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Dissertations / Theses on the topic 'Myasthena gravis'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Barrett-Jolley, Richard A. "Effects of plasma from seronegative myasthenics and controls on receptor and voltage gated membrane currents of TE671 cells." Thesis, University of Oxford, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.334228.

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2

Simpson, John Alexander. "Myasthenia gravis." Thesis, University of Edinburgh, 1991. http://hdl.handle.net/1842/27395.

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The thesis is 72 publications describing the author's hypothesis of an autoimmune basis for myasthenia gravis (MG) and its validation. At the time of the first 24 papers many considered that MG was a syndrome rather than a disease entity, due to a biochemical disorder of the neuromuscular junction. Favoured models were a circulating 'curare-like' substance released from the thymus gland, or a pre-junctional abnormality, possibly causing release of small quanta of acetylcholine at the motor nerve terminals. Endplate receptor substance was speculative. The immunological role of the thymus was un

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3

Carr, A. S. "An epidemiological study of myasthenia gravis and congenital myasthenic syndromes in Northern Ireland." Thesis, Queen's University Belfast, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.546021.

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4

da, Silva Leite Maria Isabel. "Myasthenia Gravis: Investigations into Seronegative Myasthenia." Thesis, University of Oxford, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.490100.

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Myasdienia gravis (MG) is an antibody-mediated autoimmune disease causing muscle weakness and fatigue. Over 80% of patients with generalised MG have IgG autoantibodies (mostly IgUlj to the native muscle acetylcholine receptor (AChR) at the neuromuscular junction (NM]). AChR-antibody-positive MG (AChR-MG) patients often benefit from thymectomy. Their thymus usually has epithelial hyperplasia and the thymus is thought to be the site of autoimmunisation against AChR. Of the remaining 15-20% of patients with generalised MG, a variable proportion (0-50%) have autoantibodies to muscle-specific kinas

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5

Verschuuren, Johannes Justus Gerard Maria. "Experimental autoimmune myasthenia gravis." Maastricht : Maastricht : Datawyse ; University Library, Maastricht University [Host], 1989. http://arno.unimaas.nl/show.cgi?fid=5471.

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6

Schaffert, Hanne. "Immunpathogenese der Myasthenia gravis." Doctoral thesis, Humboldt-Universität zu Berlin, Lebenswissenschaftliche Fakultät, 2015. http://dx.doi.org/10.18452/17213.

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Die Myasthenia Gravis (MG) ist ein Prototyp einer Antikörper-vermittelte Autoimmunerkrankung. Die Autoantikörper richten sich hauptsächlich gegen den Acetylcholinrezeptor (AChR). Welche Bedeutung TH17-Zellen für die Pathogenese der MG haben, konnte bisher noch nie direkt gezeigt werden. Mithilfe des Tiermodells Experimentelle Autoimmune Myasthenia Gravis (EAMG) sollte die Rolle der TH17-Zellen im Rahmen dieser Arbeit analysiert werden. Eine signifikante Anzahl tAChR-spezifischer CD4+ T-Zellen, die IL17 exprimieren, konnte nach der Immunisierung mit torpedo AChR in CFA in Wildtyp-Mäusen (WT) be

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7

Kaufman, Robin L. "Immunoregulation in myasthenia gravis." Thesis, University of British Columbia, 1989. http://hdl.handle.net/2429/30683.

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Myasthenia Gravis (MG) is an autoimmune disorder of neuromuscular transmission. Clinically, the disease is manifested by abnormal muscle fatigue with recovery on resting. Circulating nicotinic acetylcholine receptor antibodies (nAchR Ab) are highly characteristic of myasthenia gravis. These antibodies have been shown to be directly pathogenic at the muscle endplate and are responsible for impaired neuromuscular transmission through several mechanisms. While it is clear that the immune system does not function normally in MG, the mechanisms by which the response to nAchR is initiated and perpe

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8

McConville, John Paul. "Autoantibodies in seronegative myasthenia gravis." Thesis, University of Oxford, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.400295.

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9

Shi, Fu-Dong. "Immunopathogenesis and nasal tolerance in experimental autoimmune myasthenia gravis /." Stockholm, 1998. http://diss.kib.ki.se/search/diss.se.cfm?19980525shi.

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10

VILQUIN, JEAN-THOMAS. "Contribution a la comprehension, au traitement et au diagnostic de la myasthenia gravis." Strasbourg 1, 1992. http://www.theses.fr/1992STR15067.

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11

Wang, Hua-Bing. "Immunoregulation in experimental autoimmune myasthenia gravis /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4437-7/.

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12

Buckley, Camilla. "Autoimmunity in thymoma-associated Myasthenia gravis." Thesis, University of Oxford, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.394014.

