Relevant bibliographies by topics / Myocardial

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Myocardial'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "Myocardial"

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A. Meenakshi, Martin, and Erik G. Seth. "Protective role of TAT-HSP70 after myocardial I/R injury." American Journal of BioMedicine 5, no. 3 (2017): 279–84. http://dx.doi.org/10.18081/2333-5106/015-04/289-294.

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Myocardial ischemia reperfusion injury I/R adversely affects cardiac function. Heat shock proteins (HSPs) are a highly conserved family of proteins with diverse functions expressed by all cells exposed to environmental stress including myocardila injury. We investigated release of small constitutive heat shock proteins (HSPs) from mouse myocardium and the effects of TAT-HSP70 after myocardial I/R via occluding the left coronary artery (LAD). The results support the hypothesis that elevated HSPs in myocardium after ischemia and reperfusion and contributes to the inflammatory mechanism of myocar
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Micic-Labudovic, Jelena, Tatjana Atanasijevic, Vesna Popovic, Zoran Mihailovic, Slobodan Nikolic, and Dragana Puzovic. "Myocardial bridges: A prospective forensic autopsy study." Srpski arhiv za celokupno lekarstvo 143, no. 3-4 (2015): 153–57. http://dx.doi.org/10.2298/sarh1504153m.

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Introduction. When the coronary artery, located subepicardially, submerges into the myocardium and appears again subepicardially after a short intramural course, it represents an embedded coronary artery, while the part of the myocardium above is a myocardial bridge. Objective. We investigated the frequency of the embedded left coronary artery (LAD) in the autopsy material considering the descending branch of the LAD to be the most important one in the nourishment of the myocardium and myocardial bridges to be the most frequent in its area, as well as clinically important. Methods. A prospecti
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Zhong, Ze, Jia-qing Hu, Xin-dong Wu, Yong Sun, and Jun Jiang. "Anti-apoptotic effects of myocardin-related transcription factor-A on rat cardiomyocytes following hypoxia-induced injury." Canadian Journal of Physiology and Pharmacology 94, no. 4 (2016): 379–87. http://dx.doi.org/10.1139/cjpp-2014-0461.

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Myocardin-related transcription factor-A (MRTF-A) can transduce both biomechanical and humoral signals, which can positively modulate cardiac damage induced by acute myocardial infarction. However, the molecular mechanism that underlies the contribution that MRTF-A provides to the myocardium is not completely understood. The objective of this study was to investigate the effects of MRTF-A on myocardium apoptosis and its mechanisms. Our experiment results showed that MRTF-A expression increased and Bcl-2 expression reduced during myocardial ischemia–reperfusion in rat. Meanwhile, primary cardio
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Khubulava, G. G., A. N. Shishkevich, S. S. Mikhailov, and E. Yu Bessonov. "Myocardial reperfusion syndrome. Pathogenesis, clinic, diagnosis." Bulletin of the Russian Military Medical Academy 22, no. 1 (2020): 196–200. http://dx.doi.org/10.17816/brmma25992.

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The basics of pathogenesis, clinic and diagnosis of myocardial reperfusion syndrome are considered. Myocardial reperfusion syndrome is defined. Its relevance as one of the most poorly studied and formidable complications of cardiac reperfusion in myocardial infarction with elevation of the S-T segment has been explained. A brief review of the historical review of this problem and such types of manifestations of myocardial reperfusion syndrome as: diastolic myocardial dysfunction, post-reperfusion disturbances of the heart rhythm, the phenomenon of no-reflow and irreversible damage to the myoca
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Bhattacharya, Aniket, Nadia Al-Sammarraie, Mengistu G. Gebere, John Johnson, John F. Eberth та Mohamad Azhar. "Myocardial TGFβ2 Is Required for Atrioventricular Cushion Remodeling and Myocardial Development". Journal of Cardiovascular Development and Disease 8, № 3 (2021): 26. http://dx.doi.org/10.3390/jcdd8030026.

