Relevant bibliographies by topics / MYB42

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'MYB42'

Author: Grafiati
Published: 4 June 2021
Last updated: 3 February 2022

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Journal articles on the topic "MYB42"

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Geng, Pan, Su Zhang, Jinyue Liu, Cuihuan Zhao, Jie Wu, Yingping Cao, Chunxiang Fu, Xue Han, Hang He, and Qiao Zhao. "MYB20, MYB42, MYB43, and MYB85 Regulate Phenylalanine and Lignin Biosynthesis during Secondary Cell Wall Formation." Plant Physiology 182, no. 3 (December 23, 2019): 1272–83. http://dx.doi.org/10.1104/pp.19.01070.

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Zhang, Zuying, Changtao Li, Hui Zhang, Yeqing Ying, Yuanyuan Hu, and Lili Song. "Comparative Analysis of the Lignification Process of Two Bamboo Shoots Stored at Room Temperature." Plants 9, no. 10 (October 21, 2020): 1399. http://dx.doi.org/10.3390/plants9101399.

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Two types of bamboo shoots, high bamboo (Phyllostachys prominens) shoots (HBSes) and moso bamboo (Phyllostachys edulis) shoots (MBSes), underwent a fast post-harvest lignification process under room temperature storage. To explore the mechanism of lignification in two types of bamboo shoots after post-harvest during room temperature storage, the measurement of cell wall polymers (lignin and cellulose) and enzyme activities of phenylalanine ammonialyase (PAL) and peroxidase (POD), and relative expression of related transcription networks factors (TFs) were performed. The results suggested that the lignification process in HBSes is faster than that in MBSes because of incremental increase in lignin and cellulose contents within 6 days and the shorter shelf-life. Additionally, compared with the expression pattern of lignification-related TFs and correlation analysis of lignin and cellulose contents, MYB20, MYB43, MYB85 could function positively in the lignification process of two types of bamboo shoots. A negative regulator, KNAT7, could negatively regulate the lignin biosynthesis in two types of bamboo shoots. In addition, MYB63 could function positively in HBSes, and NST1 could function negatively in MBSes. Notably, MYB42 may function differently in the two types of bamboo shoots, that is, a positive regulator in HBSes, but a negative regulator in MBSes. Transcription networks provide a comprehensive analysis to explore the mechanism of lignification in two types of bamboo shoots after post-harvest during room temperature storage. These results suggest that the lignification of bamboo shoots was mainly due to the increased activity of POD, higher expression levels of MYB20, MYB43, MYB63, and MYB85 genes, and lower expression levels of KNAT7 and NST1 genes, and the lignification process of HBSes and MBSes had significant differences.
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Sun, Yuhui, Jun Zhao, Xinyue Li, and Yingzhang Li. "E2 conjugases UBC1 and UBC2 regulate MYB42‐mediated SOS pathway in response to salt stress in Arabidopsis." New Phytologist 227, no. 2 (April 19, 2020): 455–72. http://dx.doi.org/10.1111/nph.16538.

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Wang, Nan, Haifeng Xu, Shenghui Jiang, Zongying Zhang, Ninglin Lu, Huarong Qiu, Changzhi Qu, Yicheng Wang, Shujing Wu, and Xuesen Chen. "MYB12 and MYB22 play essential roles in proanthocyanidin and flavonol synthesis in red-fleshed apple (Malus sieversiif. niedzwetzkyana)." Plant Journal 90, no. 2 (February 27, 2017): 276–92. http://dx.doi.org/10.1111/tpj.13487.

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Qi, Xin, Wensi Tang, Weiwei Li, Zhang He, Weiya Xu, Zhijin Fan, Yongbin Zhou, et al. "Arabidopsis G-Protein β Subunit AGB1 Negatively Regulates DNA Binding of MYB62, a Suppressor in the Gibberellin Pathway." International Journal of Molecular Sciences 22, no. 15 (July 31, 2021): 8270. http://dx.doi.org/10.3390/ijms22158270.