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13

Pereira, Antonio. "Myasthenia gravis : l'histoire de madame h." Lille 2, 1988. http://www.theses.fr/1988LIL2M375.

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14

Koneczny, Inga. "Potential mechanisms in MuSK-myasthenia gravis." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:7b81b941-92c0-47ae-a747-62277394638e.

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Autoimmunity is a failure to tolerate circulating or cell surface expressed self antigens,leading to activation of the immune system and attack of self tissues. Muscle-specific kinase (MuSK) myasthenia gravis (MG) is a disease caused by antibodies to MuSK and hallmarked by fatigable muscle weakness. MuSK is a tyrosine kinase that interacts with low-density lipoprotein receptor-related protein 4 (LRP4), resulting in maintenance of the high density of acetylcholine receptors (AChRs) at the neuromuscular junction; this high density is essential for efficient transmission of signals from nerve to

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15

Xu, Biying. "Studies of immune mechanisms in myasthenia gravis /." Stockholm, 1998. http://diss.kib.ki.se/1998/91-628-3265-4/.

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16

Wang, XiongBiao. "CTLA-4 expression, regulation and associations in autoimmune myasthenia gravis /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-684-7/.

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17

Zhao, Xiaoyan. "Genetic and immunological control of human myasthenia gravis /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-494-5/.

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18

Plested, Charles Paul. "Mechanism of action of seronegative myasthenia gravis." Thesis, University of Oxford, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.301392.

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19

Moody, Anne Marie. "T-cell receptor studies in myasthenia gravis." Thesis, University of Oxford, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337448.

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20

Lotwick, Helen Sylvia. "Anti-(acetylcholine receptor) antibodies in myasthenia gravis." Thesis, University of Bath, 1985. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.351788.

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Levels of anti-(AChR) antibodies were determined in serial serum samples from 14 myasthenic patients over a period of several months, using detergent-solubilized muscle extracts of junctional rat AChR, extra-junctional rat AChR and human adult AChR as antigens. Anti-(AChR) antibody titres obtained using human adult AChR were always higher than those obtained using extra-junctional rat AChR, which were, in turn, always higher than those obtained using junctional rat AChR. Ihe ratios of antibody titres obtained by using the different antigens varied between patients, but were constant for an ind

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21

Richards, Ian. "Autologous mixed lymphocyte reaction in myasthenia gravis." Thesis, University of Bath, 1987. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.379569.

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22

Connor, Ruth Redlich. "Autoimmunity to cardiac muscle in myasthenia gravis /." The Ohio State University, 1988. http://rave.ohiolink.edu/etdc/view?acc_num=osu148759165817591.

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23

Palace, Jacqueline Ann. "Murine T and B cell epitodes within the human recombinant acetylcholine receptor alpha-subunit." Thesis, University of Oxford, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.387070.

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24

Nicolle, Michael W. "The induction of tolerance in CD4+ lymphocytes in vitro using soluble MHC class II : peptide complexes; a model for specific immunotherapy in autoimmune disease." Thesis, University of Oxford, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.386587.

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25

Kakoulidou, Maria. "T cells and costimulatory factors in myasthenia gravis /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-470-8/.

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26

Döring, Christian [Verfasser]. "Thymektomie bei Kindern mit Myasthenia gravis / Christian Döring." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2012. http://d-nb.info/1030382662/34.

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27

Jordan, Berit [Verfasser]. "Vorteile erweiterter Diagnostik bei Myasthenia gravis / Berit Jordan." Halle, 2017. http://d-nb.info/1132840449/34.

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28

Cowland, Alan. "Investigations into the genetic associations in myasthenia gravis." Thesis, University of Oxford, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.393351.

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29

Farrugia, Maria Elena. "Clinical and experimental investigations in seronegative myasthenia gravis." Thesis, University of Oxford, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.414087.

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30

Jacob, Saiju. "Investigations of seronegative generalised and ocular myasthenia gravis." Thesis, University of Oxford, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.547600.

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31

Nickless, Jane Christina. "Cellular immunity to acetylcholine receptor in myasthenia gravis." Thesis, University of Bath, 1985. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767550.

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Nicotinic acetylcholine receptor (AChR) has been purified from Torpedo electric organ, foetal calf muscle, adult human leg muscle, and foetal human skeletal muscle, by extraction in non-ionic detergent followed by affinity purification on immobilised a-toxin. The purified AChR preparations were used to study cellular responses in vitro from patients with myasthenia gravis. In addition, several characterisation studies were carried out on the foetal calf AChR preparation. Purified foetal calf AChR was shown in isoelectric focussing experiments to focus as a single sharp peak at pH 5.2 +/- 0.1,

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32

Hoedemaekers, Cornelia Wilhelmina Elisabeth. "Age-related resistance to experimental autoimmune myasthenia gravis immunological and neurobiological aspects /." [Maastricht : Maastricht : Universiteit Maastricht] ; University Library, Maastricht University [Host], 1997. http://arno.unimaas.nl/show.cgi?fid=5923.

