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1

BEN, HADJALI BEN KHALIFA JAMEL. "Les myoclonies squelettiques." Lille 2, 1991. http://www.theses.fr/1991LIL2M234.

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2

Lantéri-Minet, Michel. "Traitement et pathogenie des myoclonies post-hypoxiques : etude prospective de l'effet du piracetam." Nice, 1991. http://www.theses.fr/1991NICE6829.

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3

GRANDSIRE, DIDIER. "Les myoclonies neonatales benignes du sommeil : un diagnostic differentiel des convulsions du nouveau-ne." Amiens, 1990. http://www.theses.fr/1990AMIEM082.

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4

Vellieux, Geoffroy. "Syndrome de Lance-Adams : étude translationnelle de l’homme à l’animal." Electronic Thesis or Diss., Sorbonne université, 2024. http://www.theses.fr/2024SORUS432.

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Le syndrome de Lance-Adams est une pathologie neurologie chronique très handicapante rencontrée chez les survivants d'anoxie cérébrale. Il est caractérisé essentiellement par des myoclonies positives, d'action, multifocales ou généralisées, et des myoclonies négatives. Les mécanismes sous-tendant les myoclonies de cette pathologie sont peu connus. De multiples hypothèses ont été proposées depuis la description initiale de ce syndrome. L'étude multimodale d'une large cohorte de patients avec un syndrome de Lance-Adams a montré que les myoclonies sont générées dans le cortex cérébral, en particu
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5

Carr, Jonathan. "Familial adult myoclonus epilepsy : a clinical, neurophysiological and genetic study of a familial form of myoclonic epilepsy." Thesis, Stellenbosch : University of Stellenbosch, 2009. http://hdl.handle.net/10019.1/1201.

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Thesis (DMed (Medicine. Internal Medicine))--University of Stellenbosch, 2009.<br>ENGLISH ABSTRACT: Progressive Myoclonic Epilepsies (PME) are characterized by progressive neurological impairment with myoclonus, seizures and dementia. In contradistinction, Familial Adult Myoclonic Epilepsy (FAME) is characterized by a benign course with rare seizures and cortical tremor. Both conditions have neurophysiological features suggestive of a cortical origin for their myoclonus. This dissertation reports on a novel form of PME. Many of those who were affected had no or minimal progression of the
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6

Lafrenière, Ronald G. "Molecular and genetic studies of progressive myoclonus epilepsy type 1 (EPM1)." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape16/PQDD_0021/NQ36996.pdf.

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7

Waite, Adrian. "The molecular genetics of myoclonus-dystonia syndrome." Thesis, University of Oxford, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.531805.

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8

Elmslie, Frances Veryan. "Molecular genetic analysis of juvenile myoclonic epilepsy." Thesis, University of Bristol, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.299535.

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9

DASSO, MARTIN BRIGITTE. "Encephalopathie myoclonique precoce a bouffees electroencephalographiques periodiques : a propos de trois observations." Toulouse 3, 1988. http://www.theses.fr/1988TOU31062.

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10

VAUGIN-BOULANGER, VERONIQUE. "Effets indesirables des nouvelles quinolones." Clermont-Ferrand 1, 1989. http://www.theses.fr/1989CLF13005.

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11

Houdayer, Elise. "Rôle des informations somesthésiques dans la modulation de l'excitabilité des aires corticales motrices et influence de la stimulation magnétique répétitive." Lille 2, 2007. http://www.theses.fr/2007LIL2S025.

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La réalisation d'un mouvement volontaire implique la mise en jeu de structures cérébrales responsables des étapes de planification, de programmation, d'exécution et d'arrêt du mouvement. Par leur action via des circuits intracorticaux excitateurs et inhibiteurs, les influx sensoriels afférents générés au cours du mouvement semblent jouer un rôle primordial dans l'exécution et l'arrêt du mouvement, en limitant en particulier la survenue de mouvements parasites. Les objectifs de ce travail de thèse étaient d'étudier l'activité électrique des aires cérébrales impliquées dans le contrôle moteur et
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12

Read, Tara. "Elucidating a novel gene associated with myoclonus dystonia." Thesis, University of Ottawa (Canada), 2009. http://hdl.handle.net/10393/28248.

