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Journal articles on the topic 'Mutagenicity and genotoxicity'

Author: Grafiati

Published: 4 June 2021

Last updated: 10 February 2022

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1

Maurici, Daniela, Marilyn Aardema, Raffaella Corvi, et al. "3.7. Genotoxicity and Mutagenicity." Alternatives to Laboratory Animals 33, no. 1_suppl (2005): 117–30. http://dx.doi.org/10.1177/026119290503301s13.

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2

Chung, Youn-Son, Kazuhiro Ichikawa, and Hideo Utsumi. "Application of micronucleus in vitro assay to micropollutants in river water." Water Science and Technology 35, no. 8 (1997): 9–13. http://dx.doi.org/10.2166/wst.1997.0291.

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To determine the genotoxicity of river water towards mammalian cells, we applied Micronucleus in vitro test using mammalian cells to the samples taken from river Tamagawa located between Tokyo and Kanagawa prefecture. Water samples were condensed by Sep-pak cartridges and extracted by dichloromethane and methanol. Positive genotoxicity was observed in methanol extracts from sampling stations of Hinobashi and Marukobashi, while no dichloromethane extracts showed genotoxicity, suggesting that polar genotoxic micropollutants may be contained in the water of Tamagawa, at least in its down-stream.

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3

Soni, B. K., and J. P. Langan. "Mutagenicity and genotoxicity of ClearTaste." Toxicology Reports 5 (2018): 196–206. http://dx.doi.org/10.1016/j.toxrep.2017.12.015.

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4

Tobólska, Sylwia, Sylwia Terpiłowska, Jerzy Jaroszewski, and Andrzej Krzysztof Siwicki. "Genotoxicity and mutagenicity of inosine pranobex." Journal of Veterinary Research 62, no. 2 (2018): 207–13. http://dx.doi.org/10.2478/jvetres-2018-0030.

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AbstractIntroductionInosine pranobex (Methisoprinol, ISO, Isoprinosine) is an immuno-modulatory antiviral drug that has been licensed since 1971 in several countries worldwide. In humans, the drug is approved for the treatment of viral infections, and it might also have therapeutic use in animals. The aims of the presented work were to investigate the genotoxicity of inosine pranobex on BALB/3T3 clone A1 and HepG2 cell lines and to elucidate its mutagenicity using the Ames test.Material and MethodsThe BALB/3T3 clone A1 and HepG2 cells were incubated with inosine pranobex at concentrations from

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5

Otabe, Akira, Fumio Ohta, Asuka Takumi, and Barry Lynch. "Mutagenicity and genotoxicity studies of aspartame." Regulatory Toxicology and Pharmacology 103 (April 2019): 345–51. http://dx.doi.org/10.1016/j.yrtph.2018.01.023.

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6

Rossi, C., P. Poli, A. Buschini, et al. "Occupational genotoxicity assessment by mutagenicity assays." Toxicology Letters 77, no. 1-3 (1995): 289–98. http://dx.doi.org/10.1016/0378-4274(95)03309-2.

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7

Thiel, A., A. C. M. Schoenmakers, I. A. J. Verbaan, E. Chenal, S. Etheve, and P. Beilstein. "3-NOP: Mutagenicity and genotoxicity assessment." Food and Chemical Toxicology 123 (January 2019): 566–73. http://dx.doi.org/10.1016/j.fct.2018.11.010.

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8

Liman, Recep. "Mutagenicity and genotoxicity of dicapthon insecticide." Cytotechnology 66, no. 5 (2014): 741–51. http://dx.doi.org/10.1007/s10616-013-9623-x.

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9

Masood, Farhana, and Abdul Malik. "Mutagenicity and genotoxicity assessment of industrial wastewaters." Environmental Science and Pollution Research 20, no. 10 (2013): 7386–97. http://dx.doi.org/10.1007/s11356-013-1756-0.

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10

Илюшина, Н. А. "Systemic assessment of pesticide genotoxicity." Nauchno-prakticheskii zhurnal «Medicinskaia genetika», no. 9(218) (September 30, 2020): 67–69. http://dx.doi.org/10.25557/2073-7998.2020.09.67-69.