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33

高崚. "重症肌無力的用藥規律的文獻研究". HKBU Institutional Repository, 2016. https://repository.hkbu.edu.hk/etd_oa/241.

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重症肌無力是一種全身性的獲得性自身免疫性疾病，傳統的分型有眼肌型、延髓肌型和全身型。西醫目前主要以藥物治療和手術治療為主要方法，尚無特效藥物根治本病。中醫歷代文獻中並無“重症肌無力’，病名的記載，重症肌無力屬中醫學中“鞘、“棲症、“唯目、“胞垂、“臉廢等範疇。本病為現代難治性病症之一，而中醫對本病的治療有一定優勢。在治療上加用中醫中藥，可以減少免疫抑制劑帶來的副作用，中藥在重症肌無力的治療上起著保駕護航的作用，而且有重建自身免疫功能之功效。通過對現代文獻中重症肌無力的中醫用藥進行統計，瞭解重症肌無力在醫學發展史上的歷史沿革與研究進展，汲取百家用藥經驗，對其病因病機，辨證分型進行探討，總結出中醫治療重症肌無力的用藥規律，為今後的學習、研究及應用提供一個思路和借鑒。通過進人香港浸會大學圖書館“中文科技期刊資料庫（醫藥衛生及自然科學的生物學專輯）的“維普資訊網以“中藥治療重症肌無力’，為關鍵字進行搜索，搜索到近十年相關期刊文獻183 篇，根據文獻的納入標準與排除標準進行篩選後，得到5 0 篇符合標準的有關文獻，繼而進行資料整理和分析，對每一篇文獻中重症肌無力的治療方法、中醫用藥及研究進展進行記錄，針對中醫用藥情況建立頻數分佈表以分析其構成比，從而探討中藥治療重症肌無力的用藥規律。最常見的重症肌無力中醫用藥有1 0 個，分別是黃、黨參、臼朮、當歸、升麻、甘草、拘把子、山藥、柴胡、陳皮

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34

Reis, Tarcísio Albertin dos. "Efetividade da plasmaférese no pré-operatório de timectomia em pacientes com miastenia gravis - revisão sistemática e metanálise." Universidade Estadual Paulista (UNESP), 2018. http://hdl.handle.net/11449/154816.

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Submitted by Tarcisio Albertin Dos Reis (ta.reis@unesp.br) on 2018-08-06T20:30:51Z No. of bitstreams: 1 Mestrado ultima versão.pdf: 801214 bytes, checksum: edb9c814c46ecb1d03d1e2a67318e15a (MD5)<br>Approved for entry into archive by Sulamita Selma C Colnago null (sulamita@btu.unesp.br) on 2018-08-08T16:30:20Z (GMT) No. of bitstreams: 1 reis_dc_me_bot.pdf: 801214 bytes, checksum: edb9c814c46ecb1d03d1e2a67318e15a (MD5)<br>Made available in DSpace on 2018-08-08T16:30:20Z (GMT). No. of bitstreams: 1 reis_dc_me_bot.pdf: 801214 bytes, checksum: edb9c814c46ecb1d03d1e2a67318e15a (MD5) Previous

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35

OKAMOTO, SUSUMU, AKlRA TAKAHASHI, TOSHIHIKO TAKEGAMI, and KAZUO MANO. "Remission of Myasthenia Gravis: Clinical, Electrophysiological and Immunological Studies." Nagoya University School of Medicine, 1993. http://hdl.handle.net/2237/17540.

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36

Graus, Ivo Maria Franciscus. "Experimental autoimmune myasthenia gravis target organ resistance and immunogenetics /." Maastricht : Maastricht : Universitaire Pers Maastricht ; University Library, Maastricht University [Host], 1992. http://arno.unimaas.nl/show.cgi?fid=6512.

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37

Meng, Fanping. "Anti-acetylcholine receptor autoantibodies in myasthenia gravis pathogenicity and specificity related to their structure /." [Maastricht : Maastricht : Universiteit Maastricht] ; University Library, Maastricht University [Host], 2001. http://arno.unimaas.nl/show.cgi?fid=5983.

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38

MacLennan, Calman Alexander. "Acetylcholine receptor subunit gene expression in different muscle groups and the thymus : a study of healthy subjects and of those with disordered neuromuscular transmission." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.390531.

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39

Thangarajh, Mathula. "B-cell-survival factors in multiple sclerosis and myasthenia gravis /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-097-8/.

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40

Dittner, Martin [Verfasser], and M. [Akademischer Betreuer] Winterholler. "Atemmuskelfunktion bei Myasthenia gravis pseudoparalytica / Martin Dittner. Betreuer: M. Winterholler." Erlangen : Universitätsbibliothek der Universität Erlangen-Nürnberg, 2011. http://d-nb.info/1015783287/34.

