Relevant bibliographies by topics / Murine macrophages (RAW 264.7)

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Academic literature on the topic 'Murine macrophages (RAW 264.7)'

Author: Grafiati
Published: 4 June 2025
Last updated: 23 June 2025

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Journal articles on the topic "Murine macrophages (RAW 264.7)"

1

Trivedi, Mahendra Kumar, Alice Branton, Dahryn Trivedi, Gopal Nayak, Sambhu Charan Mondal, and Snehasis Jana. "Immunomodulatory Effect of the Biofield Energy Treated Cell Growth Medium (DMEM) and FBS in Immune Cells (Murine Macrophage RAW 264.7)." Journal of Cellular Immunology and Serum Biology 3, no. 2 (2018): 1–5. https://doi.org/10.15436/2471-5891.18.1946.

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The objective of the present study was to evaluate the immunomodulatory effect of Biofield Energy Treatment (The Trivedi Effect&reg;) on Dulbecco&rsquo;s Modified Eagle&rsquo;s Medium (DMEM) and fetal bovine serum (FBS) in murine macrophage cells (RAW 264.7). The study parameters were evaluated using cell viability by MTT assay and estimation of proinflammatory cytokine like tumor necrosis factor - alpha (TNF-&alpha;) on immunomodulation using enzyme linked immune sorbent assay (ELISA). The cell viability using MTT assay data showed that more than 80% cell viability was observed in all the tested groups compared to the baseline control group (G1). The characteristics of cell morphology in the Biofield Energy Treated DMEM without FBS (G6) group showed some changes in the cell morphology as evidenced a transition from spherical to elongate and also showed maturation compared to the G1 group. The level of TNF-&alpha; expression was significantly reduced by 23.15% in the G3 group compared to the G1 group. Moreover, the secretion of TNF-&alpha; was altered by 38.06%, 35.77%, and 282.49% in the G4, G5, and G6 groups, respectively compared to the G1 group. The overall results demonstrated that the Trivedi Effect&reg; - Consciousness Energy Healing Treatment has an impact on DMEM and FBS by alteration of cell morphology and reducing the level of TNF-&alpha; expression in immune cell (RAW 264.7). Therefore, the Trivedi Effect&reg; treated DMEM and FBS might be useful as an immunomodulator for various immune-related disorders like Graves&rsquo; disease, rheumatoid arthritis, multiple sclerosis, etc. <strong>Source:</strong> https://www.trivedieffect.com/science/immunomodulatory-effect-of-the-biofield-energy-treated-cell-growth-medium-dmem-and-fbs-in-immune-cells-murine-macrophage-raw-264-7/ https://www.ommegaonline.org/article-details/Immunomodulatory-Effect-of-the-Biofield-Energy-Treated-Cell-Growth-Medium-DMEM-and-FBS-in-Immune-Cells-Murine-Macrophage-RAW-2647/1968
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2

Tellez, Angela, Milena Corredig, Lubov Y. Brovko, and Mansel W. Griffiths. "Characterization of immune-active peptides obtained from milk fermented byLactobacillus helveticus." Journal of Dairy Research 77, no. 2 (2010): 129–36. http://dx.doi.org/10.1017/s002202990999046x.

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The objectives of this research were to confirm the effect of compounds derived from milk fermented byLactobacillus helveticus(LH-2) on the nonspecific host defence system, and isolate and characterize the active peptides that mediate the immune response. The cell-free supernatant obtained from the fermented milk and its fractions were testedin vitrofor immuno-modulating activity using murine macrophages (RAW 264·7 cell line). Cytokine production (Interleukin-6 (IL-6), Tumor Necrosis Factor-α (TNF-α), and Interleukin-1β (IL1-β)), nitric oxide (NO) production and phagocytosis were used as biomarkers. Macrophages stimulated with cell-free supernatant of fermented milk showed higher production of cytokines and NO compared with macrophages stimulated with LPS (Lipopolysaccharide) and a commercial immunomodulator derived from β-casein (f54-59). Phagocytosis was observed by macrophages stimulated with the supernatant. Two of nine fractions collected from the supernatant using size exclusion chromatography produced the highest response when used to stimulate macrophages. The results of the dose-response study of the effect of the fraction with the highest stimulation effect on the production of TNF-α showed a direct correlation between protein concentration and TNF-α release. The fraction contained four novel peptides, three derived from the hydrolysis of β-casein and one from the hydrolysis of α-lactalbumin. These results confirm that fermentation of milk byLactobacillus helveticus(LH-2) results in the production of specific peptides capable of modulating macrophage activity.
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3

Jofre-Monseny, Laia, Sonia de Pascual-Teresa, Eva Plonka, et al. "Differential effects of apolipoprotein E3 and E4 on markers of oxidative status in macrophages." British Journal of Nutrition 97, no. 5 (2007): 864–71. http://dx.doi.org/10.1017/s0007114507669219.