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Myoclonus Dystonia (MD) is an autosomal dominant disease with high but incomplete penetrance and is characterized by both involuntary myoclonic jerks and dystonic posturing. Our group has found that mutations within the epsilon sarcoglycan (SGCE) gene on chromosome 7q21 are associated with MD in 30-40% of affected individuals in 31 families studied, supporting the basis for genetic heterogeneity. Novel mutations have been found in SGCE by screening these families for point mutations and large deletions and duplications through the use of sequencing, high performance liquid chromatography (HPLC
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13

Russell, Jonathan Foster. "Familial Cortical Myoclonus Caused by Mutation in NOL3." Thesis, University of California, San Francisco, 2013. http://pqdtopen.proquest.com/#viewpdf?dispub=3558440.

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<p> Many neurologic diseases cause discrete episodic impairment. Study of the genes and mechanisms underlying these diseases has informed our understanding of the nervous system. Here we describe a novel episodic neurologic disorder, which we term familial cortical myoclonus (FCM). FCM is characterized by adult onset, slowly progressive, multifocal, cortical myoclonus, inherited as an autosomal dominant trait. On the basis of clinical, electrophysiological, and genetic data, FCM is nosologically distinct. We utilized genome-wide single nucleotide polymorphism genotyping, microsatellite linkage
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14

DELOLME, CORINNE. "Syndrome de kinsbourne (syndrome ataxo-opso-myoclonique) : son evolution etudiee a partir de 45 observations personnelles." Lyon 1, 1992. http://www.theses.fr/1992LYO1M212.

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15

LECLERE, SYLVIE. "Epilepsie myoclonique progressive et localisations cerebrales d'un lymphome : etude d'un cas." Clermont-Ferrand 1, 1993. http://www.theses.fr/1993CLF1M013.

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16

Tang, Shan. "Expanding the phenotype and genetic spectrum of myoclonic astatic epilepsy." Thesis, King's College London (University of London), 2017. https://kclpure.kcl.ac.uk/portal/en/theses/expanding-the-phenotype-and-genetic-spectrum-of-myoclonic-astatic-epilepsy(991de1f3-e1a5-49d7-b68a-43abb859fb39).html.

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Myoclonic astatic epilepsy (MAE) is a rare generalised childhood epilepsy with variable but poorly described neurodevelopmental outcome. Family studies suggest a major genetic influence as up to two thirds of relatives have seizures, or electroencephalographic (EEG) abnormalities. MAE is associated with 10 different genes, yet these genes account for less than 20% of the genetic aetiology of MAE leaving the majority unexplained. The aims of this thesis were (1) describe the epilepsy and neurodevelopmental phenotype of MAE cases, (2) perform EEG studies on first degree family members for famili
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17

Lowe, Simon. "Using Drosophila to model Down syndrome and progessive myoclonus epilepsy." Thesis, University of Bristol, 2017. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.752734.

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18

McDonald, Lita E. C. "The establishment of in vivo and in vitro models for myoclonus dystonia." Thesis, University of Ottawa (Canada), 2007. http://hdl.handle.net/10393/27889.

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Myoclonus dystonia (DYT11, OMIM 159900) (MD) is a movement disorder characterized by bilateral alcohol responsive myoclonic jerks often seen in combination with dystonia. MD is inherited as an autosomal dominant trait with reduced penetrance upon maternal transmission. Patients with this disorder are not diminished in their intellectual capacity and have a normal life span. In 2001, mutations in the epsilon-sarcoglycan gene on human chromosome 7q21 were implicated in causing this disorder. Our laboratory identified a 2nd locus on chromosome 18p11 that co-segregates with this disorder, however,
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19

Van, Coller Riaan. "A clinical, neurophysiological and genetic study of South African familial combined myoclonic syndromes." Thesis, University of Pretoria, 2021. http://hdl.handle.net/2263/78827.

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Myoclonus is a complex disorder of rapid repetitive muscle jerks that can occur in proximal or distal appendicular or axial muscles. It can be of cortical, sub-cortical or spinal cord origin; part of progressive and severely disabling epilepsy syndromes, basal ganglia conditions, and physiological or even functional (psychogenic)1. A systematic review of the literature shows the knowledge gap of the genetic causes of myoclonus in South Africa with 25 identified publications from Africa of which eleven were from South Africa. Publications varied from case studies to case series and included
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20

Zanarella, Erica. "Functional analysis of EFHC1, a gene involved in Juvenile Myoclonic Epilepsy, in Drosophila." Doctoral thesis, Università degli studi di Padova, 2011. http://hdl.handle.net/11577/3421984.

