Log in

Relevant bibliographies by topics / MOZ / Journal articles

To see the other types of publications on this topic, follow the link: MOZ.

Journal articles on the topic 'MOZ'

Author: Grafiati

Published: 4 June 2021

Last updated: 26 July 2025

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'MOZ.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

Saric-Krsmanovic, Marija, Dragana Bozic, Ljiljana Radivojevic, Jelena Gajic-Umiljendic, Ljiljana Santric, and Sava Vrbnicanin. "Effects of plant growth promoting rhizobacteria (PGPR) and cover crops on seed germination and early establishment of field dodder (Cuscuta campestris Yunk.)." Pesticidi i fitomedicina 32, no. 2 (2017): 105–11. http://dx.doi.org/10.2298/pif1702105s.

Full text

Abstract:

Several bacterial cultures: Bacillus licheniformis (MO1), B. pumilus (MO2), and B. amyloliquefaciens (MO3), isolated from manure; B. megatherium ZP6 (MO4) isolated from maize rhizosphere; Azotobacter chroococcum Ps1 (MO5) and Pseudomonas fluorescens (MO6), were used to test the influence of plant growth promoting rhizobacteria (PGPR) on seed germination and germination rate of field dodder (Cuscuta campestris Yunk.). Also, to examine the effect of host seeds on germination and initial growth of seedlings of field dodder plants in the dark and under white light, the seeds of four host plants we

APA, Harvard, Vancouver, ISO, and other styles

2

Katsumoto, Takuo, and Issay Kitabayashi. "MOZ Is Critical for MOZ/MLL-Fusion-Induced HoxA9/Meis1 Expression and Leukemia Development,." Blood 118, no. 21 (2011): 3467. http://dx.doi.org/10.1182/blood.v118.21.3467.3467.

Full text

Abstract:

Abstract Abstract 3467 Monocytic leukemia Zinc finger (MOZ), a Myst-type histone acetyltransferase, is involved in chromosome translocations associated with FAB M4/M5 subtypes of acute myeloid leukemia (AML). We have shown that MOZ is critical for hematopoietic cell development and self-renewal of hematopoietic stem cells (HSCs). Recent reports suggest that hematopoietic and leukemic stem cells shares molecular basis for their self-renewal. To clarify the roles of endogenous MOZ in malignant hematopoiesis, we tested MOZ-deficient cells for leukemogenesis by leukemia-associated fusion genes suc

APA, Harvard, Vancouver, ISO, and other styles

3

Katsumoto, Takuo, Kazutsune Yamagata, Yoko Ogawara, Takuro Nakamura, and Issay Kitabayashi. "Essential Role of Endogenous MOZ in Aberrant HoxA9 and Meis1 Expressions Induced By MOZ Fusion Gene." Blood 124, no. 21 (2014): 2191. http://dx.doi.org/10.1182/blood.v124.21.2191.2191.

Full text

Abstract:

Abstract Monocytic leukemia Zinc finger protein (MOZ), a histone acetyltransferase, is involved in chromosome translocations associated with FAB M4/M5 types of acute myeloid leukemia (AML). In normal hematopoiesis, MOZ is essential for self-renewal of hematopoietic stem cells (HSCs) and for expression of HoxA9/Meis1 in hematopoietic stem/progenitor cells (HSPCs). Previously we found that endogenous MOZ is critical for MOZ-TIF2-induced AML. Although MOZ-/- cells expressing the MOZ-fusion serially generated colonies in vitro, they did not induce AML after transplantation into recipient mice. In

APA, Harvard, Vancouver, ISO, and other styles

4

Yin, Hong, Kerry L. Blanchard, and Jonathan Glass. "Altered Expression Profile of Global Genes in U937 Cells Stably Transfected with a Leukemia Fusion Gene: MOZ-TIF2." Blood 104, no. 11 (2004): 4284. http://dx.doi.org/10.1182/blood.v104.11.4284.4284.

Full text

Abstract:

Abstract Chromosome translocations involving the monocytic leukemia zinc finger (MOZ) gene occurs often in patients with acute myeloid leukemia (AML) of monocytoid phenotype (FAB types M4 or M5). The resulting MOZ-related fusions include MOZ-CBP, MOZ-P300, and MOZ-TIF2. MOZ has been identified as a histone acetyltransferase of unknown function. MOZ has been demonstrated to interact directly with RUNX1 to regulate RUNX1-mediated transcription and the MOZ-CBP fusion disrupted RUNX1-mediated transcription activity. The MOZ fusion partners CBP and p300 are transcription coactivators with histone a

APA, Harvard, Vancouver, ISO, and other styles

5

Chan, Edward M., Rebecca J. Chan, Elisha M. Comer та ін. "MOZ and MOZ-CBP Cooperate with NFκB To Induce Expression of NFκB-Dependent Genes." Blood 110, № 11 (2007): 887. http://dx.doi.org/10.1182/blood.v110.11.887.887.

