Relevant bibliographies by topics / Mouse model and breast cancer

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Mouse model and breast cancer'

Author: Grafiati
Published: 10 December 2022
Last updated: 31 July 2025

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Journal articles on the topic "Mouse model and breast cancer"

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Wu, Min, and Murray O. Robinson. "Human-in-Mouse breast cancer model." Cell Cycle 8, no. 15 (2009): 2317–18. http://dx.doi.org/10.4161/cc.8.15.9206.

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Dabydeen, Sarah A., та Priscilla A. Furth. "Genetically engineered ERα-positive breast cancer mouse models". Endocrine-Related Cancer 21, № 3 (2014): R195—R208. http://dx.doi.org/10.1530/erc-13-0512.

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The majority of human breast cancers are estrogen receptor-positive (ER+), but this has proven challenging to model in genetically engineered mice. This review summarizes information on 21 mouse models that develop ER+ mammary cancer. Where available, information on cancer pathology and gene expression profiles is referenced to assist in understanding which histological subtype of ER+ human cancer each model might represent.ESR1,CCDN1, prolactin,TGFα,AIB1,ESPL1, andWNT1overexpression,PIK3CAgain of function, as well as loss ofP53(Trp53) orSTAT1are associated with ER+ mammary cancer. Treatment w
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Zhao, Minjun, Haizhi Qiao, and Jinku Zhang. "Research Progress on Mouse Models of Breast Cancer Metastasis." Proceedings of Anticancer Research 8, no. 6 (2024): 129–37. http://dx.doi.org/10.26689/par.v8i6.8882.

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Breast cancer metastasis is a major cause of treatment failure and patient mortality. Mouse tumor models largely replicate the pathophysiological processes of human tumors. Establishing mouse models of breast cancer metastasis helps to elucidate metastatic mechanisms, and in vivo imaging techniques enable dynamic monitoring of tumor cell metastasis in animals. This paper summarizes the mechanisms of breast cancer metastasis, the development, and application of various mouse breast cancer distant metastasis models over the past decade, and evaluates the characteristics and efficacy of each mode
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Hennighausen, Lothar. "Mouse models for breast cancer." Oncogene 19, no. 8 (2000): 966–67. http://dx.doi.org/10.1038/sj.onc.1203346.

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Alfred, Jane. "A new mouse model of BRCA1 breast cancer?" Molecular Medicine Today 5, no. 7 (1999): 284. http://dx.doi.org/10.1016/s1357-4310(99)01520-8.

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Burney, Maryam, Lata Mathew, Anjali Gaikwad, Elizabeth K. Nugent, Anneliese O. Gonzalez, and Judith A. Smith. "Evaluation Fucoidan Extracts From Undaria pinnatifida and Fucus vesiculosus in Combination With Anticancer Drugs in Human Cancer Orthotopic Mouse Models." Integrative Cancer Therapies 17, no. 3 (2017): 755–61. http://dx.doi.org/10.1177/1534735417740631.

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Objective: To determine the activity of fucoidan from Undaria pinnatifida (UPF) and Fucus vesiculosus (FVF) when given in combination of chemotherapy drugs using selected human breast or ovarian cancer orthotopic mouse models. Methods: Mice were inoculated with 1 × 106 cells of TOV-112d, MCF-7, or ZR-75 subcutaneously or SKOV3-GFP-Luc intraperitoneally on day 0. MCF-7 and ZR-75 mice were administered with estradiol valerate 2 mg/kg in 0.2 mL castor oil subcutaneously two days prior to cell inoculation. Mice were randomized to one of six arms (N = 10/arm) paclitaxel, UPF/paclitaxel, FVF/paclita
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Ye, Genlan, Chuangkun Li, Xiaoliang Zhao, Feng Wen, Leyu Wang, and Xiaozhong Qiu. "A Humanized Cancer-Bone Metastasis Mouse Model Based on Silica Nanoparticles-Incorporated Human Demineralized Bone Matrix." Journal of Biomedical Nanotechnology 15, no. 12 (2019): 2363–75. http://dx.doi.org/10.1166/jbn.2019.2860.

