Relevant bibliographies by topics / Myalgi

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  2. Dissertations / Theses
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  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Myalgi'

Author: Grafiati
Published: 4 June 2021
Last updated: 6 February 2022

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Journal articles on the topic "Myalgi"

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Smith, Jerry D., Ku-Lang Chang, and John G. Gums. "Possible Lansoprazole-Induced Eosinophilic Syndrome." Annals of Pharmacotherapy 32, no. 2 (February 1998): 196–200. http://dx.doi.org/10.1345/aph.17190.

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OBJECTIVE: To report a case of myalgia with eosinophilia related to lansoprazole administration. CASE SUMMARY: A 50-year-old white woman developed severe myalgia 1 week after starting lansoprazole. During the treatment course, the patient was also found to have eosinophilia. The myalgia and eosinophilia resolved 40 days after lansoprazole was stopped and 18 days after prednisone therapy was begun. The patient was not rechallenged with lansoprazole. DISCUSSION: To our knowledge, this is the first reported case of lansoprazole-induced eosinophilic syndrome. Clinically, it is difficult to distinguish between eosinophilia–myalgia syndrome and eosinophilic fasciitis, which are probably part of a continuum of eosinophilic disorders. This patient presented with symptoms of both syndromes. Although other causes cannot be completely ruled out, the time course strongly suggests that lansoprazole was the causative agent. CONCLUSIONS: It is important to consider medications when diagnosing patients with hypereosinophilia and/or myalgia. OBJETIVO: Informar un caso de mialgia con eosinofilia relacionado con la administración de lansoprazol. RESUMEN DEL CASO: Una mujer de 50 años desarrolló una mialgia severa 1 semana después de empezar terapia con lansoprazol. Además se encontró que diagnosticaba de eosinofilia. La mialgia y la eosinofilia se resolvieron 40 días después de detener la terapia con lansoprazol y 18 días después de empezar terapia con prednisona. No se le volvió a administrar lansoprazol a la paciente. DISCUSIÓN: Según nuestro conocimiento, este es el primer caso informado del síndrome de eosinofilia inducida por lansoprazol. Clínicamente, es difícil distinguir entre el síndrome de eosinofilia-mialgia y fascitis eosinofilica, las que probablemente forman parte de desórdenes eosinofílicos. El paciente presentaba síntomas similares a ambos síndromes. Aunque no se pueden eliminar otras causas, el curso clínico de este caso sugiere que lansoprazol era el agente causante. CONCLUSIONES: Es importante tomar en consideración los medicamentos cuando se diagnostican pacientes con hipereosinofilia y/o mialgia. OBJECTIF: Présenter un cas de myalgie avec éosinophile associée à l'administration de lansoprazole. RÉSUMÉ DU CAS: Une patiente de 50 ans, a développé des myalgies importantes 1 semaine après le début d'un traitement au lansoprazole. On a aussi découvert une éosinophilie chez cette patiente. Les myalgies et l'éosinophilie ont disparu 40 jours après l'arrêt du lansoprazole et 18 jours après le début d'un traitement à la prednisone. La patiente n'a pas été réexposée au lansoprazole. DISCUSSION: Ceci est le premier cas de syndrome d'éosinophilie induit par le lansoprazole rapporté dans la littérature. En clinique, il est difficile de distinguer entre un syndrome éosinophilie-myalgie et une fasciite à éosinophiles. Ces deux diagnostics faisant probablement partie d'un continuum de maladies touchant les éosinophiles. Cette patiente s'est présentée avec des symptômes compatibles avec les deux diagnostics. Bien que d'autres causes n'aient pas été complètement éliminées, l'évolution temporelle suggère fortement un lien de causalité avec le lansoprazole. CONCLUSIONS: Il demeure important de suspecter la médication lorsqu'on doit évaluer un patient qui se présente avec une hyperéosinophilie et/ou une myalgie.
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Sjøgaard, Gisela, Mette K. Zebis, Kristian Kiilerich, Bengt Saltin, and Henriette Pilegaard. "Exercise Training and Work Task Induced Metabolic and Stress-Related mRNA and Protein Responses in Myalgic Muscles." BioMed Research International 2013 (2013): 1–12. http://dx.doi.org/10.1155/2013/984523.