Full text

Abstract:

Abstract Although monocytic zinc finger protein (MOZ/MYST3) maintains normal hematopoietic stem cells, its fusion to the coactivator CREB-binding protein (CBP/CREBBP) induces acute myeloid leukemia (AML). Since leukemic stem cells in AML often exhibit excessive signal-dependent activity of the transcription factor NFκB, we hypothesized that cooperation between NFκB and MOZ-CBP represents an alternative mechanism for enhancing NFκB transcriptional activity. In reporter assays, MOZ and CBP separately induce transcription from the NFκB-dependent interleukin-8 promoter; however, these two proteins

APA, Harvard, Vancouver, ISO, and other styles

6

Yin, Hong, Jonathan Glass, and Kerry L. Blanchard. "The Interaction of MOZ-TIF2 with Histone Chaperon Proteins CAF-1A and Asf1b Alters MOZ Regulated Gene Expression." Blood 108, no. 11 (2006): 2208. http://dx.doi.org/10.1182/blood.v108.11.2208.2208.

Full text

Abstract:

Abstract We have identified a MOZ-TIF2 (MT2) fusion gene containing the N-terminal portion of MOZ and the C-terminal portion of TIF2 from a patient with acute leukemia with a chromosome 8 translocation. We report here that MOZ portion of MOZ-TIF2 associates with chromatin assembly factors, CAF1 (chromatin assembly factor 1) and ASF1 (anti-silencing factor 1) in mammalian cells. Both proteins not only bring histones to newly synthesized DNA to create chromatin structure in the replication of chromosomes and DNA damage-repair processes but also contribute to regulation of global gene expression.

APA, Harvard, Vancouver, ISO, and other styles

7

Bozic, Dragana, Ljubinko Jovanovic, Vera Raicevic, Danijela Pavlovic, Marija Saric-Krsmanovic, and Sava Vrbnicanin. "The effect of plant growth promoting rhizobacteria on Datura stramonium L., Abutilon theophrasti Med., Onopordon acanthium L. and Verbascum thapsus L. seed germination." Pesticidi i fitomedicina 29, no. 3 (2014): 205–12. http://dx.doi.org/10.2298/pif1403205b.

Full text

Abstract:

The effects of several bacterial media [Bacillus licheniformis population 1 (MO1); B. licheniformis population 2 (MO2); B. subtilis (MO3); B. megatherium (MO4); humates (MO5)] on seed germination of Datura stramonium L., Abutilon theophrasti Med., Onopordon acanthium L. and Verbascum thapsus L. were tested. Seeds were germinated in Petri dishes containing solutions with different bacterial media. The highest germination percentage in all treatments was recorded for V. thapsus seeds (100.0%). Different treatments had diverse effects (stimulative or inhibitory) on seed germination of D. stramoni

APA, Harvard, Vancouver, ISO, and other styles

8

Paggetti, Jérôme, Romain Aucagne, Arnaud Jacquel, et al. "The Histone Acetyl-Transferase MOZ Cooperates with the Histone Methyl-Transferase MLL to Regulate HOX Gene Expression in Human Hematopoietic Stem Cells." Blood 112, no. 11 (2008): 2431. http://dx.doi.org/10.1182/blood.v112.11.2431.2431.

Full text

Abstract:

Abstract MOZ (MOnocytic leukaemia Zinc finger protein) (also called MYST3 or KAT6A) is a member of the MYST family of HATs which likely acetylate H4K16. The MLL (MixedLineageLeukemia) gene is a frequent target for recurrent chromosomal translocations found in AML and ALL. MLL (KMT2A) is a methyl-transferase targeting H3K4. It was shown that MOZ/CBP leukemia, as observed in MLL-rearranged leukemias, harbors abnormal levels of homeobox (HOX) genes expression. HOX transcription factors have a crucial function in hematopoiesis regulation. In addition, HOXA5, HOXA7, and HOXA9 are often considered t

APA, Harvard, Vancouver, ISO, and other styles

9

Ohta, Kumiko, Megumi Ohigashi, Ayako Naganawa, et al. "Histone acetyltransferase MOZ acts as a co-activator of Nrf2–MafK and induces tumour marker gene expression during hepatocarcinogenesis." Biochemical Journal 402, no. 3 (2007): 559–66. http://dx.doi.org/10.1042/bj20061194.

Full text

Abstract:

HATs (histone acetyltransferases) contribute to the regulation of gene expression, and loss or dysregulation of these activities may link to tumorigenesis. Here, we demonstrate that expression levels of HATs, p300 and CBP [CREB (cAMP-response-element-binding protein)-binding protein] were decreased during chemical hepatocarcinogenesis, whereas expression of MOZ (monocytic leukaemia zinc-finger protein; MYST3) – a member of the MYST [MOZ, Ybf2/Sas3, Sas2 and TIP60 (Tat-interacting protein, 60 kDa)] acetyltransferase family – was induced. Although the MOZ gene frequently is rearranged in leukaem

APA, Harvard, Vancouver, ISO, and other styles

10

Kindle, Karin B., Philip J. F. Troke, Hilary M. Collins, et al. "MOZ-TIF2 Inhibits Transcription by Nuclear Receptors and p53 by Impairment of CBP Function." Molecular and Cellular Biology 25, no. 3 (2005): 988–1002. http://dx.doi.org/10.1128/mcb.25.3.988-1002.2005.