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Изучена генотоксичность более 200 технических продуктов (ТП) пестицидов. Разработан алгоритм оценки эквивалентности ТП оригинальным действующим веществам (ДВ) по критерию «мутагенность». Обоснована необходимость изучения генотоксической активности комбинаций ДВ пестицидов. Установлены ограничения тестов на генотоксичность, обусловленные токсичностью пестицидов разных химических классов. Выявлена зависимость ингибирующего эритропоэз действия триазоловых пестицидов от структуры ДВ. Изучено влияние и предложен общий механизм действия карбендазима на процессы кариокинеза, экструзии ядер и цитокине

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11

Charehsaz, M., FE Onen-Bayram, H. Sipahi, K. Buran, AK Giri, and A. Aydin. "Evaluation of the mutagenic and genotoxic effects of the ALC67 thiazolidine compound in Salmonella strains and human lymphocytes in vitro." Human & Experimental Toxicology 35, no. 10 (2016): 1108–15. http://dx.doi.org/10.1177/0960327115621365.

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ALC67 is an N-acylated thiazolidine compound with promising anticancer activity that led to the recent discovery of a series of 3-propionyl thiazolidine-4-carboxylic acid ethyl esters as a family of novel antiproliferative agents. Since the mutagenic and genotoxic properties of marketed anticancer molecules constitute a main issue to be addressed, this study focused on the analysis of the mutagenicity, antimutagenecity, and genotoxicity of this molecule. The mutagenicity and antimutagenicity of ALC67 were evaluated by Ames test performed on Salmonella TA98 and TA100 strains. The genotoxicity o

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12

Pruul, Reet, Hubert Kahn, and Anne Kahru. "Application of the Ames Genotoxicity Assay and the Bioluminescent Toxicity Assay in the Testing of Urine Samples." Alternatives to Laboratory Animals 21, no. 2 (1993): 225–32. http://dx.doi.org/10.1177/026119299302100218.

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Urine samples from ski factory, shoe factory and kindergarten workers were analysed using the Ames genotoxicity assay and Biotox™ toxicity test (BioOrbit, Turku, Finland), to screen the occupational exposure of these people to mutagenic and toxic chemicals. Different strains of Salmonella typhimurium were used for the screening of different groups. The assay was performed without S9 mix, i.e. directly acting mutagens were tested. The ski factory workers were grouped according to their specific tasks (compressors and polishers). The urinary mutagenicity of compressors analysed using Salmonella

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13

Pruul, Reet, Lars Nyland, Kimmo Peltonen, Marja Sorsa, and Toomas Veidebaum. "Environmental Genotoxicity in an Estonian Oil Shale Industrial Area." Alternatives to Laboratory Animals 24, no. 3 (1996): 419–22. http://dx.doi.org/10.1177/026119299602400317.

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The genotoxicity of environmental samples (ambient air, drinking and river waters, purified waste water and oil shale ash) from an oil shale mining and processing area was studied by using the Ames Salmonella/microsome assay. Salmonella typhimurium strains TA98 and YG1021 were used, with and without metabolic activation with rat liver homogenate S9. The water samples were treated with amberlite adsorbent XAD-2 for concentrating non-polar compounds. The air samples were collected on glass fibre filters by using a high volume air sampler, and extracted with dichloromethane by using a Soxhlet app

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14

Goncharuk, V. V., M. R. Vergolas, and I. V. Boltina. "Investigation of mutagenicity and genotoxicity of drinking water." Journal of Water Chemistry and Technology 35, no. 5 (2013): 238–42. http://dx.doi.org/10.3103/s1063455x1305007x.

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15

Terpilowska, S., and A. Siwicki. "The genotoxicity and mutagenicity of chromium and iron." Toxicology Letters 238, no. 2 (2015): S295—S296. http://dx.doi.org/10.1016/j.toxlet.2015.08.848.