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Plant G proteins are versatile components of transmembrane signaling transduction pathways. The deficient mutant of heterotrimeric G protein leads to defects in plant growth and development, suggesting that it regulates the GA pathway in Arabidopsis. However, the molecular mechanism of G protein regulation of the GA pathway is not understood in plants. In this study, two G protein β subunit (AGB1) mutants, agb1-2 and N692967, were dwarfed after exogenous application of GA3. AGB1 interacts with the DNA-binding domain MYB62, a GA pathway suppressor. Transgenic plants were obtained through overexpression of MYB62 in two backgrounds including the wild-type (MYB62/WT Col-0) and agb1 mutants (MYB62/agb1) in Arabidopsis. Genetic analysis showed that under GA3 treatment, the height of the transgenic plants MYB62/WT and MYB62/agb1 was lower than that of WT. The height of MYB62/agb1 plants was closer to MYB62/WT plants and higher than that of mutants agb1-2 and N692967, suggesting that MYB62 is downstream of AGB1 in the GA pathway. qRT-PCR and competitive DNA binding assays indicated that MYB62 can bind MYB elements in the promoter of GA2ox7, a GA degradation gene, to activate GA2ox7 transcription. AGB1 affected binding of MYB62 on the promoter of GA2ox7, thereby negatively regulating th eactivity of MYB62.
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Im, Jong Hee, Jae-Heung Ko, Won-Chan Kim, Brent Crain, Daniel Keathley, and Kyung-Hwan Han. "Mitogen-activated protein kinase 6 negatively regulates secondary wall biosynthesis by modulating MYB46 protein stability in Arabidopsis thaliana." PLOS Genetics 17, no. 4 (April 7, 2021): e1009510. http://dx.doi.org/10.1371/journal.pgen.1009510.

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The R2R3-MYB transcription factor MYB46 functions as a master switch for secondary cell wall biosynthesis, ensuring the exquisite expression of the secondary wall biosynthetic genes in the tissues where secondary walls are critical for growth and development. At the same time, suppression of its function is needed when/where formation of secondary walls is not desirable. Little is known about how this opposing control of secondary cell wall formation is achieved. We used both transient and transgenic expression of MYB46 and mitogen-activated protein kinase 6 (MPK6) to investigate the molecular mechanism of the post-translational regulation of MYB46. We show that MYB46 is phosphorylated by MPK6, leading to site specific phosphorylation-dependent degradation of MYB46 by the ubiquitin-mediated proteasome pathway. In addition, the MPK6-mediated MYB46 phosphorylation was found to regulate in planta secondary wall forming function of MYB46. Furthermore, we provide experimental evidences that MYB83, a paralog of MYB46, is not regulated by MPK6. The coupling of MPK signaling to MYB46 function provides insights into the tissue- and/or condition-specific activity of MYB46 for secondary wall biosynthesis.
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Chen, Jianli, and Xiaowen Chen. "MYBL2 Is Targeted by miR-143-3p and Regulates Breast Cancer Cell Proliferation and Apoptosis." Oncology Research Featuring Preclinical and Clinical Cancer Therapeutics 26, no. 6 (July 5, 2018): 913–22. http://dx.doi.org/10.3727/096504017x15135941182107.

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Breast cancer remains a public health issue on a global scale. The present study aimed to explore the functional role of MYB proto-oncogene like 2 (MYBL2) in breast cancer, as well as underlying mechanisms. The regulatory relationship between miR-143-3p and MYBL2 was analyzed, and the effects of dysregulation of miR-143-3p and MYBL2 on cell proliferation and apoptosis were investigated. The results showed that MYBL2 and miR-143-3p were inversely expressed in breast cancer tissues and cells: MYBL2 was highly expressed, whereas miR-143-3p was lowly expressed. MYBL2 was confirmed as a target gene of miR-143-3p. Suppression of MYBL2 inhibited proliferation and induced apoptosis of breast cancer cells, which was similar to the effects of overexpression of miR-143-3p. Our findings reveal that MYBL2 is targeted by miR-143-3p and regulates breast cancer cell proliferation and apoptosis.
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Yoshikawa, Yuki, Xin Victoria Wang, Yu-Hui Chen, Ying Zhang Mazzu, Goutam Chakraborty, Lina E. Jehane, Sai Harisha Rajanala, et al. "The impact of the expression of the transcription factor MYBL2 on outcomes of patients with localized and advanced prostate cancer." Journal of Clinical Oncology 38, no. 6_suppl (February 20, 2020): 149. http://dx.doi.org/10.1200/jco.2020.38.6_suppl.149.

