Books: 'Myc Genes'

1

Perry, Claire Louise. A search for novel MHC-encoded IDDM susceptibility genes using oriental haplotypes. Birmingham: University of Birmingham, 1997.

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2

The Myc Gene Methods And Protocols. Humana Press Inc., 2013.

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3

Soucie, Erinn Louisa. Myc and apoptosis: Identifying key regulators and target genes. 2004.

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4

Eisenman, Robert N., and Chi V. Dang. MYC and the Pathway to Cancer. Cold Spring Harbor Laboratory Press, 2014.

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5

Potter, M. C-myc In B-cell Neoplasia: 14TH WORKSHOP ON MECHANISMS IN B-CELL NEOPLASIA (Current Topics in Microbiology & Immunology). Edited by M. Potter. SPRINGER-VERLAG, 1997.

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6

Michael, Browning, and McMichael Andrew J, eds. HLA and MHC: Genes, molecules and function. Oxford: BIOS Scientific, 1996.

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7

Yang, Wei-Cheng. Sequence Analysis of MHC Class II Genes in Cetaceans. INTECH Open Access Publisher, 2012.

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8

Browning, Michael. HLA AND MHC GENES MOLECULES AND FUNCTIONS (Human Molecular Genetics). Routledge, 1996.

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9

Browning, Michael, and Andrew McMichael. HLA and MHC: Genes, Molecules and Function (Human Molecular Genetics Series). Academic Pr, 1999.

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10

Aptsiauri, Natalia, Angel Miguel Garcia-Lora, and Teresa Cabrera. MHC Class I Antigens In Malignant Cells: Immune Escape And Response To Immunotherapy. Springer, 2013.

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11

Eric, Blair G., Pringle Craig R, and Maudsley D. John, eds. Modulation of MHC antigen expression and disease. Cambridge: Cambridge University Press, 1995.

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12

Modulation of MHC Antigen Expression and Disease. Cambridge University Press, 2005.

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13

Lewis, Myles, and Tim Vyse. Genetics of connective tissue diseases. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0042.

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The advent of genome-wide association studies (GWAS) has been an exciting breakthrough in our understanding of the genetic aetiology of autoimmune diseases. Substantial overlap has been found in susceptibility genes across multiple diseases, from connective tissue diseases and rheumatoid arthritis (RA) to inflammatory bowel disease, coeliac disease, and psoriasis. Major technological advances now permit genotyping of millions of single nucleotide polymorphisms (SNPs). Group analysis of SNPs by haplotypes, aided by completion of the Hapmap project, has improved our ability to pinpoint causal genetic variants. International collaboration to pool large-scale cohorts of patients has enabled GWAS in systemic lupus erythematosus (SLE), systemic sclerosis and Behçet's disease, with studies in progress for ANCA-associated vasculitis. These 'hypothesis-free' studies have revealed many novel disease-associated genes. In both SLE and systemic sclerosis, identified genes map to known pathways including antigen presentation (MHC, TNFSF4), autoreactivity of B and T lymphocytes (BLK, BANK1), type I interferon production (STAT4, IRF5) and the NFκ‎B pathway (TNIP1). In SLE alone, additional genes appear to be involved in dysregulated apoptotic cell clearance (ITGAM, TREX1, C1q, C4) and recognition of immune complexes (FCGR2A, FCGR3B). Future developments include whole-genome sequencing to identify rare variants, and efforts to understand functional consequences of susceptibility genes. Putative environmental triggers for connective tissue diseases include infectious agents, especially Epstein-Barr virus; cigarette smoking; occupational exposure to toxins including silica; and low vitamin D, due to its immunomodulatory effects. Despite numerous studies looking at toxin exposure and connective tissue diseases, conclusive evidence is lacking, due to either rarity of exposure or rarity of disease.

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14

Winchester, Robert, Darren D. O’Rielly, and Proton Rahman. Genetics of psoriatic arthritis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198737582.003.0006.

