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Relevant bibliographies by topics / Multiple Organ Dysfunction Syndrome (MODS)

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Academic literature on the topic 'Multiple Organ Dysfunction Syndrome (MODS)'

Author: Grafiati

Published: 5 June 2025

Last updated: 2 August 2025

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Journal articles on the topic "Multiple Organ Dysfunction Syndrome (MODS)"

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Gourd, Nicholas M., and Nikitas Nikitas. "Multiple Organ Dysfunction Syndrome." Journal of Intensive Care Medicine 35, no. 12 (2019): 1564–75. http://dx.doi.org/10.1177/0885066619871452.

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Multiple organ dysfunction syndrome (MODS) is one of the most common syndromes of critical illness and the leading cause of mortality among critically ill patients. Multiple organ dysfunction syndrome is the clinical consequence of a dysregulated inflammatory response, triggered by clinically diverse factors with the main pillar of management being invasive organ support. During the last years, the advances in the clarification of the molecular pathways that trigger, mitigate, and determine the outcome of MODS have led to the increasing recognition of MODS as a distinct disease entity with dis

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Weiss, Scott L., Joseph A. Carcillo, Francis Leclerc, et al. "Refining the Pediatric Multiple Organ Dysfunction Syndrome." Pediatrics 149, Supplement_1 (2022): S13—S22. http://dx.doi.org/10.1542/peds.2021-052888c.

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Since its introduction into the medical literature in the 1970s, the term multiple organ dysfunction syndrome (or some variant) has been applied broadly to any patient with &gt;1 concurrent organ dysfunction. However, the epidemiology, mechanisms, time course, and outcomes among children with multiple organ dysfunction vary substantially. We posit that the term pediatric multiple organ dysfunction syndrome (or MODS) should be reserved for patients with a systemic pathologic state resulting from a common mechanism (or mechanisms) that affects numerous organ systems simultaneously. In contra

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Pang, Diana, Dalia Bashir, Joseph Carcillo, Trung Nguyen, Rajesh Aneja, and Alicia Alcamo. "Role of Damage-Associated Molecular Patterns and Uncontrolled Inflammation in Pediatric Sepsis-Induced Multiple Organ Dysfunction Syndrome." Journal of Pediatric Intensive Care 08, no. 01 (2018): 025–31. http://dx.doi.org/10.1055/s-0038-1675639.

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AbstractThe incidence of multiple organ dysfunction syndrome (MODS) in sepsis varies from 17 to 73% and furthermore, increases the risk of death by 60% when controlled for the number of dysfunctional organs. Several MODS phenotypes exist, each unique in presentation and pathophysiology. Common to the phenotypes is the stimulation of the immune response by pathogen-associated molecular patterns (PAMPs), or danger-associated molecular patterns (DAMPs) causing an unremitting inflammation. Two of the MODS phenotypes are discussed in detail, thrombocytopenia-associated multiple organ failure (TAMOF

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Alam, Mohammad Shaha, and Syeda Humaira Begum. "Multiple Organ Dysfunction Syndrome In Major Burns Patients." Medicine Today 22, no. 2 (2012): 75–79. http://dx.doi.org/10.3329/medtoday.v22i2.12438.

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Organ dysfunctions are frequent after major burn trauma. Development of MODS in severely burned patients is highly fatal. The present study was conducted to elucidate the etiology and quantify the Multiple Organ Dysfunction (MODS) in major burn patients. Multiple organ dysfunctions in major burnt patients was a prospective analytic study. A total of thirty patients were included in the study, which was conducted in the Burn and Plastic Surgery unit, Bir Hospital, Kathmondu, Nepal over the period of eighteen months from January 2002 to June 2003. Patients with acute major burn i.e. 24 hours f

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Golomidov, A. V., E. V. Grigoriev, V. G. Moses, and K. B. Moses. "Multiple organ dysfunction syndrome prediction in newborn children." Innovative Medicine of Kuban, no. 1 (March 30, 2022): 83–89. http://dx.doi.org/10.35401/2500-0268-2022-25-1-83-89.

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There are several directions for predicting multiple organ dysfunction syndrome (MODS), but almost all of them are poorly tested in neonatology. This review is presented to indicate the problem of the condition severity objectification of newborns and the possibility of predicting the development of MODS. Scales for assessing the severity of MODS in critically ill children have been developed and used since the end of the last century, but their validation in the newborns faces certain difficulties. Prognostic nosospecific scales: NICHD (National Institute of Child Health and Human Development

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Akhila, Anil1 Anjitha Santhosh1 Manju Thampi2 Shaiju. S. Dharan3 Drishya L.4*. "TROPIC FEVER WITH MULTIPLR ORGAN DYSFUNCTION SYNDROME : A CASE REPORT." International Journal in Pharmaceutical Sciences 2, no. 4 (2024): 1207–10. https://doi.org/10.5281/zenodo.11080552.