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149 Background: Patients with metastatic hormone-sensitive prostate cancer (mHSPC) have a variable response to ADT and some benefit from the addition of docetaxel or an androgen signaling pathway inhibitor. We found that loss of the epigenetic regulator KDM5D is associated with more aggressive prostate cancer (PC). We sought to determine whether MYBL2 which is regulated by KDM5D mediates this effect. MYBL2 is a transcription factor which controls key genes (e.g. FOXM1) and increases cell cycle progression and survival. Methods: AR expressing hormone-sensitive cell lines, LNCaP and LAPC4 were used. Motif analysis and CHiPseq of LNCaP with and without siKDM5D was performed and impact of modulation of KDM5D and MYBL2 in both cells on cell survival was assessed. Gene expression profiling (GEP) data assessed MYBL2’s association with KDM5D levels in localized disease (publicly available data) and mHSPC (Decipher whole Affymetrix platform on CHAARTED samples). Results: Silencing KDM5D increased H3K4me3 and increased MYBL2 expression. GEP showed a strong negative correlation between KDM5D and MYBL2 in patients with localized PC (-0.66; TCGA) but not from primary prostate cancer tissue with mHSPC (-0.03; CHAARTED). Cells with low KDM5D and high MYBL2 were androgen independent and more resistant to docetaxel. In TCGA, patients with high MYBL2 had a higher rate of relapse from localized disease. In patients with metastatic disease (CHAARTED) low MYBL2 was associated with a better overall survival (OS) on multivariable analysis when treated with ADT or ADT + docetaxel. Conclusions: Low MYBL2 is associated with a longer OS with ADT alone and ADT and docetaxel independent of clinical variables. Patients with high MYBL2 expression had better OS with ADT plus docetaxel compared with patients with high MYBL2 treated with ADT alone.[Table: see text]
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Zhang, Xueying, Yuqing He, Linying Li, Hongru Liu, and Gaojie Hong. "Involvement of the R2R3-MYB transcription factor MYB21 and its homologs in regulating flavonol accumulation in Arabidopsis stamen." Journal of Experimental Botany 72, no. 12 (April 8, 2021): 4319–32. http://dx.doi.org/10.1093/jxb/erab156.

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Abstract Commonly found flavonols in plants are synthesized from dihydroflavonols by flavonol synthase (FLS). The genome of Arabidopsis thaliana contains six FLS genes, among which FLS1 encodes a functional enzyme. Previous work has demonstrated that the R2R3-MYB subgroup 7 transcription factors MYB11, MYB12, and MYB111 redundantly regulate flavonol biosynthesis. However, flavonol accumulation in pollen grains was unaffected in the myb11myb12myb111 triple mutant. Here we show that MYB21 and its homologs MYB24 and MYB57, which belong to subgroup 19, promote flavonol biosynthesis through regulation of FLS1 gene expression. We used a combination of genetic and metabolite analysis to identify the role of MYB21 in regulating flavonol biosynthesis through direct binding to the GARE cis-element in the FLS1 promoter. Treatment with kaempferol or overexpression of FLS1 rescued stamen defects in the myb21 mutant. We also observed that excess reactive oxygen species (ROS) accumulated in the myb21 stamen, and that treatment with the ROS inhibitor diphenyleneiodonium chloride partly rescued the reduced fertility of the myb21 mutant. Furthermore, drought increased ROS abundance and impaired fertility in myb21, myb21myb24myb57, and chs, but not in the wild type or myb11myb12myb111, suggesting that pollen-specific flavonol accumulation contributes to drought-induced male fertility by ROS scavenging in Arabidopsis.
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Shukla, Vinay, Jian-Pu Han, Fabienne Cléard, Linnka Lefebvre-Legendre, Kay Gully, Paulina Flis, Alice Berhin, et al. "Suberin plasticity to developmental and exogenous cues is regulated by a set of MYB transcription factors." Proceedings of the National Academy of Sciences 118, no. 39 (September 22, 2021): e2101730118. http://dx.doi.org/10.1073/pnas.2101730118.