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Among the three transforming growth factor beta (TGFβ) ligands, TGFβ2 is essential for heart development and is produced by multiple cell types, including myocardium. Heterozygous mutations in TGFB2 in patients of connective tissue disorders result in congenital heart defects and adult valve malformations, including mitral valve prolapse (MVP) with or without regurgitation. Tgfb2 germline knockout fetuses exhibit multiple cardiac defects but the role of myocardial-TGFβ2 in heart development is yet to be elucidated. Here, myocardial Tgfb2 conditional knockout (CKO) embryos were generated by cro
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Hirsch, Alan T., John A. Opsahl, Mary M. Lunzer, and Stephen A. Katz. "Active renin and angiotensinogen in cardiac interstitial fluid after myocardial infarction." American Journal of Physiology-Heart and Circulatory Physiology 276, no. 6 (1999): H1818—H1826. http://dx.doi.org/10.1152/ajpheart.1999.276.6.h1818.

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The renin-angiotensin system promotes cardiac hypertrophy after myocardial infarction. The purpose of this study was to measure renin and angiotensinogen in plasma and myocardium 10 days after myocardial infarction. Infarction involving 45 ± 4% of left ventricular circumference with accompanying hypertrophy was induced in rats ( n = 14). Plasma and myocardial renin were increased after infarction compared with sham controls ( n = 8) (27.4 ± 3.2 vs. 7.5 ± 1.8 ng ANG I ⋅ ml plasma ⋅ h−1, P < 0.0002; and 8.8 ± 1.6 vs. 2.5 ± 0.1 ng ANG I ⋅ g myocardium−1 ⋅ h−1, P < 0.008, respectively). Afte
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Brown, TA. "Hibernating myocardium." American Journal of Critical Care 10, no. 2 (2001): 84–91. http://dx.doi.org/10.4037/ajcc2001.10.2.84.

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According to estimates, up to 50% of patients with coronary artery disease and impaired left ventricular function have areas of viable myocardium. This dysfunctional, yet viable myocardial tissue, which can improve functionally after myocardial oxygen supply is reestablished, has been called hibernating myocardium. The possible pathophysiological mechanism that leads to hibernating myocardium is controversial: is the phenomenon due to persistent ischemia or is it the result of repetitive episodes of ischemia and reperfusion, such as myocardial stunning? Regardless of the mechanism, the presenc
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Li, Hongyu, Jimiao Qiu, Chang Liu, et al. "MicroRNA-221 protects myocardial contractility in myocardial ischemia/reperfusion injury through phospholamban." PLOS ONE 20, no. 1 (2025): e0316887. https://doi.org/10.1371/journal.pone.0316887.

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Objective To investigate the effects and mechanisms of miRNA 221 on myocardial ischemia/reperfusion injury (MIRI) in mice through the regulation of phospholamban (PLB) expression. Methods The MIRI mouse model was created and mice were divided into sham, MIRI, MIRI+ 221, and MIRI+ scr groups, with miRNA 221 overexpression induced in the myocardium of MIRI mice by targeted myocardial injection. Quantitative RT-PCR analysis was performed to observe the variation in miRNA 221, PLB, SERCA2, RYR2, NCX1, Cyt C and caspase 3 mRNA levels in myocardium, while Western blot assessed the levels of PLB, p-P
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Abdrahmanova, A. I., N. B. Amirov, and N. A. Cibulkin. "Application of Perfusion Single Photon Emission Computed Tomography of the Myocardium in Pain-Free Myocardial Ischemia." Russian Archives of Internal Medicine 10, no. 5 (2020): 340–47. http://dx.doi.org/10.20514/2226-6704-2020-10-5-340-347.

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This literature review provides data on the use of single-photon emission computed tomography of myocardium in silent myocardial ischemia. The presence of silent myocardial ischemia increases the risk of cardiovascular complications several times and may be the first manifestation of coronary heart disease. Assessing the state of morphofunctional processes in the myocardium is the main goal of diagnostic imaging using singlephoton emission computed tomography of the myocardium. This allows to get three-dimensional image of left ventricle with information about distribution of perfusion volume
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Berry, Mark F., Adam J. Engler, Y. Joseph Woo, et al. "Mesenchymal stem cell injection after myocardial infarction improves myocardial compliance." American Journal of Physiology-Heart and Circulatory Physiology 290, no. 6 (2006): H2196—H2203. http://dx.doi.org/10.1152/ajpheart.01017.2005.