Full text

Abstract:

ABSTRACT Chromosomal rearrangements associated with acute myeloid leukemia (AML) include fusions of the genes encoding the acetyltransferase MOZ or MORF with genes encoding the nuclear receptor coactivator TIF2, p300, or CBP. Here we show that MOZ-TIF2 acts as a dominant inhibitor of the transcriptional activities of CBP-dependent activators such as nuclear receptors and p53. The dominant negative property of MOZ-TIF2 requires the CBP-binding domain (activation domain 1 [AD1]), and coimmunoprecipitation and fluorescent resonance energy transfer experiments show that MOZ-TIF2 interacts with CBP

APA, Harvard, Vancouver, ISO, and other styles

11

Katsumoto, Takuo, Yukiko Aikawa, Takahiro Ochiya, and Issay Kitabayashi. "Roles of MOZ in Hematopoiesis." Blood 106, no. 11 (2005): 2269. http://dx.doi.org/10.1182/blood.v106.11.2269.2269.

Full text

Abstract:

Abstract The AML1-CBFβ transcription factor complex is the most frequent target of specific chromosome translocations in acute myeloid leukemia (AML). The monocytic leukemia zinc finger (MOZ) gene, which encodes a MYST-type histone acetyltransferase (HAT), is also involved in leukemia-associated translocations such as t(8;16), t(8;22) and inv(8), which are associated with acute myeloid leukemia with M4/5 subtypes. We previously found that MOZ functions as a potent coactivator for AML1. To investigate roles of MOZ in normal hematopoiesis, we generated MOZ-deficient mice using gene-targeting met

APA, Harvard, Vancouver, ISO, and other styles

12

Tam, Winnie F., Jennifer Rocnik, Sarah Wojiski, et al. "FLT3-ITD Cooperates with MOZ-TIF2 in Leukemogenesis in a Murine Bone Marrow Transplantation Model." Blood 108, no. 11 (2006): 1427. http://dx.doi.org/10.1182/blood.v108.11.1427.1427.

Full text

Abstract:

Abstract MOZ-TIF2 is expressed as a consequence of the chromosomal inversion inv(8)(p11q13) and is associated with AML FAB subtypes M4 and M5. Mice transplanted with MOZ-TIF2 succumb to monoclonal or oligoclonal leukemias with a long median disease latency, indicating that cooperating mutations are required for MOZ-TIF2 mediated leukemogenesis. The presence of a FLT3-ITD mutation in a patient with inv(8)(p11q13) has previously been reported, suggesting FLT3-ITD as a candidate cooperating mutation. Here we report that FLT3-ITD functionally cooperates with MOZ-TIF2 in co-transduction experiments

APA, Harvard, Vancouver, ISO, and other styles

13

Katsumoto, Takuo, and Issay Kitabayashi. "Roles of Histone Acetyltransferase MORF and MOZ in Hematopoiesis." Blood 114, no. 22 (2009): 2525. http://dx.doi.org/10.1182/blood.v114.22.2525.2525.

Full text

Abstract:

Abstract Abstract 2525 Poster Board II-502 MOZ (MOnocytic leukemia Zinc finger protein) and MORF (MOz Related Factor), Myst-type histone acetyltransferases, are involved in chromosome translocations associated with FAB-M4/5 subtypes of acute myeloid leukemia. We have reported that MOZ is essential for hematopoietic cell development and self-renewal of hematopoietic stem cells. To explore the possibility MORF also plays important roles in hematopoiesis, we generated Morf-deficient mice with homologous recombination methods. Morf−/− mice were smaller than their wildtype littermates and died with

APA, Harvard, Vancouver, ISO, and other styles

14

Perez-Campo, Flor M., Julian Borrow, Valerie Kouskoff, and Georges Lacaud. "The histone acetyl transferase activity of monocytic leukemia zinc finger is critical for the proliferation of hematopoietic precursors." Blood 113, no. 20 (2009): 4866–74. http://dx.doi.org/10.1182/blood-2008-04-152017.

Full text

Abstract:

The monocytic leukemia zinc finger (MOZ) gene encodes a large multidomain protein that contains, besides other domains, 2 coactivation domains for the transcription factor Runx1/acute myeloid leukemia 1 and a histone acetyl transferase (HAT) catalytic domain. Recent studies have demonstrated the critical requirement for the complete MOZ protein in hematopoietic stem cell development and maintenance. However, the specific function of the HAT activity of MOZ remains unknown, as it has been shown that MOZ HAT activity is not required either for its role as Runx1 coactivator or for the leukemic tr

APA, Harvard, Vancouver, ISO, and other styles

15

Troke, Philip J. F., Karin B. Kindle, Hilary M. Collins, and David M. Heery. "MOZ fusion proteins in acute myeloid leukaemia." Biochemical Society Symposia 73 (January 1, 2006): 23–39. http://dx.doi.org/10.1042/bss0730023.

Full text

Abstract:

MOZ (monocytic leukaemia zinc finger protein; also known as ZNF220 or MYST3) is a member of the MYST family of protein acetyltransferases. Chromosomal translocations involving the MOZ gene are associated with AML (acute myeloid leukaemia), suggesting that it has a role in haematopoiesis. Recurrent reciprocal translocations fuse the MOZ gene [or the gene encoding MORF (MOZ-related factor); also known as MYST4] to genes encoding the nuclear receptor co-activators CBP [CREB (cAMP response element-binding protein)-binding protein], p300 or the p160 protein TIF2 (transcription intermediary factor 2

APA, Harvard, Vancouver, ISO, and other styles

16

Sheikh, Bilal N., Natalie L. Downer, Belinda Phipson, et al. "MOZ and BMI1 play opposing roles during Hox gene activation in ES cells and in body segment identity specification in vivo." Proceedings of the National Academy of Sciences 112, no. 17 (2015): 5437–42. http://dx.doi.org/10.1073/pnas.1422872112.