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Suberin is a hydrophobic biopolymer that can be deposited at the periphery of cells, forming protective barriers against biotic and abiotic stress. In roots, suberin forms lamellae at the periphery of endodermal cells where it plays crucial roles in the control of water and mineral transport. Suberin formation is highly regulated by developmental and environmental cues. However, the mechanisms controlling its spatiotemporal regulation are poorly understood. Here, we show that endodermal suberin is regulated independently by developmental and exogenous signals to fine-tune suberin deposition in roots. We found a set of four MYB transcription factors (MYB41, MYB53, MYB92, and MYB93), each of which is individually regulated by these two signals and is sufficient to promote endodermal suberin. Mutation of these four transcription factors simultaneously through genome editing leads to a dramatic reduction in suberin formation in response to both developmental and environmental signals. Most suberin mutants analyzed at physiological levels are also affected in another endodermal barrier made of lignin (Casparian strips) through a compensatory mechanism. Through the functional analysis of these four MYBs, we generated plants allowing unbiased investigation of endodermal suberin function, without accounting for confounding effects due to Casparian strip defects, and were able to unravel specific roles of suberin in nutrient homeostasis.
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Dissertations / Theses on the topic "MYB42"

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Agarwal, Tina R. "Syntelogs of MYB31 and MYB42 Exhibit Divergent Roles in Phenylpropanoid Pathway Regulation in Maize, Sorghum, and Rice." University of Toledo / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1452181737.

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Shi, Xinhui. "Regulatory Functions of ZmMYB31 and ZmMYB42 in Maize Phenylpropanoid Pathway." University of Toledo / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1302295047.

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Severmann, Julia [Verfasser], and Peter A. [Akademischer Betreuer] Horn. "Cell cycle-dependent gene expression by Mybl2 - a tumor suppressor in MDS / Julia Severmann ; Betreuer: Peter A. Horn." Duisburg, 2018. http://d-nb.info/1161341358/34.

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Krause, Theresa Margret Erika [Verfasser]. "Prognostische Bedeutung von Avian Myeloblastosis Viral Oncogene Homolog-Like 2 -Protein (MYBL2) beim Prostatakarzinom / Theresa Margret Erika Krause." Hamburg : Staats- und Universitätsbibliothek Hamburg Carl von Ossietzky, 2020. http://d-nb.info/1234150409/34.

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Kalemtas, Gulsum. "Transformation Of Potato With Myb4 Transcription Factor And Evaluation Of Abiotic Stress Tolerance And Gene Expression Profiles In Transgenic Plants." Phd thesis, METU, 2011. http://etd.lib.metu.edu.tr/upload/12613042/index.pdf.

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ABSTRACT TRANSFORMATION OF POTATO WITH MYB4 TRANSCRIPTION FACTOR AND EVALUATION OF ABIOTIC STRESS TOLERANCE AND GENE EXPRESSION PROFILES IN TRANSGENIC PLANTS Kalemtas, Gü
lsü
m Ph.D., Department of Biology Supervisor: Prof. Dr. Hü
seyin Avni Ö
ktem February 2011, 257 pages Potato (Solanum tuberosum L. cv. Kennebec) was transformed via Agrobacterium tumefaciens (EHA105) harbouring two different binary vectors containing Oryza sativa myb4 gene, which encodes MYB4 transcription factor
under the control of CaMV35S promoter or cold inducible COR15a promoter. The transgenic plants were not growth retarded and there was no significant difference (p<
0.05) in their tuber yield compared to wild-type plants. Wild-type and transgenic plants were subjected to abiotic stresses to compare their stress tolerances. There was no significant difference in boron, freezing and drought tolerances of wild-type and transgenic lines. Two of the transgenic lines were more salt tolerant than wild-type with respect to growth parameters. Transcriptomes of wild-type and these two lines, one expressing myb4 under the control of 35S promoter and the other COR15a promoter, were analyzed to elucidate the myb4-regulated processes and downstream target genes in potato. Differentially regulated genes in transgenic lines showed that myb4 controls a large and complex transcriptional network associated with diverse cellular processes, primarily defense and rescue, metabolism and development. Genes involved in sucrose synthesis, some peroxidases and CBF3 transcription factor were up-regulated in transgenic plants upon exposure to freezing stress. This suggested that myb4 may configure freezing response in potato primarily by oxidative stress defence mechanisms, osmotic adjustment or activation of CBF3 regulated genes that may confer cold tolerance. Despite up-regulation of these stress related genes, transgenic potato was not more drought or freezing tolerant compared to WT under the tested conditions. Further experiments should be conducted to better elucidate the involvement of these genes in regulation of stress response in transgenic potato expressing myb4.
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Rafatian, Ghazaleh. "Rejuvenation of Aged Heart Explant-Derived Cells for Repair of Ischemic Cardiomyopathy." Thesis, Université d'Ottawa / University of Ottawa, 2019. http://hdl.handle.net/10393/38850.