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Tropic  fever with multiple organ dysfunction is often seen as undifferentiated fevers with organ failure. Dengue, scrub typhus, leptospirosis, malaria, typhoid fever and some other fevers are incorporated in Tropic fever. These fevers are prevalent in tropical and subtropical regions. Symptoms of tropic fever includes sudden onset of fever, headache, chills, myalgia, transient skin rashes, conjunctival suffusion. A variety of acute insults, including sepsis, can cause progressive and potentially reversible physiologic dysfunction in two or organs or organ systems which is what is known a

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Royston, Rachel. "Multiple organ dysfunction syndrome: an extended patient care report." Veterinary Nurse 12, no. 2 (2021): 85–91. http://dx.doi.org/10.12968/vetn.2021.12.2.85.

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This report describes and reflects on the intensive nursing care provided to a patient with multiple organ dysfunction syndrome (MODS) and systemic inflammatory response syndrome (SIRS). Veterinary nurses are at the forefront of supportive care for these critical patients and nursing interventions should focus on close and frequent monitoring for early detection of sepsis. An understanding of the physiological processes that occur with these diseases can increase awareness of the potential life-threatening complications associated with MODS, thus, allowing early recognition and intervention to

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Baue, Arthur E., Rodney Durham, and Eugen Faist. "SYSTEMIC INFLAMMATORY RESPONSE SYNDROME (SIRS), MULTIPLE ORGAN DYSFUNCTION SYNDROME (MODS), MULTIPLE ORGAN FAILURE (MOF)." shock 10, no. 2 (1998): 79–89. http://dx.doi.org/10.1097/00024382-199808000-00001.

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Halim, Hendy, Ida Bagus Gede Suparyatha, I. Made Arimbawa, and I. Nyoman Budi Hartawan. "Multiple organ dysfunction syndrome associated with hyperglycemia in children requiring intensive care." Paediatrica Indonesiana 55, no. 4 (2015): 230. http://dx.doi.org/10.14238/pi55.4.2015.230-4.

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Background Hyperglycemia can be caused by three or more organ dysfunctions and occurs in children requiring intensive care in the first 48 hours. Blood sugar level higher than 140 mg/dl is considered as hyperglycemia in children requiring intensive care.Objective To determine the association between multiple organ dysfunction syndrome (MODS) in children requiring intensive care and hyperglycemia with blood sugar level higher than 140 mg/dl.Methods This case control study without matching was conducted on children aged 1 month-12 years from pediatric ward at Sanglah hospital during June-August

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Whitmore, Laura C., Kelli L. Goss, Elizabeth A. Newell, Brieanna M. Hilkin, Jessica S. Hook, and Jessica G. Moreland. "NOX2 protects against progressive lung injury and multiple organ dysfunction syndrome." American Journal of Physiology-Lung Cellular and Molecular Physiology 307, no. 1 (2014): L71—L82. http://dx.doi.org/10.1152/ajplung.00054.2014.

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Systemic inflammatory response syndrome (SIRS) is a common clinical condition in patients in intensive care units that can lead to complications, including multiple organ dysfunction syndrome (MODS). MODS carries a high mortality rate, and it is unclear why some patients resolve SIRS, whereas others develop MODS. Although oxidant stress has been implicated in the development of MODS, several recent studies have demonstrated a requirement for NADPH oxidase 2 (NOX2)-derived oxidants in limiting inflammation. We recently demonstrated that NOX2 protects against lung injury and mortality in a murin

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Dissertations / Theses on the topic "Multiple Organ Dysfunction Syndrome (MODS)"

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Sydorchuk, Ryslan Ihorovuch, Oleg Yosypovych Khomko, Oleksandr Matviyovych Plehutsa, Oleg Ihorovich Plyansky, Volodymir Dmytrovich Shafranyuk, and Svitlana Illivna Raileanu. "Acute hepatic failure under abdominal sepsis: multiple organ dysfunction syndrome." Thesis, Матерiали 97-ї пiдсумковоi' наукової конференцiї професорсько-викладацького персоналу вищого державного навчального закладу України «Буковинський державний медичний унiверситет» (Чернiвцi, 15, 17 ,22 лютого 2016 р.) - Чернiвцi: Медунiверситет, 2016, 2016. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/10450.