Full text

Abstract:

Hox genes underlie the specification of body segment identity in the anterior–posterior axis. They are activated during gastrulation and undergo a dynamic shift from a transcriptionally repressed to an active chromatin state in a sequence that reflects their chromosomal location. Nevertheless, the precise role of chromatin modifying complexes during the initial activation phase remains unclear. In the current study, we examined the role of chromatin regulators during Hox gene activation. Using embryonic stem cell lines lacking the transcriptional activator MOZ and the polycomb-family repressor

APA, Harvard, Vancouver, ISO, and other styles

17

Ullah, Mukta, Nadine Pelletier, Lin Xiao, et al. "Molecular Architecture of Quartet MOZ/MORF Histone Acetyltransferase Complexes." Molecular and Cellular Biology 28, no. 22 (2008): 6828–43. http://dx.doi.org/10.1128/mcb.01297-08.

Full text

Abstract:

ABSTRACT The monocytic leukemia zinc finger protein MOZ and the related factor MORF form tetrameric complexes with ING5 (inhibitor of growth 5), EAF6 (Esa1-associated factor 6 ortholog), and the bromodomain-PHD finger protein BRPF1, -2, or -3. To gain new insights into the structure, function, and regulation of these complexes, we reconstituted them and performed various molecular analyses. We found that BRPF proteins bridge the association of MOZ and MORF with ING5 and EAF6. An N-terminal region of BRPF1 interacts with the acetyltransferases; the enhancer of polycomb (EPc) homology domain in

APA, Harvard, Vancouver, ISO, and other styles

18

SHATWELL, D., J. A. CLIFFORD, D. ECHAVARRÍA, G. IRUSTA, and D. LOPEZ. "Discoveries of Low-Sulfidation Epithermal Au-Ag Veins at Cerro Negro, Deseado Massif, Argentina." SEG Discovery, no. 85 (April 1, 2011): 1–23. http://dx.doi.org/10.5382/segnews.2011-85.fea.

Full text

Abstract:

ABSTRACT Exploration by Andean Resources Ltd. in the Cerro Negro district of southern Argentina between March 2005 and July 2010 delineated resources of 2.54 Moz gold and 23.5 Moz silver in three low-sulfidation epithermal vein deposits. Two of these, Eureka West and Bajo Negro, are new discoveries; the third deposit, Vein Zone, had been explored previously. Additional measured and indicated resources of 2.7 Moz Au have been estimated by current owners Goldcorp Inc. for three other discoveries at Cerro Negro: San Marcos, Mariana Norte, and Mariana Central. The total gold resource for the proje

APA, Harvard, Vancouver, ISO, and other styles

19

Susiana, Andi Niartiningsih, Muh Anshar Amran, and Rochmady. "SUITABILITY OF LOCATION FOR RESTOCKING CLAMS TRIDACNIDAE IN THE SPERMONDE ARCHIPELAGO." Jurnal Ilmu dan Teknologi Kelautan Tropis 9, no. 2 (2018): 475–90. http://dx.doi.org/10.29244/jitkt.v9i2.19284.

Full text

Abstract:

Efforts to protect giant clams from extinction one of them through conservation, which set a region as a restocking area. The research aimed to analyze the suitability environments with the density giant clams in the aquatic. The research conducted in September 2013-January 2014. The research site was determined by purposive sampling of middle inner zone = MIZ, middle outer zone = MOZ, and outer zone = OZ in the aquatic of Spermonde Archipelago. The research conducted was using observation method of biology, physics, and chemistry parameters of aquatic at mintakat reef flats, reef slope and re

APA, Harvard, Vancouver, ISO, and other styles

20

Voss, Anne K., Hannah K. Vanyai, Caitlin Collin, et al. "MOZ Regulates the Tbx1 Locus, and Moz Mutation Partially Phenocopies DiGeorge Syndrome." Developmental Cell 23, no. 3 (2012): 652–63. http://dx.doi.org/10.1016/j.devcel.2012.07.010.

Full text

APA, Harvard, Vancouver, ISO, and other styles

21

Aikawa, Yukiko, Takuo Katsumoto, Daniel G. Tenen, and Issay Kitabayashi. "PU.1-Mediated Upregulation of M-CSFR Is Critical for MOZ-Leukemia Stem Cell Potential." Blood 114, no. 22 (2009): 216. http://dx.doi.org/10.1182/blood.v114.22.216.216.

Full text

Abstract:

Abstract Abstract 216 Leukemias and other cancers possess self-renewing stem cells that help to maintain the cancer. The eradication of cancer stem cells is thought to be critical for successful anti-cancer therapy. However, there is little evidence for this. Using an acute myeloid leukemia (AML) model by introducing the leukemia-associated monocytic leukemia zinc finger (MOZ)-TIF2 fusion protein, we show here that AML can be cured by the ablation of leukemia stem cells. Chromosomal translocations that involve the MOZ gene are typically associated with the FAB-M4 or -M5 subtype of human AML an

APA, Harvard, Vancouver, ISO, and other styles

22

Sheikh, Bilal N., Stanley C. W. Lee, Farrah El-Saafin, et al. "MOZ regulates B-cell progenitors and, consequently, Moz haploinsufficiency dramatically retards MYC-induced lymphoma development." Blood 125, no. 12 (2015): 1910–21. http://dx.doi.org/10.1182/blood-2014-08-594655.