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Breast cancer tends to spread to other organs and bone metastasis has the highest frequency in breast cancer metastasis, while its mechanisms are not clear and the current treatments are not very effective. To better study the mechanisms and facilitate drug screening for breast cancer bone metastasis, an in vivo mouse model needs to be constructed. However, the construction of the humanized mouse model for cancer bone metastasis which will mimick real interactions between cancer tissue and bone tissue in the human microenvironment remains a challenge. In this study, we constructed a human engi
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Hámori, Lilla, Gyöngyi Kudlik, Kornélia Szebényi, et al. "Establishment and Characterization of a Brca1−/−, p53−/− Mouse Mammary Tumor Cell Line." International Journal of Molecular Sciences 21, no. 4 (2020): 1185. http://dx.doi.org/10.3390/ijms21041185.

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Breast cancer is the most commonly occurring cancer in women and the second most common cancer overall. By the age of 80, the estimated risk for breast cancer for women with germline BRCA1 or BRCA2 mutations is around 80%. Genetically engineered BRCA1-deficient mouse models offer a unique opportunity to study the pathogenesis and therapy of triple negative breast cancer. Here we present a newly established Brca1−/−, p53−/− mouse mammary tumor cell line, designated as CST. CST shows prominent features of BRCA1-mutated triple-negative breast cancers including increased motility, high proliferati
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Piazza, Gary A., Khalda Fadlalla, Adam B. Keeton та ін. "Abstract P1-02-27: A novel 1st-in-class RAS/β-catenin inhibitor concurrently targets cancer cells and MDSC to reverse the immunosuppressive tumor microenvironment: antitumor activity in mouse models of breast and other cancers". Clinical Cancer Research 31, № 12_Supplement (2025): P1–02–27—P1–02–27. https://doi.org/10.1158/1557-3265.sabcs24-p1-02-27.

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Abstract While RAS mutations rarely occur in breast cancers, RAS signaling is well-known to be involved in breast cancer development resulting from constitutive activation of receptor tyrosine kinases. The Wnt/β-catenin pathway is also activated and associated with more aggressive forms of breast cancer. We found that the cyclic nucleotide degrading enzyme, phosphodiesterase 10A (PDE10), is overexpressed in breast cancer cell lines and tumors compared with normal human mammary epithelial cells (HMEC) or tissues, respectively. PDE10 selective inhibitors and gene silencing inhibited the growth o
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Cuellar-Vite, Leslie, Kristen L. Weber-Bonk, Fadi W. Abdul-Karim, Christine N. Booth, and Ruth A. Keri. "Focal Adhesion Kinase Provides a Collateral Vulnerability That Can Be Leveraged to Improve mTORC1 Inhibitor Efficacy." Cancers 14, no. 14 (2022): 3374. http://dx.doi.org/10.3390/cancers14143374.

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The PI3K/AKT/mTORC1 pathway is a major therapeutic target for many cancers, particularly breast cancer. Everolimus is an mTORC1 inhibitor used in metastatic estrogen receptor-positive (ER+) and epidermal growth factor receptor 2-negative (HER2-) breast cancer. However, mTORC1 inhibitors have limited efficacy in other breast cancer subtypes. We sought to discover collateral sensitivities to mTORC1 inhibition that could be exploited to improve therapeutic response. Using a mouse model of breast cancer that is intrinsically resistant to mTORC1 inhibition, we found that rapamycin alters the expres
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Dissertations / Theses on the topic "Mouse model and breast cancer"

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Herschkowitz, Jason I. Perou Charles M. "Breast cancer subtypes, mouse models, and microarrays." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2007. http://dc.lib.unc.edu/u?/etd,1728.

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Thesis (Ph. D.)--University of North Carolina at Chapel Hill, 2008.<br>Title from electronic title page (viewed Sep. 16, 2008). "... in partial fulfillment of the requirements for the degree of Doctor of Philosophy in the Curriculum of Genetics and Molecular Biology." Discipline: Genetics and Molecular Biology; Department/School: Medicine.
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Lesurf, Robert. "Molecular pathway analysis of mouse models for breast cancer." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=32499.

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Human breast cancer is an extremely heterogeneous disease, consisting of a number of different subtypes with varying levels of aggressiveness reflected by distinct, but largely undefined, molecular profiles. Here we have analyzed several novel mouse models for breast cancer in the context of the human subtypes, and have shown parallels between the mice and humans at numerous biologically relevant levels. In addition, we have developed a statistical framework to help elucidate the individual molecular comp
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Simpson, Peter Thomas. "Differential gene expression analysis in a transgenic mouse model of metastatic breast cancer." Thesis, University of Liverpool, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.343681.