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41

Hawke, Simon. "Cellular immunity to the human acetylcholine receptor in myasthenia gravis." Thesis, University of Oxford, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.239315.

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42

Farrar, Jeremy. "Analysis of combinatorial immunoglobulin libraries from a myasthenia gravis patient." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.339366.

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43

Roxanis, Ioannis. "Studies in the thymus of early-onset myasthenia gravis patients." Thesis, University of Oxford, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.301233.

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44

Sollberger, Claudia Elisabeth. "The contribution of oculography to early diagnosis of myasthenia gravis /." [S.l : s.n.], 1986. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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45

Huda, Saif. "Clinical and functional characteristics of seronegative and musk myasthenia gravis." Thesis, University of Oxford, 2017. https://ora.ox.ac.uk/objects/uuid:597a563c-89bb-4fb6-85a8-ae1a28c5bed6.

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Myasthenia gravis (MG) is an autoimmune disorder of the neuromuscular junction (NMJ). Most patients have antibodies (Abs) against the acetylcholine receptor (AChR) or muscle specific kinase (MuSK) detected by radioimmunoassays (RIAs). Cell-based assays can more sensitively detect these and other Abs (e.g. to low density lipoprotein receptor-related protein 4 (LRP4)) but no large cohorts of MG patients have been systematically studied. RIA positive MuSK-Abs are mainly IgG4 subclass and block the interaction between LRP4 and MuSK, resulting in reduced MuSK phosphorylation and agrin-induced AChR

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46

Freeman, Carla Patricia. "Neuropsychiatric symptoms in thymoma-associated and non-thymoma myasthenia gravis." Master's thesis, University of Cape Town, 2012. http://hdl.handle.net/11427/11996.

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Includes abstract.<br>Includes bibliographical references.<br>Myasthenia gravis (MG) is an acetylcholine receptor antibody- mediated disease targeting the neuromuscular junction resulting in fatigable muscle weakness. A number of reports have suggested a high prevalence of psychiatric symptoms amongst MG patients. Approximately 10% of MG subjects are found to have an associated thymoma and despite thymomectomy, the MG persists. The presence of thymoma may lead to other antibody-mediated neuropsychiatric manifestations including limbic encephalitis. We hypothesized that the prevalence of neurop

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47

Fitzpatrick, Elizabeth. "Analysis of human skeletal muscle autoantibodies in myasthenia gravis patients /." The Ohio State University, 1990. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487678444257981.

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48

Carvalho, Nise de Brito. "Influência da terapêutica sobre a qualidade de vida do paciente com miastenia gravis." Universidade de São Paulo, 2006. http://www.teses.usp.br/teses/disponiveis/5/5138/tde-01112006-094252/.

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INTRODUÇÃO: A Miastenia gravis (MG) é uma desordem imunológica com antígenos alvos conhecidos, com produção de anticorpos contra o receptor nicotínico de acetilcolina, AAChR e Musk, na junção neuromuscular, dificultando a transmissão do impulso nervoso e provocando fadiga e fraqueza flutuantes na musculatura ocular, facial, dos membros e respiratória. A terapêutica sintomática com inibidores de acetilcolinesterase e a etiopatogênica como a timectomia, corticosteróides, agentes citostásticos e imunoglobulinas são utilizadas e indicadas em acordo com a incapacidade e gravidade clínicas. A qualid

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49

Cron, Mélanie. "Implication of microRNAs in the pathophysiology of autoimmune Myasthenia Gravis Analysis of microRNA expression in the thymus of Myasthenia Gravis patients opens new research avenues Use of Toll-like receptor agonists to induce ectopic lymphoid structures in Myasthenia gravis Mouse Models." Thesis, Sorbonne université, 2018. http://www.theses.fr/2018SORUS395.

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La Myasthénie auto-immune (MG), caractérisée par des auto-anticorps dirigés contre le récepteur à l’acétylcholine à la jonction neuromusculaire, est une maladie rare entraînant des faiblesses musculaires. Cette étude présente l’implication de petits ARN non-codants, les microARN, dans la physiopathologie de la MG. Nous avons étudié l’expression de microARN dérégulés dans l’organe effecteur de la maladie, le thymus, via une analyse du miRnome. J’ai mis en évidence 1) que miR-7-5p participerait à la mise en place des anomalies thymiques observées chez les patients de par son action sur CCL21, 2)

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50

Chan, Koon-ho. "Neuronal and muscle autoantibodies in paraneoplastic neurological disorders and autoimmune myasthenia gravis." Click to view the E-thesis via HKUTO, 2007. http://sunzi.lib.hku.hk/HKUTO/record/B39557091.

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