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16

Pinhatti, Valéria Rodrigues, Juliana da Silva, Tales Leandro Costa Martins, et al. "Cytotoxic, mutagenicity, and genotoxicity effects of guanylhydrazone derivatives." Mutation Research/Genetic Toxicology and Environmental Mutagenesis 806 (August 2016): 1–10. http://dx.doi.org/10.1016/j.mrgentox.2016.06.001.

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17

More, Amol B., Mitesh D. Patel, Vinod C. Malshe, Padma V. Devarajan, and Geeta R. Vanage. "Genotoxicity and Mutagenicity Evaluation of Polyethylene Sebacate Nanoparticles." Journal of Nanopharmaceutics and Drug Delivery 1, no. 3 (2013): 301–10. http://dx.doi.org/10.1166/jnd.2013.1023.

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18

de Groene, Els M., Andrea Jahn, G. Jean Horbach, and Johanna Fink-Gremmels. "Mutagenicity and genotoxicity of the mycotoxin ochratoxin A." Environmental Toxicology and Pharmacology 1, no. 1 (1996): 21–26. http://dx.doi.org/10.1016/1382-6689(95)00005-4.

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19

Bomhard, E. M., J. N. Bremmer, and B. A. Herbold. "Review of the mutagenicity/genotoxicity of butylated hydroxytoluene." Mutation Research/Reviews in Genetic Toxicology 277, no. 3 (1992): 187–200. http://dx.doi.org/10.1016/0165-1110(92)90043-9.

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20

Parrella, Alfredo, Margherita Lavorgna, Emma Criscuolo, and Marina Isidori. "Mutagenicity, Genotoxicity, and Estrogenic Activity of River Porewaters." Archives of Environmental Contamination and Toxicology 65, no. 3 (2013): 407–20. http://dx.doi.org/10.1007/s00244-013-9928-y.

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21

Liu, Wen, Yang Yang, and Qing Fang Liu. "Genotoxicity Assessment of Advanced Composite Material – Silicone Gel-Filled Breast Implant after Radiation Sterilization." Advanced Materials Research 583 (October 2012): 95–99. http://dx.doi.org/10.4028/www.scientific.net/amr.583.95.

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The purpose of this study was to evaluate the genotoxicity of advanced composed material—silicone gel-filled breast implant after irradiation sterilization, which will provide basis for the safety of such medical material in clinical application. The irradiation dose for sterilization was set according to ISO11137-2 standard. Ames test, micronucleus test and chromosome aberration test were performed to detect the genotoxicity of silicone gel-filled breast implant. No genotoxicity, no mutagenicity and no significant increase in micronucleus rate in polychromatic erythrocytes of bone marrow were

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22

Hoshina, Márcia Miyuki, and Maria Aparecida Marin-Morales. "Anti-genotoxicity and anti-mutagenicity of Apis mellifera venom." Mutation Research/Genetic Toxicology and Environmental Mutagenesis 762 (March 2014): 43–48. http://dx.doi.org/10.1016/j.mrgentox.2013.11.005.

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23

Chen, Tai-I., Hui-Wen Zhuang, Yu-Chung Chiao, and Chin-Chu Chen. "Mutagenicity and genotoxicity effects of Lignosus rhinocerotis mushroom mycelium." Journal of Ethnopharmacology 149, no. 1 (2013): 70–74. http://dx.doi.org/10.1016/j.jep.2013.06.001.

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24

Ferrand, C., F. Marc, P. Fritsch, P. Cassand, and G. de Saint Blanquat. "Mutagenicity and genotoxicity of sorbic acid–amine reaction products." Toxicology in Vitro 14, no. 5 (2000): 423–28. http://dx.doi.org/10.1016/s0887-2333(00)00035-7.

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25

Deininger, Christoph, Erwin Eder, Tilmann Neudecker, and Christian Hoffman. "Mutagenicity and genotoxicity of ethylvinyl ketone in bacterial tests." Journal of Applied Toxicology 10, no. 3 (1990): 167–71. http://dx.doi.org/10.1002/jat.2550100305.

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26

A, A. Bakare, G. Alimba C, and A. Alabi O. "Genotoxicity and mutagenicity of solid waste leachates: A review." African Journal of Biotechnology 12, no. 27 (2013): 4206–20. http://dx.doi.org/10.5897/ajb2013.12014.