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Sydorchuk, Larysa Petrivna, Oleg Yosypovych Khomko, Oleksandr Matviyovych Plehutsa, Oleh Ihorovych Polyanskyy, Volodymir Dmytrovich Shafranyuk, and S. I. Raileanu. "Acute hepatic failure under abdominal sepsis: multiple organ dysfunction syndrome." Thesis, Матерiали 97-ї пiдсумковоi' наукової конференцiї професорсько-викладацького персоналу вищого державного навчального закладу України «Буковинський державний медичний унiверситет» (Чернiвцi, 15, 17 ,22 лютого 2016 р.) - Чернiвцi: Медунiверситет, 2016, 2016. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/10402.

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Ruetten, Hartmut. "Role of nitric oxide and endothelin-1 in the multiple organ dysfunction syndrome in sepsis." Thesis, Oxford Brookes University, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.267520.

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Reid, Clare Louise. "Immunological and metabolic features of multiple organ dysfunction syndrome : a study of their association and potential modification by 'immunonutrition'." Thesis, University of Manchester, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.502915.

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Rationale: 1. To characterise and quantify the cytokine response to critical illness and relate this response to common metabolic features of the illness; 2. To determine whether these immunologic and metabolic responses can be modified using enteral immunonutrition. Methods: Circulating cytokine concentrations (TNFα, IL-6, IL-10, TGF-β1, IL-lra, sTNF-RI and sTNF-RII), c-reactive protein and cortisol levels were determined using the ELISA technique. Cytokine genotypes (IL-10, TNFα and TNFβ) were determined following the amplification of DNA using polymerase chain reaction (PCR) then by using s

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von, Seth Magnus. "Oxygen delivery and mitochondrial dysfunction as assessed by microdialysis during interventions in experimental sepsis." Doctoral thesis, Uppsala universitet, Anestesiologi och intensivvård, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-326788.

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Early administration of broad-spectrum antibiotics is the first goal in sepsis treatment. Besides from bacteriostatic/bactericidal effects, some antibiotics may also modify the host´s response to infection. The novel antibiotic tigecycline may exert such properties; however, this property has not been evaluated in large-animal trials. We compared tigecycline with doxycycline and placebo in relation to anti-inflammatory, circulatory and organ dysfunction effects in a sterile pig model of sepsis. Doxycycline, but not tigecycline, reduced the inflammatory response as manifested by tumor necrosis

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Rocha, Tais Sica da. "Marcadores de síndrome da resposta inflamatória sistêmica e sepse no pós-operatório de cirurgia cardíaca em crianças." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2012. http://hdl.handle.net/10183/77225.

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Objetivo geral: estudar a síndrome da resposta inflamatória sistêmica após a cirurgia cardíaca com circulação extracorpórea (CEC) e a sua relação com marcadores inflamatórios. Objetivos específicos: 1) avaliar a prevalência de síndrome da resposta inflamatória sistêmica (SIRS), sepse e disfunção de múltiplos órgãos (DMO); 2) avaliar a relação da SIRS, sepse e DMO com certos biomarcadores; 3) avaliar a relação desses biomarcadores com mortalidade no pós-operatório de cirurgia cardíaca com CEC em crianças; 4) estudar a cinética do soluble triggering receptor on myeloid cells-1 (sTREM-1), procalc

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Lin, Ya-Ying, and 林雅瑩. "Therapeutic Effects of Hydralazine on Peritonitis-Induced Septic Shock with Multiple Organ Dysfunction Syndrome in Rats." Thesis, 2011. http://ndltd.ncl.edu.tw/handle/51780200354191127459.

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碩士<br>國防醫學院<br>藥理學研究所<br>99<br>Sepsis and its sequelae, multiple organ dysfunction syndrome (MODS), are major contributors of mortality in critical ill. Global tissue hypoxia results from an imbalance between systemic oxygen delivery and demand, and is a key development preceding multiple organ failure and death. Hydralazine preferentially dilates the arterioles and increases renal blood flow. In addition, hydralazine per se has powerful free radical scavenger properties. Therefore, this study was to examine the effect of hydralazine on peritonitis- induced sepsis with MODS in Wistar rats. In

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LI, Kai-Yi, and 李凱益. "Therapeutic Effects of Levosimendan on Peritonitis-Induced Septic Shock with Multiple Organ Dysfunction Syndrome in Rats." Thesis, 2010. http://ndltd.ncl.edu.tw/handle/34075137309031189172.