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In autologous stem cell therapy, cell characteristics determine the potency of stem cells for regeneration. Aging and ischemia are two factors that are often neglected in pre-clinical tests for stem cell therapy. Here, we characterized cardiac explant-derived cells (EDCs) with a focus on distinguishing the effect of age and ischemia and then we looked for the effects of the combination of the two factors. We observed that ischemia worsens the age effect on EDCs. EDCs that were derived from aged mice with a history of myocardial infarction showed the highest number of senescent cells with dysregulation of the DNA repair system resulting in activation of cell cycle checkpoints. We over-expressed the anti-senescence Mybl2 transcription factor in EDCs from ischemic aged mice. The senescent state, paracrine profile and superoxide dismutase antioxidant enzyme activity improved in these cells. In vivo, we observed a boost in the potency of the Mybl2-modified EDCs, with an increase in short-term engraftment leading to improved heart function in infarcted mice. In general, Mybl2 over-expression rejuvenates senescent EDCs.
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Wang, Szu-Huan, and 王思寰. "Probing the Protein Dynamics of Myb2 from Human Trichomonas vaginalis." Thesis, 2008. http://ndltd.ncl.edu.tw/handle/41603278694725526233.

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碩士
國立中央大學
生物資訊與系統生物研究所
96
Trichomonas vaginalis, an anaerobic, parasitic flagellated protozoan resides mostly in female vagina, urethra, uterus as well as male prostate gland, is the causative agent of the most common nonviral sexually transmitted infections (STIs) in the world, human trichomoniasis. Recently, multiple surface adhesion proteins have been shown to be engaged in Cytoadherence, an essential step, of the T. vaginalis infection. The ap65-1 gene, a member of the adhesion protein 65 (ap65) multigene family, encodes for multiple homologous 65-kDa proteins, was found to be regulated by transcription factors, Myb1 and Myb2 proteins, in T. vaginalis. Two DNA sequences, MRE1/MRE2r and MRE2f, on the gene, ap65-1, is recognized by Myb protein; this discovery deduce the possible involvement of Myb-like transcription factors related to the transcription mechanism within T. vaginalis. From various experiments, evidences show a specific interaction between full-length Myb2 protein with MRE1/MRE2r and MRE2f. We have found that a truncated fragment of Myb2, designated as Myb2x, consisting of amino acid V40–M156 displays similar DNA affinity. This fragment was employed for affinity binding and NMR structure-dynamic studies. By NMR relaxation technology, the difference in dynamics between Myb2x free, Myb2x-MRE1/MRE2r and Myb2x- MRE2f complex forms was resolved. 15N spin relaxation rates and heternuclear (15N-1H) NOE were measured by standard pulse sequences at static magnetic field of 14.7 Tesla. The relaxation data were further analyzed with reduced spectral density mapping approach to deduce the spectral density functions: J(0), J(ωN) and J(ω0.87H) . The protein dynamics of Myb2x, as revealed by the reduced spectral density functions, are mapped onto the structures of the free and DNA-bound forms of Myb2x. The results showed that binding of DNA tighten the protein structure considerably. Such information will be useful for design of effective drugs for the treatment of human trichomoniasis.
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Chang, Lung-Chun, and 張隆俊. "Define the nuclear translocation domain of Myb2 protein in Trichomonas vaginalis." Thesis, 2009. http://ndltd.ncl.edu.tw/handle/49029880123501100652.