Full text

APA, Harvard, Vancouver, ISO, and other styles

23

Shima, Haruko, Emi Takamatsu-Ichihara, Mika Shino, et al. "Ring1A and Ring1B inhibit expression of Glis2 to maintain murine MOZ-TIF2 AML stem cells." Blood 131, no. 16 (2018): 1833–45. http://dx.doi.org/10.1182/blood-2017-05-787226.

Full text

APA, Harvard, Vancouver, ISO, and other styles

24

Aguiar, Ricardo C. T., Andrew Chase, Stephanie Coulthard, et al. "Abnormalities of Chromosome Band 8p11 in Leukemia: Two Clinical Syndromes Can Be Distinguished on the Basis of MOZ Involvement." Blood 90, no. 8 (1997): 3130–35. http://dx.doi.org/10.1182/blood.v90.8.3130.

Full text

Abstract:

Abstract Two distinct leukemia syndromes are associated with abnormalities of chromosome band 8p11. First, a myeloproliferative disorder with features characteristic of both chronic myeloid leukemia and non-Hodgkin's lymphoma and second, an acute myeloid leukemia (AML) with French-American-British (FAB) M4/5 morphology and prominent erythrophagocytosis. The two syndromes are exemplified by a t(8; 13)(p11; q12) and a t(8; 16)(p11; p13), respectively, but cytogenetic variants of both have been described. Recently, the t(8; 16) has been cloned and shown to fuse the MOZ gene at 8p11 to the CBP gen

APA, Harvard, Vancouver, ISO, and other styles

25

Shima, Haruko, Mika Shino, Kazutsune Yamagata, Yukiko Aikawa, Haruhiko Koseki, and Issay Kitabayashi. "Roles of Ring1A/B on Stem Cell Potential of MOZ and Other Acute Myeloid Leukemias,." Blood 118, no. 21 (2011): 3998. http://dx.doi.org/10.1182/blood.v118.21.3998.3998.

Full text

Abstract:

Abstract Abstract 3998 Leukemia and other cancers possess self-renewing stem cells that help maintain cancer. Chromosomal translocations are often involved in the development of human acute myeloid leukemia (AML). The monocytic leukemia zinc finger (MOZ) gene is one of the targets of such translocations. While MOZ is essential for the self-renewal of hematopoietic stem cells, the leukemia associated MOZ-fusion proteins enable the transformation of non–self-renewing myeloid progenitors into leukemia stem cells. Ring1A and Ring1B are catalytic subunits of the polycomb-group repressive complex 1

APA, Harvard, Vancouver, ISO, and other styles

26

Falk, Hendrik, Theresa Connor, Hong Yang, et al. "An Efficient High-Throughput Screening Method for MYST Family Acetyltransferases, a New Class of Epigenetic Drug Targets." Journal of Biomolecular Screening 16, no. 10 (2011): 1196–205. http://dx.doi.org/10.1177/1087057111421631.

Full text

Abstract:

Epigenetic aberrations are increasingly regarded as key factors in cancer progression. Recently, deregulation of histone acetyltransferases (HATs) has been linked to several types of cancer. Monocytic leukemia zinc finger protein (MOZ) is a member of the MYST family of HATs, which regulate gene expression in cell proliferation and differentiation. Deregulation of these processes through constitutively active MOZ fusion proteins gives rise to the formation of leukemic stem cells, rendering MOZ an excellent target for treating myeloid leukemia. The authors implemented a hit discovery campaign to

APA, Harvard, Vancouver, ISO, and other styles

27

Ayu Putri Aryani and R. Hiru Muhammad. "Proses Produksi Program Siaran “Get In The Moz“Di 90.8 FM OZ Radio Jakarta." Harmoni: Jurnal Ilmu Komunikasi dan Sosial 1, no. 4 (2023): 96–102. http://dx.doi.org/10.59581/harmoni-widyakarya.v1i4.1626.

Full text

Abstract:

Radio is still one of the mass communication media that still exists in society today. Even though many other media have emerged that provide broadcasts other than audio, radio is still in demand by many fans. This research focuses on the Production Process of the Get In The Moz Program on 90.8 FM OZ Radio Jakarta. The aim of this research is to determine the production process of the Get In The Moz broadcast program on 90.8 FM OZ Radio Jakarta starting from pre-production, production, post-production, supporting factors and inhibiting factors in the production process of the Get In The Moz br

APA, Harvard, Vancouver, ISO, and other styles

28

Su, Jingtian, Xuan Wang, Yuwei Bai, Moran Sun, Yongfang Yao, and Yongtao Duan. "The role of MOZ/KAT6A in hematological malignancies and advances in MOZ/KAT6A inhibitors." Pharmacological Research 174 (December 2021): 105930. http://dx.doi.org/10.1016/j.phrs.2021.105930.

Full text

APA, Harvard, Vancouver, ISO, and other styles

29

Cirera, Laia, Charfudin Sacoor, Martin Meremikwu, et al. "The economic costs of malaria in pregnancy: evidence from four sub-Saharan countries." Gates Open Research 7 (May 5, 2023): 47. http://dx.doi.org/10.12688/gatesopenres.14375.2.