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Mutations in the EFHC1 gene, encoding a novel microtubule binding protein, have been linked to juvenile myoclonic epilepsy. EFHC1 has been proposed to regulate cell division by controlling mitotic spindle organization and cerebral cortex lamination by modulating neuroblast migration. To understand EFHC1 function in vivo we generated knock-out Drosophila for the fly homolog Defhc1. We found that the NMJ synapse of Defhc1 mutants display an increased number of satellite boutons and increased spontaneous neurotransmitter release. Defhc1 binds to microtubules in vitro and overlaps in vivo with axo
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21

Guipponi, Michel. "Cartographie génétique de facteurs de susceptibilité à deux syndromes épileptiques idiopathiques : les convulsions infantiles familiales bénignes et l'épilepsie myoclonique juvénile." Montpellier 1, 1996. http://www.theses.fr/1996MON1T009.

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22

Mory, Susana Barretto. "Alteração morfofuncional talamica em epilepsia mioclonica juvenil." [s.n.], 2009. http://repositorio.unicamp.br/jspui/handle/REPOSIP/310347.

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Orientadores: Li Li Min, Fernando Cendes<br>Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas<br>Made available in DSpace on 2018-08-14T05:26:43Z (GMT). No. of bitstreams: 1 Mory_SusanaBarretto_D.pdf: 7381839 bytes, checksum: 24b63af2fd5b4745e23d8c1487300689 (MD5) Previous issue date: 2009<br>Resumo: A epilepsia mioclônica juvenil é o subtipo mais frequente entre as epilepsias generalizadas idiopáticas. Apresenta bom prognóstico e fácil controle em relação às crises epilépticas. O nosso objetivo foi a análise do tálamo combinada com métodos funcional e estru
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23

Preto, Paula Maria. "Aspectos clínicos e eletrencefalográficos da Síndrome de Dravet e da Síndrome de Doose." [s.n.], 2010. http://repositorio.unicamp.br/jspui/handle/REPOSIP/311191.

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Orientadores: Marilisa Mantovani Guerreiro, Maria Augusta Montenegro<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas<br>Made available in DSpace on 2018-08-16T12:47:22Z (GMT). No. of bitstreams: 1 Preto_PaulaMaria_M.pdf: 6934972 bytes, checksum: e8531a69068b5848bc523e1dce28ee1c (MD5) Previous issue date: 2010<br>Resumo: A Epilepsia Mioclônica Grave da Infância ou Síndrome de Dravet (SDr) e a Epilepsia Miclônico-Astática ou Síndrome de Doose (SDo) são epilepsias raras da infância, que cursam com crises epilépticas refratárias, nas quais há predomín
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24

Jamieson-Williams, Rhiannon. "Investigating the Relationship and Potential Interactions of CD108131 and SGCE." Thesis, Université d'Ottawa / University of Ottawa, 2019. http://hdl.handle.net/10393/39418.

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Myoclonus dystonia (MD) is a rare autosomal-dominant combined dystonia movement disorder characterised by quick, involuntary muscle jerks (myoclonus) paired with sustained muscular contraction (dystonia). Although known to be genetically heterogeneous, the most common genetic factor is mutations within SGCE, the gene encoding ε-sarcoglycan, accounting for approximately 45% of cases. Previous linkage analyses conducted on a family displaying inherited MD without SGCE mutations lead to the identification of another critical region, DYT15. Preliminary data suggested that mutations within the long
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25

Praschberger, Roman. "Expanding the mutational spectrum and investigating the pathophysiology of GOSR2 mediated progressive myoclonus epilepsy." Thesis, University College London (University of London), 2018. http://discovery.ucl.ac.uk/10041248/.

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In this PhD thesis I summarize my research into the genetics and pathophysiology of progressive myoclonus epilepsy (PME) associated with mutations in GOSR2. This disorder is characterized by early disease onset with ataxia around 3 years of age, followed by development of cortical myoclonus, generalized epilepsy and a rapid deterioration of motor function. Upon beginning my PhD, only one homozygous GOSR2 mutation – c.430G > T (p.G144W) – had been shown to cause PME. Furthermore, because GOSR2 encodes a Golgi SNARE protein (termed Membrin) that mediates ER-to-Golgi trafficking in every cell of
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26

Greechan, Martha. "Illness meaning and perceived support of parents of children with severe myoclonic epilepsy of infancy." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape17/PQDD_0002/MQ36451.pdf.

