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Books on the topic 'Mycosis - Treatment'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Hospenthal, Duane R., and Michael G. Rinaldi, eds. Diagnosis and Treatment of Human Mycoses. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-325-7.

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2

Feigenbaum, Ernest. Debridement and other treatment of mycotic toenails. Rockville, MD: U.S. Dept. of Health and Human Services, Public Health Service, Office of the Assistant Secretary for Health, National Center for Health Services Research and Health Care Technology Assessment, 1985.

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3

FRCPath, Jones Brian L., and Rautemaa Riina, eds. Therapeutic guidelines in systemic fungal infections. 4th ed. London: Remedica, 2007.

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4

Buddle, J. R. Bacterial and fungal diseases of pigs. Canberra: Australian Govt. Pub. Service, 1985.

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5

Hall, Gerri S. Interactions of yeasts, moulds, and antifungal agents: How to detect resistance. New York: Humana Press, 2012.

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6

Interactions of yeasts, moulds, and antifungal agents: How to detect resistance. New York: Humana Press, 2012.

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7

The Treatment of Mycosis with Imidazole Derivatives. Springer, 2012.

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8

Sokol, Lubomir. Mycosis Fungoides and Sézary Syndrome: Pathogenesis, Diagnosis and Treatment. Edited by Xiaohui Zhang. Nova Science Publishers, 2021. http://dx.doi.org/10.52305/lfse9103.

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9

R, Hospenthal Duane, and Rinaldi Michael G, eds. Diagnosis and treatment of human mycoses. Totowa, N.J: Humana, 2008.

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10

(Editor), Duane R. Hospenthal, and Michael G. Rinaldi (Editor), eds. Diagnosis and Treatment of Human Mycoses (Infectious Disease). Humana Press, 2007.

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11

Rinaldi, Michael G., and Duane R. Hospenthal. Diagnosis and Treatment of Fungal Infections. Springer, 2016.

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12

Rinaldi, Michael G., and Duane R. Hospenthal. Diagnosis and Treatment of Fungal Infections. Springer, 2015.

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13

Invasive Fungal Infection (Round Table Series (RTS)). Royal Society of Medicine Press Ltd, 1999.

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14

W, Denning David, Dupont Bertrand, and Pauw B. de, eds. Invasive fungal infection. London: Royal Society of Medicine Press, 1999.

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15

1918-, Jucker Ernst, ed. Antifungal agents: Advances and problems. Basel: Birkhauser Verlag, 2003.

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16

Andrew, Miles, Prentice Archie, Rogers Tom, British Society for Antimicrobial Chemotherapy., British Society for Haematology, and University of East London. University Centre for Health Services Research., eds. The effective prevention and management of systemic fungal infection in haematological malignancy. London: Aesculapius Medical Press, 2001.

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17

Clark, Heather L., and Eric Pearlman. Fungal eye infections. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0028.

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Mycotic keratitis is a fungal infection of the cornea that leads to severe, painful ulceration and loss of vision, and is a major cause of blindness worldwide, particularly in the developing world. Major risk factors for mycotic keratitis include ocular trauma and contact lens use. Both yeasts and moulds can cause mycotic keratitis, with the filamentous moulds of the Fusarium and Aspergillus genera the most common aetiological agents worldwide. Fungi, particularly Candida spp. yeasts, can also cause endophthalmitis—a painful, blinding infection of the posterior eye. Treatment of these infections is challenging owing to a limited arsenal of antifungal agents and highly variable susceptibility among causative fungi. Furthermore, associated inflammation contributes greatly to tissue damage and permanent blindness. Studies using experimental models of mycotic keratitis have revealed new targets for novel antifungal agents and anti-inflammatory therapies that have the potential to reduce the impact of these devastating infections.

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18

Combating Fungal Infections Problems And Remedy. Springer, 2010.

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19

Hay, Roderick J. Fungal infections of the skin and subcutaneous tissue. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0023.