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27

Ferrand, Carine, FranÇoise Marc, Pierre Fritsch, Pierrette Cassand, and Georges Saint de Blanquat. "Mutagenicity and genotoxicity of sorbic acid-amine reaction products." Food Additives & Contaminants 17, no. 11 (2000): 895–901. http://dx.doi.org/10.1080/026520300750038063.

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28

Thompson, E. D., J. A. McDermott, T. B. Zerkle, J. A. Skare, B. L. B. Evans, and D. B. Cody. "Genotoxicity of zinc in 4 short-term mutagenicity assays." Mutation Research/Genetic Toxicology 223, no. 3 (1989): 267–72. http://dx.doi.org/10.1016/0165-1218(89)90119-5.

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29

Prá, Daniel, Silvia Isabel Rech Franke, Raquel Giulian, et al. "Genotoxicity and mutagenicity of iron and copper in mice." BioMetals 21, no. 3 (2007): 289–97. http://dx.doi.org/10.1007/s10534-007-9118-3.

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30

Chakraborty, Rajarshi, and Anita Mukherjee. "Mutagenicity and genotoxicity of coal fly ash water leachate." Ecotoxicology and Environmental Safety 72, no. 3 (2009): 838–42. http://dx.doi.org/10.1016/j.ecoenv.2008.09.023.

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31

Hsieh, MK, CL Shyu, JW Liao, et al. "Correlation analysis of heat stability of veterinary antibiotics by structural degradation, changes in antimicrobial activity and genotoxicity." Veterinární Medicína 56, No. 6 (2011): 274–85. http://dx.doi.org/10.17221/1548-vetmed.

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The relationship between the structural degradation of veterinary antibiotics, their antimicrobial activity, and possible mutagenicity after heating have not been well investigated sequentially. This study aimed to evaluate the heat stability of 14 veterinary antibiotics under a short-term heating scenario by characterization of their structural degradation and their relationship to resultant changes in antimicrobial activity. Mutagenicity was also examined in four representative antibiotics after 15-min-heat treatments at two temperatures (100 &deg;C and 121 &deg;C). Differential heat

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32

Kołodziejski, Dominik, Izabela Koss-Mikołajczyk, Ahmad Y. Abdin, Claus Jacob, and Agnieszka Bartoszek. "Chemical Aspects of Biological Activity of Isothiocyanates and Indoles, the Products of Glucosinolate Decomposition." Current Pharmaceutical Design 25, no. 15 (2019): 1717–28. http://dx.doi.org/10.2174/1381612825666190701151644.

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There is growing evidence that cancer chemoprevention employing natural, bioactive compounds may halt or at least slow down the different stages of carcinogenesis. A particularly advantageous effect is attributed to derivatives of sulfur-organic phytochemicals, such as glucosinolates (GLs) synthesized mainly in Brassicaceae plant family. GLs are hydrolysed enzymatically to bioactive isothiocyanates (ITC) and indoles, which exhibit strong anti-inflammatory and anti-carcinogenic activity. Highly bioavailable electrophilic ITC are of particular interest, as they can react with nucleophilic groups

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33

van Beerendonk, G. J. M., S. D. Nelson, and J. H. N. Meerman. "Metabolism and Genotoxicity of the Halogenated Alkyl Compound Tris(2,3-Dibromopropyl)phosphate." Human & Experimental Toxicology 13, no. 12 (1994): 861–65. http://dx.doi.org/10.1177/096032719401301208.

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1 The genotoxicity of the flame retardant tris(2,3-dibromopropyl)phosphate (Tris-BP) was studied in vivo. Results showed that Tris-BP was highly clasfogenic, but it could only initiate a low number of preneoplastic foci in the rat liver in vivo. In Drosophila, Tris-BP could be classified as a cross-linking agent, because it was more clastogenic than mutagenic. The use of completely deuterated Tris-BP as a metabolic probe revealed that cytochrome P450 and most likely the formation of 2-bromoacrolein (2BA) from Tris-BP is important for the observed genotoxic effects. 2 In contrast to the high mu

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34

Cândido-Bacani, Priscila de Matos, Mariana Bisarro dos Reis, Juliana Mara Serpeloni, et al. "Mutagenicity and genotoxicity of isatin in mammalian cells in vivo." Mutation Research/Genetic Toxicology and Environmental Mutagenesis 719, no. 1-2 (2011): 47–51. http://dx.doi.org/10.1016/j.mrgentox.2010.11.006.