Full text

Abstract:

Background Malaria in pregnancy is a major public health problem in sub-Saharan Africa (SSA), which imposes a significant economic burden. We provide evidence on the costs of malaria care in pregnancy to households and the health system in four high-burden countries in SSA. Methods Household and health system economic costs associated with malaria control in pregnancy were estimated in selected areas of the Democratic Republic of Congo (DRC), Madagascar (MDG), Mozambique (MOZ) and Nigeria (NGA). An exit survey was administered to 2,031 pregnant women when leaving the antenatal care (ANC) clini

APA, Harvard, Vancouver, ISO, and other styles

30

Cirera, Laia, Charfudin Sacoor, Martin Meremikwu, et al. "The economic costs of malaria in pregnancy: evidence from four sub-Saharan countries." Gates Open Research 7 (February 15, 2023): 47. http://dx.doi.org/10.12688/gatesopenres.14375.1.

Full text

Abstract:

Background Malaria in pregnancy is a major public health problem in sub-Saharan Africa (SSA), which imposes a significant economic burden. We provide evidence on the costs of malaria care in pregnancy to households and the health system in four high-burden countries in SSA. Methods Household and health system economic costs associated with malaria control in pregnancy were estimated in selected areas of the Democratic Republic of Congo (DRC), Madagascar (MDG), Mozambique (MOZ) and Nigeria (NGA). An exit survey was administered to 2,031 pregnant women when leaving the antenatal care (ANC) clini

APA, Harvard, Vancouver, ISO, and other styles

31

Carapeti, Melina, Ricardo C. T. Aguiar, John M. Goldman, and Nicholas C. P. Cross. "A Novel Fusion Between MOZ and the Nuclear Receptor Coactivator TIF2 in Acute Myeloid Leukemia." Blood 91, no. 9 (1998): 3127–33. http://dx.doi.org/10.1182/blood.v91.9.3127.

Full text

Abstract:

Abstract Chromosomal abnormalities of band 8p11 are associated with a distinct subtype of acute myeloid leukemia with French-American-British M4/5 morphology and prominent erythrophagocytosis by the blast cells. This subtype is usually associated with the t(8;16)(p11;p13), a translocation that has recently been shown to result in a fusion between the MOZ and CBP genes. We have cloned the inv(8)(p11q13), an abnormality associated with the same leukemia phenotype, and found a novel fusion between MOZ and the nuclear receptor transcriptional coactivatorTIF2/GRIP-1/NCoA-2. This gene has not previo

APA, Harvard, Vancouver, ISO, and other styles

32

Carapeti, Melina, Ricardo C. T. Aguiar, John M. Goldman, and Nicholas C. P. Cross. "A Novel Fusion Between MOZ and the Nuclear Receptor Coactivator TIF2 in Acute Myeloid Leukemia." Blood 91, no. 9 (1998): 3127–33. http://dx.doi.org/10.1182/blood.v91.9.3127.3127_3127_3133.

Full text

Abstract:

Chromosomal abnormalities of band 8p11 are associated with a distinct subtype of acute myeloid leukemia with French-American-British M4/5 morphology and prominent erythrophagocytosis by the blast cells. This subtype is usually associated with the t(8;16)(p11;p13), a translocation that has recently been shown to result in a fusion between the MOZ and CBP genes. We have cloned the inv(8)(p11q13), an abnormality associated with the same leukemia phenotype, and found a novel fusion between MOZ and the nuclear receptor transcriptional coactivatorTIF2/GRIP-1/NCoA-2. This gene has not previously been

APA, Harvard, Vancouver, ISO, and other styles

33

Kitabayashi, I. "Activation of AML1-mediated transcription by MOZ and inhibition by the MOZ-CBP fusion protein." EMBO Journal 20, no. 24 (2001): 7184–96. http://dx.doi.org/10.1093/emboj/20.24.7184.

Full text

APA, Harvard, Vancouver, ISO, and other styles

34

Chan, Edward M., Rebecca J. Chan, Elisha M. Comer та ін. "MOZ and MOZ-CBP cooperate with NF-κB to activate transcription from NF-κB–dependent promoters". Experimental Hematology 35, № 12 (2007): 1782–92. http://dx.doi.org/10.1016/j.exphem.2007.07.015.

Full text

APA, Harvard, Vancouver, ISO, and other styles

35

Schmidt, H. H. "MYST3/CREBBP (MOZ/CBP) andCREBBP/MYST3 (CBP/MOZ) transcripts in AML with t(8;16)(p11;p13)." Genes, Chromosomes and Cancer 42, no. 2 (2005): 207–8. http://dx.doi.org/10.1002/gcc.20112.

Full text

APA, Harvard, Vancouver, ISO, and other styles

36

Tavakoli, Samon, Stephanie A. Armstrong, Christina Feller, Sang Hun Hong, and Nathan T. Zwagerman. "Transpalpebral mini-orbitozygomatic approach for nonvascular skull base lesions: a single neurosurgeon’s experience." Neurosurgical Focus 56, no. 4 (2024): E11. http://dx.doi.org/10.3171/2024.1.focus23875.

