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Relevant bibliographies by topics / Mouse leukemia viruses / Dissertations / Theses

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Dissertations / Theses on the topic 'Mouse leukemia viruses'

Author: Grafiati

Published: 4 June 2021

Last updated: 31 July 2024

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1

Najmabadi, Hossein. "Characterization of the Self-Replicating Kirsten Murine Leukemia Viral DNA: Replication and Tetracycline Resistance." Thesis, University of North Texas, 1989. https://digital.library.unt.edu/ark:/67531/metadc798479/.

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This research project deals with the characterization of self-replicating Kirsten murine viral DNA. The replication of this viral DNA and tetracycline resistance conferred to bacteria by this viral DNA will be studied. The restriction endonuclease and Southern blot analysis revealed a fragment of pBR322 from the Hind III and Pst I site that is located in the 3' end of the MLV-K:E molecule. Single stranded sequencing of the two terminal ends of this fragment verified that the 3' end of MLV-K:E contains identical sequence homology to pBR322. The presence of this pBR322 fragment explains the unus

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2

BOI, STEFANO. "Differential effects of mouse APOBEC3 after incorporation into murine leukemia viruses." Doctoral thesis, Università degli Studi di Cagliari, 2014. http://hdl.handle.net/11584/266459.

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APOBEC3 proteins are cytidine deaminases that are potent inhibitors of retrovirus replication. These restriction factors induce G→A hypermutation as a result of deamination of cytidine in the single stranded transcripts generated by reverse transcription during replication. Mouse APOBEC3 (mA3) effectively inhibits the replication of endogenous retroelements whereas most exogenous murine leukemia viruses (MuLV) are largely resistant to the action(s) of mA3. However, several studies have reported significant inhibition of infectivity by virion-associated mA3 that did not appear to be the result

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3

Kabdulov, Timur O. "Mechanisms of retroviral replication." Morgantown, W. Va. : [West Virginia University Libraries], 2001. https://etd.wvu.edu/etd/controller.jsp?moduleName=documentdata&jsp%5FetdId=2256.

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4

Nardiello, Tricia Lynn. "Protease activity in lymphoid organs of BALB/C and C57BL/6 mice following murine leukemia virus /." Connect to online version, 2007. http://ada.mtholyoke.edu/setr/websrc/pdfs/www/2007/214.pdf.

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5

Halvas, Elias Konstantine. "Structural determinants of murine leukemia virus (MLV) reverse transcriptase (RT) important for fidelity and drug-resistance in vivo." Morgantown, W. Va. : [West Virginia University Libraries], 2000. http://etd.wvu.edu/templates/showETD.cfm?recnum=1518.

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Thesis (Ph. D.)--West Virginia University, 2000.<br>Title from document title page. Document formatted into pages; contains x, 231 p. : ill. (some col.). Vita. Includes abstract. Includes bibliographical references (p. 188-203).

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6

Socha, Amanda L. "Differential expression of an endogenous retrovirus in MAIDS susceptible (C57BL/6) versus resistant (BALB/C) mice /." Connect to online version, 2006. http://ada.mtholyoke.edu/setr/websrc/pdfs/www/2006/159.pdf.

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7

Krauchunas, Amber R. M. "CD8⁺ T-cell response potential, as determined by expression of the high affinity interleukin-2 receptor, in murine AIDS /." Connect to online version, 2006. http://ada.mtholyoke.edu/setr/websrc/pdfs/www/2006/149.pdf.

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8

Haworth, Richard Ian. "The role of macrophage scavenger receptors in host defence : studies in normal and genetically deficient murine models." Thesis, University of Oxford, 1997. http://ora.ox.ac.uk/objects/uuid:cc184f02-1393-407c-b241-a44ab3e0215c.

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9

Voronin, Yegor A. "Investigation of initiation of reverse transcription in retroviruses using vectors with two primer-binding sites." Morgantown, W. Va. : [West Virginia University Libraries], 2003. http://etd.wvu.edu/templates/showETD.cfm?recnum=3136.

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10

Berjanskii, Mark. "Structure and dynamics of the N-terminal J-domain of T antigens of murine polyomavirus." MU online access free, to others for fee Free online access, 2002. http://wwwlib.umi.com/cr/mo/preview?3052146.