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The aim was to assess mRNA and/or protein levels of heat shock proteins, cytokines, growth regulating, and metabolic proteins in myalgic muscle at rest and in response to work tasks and prolonged exercise training. A randomized controlled trial included 28 females with trapezius myalgia and 16 healthy controls. Those with myalgia performed ~7 hrs repetitive stressful work and were subsequently randomized to 10 weeks of specific strength training, general fitness training, or reference intervention. Muscles biopsies were taken from the trapezius muscle at baseline, after work and after 10 weeks intervention. The main findings are that the capacity of carbohydrate oxidation was reduced in myalgic compared with healthy muscle. Repetitive stressful work increased mRNA content for heat shock proteins and decreased levels of key regulators for growth and oxidative metabolism. In contrast, prolonged general fitness as well as specific strength training decreased mRNA content of heat shock protein while the capacity of carbohydrate oxidation was increased only after specific strength training.
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Byrne, Edward. "Idiopathic chronic fatigue and myalgia syndrome (myalgic encephalomyelitis): some thoughts on nomenclature and aetiology." Medical Journal of Australia 148, no. 2 (January 1988): 80–82. http://dx.doi.org/10.5694/j.1326-5377.1988.tb104513.x.

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Palmio, Johanna, Satu Sandell, Michael G. Hanna, Roope Männikkö, Sini Penttilä, and Bjarne Udd. "Predominantly myalgic phenotype caused by the c.3466G>A p.A1156T mutation in SCN4A gene." Neurology 88, no. 16 (March 22, 2017): 1520–27. http://dx.doi.org/10.1212/wnl.0000000000003846.

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Objective:To characterize the clinical phenotype in patients with p.A1156T sodium channel mutation.Methods:Twenty-nine Finnish patients identified with the c.3466G>A p.A1156T mutation in the SCN4A gene were extensively examined. In a subsequent study, 63 patients with similar myalgic phenotype and with negative results in myotonic dystrophy type 2 genetic screening (DM2-neg group) and 93 patients diagnosed with fibromyalgia were screened for the mutation. Functional consequences of the p.A1156T mutation were studied in HEK293 cells with whole-cell patch clamp.Results:The main clinical manifestation in p.A1156T patients was not myotonia or periodic paralysis but exercise- and cold-induced muscle cramps, muscle stiffness, and myalgia. EMG myotonic discharges were detected in most but not all. Electrophysiologic compound muscle action potentials exercise test showed variable results. The p.A1156T mutation was identified in one patient in the DM2-neg group but not in the fibromyalgia group, making a total of 30 patients so far identified. Functional studies of the p.A1156T mutation showed mild attenuation of channel fast inactivation.Conclusions:The unspecific symptoms of myalgia stiffness and exercise intolerance without clinical myotonia or periodic paralysis in p.A1156T patients make the diagnosis challenging. The symptoms of milder SCN4A mutations may be confused with other similar myalgic syndromes, including fibromyalgia and myotonic dystrophy type 2.
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O-Lee, Tsungju, Charles E. Stewart, Loren Seery, and Christopher A. Church. "Fluoroquinolone-Induced Arthralgia and Myalgia in the Treatment of Sinusitis." American Journal of Rhinology 19, no. 4 (July 2005): 395–99. http://dx.doi.org/10.1177/194589240501900413.

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Background Because of their broad-spectrum coverage, fluoroquinolone antibiotics are widely used in the treatment of acute sinusitis and acute exacerbations of chronic sinusitis. Generally, they are well tolerated, and adverse effects are usually mild. In our experience with quinolones, patients have frequently complained of arthralgias and/or myalgias. Although tendon rupture has been described as one of the more severe side effects, there are few published reports of arthralgias and/or myalgias associated with these drugs. Methods A retrospective review of fluoroquinolone prescriptions in a tertiary rhinology clinic was completed. Patients treated with oral levofloxacin for sinusitis over a 12-month period were contacted by mail and asked to complete an anonymous survey regarding adverse effects. Of 81 patients identified, 36 responses were received. Results Among respondents, the incidence of arthralgias and/or myalgias was 25%, which was more than twice the incidence of any other adverse effect reported. Occurrence of arthralgias and/or myalgias did not appear to be influenced by respondent age, course length, concurrent use of oral steroids, or a history of arthritis. Among those reporting arthralgia or myalgia, symptoms began after an average of 3 days of therapy and resolved an average of 7.5 days after cessation of treatment. Fourteen percent of respondents were unable to complete their course of therapy because of arthralgias and/or myalgias. Conclusion Although effective and generally well tolerated in the treatment of sinusitis, fluoroquinolone antibiotics may produce adverse effects of arthralgia and/or myalgia.
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Schor, Anna M., and Ann Hellerstein. "Rhabdomyolysis Following a Short Course of Clarythromycin." Journal of Pediatric Pharmacology and Therapeutics 16, no. 3 (July 1, 2011): 216–17. http://dx.doi.org/10.5863/1551-6776-16.3.216.