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碩士
國立臺灣大學
微生物學研究所
97
Myb2, which regulates iron inducible transcription of the ap65-1 gene in the protozoan parasite Trichomonas vaginalis, was persistently detected in the nucleus. In the study, the nuclear localization signal of Myb2 was investgated exploiting immuneflouorescence assay to monitor subcellular localization of Myb2 and its mutant proteins. The region 48-143 was found to be sufficient, while 55EEDE58 as well as anyone of the structure elements in the R2R3 DNA-binding domain to be essential for nuclear import. Moreover, 51KF52, 139NRW141, and 143T/S145 together may also play important roles in Myb2 nuclear import. As examined by Western blotting, the region spanning 144-179 may contain a site for post-translational modification, which is not required for Myb2 nuclear import. Signal transduction pathway involve in Myb2 nuclear import were not identified. These observations suggest that Myb2 nuclear translocation is controlled by a novel nuclear localization signal.
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Liu, I.-Fen, and 劉怡棻. "Molecular cloning and biochemical characterization of a myb4 gene in Trichomonas vaginalis." Thesis, 2002. http://ndltd.ncl.edu.tw/handle/54662965101237888963.

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碩士
國立臺灣大學
微生物學研究所
91
MYB proteins are a group of conserved nuclear DNA-binding proteins, which involve in certain gene regulation during cell proliferation and differentiation in eukaryotes. The MYB proteins in Trichomonas vaginalis were previously shown to interact with the MREs (-44TATCGT-39) and MREl (-95TAACGATAT—87) regions of the ap65-1 promoter. In the present study, a tvmyb4 gene was cloned by degenerated polymerase chain reaction (PCR) in conjunction with 5’-rapid amplification of cDNA ends (RACE) and 3’-nested PCR. The tvmyb4 gene encodes a 31 kDa open reading frame. Using a prokaryotic gene expression system (pET 30), the recombinant tvMYB4 protein was produced and purified to raise antiserum in rabbits. The role of the tvMYB4 protein in transcriptional regulation of the ap65 gene remain to be investigated by promoter transactivation assay.
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Tsai, Cheng-Kun, and 蔡丞坤. "DNA Recognition Mechanism of Myb2 Protein Derived from Trichomonas vaginalis by NMR technology." Thesis, 2009. http://ndltd.ncl.edu.tw/handle/80596090569878157505.

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博士
國立臺灣師範大學
物理學系
97
The Myb2 protein from Trichomonas vaginalis was found to interact with specific sequence contexts MRE2r and MRE2f, and is involved in activation of both iron-inducible and growth-related transcription of the ap65-1 gene. The truncated Myb2 protein, Myb2x, spanning amino acid sequence 40-156, has been found to retain similar DNA binding affinity. We have determined Myb2x solution structures on free form and MRE2f-bound form by NMR. Both structures contain three α-helices at R2 domain and three α-helices at R3 domain. In free form the third helix and the linker are more flexible than in complex. Upon binding to MRE2f, the backbone dynamics of Myb2x protein become less flexible. Our results shown that the MRE2r and MRE2f share the same binding site on Myb2x, and the protein to DNA binding ratio is about one to one. On the other hand, the iron does not cause overall structure change in free Myb2x and two complexes, and does not interfere the process of MRE2r/MRE2f binding to Myb2x protein. The larger chemical shift perturbation of MRE2f on Myb2x-bound form appears at four bases ATAC. According to the 2D/3D-filtered NOESY experiments and the perturbation analysis of side-chain chemical shift, the DNA binding site is most likely distributed in the N-terminal, α3-helix of R2 domain, and α6-helix of R3 domain. Moreover, the N-terminal head directly contact to MRE2f DNA. The NOE signal between protein residue and DNA base can’t be well-defined, and the real MRE2f solution structure on Myb2x-bound form isn’t available yet. However, the Myb2x-MRE2f complex model was simulated using solved Myb2x protein structure and modeled B-form MRE2f.
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Book chapters on the topic "MYB42"

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Martin, Cara M., Katharine Astbury, Louise Kehoe, Jacqueline Barry O’Crowley, Sharon O’Toole, and John J. O’Leary. "The Use of MYBL2 as a Novel Candidate Biomarker of Cervical Cancer." In Methods in Molecular Biology, 241–51. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-2013-6_18.