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The psoriatic phenotype is clinically heterogeneous with psoriatic arthritis (PsA) itself being heterogeneous. Studies have consistently demonstrated that PsA has a strong genetic component and disease pathogenesis encompasses a complex interplay between genetic, immunological, and environmental factors. In this chapter, we will review the genetics of PsA including the major histocompatibility complex (MHC) region and non-MHC loci. We will detail how susceptibility genes can be grouped into barrier integrity, innate immune response, and adaptive immune response (particularly Th-17 lymphocyte signalling). We will articulate how these studies strongly support PsA as genetically different from PsV and that the genetic heterogeneity is likely attributed to different HLA susceptibility alleles within the MHC region that an individual carries. Furthermore, we will highlight new emerging technologies, in particular, next-generation sequencing, which may lead to new genetic discoveries in PsA.

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15

Kinsman, Stephen L. Spina Bifida and Related Conditions. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0077.

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The term “spinal dysraphism” encompasses the broadest array of the conditions known as the neural tube defects. The open neural tube defects (spina bifida aperta and cystica) include both disorders of primary and/or secondary neuralation and are best defined as myelomeningocele complex (MMC) due to their protean nervous system manifestations beyond the spinal lesion. Closed spinal dysraphisms (so-called spina bifida occulta) include lipomatous lesions, forms of tethered spinal cord, sinus tracts, and forms of split spinal cord (diastematomyelia). Both genetic and environmental etiologies have been identified. Gene-environment and gene-gene interactions are also important in the pathobiology of these conditions.

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16

Sykes, Jim. Beravā Secrecy and the Hoarding of Musical Gifts. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190912024.003.0004.

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This chapter provides an introduction to Sinhala Buddhist music-making, focusing on the domain of drumming in religious ritual. These genres, with their accompanying dances, have become the most esteemed traditional music genres in Sri Lanka. The chapter also considers the ways that nationalism has influenced the use and placement of these genres, discussing a riot that broke out when Sinhala students agitated for Sinhala drumming to be performed at the head of a graduation ceremony at the Tamil-dominated University of Jaffna. The chapter provides basic information on drumming for the caste of drummers, dancers, and ritualists called the Beravā, and discusses the life and work of my drum teacher, a performer of the low-country Sinhala drum (pahata rata beraya, yak beraya).

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17

Watson, JAL, and HM Abbey. Atlas of Australian Termites. CSIRO Publishing, 1993. http://dx.doi.org/10.1071/9780643100657.

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This Atlas provides, for the first time, comprehensive maps showing the distribution of all named species of termite found in Australia. Based on records associated with the Australian National Insect Collection, it also provides a checklist of species and notes on the outstanding taxonomic problems in each genus. It answers the questions that administrators and pest controllers often ask: which troublesome termites are found in my area?

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18

Hinks, Anne, and Wendy Thomson. Genetics of juvenile rheumatic diseases. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0043.

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Juvenile rheumatic diseases are heterogeneous, complex genetic diseases; to date only juvenile idiopathic arthritis (JIA) has been extensively studied in terms of identifying genetic risk factors. The MHC region is a well-established risk factor but in the last few years candidate gene and genome-wide association studies have been utilized in the search for non-HLA risk factors. There are now an additional 12 JIA susceptibility loci with evidence for association in more than one study. In addition, some subtype-specific associations are emerging. These risk loci now need to be investigated further using fine-mapping strategies and then appropriate functional studies to show how the variant alters the gene function. This knowledge will not only lead to a better understanding of disease pathogenesis for juvenile rheumatic diseases but may also aid in the classification of these heterogeneous diseases. It may identify new pathways for potential therapeutic targets and help in the prediction of disease outcome and response to treatment.

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19

Hinks, Anne, and Wendy Thomson. Genetics of juvenile rheumatic diseases. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199642489.003.0043_update_002.