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A healthy 5-year-old girl developed myalgia and an elevated creatine kinase concentration after 4 days of normal doses of clarithromycin. Myalgias resolved and creatine kinase values returned to normal following discontinuation of clarithromycin. No other precipitating factors or interacting drugs were identified; hence, clarithromycin was suspected as the cause of the rhabdomyolysis.
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Oliver, Nathan D., Auleen Millar, and Adrian Pendleton. "A Case Report on Parvovirus B19 Associated Myositis." Case Reports in Rheumatology 2012 (2012): 1–2. http://dx.doi.org/10.1155/2012/250537.

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Introduction. Whilst there are reports of viral myopathies affecting children and the immunocompromised, infective myositis is a relatively rare inflammatory myopathy in adults. The clinical spectrum can range from benign myalgias to more serious complications in certain risk groups.Case Presentation. We present two cases of myositis as a result of parvovirus B19 infection.Conclusion. Viral myositis and parvovirus B19 associated myositis should be considered in adults presenting with significant myalgia.
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Jensen, L., L. L. Andersen, H. D. Schrøder, U. Frandsen, and G. Sjøgaard. "Neuronal Nitric Oxide Synthase Is Dislocated in Type I Fibers of Myalgic Muscle but Can Recover with Physical Exercise Training." BioMed Research International 2015 (2015): 1–11. http://dx.doi.org/10.1155/2015/265278.

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Trapezius myalgia is the most common type of chronic neck pain. While physical exercise reduces pain and improves muscle function, the underlying mechanisms remain unclear. Nitric oxide (NO) signaling is important in modulating cellular function, and a dysfunctional neuronal NO synthase (nNOS) may contribute to an ineffective muscle function. This study investigated nNOS expression and localization in chronically painful muscle. Forty-one women clinically diagnosed with trapezius myalgia (MYA) and 18 healthy controls (CON) were included in the case-control study. Subsequently, MYA were randomly assigned to either 10 weeks of specific strength training (SST,n=18), general fitness training (GFT,n=15), or health information (REF,n=8). Distribution of fiber type, cross-sectional area, and sarcolemmal nNOS expression did not differ between MYA and CON. However, MYA showed increased sarcoplasmic nNOS localization (18.8 ± 12 versus 12.8 ± 8%,P=0.049) compared with CON. SST resulted in a decrease of sarcoplasm-localized nNOS following training (before 18.1 ± 12 versus after 12.0 ± 12%;P=0,027). We demonstrate that myalgic muscle displays altered nNOS localization and that 10 weeks of strength training normalize these disruptions, which supports previous findings of impaired muscle oxygenation during work tasks and reduced pain following exercise.
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Roy, E. Pierre, and Ludwig Gutmann. "Myalgia." Neurologic Clinics 6, no. 3 (August 1988): 621–36. http://dx.doi.org/10.1016/s0733-8619(18)30864-8.

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Gerwin, Robert D., Barbara Cagnie, Mirko Petrovic, Jo Van Dorpe, Patrick Calders, and Kayleigh De Meulemeester. "Foci of Segmentally Contracted Sarcomeres in Trapezius Muscle Biopsy Specimens in Myalgic and Nonmyalgic Human Subjects: Preliminary Results." Pain Medicine 21, no. 10 (April 14, 2020): 2348–56. http://dx.doi.org/10.1093/pm/pnaa019.