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ApoE is secreted by macrophages at the lesion site of the atherosclerotic plaque, where it is thought to play a protective role against atherosclerosis independently of its effects on lipid metabolism. Of the three common isoforms for apoE, apoE4 is associated with higher risk of cardiovascular disease (CVD). In vitro studies have shown that recombinant apoE may act as an antioxidant in an isoform-dependent manner (E2&gt;E3&gt;E4). The oxidative status of the macrophages plays a key role in the process of atherosclerosis. In the present study the possible differential actions of apoE3 and apoE4 on several parameters of oxidative status were determined in stably transfected murine macrophages (RAW 264·7-apoE3 and -apoE4). No differences between genotypes were observed after peroxide challenge in either protection against cytotoxicity or in cell membrane oxidation, and modest differences were observed in the non-enzymatic antioxidants (glutathione and α-tocopherol) in apoE3 v. apoE4 macrophages. Importantly, cells secreting apoE4 showed increased membrane oxidation under basal conditions, and produced more NO and superoxide anion radicals than the apoE3 macrophages after stimulation. The present data suggest that apoE genotype influences the oxidative status of macrophages, and this could partly contribute to the higher CVD risk observed in apoE4 carriers.
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4

Tabouret, G., I. Vouldoukis, C. Duranton, et al. "Oestrus ovis (Diptera: Oestridae): effects of larval excretory/secretory products on nitric oxide production by murine RAW 264·7 macrophages." Parasite Immunology 23, no. 3 (2001): 111–19. http://dx.doi.org/10.1046/j.1365-3024.2001.00355.x.

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5

Chon, H., B. Choi, E. Lee, S. Lee, and G. Jeong. "Immunomodulatory effects of specific bacterial components ofLactobacillus plantarumKFCC11389P on the murine macrophage cell line RAW 264·7." Journal of Applied Microbiology 107, no. 5 (2009): 1588–97. http://dx.doi.org/10.1111/j.1365-2672.2009.04343.x.

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6

Koc, A., T. Ozkan, A. Z. Karabay, A. Sunguroglu, and F. Aktan. "Effect of L-carnitine on the synthesis of nitric oxide in RAW 264·7 murine macrophage cell line." Cell Biochemistry and Function 29, no. 8 (2011): 679–85. http://dx.doi.org/10.1002/cbf.1807.

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7

Keen, Imelda, Traian V. Chirila, Zeke Barnard, Z. Zainuddin, and Andrew K. Whittaker. "Degradable Hydrogels for Tissue Engineering - Part II: Responses of Fibroblasts and Macrophages to Linear PHEMA." Journal of Biomimetics, Biomaterials and Tissue Engineering 8 (November 2010): 91–104. http://dx.doi.org/10.4028/www.scientific.net/jbbte.8.91.

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A series of linear poly(2-hydroxyethyl methacrylate) (PHEMA) with defined molecular weights (MW) and narrow molecular distributions were synthesized by reversible addition-fragmentation chain transfer (RAFT) polymerization using cumyl dithiobenzoate (CDB) as a chain transfer agent. Murine fibroblasts (3T3) were exposed to eluates from various PHEMA samples, washed or unwashed, and with or without dithioester end groups. After 72 hrs in cell culture, no cytotoxic response was elicited by the polymer samples devoid of dithioester end groups, and which also underwent a thorough washing regime. Specimens throughout the entire MW range were internalized by a macrophage (cell line Raw 264), suggesting that such polymers can be used as models for studying the biodegradation of PHEMA.
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8

Tjandrawinata, R. R., L. Hawel, and C. V. Byus. "Regulation of putrescine export in lipopolysaccharide or IFN-gamma-activated murine monocytic-leukemic RAW 264 cells." Journal of Immunology 152, no. 6 (1994): 3039–52. http://dx.doi.org/10.4049/jimmunol.152.6.3039.

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Abstract The regulation of putrescine/polyamine export out of the cell was investigated during activation of monocytic-leukemic RAW 264 cells with LPS and IFN-gamma. The RAW 264 cells exported putrescine constitutively at a significant rate into the culture medium. This export process appeared to be selective for putrescine in that only a small amount of other polyamines (spermidine and N1-acetylspermidine) was found in the culture medium. LPS and IFN-gamma alone and in combination markedly stimulated putrescine export and nitrite production throughout a 24-h period. The efflux of putrescine but not nitrite was further increased by the addition of ornithine (the amino acid precursor of putrescine) to the culture medium. LPS and ornithine also stimulated the intracellular accumulation of putrescine in primary inflammatory macrophages and the export of putrescine into the peritoneal exudate of the mouse. A detailed comparison of the steady state rates of accumulation of intracellular putrescine/polyamines and the rate of putrescine efflux from the cells constitutively and after LPS, IFN-gamma, and ornithine indicated that a surprisingly large fraction of total polyamine biosynthesis is comprised of exported putrescine. The observed dose-dependent inhibition of putrescine export with the drug verapamil implicated the involvement of a specific membrane transport system sensitive to calcium influx in this process. The data are discussed in regard to the potential involvement of putrescine export in the regulation of intracellular polyamine levels, cell differentiation, and macrophage-mediated cytotoxicity.
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9

Homsani, Fortune, Gleyce Moreno, Camila Siqueira, Juliana Grechi, André Luis Santos, and Carla Holandino. "Cellular Alterations Induced by Candida albicans RC Nosodes: an in vitro Study." International Journal of High Dilution Research - ISSN 1982-6206 11, no. 40 (2021): 209–10. http://dx.doi.org/10.51910/ijhdr.v11i40.600.