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27

Given, Alexis. "Models of Epsilon-Sarcoglycan Gene Inactivation and their Implications for the Pathology of Myoclonus Dystonia." Thèse, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/23790.

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Myoclonus Dystonia (MD) is an autosomal dominant movement disorder characterized by bilateral myoclonic jerks paired with dystonia 1. Mutations have been mapped to the ε-sarcoglycan (SGCE) gene in about 40% of patients 2,92. The purpose of this project was to examine the properties of SGCE in the central nervous system (CNS) and use this knowledge to elucidate the pathology of MD. Although Sgce is a member of the sarcoglycan complex (SGC) in other tissues, little is known about its interactions in the CNS. The vast majority of mutations in SGCE alter the translational reading frame. Proteins a
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28

Oelbe, geb Wagner Miriam Svenja [Verfasser]. "LIMP2-Defizienz und das Action Myoclonus and Renal Failure (AMRF) Syndrom / Miriam Svenja Oelbe, geb. Wagner." Kiel : Universitätsbibliothek Kiel, 2013. http://d-nb.info/1037109430/34.

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29

Silén, Teija. "Somatosensory and motor cortical activity in patients and carriers of Unverricht-Lundborg type progressive myoclonus epilepsy." Helsinki : University of Helsinki, 2003. http://ethesis.helsinki.fi/julkaisut/laa/kliin/vk/silen/.

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30

Wieländer, Franziska Verfasser], and Andrea [Akademischer Betreuer] [Fischer. "Clinical and electroencephalographic characterization of juvenile myoclonic epilepsy in Rhodesian Ridgebacks / Franziska Wieländer ; Betreuer: Andrea Fischer." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2018. http://d-nb.info/1165503743/34.

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31

Vanstone, Megan. "Identification, Validation and Characterization of the Mutation on Chromosome 18p which is Responsible for Causing Myoclonus-Dystonia." Thèse, Université d'Ottawa / University of Ottawa, 2012. http://hdl.handle.net/10393/23486.

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Myoclonus-Dystonia (MD) is an inherited, rare, autosomal dominant movement disorder characterized by quick, involuntary muscle jerking or twitching (myoclonus) and involuntary muscle contractions that cause twisting and pulling movements, resulting in abnormal postures (dystonia). The first MD locus was mapped to 7q21-q31 and called DYT11; this locus corresponds to the SGCE gene. Our group previously identified a second MD locus (DYT15) which maps to a 3.18 Mb region on 18p11. Two patients were chosen to undergo next-generation sequencing, which identified 2,292 shared novel variants within th
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32

Labauge, Pierre. "Etude génétique des épilepsies myocloniques progressives : localisation génétique de la maladie d'Unverricht-Lundborg et de la maladie de Lafora." Montpellier 1, 1996. http://www.theses.fr/1996MON1T018.

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33

Soares, Eric. "Syndrome opso-myoclonique et ganglioneurome." Bordeaux 2, 1990. http://www.theses.fr/1990BOR25070.

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34

Oi, Kazuki. "Low-dose perampanel improves refractory cortical myoclonus by the dispersed and suppressed paroxysmal depolarization shifts in the sensorimotor cortex." Doctoral thesis, Kyoto University, 2021. http://hdl.handle.net/2433/263565.

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35

Murai, Marcelo Jun. "Expressão e purificação de proteinas relacionadas a epilepsia." [s.n.], 2007. http://repositorio.unicamp.br/jspui/handle/REPOSIP/316860.