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Alhazmi, Aiman. "Role of Nucleosome Remodeling Factor (NURF) in Tumorigenesis Using a Breast Cancer Mouse Model." VCU Scholars Compass, 2012. http://scholarscompass.vcu.edu/etd/379.

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Understanding the impact of epigenetic mechanisms on tumorigenesis is essential, as epigenetic alterations are associated with tumor initiation and progression. Because epigenetic changes are reversible, they are potential targets for cancer therapy. Nucleosome Remodeling Factor (NURF) is a chromatin-remodeling complex that regulates gene expression by changing nucleosome positioning along the DNA sequence. Previous studies have shown a role for NURF in embryonic development as well as regulating genes involved in tumor progression. In this work we investigated the impact of eliminating NU
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Robey, Ian, and Natasha Martin. "Bicarbonate and dichloroacetate: Evaluating pH altering therapies in a mouse model for metastatic breast cancer." BioMed Central, 2011. http://hdl.handle.net/10150/610344.

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BACKGROUND:The glycolytic nature of malignant tumors contributes to high levels of extracellular acidity in the tumor microenvironment. Tumor acidity is a driving force in invasion and metastases. Recently, it has been shown that buffering of extracellular acidity through systemic administration of oral bicarbonate can inhibit the spread of metastases in a mouse model for metastatic breast cancer. While these findings are compelling, recent assessments into the use of oral bicarbonate as a cancer intervention reveal limitations.METHODS:We posited that safety and efficacy of bicarbonate could b
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Pochampalli, Mamata Rani. "Characterization of Effects of Muc1 Expression on Epidermal Growth Factor Receptor Signaling in Breast Cancer." Diss., The University of Arizona, 2006. http://hdl.handle.net/10150/194355.

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EGF receptors are key regulators of cell survival and growth in normal and transformed tissues. Ligand binding results in formation of homo/hetero dimers of these receptors, followed by activation of the kinase activity and subsequent tyrosine phosphorylation of many downstream molecules. The activation of these receptors is not only mediated by the binding of their cognate ligands, but by transactivaton by other molecules as well. Recent studies have identified an oncogenic glycoprotein MUC1 as a binding partner for EGFR and that MUC1 expression can potentiate EGFR-dependent signal transducti
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Ke, Jia-Yu. "Bioactivity of Naringenin in Metabolic Dysregulation and Obesity-Associated Breast Cancer in a Mouse Model of Postmenopause." The Ohio State University, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=osu1437479457.

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Heilmann, Katharina [Verfasser], and Karin [Akademischer Betreuer] Müller-Decker. "Epigenetic characterization of the C3(1) SV40T mouse model of human breast cancer / Katharina Heilmann ; Betreuer: Karin Müller-Decker." Heidelberg : Universitätsbibliothek Heidelberg, 2017. http://d-nb.info/1178008134/34.

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Balderstone, Lucy Anne. "Use of fluorescent imaging to monitor drug responses in mouse models of tumourigenesis." Thesis, University of Edinburgh, 2014. http://hdl.handle.net/1842/17859.

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As our understanding of the complexities of cancer biology has increased, the ability to exploit unique features of tumour cells with molecularly targeted therapies has become a reality. However, despite unprecedented volumes of new molecules in clinical trials, the number of highly effective drugs approved by the regulatory authorities remains disappointingly low. Moreover, oncology drug development is plagued by high levels of attrition in late phase clinical development. Failure due to poor efficacy and toxicity issues are not believed to be a result of the development of molecules with ina
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Milliken, Erin L. "USE OF A TRANSGENIC MOUSE MODEL OF OVARIAN HYPERSTIUMLUATION TO IDENTIFY THERAPEUTIC TARGETS AND MECHANISMS IN HORMONE-INDUCED MAMMARY CANCER." Case Western Reserve University School of Graduate Studies / OhioLINK, 2005. http://rave.ohiolink.edu/etdc/view?acc_num=case1121273034.

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Books on the topic "Mouse model and breast cancer"

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O'Connell, Fiona Claire. Morphology and gene expression in the postnatal mouse mammary gland. University College Dublin, 1997.

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Rintala, Anne C. DNA repair in a radioresistant breast cancer model system. Laurentian University, 2000.

