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Dissertations / Theses on the topic 'Motor neurons'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

PLETTO, Daniela Rita. "CHARACTERIZATION OF MOLECULAR ISOFORMS AND ROLE OF THE SURVIVAL MOTOR NEURON (SMN) IN MOTOR NEURONS DISEASES." Doctoral thesis, Università degli Studi di Palermo, 2014. http://hdl.handle.net/10447/91238.

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La sclerosi Laterale Amiotrofica (SLA) e l'Atrofia Muscolare Spinale (SMA) sono malattie neurodegenerative caratterizzate dalla perdita progressiva dei motoneuroni. La SMA è generalmente causata da delezione in omozigosi o mutazione del gene SMN, che codifica per una proteina ubiquitaria e multifunzionale, altamente espressa nel midollo spinale. La SLA è una malattia che può essere familiare o sporadica.Il 20% dei casi familiari è causato da una mutazione dominante nel gene SOD1. Inoltre ci sono altri geni coinvolti in questa malattia, tra cui FUS e TDP43. Lo scopo principale della tesi è

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2

Christou, Yiota Apostolou. "Generation of motor neurons from embryonic stem cells : application in studies of the motor neuron disease mechanism." Thesis, University of Sheffield, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.505426.

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Embryonic stem cells are pluripotent cells with the potential to differentiate into any cell type in the presence of appropriate stimulatory factors and environmental cues. Their broad developmental potential has led to the proposal that in the future, the use of human embryonic stem cells or their differentiated progeny may be beneficial in regenerative medicine. In particular, a current goal in the field of clinical neurology is to use stem cells in cell-based therapies for motor neuron disease (MND) or amyotrophic lateral ~clerosis. MND is a progressive neurodegenerative disease that specif

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3

Shaw, Ivan Ting-kun. "Cell death in motor neurons, two complementary models." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape11/PQDD_0028/NQ50259.pdf.

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4

Shaw, Ivan Ting-kun 1966. "Cell death in motor neurons : two complementary models." Thesis, McGill University, 1998. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=35486.

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Target-dependent cell death is an important embryogenic mechanism for regulating and sculpting the developing motor system. Efficient characterization of apoptosis has been more difficult in the nervous system than in other systems due to the use of several different primary culture systems as well as with heterogeneity of neuronal cell populations. We have developed a simple in vitro model of apoptosis with the motor neuron hybrid NSC34, a cell line which expresses much of the motor neuron phenotype (Cashman et al. 1992). Serum-deprived NSC 34 cells in bulk culture undergo cell death, likely

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5

Stephens, Benjamin. "Pathology of spinal interneurons in motor neuron disease." Thesis, Imperial College London, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.251759.

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6

Taylor, David M. 1977 Nov 23. "Understanding the regulation of molecular chaperones in motor neurons." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111857.

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Cells are constantly challenged by acute and chronic stresses that must be counteracted by upregulation of protective pathways. The premise of this thesis is that motor neurons have an impaired ability to trigger these protective mechanisms, which may contribute to their preferential vulnerability in the neurodegenerative disease, amyotrophic lateral sclerosis (ALS). The first objective was to study the involvement of metallochaperones in motor neuronal stress response, including their potential for rescuing motor neurons from toxicity conferred by a mutant Cu/Zn-superoxide dismutase (SOD1G93A

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7

Deinhardt, Katrin. "The endocytic pathway of tetanus neurotoxin in motor neurons." Thesis, University College London (University of London), 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.428573.

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8

Murtha, Matthew J. III. "Transcriptional Programming of Spinal Motor Neurons from Stem Cells." The Ohio State University, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=osu1261416295.

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9

Wertz, Mary Helene. "Aberrant microRNA Expression in Spinal Muscular Atrophy Motor Neurons." Thesis, Harvard University, 2015. http://nrs.harvard.edu/urn-3:HUL.InstRepos:17464519.

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Spinal Muscular Atrophy (SMA) is a devastating autosomal-recessive pediatric neurodegenerative disease characterized by loss of spinal motor neurons. It is caused by mutation in the survival of motor neuron 1, SMN1, gene and leads to loss of function of the full-length SMN protein. SMN has a number of functions related to RNA processing in neurons, including RNA trafficking in neurites, and RNA splicing and snRNP biogenesis in the nucleus. While previous work has focused on the alternative splicing and expression of traditional mRNAs, our lab has focused on the contribution of another RNA spec

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10

Nakamizo, Tomoki. "Phosphodiesterase inhibitors are neuroprotective to cultured spinal motor neurons." Kyoto University, 2003. http://hdl.handle.net/2433/148692.