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Introduction: Candidiasis is an opportunist infection, caused by yeast of the genus Candida, which emerges as one of the main causes of systemic infections in hospitalized patients. Candida albicans is the most common causing agent of these infections. According to the Brazilian Homeopathic Pharmacopeia[1], nosodes are medicines compounded from chemically undefined biological products. Living nosodes are prepared using the etiologic agent of an illness in its infective form, were first developed by Brazilian physician Roberto Costa (RC). Roberto Costa’s research indicated that living nosodes present a higher capability to stimulate the host’s immunological system [2]. &#x0D; Aim: This study aims to evaluate cellular alterations induced in C. albicans yeasts and RAW 264-7 macrophages by Candida albicans RC. &#x0D; Methodology: To prepare Candida albicans RC, one part of C. albicans infective yeast suspension (108 cell/ml) was diluted in 9 parts of sterile distilled water and submitted to 100 mechanical succussions. This process was successively repeated to the potencies of 12x and 30x1. Water 30x was prepared by the same technique, as control. The cell viability of C. albicans previously treated with nosodes in both potencies and respective controls was evaluated using the samples at the concentration of 10% (V/V), in a volume of 1ml, distributed in 1-3 days. The viability of the yeast cells was analyzed by MTT (3-(4,5-dimetiltiazol-2-il)-2,5-difeniltetrazolic) (5mg/ml) assay [3] and by Propidium Iodide (PI) incorporation methods. Additionally, using macrophages RAW 264-7 as a cell model, Nitric Oxide (NO) production and cell viability were also evaluated. For this, the following protocol of cell treatment was employed: on each experimental day, RAW 264-7 cells were treated 4 times (4 stimuli) with RC nosode 30x at the concentration of 10% (V/V). &#x0D; Results: The nosodes (12x and 30x) did not present cytotoxic effects on macrophage cells (n=1), or on C. albicans yeasts (n=2), as detected by MTT and PI methods. Moreover, no statistically significant differences on NO production were detected among the experimental groups (n=6). &#x0D; Conclusion: Preliminary results of in vitro assays indicate that nosodes (12x and 30x) do not alter mitochondrial activity or cell viability of C. albicans. Similarly, treatment by RC nosodes does not seem to alter NO release and mitochondrial activity of RAW macrophages. New experiments are being performed to confirm these preliminary data.
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10

Rao, P., L. A. Falk, S. F. Dougherty, T. Sawada, and D. H. Pluznik. "Colchicine down-regulates lipopolysaccharide-induced granulocyte-macrophage colony-stimulating factor production in murine macrophages." Journal of Immunology 159, no. 7 (1997): 3531–39. http://dx.doi.org/10.4049/jimmunol.159.7.3531.

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Abstract Activation of macrophages by LPS and taxol results in production of IL-1, IL-6, TNF-alpha, and granulocyte-macrophage CSF (GM-CSF), which are involved in regulating hemopoiesis, inflammation, and immune responses. Microtubules are proposed as a target site for LPS interaction(s), based on similarities between the effects of the tubulin-binding drug taxol and LPS. To clarify the role of microtubules in LPS-induced GM-CSF expression in macrophages, we examined whether microtubule depolymerizing agents affect GM-CSF production in macrophages. Pretreatment with colchicine impaired LPS induction of GM-CSF in RAW 264 cells, and studies using stable transfectants revealed that colchicine impaired the transcriptional responsiveness of a reporter gene driven by a GM-CSF promoter sequence. Colchicine inhibition of the GM-CSF response correlated with decreases in the mRNA levels of beta-tubulin; maximal inhibition of both events was observed 4 h after addition of colchicine. Microtubule agents inhibited LPS induction of IL-6 and TNF-alpha, while the induction of both IL-1beta and inducible nitric oxide synthase was unaltered, suggesting that LPS activates microtubule-dependent and -independent pathways. Interestingly, LPS stimulation of macrophages down-regulated levels of beta-tubulin transcripts, implying that LPS interacts with an element(s) of the microtubule network in vivo, activating pathways regulating transcription of beta-tubulin. The ability of both colchicine and LPS to modulate transcription of beta-tubulin suggests that this event does not per se underlie the inhibitory effect of colchicine on LPS-induced GM-CSF expression. These data led us to conclude that colchicine inhibits LPS induction of GM-CSF by affecting microtubule-dependent costimulatory signaling pathways that synergize with primary LPS-triggered responses.
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Dissertations / Theses on the topic "Murine macrophages (RAW 264.7)"

1

Alruwaili, Muhannad Falah. "The Impact of Cytokines and HSV-1 on Rab5 Protein Expression, F-actin Cytoskeleton Rearrangement, and Cell Viability of Uninfected and Virus-Infected M0, M1, and M2 RAW264.7 Murine Macrophages." Wright State University / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=wright1526015378786658.

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