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Orientador: Iscia Teresinha Lopes-Cendes<br>Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia<br>Made available in DSpace on 2018-08-09T12:37:49Z (GMT). No. of bitstreams: 1 Murai_MarceloJun_D.pdf: 2432622 bytes, checksum: 77473c64f1b333e9164100a3c525b735 (MD5) Previous issue date: 2007<br>Resumo: Os genes LGI1 e EFHC1 não codificam canais iônicos; entretanto, afetam indiretamente a corrente nesses canais em síndromes epilépticas determinadas geneticamente. O gene LGI1 (do inglês Leucine-rich, glioma inactivated gene 1) está relacionado à epilepsia parcial autossômi
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36

Tudesq, Nicolas. "Encéphalomyopathies mitochondriales : à propos d'un cas de MERRF avec déficit en NADH Coenzyme Q Reductase : revue de la littérature." Bordeaux 2, 1988. http://www.theses.fr/1988BOR23006.

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37

Carr, Jonathan Ambrose. "Juvenile myoclonic epilepsy : characterisation of the syndrome and examination of linkage to the HLA locus in families from the Western Cape." Master's thesis, Faculty of Health Sciences, 1997. https://hdl.handle.net/11427/31753.

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The primary purpose of this study was the identification of patients with JME, in order to identify suitable families for studying the nature of the linkage of the syndrome of JME to the HLA (Human Leukocyte Antigen) locus. This search for suitable candidate families necessitated examination of a large number of individuals with a range of IGE syndromes, and patients were classified into these various syndromes. Patients with JME were thereafter investigated further. The study could thus be broken up into three sections: i) Determination of the various syndromes of IGE. ii) Characterisation of
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38

Togo, Masaya. "Short “Infraslow” Activity (SISA) With Burst Suppression in Acute Anoxic Encephalopathy: A Rare, Specific Ominous Sign With Acute Posthypoxic Myoclonus or Acute Symptomatic Seizures." Kyoto University, 2019. http://hdl.handle.net/2433/242412.

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39

Thomas, Saralene Iona. "Genetic markers in the differential diagnosis in a family setting of episodic loss of consciousness." Thesis, Stellenbosch : Stellenbosch University, 2000. http://hdl.handle.net/10019.1/51777.

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40

Moschetta, Sylvie Carolina Paes. "Epilepsia mioclônica juvenil: avaliação das funções atencionais e executivas, traços de personalidade e adequação social." Universidade de São Paulo, 2010. http://www.teses.usp.br/teses/disponiveis/5/5142/tde-10052010-144855/.

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A Epilepsia Mioclônica Juvenil (EMJ) é uma epilepsia generalizada idiopática geralmente associada à ausência de alterações estruturais. Estudos neuropsicológicos, com um número restrito de paradigmas, sugerem que indivíduos com EMJ apresentam pior desempenho nos testes que avaliam funções executivas. De maneira complementar, a descrição da personalidade nos pacientes com EMJ corrobora os estudos neuropsicológicos, uma vez que relata maior exacerbação da impulsividade nestes pacientes, o que refletiria uma possível disfunção de lobo frontal. Estes estudos baseiam-se ou em observações clínicas o
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41

Staege, Selma [Verfasser], and Florian [Akademischer Betreuer] Wegner. "Functional and molecular properties of DYT-THAP1 dystonia and DYT-SGCE myoclonus-dystonia using patient-derived striatal medium spiny neurons / Selma Staege ; Betreuer: Florian Wegner." Hannover : Stiftung Tierärztliche Hochschule Hannover, 2021. http://d-nb.info/1237684641/34.

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42

Ciumas, Carolina. "Multimethodological brain imaging studies of human epilepsy /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-268-2/.

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43

Pataskar, Shashank S. "Structure Function Studies Of Biologically Important Simple Repetitive DNA Sequences." Thesis, Indian Institute of Science, 2000. http://hdl.handle.net/2005/261.

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The recent explosion of DNA sequence information has provided compelling evidence for the following facts. (1) Simple repetitive sequences-microsatellites and minisatellites occur commonly in the human genome and (2) these repetitive DNA sequences could play an important role in the regulation of various genetic processes including modulation of gene expression. These sequences exhibit extensive polymorphism in both length and the composition between species and between organisms of the same species and even cells of the same organism. The repetitive DNA sequences also exhibit structural polym
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44

Pataskar, Shashank S. "Structure Function Studies Of Biologically Important Simple Repetitive DNA Sequences." Thesis, Indian Institute of Science, 2001. https://etd.iisc.ac.in/handle/2005/261.

