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Books on the topic 'Multi-sensitive'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Lane, Leigh Blackmon. Multi-disciplinary teams in context-sensitive solutions. Washington, D.C: Transportation Research Board, 2007.

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2

Spiritually sensitive caregiving: A multi-faith handbook. Burnsville, NC: Compassion Press, 2008.

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3

Mrozek, Ireneusz. Multi-run Memory Tests for Pattern Sensitive Faults. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-91204-2.

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4

Hotel, New Delhi) National Consultation on Women Living with HIV (2004 Samrat. Shaping a new reality for women living with HIV/AIDS: Towards a gender sensitive multi-sectoral response. New Delhi: Positive Women Network, 2005.

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5

Bayliss, Julian Luke. Use of GIS, geostatistics, and multilevel modelling for biodiversity action planning: The use of habitat association models for multi-species habitat conservation in the Upper Thames Tributaries Environmentally Sensitive Area. [Oxford]: Oxford Brookes University, 2002.

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6

Prampolini, Gaetano, and Annamaria Pinazzi, eds. The Shade of the Saguaro / La sombra del saguaro. Florence: Firenze University Press, 2013. http://dx.doi.org/10.36253/978-88-6655-393-9.

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This volume springs from that fruitful project of scientific cooperation between the humanities departments of Università di Firenze and University of Arizona which was the Forum for the Study of the Literary Cultures of the Southwest (2000-2007). Tri-cultural, at least (Native, Hispanic and Anglo-American), and multi-lingual, today’s Southwest presents a complex coexistence of different cultures, the equal of which would be hard to find elsewhere in the United States. Of this virtually inexhaustible object of study, the essays here collected tackle an ample range of themes. While the majority of them are concerned with the literatures of the Southwest, still a good third falls into the fields of history, art history, ethnography, sociology or cultural studies. They are partitioned in four sections, the first three reflecting the chronology of the stratification of the three major cultures and the fourth highlighting one of the most sensitive topics in and about contemporary Southwest – the borderlands/la frontera.

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7

Multi-Disciplinary Teams in Context-Sensitive Solutions. Washington, D.C.: Transportation Research Board, 2007. http://dx.doi.org/10.17226/23123.

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8

Mrozek, Ireneusz. Multi-run Memory Tests for Pattern Sensitive Faults. Springer, 2019.

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9

Mrozek, Ireneusz. Multi-run Memory Tests for Pattern Sensitive Faults. Springer, 2018.

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10

Council, Zambia National HIV/AIDS/STD/TB, ed. Facilitator's manual on planning for gender sensitive multi-sectoral response to HIV/AIDS initiatives. [Lusaka]: National HIV/AIDS/STD/TB Council, 2005.

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11

Council, Zambia National HIV/AIDS/STD/TB, ed. Facilitator's manual on planning for gender sensitive multi-sectoral response to HIV/AIDS initiatives. Lusaka: National HIV/AIDS Council, 2005.

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12

Alqassas, Ahmad. A Multi-locus Analysis of Arabic Negation. Edinburgh University Press, 2019. http://dx.doi.org/10.3366/edinburgh/9781474433143.001.0001.

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This book studies the micro-variation in the syntax of negation of Southern Levantine, Gulf and Standard Arabic. By including new and recently published data that support key issues for the syntax of negation, the book challenges the standard parametric view that negation has a fixed parametrized position in syntactic structure. It particularly argues for a multi-locus analysis with syntactic, semantic, morphosyntactic and diachronic implications for the various structural positions. Thus accounting for numerous word order restrictions, semantic ambiguities and pragmatic interpretations without complicating narrow syntax with special operations, configurations or constraints. The book includes data from Southern Levantine, Gulf and Standard Arabic, which shed light on word order contrasts in negative clauses and their interaction with tense/aspect, mood/modality, semantic scope over adverbs, and negative sensitive items. It also has new data challenging the standard claim in Arabic linguistics literature that negation has a fixed parametrized position in the clause structure. The book brings a new perspective on the role of negation in licensing negative sensitive items, scoping over propositions and interacting with pragmatic notions such as presupposition and speech acts.

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13

Fuglsang-Frederiksen, Anders, Kirsten Pugdahl, and Hatice Tankisi. Quantitative electromyography. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0008.