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35

Claxton, Larry D., Peggy P. Matthews, and Sarah H. Warren. "The genotoxicity of ambient outdoor air, a review: Salmonella mutagenicity." Mutation Research/Reviews in Mutation Research 567, no. 2-3 (2004): 347–99. http://dx.doi.org/10.1016/j.mrrev.2004.08.002.

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36

Alabi, Okunola A., and Adekunle A. Bakare. "Genotoxicity and mutagenicity of electronic waste leachates using animal bioassays." Toxicological & Environmental Chemistry 93, no. 5 (2011): 1073–88. http://dx.doi.org/10.1080/02772248.2011.561949.

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37

Vidal, Leonardo S., Adriana M. Alves, Ricardo M. Kuster, Claudia Lage, and Alvaro C. Leitão. "Genotoxicity and mutagenicity of Echinodorus macrophyllus (chapéu-de-couro) extracts." Genetics and Molecular Biology 33, no. 3 (2010): 549–57. http://dx.doi.org/10.1590/s1415-47572010005000060.

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38

Guan, Davy, Kevin Fan, Ian Spence, and Slade Matthews. "QSAR ligand dataset for modelling mutagenicity, genotoxicity, and rodent carcinogenicity." Data in Brief 17 (April 2018): 876–84. http://dx.doi.org/10.1016/j.dib.2018.01.077.

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39

Hamad Mohamed, Hanan Ramadan. "Daily used silver nanoparticles induced persistent accumulative genotoxicity and mutagenicity." Recent Research in Genetics and Genomics 2019, no. 1 (2019): 48–55. http://dx.doi.org/10.21608/rrgg.2019.56368.

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40

Reifferscheid, Georg, and Britta v. Oepen. "Genotoxicity and Mutagenicity of Suspended Particulate Matter of River Water and Waste Water Samples." Scientific World JOURNAL 2 (2002): 1036–39. http://dx.doi.org/10.1100/tsw.2002.206.

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Suspended particulate matter of samples of river water and waste water treatment plants was tested for genotoxicity and mutagenicity using the standardized umu assay and two versions of the Ames microsuspension assay. The study tries to determine the entire DNA-damaging potential of the water samples and the distribution of DNA-damaging substances among the liquid phase and solid phase. Responsiveness and sensitivity of the bioassays are compared.

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41

Karlina, Irene, Rahmi Amtha, Boedi Oetomo Roeslan, and Yuniar Zen. "The Release of Total Metal Ion and Genotoxicity of Stainless Steel Brackets: Experimental Study Using Micronucleus Assay." Indonesian Biomedical Journal 8, no. 2 (2016): 97. http://dx.doi.org/10.18585/inabj.v8i2.193.

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BACKGROUND: Stainless steel brackets are composed of various metal that may corrode in oral cavity. Corrosion is caused by the release of metal ions such as chromium, nickel, and iron. The release of metal ions can cause adverse effects such as toxicity, allergic, and mutagenicity. To evaluate the biocompatibility of stainless steel brackets, micronucleus assay as one of genotoxicity assay is used in this study. To determine the differences and the correlation of metal ions release and genotoxic activity among three brand stainless steel brackets.METHODS: Three brands of brackets were immersed

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42

Cho, Chang-Won, Young-Ran Song, Won-Chul Lim, et al. "Acute Oral Toxicity and Genotoxicity of Polysaccharide Fraction from Young Barley Leaves (Hordeum vulgare L.)." Foods 9, no. 6 (2020): 809. http://dx.doi.org/10.3390/foods9060809.