Full text

Abstract:

OBJECTIVE The authors aim to describe the advantages, utility, and disadvantages of the transpalpebral mini-orbitozygomatic (MOZ) approach for tumors of the lateral and superior orbit, orbital apex, anterior clinoid, anterior cranial fossa, middle cranial fossa, and parasellar region. METHODS The surgical approach from skin incision to closure is described while highlighting key technical and anatomical considerations, and cadaveric dissection demonstrates the surgical steps and focuses on important anatomy. Intraoperative images were included to supplement the cadaveric dissection. A retrospe

APA, Harvard, Vancouver, ISO, and other styles

37

Liang, Jian, Leonard Prouty, B. Jill Williams, Mark A. Dayton, and Kerry L. Blanchard. "Acute Mixed Lineage Leukemia With an inv(8)(p11q13) Resulting in Fusion of the Genes for MOZ and TIF2." Blood 92, no. 6 (1998): 2118–22. http://dx.doi.org/10.1182/blood.v92.6.2118.

Full text

Abstract:

Abstract Chromosomal abnormalities in acute leukemia have led to the discovery of many genes involved in normal hematopoiesis and in malignant transformation. We have identified the fusion partners in an inv(8)(p11q13) from a patient with acute mixed lineage leukemia. We show by fluorescence in situ hybridization (FISH) analysis, Southern blotting, and reverse transcriptase-polymerase chain reaction (RT-PCR) that the genes for MOZ, monocytic leukemiazinc finger protein, and TIF2,transcriptional intermediary factor 2, are involved in the inv(8)(p11q13). We demonstrate that the inversion creates

APA, Harvard, Vancouver, ISO, and other styles

38

Liang, Jian, Leonard Prouty, B. Jill Williams, Mark A. Dayton, and Kerry L. Blanchard. "Acute Mixed Lineage Leukemia With an inv(8)(p11q13) Resulting in Fusion of the Genes for MOZ and TIF2." Blood 92, no. 6 (1998): 2118–22. http://dx.doi.org/10.1182/blood.v92.6.2118.418k09_2118_2122.

Full text

Abstract:

Chromosomal abnormalities in acute leukemia have led to the discovery of many genes involved in normal hematopoiesis and in malignant transformation. We have identified the fusion partners in an inv(8)(p11q13) from a patient with acute mixed lineage leukemia. We show by fluorescence in situ hybridization (FISH) analysis, Southern blotting, and reverse transcriptase-polymerase chain reaction (RT-PCR) that the genes for MOZ, monocytic leukemiazinc finger protein, and TIF2,transcriptional intermediary factor 2, are involved in the inv(8)(p11q13). We demonstrate that the inversion creates a fusion

APA, Harvard, Vancouver, ISO, and other styles

39

Mishima, Yuta, Satoru Miyagi, Atsunori Saraya, et al. "The Hbo1-Brd1/Brpf2 complex is responsible for global acetylation of H3K14 and required for fetal liver erythropoiesis." Blood 118, no. 9 (2011): 2443–53. http://dx.doi.org/10.1182/blood-2011-01-331892.

Full text

Abstract:

Abstract The histone acetyltransferases (HATs) of the MYST family include TIP60, HBO1, MOZ/MORF, and MOF and function in multisubunit protein complexes. Bromodomain-containing protein 1 (BRD1), also known as BRPF2, has been considered a subunit of the MOZ/MORF H3 HAT complex based on analogy with BRPF1 and BRPF3. However, its physiologic function remains obscure. Here we show that BRD1 forms a novel HAT complex with HBO1 and regulates erythropoiesis. Brd1-deficient embryos showed severe anemia because of impaired fetal liver erythropoiesis. Biochemical analyses revealed that BRD1 bridges HBO1

APA, Harvard, Vancouver, ISO, and other styles

40

Huntly, Brian J. P., Hirokazu Shigematzu, Kenji Deguchi, et al. "MOZ-TIF2, but Not BCR-ABL, Confers Properties of Leukemic Stem Cells to Committed Murine Hematopoietic Progenitors." Blood 104, no. 11 (2004): 62. http://dx.doi.org/10.1182/blood.v104.11.62.62.

Full text

Abstract:

Abstract The existence of leukemia stem cells has been demonstrated in acute myeloid and lymphoblastic leukemias (AML and ALL). The origins of these cells are unknown, but it has been suggested that they result from the transformation of adult hematopoietic stem cells (HSC). To challenge this hypothesis we tested the ability of representative leukemia oncogenes to transform committed myeloid progenitor cells that lack the capacity for self-renewal. Flow-sorted populations of common myeloid progenitors (CMP), and granulocyte-monocyte progenitors (GMP) were transduced with the fusion oncogenes M

APA, Harvard, Vancouver, ISO, and other styles

41

Maki, Kazuhiro, Fumihiko Nakamura, Tetsuya Yamagata, Issay Kitabayashi, and Kinuko Mitani. "Functional Regulation of TEL through MOZ-Induced Acetylation." Blood 110, no. 11 (2007): 2647. http://dx.doi.org/10.1182/blood.v110.11.2647.2647.

