Action by the Serotonergic System: Behavior and Neurophysiology inDrosophila melanogaster." Neural Plasticity 2016 (2016): 1–23. http://dx.doi.org/10.1155/2016/7291438.

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Serotonin modulates various physiological processes and behaviors. This study investigates the role of 5-HT in locomotion and feeding behaviors as well as in modulation of sensory-motor circuits. The 5-HT biosynthesis was dysregulated by feedingDrosophilalarvae 5-HT, a 5-HT precursor, or an inhibitor of tryptophan hydroxylase during early stages of development. The effects of feeding fluoxetine, a selective serotonin reuptake inhibitor, during early second instars were also examined. 5-HT receptor subtypes were manipulated using RNA interference mediated knockdown and 5-HT receptor insertional

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Pflüger, Hans-Joachim, and Carsten Duch. "Dynamic Neural Control of Insect Muscle Metabolism Related to Motor Behavior." Physiology 26, no. 4 (2011): 293–303. http://dx.doi.org/10.1152/physiol.00002.2011.

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Skeletal muscle innervation differs between vertebrates and insects. Insect muscle fibers exhibit graded electrical potentials and are innervated by excitatory, inhibitory, and also neuromodulatory motoneurons. The latter form a unique class of unpaired neurons with bilaterally symmetrical axons that release octopamine to alter the efficacy of synaptic transmission and regulate muscle energy metabolism by activating glycolysis. Octopaminergic neurons that innervate muscles with a high energy demand, for example, flight muscles that move the wings of a locust up and down, are active during rest

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3

Fung, Candice, Petra Unterweger, Laura J. Parry, Joel C. Bornstein, and Jaime P. P. Foong. "VPAC1 receptors regulate intestinal secretion and muscle contractility by activating cholinergic neurons in guinea pig jejunum." American Journal of Physiology-Gastrointestinal and Liver Physiology 306, no. 9 (2014): G748—G758. http://dx.doi.org/10.1152/ajpgi.00416.2013.

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In the gastrointestinal tract, vasoactive intestinal peptide (VIP) is found exclusively within neurons. VIP regulates intestinal motility via neurally mediated and direct actions on smooth muscle and secretion by a direct mucosal action, and via actions on submucosal neurons. VIP acts via VPAC1 and VPAC2 receptors; however, the subtype involved in its neural actions is unclear. The neural roles of VIP and VPAC1 receptors (VPAC1R) were investigated in intestinal motility and secretion in guinea pig jejunum. Expression of VIP receptors across the jejunal layers was examined using RT-PCR. Submuco

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4

Smith, Terence Keith, and Sang Don Koh. "A model of the enteric neural circuitry underlying the generation of rhythmic motor patterns in the colon: the role of serotonin." American Journal of Physiology-Gastrointestinal and Liver Physiology 312, no. 1 (2017): G1—G14. http://dx.doi.org/10.1152/ajpgi.00337.2016.

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We discuss the role of multiple cell types involved in rhythmic motor patterns in the large intestine that include tonic inhibition of the muscle layers interrupted by rhythmic colonic migrating motor complexes (CMMCs) and secretomotor activity. We propose a model that assumes these motor patterns are dependent on myenteric descending 5-hydroxytryptamine (5-HT, serotonin) interneurons. Asynchronous firing in 5-HT neurons excite inhibitory motor neurons (IMNs) to generate tonic inhibition occurring between CMMCs. IMNs release mainly nitric oxide (NO) to inhibit the muscle, intrinsic primary aff

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5

Kim, Jin Wook, Su Jin Kim, and Khae Hawn Kim. "Past, Present, and Future in the Study of Neural Control of the Lower Urinary Tract." International Neurourology Journal 24, no. 3 (2020): 191–99. http://dx.doi.org/10.5213/inj.2040318.159.

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The neurological coordination of the lower urinary tract can be analyzed from the perspective of motor neurons or sensory neurons. First, sensory nerves with receptors in the bladder and urethra transmits stimuli to the cerebral cortex through the periaqueductal gray (PAG) of the midbrain. Upon the recognition of stimuli, the cerebrum carries out decision-making in response. Motor neurons are divided into upper motor neurons (UMNs) and lower motor neurons (LMNs) and UMNs coordinate storage and urination in the brainstem for synergic voiding. In contrast, LMNs, which originate in the spinal cor

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6

Fox, Lyle E., and Philip E. Lloyd. "Glutamate is a Fast Excitatory Transmitter at Some Buccal Neuromuscular Synapses in Aplysia." Journal of Neurophysiology 82, no. 3 (1999): 1477–88. http://dx.doi.org/10.1152/jn.1999.82.3.1477.