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Cellular therapy for myocardial injury has improved ventricular function in both animal and clinical studies, though the mechanism of benefit is unclear. This study was undertaken to examine the effects of cellular injection after infarction on myocardial elasticity. Coronary artery ligation of Lewis rats was followed by direct injection of human mesenchymal stem cells (MSCs) into the acutely ischemic myocardium. Two weeks postinfarct, myocardial elasticity was mapped by atomic force microscopy. MSC-injected hearts near the infarct region were twofold stiffer than myocardium from noninfarcted
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Dissertations / Theses on the topic "Myocardial"

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Egan, Jonathan Rogers. "The role of myocardial membrane proteins and myocardial oedema in postoperative myocardial dysfunction." Thesis, The University of Sydney, 2009. http://hdl.handle.net/2123/5975.

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The vast majority of children undergoing surgical repair of cardiac lesions do spectacularly well. However a significant proportion, ~ 25%, struggle to progress in the early postoperative period and require additional pharmacological and occasionally mechanical circulatory support. All children typically have some degree of postoperative myocardial dysfunction, with the severe spectrum termed the low cardiac output state (LCOS). LCOS is clinically defined as the requirement for new or escalated inotrope therapy, a widened arteriovenous oxygen difference, cardiac arrest or the need for reinstit
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Egan, Jonathan Rogers. "The role of myocardial membrane proteins and myocardial oedema in postoperative myocardial dysfunction." Faculty of Medicine, 2009. http://hdl.handle.net/2123/5975.

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Doctor of Philosophy(PhD)<br>The vast majority of children undergoing surgical repair of cardiac lesions do spectacularly well. However a significant proportion, ~ 25%, struggle to progress in the early postoperative period and require additional pharmacological and occasionally mechanical circulatory support. All children typically have some degree of postoperative myocardial dysfunction, with the severe spectrum termed the low cardiac output state (LCOS). LCOS is clinically defined as the requirement for new or escalated inotrope therapy, a widened arteriovenous oxygen difference, cardiac ar
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Löwbeer, Christian. "Cardiac troponin T in clinical and experimental studies /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-426-6/.

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Zhou, Xiaopeng. "Myocardial T1 Mapping Techniques for Quantification of Myocardial Fibrosis." Cleveland State University / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=csu1355418392.

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Park, Jade. "Myocardial fibrosis and effect of AZT in myocardium of Y995CB mouse." Thesis, Boston University, 2012. https://hdl.handle.net/2144/12581.

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Thesis (M.A.)--Boston University PLEASE NOTE: Boston University Libraries did not receive an Authorization To Manage form for this thesis or dissertation. It is therefore not openly accessible, though it may be available by request. If you are the author or principal advisor of this work and would like to request open access for it, please contact us at open-help@bu.edu. Thank you.<br>Pyrimidine nucleoside reverse transcriptase inhibitors (NRTIs), one of the primary classes of HIV/AIDS antiretroviral drugs, are known to cause mitochondrial toxicity by inhibiting polymerase gamma during extend
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Dwivedi, Girish. "A Comparison between Myocardial Contrast Echocardiography and Radionuclide Myocardial perfusion Imaging in Patients with Acute Myocardial Infarction." Thesis, University of Manchester, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.521583.

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Treibel, Thomas Alexander. "Aortic stenosis : a myocardial disease : insights from myocardial tissue characterisation." Thesis, University College London (University of London), 2017. http://discovery.ucl.ac.uk/1574742/.

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Aortic stenosis (AS) is a disease of not just the valve, but also of the myocardium. Patient symptoms and outcome are determined by the myocardial response; a crucial but poorly understood process. Diffuse and focal myocardial fibrosis play a key role. Until recently, both could only be assessed using invasive histology, but now cardiovascular magnetic resonance (CMR) offers late gadolinium enhancement (LGE) and extracellular volume fraction (ECV) techniques. In this thesis, I developed new methods to quantify ECV by synthetic ECV and cardiac CT. I then explored myocardial remodelling and fibr
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White, Melanie Yvonne. "Proteomics of ischemia/reperfusion injury in rabbit myocardium." Thesis, The University of Sydney, 2006. https://hdl.handle.net/2123/27890.

