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Books on the topic 'Multiple Organ Dysfunction Syndrome (MODS)'

Author: Grafiati
Published: 5 June 2025
Last updated: 2 August 2025

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1

F, Hammerle A., ed. MODS: Multiple-organ dysfunction syndrome : intensive care medicine in dialogue, international workshop, Schloss Wilhelminenberg, Vienna, December 10-12, 1993. Verlag Wilhelm Maudrich, 1994.

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2

Heinz-Günter, Sieberth, Stummvoll H. K, and Kierdorf H. 1957-, eds. Continuous extracorporeal treatment in multiple organ dysfunction syndrome. S. Karger, 1995.

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3

Ruetten, Hartmut. Role of nitric oxide and endothelin-1 in the multiple organ dysfunction syndrome in sepsis. Oxford Brookes University, 1998.

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4

Adam, Sheila, Sue Osborne, and John Welch. Sepsis and multiple organ dysfunction. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199696260.003.0010.

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Sepsis—a dysregulated systemic inflammatory response to infection—occurs at some point in most critical illnesses, and is the most common cause of multiple organ dysfunction syndrome (MODS). Patients with MODS always present great challenges to the critical care team and are often at high risk of death. This chapter discusses the latest thinking about the infective causes and complex evolution of sepsis, with details of how each of the main body systems can be affected (e.g. in the form of acute respiratory distress syndrome) and how different organ functions can be assessed and dysfunction re
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5

Fischer, Kevin M., and Shannon S. Carson. Chronic Multiple Organ Dysfunction. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0013.

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This chapter reviews the clinical syndrome of chronic multiple organ dysfunction (MOD) following acute critical illness. Chronic MOD, also referred to as chronic critical illness, occurs in patients who have survived the acute phase of their illness or injury but remain dependent on life support for weeks or months. This condition presents unique physiologic and metabolic abnormalities distinct from those encountered in the acute illness. These include neuroendocrine and immune dysregulation, ICU-acquired weakness, persistent respiratory failure, and brain dysfunction. The symptom burden for t
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6

Multiple organ dysfunction syndrome: Examining the role of eicosanoids and procoagulants. Chapman & Hall, 1996.

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7

Multiple Organ Dysfunction Syndrome: Examining the Role of Eicosanoids and Procoagulants (Medical Intelligence Unit). Landes Bioscience, 1996.

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8

Williams, John. Multiple Organ Dysfunction Syndrome: Examining the Role of Eicosanoids and Procoagulants (Medical Intelligence Unit). Landes Bioscience, 1996.

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9

Williams, J. G. Multiple Organ Dysfunction Syndrome: Examining the Role of Eicosanoids and Procoagulants (Medical Intelligence Unit). Springer, 1996.

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10

Ortiz, Julian Arias, Raphaël Favory, and Jean-Louis Vincent. Infection, sepsis, and multiorgan dysfunction syndrome. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0072.

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Sepsis is the main cause of multiple organ failure and remains a concern because of the associated high morbidity and mortality. In recent years, important advances have been made in the understanding of the pathophysiology of sepsis. Sepsis and septic shock are the end result of complex interactions between infecting organisms and various elements of the host response. A key feature of the common sequence of organ failure is dysfunction of the cardiovascular system, including microcirculatory elements. Outcome improvement in sepsis is based on recognizing the process early and instituting eff
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11

Favory, Raphaël, and Jean-Louis Vincent. Infection, sepsis, and multiorgan dysfunction syndrome. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0072_update_001.

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Sepsis is the main cause of multiple organ failure and remains a concern because of the associated high morbidity and mortality. In recent years, important advances have been made in the understanding of the pathophysiology of sepsis. Sepsis and septic shock are the end result of complex interactions between infecting organisms and various elements of the host response. A key feature of the common sequence of organ failure is dysfunction of the cardiovascular system, including microcirculatory elements. Outcome improvement in sepsis is based on recognizing the process early and instituting eff
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12

Favory, Raphaël, and Jean-Louis Vincent. Infection, sepsis, and multiorgan dysfunction syndrome. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0072_update_002.