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碩士<br>國防醫學院<br>藥理學研究所<br>98<br>Sepsis/septic shock and its sequelae, multiple organ dysfunction syndrome (MODS), are major contributors of mortality in critically ill patients. In sepsis, bacterial products and toxins activate cells of innate immune system (macrophages and neutrophils) to release amount of inflammatory mediator and the formation of free radicals results in producing large quantity of nitric oxide (NO), leading to circulatory failure and an imbalance between systemic oxygen delivery and demand, and finally causing MODS. Although the use of vasopressors is recommended, but there

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Chen, Jui-Ling, and 陳瑞鈴. "Effects of 1,25-dihydroxycholecaliferol on multiple organ dysfunction syndrome of peritonitis-induced septic shock with in rats." Thesis, 2010. http://ndltd.ncl.edu.tw/handle/79675090426143279715.

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碩士<br>國防醫學院<br>藥理學研究所<br>97<br>Septic shock is characterized by a decrease in systemic vascular resistance and its main cause of death is due to multiple organ dysfunction syndrome (MODS). The pathogenesis of sepsis is related to the release of inflammatory mediators and the formation of free radicals and nitric oxide (NO), leading to circulatory failure and an imbalance of between systemic oxygen delivery and demand, and finally causing MODS. 1,25-Dihydroxy-vitamin D3 (1,25(OH)2D3), an active form of vitamin D, is an important regulator of the calcium-phosphate homeostasis. The administration

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Villeneuve, Andréanne. "Transfusions de globules rouges en néonatologie et syndrome de défaillance multiviscérale aiguë." Thèse, 2016. http://hdl.handle.net/1866/18887.

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Le niveau d’hémoglobine (Hb) d’un nouveau-né diminue dans les premiers mois de vie. Cette anémie dite physiologique est plus sévère chez les nourrissons admis aux soins intensifs néonataux (SIN), et ceux-ci nécessitent souvent une transfusion sanguine. En néonatalogie, les indications de transfuser sont controversées et les pratiques transfusionnelles sont très variables. Pour mieux comprendre ces pratiques, nous avons mené l’étude prospective «Epidemiology and determinants of red blood cells transfusion in a neonatal intensive care unit: a cohort study». 13.4% des patients consécutifs a

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Books on the topic "Multiple Organ Dysfunction Syndrome (MODS)"

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F, Hammerle A., ed. MODS: Multiple-organ dysfunction syndrome : intensive care medicine in dialogue, international workshop, Schloss Wilhelminenberg, Vienna, December 10-12, 1993. Verlag Wilhelm Maudrich, 1994.

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Heinz-Günter, Sieberth, Stummvoll H. K, and Kierdorf H. 1957-, eds. Continuous extracorporeal treatment in multiple organ dysfunction syndrome. S. Karger, 1995.

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Ruetten, Hartmut. Role of nitric oxide and endothelin-1 in the multiple organ dysfunction syndrome in sepsis. Oxford Brookes University, 1998.

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Adam, Sheila, Sue Osborne, and John Welch. Sepsis and multiple organ dysfunction. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199696260.003.0010.

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Sepsis—a dysregulated systemic inflammatory response to infection—occurs at some point in most critical illnesses, and is the most common cause of multiple organ dysfunction syndrome (MODS). Patients with MODS always present great challenges to the critical care team and are often at high risk of death. This chapter discusses the latest thinking about the infective causes and complex evolution of sepsis, with details of how each of the main body systems can be affected (e.g. in the form of acute respiratory distress syndrome) and how different organ functions can be assessed and dysfunction re

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Fischer, Kevin M., and Shannon S. Carson. Chronic Multiple Organ Dysfunction. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0013.

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This chapter reviews the clinical syndrome of chronic multiple organ dysfunction (MOD) following acute critical illness. Chronic MOD, also referred to as chronic critical illness, occurs in patients who have survived the acute phase of their illness or injury but remain dependent on life support for weeks or months. This condition presents unique physiologic and metabolic abnormalities distinct from those encountered in the acute illness. These include neuroendocrine and immune dysregulation, ICU-acquired weakness, persistent respiratory failure, and brain dysfunction. The symptom burden for t

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Multiple organ dysfunction syndrome: Examining the role of eicosanoids and procoagulants. Chapman & Hall, 1996.

