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Relevant bibliographies by topics / Motorneurone

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Academic literature on the topic 'Motorneurone'

Author: Grafiati

Published: 16 November 2024

Last updated: 31 July 2025

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Journal articles on the topic "Motorneurone"

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Braak, H., M. Neumann, A. Ludolph, and K. Del Tredici. "Breitet sich die sporadisch auftretende amyotrophe Lateralsklerose über axonale Verbindungen aus?" Aktuelle Neurologie 44, no. 06 (2017): 409–14. http://dx.doi.org/10.1055/s-0043-111405.

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ZusammenfassungDer pathologische Prozess einer sporadisch auftretenden amyotrophen Lateralsklerose (sALS) ist mit dem Auftreten zytoplasmatischer Einschlusskörper eines normalerweise im Zellkern vorkommenden Proteins (TDP-43) verbunden und ergreift nur wenige Arten langaxoniger Projektionsneurone. Die Riesenpyramidenzellen von Betz im primären motorischen Neokortex und die α-Motorneurone im unteren Hirnstamm und Rückenmark sind früh ergriffene Zellformen. Im zentralen Nervensystem des Menschen sind diese beiden Zellarten durch axonale Projektionen monosynaptisch verbunden. Im Verlauf einer sAL

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GUILOFF, R. J. "Use of TRH Analogues in Motorneurone Disease." Annals of the New York Academy of Sciences 553, no. 1 Thyrotropin-R (1989): 399–421. http://dx.doi.org/10.1111/j.1749-6632.1989.tb46662.x.

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Pall, HardevS, AdrianC Williams, Rosemary Waring, and Elwyn Elias. "MOTORNEURONE DISEASE AS MANIFESTATION OF PESTICIDE TOXICITY." Lancet 330, no. 8560 (1987): 685. http://dx.doi.org/10.1016/s0140-6736(87)92468-8.

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Jacobs, K., M. G. Todman, M. J. Allen, J. A. Davies, and J. P. Bacon. "Synaptogenesis in the giant-fibre system of Drosophila: interaction of the giant fibre and its major motorneuronal target." Development 127, no. 23 (2000): 5203–12. http://dx.doi.org/10.1242/dev.127.23.5203.

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The tergotrochanteral (jump) motorneuron is a major synaptic target of the Giant Fibre in Drosophila. These two neurons are major components of the fly's Giant-Fibre escape system. Our previous work has described the development of the Giant Fibre in early metamorphosis and the involvement of the shaking-B locus in the formation of its electrical synapses. In the present study, we have investigated the development of the tergotrochanteral motorneuron and its electrical synapses by transforming Drosophila with a Gal4 fusion construct containing sequences largely upstream of, but including, the

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Mills, K. "Update on ALS: assessing the upper motorneurone component." Clinical Neurophysiology 119 (May 2008): S8. http://dx.doi.org/10.1016/s1388-2457(08)60035-8.

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Leigh, P. N. "DS1.1 Motorneurone degeneration: ALS and its clinical variants." Clinical Neurophysiology 117 (September 2006): 1. http://dx.doi.org/10.1016/j.clinph.2006.07.049.

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Tissenbaum, H. A., and D. J. Parry. "The effect of partial denervation of tibialis anterior (TA) muscle on the number and sizes of motorneurons in TA motornucleus of normal and dystrophic (C57BL dy2j/dy2j) mice." Canadian Journal of Physiology and Pharmacology 69, no. 11 (1991): 1769–73. http://dx.doi.org/10.1139/y91-261.

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The tibialis anterior (TA) muscle in one leg of normal (C57BL) and dystrophic (dy2j) mice was partially denervated by resection of a part of the lateral popliteal nerve. Two months later the muscle was injected with horseradish peroxidase to permit visualization of the motorneurons that survived. Partial denervation in both C57 and dy2j mice resulted in reduction of the number of motorneurons that supplied the muscle to approximately one-half the normal complement. The surviving motorneurons were found to be significantly larger (about 25%) than their contralateral counterparts. This condition

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Abbott, R. J., D. Holder, and S. Currie. "FALSE POSITIVE ANTI ACETYLCHOLINE RECEPTOR ANTIBODIES IN MOTORNEURONE DISEASE." Lancet 327, no. 8486 (1986): 906–7. http://dx.doi.org/10.1016/s0140-6736(86)91005-6.