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Juvenile rheumatic diseases are heterogeneous, complex genetic diseases; to date only juvenile idiopathic arthritis (JIA) has been extensively studied in terms of identifying genetic risk factors. The MHC region is a well-established risk factor but in the last few years candidate gene and large-scale genome-wide association studies have been utilized in the search for non-HLA risk factors. There are now 17 JIA susceptibility loci which reach the genome-wide significance threshold for association and a further 7 regions with evidence for association in more than one study. In addition, some subtype-specific associations are emerging. These risk loci now need to be investigated further using fine-mapping strategies and then appropriate functional studies to show how the variant alters the gene function. This knowledge will not only lead to a better understanding of disease pathogenesis for juvenile rheumatic diseases but may also aid in the classification of these heterogeneous diseases. It may identify new pathways for potential therapeutic targets and help in the prediction of disease outcome and response to treatment.

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20

Hinks, Anne, and Wendy Thomson. Genetics of juvenile rheumatic diseases. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199642489.003.0043_update_003.

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Juvenile rheumatic diseases are heterogeneous, complex genetic diseases; to date only juvenile idiopathic arthritis (JIA) has been extensively studied in terms of identifying genetic risk factors. The MHC region is a well-established risk factor but in the last few years candidate gene and large-scale genome-wide association studies have been utilized in the search for non-HLA risk factors. There are now 17 JIA susceptibility loci which reach the genome-wide significance threshold for association and a further 7 regions with evidence for association in more than one study. In addition, some subtype-specific associations are emerging. These risk loci now need to be investigated further using fine-mapping strategies and then appropriate functional studies to show how the variant alters the gene function. This knowledge will not only lead to a better understanding of disease pathogenesis for juvenile rheumatic diseases but may also aid in the classification of these heterogeneous diseases. It may identify new pathways for potential therapeutic targets and help in the prediction of disease outcome and response to treatment.

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21

International Laboratory for Research on Animal Diseases. and International Society for Animal Blood Group Research., eds. MHC class II genes and products and their significance for disease research in livestock species: Proceedings of a workshop held at the International Laboratory for Research on Animal Diseases, Nairobi, Kenya. 27 September-1 October 1987. Oxford: Published by Blackwell Scientific Publications for the International Society for Animal Blood Group Research, 1988.

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22

Bakan, Michael B. Graeme Gibson. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190855833.003.0007.

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Graeme Gibson is a man of many talents. He curates an online museum of more than four hundred world music instruments, plays many of those instruments himself, designs and builds others, and conducts copious research on music traditions worldwide. Because “world music involves numerous traditions [up] to contemporary musics,” Graeme says, “I prefer to think of it as a spectrum that includes numerous genres.” He sees parallels between the spectrum of world music and the autism spectrum, where he states that he “also found that everyone is different from their case, to my case and so on. I do agree with the term ‘spectrum,’ ” he adds, “but we still have lots to learn.” Living with “classic autism” all his life, Graeme has faced formidable challenges. Yet he would not rid himself of his condition even given the chance: “Some symptoms I would like to have gone, like stress triggers and so on, but I fear in the end my music may be lost too.”

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23

Kaplan, E. Ann. Troubling Genre/Reconstructing Gender. University of Illinois Press, 2017. http://dx.doi.org/10.5406/illinois/9780252036613.003.0005.

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This chapter explores the cultural work that feminist critics performed in “inventing” the genre of the woman's film, and how genre impacts on feminist cinema practices in the current postmodern moment. It argues first that historically, genre was important in providing a useful pathway through which feminist film theorists could assert a critical position vis-à-vis dominant cinematic and critical strategies. That is, feminist critics used genre as a concept to invent a new genre—the women's picture or woman's film—thereby drawing attention to aspects of Hollywood melodrama that had been neglected by (largely male) critics. Secondly, through the examples Sister My Sister (Nancy Meckler, 1994) and Memsahib Rita (Pratibha Parmar, 1994), the chapter shows how some female directors have drawn on traditional Hollywood genres for feminist ends.

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24

d'Hubert, Thibaut. Ālāol’s Poetry and Mrauk U’s Political Turmoil. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190860332.003.0005.