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Abstract Objective The myofascial trigger point hypothesis postulates that there are small foci of contracted sarcomeres in resting skeletal muscle. Only one example, in canine muscle, has been published previously. This study evaluated human muscle biopsies for foci of contracted sarcomeres. Setting The Departments of Rehabilitation Sciences and Physiotherapy at Ghent University, Ghent, Belgium. Subjects Biopsies from 28 women with or without trapezius myalgia were evaluated, 14 in each group. Methods Muscle biopsies were obtained from regions of taut bands in the trapezius muscle and processed for light and electron microscopy and for histochemical analysis. Examination of the biopsies was blinded as to group. Results A small number of foci of segmentally contracted sarcomeres were identified. One fusiform segmental locus involved the entire muscle fiber in tissue from a myalgic subject. Several transition zones from normal to contracted sarcomeres were found in both myalgic and nonmyalgic subjects. The distance between Z-lines in contracted sarcomeres was about 25–45% of the same distance in normal sarcomeres. Z-lines were disrupted and smeared in the contracted sarcomeres. Conclusions A small number of foci of segmentally contracted sarcomeres were found in relaxed trapezius muscle in human subjects, a confirmation of the only other example of spontaneous segmental contraction of sarcomeres (in a canine muscle specimen), consistent with the hypothesis of trigger point formation and with the presence of trigger point end plate noise.
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Dissertations / Theses on the topic "Myalgi"

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Pettersson, Maria. "Förebyggande av postoperativ myalgi." Thesis, Halmstad University, School of Social and Health Sciences (HOS), 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:hh:diva-5222.

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Succinylcolin är ett icke-depolariserande muskelrelaxantia som används inom anestesisjukvård. En vanlig biverkning är postoperativ myalgi. Varför smärtan uppstår är inte helt klarlagd. Under många år har forskare runt om i världen försökt komma till rätta med problemet utan att helt lyckas.Olika läkemedel och strategier har prövats. En av de viktigaste uppgifter en sjuksköterska har är att förebygga och lindra lidande. Som anestesisjuksköterska finns det möjlighet att påverka den vård som ordineras. Syftet med studien var att undersöka vilka metoder som kan förebygga postoperativ myalgi orsakad av succinylcolin. En litteraturstudie baserad på tio vetenskapliga artiklar genomfördes. Resultatet visade att parecoxib preoperativt samt premedicinering med diklofenakplåster gav det bästa resultatet när det gäller reducerande av myalgi. Med hjälp av dessa så vanliga läkemedel kan onödigt lidande förebyggas och samhällsekonomiska resurser sparas.

 

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Jansson, Helena. "Har Q10 någon effekt på smärtan vid statininducerad myopati/myalgi?" Thesis, Linnéuniversitetet, Institutionen för kemi och biomedicin (KOB), 2018. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-72831.

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Sammanfattning Bakgrund:Koenzym Q10 (Q10) syntetiseras i kroppen och återfinns med de högsta koncentrationerna i hjärta, lever och immunförsvarets celler. Det har en viktig roll när det agerar transportör av elektroner i andningskedjan. Q10 har även antioxiderande egenskaper, stärker cellmembran och motverkar fettsyraoxidation i mitokondrien och cellmembran. Nyare studier visar att ett tillskott av Q10 även förbättrar symptomen vid kronisk hjärtsvikt och minskar kardiovaskulära händelser.Vid förhöjda blodfetter är det mest använda läkemedlet statiner. De hämmar kolesterolsyntesen i levern och de påverkar även syntesen av Q10 vilken också sker mevalonatvägen. En av de vanligaste biverkningarna vid statinbehandling är muskelbesvären myopati och myalgi. Studier visar att dåligt fungerande mitokondrier och reducering av Q10 i serum kan relateras till statinbehandling. Syfte:Syftet med detta arbete har varit att undersöka om ett tillskott av Q10 har någon reducerande effekt på smärtan vid statininducerad myopati (SIM).  Metod:Denna litteraturstudie baserades på sex vetenskapliga artiklar sökta via PubMed. Studierna skulle vara randomiserade, dubbelblinda kliniska studier gjorda på människor. Av sökningens 10 träffar erhölls 6 artiklar vilka var relevanta för att besvara denna studiens frågeställning. Resultat:Resultatet visade att Q10 inte har någon större effekt på smärtan vid SIM. Endast 2 av 6 studier visar att Q10 har en god effekt.  Slutsats: För att kunna dra slutsats om Q10 har effekt eller inte vid SIM behövs fler randomiserade placebokontrollerade studier med jämförbara grupper och fler antal deltagare.
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Nygren, Karin, and Charlotte Glimstedt. "Pain modulation in patients with chronic lumbar myalgia : An experimental study." Thesis, Linnéuniversitetet, Institutionen för idrottsvetenskap (ID), 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-26833.