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Ashizawa, T. "FALSE POSITIVE ANTI-ACETYLCHOLINE RECEPTOR ANTIBODIES IN MOTORNEURONE DISEASE." Lancet 327, no. 8492 (1986): 1272. http://dx.doi.org/10.1016/s0140-6736(86)91408-x.

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Spencer, PeterS, PeterB Nunn, Jacques Hugon, Albert Ludolph, and DwijendraN Roy. "MOTORNEURONE DISEASE ON GUAM: POSSIBLE ROLE OF A FOOD NEUROTOXIN." Lancet 327, no. 8487 (1986): 965. http://dx.doi.org/10.1016/s0140-6736(86)91059-7.

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Dissertations / Theses on the topic "Motorneurone"

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Esmaeili, Behrooz. "The C.elegans even-skipped homologue vab-7 controls DB motorneurone fate." Thesis, University of Cambridge, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.621089.

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Pinkernelle, Josephine [Verfasser], and Gerburg [Akademischer Betreuer] Keilhoff. "Etablierung eines in vitro-Modells zur Untersuchung der Regeneration spinaler Motorneurone und ihrer Axone nach Axotomie und Neurodegeneration / Josephine Pinkernelle. Betreuer: Gerburg Keilhoff." Magdeburg : Universitätsbibliothek, 2015. http://d-nb.info/1070276979/34.

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Draper, Christiana S. I. "ALS-induced Excitability Changes in Individual Motorneurons and the Spinal Motorneuron Network in SOD1-G93A Mice at Symptom Onset." Wright State University / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=wright1621064515386592.

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Hancox, Julian C. "Non-linear membrane properties of insect motorneurones." Thesis, University of St Andrews, 1991. http://hdl.handle.net/10023/15029.

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1. Electrical characteristics of the cell body of an identified motoneurone, the 'fast' coxal depressor motoneurone (Df), from the cockroach (Periplaneta americana) have been studied under current- and voltage-clamp. 2. In response to low magnitude, relatively long duration depolarising current pulses, Df could generate plateau potentials, regenerative events which often far outlived the duration of the applied depolarisation. 3. Plateau potentials constitute an inherent property of the neurone because they could be evoked in somata that had been surgically isolated from other parts of the neu

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Uhler, Jennifer Pamela. "The development of dendritic arbors in Drosophila motorneurons." Thesis, University of Cambridge, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.621930.

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Blair, Alex B. "Nervous System Remodeling in Drosophila: The fate of larval motorneurons." Miami University Honors Theses / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=muhonors1272045959.

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Mauss, Alex Stefan. "Development and patterning of motorneuron dendrites in the Drosophila embryo." Thesis, University of Cambridge, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.611196.

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Clerc, Zoé. "Identification des mécanismes moléculaires de neuroprotection modulés par l’activité dans deux maladies du motoneurone." Electronic Thesis or Diss., Université Paris Cité, 2024. http://www.theses.fr/2024UNIP5087.

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La sclérose latérale amyotrophique (SLA) et l'amyotrophie spinale (SMA) sont deux maladies du motoneurone (MN) se caractérisant par une dénervation musculaire progressive, pouvant être fatale par insuffisance respiratoire. Dans la SLA, les MN rapides (MNr) sont principalement affectés, tandis que dans la SMA, les MNr et les MN lents (MNl) dégénèrent. Le laboratoire a montré que soumettre des modèles murins adultes de la SLA (B6SJL-Tg(SOD1-G93A)1Gur/J) et de la SMA de type 3 (FVB/NRj-SmnDelta7/Delta7,huSMN2+/+) à un exercice de nage, activant les MNr, induisait une neuroprotection spécifique de

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Zee, Michele Chi-Wai. "Steroid hormones and cell death : analysis of motorneuron and muscle fates during insect metamorphosis /." view abstract or download file of text, 2004. http://wwwlib.umi.com/cr/uoregon/fullcit?p3136456.