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11

Wang, Hsien-Yi Sabrina. "Motor neurons and motor patterns underlying phonotaxis during flight of the cricket, Teleogryllus oceanicus." Thesis, McGill University, 1988. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=63869.

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12

Gopinath, Sumana. "Finding new genes causing motor neuron diseases." Thesis, The University of Sydney, 2006. http://hdl.handle.net/2123/1624.

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Abstract Neurodegenerative disorders are a diverse group of disorders that affect specific subsets of neurons. Motor neuron diseases, neurodegenerative disorders of motor neurons, are seen commonly as sporadic cases and less frequently as familial disease forms. The familial forms show genetic and phenotypic heterogeneity. Clinically motor neuron diseases may be seen as rapidly progressive disorders like amyotrophic lateral sclerosis, ALS or slowly progressive disorders like hereditary motor neuropathies, HMN. The only proven causes for motor neuron diseases are gene mutations that lead to m

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13

Gopinath, Sumana. "Finding new genes causing motor neuron diseases." University of Sydney, 2006. http://hdl.handle.net/2123/1624.

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Doctor of Philosophy<br>Abstract Neurodegenerative disorders are a diverse group of disorders that affect specific subsets of neurons. Motor neuron diseases, neurodegenerative disorders of motor neurons, are seen commonly as sporadic cases and less frequently as familial disease forms. The familial forms show genetic and phenotypic heterogeneity. Clinically motor neuron diseases may be seen as rapidly progressive disorders like amyotrophic lateral sclerosis, ALS or slowly progressive disorders like hereditary motor neuropathies, HMN. The only proven causes for motor neuron diseases are gene m

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14

Li, Mei. "Expression of chondroitin sulfotransferases in relation to cranial motor neuron movements in the embryonic hindbrain." Click to view the E-thesis via HKUTO, 2010. http://sunzi.lib.hku.hk/hkuto/record/B44236736.

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15

Krieger, Patrik. "On the role of metabotropic glutamate receptors in motor control : analysis of synaptic, cellular and network properties /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4449-0/.

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16

Chan, Alexander Kam Shing. "The effect of acupuncture on alpha-motoneuron excitability thesis submission to Auckland University of Technology for the degree of Master in Health Science, December 2002." Full thesis. Abstract, 2002.

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17

Li, Mei, and 李玫. "Expression of chondroitin sulfotransferases in relation to cranial motor neuron movements in the embryonic hindbrain." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B44236736.

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18

Ming, Yu. "Regulation of neurotrophic signaling molecules in motor neurons, primary sensory neurons and target tissues in senescence /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-591-3.

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19

Gill, Mark D. "Aminergic modulation of spontaneous and reflexly generated motor output of crayfish walking leg motor neurons." Thesis, University of Bristol, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.262842.

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20

O'Brien, Laura. "Mitochondrial biogenesis and electrical properties of hPSC-derived motor neurons." VCU Scholars Compass, 2015. http://scholarscompass.vcu.edu/etd/3804.

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Human pluripotent stem cells (hPSCs), including human embryonic stem cells (hESCs) and induced pluripotent stem cells (iPSCs) hold great promise in the fields of drug development and regenerative medicine. If iPSCs reprogrammed from patient cells replicate what is seen in vivo they may be used as a model of disease. A process that is disrupted in many neurodegenerative diseases is mitochondrial biogenesis. One of these diseases is amyotrophic lateral sclerosis (ALS), which is characterized by loss of motor neurons in the brain and spinal cord. Differentiation of hPSCs into motor neurons offers

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21

Cherry, Cortnie Lauren. "Mechanisms of Depolarization Induced Dendritic Growth of Drosophila Motor Neurons." Diss., The University of Arizona, 2006. http://hdl.handle.net/10150/195475.