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11

Svarovskaia, Evguenia S. "Structural determinants of murine leukemia virus reverse transcriptase that are important for template switching, fidelity, and drug-resistance." Morgantown, W. Va. : [West Virginia University Libraries], 2000. http://etd.wvu.edu/templates/showETD.cfm?recnum=1538.

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Thesis (Ph. D.)--West Virginia University, 2000.<br>Title from document title page. Document formatted into pages; contains xi, 185 p. : ill. (some col.). Vita. Includes abstract. Includes bibliographical references.

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12

Bagalb, Hussein Saeed. "Cellular and molecular biological studies of a retroviral induced lymphoma transmitted via breast milk in a mouse model." Connect to full text in OhioLINK ETD Center, 2008. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=mco1225294363.

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Thesis (M.S.)--University of Toledo, 2008.<br>"In partial fulfillment of the requirements for the degree of Master of Science in Biomedical Sciences." Title from title page of PDF document. Bibliography: pages 82-88, 111-116.

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13

Johansson, Ann-Sofie. "Establishment and characterization of a murine T-cell lymphoma/leukemia model." Doctoral thesis, Umeå universitet, Institutionen för strålningsvetenskaper, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-35195.

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Mouse models of human disease are valuable tools for studying pathogenesis and for evaluating novel therapies. T-cell lymphoma is a relatively rare disease in humans, affecting 100-150 persons yearly in Sweden. It exists in both aggressive and more indolent forms. We have established a mouse model for an aggressive T-cell lymphoma, the T-cell lymphoma/leukemia (TLL) mouse. In the present thesis, the TLL mouse model was characterized and used for experimental therapeutic and primary prevention studies. The TLL mouse was established unintentionally in our laboratory during work on VH-gene replac

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14

Rosales, Gerpe María Carla. "The Role of APOBEC3 in Controlling Retroviral Spread and Zoonoses." Thesis, Université d'Ottawa / University of Ottawa, 2014. http://hdl.handle.net/10393/31484.

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APOBEC3 (A3) proteins are a family of host-encoded cytidine deaminases that protect against retroviruses and other viral intruders. Retroviruses, unlike other viruses, are able to integrate their genomic proviral DNA within hours of entering host cells. A3 proteins hinder retroviral infectivity by editing retroviral replication intermediates, as well as by inhibiting retroviral replication and integration through deamination-independent methods. These proteins thus constitute the first line of immune defense against endogenous and exogenous retroviral pathogens. The overall goal of my Master's

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15

Villaudy, Julien. "Challenging Development of a Humanized Mouse Model for Evaluating the HTLV-1 Infection and Leukemogenic Process in vivo." Phd thesis, Ecole normale supérieure de lyon - ENS LYON, 2011. http://tel.archives-ouvertes.fr/tel-00682482.

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Human T-cell Leukemia Virus type 1 (HTLV-1) is the etiologic agent of the Adult T-cell Leukemia (ATL), an aggressive lymphoproliferation of activated CD4+ T cells. The lack of a reliable small animal model to reproduce in vivo the leukemogenic process associated with HTLV-1 infection has impaired the understanding of the early stages of this process as well as the discovery of effective therapeutic approaches. Recently, improvement in the models of humanized mouse models were achieved allowing the development of a human immune system in mice. Injection of human hematopoietic stem and progenito

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16

DELLA-VALLE, VERONIQUE. "1) etude moleculaire et physiopathologique des virus mcf isoles de proliferations malignes induites chez la souris par le virus auxiliaire de la leucemie de friend (f-mul v) : 2) les autoanticorps : un outil essentiel de la biologie cellulaire." Paris 6, 1988. http://www.theses.fr/1988PA066675.

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La leucemogenese induite chez la souris par le virus auxiliaire de la leucemie de friend est accompagnee de la formation "in vivo" de retrovirus mcf dans les proliferations malignes de lignees erythroides et lymphoides mais pas dans celles des lignees de type myelomonocytaire. L'identification des virus mcf isoles montre que leur genome viral comporte une region "eno" recombinee avec des sequences endogenes murins. In vivo ces virus f-mcf entrainent l'apparition d'erythroblastoses et de lymphoblastoses

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17

Pérès, Eléonore. "La souris humanisée : modèle d'étude in vivo du processus leucémogène induit par HTLV-1." Thesis, Lyon, 2017. http://www.theses.fr/2017LYSEN043/document.