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Moelleken, Brent Roderick Wilfred. Tamoxifen - 5-fluorouracil synergy in human breast cancer cell lines: Correlating in vitro synergy with the current estrogen receptor model. s.n.], 1985.

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Freed-Pastor, William Allen. Gain-of-Function Effects of Mutant p53 Explored Using a Three-Dimensional Culture Model of Breast Cancer. [publisher not identified], 2012.

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Pearce, Andrews G. The generation and characterization of a radiation resistant model system to study radioresistance in human breast cancer cells. Laurentian University, Chemistry and Biochemistry Department, 2000.

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Kogan, Ilana. An in vivo model for PSA production by breast cancer cell-lines growing as xenografts in scid mice. National Library of Canada = Bibliothèque nationale du Canada, 1999.

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Cheung, Alison Min Yan. Characterization of the biological functions of breast cancer gene BRCA2 using conditionally-inactivated mouse models. 2003.

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Tamimi, Rulla, Susan Hankinson, and Pagona Lagiou. Breast Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0016.

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Most of the established reproductive risk factors for breast cancer, like age at menarche or parity, are not appropriate for public health intervention. Several lines of evidence, like the associations with birthweight and early exposure to radiation, support an important influence of early-life events on subsequent breast cancer risk. The best established modifiable risk factors for the disease include postmenopausal hormone use, moderate alcohol intake, and adult weight gain. More recently, we have come to appreciate that instead of a single disease, breast cancer is rather a heterogeneous g
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Zai, Clement. Generation of a mouse model for colorectal cancer. 2004.

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Yin, Hong. Human Mouse Mammary Tumor Virus-Like Elements and Their Relation to Breast Cancer. Uppsala Universitet, 1999.

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Book chapters on the topic "Mouse model and breast cancer"

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Couto, Joana Pinto, and Mohamed Bentires-Alj. "Mouse Models of Breast Cancer: Deceptions that Reveal the Truth." In Breast Cancer. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-48848-6_6.

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Sakamoto, Kazuhito, Jeffrey W. Schmidt, and Kay-Uwe Wagner. "Mouse Models of Breast Cancer." In Methods in Molecular Biology. Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2297-0_3.

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McAllister, Sandra S. "Systemic Instigation: A Mouse Model to Study Breast Cancer as a Systemic Disease." In Mouse as a Model Organism. Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0750-4_9.

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Medina, Daniel. "Mouse Models for Mammary Cancer." In Methods in Mammary Gland Biology and Breast Cancer Research. Springer US, 2000. http://dx.doi.org/10.1007/978-1-4615-4295-7_1.

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Zhou, Jiabao, Diane Lefley, and Penelope Ottewell. "Mouse Models of Breast Cancer Bone Metastasis." In Methods in Molecular Biology. Springer US, 2025. https://doi.org/10.1007/978-1-0716-4306-8_21.

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Chakrabarti, Rumela, and Yibin Kang. "Transplantable Mouse Tumor Models of Breast Cancer Metastasis." In Methods in Molecular Biology. Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2297-0_18.

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Imagawa, W., G. Bandyopadhyay, M. Spencer, J. Li, and S. Nandi. "Regulation of Mammary Epithelial Cell Proliferation: An In Vitro Mouse Mammary Epithelial Cell Model System." In Breast Cancer: Origins, Detection, and Treatment. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2309-9_3.

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Thiagarajan, Praveena S., and Ofer Reizes. "Mouse Models to Study Leptin in Breast Cancer Stem Cells." In Energy Balance and Cancer. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-16733-6_7.

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Quinn, Hazel M., Laura Battista, Valentina Scabia, and Cathrin Brisken. "Preclinical Mouse Intraductal Model (MIND) to Study Metastatic Dormancy in Estrogen Receptor-Positive Breast Cancer." In Cancer Cell Dormancy. Springer US, 2024. http://dx.doi.org/10.1007/978-1-0716-3882-8_7.

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Rottenberg, Sven, Marina Pajic, and Jos Jonkers. "Studying Drug Resistance Using Genetically Engineered Mouse Models for Breast Cancer." In Methods in Molecular Biology. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-416-6_3.