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MECHANISMS OF DEPOLARIZATION INDUCED DENDRITIC GROWTH OF DROSOPHILA MOTOR NEURONS Cortnie Lauren Cherry The University of Arizona, 2006 Director: Richard B. Levine The study of the cellular mechanisms underlying dendritic growth contributes to our understanding of nervous system development, function and disease. Electrical activity is a fundamental property of neurons, and this property is utilized to influence the mechanisms involved in dendrite formation and maturation. Here we employ the Drosophila transgenic system to quantify dendritic growth of identified motor neurons using bo

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22

Dacre, Joshua Rupert Heaton. "Thalamic control of motor behaviour." Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/29530.

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The primary motor cortex (M1) is a key brain area for the generation and control of motor behaviour. Output from M1 can be driven in part by long-range inputs from a collection of thalamic nuclei termed the motor thalamus (MTh), but how MTh input shapes activity in M1 and forelimb motor behaviour remains largely unresolved. To address this issue, we first defined the 3D anatomical coordinates of mouse forelimb motor thalamus (MThFL) by employing conventional retrograde and virus-based tracing methods targeted to the forelimb region of M1 (M1FL). These complimentary approaches defined MThFL as

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23

Jacob, Sanjeev John. "Segmental origins and axon pathfinding of branchial motor and visceral motor neurons of the facial nerve." Thesis, King's College London (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.393791.

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24

Barbagallo, Belinda. "Activity Regulates Neuronal Connectivity and Function in the C. elegans Motor Circuit: A Dissertation." eScholarship@UMMS, 2014. https://escholarship.umassmed.edu/gsbs_diss/728.

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Activity plays diverse roles in shaping neuronal development and function. These roles range from aiding in synaptic refinement to triggering cell death during traumatic brain injury. Though the importance of activity-dependent mechanisms is widely recognized, the genetic underpinnings of these processes have not been fully described. In this thesis, I use the motor circuit of Caenorhabditis elegans as a model system to explore the functional and morphological consequences of modulating neuronal activity. First, I used a gain-of-function ionotropic receptor to hyperactivate motor neurons and a

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25

Barbagallo, Belinda. "Activity Regulates Neuronal Connectivity and Function in the C. elegans Motor Circuit: A Dissertation." eScholarship@UMMS, 2007. http://escholarship.umassmed.edu/gsbs_diss/728.

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Activity plays diverse roles in shaping neuronal development and function. These roles range from aiding in synaptic refinement to triggering cell death during traumatic brain injury. Though the importance of activity-dependent mechanisms is widely recognized, the genetic underpinnings of these processes have not been fully described. In this thesis, I use the motor circuit of Caenorhabditis elegans as a model system to explore the functional and morphological consequences of modulating neuronal activity. First, I used a gain-of-function ionotropic receptor to hyperactivate motor neurons and a

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26

Banks, Glen B. "The role of synapse formation on motoneuron survival during embryonic development /." [St. Lucia, Qld.], 2003. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17453.pdf.

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27

Li, Lai-fung. "Survival and regeneration of adult spinal motoneurons after root avulsion : a comparison of influence from different targets /." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B3984903X.

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28

Jackson, Mandy. "Screening of familial and sporadic amyotrophic lateral sclerosis patients for mutations in CuZn superoxide dismutase (SOD-1) and other candidate genes." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.363787.

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29

Simon, Horst Hubertus. "Development of the efferent system in the segmented chick brainstem." Thesis, King's College London (University of London), 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.307096.

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30

Uhler, Jennifer Pamela. "The development of dendritic arbors in Drosophila motorneurons." Thesis, University of Cambridge, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.621930.

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31

Musick, James R. "Mechanisms of spike-frequency adaptation in hypoglossal motoneurons /." Thesis, Connect to this title online; UW restricted, 1999. http://hdl.handle.net/1773/10550.

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32

Odden, Joanne Pamela. "Determined to be a Drosophila motor neuron : identification of subtype- and lineage-specific genetic components /." view abstract or download file of text, 2003. http://wwwlib.umi.com/cr/uoregon/fullcit?p3113020.

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Thesis (Ph. D.)--University of Oregon, 2003.<br>Typescript. Includes vita and abstract. Includes bibliographical references (leaves 96-105). Also available for download via the World Wide Web; free to University of Oregon users.

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33

Patani, Rickie. "Generating motor neuron subtype diversity from human pluripotent stem cells." Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610349.