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Syfte: Syftet med denna studie var att undersöka hur statisk muskelkontraktion och cold pressor test påverkar kroppsegna smärtreglerande system (”Exercise induced analgesia” (EIA) och ”Conditioned pain modulation” (CPM)) hos patienter med kronisk ländryggssmärta kännetecknad av lumbal myalgi (LM) jämfört med friska kontroller. Försökspersoner och metod: Tjugosex friska köns- och åldersmatchade personer och tjugosex LM-patienter deltog. De utförde standardiserad statisk muskelkontraktion med m. Erector spinae (ME) i form av rygglyft och kontraktion av m. Quadriceps femoris (MQ) i form av knäledsextension. För att bedöma CPM användes sk cold pressor test. Smärttrösklar för tryck (PPTs) mättes över m. Deltoideus (MD), m. Erector spinae (ME) samt över m. Quadriceps (MQ) i vila och under resp. efter kontraktionen/cold pressor test. Under kontraktion mättes PPTs över den arbetande muskeln respektive över de två vilande musklerna. Dessutom undersöktes PPTs och känsligheten för övertrösklig trycksmärta (P7) i vila på 8 olika punkter på kroppen. Resultat: Kvinnliga LM-patienter hade ökad känslighet för trycksmärta (PPT) och övertrösklig trycksmärta (P7) jämfört med köns- och åldermatchade friska kontroller, medan manliga LM-patienter paradoxalt nog hade minskad känslighet för övertrösklig trycksmärta. Beträffande EIA fann vi att LM-patienter och kontroller kunde aktivera lokal EIA under kontraktion med ME. Vi fann dessutom en minskad förmåga hos LM-patienter att rekrytera generaliserad EIA under kontraktion med MQ. Slutligen hade LM-patienterna en normal funktion av CPM. Slutsats: LM-patienter kunde aktivera lokal EIA under kontraktion av ME, men hade mindre effektiv generaliserad EIA jämfört med kontrollerna, trots normal funktion av CPM. Våra resultat tyder på att muskelarbete med smärtande ryggmuskler skulle kunna användas för att minska smärtkänslighet i det drabbade området.
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Olivier, Catherine. "Le syndrome "éosinophilie-myalgies" lié à la consommation de L. Tryptophane : à propos d'une observation." Montpellier 1, 1992. http://www.theses.fr/1992MON11084.

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Ross, Jessica L. "Peripheral Mechanisms of Ischemic Myalgia." University of Cincinnati / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1504803397585968.

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Frey, Daniela. "An experimental model of chronic myalgia." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=66971.

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Objective: To develop a model of chronic jaw muscle allodynia using acidic saline infusion in humans. Methods: Buffered acidic (pH 5.1) or normal (pH 7.2) saline was infused into the masseter of 20 healthy subjects for 20 minutes. Sensory and motor tasks were conducted prior to infusion and 1, 24, 48, and 72 hours post-infusion. Results: Acidic saline induced mild pain during infusion, discomfort during motor tasks and mild allodynia that lasted for at least 24 hours. These effects were not different from those produced by normal saline. Sensory intensity during infusion was associated with levels of allodynia and sensory intensity during motor tasks for up to 3 days. Conclusion: Infusion of buffered solutions at pH 5.1 or 7.2 into the masseter produce mild pain and long-lasting allodynia and discomfort during jaw function. The intensity of these after-effects was strongly associated with the sensory intensity evoked by the infusion.
Objectif: Le but de cette étude est de développer un modèle humain d'allodynie chronique des muscles de la mâchoire grâce à une perfusion de solution saline acide. Méthodes : Nous avons fait une perfusion pendant 20 minutes soit d'une solution tampon acide (pH 5.1) ou d'une solution saline (pH 7.2) au niveau du masséter chez 20 individus en santé. Des tâches motrices et sensorielles ont été effectuées avant et 1h, 24h, 48h et 72 heures après l'injection. Résultats: La solution tampon acide a induit une douleur modérée pendant la perfusion, un inconfort durant les tâches motrices et de l'allodynie pendant au moins 24 heures. Ces effets ne sont pas différents de ceux produit par l'injection de solution saline. L'intensité sensorielle ressentie pendant la perfusion est associée au niveau d'allodynie et à l'intensité sensorielle au cours des tâches motrices au cours des 3 jours suivants. Conclusion: La perfusion de solution tampon acide (pH 5.1) ou saline (pH 7.2) dans le muscle masséter a produit une douleur modérée, de l'inconfort et de l'allodynie persistante lors du fonctionnement de la mâchoire. L'intensité de ces répercussions est fortement liée à l'intensité sensorielle évoquée par la perfusion.
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Roumier, Anne-Sophie. "Rôle des chimiokines et de leurs récepteurs dans les hyperéosinophilies." Lille 2, 2003. http://www.theses.fr/2003LIL2P011.

