Relevant bibliographies by topics / Murine sarcoma virus

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'Murine sarcoma virus'

Author: Grafiati
Published: 4 June 2021
Last updated: 12 February 2022

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Journal articles on the topic "Murine sarcoma virus"

1

van der Hoorn, F. A., and V. Müller. "Differential transformation of C3H10T1/2 cells by v-mos: sequential expression of transformation parameters." Molecular and Cellular Biology 5, no. 9 (1985): 2204–11. http://dx.doi.org/10.1128/mcb.5.9.2204.

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Extremely small quantities of the product of the transforming gene v-mos of Moloney murine sarcoma virus are able to efficiently transform cells. Recent data indicate the existence of a threshold level for v-mos transformation of NIH3T3 cells. Using mouse mammary tumor virus long terminal repeat sequences or hybrid promoters consisting of mouse mammary tumor virus and Moloney murine sarcoma virus long terminal repeat elements to express v-mos in C3H10T1/2 cells, we established cell lines representing different stages of morphological transformation in vitro. The threshold level for v-mos trans
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van der Hoorn, F. A., and V. Müller. "Differential transformation of C3H10T1/2 cells by v-mos: sequential expression of transformation parameters." Molecular and Cellular Biology 5, no. 9 (1985): 2204–11. http://dx.doi.org/10.1128/mcb.5.9.2204-2211.1985.

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Extremely small quantities of the product of the transforming gene v-mos of Moloney murine sarcoma virus are able to efficiently transform cells. Recent data indicate the existence of a threshold level for v-mos transformation of NIH3T3 cells. Using mouse mammary tumor virus long terminal repeat sequences or hybrid promoters consisting of mouse mammary tumor virus and Moloney murine sarcoma virus long terminal repeat elements to express v-mos in C3H10T1/2 cells, we established cell lines representing different stages of morphological transformation in vitro. The threshold level for v-mos trans
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3

Graves, B. J., S. P. Eisenberg, D. M. Coen, and S. L. McKnight. "Alternate utilization of two regulatory domains within the Moloney murine sarcoma virus long terminal repeat." Molecular and Cellular Biology 5, no. 8 (1985): 1959–68. http://dx.doi.org/10.1128/mcb.5.8.1959.

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The Moloney murine sarcoma virus long terminal repeat (LTR) harbors two distinct positive activators of transcription, namely, a distal signal and an enhancer. In this report we demonstrate that infection by herpes simplex virus (HSV) can markedly affect the utilization of these two Moloney murine sarcoma virus transcription signals. We investigated the HSV-mediated trans-acting effects with two goals in mind: first, to gain insight into LTR function, and second, to probe the mechanisms used by HSV to establish its own transcription cascade. In mock-infected cells, LTR-mediated expression was
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Graves, B. J., S. P. Eisenberg, D. M. Coen, and S. L. McKnight. "Alternate utilization of two regulatory domains within the Moloney murine sarcoma virus long terminal repeat." Molecular and Cellular Biology 5, no. 8 (1985): 1959–68. http://dx.doi.org/10.1128/mcb.5.8.1959-1968.1985.

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The Moloney murine sarcoma virus long terminal repeat (LTR) harbors two distinct positive activators of transcription, namely, a distal signal and an enhancer. In this report we demonstrate that infection by herpes simplex virus (HSV) can markedly affect the utilization of these two Moloney murine sarcoma virus transcription signals. We investigated the HSV-mediated trans-acting effects with two goals in mind: first, to gain insight into LTR function, and second, to probe the mechanisms used by HSV to establish its own transcription cascade. In mock-infected cells, LTR-mediated expression was
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5

Inayoshi, Yujin, Yuuki Okino, Katsuhide Miyake, et al. "Transcription Factor YY1 Interacts with Retroviral Integrases and Facilitates Integration of Moloney Murine Leukemia Virus cDNA into the Host Chromosomes." Journal of Virology 84, no. 16 (2010): 8250–61. http://dx.doi.org/10.1128/jvi.02681-09.