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34

Spielmann, John Michael. "Quantification of motor neuron adaptation to sustained and intermittent stimulation." Diss., The University of Arizona, 1991. http://hdl.handle.net/10150/185411.

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In deeply anesthetized mammals, as typified by the adult cat, there is limited evidence that the firing-rate response of spinal motor neurons to sustained simulation usually features a progressive reduction in firing rate, termed late adaptation, that begins 1-2 s after the onset of sustained stimulation. The fullest description of late adaptation has been provided by Kernell & Monster (1982a,b) who evoked repetitive firing in spiral motor neurons of deeply anesthetized cats by the conventional procedure of an intracellular injection of a sustained depolarizing current. The main purpose of

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35

Jerregård, Helena. "Factors influencing nerve growth in situ and in vitro /." Linköping : Univ, 2001. http://www.bibl.liu.se/liupubl/disp/disp2001/med693s.pdf.

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36

Tokuno, Craig. "Neural control of standing posture." Doctoral thesis, Stockholm : Karolinska institutet, 2007. http://diss.kib.ki.se/2007/978-91-7357-396-2/.

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37

Wang, Pei-Yu, and n/a. "Novel survival factors with a gender specific twist for motor neurons." University of Otago. Department of Anatomy & Structural Biology, 2006. http://adt.otago.ac.nz./public/adt-NZDU20070504.143741.

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The survival of motor neurons is controlled by multiple factors, which regulate different aspects of their physiology. The identification of these factors is important because of their relevance for motor neuron disease. This thesis began with a search for novel growth factors that naturally keep these neurons alive. Members of the TGF-β superfamily, including Mullerian inhibiting substance (MIS) and bone morphogenetic protein 6 (BMP6), were identified as putative survival factors following a cDNA microarray analysis of a mouse model of motor neuron disease. MIS is a gonad-derived hormone wi

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38

Benn, Susanna Clare. "Neuroprotection by heat shock protein 27 in sensory and motor neurons." Thesis, University College London (University of London), 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.271424.

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39

Siahpoosh, Yasmin(Yasmin H. ). "Investigating mechanisms of biophysical diversity between phasic and tonic motor neurons." Thesis, Massachusetts Institute of Technology, 2020. https://hdl.handle.net/1721.1/130197.

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Thesis: M. Eng., Massachusetts Institute of Technology, Department of Electrical Engineering and Computer Science, May, 2020<br>Cataloged from student-submitted PDF of thesis.<br>Includes bibliographical references (pages 65-67).<br>Neurons exhibit striking diversity in core neuronal properties (intrinsic biophysical and synaptic properties), which are the building blocks of brain function and computation. Despite the central role of these properties in brain function, the underlying molecular and biophysical mechanisms which generate this diversity remain incompletely understood. In the Droso

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40

Sances, Samuel. "Influence of Microvascular Cells on Stem Cell Derived Spinal Motor Neurons." Thesis, Cedars-Sinai Medical Center, 2017. http://pqdtopen.proquest.com/#viewpdf?dispub=10231221.

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<p> Human ALS patient samples can be converted into motor neurons for the study of ALS pathogenesis <i>in vitro.</i> A major challenge, however is the lack of maturation of the stem cell-derived tissue, representing a major barrier to therapy development. The body of work herein details a series of studies to interrogate the relative maturation of induced pluripotent stem cell derived MNs and determine the effects of brain microvascular endothelial cells on their maturation. Finally, a novel microphysiological system for the modeling of ALS and the blood brain barrier is developed that include

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41

Vanderlaan, Gary. "Differential roles for hedgehog signaling in motor neuron development." Diss., Columbia, Mo. : University of Missouri-Columbia, 2006. http://hdl.handle.net/10355/4461.

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Thesis (Ph.D.)--University of Missouri-Columbia, 2006.<br>The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from title screen of research.pdf file (viewed on April 29, 2009) Vita. Includes bibliographical references.

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42

Gill, Nishi. "The morphology of C3, a motoneuron mediating the tentacle withdrawal reflex in the snail Helix aspersa /." Thesis, McGill University, 1996. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=27326.