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Mörk, Camilla, and Dahlberg Linda Welin. "Att leva med myalgisk encefalomyelit." Thesis, Sophiahemmet Högskola, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:shh:diva-3854.

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Bakgrund Myalgisk encefalomyelit är en komplex, kronisk, neurologisk sjukdom som kännetecknas av långvarig psykisk och fysisk utmattning. Detta tillsammans med många andra symtom som smärta, yrsel och influensaliknande symtom, där symtomen förvärras vid ansträngning. Sjukdomens etiologi och patofysiologi är fortfarande okänd vilket leder till misstro och missuppfattningar från sjukvården för personen som lever med myalgisk encefalomyelit. Syfte Syftet var att belysa personers erfarenheter av att leva med myalgisk encefalomyelit. Metod Litteraturöversikten är baserad på 18 vetenskapliga kvalitativa och kvantitativa originalartiklar. Dessa återfanns i databaserna CINAHL, PubMed och PsycINFO. De kvalitetsgranskades enligt Sophiahemmet Högskolas bedömningsunderlag modifierat utifrån Berg et al. (1999) och Willman et al. (2011). Dataanalysen gjordes med inspiration från Kristenssons (2014) integrerade analys. Resultat Tre kategorier framträdde i resultatet med utgångspunkt i personernas erfarenhet av: mötet med sjukvården, påverkan på det dagliga livet samt copingstrategier och framtidstro. Resultatet visade att personerna hade erfarenheter av brister i mötet med sjukvården, social isolering och resultatet visade vikten av att ha strategier för att hantera sin vardag. Slutsats Litteraturöversiktens fynd visar vad det innebär för personen att leva med myalgisk encefalomyelit och hur det är att dagligen påverkas av de karaktäristiska symtomen fatigue, smärta och i och med det isolering. Samtidigt har behovet synliggjorts av ökad kunskap, rättvis vård och bättre behandling.
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Andersson, Daniel, and Emma Hellmark. "Arbetsterapeutens roll för personer med diagnosen myalgisk encefalomyelit/kroniskt trötthetssyndrom : En litteraturöversikt." Thesis, Luleå tekniska universitet, Institutionen för hälsovetenskap, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:ltu:diva-79099.

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Bakgrund: ME/CFS är en allvarlig, kronisk och komplex multisystemsjukdom som ofta och dramatiskt begränsar de drabbade personernas aktivitet. De vetenskapliga beläggen gällande effekten av interventioner riktade mot funktion och funktionsnedsättning är begränsade. Nuvarande kunskapsläge indikerar att arbetsterapeuten kan bidra i vården av personer med ME/CFS, men behov av vidare forskning finns. Syfte: Att med denna litteraturöversikt kartlägga och beskriva aktuell forskning gällande arbetsterapeutens roll för personer med diagnosen ME/CFS. Metod: Datainsamling för litteraturöversikten genomfördes baserat på utarbetade urvalskriterier i tre relevanta databaser; PubMed, CINAHL och PsycINFO och resulterade i tio artiklar, sju kvantitativa och tre kvalitativa studier. Studiernas kvalitet granskades och sedan utfördes en latent innehållsanalys. Resultat: Analysen resulterade i fyra kategorier: Att ge klientcentrerat stöd för strategier i aktivitet, Att justera terapeutiskt förhållningssätt vid aktivitetsanpassning, Att beakta gruppbehandlingens terapeutiska värde samt Att bidra till professionernas teamsamverkan. Slutsats: Arbetsterapeutens kompetens är ett viktigt bidrag i rehabiliteringen på grund av den komplexa aktivitetssituation som diagnosen innebär.
Background: ME/CFS is a serious, chronic and complex systemic disease which often and dramatically limits the activity of the affected. The existing scientific evidence of interventions regarding function and disability is limited. The current level of knowledge indicates that the occupational therapist can contribute to the care for people with ME/CFS, but there is a need for further research. Aim: The aim of this literature overview was to map out and describe current research regarding the role of the occupational therapist for people diagnosed with ME/CFS. Method: Data collection for the literature overview was conducted based on developed selection criterias in three relevant databases; PubMed, CINAHL and PsycINFO which resulted in ten articles, seven quantitative and three qualitative studies. The quality of the included studies were assessed and finally a latent content analysis was completed which resulted in four categories. Result: The content analysis resulted in four categories: to supply a client centered support for strategies in activity, to adjust therapeutic approach in occupational adaptation, to consider the therapeutic value of the group treatment, and to contribute to the professional team collaboration. Conclusion: The expertise of the occupational therapist is an important contribution to rehabilitation due to the complex occupational situation for people diagnosed with ME/CFS.
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Wolfe, Patricia Jean de. "I'm really ill, I've got M.E. : bodily disorder and the quest for disease." Thesis, Goldsmiths College (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.298292.