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The recent explosion of DNA sequence information has provided compelling evidence for the following facts. (1) Simple repetitive sequences-microsatellites and minisatellites occur commonly in the human genome and (2) these repetitive DNA sequences could play an important role in the regulation of various genetic processes including modulation of gene expression. These sequences exhibit extensive polymorphism in both length and the composition between species and between organisms of the same species and even cells of the same organism. The repetitive DNA sequences also exhibit structural polym
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45

Carvajal, González Alexander. "Glycine receptor antibodies : pathogenic mechanisms and clinical correlates." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:8d9e92c6-6c02-4d62-b3fc-086d8dd297a5.

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Glycine receptor antibodies have been identified in a few patients with progressive encephalomyelitis with rigidity and myoclonus (PERM), a highly disabling disorder characterised by rigidity, spasm and brainstem symptomatology. The clinical characteristics of patients with glycine receptor antibodies have not yet been fully described and it is not clear whether GlyR-Abs are pathogenic or just an epiphenomenon. This study examined the clinical features and immunotherapy responses of 45 patients; characterised the GlyR-Ab pathogenicity, subunit specificity and binding to different brain region
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46

Lin, Katia [UNIFESP]. "Estudo anátomo-funcional por ressonância magnética em pacientes com epilepsia mioclônica juvenil." Universidade Federal de São Paulo (UNIFESP), 2009. http://repositorio.unifesp.br/handle/11600/9493.

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Lobato, Mauricio Lima. "Epilepsias generalizadas idiopáticas: fatores clínicos e de neuroimagem relacionados ao difícil controle medicamentoso." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/5/5138/tde-23102018-103029/.

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As epilepsias generalizadas idiopáticas (EGIs) associam-se a controle satisfatório de crises e a exames de neuroimagem convencionais normais. Métodos de neuroimagem avançada, como DTI (diffusion tensor imaging) e VBM (voxel based morphometry), permitiram melhor compreensão dos mecanismos envolvidos no comportamento clínico das EGIs. O objetivo do estudo foi avaliar diferenças clínicas entre pacientes com EGI não refratária e refratária, assim como avaliar as diferenças entre pacientes com EGI não refratária, refratária e indivíduos saudáveis através de ressonância por DTI e VBM. Avaliamos 40 p
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48

Gomes, Sidcley Pereira. "Localização de Fontes de Descargas Generalizadas em Pacientes com Epilepsia Mioclônica Juvenil." Universidade Federal do Maranhão, 2010. http://tedebc.ufma.br:8080/jspui/handle/tede/441.

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Made available in DSpace on 2016-08-17T14:53:09Z (GMT). No. of bitstreams: 1 Sidcley Pereira Gomes.pdf: 4576085 bytes, checksum: 5969f56106d77776c17330c476ba26de (MD5) Previous issue date: 2010-05-28<br>One important information for the classification of epilepsy is the cortical localization of the discharges source. Juvenile myoclonic epilepsy (JME) is an idiopathic generalized epilepsy (IGE) that typically presents generalized tonic-clonic, myoclonic, or absence seizures, or a combination of these. In typical cases of JME, the seizures are usually bilateral and symmetric, and EEG shows g
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Mechler, Marina Lopes. "Tremor congênito em suínos : o vírus da diarreia viral bovina é um agente etiológico? /." Universidade Estadual Paulista (UNESP), 2018. http://hdl.handle.net/11449/152767.

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Submitted by MARINA LOPES MECHLER null (mlopesvet@gmail.com) on 2018-02-19T14:08:06Z No. of bitstreams: 1 Dissertação_Marina Lopes Mechler.pdf: 2026984 bytes, checksum: ffc0d70577dd42d4b80d5ee121754dbd (MD5)<br>Approved for entry into archive by Alexandra Maria Donadon Lusser Segali null (alexmar@fcav.unesp.br) on 2018-02-20T12:49:32Z (GMT) No. of bitstreams: 1 mechler_ml_me_jabo.pdf: 2026984 bytes, checksum: ffc0d70577dd42d4b80d5ee121754dbd (MD5)<br>Made available in DSpace on 2018-02-20T12:49:32Z (GMT). No. of bitstreams: 1 mechler_ml_me_jabo.pdf: 2026984 bytes, checksum: ffc0d70577dd42d
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Grosse, Pascal. "Diagnostic and experimental applications of cortico-muscular and intermuscular frequency analysis." Doctoral thesis, [S.l.] : [s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=972661360.

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