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The morphology of C3, a motoneuron mediating the tentacle withdrawal reflex, was investigated in the snail Helix aspersa by intracellular injections of the tracers Neurobiotin and biocytin. Axonal projections were identified in the optic nerve, the olfactory nerve, the internal peritentacular nerve, the external peritentacular nerve, the cerebral-pedal connective and the cerebral commissure. A rare characteristic of the cell was the multibranching of axons in the neuropil and the exiting of this bundle of fibres into the cerebral-pedal connective. Dendritic arborizations were observed branchin

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43

Chaube, Sanjay. "Ubiquitin Expression in the Lumbar Spinal Cord Motoneurons of Postnatal Mice-- an Immunohistochemical Study." Thesis, University of North Texas, 1994. https://digital.library.unt.edu/ark:/67531/metadc332620/.

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Maturation of spinal motoneurons in rodents is characterized by a period of cell loss in the embryo, but researchers have claimed that some cell death occurs postnatally. This form of cell death is called apoptosis and involves active participation of the cell. Apoptotic cells have certain recognizable morphological and molecular features. I have used a monoclonal antibody against ubiquitin, (a putative marker of apoptotic cells), to do immunochemistry on mouse spinal cords at various postnatal ages till early adulthood. Staining is seen in large amotoneurons in the ventral horn. Staining is i

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44

Boatin, William. "Characterization of neuron models." Thesis, Available online, Georgia Institute of Technology, 2005, 2005. http://etd.gatech.edu/theses/available/etd-04182005-181732/.

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Thesis (M. S.)--Electrical and Computer Engineering, Georgia Institute of Technology, 2006.<br>Dr. Robert H. Lee, Committee Member ; Dr. Kurt Wiesenfeld, Committee Member ; Dr Robert J. Butera, Committee Member.

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45

Fellows, Matthew R. "Spatiotemporal tuning for position and velocity in primate primary motor cortex neurons /." View online version; access limited to Brown University users, 2005. http://wwwlib.umi.com/dissertations/fullcit/3174598.

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46

Garcia, Paul Anthony. "Modeling the Intersegmental Coordination of Heart Motor Neurons in the Medicinal Leech." Diss., Georgia Institute of Technology, 2004. http://hdl.handle.net/1853/5064.

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We constructed a model of the coordination of segmental heart motor neurons driving blood circulation in leeches. The heart motor neuron models were conductance-based; conductances of voltage-gated and synaptic currents were adjusted to match the firing pattern of heart motor neurons from the living system. Each motor neuron receives a specific pattern of inhibitory input from rhythmic premotor heart interneurons and translates this spatiotemporal pattern into the fictive heartbeat motor pattern. The temporal pattern of synaptic input to the model was derived from extracellularly recorded spi

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47

Roy, Jefferson Edward. "Signal processing by vestibular nuclei neurons : dissociating sensory, motor, and cognitive influences." Thesis, McGill University, 2002. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=84430.

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The vestibular sensory apparatus and associated vestibular nuclei are generally thought to encode angular head velocity during our daily activities. However, in addition to direct inputs from vestibular afferents, the vestibular nuclei receive substantial projections from cortical, cerebellar, and other brainstem structures. Given this diversity of inputs we asked: how are the responses of vestibular nuclei neurons to head velocity modified by these additional inputs during naturally occurring behaviours? Here we have focused on three specific classes of neurons in the vestibular nuclei

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48

Friel, Ruairi Donal. "The generation of a herpes simplex virus vector to target motor neurons." Thesis, University of Glasgow, 2001. http://theses.gla.ac.uk/3960/.

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Herpes simplex virus (HSV) is a neurovirulent virus that in the course of natural infection of man predominantly infects sensory neurons. The aim of this project was to develop a safe, nonvirulent HSV, capable of expressing exogenous genes which altered the binding characteristics of the virus so that tropism was directed predominantly to motor nerves. It was envisaged that these viruses could then act as prototypes for gene therapy vectors targeted to the treatment of motor nerve diseases. To achieve this, two mutant viruses were created, RFa and RFb. These contained deletions of the main HSV

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49

Rakowicz, Wojciech Piotr. "The regulation of death in retinal ganglion cells and spinal motor neurons." Thesis, University of Cambridge, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.621305.

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50

Banerjee, Soumya. "PERSISTENCE OF DROSOPHILA LARVAL MOTOR NEURONS INTO THE ADULT-IMPLICATIONS FOR BEHAVIOR." Miami University / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=miami1379680527.

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