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Multiple Organ Dysfunction Syndrome: Examining the Role of Eicosanoids and Procoagulants (Medical Intelligence Unit). Landes Bioscience, 1996.

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Williams, John. Multiple Organ Dysfunction Syndrome: Examining the Role of Eicosanoids and Procoagulants (Medical Intelligence Unit). Landes Bioscience, 1996.

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Williams, J. G. Multiple Organ Dysfunction Syndrome: Examining the Role of Eicosanoids and Procoagulants (Medical Intelligence Unit). Springer, 1996.

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Ortiz, Julian Arias, Raphaël Favory, and Jean-Louis Vincent. Infection, sepsis, and multiorgan dysfunction syndrome. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0072.

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Sepsis is the main cause of multiple organ failure and remains a concern because of the associated high morbidity and mortality. In recent years, important advances have been made in the understanding of the pathophysiology of sepsis. Sepsis and septic shock are the end result of complex interactions between infecting organisms and various elements of the host response. A key feature of the common sequence of organ failure is dysfunction of the cardiovascular system, including microcirculatory elements. Outcome improvement in sepsis is based on recognizing the process early and instituting eff

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Book chapters on the topic "Multiple Organ Dysfunction Syndrome (MODS)"

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Marshall, J. C. "Multiple Organ Dysfunction Syndrome (MODS)." In Update in Intensive Care and Emergency Medicine. Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79224-3_8.

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Brogi, Etrusca, Federico Coccolini, and Francesco Forfori. "The Pathomechanism of Post-Injury Multiple Organ Dysfunction Syndrome (MODS)." In Postinjury Multiple Organ Failure. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-92241-2_2.

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Berlot, G., L. Silvestri, F. Iscra, G. Sganga, and A. Gullo. "Lights and Shadows in Sepsis and Multiple Organ Dysfunction Syndrome (MODS)." In Sepsis and Organ Dysfunction. Springer Milan, 1998. http://dx.doi.org/10.1007/978-88-470-2271-3_16.

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Chen, Peng, and Timothy Billiar. "Gut Microbiota and Multiple Organ Dysfunction Syndrome (MODS)." In Advances in Experimental Medicine and Biology. Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-2385-4_11.

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Pulido, Cindy G., and Eli D. Ehrenpreis. "Role of the Mesentery in Systemic Inflammation Response Syndrome (SIRS) and Multiple Organ Dysfunction Syndrome (MODS)." In The Mesenteric Organ in Health and Disease. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-71963-0_18.

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Proulx, François, Stéphane Leteurtre, Jean Sébastien Joyal, and Philippe Jouvet. "Multiple Organ Dysfunction Syndrome." In Pediatric Critical Care Medicine. Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-6362-6_35.

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Buchman, Timothy G. "Multiple Organ Dysfunction Syndrome." In Surgery. Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-57282-1_19.

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Bensard, Denis D., Philip F. Stahel, Jorge Cerdá, et al. "Multiple Organ Dysfunction Syndrome." In Encyclopedia of Intensive Care Medicine. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_1904.

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Zhou, Jian. "Multiple Organ Dysfunction Syndrome." In Explosive Blast Injuries. Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-19-2856-7_17.

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Fry, Donald E. "Multiple Organ Dysfunction Syndrome." In Surgery. Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-68113-9_31.

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Conference papers on the topic "Multiple Organ Dysfunction Syndrome (MODS)"

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GORECKI, Gabriel-Petre, Dana-Rodica TOMESCU, and Daniel COCHIOR. "Early diagnosis and optimal management of objective microcirculatory dysfunction to prevent the development of multiple organic dysfunction syndrome." In International congress "Research-Innovation-Inovative Entreneurship". Ion Creangă Pedagogical State University, 2024. http://dx.doi.org/10.46727/c.13-14-10-2023.p37-43.

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Microcirculatory dysfunction plays a central role in the genesis of multiple organ dysfunction syndrome (MODS), being a pathophysiological milestone of it. The mechanism of producing MODS is complex and still incompletely studied, having enough unknowns. It is now known that, despite adequate treatment of macrohemodynamics, there are few correlations between systemic hemodynamic improvement and correction of microcirculatory abnormalities, and the presence of these dysfunctions is more common among patients who do not survive than in survivors. Therapeutic intervention based on evaluation and

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Pidaparti, Ramana M., and Kevin R. Ward. "Airway Inflammation Induced by Mechanical Ventilation Through Multiscale Modeling." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80174.