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Thesis (Ph. D.)--University of Oregon, 2004.<br>Typescript. Includes vita and abstract. Includes bibliographical references (leaves 99-113). Also available for download via the World Wide Web; free to University of Oregon users.

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Sánchez-Alvarez, Leticia. "Planar Cell Polarity Genes prkl-1 and dsh-1 Polarize C. Elegans Motorneurons during Organogenesis." Thesis, Université d'Ottawa / University of Ottawa, 2012. http://hdl.handle.net/10393/23513.

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The correct polarity of a neuron underlies its ability to integrate precise circuitries in the nervous system. The goal of my thesis was to investigate the pathways that establish and maintain neuron polarity/orientation in vivo. To accomplish this, I used bipolar VC4/5 motor neurons, which innervate the C. elegans egg-laying musculature, as a model system. Vulval proximal VC4/5 neurons extend axons in the left-right (LR) orientation, around the vulva; whereas vulval distal VC1-3,6 neurons extend axons along the anterior-posterior (AP) axis. A previous study showed that vang-1, a core planar c

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Books on the topic "Motorneurone"

1

Larsen, DeLaine D. The genetic regulation of sex-specific motorneurons by the doublesex gene in Drosophila melanogaster and the genetic characterization of an interaction with the sex determination hierarchy. 1998.

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Book chapters on the topic "Motorneurone"

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Pinelli, P., C. Pasetti, L. Mazzini, F. Pisano, and A. Villani. "Motorneuron Sprouting and Spinal Plasticity in Amyotrophic Lateral Sclerosis: The “Window of Opportunity” for a Ganglioside Treatment." In Gangliosides and Neuronal Plasticity. Springer New York, 1986. http://dx.doi.org/10.1007/978-1-4757-5309-7_37.

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Pellizzari, R., and O. Rossetto. "Tetanus neurotoxin (Clostridium tetani)." In Guidebook to Protein Toxins and Their Use in Cell Biology. Oxford University PressOxford, 1997. http://dx.doi.org/10.1093/oso/9780198599555.003.0034.

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Abstract Tetanus neurotoxin (TeNT) is the cause of the spastic paralysis of tetanus (van Heyningen 1968; Montecucco and Schiavo 1995). It is produced as a single polypeptide chain of 150 kDa (1315 amino acid residues, sequence accession number to the Swiss Prot data bank: P04958ITETX_CLOTE). As shown in Fig. 1, it consists of three 50 kDa domains. Selective proteolysis generates within the bacterial culture the active di-chain toxin with a single interchain disulfide bond, whose reduction leads to total loss of neurotoxicity (Schiavo et al. 1990). The dichain toxin is the commercially availabl

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Rossetto, O., and R. Pellizzari. "Botulinum neurotoxins type A and E (Clostridium botulinum)." In Guidebook to Protein Toxins and Their Use in Cell Biology. Oxford University PressOxford, 1997. http://dx.doi.org/10.1093/oso/9780198599555.003.0036.

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Abstract Botulinum neurotoxins are structurally related protein toxins (BoNTs) produced by Clostridia in seven different serotypes: A, B, C, D, E, F, and G. BoNTs penetrate motorneurons at the neuromuscular junction and block acetylcholine release, thus causing the flaccid paralysis of botulism. They impair neurotransmission via the selective cleavage of proteins of the synaptic vesicles docking and fusion complex. BoNT/A and E are involved in human botulism and cleave SNAP-25 (synaptosomal-associated protein of 25 kDa), a protein bound to the cytosolic face of the plasma membrane. Botulinum n

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Conference papers on the topic "Motorneurone"

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Buono, Pietro-Luciano. "Heart Motorneuron Dynamics of Leeches." In EXPERIMENTAL CHAOS: 7th Experimental Chaos Conference. AIP, 2003. http://dx.doi.org/10.1063/1.1612221.

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Garavaglia, Lorenzo, Erika Molteni, Elena Beretta, Elena Vassena, Sandra Strazzer, and Simone Pittaccio. "Pilot study of the cortical correlates and clinical effects of passive ankle mobilisation in children with upper motorneuron lesions." In 2015 37th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2015. http://dx.doi.org/10.1109/embc.2015.7319909.

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