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ABSTRACT Retroviral integrases associate during the early viral life cycle with preintegration complexes that catalyze the integration of reverse-transcribed viral cDNA into the host chromosomes. Several cellular and viral proteins have been reported to be incorporated in the preintegration complex. This study demonstrates that transcription factor Yin Yang 1 binds to Moloney murine leukemia virus, human immunodeficiency virus type 1, and avian sarcoma virus integrases. The results of coimmunoprecipitation and in vitro pulldown assays revealed that Yin Yang 1 interacted with the catalytic core
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6

Lichtman, A. H., D. S. Reynolds, D. V. Faller, and A. K. Abbas. "Mature murine B lymphocytes immortalized by Kirsten sarcoma virus." Nature 324, no. 6096 (1986): 489–91. http://dx.doi.org/10.1038/324489a0.

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SCHULZE, FRANK, ERNST BOEHNLEIN, and PETER GRUSS. "Mutational Analyses of the Moloney Murine Sarcoma Virus Enhancer." DNA 4, no. 3 (1985): 193–202. http://dx.doi.org/10.1089/dna.1985.4.193.

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Escobedo, Jaime, Balraj Singh, Dino Dina, and Ralph B. Arlinghaus. "Temperature-dependent cytocidal effects of Moloney murine sarcoma virus." Virus Research 6, no. 1 (1986): 75–84. http://dx.doi.org/10.1016/0168-1702(86)90058-4.

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9

Hatziioannou, Theodora, and Stephen P. Goff. "Infection of Nondividing Cells by Rous Sarcoma Virus." Journal of Virology 75, no. 19 (2001): 9526–31. http://dx.doi.org/10.1128/jvi.75.19.9526-9531.2001.

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ABSTRACT A direct comparison demonstrates that Rous sarcoma virus is capable of infecting aphidicolin-arrested cells 10-fold more efficiently than murine leukemia virus but less efficiently than human immunodeficiency virus. The efficiency of infection of nondividing cells by the three viruses correlates with the respective ability of each viral DNA to enter the nucleus.
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Graves, B. J., R. N. Eisenman, and S. L. McKnight. "Delineation of transcriptional control signals within the Moloney murine sarcoma virus long terminal repeat." Molecular and Cellular Biology 5, no. 8 (1985): 1948–58. http://dx.doi.org/10.1128/mcb.5.8.1948.

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We identified three distinct elements within the Moloney murine sarcoma virus long terminal repeat that control transcription. The phenotypes of unidirectional deletion mutants of the long terminal repeat were assayed in microinjected frog oocytes and in transfected mouse fibroblasts. Steady-state levels of RNA bearing the same 5' terminus as the authentic Moloney murine sarcoma viral transcripts were measured by primer extension in assays that included a pseudo-wild-type internal reference. Mutant phenotypes define the boundaries of three functional elements. A region between 21 and 31 base p
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Dissertations / Theses on the topic "Murine sarcoma virus"

1

Souyri-Caporale, Michèle. "Etude du pouvoir tumorigene de l'oncogene n-ras." Paris 7, 1987. http://www.theses.fr/1987PA077083.

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Le, Bousse-Kerdiles Caroline. "Etude physiopathologique du syndrome myeloproliferatif provoque par le virus sarcomatogene myeloproliferatif murin : mise en evidence d'une activite stimulant la proliferation et la differenciation des cellules souches hematopoietiques pluripotentes." Paris 7, 1987. http://www.theses.fr/1987PA077220.

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Hamelin, Richard. "Le Virus du sarcome murin de Moloney ts110 un mutant thermosensible d'épissage." Grenoble 2 : ANRT, 1986. http://catalogue.bnf.fr/ark:/12148/cb37598218m.

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Book chapters on the topic "Murine sarcoma virus"

1

"Harvey Murine Sarcoma Virus Oncogene." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-46875-3_101053.

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"Harvey Murine Sarcoma Virus (transformation gene)." In Encyclopedia of Genetics, Genomics, Proteomics and Informatics. Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6754-9_7362.

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