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Polysaccharides isolated from various plants are considered precious bioactive materials owing to their potent biological activities. Previously, we prepared a polysaccharide fraction (BLE0) isolated from young barley leaves (Hordeum vulgare L.), demonstrating its anti-osteoporotic and immunostimulatory activities. However, data regarding BLE0 toxicity is lacking. To establish its safety, in vitro genotoxicity (chromosomal aberration and bacterial reverse mutation assays) and acute oral toxicity assays were conducted. In the in vitro genotoxicity assays, bacterial reverse mutation and chromoso

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43

Chłopkiewicz, B., A. Ejchart, and J. Marczewska. "Studies on the mechanism of mutagenicity and genotoxicity induced by dihydralazine." Acta Biochimica Polonica 42, no. 3 (1995): 291–95. http://dx.doi.org/10.18388/abp.1995_4623.

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Dihydralazine was found to be mutagenic towards S. typhimurium TA1537, TA97, TA1538 and TA98 and genotoxic towards E. coli PQ37. Using the nitro blue tetrazolium reduction method we have found that dihydralazine can generate active oxygen species. The possible role of active oxygen species in mutagenicity (Ames test) and genotoxicity (SOS Chromotest) of dihydralazine was studied by testing the influence of the different active oxygen species scavengers on these two processes. Of the active oxygen scavengers tested, only superoxide dismutase suppressed partially the mutagenic and genotoxic acti

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44

Yujra, Veronica Quispe, Eduardo Gregolin Moretti, Samuel Rangel Claudio, et al. "Genotoxicity and mutagenicity induced by acute crack cocaine exposure in mice." Drug and Chemical Toxicology 39, no. 4 (2015): 388–91. http://dx.doi.org/10.3109/01480545.2015.1126843.

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45

Ejchart, A., and J. Koziorowska. "Strain-dependent differences in mutagenicity and genotoxicity of cyclophosphamide in mice." Acta Biochimica Polonica 40, no. 1 (1993): 54–56. http://dx.doi.org/10.18388/abp.1993_4844.

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46

Ruiz, M. J., and D. Marzin. "Genotoxicity of six pesticides by Salmonella mutagenicity test and SOS chromotest." Mutation Research/Genetic Toxicology and Environmental Mutagenesis 390, no. 3 (1997): 245–55. http://dx.doi.org/10.1016/s1383-5718(97)00021-1.

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47

Mersch-Sundermann, V., K. H. Hauff, and P. Braun. "Mutagenicity, genotoxicity and structure-activity of mono-, poly- and heterocyclic aminoaromates." Mutation Research/Environmental Mutagenesis and Related Subjects 360, no. 3 (1996): 218. http://dx.doi.org/10.1016/s0165-1161(96)90050-3.

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48

Ashby, John. "Gonadal genotoxicity assays as practical surrogates for germ-cell mutagenicity assays." Environmental Mutagenesis 7, no. 3 (1985): 263–66. http://dx.doi.org/10.1002/em.2860070303.

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49

Mansour, Hedi Ben, Ridha Mosrati, David Corroler, Kamel Ghedira, Daniel Barillier, and Leila Chekir-Ghedira. "Mutagenicity and genotoxicity of acid yellow 17 and its biodegradation products." Drug and Chemical Toxicology 32, no. 3 (2009): 222–29. http://dx.doi.org/10.1080/01480540902862269.

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50

Fujita, Hiroyuki, and Tomohide Yamagami. "Absence of Mutagenicity, Genotoxicity, and Subchronic Oral Toxicity of Touchi Extract." International Journal of Toxicology 26, no. 5 (2007): 465–73. http://dx.doi.org/10.1080/10915810701620374.

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Touchi, a traditional Chinese food used mainly for seasoning is obtained by first steaming soybeans followed by fermentation with Aspergillus oryzae (koji). A series of toxicological studies was conducted to evaluate the mutagenic and genotoxic potential and subchronic toxicity of a water extract of Touchi, a known inhibitor of α-glucosidase activity. Touchi extract (TE) did not induce reverse mutations in Salmonella typhimurium strains TA98, TA1537, TA100, TA1535, and Escherichia coli WP2uvrA at concentrations up to 5000 μg/plate, in either the absence or presence of exogenous metabolic activ

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