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"Myb-Related Protein B, MYBL2." In Encyclopedia of Signaling Molecules, 3270. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_102446.

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Omondi, John Okoth. "Towards the Development of Salt-Tolerant Potato." In Sustainable Potato Production and the Impact of Climate Change, 133–51. IGI Global, 2017. http://dx.doi.org/10.4018/978-1-5225-1715-3.ch006.

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Soil salinity is a major constrain to crop production and climate change accelerates it. It reduces plant water potential, causes ion imbalance, reduce plant growth and productivity, and eventually leads to death of the plant. This is the case in potato. However, potato has coping strategies such as accumulation of proline, an osmoregulator and osmoprotector. In addition, leaching of salts below the root zone is preferred, exogenous application of ascorbic acid and growth hormones are practiced to combat salinity. Breeding and genetic engineering also play key roles in salinity management of potato. Varieties such as: Amisk, BelRus, Bintje, Onaway, Sierra, and Tobique were tolerant in North America, variety Cara in Egypt, Sumi in Korea and varieties Vivaldi and Almera in Mediterranean region. Transgenic lines of Kennebec variety, lines S2 and M48 also proved tolerance due to transcription factor MYB4 encoded by rice Osmyb4 gene.
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Omondi, John Okoth. "Towards the Development of Salt-Tolerant Potato." In Research Anthology on Food Waste Reduction and Alternative Diets for Food and Nutrition Security, 850–64. IGI Global, 2021. http://dx.doi.org/10.4018/978-1-7998-5354-1.ch043.

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Soil salinity is a major constrain to crop production and climate change accelerates it. It reduces plant water potential, causes ion imbalance, reduce plant growth and productivity, and eventually leads to death of the plant. This is the case in potato. However, potato has coping strategies such as accumulation of proline, an osmoregulator and osmoprotector. In addition, leaching of salts below the root zone is preferred, exogenous application of ascorbic acid and growth hormones are practiced to combat salinity. Breeding and genetic engineering also play key roles in salinity management of potato. Varieties such as: Amisk, BelRus, Bintje, Onaway, Sierra, and Tobique were tolerant in North America, variety Cara in Egypt, Sumi in Korea and varieties Vivaldi and Almera in Mediterranean region. Transgenic lines of Kennebec variety, lines S2 and M48 also proved tolerance due to transcription factor MYB4 encoded by rice Osmyb4 gene.
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"Gibberellin Acts through Jasmonate to Control the Expression of MYB21, MYB24, and MYB57 to Promote Stamen Filament Growth in Arabidopsis." In Reproductive Physiology in Plants, 169–96. Apple Academic Press, 2011. http://dx.doi.org/10.1201/b14533-10.

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Conference papers on the topic "MYB42"

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Xie, Meng. "Arabidopsis C-terminal Binding Protein ANGUSTIFOLIA modulates transcriptional co-regulation of MYB46 and WRKY33." In ASPB PLANT BIOLOGY 2020. USA: ASPB, 2020. http://dx.doi.org/10.46678/pb.20.1048273.

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Papetti, Michael, and Leonard H. Augenlicht. "Abstract 3903: Mybl2, a link between proliferation and differentiation in maturing colon epithelial cells." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3903.

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Papetti, Michael, and Leonard H. Augenlicht. "Abstract 138: Regulation of Mybl2 and miRNAs in differentiating colon epithelial cellsin vitroandin vivo." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-138.

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Mullen, Daniel J., Chunli Yan, Diane S. Kang, Beiyun Zhou, Zea Borok, Crystal N. Marconett, Peggy J. Farnham, Ite A. Offringa, and Suhn K. Rhie. "Abstract 3584: CENPA, MYBL2, and FOXM1 are identified as key transcriptional regulators in lung adenocarcinoma using TENET 2.0." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-3584.

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