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The political crisis of 1661 that was triggered by the arrival in Arakan of Mughal Prince Shāh Shujāʿ (d. 1661), who was fleeing from his brother Awrangzeb’s (r. 1658–1707) army, marks the beginning of major shifts in Ālāol’s literary activities. In this chapter, I follow the thread of Ālāol’s poems’ stylistic evolutions during the decade spanning from 1661 to 1671—the date of the composition of Sikāndarnāmā, Ālāol’s last work. This period is characterized by the progressive degradation of the relationships between the Bengali Muslim elites and the Buddhist Arakanese ruler that deeply modified the configuration of the literary gatherings in Mrauk U, as well as the aspirations of their members. In my analysis I trace how these changes affected the poet’s recourse to the romance and epic genres, as well as lyric poetry.

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25

Bradley, Ben. Darwin's Psychology. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780198708216.001.0001.

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Darwin has long been hailed as forefather to behavioural science, and even more so nowadays, with the growing popularity of evolutionary psychologies. This is the first book to examine Darwin’s own extensive writings about psychological matters. It finds that Darwin’s fulcrum was the agency of living creatures—both in his psychology and in his theory of evolution. A careful reading of Darwin’s writings on topics from climbing plants to babies shows that no individual-based theory of evolution can explain everything about human action. The interpersonal domain, group-life and culture, are also key, whether we consider the dynamics of conscience, emotional expressions or the dramas of desire. For example, Darwin argues that the anatomy and physiology of evolutionarily ‘purposeless’ facial movements gain meaning through their perception by others. His explanation of blushing adds a layer of complexity to such recognition—my blush results from my perception of how you are reading me. A similar reflexive dynamic governs how Darwin understands sexual desire, conscience, the setting of social standards, and the place of culture in human agency. Testing the main plank of Darwin’s psychology—that a capacity for group-interaction underpins the most human aspects of human agency—has awaited contemporary research, being recently confirmed by film-studies of young babies. Darwin’s writings frame a surprisingly well-resourced arena for elaboration of a socialized, agentic account of how we and our fellow creatures live. Moreover, Darwin stands at the forefront of moves toward an evolutionary biology in which organisms lead and genes follow.

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26

Sherman, Stuart. Finding Their Accounts. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780199580033.003.0022.

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This chapter focuses on the peculiarities of the Spectator, a publication which began in 1711. The Spectator is neither autobiography nor novel; it offers, starting with its first number, a useful map through the maze of their intertwining. That the two genres were intimately enmeshed during the decades of their first emergence is a proposition at once self-evident and much canvassed. But the chapter shows how the Spectator may provide a route worth further canvassing. In the peculiar characteristics of its wildly popular authorial persona, it plays out as paradigm (and as parody too) core patterns of transaction between author and reader which had already begun to establish the narrative of ‘my own History’ (whether factual or fictive) as a newly hypnotic cultural artefact — and as a mode of writing whose powerful appeal resides in ‘separations’.

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27

Hamilton, Tom. Pierre de L’Estoile and his World in the Wars of Religion. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198800095.001.0001.

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The Wars of Religion embroiled France in decades of faction, violence, and peacemaking in the late sixteenth century. When historians interpret these events, inevitably they depend on sources of information gathered by contemporaries, none more valuable than the diaries and the collection of Pierre de L’Estoile (1546–1611), who lived through the civil wars in Paris and shaped how they have been remembered ever since. Taking him out of the footnotes, and demonstrating his significance in the culture of the late Renaissance, this book is the first life of L’Estoile in any language. It examines how he negotiated and commemorated the conflicts that divided France as he assembled an extraordinary collection of the relics of the troubles, a collection that he called ‘the storehouse of my curiosities’. The story of his life and times is the history of the civil wars in the making. Focusing on a crucial individual for understanding Reformation Europe, this book challenges historians’ assumptions about the widespread impact of confessional conflict in the sixteenth century. L’Estoile’s prudent, non-confessional responses to the events he lived through and recorded were common among his milieu of Gallican Catholics. His life writing and engagement with contemporary news, books, and pictures reveals how individuals used different genres and media to destabilize rather than fix confessional identities. Bringing together the great variety of topics in society and culture that attracted L’Estoile’s curiosity, this book rethinks his world in the Wars of Religion.

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Books on the topic 'Myc Genes'

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