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Myocardial stunning is best defined as the persistent, yet reversible, contractile dysfunction that occurs with brief myocardial ischemia / reperfusion (I/R) injury. In contrast, prolonged ischemia results in myocardial infarction that leads to cell death of necrosis of the tissue. The causes of stunning are not fully elucidated, however two major hypotheses currently exist; firstly changes to calcium handling resulting from lowered cellular pH by means of anaerobic respiration, and altered Nair/H)r antiporter kinetics, and secondly, the generation oxygen free radical (OFR) that may occu
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Singh, Hardial. "Quantitative assessment of myocardial ischaemia with thallium-201 myocardial perfusion imaging." Thesis, University of Edinburgh, 1986. http://hdl.handle.net/1842/19297.

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Frostfeldt, Gunnar. "Coagulation Inhibition and Development of Myocardial Damage in ST-Elevation Myocardial Infarction." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2002. http://publications.uu.se/theses/91-554-5322-8/.

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Books on the topic "Myocardial"

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Salerno, Tomas A., and Marco Ricci, eds. Myocardial Protection. Blackwell Publishing, 2003. http://dx.doi.org/10.1002/9780470987452.

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Iskandrian, Ami E., and Ernst E. Van Der Wall, eds. Myocardial Viability. Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-011-4080-5.

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Kaski, Juan Carlos, and David W. Holt, eds. Myocardial Damage. Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-017-2380-0.

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Cokkinos, Dennis V., ed. Myocardial Preservation. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-98186-4.

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Iskandrian, Abdulmassih S., and Ernst E. Van Der Wall, eds. Myocardial viability. Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1170-6.

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Dhalla, Naranjan S., Ian R. Innes, and Robert E. Beamish, eds. Myocardial Ischemia. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-2055-5.

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Wainwright, Cherry L., and James R. Parratt. Myocardial Preconditioning. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-662-22206-5.

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Cokkinos, Dennis V., Constantinos Pantos, Gerd Heusch, and Heinrich Taegtmeyer, eds. Myocardial Ischemia. Springer US, 2006. http://dx.doi.org/10.1007/0-387-28658-6.

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1960-, Wainwright Cherry, and Parratt James R, eds. Myocardial preconditioning. Springer, 1996.

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R, Gray Ian, and General Practitioner Writers Association, eds. Myocardial medley. Radcliffe Medical Press, 1990.

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Book chapters on the topic "Myocardial"

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Cokkinos, Dennis V. "Myocardial Hibernation." In Myocardial Preservation. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-98186-4_10.

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Cokkinos, Dennis V. "Myocardial Stunning." In Myocardial Preservation. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-98186-4_9.

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Pantos, Constantinos, Iordanis Mourouzis, and Dennis V. Cokkinos. "Myocardial Ischemia." In Myocardial Ischemia. Springer US, 2006. http://dx.doi.org/10.1007/0-387-28658-6_2.

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Schwaiger, Markus, and Ulrich Schricke. "Hibernating and stunned myocardium: Pathophysiological considerations." In Myocardial Viability. Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-011-4080-5_1.

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Van Der Wall, Ernst E., Jeroen J. Bax, Hubert W. Vliegen, Albert V. G. Bruschke, and Albert De Roos. "Role of magnetic resonance techniques in viability assessment." In Myocardial Viability. Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-011-4080-5_10.

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Iskandrian, Ami E. "Viability assessment: clinical applications." In Myocardial Viability. Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-011-4080-5_11.

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Iskandrian, Ami S., and Ernst E. Van Der Wall. "Summary." In Myocardial Viability. Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-011-4080-5_12.

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Baliga, Ragavendra R., Jutta Schaper, and Jagat Narula. "Role of apoptosis in myocardial hibernation and myocardial stunning." In Myocardial Viability. Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-011-4080-5_2.

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Schelbert, Heinrich R. "Assessment of myocardial viability with positron emission tomography." In Myocardial Viability. Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-011-4080-5_3.

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Bax, Jeroen J., Jean-Louis J. Vanoverschelde, and Ernst E. Van Der Wall. "Assessment of myocardial viability by thallium-201." In Myocardial Viability. Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-011-4080-5_4.

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Conference papers on the topic "Myocardial"

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N, Pusphalatha, Sri Dhananjayan K, Senthooriya O. S, Tharunprasath K, J.Arulvadivu, and Gowtham M. "Detection of Mishap and Myocardial Infraction." In 2024 International Conference on Recent Innovation in Smart and Sustainable Technology (ICRISST). IEEE, 2024. https://doi.org/10.1109/icrisst59181.2024.10921953.