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Conference papers on the topic "Mouse model and breast cancer"

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Sawyer, Travis W., Photini F. Rice, Jennifer W. Koevary, Jennifer K. Barton, Denise C. Connolly, and Kathy Q. Cai. "In vivo multiphoton imaging of an ovarian cancer mouse model." In Diseases in the Breast and Reproductive System V, edited by Melissa C. Skala, Darren M. Roblyer, and Paul J. Campagnola. SPIE, 2019. http://dx.doi.org/10.1117/12.2505825.

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Bowman, Tyler, Kinan Alhallak, Tanny Chavez, et al. "Terahertz imaging of freshly excised breast cancer using mouse model." In 2017 42nd International Conference on Infrared, Millimeter, and Terahertz Waves (IRMMW-THz). IEEE, 2017. http://dx.doi.org/10.1109/irmmw-thz.2017.8067153.

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Rajhans, R., V. Cortez, SS Nair, RR Tekmal, R. Kumar, and RK Vadlamudi. "Novel mouse model for studying role of ER-nongenomic actions in breast cancer." In CTRC-AACR San Antonio Breast Cancer Symposium: 2008 Abstracts. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-601.

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DeAngel, R., O. Tolstykh, K. Nameer, et al. "Effects of obesity on anastrozole response in a mouse model of postmenopausal breast cancer." In CTRC-AACR San Antonio Breast Cancer Symposium: 2008 Abstracts. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-1146.

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Bhardwaj, Anjana, Matthew D. Embury, Raniv D. Rojo, Constance Albarracin, and Isabelle Bedrosian. "Abstract 20: Fluvastatin inhibits the development of breast cancer in SV40C3Tag mouse model of triple negative breast cancer." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-20.

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Jonkers, J. "ES2-2: Mouse Models of Basal-Like Breast Cancer." In Abstracts: Thirty-Fourth Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 6‐10, 2011; San Antonio, TX. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/0008-5472.sabcs11-es2-2.

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Shaw, Aubie K., Rachelle Johnson, Julie Sterling, Greg Mundy, and Hal Moses. "Abstract 1956: A novel mouse model of breast cancer metastasis to bone." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-1956.

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Krause, Silva, Heather Tobin, Heidi L. Lurvey, and Donald E. Ingber. "Abstract 3273: A robust transgenic mouse model to study male breast cancer." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-3273.

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Chen, Wenhong, John Olson, Christine N. McMahan, Mayur Choudhary, Hannah Caldas, and Linda J. Metheny-Barlow. "Abstract 473: Generation of a mouse model of breast cancer brain metastasis." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-473.

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Bhala, Rhea, Anjana Bhardwaj, Alexander Koh, Zhenlin Ju, Jing Wang, and Isabelle Bedrosian. "Long-term avasimibe treatment abolishes the breast cancer preventative efficacy of statin in a spontaneous mouse model of breast cancer." In Leading Edge of Cancer Research Symposium. The University of Texas at MD Anderson Cancer Center, 2022. http://dx.doi.org/10.52519/00044.

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Reports on the topic "Mouse model and breast cancer"

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Parrinello, Simona, and Judith Campisi. Aging, Breast Cancer, and the Mouse Model. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada417918.

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Donehower, Laurence A. The p53-Deficient Mouse as a Breast Cancer Model. Defense Technical Information Center, 1998. http://dx.doi.org/10.21236/ada368272.

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Weilbaecher, Katherine, and Ross Cagan. Assessing a Drosophila Metastasis Model in Mouse and Human Breast Cancer. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada488819.

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Jarvis, Gary A. Efficacy of Galectin-3C in Mouse Model of Metastatic Breast Cancer. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada383096.

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Weilbaecher, Katherine, and Ross Cagan. Assessing a Drosophila Metastasis Model in Mouse and Human Breast Cancer. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada625288.

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VAN Golen, Kenneth L. The RhoC Transgenic Mouse as a Realistic Model of Inflammatory Breast Cancer. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada398977.

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Boka, Valerie. A Mouse Model to Investigate the Role of DBC2 in Breast Cancer. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada452752.

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Boka, Valerie. A Mouse Model to Investigate the Role of DBC2 in Breast Cancer. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada434065.

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Van Golen, Kenneth L. The RhoC Transgenic Mouse as a Realistic Model of Inflammatory Breast Cancer. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada411463.

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Boka, Valerie. A Mouse Model to Investigate the Role of DBC2 in Breast Cancer. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada463478.

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