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Sepsis is the main cause of multiple organ failure and remains a concern because of the associated high morbidity and mortality. In recent years, important advances have been made in the understanding of the pathophysiology of sepsis. Sepsis and septic shock are the end result of complex interactions between infecting organisms and various elements of the host response. A key feature of the common sequence of organ failure is dysfunction of the cardiovascular system, including microcirculatory elements. Outcome improvement in sepsis is based on recognizing the process early and instituting eff
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13

Favory, Raphaël, and Jean-Louis Vincent. Infection, sepsis, and multiorgan dysfunction syndrome. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0072_update_003.

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Abstract:
Sepsis is the main cause of multiple organ failure and remains a concern because of the associated high morbidity and mortality. In recent years, important advances have been made in the understanding of the pathophysiology of sepsis. Sepsis and septic shock are the end result of complex interactions between infecting organisms and various elements of the host response. A key feature of the common sequence of organ failure is dysfunction of the cardiovascular system, including microcirculatory elements. Outcome improvement in sepsis is based on recognizing the process early and instituting eff
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14

(Editor), H. G. Sieberth, H. K. Stummvoll (Editor), and H. Kierdorf (Editor), eds. Continuous Extracorporeal Treatment in Multiple Organ Dysfunction Syndrome: 3rd International Conference on Continuous Hemofiltration, Vienna, July 8, 1994 (Contributions to Nephrology). S. Karger AG (Switzerland), 1995.

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15

Franklin, Andrew. Sickle Cell Disease. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0084.

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Sickle cell disease, a hemoglobinopathy that affects multiple organ systems, is a complex disease entity that presents unique challenges during the perioperative period. The hallmark of sickle cell disease, vaso-occlusion, results from sickling of erythrocytes containing hemoglobin of abnormal conformation due to genetically mutated beta globin genes. The perioperative clinician must properly care for acute sickle cell crises including acute painful episodes and acute chest syndrome, and safely care for the sickle cell patient through the preoperative, intraoperative, and postoperative phases
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16

Blaser, Annika Reintam, and Adam M. Deane. Normal physiology of the gastrointestinal system. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0172.

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The gastrointestinal (GI) system is responsible for digestion and absorption, but also has important endocrine, immune and barrier functions. Additionally, the GI system plays a major role in fluid, electrolyte and acid-base balance. The GI system is regulated by complex myogenic, neural and humoral mechanisms, and, in health, these are affected by the presence of luminal nutrient, thereby modulating function of the GI system. Accordingly, GI function varies depending on whether a person is fasted or in the postprandial state. Adequate fasting and postprandial perfusion, motility and exocrine
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17

Ronco, Claudio, and Zaccaria Ricci. Renal support therapy. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0029.

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Renal dysfunction is known to be frequently a component of multiple organ failure, a complex syndrome affecting the most severely ill critical patients. Bidirectional interaction between the kidneys and other organs has always been suspected; evidence suggests that severe kidney injury is an important protagonist in acute illness, even when managed by dialysis. In fact, if it seems that increasing the dose of renal replacement therapy does not reduce mortality, it could be inferred that acute kidney injury influences mortality through means that are not reversed by conventional renal support,
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18

Ronco, Claudio, and Zaccaria Ricci. Renal support therapy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0029_update_001.

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Renal dysfunction is known to be frequently a component of multiple organ failure, a complex syndrome affecting the most severely ill critical patients. Bidirectional interaction between the kidneys and other organs has always been suspected; evidence suggests that severe kidney injury is an important protagonist in acute illness, even when managed by dialysis. In fact, if it seems that increasing the dose of renal replacement therapy does not reduce mortality, it could be inferred that acute kidney injury influences mortality through means that are not reversed by conventional renal support,
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19

Ronco, Claudio, and Zaccaria Ricci. Renal support therapy. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0029_update_002.

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Abstract:
Renal dysfunction is known to be frequently a component of multiple organ failure, a complex syndrome affecting the most severely ill critical patients. Bidirectional interaction between the kidneys and other organs has always been suspected; evidence suggests that severe kidney injury is an important protagonist in acute illness, even when managed by dialysis. In fact, if it seems that increasing the dose of renal replacement therapy does not reduce mortality, it could be inferred that acute kidney injury influences mortality through means that are not reversed by conventional renal support,
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