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Damit, Dayang Suhaida Awang, A’Isyah Nadiyah Hilmi, Siti Noraini Sulaiman, Muhammad Khusairi Osman, Noor Khairiah A. Karim, and Nor Adni Mat Leh. "Impact of Class Labeling on Myocardium Segmentation using Cascaded Deep Learning for Improved Myocardial Infarction Segmentation." In 2024 IEEE 14th International Conference on Control System, Computing and Engineering (ICCSCE). IEEE, 2024. http://dx.doi.org/10.1109/iccsce61582.2024.10696767.

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Discher, Dennis, and Adam Engler. "Mesenchymal Stem Cell Injection After Myocardial Infarction Improves Myocardial Compliance." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176754.

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Cellular therapy for myocardial injury has improved ventricular function in both animal and clinical studies, though the mechanism of benefit is unclear. This study was undertaken to examine the effects of cellular injection after infarction on myocardial elasticity. Coronary artery ligation of Lewis rats was followed by direct injection of human mesenchymal stem cells (MSC) into the acutely ischemic myocardium. Two weeks post-infarct, myocardial elasticity was mapped by atomic force microscopy. MSC-injected hearts near the infarct region were two-fold stiffer than myocardium from non-infarcte
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Tracy Ling, Yik Tung, Vincent Sayseng, and Elisa Konofagou. "Myocardial Elastography for Evaluating the Evolution of Strains and Strain Rates in Canine Myocardium After Myocardial Infarction." In 2022 IEEE International Ultrasonics Symposium (IUS). IEEE, 2022. http://dx.doi.org/10.1109/ius54386.2022.9957783.

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Veress, A. I., A. Giannakidis, and G. T. Gullberg. "Mechanical Effects of Myofibril Disarray on Cardiac Function." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14696.

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Myocardial disarray is a fiber distribution that deviates away from the tightly organized, parallel alignment of myocardial fibers that characterizes the normal myocardium. This coherently-organized distribution of the myofibers results in the twisting contraction of the normal left ventricle (LV). With myofiber disarray, the fibers have random directionality, either locally or globally, within the LV.
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Zhang, Song, John A. Crow, Robert C. Cooper, et al. "Detection of Myocardial Fiber Disruption in Artificial Lesions With 3D DT-MRI Tract Models." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193121.

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In the United States, it is estimated that in 2008 approximately 1.2 million people will suffer a new or recurrent myocardial infarction. In 2005, the latest full year for which statistics are available, 16 million Americans (7.3% of the population) had some form of coronary heart disease. Loss of myocardium as a result of myocardial infarction increases wall stress locally and globally and triggers adaptive responses at the molecular, cellular, and tissue levels. These adaptive responses can lead to left ventricular dilation and congestive heart failure. Accurate non-invasive evaluation of my
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Hu, Zhenhua, Dimitris Metaxas, and Leon Axel. "Heart Composite Material Model for Stress-Strain Analysis." In ASME 2003 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2003. http://dx.doi.org/10.1115/detc2003/vib-48334.

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Mechanical properties of the myocardium have been investigated intensively in the past four decades. Due to the non-linearity and history dependence of myocardial deformation, many complex strain energy functions have been used to describe the stress-strain relationship in the myocardium. These functions are good at fitting in-vitro experimental data from myocardial stretch testing into strain energy functions. However, it is difficult to model in-vivo myocardium by using strain energy functions. In a previous paper [1], we have implemented a transversely anisotropic material model to estimate
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Mehri, Sounira, Wided Khamlaoui, and Mohamed Hammami. "Acute myocardial infarction." In the Fourth International Conference. ACM Press, 2018. http://dx.doi.org/10.1145/3234698.3234741.

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Singelyn, J. M., J. A. DeQuach, and K. L. Christman. "Injectable myocardial matrix as a scaffold for myocardial tissue engineering." In 2009 Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2009. http://dx.doi.org/10.1109/iembs.2009.5334839.