Full text

Abstract:

Abstract TEL, a member of ETS transcription factors, acts as a transcriptional repressor and a tumor suppressor. TEL is inactivated by genomic alterations such as deletion and chromosomal translocation in a variety of human leukemia. TEL also has important roles in the maintenance of hematopoietic stem cells and differentiation of erythroid progenitors. Regulation of its function through phosphorylation and sumoylation has been described in the past literatures. We here demonstrated that TEL is also regulated by acetylation, another mode of post-translational modification. First, immunoprecipi

APA, Harvard, Vancouver, ISO, and other styles

42

Imataki, Osamu, and Makiko Uemura. "Sea-Blue Histiocytosis of Bone Marrow in a Patient with t(8;22) Acute Myeloid Leukemia." Case Reports in Oncology 13, no. 2 (2020): 849–52. http://dx.doi.org/10.1159/000508495.

Full text

Abstract:

An 80-year-old Japanese male was treated with chemotherapy consisting of cyclophosphamide, doxorubicin, vincristine, and prednisolone, for non-Hodgkin lymphoma. Nine months after the chemotherapy, he was diagnosed with acute myeloid leukemia (AML) (M4) with translocation 8p11 and 22q13. The patient bone marrow indicated a remarkable degree of sea-blue histiocytosis. His disease was aggressive, and he died of the disease. Sea-blue histiocytes are macrophages harboring blue vacuoles and granular deposition, which results from the phagocytosis of dead cells and the subsequent deposition of phosph

APA, Harvard, Vancouver, ISO, and other styles

43

Deguchi, Kenji, Paul M. Ayton, Melina Carapeti, et al. "MOZ-TIF2-induced acute myeloid leukemia requires the MOZ nucleosome binding motif and TIF2-mediated recruitment of CBP." Cancer Cell 3, no. 3 (2003): 259–71. http://dx.doi.org/10.1016/s1535-6108(03)00051-5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

44

Michmerhuizen, Nicole L., Emily Heikamp, Masayuki Umeda, et al. "The Histone Acetyltransferase MOZ (KAT6A) Is a Molecular Dependency and Therapeutic Target in NUP98-Rearranged Acute Myeloid Leukemia." Blood 144, Supplement 1 (2024): 825. https://doi.org/10.1182/blood-2024-210529.

Full text

Abstract:

NUP98 fusion oncoproteins(FOs) are a hallmark of high-risk leukemia and are present in approximately 5% of pediatric and 3% of adult acute myeloid leukemia (AML). NUP98 fusion oncoproteins involve the N-terminal, intrinsically disordered region of NUP98 and the C-terminal portion of one of over 30 fusion partners. Approximately one-third of fusion partners have DNA-binding homeodomains, and many others have domains involved in gene regulation. Outcomes in children with NUP98-rearranged (NUP98-r) AML are poor, with relapse rates of nearly 70%. We and others recently demonstrated the importance

APA, Harvard, Vancouver, ISO, and other styles

45

Hermans, S. J., M. C. Chung, J. B. Baell, and M. W. Parker. "Developing MOZi, a chimeric MOF protein to aid structure-based drug design in MOZ." Acta Crystallographica Section A Foundations and Advances 79, a2 (2023): C341. http://dx.doi.org/10.1107/s2053273323092744.

Full text

APA, Harvard, Vancouver, ISO, and other styles

46

Rozman, Mar�a, and Dolors Colomer. "Reply to Schmidt ?MYST3/CREBBP (MOZ/CBP) andCREBBP/MYST3(CBP/MOZ) Transcripts in AML with t(8;16)(p11;p13)?" Genes, Chromosomes and Cancer 42, no. 2 (2005): 209. http://dx.doi.org/10.1002/gcc.20111.

Full text

APA, Harvard, Vancouver, ISO, and other styles

47

Peregrino, Miriane. "Moz Slam e as novas dinâmicas da poesia falada em Moçambique no século XXI." Via Atlântica 25, no. 2 (2025): 177–99. https://doi.org/10.11606/va.v25.n2.2025.215816.

Full text

Abstract:

From the United States to the world, poetry slam competitions establish rules for oral poetry presentations and streamline an international circuit that provides meetings between poets from different realities. This article takes the Mozambican competition Moz Slam to present and discusses the dynamics of production, circulation and reception of this format of spoken poetry that has been gaining acceptance, especially among young artists. The article highlights the presence of women in Moz Slam and Slam da Guilhermina during online slams in times of pandemic and is based on interviews carried

APA, Harvard, Vancouver, ISO, and other styles

48

Plensa, Esther, Josep-Maria Ribera, José-Tomás Navarro, and Fuensanta Millà. "Leucemia aguda mieloblástica con reordenamiento de MOZ/CBP." Medicina Clínica 122, no. 16 (2004): 639. http://dx.doi.org/10.1157/13061075.

Full text

APA, Harvard, Vancouver, ISO, and other styles

49

Plensa, Esther, Josep-Maria Ribera, José-Tomás Navarro, and Fuensanta Millà. "Leucemia aguda mieloblástica con reordenamiento de MOZ/CBP." Medicina Clínica 122, no. 16 (2004): 639. http://dx.doi.org/10.1016/s0025-7753(04)74338-9.

Full text

APA, Harvard, Vancouver, ISO, and other styles

50

Yin, Hong, Jonathan Glass, and Kerry L. Blanchard. "MOZ-TIF2 repression of nuclear receptor-mediated transcription requires multiple domains in MOZ and in the CID domain of TIF2." Molecular Cancer 6, no. 1 (2007): 51. http://dx.doi.org/10.1186/1476-4598-6-51.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!