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Several quantitative electromyography (QEMG) methods are used for diagnosing and monitoring in patients with neuromuscular disorders. At weak effort of the muscle, motor unit potential (MUP) analyses as individual MUP, multi-MUP, and macro-EMG are diagnostically sensitive and well tested. At higher effort of the muscle, interference pattern analyses such as the turns amplitude analysis are also diagnostically sensitive. Other potential diagnostic methods are power spectrum analysis, muscle fibre conduction velocity analysis, and some surface EMG methods. In patients with myopathy, QEMG has an important role in the diagnosis as a supplement to blood tests, muscle biopsy, and genetic testing. In patients with neurogenic disorders such as anterior horn cell disorders, peripheral nerve lesions, or polyneuropathy, QEMG has important roles in characterizing the lesion and differential diagnosis. Furthermore, QEMG may be useful in the examination of patients with neuromuscular transmission failure, critical illness disorders, and in treatment of dystonic muscle with botulinum toxin.

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14

Jacobsson, Katarina, and Jaber Gubrium, eds. Doing Human Service Ethnography. Policy Press, 2021. http://dx.doi.org/10.47674/9781447355809.

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EPDF and EPUB available Open Access under CC-BY-NC-ND licence. Human service work is performed in many places – hospitals, shelters, households – and is characterised by a complex mixture of organising principles, relations and rules. Using ethnographic methods, researchers can investigate these site-specific complexities, providing multi-dimensional and compelling analyses. Bringing together both theoretical and practical material, this book shows researchers how ethnography can be carried out within human service settings. It provides an invaluable guide on how to apply ethnographic creativeness and offers a more humanistic and context-sensitive approach in the field of health and social care to generating valid knowledge about today’s service work.

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15

Empson, Laura. Leadership Evolution. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198744788.003.0008.

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How does the locus of power shift around a professional organization as it grows, and what are the implications for leadership? This chapter presents a multi-stage model of evolutionary and revolutionary growth in professional organizations from Founder-focused to Collegial, from Committee-based to Delegated, from Federated to Corporate. It shows how, as a professional organization increases in size and complexity over time, unresolved governance problems may precipitate organizational crises which can in turn lead to dramatic shifts in the locus of power. It explains the complex and messy reality of leadership in professional organizations, emphasizing the crises and reversals that can occur during aborted attempts at governance change. It emphasizes how leaders need to be sensitive to the consequences of these changes, and explores how they can adapt their approach to leadership accordingly.

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16

Niazi, Imran Khalid, and Navin Ramachandran. Imaging the abdomen in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0174.

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Abdominal evaluation of the critically-ill patient is challenging. The patient may have a vague presentation, sometimes with a poor clinical history, few localizing signs, multiple co-morbidities and multi-organ involvement. Often the patient will require resuscitation prior to diagnostic work-up, and support devices such as mechanical ventilators and haemofilters may hamper assessment. Such unreliability of clinical indicators and the myriad of abdominal pathologies in a critically-ill patient may lead to diagnostic uncertainty with consequent delays in treatment. These challenges make imaging one of the most critical steps in the management of such patients. The optimal imaging pathway should be sensitive, specific, and minimize delay in therapy, but should also account for the patient’s clinical state and overall radiation dose. The modalities that have a role in abdominal evaluation of the critically ill are covered in this chapter.

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17

Giannitsis, Evangelos, and Hugo A. Katus. Biomarkers in acute coronary syndromes. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0036.

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Biomarker testing in the evaluation of a patient with acute chest pain is best established for cardiac troponins that allow the diagnosis of myocardial infarction, risk estimation of short- and long-term risk of death and myocardial infarction, and guidance of pharmacological therapy, as well as the need and timing of invasive strategy. Newer, more sensitive troponin assays have become commercially available and have the capability to detect myocardial infarction earlier and more sensitively than standard assays, but they are hampered by a lack of clinical specificity, i.e. the ability to discriminate myocardial ischaemia from myocardial necrosis not related to ischaemia such as myocarditis, pulmonary embolism, or decompensated heart failure. Strategies to improve clinical specificity (including strict adherence to the universal myocardial infarction definition and the need for serial troponin measurements to detect an acute rise and/or fall of cardiac troponin) will improve the interpretation of the increasing number of positive results. Other biomarkers of inflammation, activated coagulation/fibrinolysis, and increased ventricular stress mirror different aspects of the underlying disease activity and may help to improve our understanding of the pathophysiological mechanisms of acute coronary syndromes. Among the flood of new biomarkers, there are several novel promising biomarkers, such as copeptin that allows an earlier rule-out of myocardial infarction in combination with cardiac troponin, whereas MR-proANP and MR-proADM appear to allow a refinement of cardiovascular risk. GDF-15 might help to identify candidates for an early invasive vs conservative strategy. A multi-marker approach to biomarkers becomes more and more attractive, as increasing evidence suggests that a combination of several biomarkers may help to predict individual risk and treatment benefits, particularly among troponin-negative subjects. Future goals include the acceleration of rule-in and rule-out of patients with suspected acute coronary syndrome, in order to shorten lengths of stay in the emergency department, and to optimize patient management and the use of health care resources. New algorithms using high-sensitivity cardiac troponin assays at low cut-offs alone, or in combination with additional biomarkers, allow to establish accelerated rule-out algorithms within 1 or 2 hours.