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Bradley, Joshua, J. Bradley, EB Schelbert, et al. "31 Remote myocardial fibrosis predicts adverse outcome following myocardial infarction." In British Society of Cardiovascular Magnetic Resonance (BSCMR) Annual Congress 2022. BMJ Publishing Group Ltd and British Cardiovascular Society, 2023. http://dx.doi.org/10.1136/heartjnl-2022-bscmr.30.

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Reports on the topic "Myocardial"

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Moridi, Mina, Parinaz Onikzeh, Aida Kazemi, and Hadi Zamanian. CABG versus myotomy in symptomatic myocardial bridge patients : A systematic Review and Meta-analysis protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.11.0088.

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Review question / Objective: The aim of this study is to find which surgical intervention in myocardial bridge ( myotomy or CABG) is more effective in reducing adverse outcomes in symptomatic patients resistant to optimal medical therapy ? Condition being studied: Myocardial bridge : A myocardial bridge (MB) is a congenital heart defect in which a bridge of muscle fibers (myocardium) overlying a section of a coronary artery and the artery is squeezed and normal blood flow is disrupted. Most bridges don't seem to cause symptoms. However, some people can experience angina, or chest pain. In pati
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Li, Xiao, Fayang Ling, Wenchuan Qi, et al. Preclinical Evidence of Acupuncture on infarction size of Myocardial ischemia: A Systematic Review and Meta-Analysis of Animal Studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.6.0044.

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Review question / Objective: Whether acupuncture is effective for infarction size on myocardial ischemia rat models. Condition being studied: Myocardial ischemia is a typical pathological condition of coronary heart disease (CHD), which has been a global issue with high incidence and mortality. Myocardial infarction caused by myocardial ischemia leads to cardiac dysfunction, and the size of myocardial infarction also determines the recovery and prognosis of cardiac function. Acupuncture, a long history of traditional Chinese medicine, is widely used to treat symptoms like thoracalgia and palpi
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Voynalovich-Khanova, Y. A. SYNDROME OF MYOCARDIAL REMODELING (CLINICAL OBSERVATION). "PLANET", 2019. http://dx.doi.org/10.18411/978-5-907192-54-6-2019-xxxvi-46-49.

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McDonough, Kathleen H., and Harvey I. Miller. Myocardial Dysfunction Contributes to Irreversible Hemorrhagic Shock. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada389358.

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Pickard, Jeb S., and Joe E. Burton. Flying Waivers for History of Angioplasty and Myocardial Infraction. Defense Technical Information Center, 1994. http://dx.doi.org/10.21236/ada292505.

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Hassanzadeh, Sara, Sina Neshat, Afshin Heidari, and Masoud Moslehi. Myocardial Perfusion Imaging in the Era of COVID-19. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.4.0063.

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Review question / Objective: This review studies all aspects of myocardial perfusion imaging with single-photon emission computed tomography (MPI SPECT) after the COVID-19 pandemic. Condition being studied: Many imaging modalities have been reduced after the COVID-19 pandemic. Our focus in this review is to see if the number of MPIs is lowered or not and, if so, why. Furthermore, it is possible that a combination of CT attenuation correction and MPI could yield findings. In this study, we'll also look for these probable findings. Third, we know from previous studies that COVID might cause card
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Dejong, Marla J., Kyungeh An, Candace C. Cherrington, and Debra K. Moser. Predictors of Symptom Appraisal for Patients with Acute Myocardial Infarction. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada427523.

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Crowley, James P., C. R. Valeri, and Joseph Chazan. Myocardial Infarction and Transfusion Requirements in Transfusion Dependent Anemic Patients. Defense Technical Information Center, 1990. http://dx.doi.org/10.21236/ada360239.

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QIN, Xiaoyu, Chunai WANG, Jie ZHANG, Shuwei WANG, and Weiqi ZHANG. Effectiveness and safety of electroacupuncture for myocardial protection in cardiopulmonary bypass patients with myocardial ischemia-reperfusion injury：a protocol for a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.4.0045.

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Naydenov, Stefan, Nikolay Runev, Emil Manov, Nadya Naydenova, Mikhail Matveev, and Plamen Krastev. Diagnostic Potential of Signal-Averaged Orthogonal Electrocardiography in Acute Myocardial Infarction. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2021. http://dx.doi.org/10.7546/crabs.2021.02.16.

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