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Inflammation has been recognized as a major integral component for most of the acute and chronic diseases. Inflammation can be initiated within the body as an innate process or by external factors such as infections and trauma. Inflammation is a complex and dynamic process, and involves nonlinearity and stochasticity. Without the inflammation, the harmful stimuli cannot be removed and the healing process cannot occur. However, an over-expression or under-expression of inflammatory responses can lead to severe consequences, such as Multiple Organ Dysfunction Syndrome (MODS), which is characteri

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Santhanakrishnan, Arvind, Trent Nestle, Brian Moore, Ajit P. Yoganathan, and Matthew L. Paden. "Characterization of a Low Extracorporeal Volume, High Accuracy Pediatric Continuous Renal Replacement Therapy Device." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80210.

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The incidence of acute kidney injury (AKI) is commonly seen in critically ill children, the origins of which may be traced to a wide range of conditions such as inborn errors of metabolism, sepsis, congenital heart defects, bone marrow and organ transplantation, and to a lesser extent from multiple organ dysfunction syndrome (MODS) [1]. It is vital to provide a form of fluid and electrolyte clearance in these patients until native renal function improves. Nearly 3,600 critically ill children per year with acute kidney injury receive life-saving continuous renal replacement therapy (CRRT) in th

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Rayasam, V., S. S. Haider, A. Mehta, A. Ladzinski, and P. P. Patel. "Thyrotoxicosis Factitia Leading to Multiple Organ Dysfunction Syndrome." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a2899.

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Stroup, Emily Kunce, Yuan Luo, and L. Nelson Sanchez-Pinto. "Phenotyping Multiple Organ Dysfunction Syndrome Using Temporal Trends in Critically Ill Children." In 2019 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2019. http://dx.doi.org/10.1109/bibm47256.2019.8983126.

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Dickson, Robert P., and John M. Lacy. "Acute Colchicine Toxicity As A Cause Of Rapid And Irreversible Multiple Organ Dysfunction Syndrome." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a3882.

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Kozlova, A., J. B. Gleason, and N. Maroz. "Multiple Organ Dysfunction Syndrome in Setting of Sweet's Syndrome Without Evidence of Underlying Infectious, Rheumatologic, or Oncologic Etiology." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6544.

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Sukamto, IGL, Bambang Purwanto, and Ambar Mudigdo. "The Optimal Dose of Intraperitoneal Heat Shock Protein (Hsp70) to Prevent Death in Sepsis Mice Model with Multiple Organ Dysfunction Syndrome." In Mid International Conference on Public Health 2018. Masters Program in Public Health Universitas Sebelas Maret, 2018. http://dx.doi.org/10.26911/mid.icph.2018.05.21.

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Lindell, R. B., S. Sayed, J. S. Campos, et al. "A Prognostic Proteomic Signature of Immune Dysregulation Is Present at Onset of Multiple Organ Dysfunction Syndrome in Pediatric Patients With and Without Sepsis." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a7149.

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Reports on the topic "Multiple Organ Dysfunction Syndrome (MODS)"

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Zeng, Siyao, Lei Ma, Lishan Yang, et al. Advantages of damage control surgery over conventional surgery inmultiple trauma: a meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.10.0006.

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Abstract:

Review question / Objective: This meta-analysis aims to explore whether damage control surgery has advantages over traditional surgery in the treatment of multiple trauma. Information sources: The Chinese Biomedical literature (CBM), Chinese National Knowledge Infrastructure (CNKI), Weipu (VIP), Duxiu, WanFang, Web of sciense, PubMed, Scopus, Ovid, EMbase, ProQuest, Cochrane, Chinese clinical trial Registry and Clinical Trials.gov databases. Main outcome(s): mortality rate, the success rate of rescue, In-hospital length of stay, ICU length of stay, the overall incidence rate of complications,

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Neodo, Anna, Fiona Augsburger, Jan Waskowski, Joerg C. Schefold, and Thibaud Spinetti. Monocytic HLA-DR expression and clinical outcomes in adult ICU patients with sepsis – a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.11.0119.

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Review question / Objective: The scope of this review was defined using PICOTS framework where 1) population: adult critically ill patients with sepsis or septic shock; 2) index prognostic factor: cell surface protein expression of mHLA-DR in blood; 3) comparative factor: none; 4) outcomes to be predicted: mortality, secondary infections, length of stay, and organ dysfunction score (sequential organ failure assessment [SOFA], multiple organ dysfunction score [MODS], logistic organ dysfunction score [LODS]), composite outcomes where component endpoints consist of at least one of the outcomes st

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