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Books on the topic "Myalgi"

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Jane, Sullivan, ed. M.E. What is it? Have you got it? How to get better. London: Century, 1989.

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Great, Britain Parliament. Myalgic encephalomyelitis Bill. London: H.M.S.O., 1991.

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Myalgic encephalomyelitis: Post-viral fatigue syndrome and how to cope with it. London: Croom Helm, 1986.

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Trust, Akamba Cultural, ed. Ngaeka waeka: Myali ya Kiikamba. Nairobi: Akamba Cultural Trust, 2010.

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Mense, Siegfried, and Robert Gerwin. Muscle pain: Diagnosis and treatment. Heidelberg: Springer, 2010.

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Shepherd, Charles. Living with M.E. London: Cedar, 1989.

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Shepherd, Charles. Living with M. E. London: Cedar, 1990.

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Shepherd, Charles. Living with M.E. London: Cedar, 1990.

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Great Britain. Department of Health. Chronic fatigue syndrome/myalgic encephalopathy (CFS/ME). London: Department of Health, 2004.

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Saunders, Keith B. Myall Road. Canberra: Aboriginal Studies Press, 1998.

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Book chapters on the topic "Myalgi"

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Gerwin, Robert D. "Myalgia." In Encyclopedia of Pain, 1996–2001. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_2550.

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Mehlhorn, Heinz. "Myalgia." In Encyclopedia of Parasitology, 1715. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_4834.

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Mehlhorn, Heinz. "Myalgia." In Encyclopedia of Parasitology, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_4834-1.

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Oette, Mark, Marvin J. Stone, Hendrik P. N. Scholl, Peter Charbel Issa, Monika Fleckenstein, Steffen Schmitz-Valckenberg, Frank G. Holz, et al. "Myalgic Encephalomyelitis." In Encyclopedia of Molecular Mechanisms of Disease, 1393. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_8006.

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Sjøgaard, Gisela, and Karen Søgaard. "Myalgia, Chronic." In Encyclopedia of Exercise Medicine in Health and Disease, 613–16. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_248.

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Nicolodi, M., and F. Sicuteri. "Eosinophilia Myalgia Syndrome." In Advances in Experimental Medicine and Biology, 351–57. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-0381-7_54.

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Criswell, L. A., and T. Pincus. "Eosinophilia-Myalgia Syndrome." In Advances in Experimental Medicine and Biology, 371. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-0381-7_57.

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Jain, Kewal K. "Eosinophilia-Myalgia Syndrome." In Drug-induced Neurological Disorders, 533–40. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-73503-6_34.

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Reimers, C. D., and E. Schmutzhard. "Myalgia, Myositis, and Fasciitis." In Aspects of Lyme Borreliosis, 185–92. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77614-4_13.

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Costa, D. C., and A. Greco. "Chronic Fatigue Syndrome/Myalgic Encephalomyelitis." In Nuclear Medicine in Psychiatry, 289–300. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18773-5_17.

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Conference papers on the topic "Myalgi"

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Schürholz, Daniel, Meruyert Nurgazy, Arkady Zaslavsky, Prem Prakash Jayaraman, Sylvain Kubler, Karan Mitra, and Saguna Saguna. "MyAQI." In IoT 2019: 9th International Conference on the Internet of Things. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3365871.3365884.

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Borsting, Jorun. "SUPPORTING RELAXATION: SLOWBREATH FOR PEOPLE WITH MYALGIC ENCEPHALOMYELITIS." In International Conference on e-Health 2019. IADIS Press, 2019. http://dx.doi.org/10.33965/eh2019_201910l019.

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Nazemroaya, Behzad, and Mehrdad Masoudifar. "A008 Effect of Preemptive Midazolam on Post-Electroconvulsive Therapy Headache, Myalgia, Nausea, and Vomiting." In 20th Annual Conference of Indian Society of Neuroanaesthesiology and Critical Care (ISNACC). Thieme Medical and Scientific Publishers Private Ltd., 2019. http://dx.doi.org/10.1055/s-0039-1684115.