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Fungal infections that involve the skin range from tinea pedis or ‘athlete’s foot’, which presents no threat to life, to less common, and often life-threatening, systemic mycoses such as cryptococcosis. The superficial infections are world-wide in distribution, and are estimated to be the fourth most common of all non-fatal causes of human disability. Diagnosis is often clinical, supported where appropriate by laboratory diagnostics. However, in those cases where deep infection is possible, screening patients for other sites of infection is key to therapeutic success. Treatment for the superficial infection is often based on topical medications obtainable without prescription, whereas the systemic infections usually require the best treatment for bloodstream-disseminated forms of infection.

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20

(Editor), Mahmoud A. Ghannoum, and John Perfect (Editor), eds. Antifungal Therapy. Informa Healthcare, 2008.

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21

Ghannoum, Mahmoud A., John R. Perfect, and Mahmoud Ghannoum. Antifungal Therapy. Taylor & Francis Group, 2017.

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22

Grossman, Jonah, Tanzila Shams, and Cathy Sila. Neurological Complications of Infective Endocarditis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0167.

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Infective endocarditis is the fourth leading cause of life-threatening infections, accounting for 40,000 annual U.S. hospital admissions. Due to decline in rheumatic heart disease, a shift in causative organisms from viridans streptococci to S. aureus, Group D Streptococcus, and multidrug-resistant species has been observed. The spectrum of neurological complications ranges widely from cerebrovascular pathologies-including septic embolization, mycotic aneurysms, and intracerebral hemorrhages-to seizures, meningitis, cerebritis, and abscess. Transthoracic echocardiogram remains the standard for initial investigation whereas CT scans, MRI with DWI sequence, and cerebral angiograms are useful for exploring neurological complications. Antibiotic regimens, tailored to culprit organisms, should be initiated early after obtaining blood cultures and continued for 4 to 6 weeks. Antithrombotic treatment may pose increased risk for intracerebral hemorrhage, even in the absence of mycotic aneurysms (MA). Unruptured MA must be treated according to risk of rupture and overall health of the patient. MAs either at risk or previously ruptured should be secured by neurosurgical or endovascular means. Early cardiac surgery is a viable option for prevention of septic embolization for high-risk cardiac diseases such as perivalvular abscess and infection with resistant organisms, but may increase mortality rates for those with decompensated heart failure.

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23

Hall, Gerri S. Interactions of Yeasts, Moulds, and Antifungal Agents: How to Detect Resistance. Springer, 2011.

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24

Hall, Gerri S. Interactions of Yeasts, Moulds, and Antifungal Agents: How to Detect Resistance. Humana, 2014.

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25

Mignon, B., and M. Monod. Zoonotic infections with dermatophyte fungi. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0077.

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Dermatophytes are highly specialized pathogenic fungi which are the most common agents of superficial mycoses. These fungi grow exclusively in the stratum corneum, nails or hair utilising them as sole nitrogen and carbon sources. Dermatophyte species are recognized and classified as antropophilic, zoophilic, or geophilic, depending on their major reservoir in nature (humans, animals, and soil, respectively). Zoophilic dermatophytes may result in zoonoses when humans are exposed to these organisms and dermatophytosis is considered to be one of the most common zoonotic diseases. The majority of zoonotic dermatophytoses are caused by four species: Microsporum canis (usually derived from pet animals, particularly cats and dogs), Trichophyton verrucosum (usually derived from cattle), Arthroderma vanbreuseghemii (usually derived from cats and dogs) and Arthroderma benhamiae (usually derived from guinea-pigs). Infection results most often from direct contact with an infected animal, but may be also acquired indirectly through contact with a contaminated environment. While clinical disease is rarely serious, the lesions can result in disfigurement and pain. Diagnosis is based on history, clinical appearance and diagnostic procedures, e.g. direct microscopic examination of scales, hair or nail and fungal culture. Specific treatment is generally required to resolve lesions, and this may be prolonged depending on the fungal species and the host status. Identifying animals as the source of infection for people can help in the prevention of recurrence or new infections, especially in children, by adequately treating affected pets and their environments. Immunoprophylaxis is an attractive means of controlling infection in animals, and the development and widespread use of efficacious T. verrucosum vaccines in certain countries has already proved valuable in the management of cattle ringworm.

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