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La leucémie T de l’adulte (ATL), caractérisée par une prolifération dérégulée de lymphocytesT CD4+ activés, se développe chez des individus infectés par le virus T-lymphotropehumain (HTLV-1). Une période asymptomatique de plusieurs décennies sépare l’infection del’apparition des symptômes cliniques de l’ATL, rendant complexe la compréhension des étapesinitiales de la leucémogenèse. Le modèle de la souris humanisée dans laquelle est reconstituéun système hémato-lymphoïde humain est pertinent pour étudier ces étapes, depuis l’infectionpar HTLV-1 jusqu’à l’apparition de la lymphoprolifération. Gr

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18

Tinaztepe, Sedef. "Biochemical and Genetic Investigation of Immature Murine Leukemia Virus Assembly." Thesis, 2017. https://doi.org/10.7916/D82V2TNG.

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Production of infectious retrovirus particles is a complex and poorly-understood process with multiple steps that are often linked to one another. Our aim in this study was to gain better understanding of the path the murine leukemia virus (MLV) structural protein Gag follows to assemble into immature capsid structures, the process of which is central to retroviral assembly and release. Extensive studies of human immunodeficiency virus type 1 (HIV-1) assembly have led to the development of a model proposing that the assembly of immature HIV-1 capsids proceeds sequentially through multiple inte

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19

Wang, Cheng. "Regulation Of Retroviral Silencing In Different Cell Types." Thesis, 2015. https://doi.org/10.7916/D8HH6JCP.

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The replication of Moloney Murine Leukemia Virus (MoMLV or MLV) is restricted in mouse embryonic stem (ES) and embryonic carcinoma (EC) cells, but not in differentiated cells. The restriction is mediated by the primer binding site (PBS) of proviral DNA of MLV. A restriction complex can bind to the PBS of MLV and block the transcription of viral genes. Two major components of the PBS-mediated silencing complex were identified in our lab, ZFP809 and Trim28. ZFP809 contains two conserved domains, a zinc finger domain responsible for DNA binding and a KRAB box recruiting Trim28, and hence other tr

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20

Segal, David Harry. "The immune response to murine retroviral infection." Phd thesis, 1996. http://hdl.handle.net/1885/143051.

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21

Martin, James Arthur. "Investigation of Ribonuclease HI handle region dynamics using Solution-state nuclear magnetic resonance spectroscopy, Molecular Dynamic simulations and X-ray crystallography." Thesis, 2020. https://doi.org/10.7916/d8-t0wr-yr67.

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Ribonuclease HI (RNase HI), a ubiquitous, non-sequence-specific endonuclease, cleaves the RNA strand in RNA/DNA hybrids. The enzyme has roles in replication, genome maintenance, and is the C-terminal domain of retroviral multi-domain reverse transcriptase (RT) proteins. Murine Leukemia Virus (MLV) and Human Immunodeficiency Virus (HIV) are two such retroviruses and their RNase HI (RNHI) domains are necessary for viral replication, making it an attractive drug target. RNase HI has a “handle region”, an extended loop with a large cluster of positive residues, that is critical for substrate recog

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22

Dias, Andreia Sofia Pires. "The effects of combinations of a green tea extract and an active ingredient thereof, with standard antiretroviral drugs on SC-1 cells infected with the LP-BM5 virus." Diss., 2008. http://upetd.up.ac.za/thesis/available/etd-01132009-092526.

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23

Alkhawaja, Mariam Jamal. "A study of Th17 axis cytokines in a mouse model of cutaneous autoimmunity and of the association of the Human T-cell Leukemia Virus Type I and mycosis fungoides." Thesis, 2014. http://hdl.handle.net/1993/23252.

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Psoriasiform diseases are a group of cutaneous disorders that are characterized by impaired keratinocyte maturation leading to epidermal hyperplasia and thickening of skin. This group of disorders includes psoriasis, seborrheic dermatitis (SD) and mycosis fungoides (MF). Psoriasis has been recently shown to be mediated by the pro-inflammatory T helper cell subset, namely Th17 cells, whereas the pathogenesis of SD and MF are still poorly understood. SD is characterized by inflamed skin that primarily manifests on areas populated with sebaceous glands. Interestingly, SD is very common amongst im

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