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Guis, S., D. Bendahan, G. Kozak-Ribbens, JP Mattei, Y. Le Fur, D. Figarella-Branger, J. Jouglard, and PJ Cozzone. "SAT0204 Investigation of fluoroquinolone-induced myalgia using 31p magnetic resonance spectroscopy andin vitrocontracture tests." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.701.

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Domingues, Tiago Dias, Helena Mouriño, and Nuno Sepúlveda. "A statistical analysis of serological data from the UK myalgic encephalomyelitis/chronic fatigue syndrome biobank." In INTERNATIONAL CONFERENCE OF NUMERICAL ANALYSIS AND APPLIED MATHEMATICS ICNAAM 2019. AIP Publishing, 2020. http://dx.doi.org/10.1063/5.0026633.

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Yardley, D., N. Green, S. Papish, S. Lake, K. Culver, E. Argonza-Aviles, G. Warsi, and L. Hart. "Rheumatologic Evaluation of Adjuvant Letrozole in Post-Menopausal Breast Cancer Patients Discontinuing Anastrozole Due to Grade 2-3 Arthralgia – Myalgia." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-805.

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Neale, FK, TY Segal, and DS Hargreaves. "G13 Prevalence and correlates of low mood, poor quality of life and high symptom impact in adolescents attending a tertiary service for chronic fatigue syndrome/myalgic encephalomyelitis." In Royal College of Paediatrics and Child Health, Abstracts of the Annual Conference, 13–15 March 2018, SEC, Glasgow, Children First – Ethics, Morality and Advocacy in Childhood, The Journal of the Royal College of Paediatrics and Child Health. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2018. http://dx.doi.org/10.1136/archdischild-2018-rcpch.13.

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Liu, Jiening, Benjamin Hall, Mary Frecker, and Edward W. Reutzel. "Compliant Articulation Structure Using Superelastic NiTiNOL." In ASME 2012 Conference on Smart Materials, Adaptive Structures and Intelligent Systems. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/smasis2012-7970.

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A device that can provide articulation to surgical tool tips is needed in natural orifice transluminal endoscopy surgery (NOTES). In this paper, we propose a compliant articulation structure that uses superelastic NiTiNOL to achieve a large deflection angle and force in a compact size. Six geometric parameters are used to define this structure, and constraints based on the fabrication process are imposed. Using finite element analysis, a family of designs is evaluated in terms of the free deflection angle and blocked force. The same family of designs is evaluated for both NiTiNOL and stainless steel. It can be seen that significant benefits are observed when using NiTiNOL compared to 316 stainless steel; a maximum free deflection angle of 64.8° and maximum blocked force of 24.7 N are predicted. The designs are refined to avoid stress concentrations, and design guidelines are recommended. The meso-scale articulation structure is fabricated using both a Coherent Avia Q-switched, 355 nm laser and a Myachi Unitek 200 W single mode pulsed fiber laser with active water cooling. Select fabricated structures are then tested to validate the finite element models.
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Ayadi O’Donnell, N., A. McCourt, and T. Segal. "G531(P) A cohort study of whether parental separation and lack of contact with a parent predicts disease severity at diagnosis in young peoples chronic fatigue syndrome/myalgic encephalomyelitis." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference–Online, 25 September 2020–13 November 2020. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2020. http://dx.doi.org/10.1136/archdischild-2020-rcpch.449.

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Hadji, P., DG Kieback, A. Hasenburg, J. Tams, and M. Ziller. "P2-17-02: Increased Progression Free and Overall Survival in Breast Cancer Patients with Menopausal Symptoms or Arthralgia/Myalgia during Adjuvant Treatment with Exemestane or Tamoxifen – Results of the German TEAM Trial." In Abstracts: Thirty-Fourth Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 6‐10, 2011; San Antonio, TX. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/0008-5472.sabcs11-p2-17-02.

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Reports on the topic "Myalgi"

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Smith, M. E. Beth, Heidi D. Nelson, Elizabeth Haney, Miranda Pappas, Monica Daeges, Ngoc Wasson, and Marian McDonagh. Diagnosis and Treatment of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome. Agency for Healthcare Research and Quality, December 2014. http://dx.doi.org/10.23970/ahrqepcerta219.

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