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Books on the topic 'Myelitis'

Author: Grafiati

Published: 4 June 2021

Last updated: 26 July 2025

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1

National Institutes of Health (U.S.). Transverse myelitis. U.S. Department of Health and Human Services, Public Health Service, National Institutes of Health, 2012.

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2

National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison. La mielitis transversa. U.S. Department of Health and Human Services, Public Health Service, National Institutes of Health, 2012.

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3

Parker, James N., and Philip M. Parker. The official patient's sourcebook on transverse myelitis. Edited by Icon Group International Inc. Icon Health Publications, 2002.

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4

Richer, Catherine Balsdon. Occupational therapy practice guidelines for adults with neurodegenerative diseases: Multiple sclerosis, transverse myelitis, and amyotrophic lateral sclerosis. American Occupational Therapy Association, 1999.

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5

Rucker, Allen. The Best Seat in the House. HarperCollins, 2007.

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6

Miller, Aaron E., and Teresa M. DeAngelis. Transverse Myelitis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199732920.003.0004.

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The differential diagnosis of transverse myelitis is lengthy and can present a daunting challenge to the evaluating physician. Emphasizing the critical importance of an initial exclusion of spinal cord compressive causes, we then present several clinical red flags, which can assist in narrowing the potential etiologies and discuss available therapeutic options.

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7

Vodopivec, Ivana, and Tracey A. Cho. Neurobiology of Transverse Myelitis and Infectious Myelopathies. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0153.

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Infectious agents cause spinal cord pathology by three different mechanisms: direct invasion/infection of neural tissues (i.e., infective myelitis), secondary inflammation and tissue bystander damage with or without autoimmune pathogenesis (parainfectious myelitis), or involvement of extra-axial structures (including the pia-arachnoid, the dura, the epidural space, or the adjacent spinal bones or intervertebral discs), resulting in compressive or ischemic myelopathy. This chapter describes the pathogenesis and treatment of these disorders.

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8

Nahar, Yasmin. Transverse Myelitis: My Journey to Self Discovery. Olympia Publishers, 2022.

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9

Malecki, Damon. Coloring Book - You Will Get Better - Transverse Myelitis. Independently Published, 2021.

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10

MORTON, Charles. Acute Flaccid Myelitis: A Simple Care Guide for Afm Children. Independently Published, 2021.

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11

Jezdimir, Mike. I Will Not Sell Pencils; My Journey, Living with Transverse Myelitis. Lulu Press, Inc., 2013.

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12

Publications, ICON Health. Myelitis - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References. ICON Health Publications, 2004.

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13

E. C. (Edward Constant) B. 1843 Seguin. Myelitis of the Anterior Horns, or, Spinal Paralysis of the Adult and Child. Creative Media Partners, LLC, 2015.

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14

Misulis, Karl E., and E. Lee Murray. Demyelinating Diseases. Edited by Karl E. Misulis and E. Lee Murray. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190259419.003.0022.

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The hospital neurologist is often consulted to coordinate the evaluation and management of a host of known or suspected demyelinating diseases. Among the most common are multiple sclerosis, acute disseminated encephalomyelitis, optic neuritis, and transverse myelitis.

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15

Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson, and David Ledingham. Neurological infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0015.

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This chapter reviews the clinical features and management of meningitis (community-acquired bacterial meningitis and chronic meningitis), Mycobacterium tuberculosis, encephalitis and myelitis, Lyme disease, brain abscess and parameningeal infection, neurological infections in the immunocompromised, fungal infection, parasitic infection, and bacterial neurotoxins.

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16

Publications, ICON Health. The Official Patient's Sourcebook on Transverse Myelitis: A Revised and Updated Directory for the Internet Age. Icon Health Publications, 2002.

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17

Miller, Aaron E., and Teresa M. DeAngelis. Varicella Zoster Myelopathy. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199732920.003.0010.

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Varicella zoster virus (VZV) myelopathy is a rare cause of infectious myelitis and can present in both immunocompetent as well as immunocompromised individuals. In this chapter, we review the two most common presentations of VZV myelopathy, the optimal diagnostic testing and current therapeutic approaches.

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18

Coyle, Patricia K. Immune-mediated Disorders of the Central Nervous System. Edited by Emma Ciafaloni, Cheryl Bushnell, and Loralei L. Thornburg. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190667351.003.0010.

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This chapter reviews pregnancy in multiple sclerosis (MS), neuromyelitis optica spectrum disorder (NMOSD), and acute transverse myelitis (ATM) syndrome. MS is a major acquired disease of young adults, with a rising female predominance. MS has no direct negative consequences on fertility or pregnancy. Pregnancy has a profound effect on MS, with decrease in disease activity during the last trimester counteracted by a three-month postpartum increase in disease activity. With the development of disease-modifying therapies, important questions arise about washout periods, the feasibility and risks

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19

Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson, and David Ledingham. Inflammatory disorders of the central nervous system. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0006.