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18

Giannitsis, Evangelos, and Hugo A. Katus. Biomarkers in acute coronary syndromes. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0036_update_001.

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Abstract:

Biomarker testing in the evaluation of a patient with acute chest pain is best established for cardiac troponins that allow the diagnosis of myocardial infarction, risk estimation of short- and long-term risk of death and myocardial infarction, and guidance of pharmacological therapy, as well as the need and timing of invasive strategy. Newer, more sensitive troponin assays have become commercially available and have the capability to detect myocardial infarction earlier and more sensitively than standard assays, but they are hampered by a lack of clinical specificity, i.e. the ability to discriminate myocardial ischaemia from myocardial necrosis not related to ischaemia such as myocarditis, pulmonary embolism, or decompensated heart failure. Strategies to improve clinical specificity (including strict adherence to the universal myocardial infarction definition and the need for serial troponin measurements to detect an acute rise and/or fall of cardiac troponin) will improve the interpretation of the increasing number of positive results. Other biomarkers of inflammation, activated coagulation/fibrinolysis, and increased ventricular stress mirror different aspects of the underlying disease activity and may help to improve our understanding of the pathophysiological mechanisms of acute coronary syndromes. Among the flood of new biomarkers, there are several novel promising biomarkers, such as copeptin that allows an earlier rule-out of myocardial infarction in combination with cardiac troponin, whereas MR-proANP and MR-proADM appear to allow a refinement of cardiovascular risk. GDF-15 might help to identify candidates for an early invasive vs conservative strategy. A multi-marker approach to biomarkers becomes more and more attractive, as increasing evidence suggests that a combination of several biomarkers may help to predict individual risk and treatment benefits, particularly among normal-troponin subjects. Future goals include the acceleration of rule-in and rule-out of patients with suspected acute coronary syndrome, in order to shorten lengths of stay in the emergency department, and to optimize patient management and the use of health care resources. New algorithms using high-sensitivity cardiac troponin assays at low cut-offs alone, or in combination with additional biomarkers, allow to establish accelerated rule-out algorithms within 1 or 2 hours.

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19

Giannitsis, Evangelos, and Hugo A. Katus. Biomarkers in acute coronary syndromes. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0036_update_002.

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Abstract:

Biomarker testing in the evaluation of a patient with acute chest pain is best established for cardiac troponins that allow the diagnosis of myocardial infarction, risk estimation of short- and long-term risk of death and myocardial infarction, and guidance of pharmacological therapy, as well as the need and timing of invasive strategy. Newer, more sensitive troponin assays have become commercially available and have the capability to detect myocardial infarction earlier and more sensitively than standard assays, but they are hampered by a lack of clinical specificity, i.e. the ability to discriminate myocardial ischaemia from myocardial necrosis not related to ischaemia such as myocarditis, pulmonary embolism, or decompensated heart failure. Strategies to improve clinical specificity (including strict adherence to the universal myocardial infarction definition and the need for serial troponin measurements to detect an acute rise and/or fall of cardiac troponin) will improve the interpretation of the increasing number of positive results. Other biomarkers of inflammation, activated coagulation/fibrinolysis, and increased ventricular stress mirror different aspects of the underlying disease activity and may help to improve our understanding of the pathophysiological mechanisms of acute coronary syndromes. Among the flood of new biomarkers, there are several novel promising biomarkers, such as copeptin that allows an earlier rule-out of myocardial infarction in combination with cardiac troponin, whereas MR-proANP and MR-proADM appear to allow a refinement of cardiovascular risk. GDF-15 might help to identify candidates for an early invasive vs conservative strategy. A multi-marker approach to biomarkers becomes more and more attractive, as increasing evidence suggests that a combination of several biomarkers may help to predict individual risk and treatment benefits, particularly among normal-troponin subjects. Future goals include the acceleration of rule-in and rule-out of patients with suspected acute coronary syndrome, in order to shorten lengths of stay in the emergency department, and to optimize patient management and the use of health care resources. New algorithms using high-sensitivity cardiac troponin assays at low cut-offs alone, or in combination with additional biomarkers, allow to establish accelerated rule-out algorithms within 1 or 2 hours.

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