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Studies of the modulation of synaptic transmission in buccal muscle of Aplysiawere limited because the conventional fast transmitter used by a number of large buccal motor neurons was unknown. Most of the identified buccal motor neurons are cholinergic because they synthesize acetylcholine (ACh) and their excitatory junction potentials (EJPs) are blocked by the cholinergic antagonist hexamethonium. However, three large identified motor neurons (B3, B6, and B38) do not synthesize ACh and their EJPs are not inhibited by hexamethonium. To identify the fast excitatory transmitter used by these non

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7

Yavuz, Utku Ş., Francesco Negro, Robin Diedrichs, and Dario Farina. "Reciprocal inhibition between motor neurons of the tibialis anterior and triceps surae in humans." Journal of Neurophysiology 119, no. 5 (2018): 1699–706. http://dx.doi.org/10.1152/jn.00424.2017.

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Motor neurons innervating antagonist muscles receive reciprocal inhibitory afferent inputs to facilitate the joint movement in the two directions. The present study investigates the mutual transmission of reciprocal inhibitory afferent inputs between the tibialis anterior (TA) and triceps surae (soleus and medial gastrocnemius) motor units. We assessed this mutual mechanism in large populations of motor units for building a statistical distribution of the inhibition amplitudes during standardized input to the motor neuron pools to minimize the effect of modulatory pathways. Single motor unit a

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8

Wang, Zhong-Min, Anna Carolina Zaia Rodrigues, María Laura Messi та Osvaldo Delbono. "Aging Blunts Sympathetic Neuron Regulation of Motoneurons Synaptic Vesicle Release Mediated by β1- and α2B-Adrenergic Receptors in Geriatric Mice". Journals of Gerontology: Series A 75, № 8 (2020): 1473–80. http://dx.doi.org/10.1093/gerona/glaa022.

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Abstract This study was designed to determine whether and how the sympathetic nervous system (SNS) regulates motoneuron axon function and neuromuscular transmission in young (3–4-month) and geriatric (31-month) mice. Our approach included sciatic-peroneal nerve immunolabeling coregistration, and electrophysiological recordings in a novel mouse ex-vivo preparation, the sympathetic-peroneal nerve-lumbricalis muscle (SPNL). Here, the interaction between the motoneuron and SNS at the neuromuscular junction (NMJ) and muscle innervation reflect the complexity of the living mouse. Our data show that

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9

Perez-Medina, Alberto L., and James J. Galligan. "Optogenetic analysis of neuromuscular transmission in the colon of ChAT-ChR2-YFP BAC transgenic mice." American Journal of Physiology-Gastrointestinal and Liver Physiology 317, no. 5 (2019): G569—G579. http://dx.doi.org/10.1152/ajpgi.00089.2019.

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Propulsion of luminal content along the gut requires coordinated contractions and relaxations of gastrointestinal smooth muscles controlled by the enteric nervous system. Activation of excitatory motor neurons (EMNs) causes muscle contractions, whereas inhibitory motor neuron (IMN) activation causes muscle relaxation. EMNs release acetylcholine (ACh), which acts at muscarinic receptors on smooth muscle cells and adjacent interstitial cells of Cajal, causing excitatory junction potentials (EJPs). IMNs release ATP (or another purine) and nitric oxide to cause inhibitory junction potentials (IJPs

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Carpenter, David O. "Neural mechanisms of emesis." Canadian Journal of Physiology and Pharmacology 68, no. 2 (1990): 230–36. http://dx.doi.org/10.1139/y90-036.

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Emesis is a reflex, developed to different degrees in different species, that allows an animal to rid itself of ingested toxins or poisons. The reflex can be elicited either by direct neuronal connections from visceral afferent fibers, especially those from the gastrointestinal tract, or from humoral factors. Emesis from humoral factors depends on the integrity of the area postrema; neurons in the area postrema have excitatory receptors for emetic agents. Emesis from gastrointestinal afferents does not depend on the area postrema, but probably the reflex is triggered by projections to some par

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