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Primary neuroinflammatory disorders of the central nervous system (multiple sclerosis, neuromyelitis optica (NMO), transverse myelitis, optic neuritis, acute disseminated encephalomyelitis (ADEM), primary angiitis of the central nervous system, autoimmune limbic encephalitis, and Susac’s syndrome) and multisystem diseases with inflammatory involvement of the central nervous system (sarcoidosis, systemic lupus erythematosus (SLE), giant cell arteritis, Behçet’s disease, Sjögren’s, and other vasculitides) are discussed in depth, covering the aetiology, clinical features and evidence-based treatm

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20

Andiel, Ernst. Nachweis von equinem Herpesvirus 1 mit Hilfe der Immunhistochemie und der In situ Hybridisierung unter besonderer Berücksichtigung der Myelitis disseminata Dexler. 1992.

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21

Rucker, Allen. Best Seat in the House. HarperCollins Publishers, 2007.

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22

Rucker, Allen. The Best Seat in the House: How I Woke Up One Tuesday and Was Paralyzed for Life. HarperCollins, 2007.

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23

Rucker, Allen. Best Seat in the House: How I Woke up One Tuesday and Was Paralyzed for Life. HarperCollins Publishers, 2009.

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24

Rucker, Allen. The Best Seat in the House: How I Woke Up One Tuesday and Was Paralyzed for Life. Harper Paperbacks, 2008.

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25

Rucker, Allen. The Best Seat in the House: How I Woke Up One Tuesday and Was Paralyzed for Life. HarperCollins, 2007.

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26

Rucker, Allen. Best Seat in the House. HarperCollins Publishers, 2007.

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27

Rucker, Allen. Best Seat in the House. HarperCollins Publishers, 2007.

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28

Rucker, Allen. Best Seat in the House. HarperCollins Publishers, 2007.

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29

Schreiner, Teri L., and Jeffrey L. Bennett. Neuromyelitis Optica. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0088.

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Neuromyelitis optica (NMO), or Devic’s disease is an inflammatory disorder of the central nervous system that preferentially affects the optic nerves and spinal cord. Initially considered a variant of multiple sclerosis (MS), NMO is now clearly recognized to have distinct clinical, radiographic, and pathologic characteristics. Historically, the diagnosis of NMO required bilateral optic neuritis and transverse myelitis; however, the identification of a specific biomarker, NMO-IgG, an autoantibody against the aquaporin-4 (AQP4) water channel, has broadened NMO spectrum disease to include patient

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30

McCormick, Paul C. Intramedullary Spinal Cord Tumors. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696696.003.0020.

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Intramedullary spinal cord tumors represent a heterogeneous group of neoplasms that may cause progressive spinal cord dysfunction. With few exceptions, however, most are slow-growing, biologically indolent neoplasms for which surgery is the treatment of choice. Long-term tumor control with preservation of neurological function is achievable in most patients. Challenges to the evaluation and management of these lesions include accurate differentiation of these tumors from inflammatory conditions such as transverse myelitis, sarcoidosis, and demyelinating conditions; the management of patients w

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31

Gilden, Don, Randall J. Cohrs, Ravi Mahalingam, and Maria A. Nagel. Varicella Zoster Virus Infection of the Nervous System. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0149.

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Varicella zoster virus (VZV) is a human herpesvirus that causes varicella (chickenpox), after which virus becomes latent in ganglionic neurons along the entire neuraxis. Reactivation of VZV due to a decline in the cell-mediated immune response to VZV in elderly or immunocompromised individuals causes zoster (shingles), frequently complicated by chronic pain (postherpetic neuralgia) and serious neurological disease (meningoencephalitis, myelitis and VZV vasculopathy due to retrograde spread of virus after zoster. Here, we describe clinical, laboratory and pathological features of neurological c

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32

Wingerchuk, Dean M. Neuromyelitis Optica Spectrum Disorders. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199341016.003.0023.

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Neuromyelitis optica (NMO) is an inflammatory demyelinating central nervous system disease. It has been classically defined as a monophasic, isolated co-occurrence of optic neuritis and transverse myelitis with uncertain relationship to multiple sclerosis. In the past decade, however, NMO has emerged as a distinct disorder associated with serum antibodies that target the astrocyte water channel aquaporin-4, distinguishing it from multiple sclerosis. The specificity of aquaporin-4 antibodies has led to appreciation of a wider spectrum of clinical and neuroimaging features, termed NMO spectrum d

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33

Solomon, Tom, and Benedict Michael. Neurological infection. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0229.

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Neurological infections can be broadly subdivided into chronic/subacute and acute. Chronic/subacute infection usually presents with global cognitive decline, with the prototypical disease being progressive multifocal leucoencephalopathy due to infection with the JC virus in immunocompromised patients. Acute neurological infections can be defined microbiologically, by the nature of the pathogen; clinically, by the presenting signs and symptoms and initial CSF findings; or anatomically. The anatomical definitions are those occurring intracranially (‘meningitis’, where infection involves the meni

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34

Graham, Andrew, and Clare Galton. Nervous system. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0018.

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Rheumatological conditions may be complicated by a variety of both central and peripheral nervous system disorder. Common complications such as entrapment neuropathies are familiar to rheumatologists but accurate diagnosis of less common neurological disorders may be challenging; careful clinical reasoning is essential, supplemented where necessary by imaging, neurophysiology, and other special investigations including cerebrospinal fluid examination. Complications vary according to the nature of background condition. In rheumatoid arthritis, neurological involvement is typically related to th

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35

Rethinking Possible: A Memoir of Resilience. She Writes Press, 2017.

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36

Current issues in clinical neurovirology: Pathogenesis, diagnosis and treatment. Saunders, 2008.

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